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Relevant bibliographies by topics / Acid peptic disorder

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Academic literature on the topic 'Acid peptic disorder'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Acid peptic disorder"

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Alfahad, Mohanad, Mahmood H. M. Jasim, Mohannad E. Qazzaz, Fawaz A. Alassaf, and Mohammed N. Abed. "Proton pump inhibitors and peptic ulcer management: Antioxidant mechanisms." Journal of Drug Delivery and Therapeutics 11, no. 4-S (2021): 242–46. http://dx.doi.org/10.22270/jddt.v11i4-s.4955.

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Peptic ulcer (P.U.) is the gastrointestinal tract's most frequent disorder affecting mainly the stomach and duodenum. Surgical intervention, ingested materials and microbial infections may trigger inflammation that further predispose to oxidative stress. Proton pump inhibitors (PPIs) are group of compounds established for suppressions of gastric acid secretions profoundly and permanently over a reasonably long period of time. Oxidative stress has been shown to be involved in the pathophysiology of various diseases and disorders, including P.U. Particularly when H. pylori infection accompanies

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Bandana Kaur, Bandana Kaur, Parul Verma, Dr Ajeet Pal Singh, Dr Amar Pal Singh, and Kiran Bala Kiran Bala. "Peptic Ulcers: Understanding Etiology, Pathophysiology, Diagnosis, Treatment of Peptic Ulcers." International Journal of Pharmaceutical Research and Applications 10, no. 2 (2025): 1172–84. https://doi.org/10.35629/4494-100211721184.

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Peptic ulcer disease (PUD) is a common gastrointestinal disorder characterized by acid-induced lesions in the digestive tract, predominantly affecting the stomach and the first segment of the duodenum. It results from extensive erosion of the mucosal lining, which can progress to the muscular layer due to the corrosive effects of gastric acid. PUD is classified into two main types: gastric ulcers, which occur in the stomach, and duodenal ulcers, which develop in the duodenum. The primary causes of PUD are infection with Helicobacter pylori and the use of non-steroidal anti-inflammatory drugs (

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Komal, Chavan Dr. Kavita Kulkarni Madhavi Kakphale*. "Review Article On Hyperacidity." International Journal of Pharmaceutical Sciences 2, no. 10 (2024): 1192–97. https://doi.org/10.5281/zenodo.13960963.

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Hyperacidity, is refers to as the excessive stomach acid, occurs when the stomach produces an abnormal amount of acid, often leading to symptoms such as heartburn, indigestion, and gastric discomfort. Common causes include diet, stress, smoking, and certain medications. Chronic hyperacidity can result in complications like gastro-esophageal reflux disease (GERD) and peptic ulcers. Management typically involves lifestyle modifications, dietary changes, and medications like antacids, proton pump inhibitors, or H2 blockers to reduce acid production and develop symptoms. Understanding hyperacidity

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Kawaguchi, Takaki, Hiroyuki Fukazawa, Yoshihisa Naito, and Kōsuke Okada. "Dermal dysplasia characterized by collagen disorder-related skin fragility in a cow." American Journal of Veterinary Research 49, no. 6 (1988): 965–71. https://doi.org/10.2460/ajvr.1988.49.06.965.

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SUMMARY Holstein cow 1 was examined because of skin fragility and delayed healing of skin wounds, which were markedly exacerbated around the time of parturition. A skin biopsy sample was obtained, and light microscopy revealed irregular deposition of thin collagen fibers in a dermal matrix. Although diffuse inflammation did not occur, the number of plump fibroblasts was increased. Electron microscopy revealed poor construction of collagen fibrils in the dermal matrix. Biochemical analysis of the dermis revealed a normal amount of collagen and uronic acid, but sodium dodecyl sulfate-polyacrylam

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Manimekalai, K., R. Sudar Codi, Navinraja Komal Veererathinakumar, and Vimala Ananthy. "Potassium-competitive Acid Blocker: A Newer Target in the Treatment of Acid Peptic Disorder." SBV Journal of Basic, Clinical and Applied Health Science 6, no. 2 (2023): 33–38. http://dx.doi.org/10.5005/jp-journals-10082-03182.

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Singh, Karanvir, Rajesh Kumar, Ajeet Pal Singh, Meenakshi Malhotra, Amar Pal Singh, and Ritu Rani. "Peptic Ulcer: A review." International Journal of Medical, Pharmacy and Drug Research 8, no. 2 (2024): 38–46. http://dx.doi.org/10.22161/ijmpd.8.2.5.

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Peptic ulcers, characterized by painful sores that develop on the lining of the stomach, small intestine, or esophagus, are a prevalent gastrointestinal condition affecting millions worldwide. This abstract provides an overview of the causes, symptoms, and treatment options for peptic ulcers. Peptic ulcers primarily stem from the imbalance between aggressive factors such as gastric acid secretion, pepsin, and Helicobacter pylori infection, and defensive mechanisms like mucosal barrier integrity and blood flow. Lifestyle factors such as excessive alcohol consumption, smoking, and stress, along

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Deshpande, Mangirish, Pearl Dighe, and Sujit Nagare. "A Review of Antiulcer Activity of Some Medicinal Plants." Journal of Drug Delivery and Therapeutics 14, no. 8 (2024): 201–13. http://dx.doi.org/10.22270/jddt.v14i8.6722.

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Peptic ulcer is a gastrointestinal disorder and with increased prevalence. Peptic ulcer is breaking of endothelial lining of stomach and exposing underlying tissues. Peptic ulcer occurs due to high secretion of acid and reduced defensive factors in stomach and duodenum. It is imbalance between aggressive and defensive factors. Non-steroidal anti-inflammatory drugs and Helicobacter pylori infection also increases the risk of peptic ulcer. Indiscriminate use of synthetic drugs leads to adverse effects and concomitant use of antibiotics potentiates drug-drug interaction thus search of drugs from

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Azyenela, Lola, Ringga Novelni, and Viora Lusiana Satri. "A Study of Potential Drug-Drug Interactions in Peptic Ulcer Patients in Dr. M. Djamil Hospital Padang in 2019." Journal of Pharmaceutical Sciences and Community 20, no. 2 (2023): 144–50. http://dx.doi.org/10.24071/jpsc.003909.

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A peptic ulcer is a gastrointestinal tract disorder caused by excessive secretion of acid and pepsin by the gastric mucosa. The combined use of drugs in peptic ulcer management may potentially cause drug-to-drug interactions. Drug interactions are caused when two or more drugs interact and may affect the effectiveness of the other drugs. This study aims to analyze the potential drug reactions in peptic ulcer patients in the internal medicine ward of Dr. M. Djamil General Hospital Padang in 2019. This study was a descriptive, non-experimental study with retrospective data, while data analysis u

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Vandana1*, Pooja Solanki2 Ankit Dahiya3 Rajashekar Kamalla4 and Varun Kumar Sarkar5. "" Zollinger-Ellison syndrome in dogs "." Trends In Agriculture Science 2, no. 7 (2023): 541–45. https://doi.org/10.5281/zenodo.8184375.

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<strong>Zollinger-Ellison syndrome </strong>is a rare disorder caused by a tumor called a gastrinoma. The tumor secretes the hormone gastrin, which causes excess production of gastric acid, leading to severe recurrent ulcers of the esophagus, stomach, duodenum, and jejunum. The syndrome is characterized by severe peptic ulcer disease caused by gastrin-secreting neuroendocrine tumors, most commonly located in the <strong>duodenum and pancreas</strong>. Frequently gastrinomas are not recognized at initial clinical presentation and are often mistreated. Symptoms that should raise suspicion of a g

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Awuchi, Chinaza Godswill, Purabi Saha, Vandana Sonaji Amle, et al. "A Study of Various Medicinal Plants used in Ulcer Treatment: A Review." Journal for Research in Applied Sciences and Biotechnology 2, no. 1 (2023): 234–46. http://dx.doi.org/10.55544/jrasb.2.1.34.

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Many people suffer from ulcer, a disorder of the digestive tract. Simply put, it's an inflamed break in the skin or the mucus membrane lining the digestive tract. Whenever there is a change in the equilibrium, such as increased aggression or decreased mucosal resistance, ulceration develops. Reasons for this could include drug use, poor diet, stress, and other similar factors. Peptic ulcers are a general term for any type of stomach or duodenal ulcer. To develop, peptic ulcers need mucosal defences to be compromised in addition to gastric juice acid and peptic activity. Ulcers can be treated w

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Dissertations / Theses on the topic "Acid peptic disorder"

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Minnie, Jacqueline Louise. "An education intervention on prescribing patterns of drugs for acid-related disorders in a clinic setting : a case study / Jacqueline Louise Minnie." Thesis, North-West University, 2007. http://hdl.handle.net/10394/1562.

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Janse, van Rensburg Hendrika Nicolien. "A longitudinal study of the usage of acid reducing medicine using a medicine claims database / Hendrika Nicolien Janse van Rensburg." Thesis, North-West University, 2007. http://hdl.handle.net/10394/1903.

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Acid-related disorders are common, chronic conditions that have considerable impact on a patient's quality of life. In a study conducted by Majumdar et al. (2003:2411) the prevalence of chronic acid-related disorders was 2.3%. Acid-related disorders represent a major financial consideration with respect to the costs of drug prescribing (Whitaker, 1998:6). Health care cost increases each year. This leads to an increased interest in economic evaluation of health care and medical technologies (Anell & Svarvar, 2000:175). Health care providers no longer make treatment decisions independent of the

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Ramraj, Varun. "Exploiting whole-PDB analysis in novel bioinformatics applications." Thesis, University of Oxford, 2014. http://ora.ox.ac.uk/objects/uuid:6c59c813-2a4c-440c-940b-d334c02dd075.

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The Protein Data Bank (PDB) is the definitive electronic repository for experimentally-derived protein structures, composed mainly of those determined by X-ray crystallography. Approximately 200 new structures are added weekly to the PDB, and at the time of writing, it contains approximately 97,000 structures. This represents an expanding wealth of high-quality information but there seem to be few bioinformatics tools that consider and analyse these data as an ensemble. This thesis explores the development of three efficient, fast algorithms and software implementations to study protein struct

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Van, Rensburg Hendrika Nicolien Janse. "A longitudinal study of the usage of acid reducing medicine using a medicine claims database / H.N. Janse van Rensburg." Thesis, 2007. http://hdl.handle.net/10394/1903.

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Books on the topic "Acid peptic disorder"

1

Arnold, Berstad, ed. Management of peptic ulcer disease and acid-related disorders: Clinical aspects of antacids in the 1990s : Bermuda, November 15-16, 1991. Raven Press, 1992.

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2

Peptic ulcer disease and other acid-related disorders. Academic Research Associates, 1991.

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3

Clearfield, Harris R. Acid-Peptic Disorders of the Upper Gastrointestinal Tract: Diagnosis and Management. Handbooks in Health Care, 1995.

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4

Freston, James W. Diseases of the Gastroesophageal Mucosa: The Acid-Related Disorders. Humana Press, 2012.

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Freston, James W. Diseases of the Gastroesophageal Mucosa: The Acid-Related Disorders. Humana Press, 2001.

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Sedlack, Robert E., Conor G. Loftus, Amy S. Oxentenko, and Thomas R. Viggiano. Gastroenterology. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0210.

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Part 1 reviews the major portions of the gastrointestinal system (esophagus, stomach, small intestine, colon, and pancreas), their function (motility, acid production, enzymatic function, and absorption), and various disorders associated with them (dysmotility, ulceration, malabsorption, inflammation, and dysplasia). Symptoms, diagnostic testing, and treatment of common gastrointestinal conditions, such as gastroesophageal reflux disease, peptic ulcer disease, diarrhea, constipation, inflammatory bowel disease, and pancreatitis, are reviewed.

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Modlin, Irvin M., and George Sachs. Acid Related Diseases: Biology and Treatment. 2nd ed. Lippincott Williams & Wilkins, 2004.

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8

(Editor), Henry Parkman, and Robert S. Fisher (Editor), eds. The Clinician's Guide to Acid/Peptic Disorders and Motility Disorders of the Gastrointestinal Tract (The Clinician's Guide to GI Series). Slack Incorporated, 2006.

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9

Wójcik-Gładysz, Anna. Ghrelin – hormone with many faces. Central regulation and therapy. The Kielanowski Institute of Animal Physiology and Nutrition, Polish Academy of Sciences, 2020. http://dx.doi.org/10.22358/mono_awg_2020.

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Discovered in 1999, ghrelin, is one of the peptides co-creating the hypothalamicgastrointestinal axis, otherwise known as the brain-gut axis. Ghrelin participates in many physiological processes and spectrum of its activity is still being discovered. This 28 amino acid peptide ‒ a product of the ghrl gene, was found in all vertebrates and is synthesized and secreted mainly from enteroendocrine X/A cells located in the gastric mucosa of the stomach. Expression of the ghrelin receptor has been found in many nuclei of the hypothalamus involved in appetite regulation. Therefore it’s presumed that

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Book chapters on the topic "Acid peptic disorder"

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Kovacs, Thomas O. G. "Acid-Peptic Disorders." In Gut Instincts. CRC Press, 2024. http://dx.doi.org/10.1201/9781003524489-11.

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Lakshmanan, Mageshwaran. "Drugs Used in Acid Peptic Disorders." In Introduction to Basics of Pharmacology and Toxicology. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-33-6009-9_34.

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Daneshmend, Tawfique, and Alasdair Dow. "Gastroenterological disorders." In Perioperative Medicine. Oxford University PressOxford, 2006. http://dx.doi.org/10.1093/oso/9780199211739.003.0030.

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Abstract A history of acid-peptic ulceration of the upper digestive tract affecting the duodenum, stomach, or oesophagus will often be obtained in patients presenting for elective or emergency surgery unrelated to the upper GI tract. Surgery for acid-peptic disease itself (elective surgery for peptic ulcer, gastro-oesophageal reflux disease; emergency surgery for perforated peptic ulcer or oesophagus; or surgery for acute GI bleeding) is not the purpose of this chapter. The management of these patients is addressed in standard surgical texts. Chronic peptic ulceration should now be rare. Since

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"Acid Peptic Disorders." In Yamada's Handbook of Gastroenterology. John Wiley & Sons, 2013. http://dx.doi.org/10.1002/9781118572610.ch21.

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Qadeer, Mohammed A., and Gary W. Falk. "Acid Peptic Disorders." In Current Clinical Medicine. Elsevier, 2010. http://dx.doi.org/10.1016/b978-1-4160-6643-9.00065-5.

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Vikraman Pillai, R. "Acid Peptic Disease." In PG Textbook of Pediatrics: Infections and Systemic Disorders (Volume 2). Jaypee Brothers Medical Publishers (P) Ltd., 2015. http://dx.doi.org/10.5005/jp/books/12854_100.

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Aridome, Kuniaki. "Benign diseases of the stomach." In Oesophagus and Stomach, edited by Matthias Reeh, Jakob R. Izbicki, Samiran Nundy, and Dirk J. Gouma. Oxford University PressOxford, 2023. http://dx.doi.org/10.1093/med/9780192863591.003.0005.

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Abstract Most benign diseases of the stomach are attributed to functional dyspepsia and acid-related disorders such as peptic ulcer disease. Dyspepsia is also the main manifestation of these acid-related conditions of the stomach. Functional dyspepsia comprises three subtypes: postprandial distress syndrome, epigastric pain syndrome, and a subtype with overlapping postprandial distress syndrome and epigastric pain syndrome features. Peptic ulcer disease is characterized by a non-malignant lesion in the gastrointestinal mucosa that may be acute, subacute, or chronic. Proton pump inhibitors sign

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Boarder, Michael, Jane Dixon, David Newby, Phyllis Navti, and Tyra Zetterström. "Upper gastrointestinal tract disorders." In Pharmacology for Pharmacy and the Health Sciences. Oxford University Press, 2016. http://dx.doi.org/10.1093/9780198728832.003.0012.

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This chapter focuses on upper gastrointestinal tract disorders. The commonly experienced symptoms of heartburn, acid regurgitation, and indigestion (dyspepsia) arise from disturbances of acid secretion in the stomach and the consequent damage to the mucosal lining of the oesophagus, stomach, or duodenum. Conditions affecting the upper gastrointestinal tract, like gastro-oesophageal reflux disease and peptic ulcer disease, are among the most prevalent illnesses in Western countries. The chapter discusses the causes and significance of nausea and vomiting. It cites that the main targets for anti

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"Gastric Physiology and Acid-Peptic Disorders." In Modern Surgical Care. CRC Press, 2006. http://dx.doi.org/10.3109/9781420016581-19.

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Garg, Dr Gopal. "Antiulcer Agents." In Edited Book of Pharmacology-III [According to Latest Syllabus of B. Pharm-VI Semester of Pharmacy Council of India]. Iterative International Publishers, Selfypage Developers Pvt Ltd, 2024. http://dx.doi.org/10.58532/nbennurphch2.

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Antiulcer agents are essential medications used to manage and treat conditions like peptic ulcers, gastroesophageal reflux disease (GERD), and other acid-related disorders of the gastrointestinal tract. These agents are classified into several categories based on their mechanisms of action: proton pump inhibitors (PPIs), H2-receptor antagonists (H2RAs), antacids, mucosal protective agents, antibiotics, and prostaglandin analogues. PPIs, such as omeprazole and lansoprazole, work by irreversibly inhibiting the hydrogen-potassium ATPase enzyme, significantly reducing gastric acid secretion. H2RAs

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Conference papers on the topic "Acid peptic disorder"

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Bauer, K. A., B. L. Kass, M. Bednarek, M. Kloczewiak, J. Hawiger, and R. D. Rosenberg. "DETECTION OF FACTOR X ACTIVATION IN HUMANS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643828.

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The activation of factor X by factor VII/VIIa-tissue factor or factor IXa plays a pivotal role in the hemostatic mechanism. This reaction results in the liberation of a peptide from the zymogen for which we have developed a sensitive and specific radioimmunoassay (RIA), The native peptide was purified from activated human factor X by hydroxylapatite chromatography and reverse-phase high pressure liquid chromatography (HPLC). Gel filtration experiments demonstrated that the peptide was not physically associated with the enzyme. A 15 amino acid peptide with the COOH-terminal sequence of the acti

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Kibitkina, A. A., E. R. Vasilevskaya, N. A. Ilin, and G. S. Tolmacheva. "CHANGES IN MILK OF TPH2 HET MICE UNDER EXPOSURE TO STRESS." In NOVEL TECHNOLOGIES IN MEDICINE, BIOLOGY, PHARMACOLOGY AND ECOLOGY. LLC Institute Information Technologies, 2024. http://dx.doi.org/10.47501/978-5-6044060-4-5.41-46.

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The relationship between neuroregulatory factors of the brain and the composition of breast milk is an interesting and little-studied topic. The article is aimed at studying changes in the chemical composition of the milk of mice prone to the development of affective disorders (heterozygous for TPH2) under the influence of stress and without stress relative to the milk of ordinary mice. Raman spectroscopy (wavelength 785 nm, focal length 50x) was used for the study. Het mouse milk has a less rich protein and peptide profile, but the range of fatty acids, especially under stress, increases.

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Müller, E., and A. Henschen. "HUMAN PLASMA FIBRINOGEN MOLECULAR WEIGHT VARIANTS:CHARACTERIZATION BY HIGH-PERFORMANCE LIQUID CHROMATOGRAPHY AND IDENTIFICATION BY SEQUENCE ANALYSIS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643328.

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Human plasma has been shown to contain several fibrinogen variants which differ from each other in molecular weight. The three most common forms, with molecular weights of 340 kD, 305 kD and 270 kD, have been reported to change considerably in their relative amounts during certain pathophysiological processes such as acute phase reactions, disseminated intravascular coagulation, fibrinolytic disorders, liver disease and cancer. Though it has been suggested that the differences in molecular weight are due to degradation of one or both, respectively, of the carboxy-terminal parts of the Aα-chain

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Lazarević, Marija. "COGNITIVE, HEMATOPOIETIC AND GASTROINTESTINAL IMPAIRMENTS CAUSED BY VITAMIN B12 DEFICIENCY – PATOGENESIS, TREATMENT AND PATIENT CARE." In Global Challenges Through the Prism of Rural Development in the Sector of Agriculture and Tourism GIRR 2024. Academy of Applied Studies Šabac, 2025. https://doi.org/10.46793/girr25.287l.

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Many studies have investigated the pathogenetic association of cognitive, hematopoietic, and gastrointestinal impairments with low vitamin B12 concentrations. The aim of our study is to point out the importance of maintaining vitamin B12 concentrations in the body, in order to preserve cognition, but also other vital parameters of the body. For a systematic literature search on studies on the association of cognitive, hematopoietic and gastrointestinal impairments with vitamin B12 deficiency, we used we used the databases: Web of Science, PubMed, Google Scholar. The search keywords included “v

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Askew, Michael J., Gary B. Schneider, Kristina J. Grecco, Jason Hsu, Emily Mugler, and Donald A. Noe. "Effect of Pharmaceutical Bone Growth Stimulation With Novel Anabolic Peptides: Biomechanical and Bone Density Measurements in a Rat Model." In ASME 2003 International Mechanical Engineering Congress and Exposition. ASMEDC, 2003. http://dx.doi.org/10.1115/imece2003-43044.

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Pharmaceutical bone growth stimulation holds promise for prevention and treatment bone disorders, and the enhancement of fracture healing. Bone growth hormones have begun to have limited clinical use, but can illicit adverse side effects. Recent studies have shown that short peptides (less than 15 amino acids) derived from the protein sequence of Vitamin D Binding Protein (DBP), can enhance bone formation (osteogenesis). These peptides may have potential as controllable bone growth stimulators without the adverse side effects and cost of bone growth hormones. Rats, injected every other day for

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Asen, Nancy, and Rotimi Aluko. "Functional Properties of Enzymatic Pea Protein Hydrolysates That Inhibit in vitro Activities of Acetylcholinesterase and Butyrylcholinesterase." In 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/ktht4252.

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Overview: Alzheimer’s disease (AD) is a neurodegenerative disorder prevalent among the aged population with morbidity and mortality rate of ~ 12%. Research has linked the cause of this disorder to the loss of acetylcholine through excessive activities of acetylcholinesterase (AChE) and butyrylcholinesterase (BChE). Therefore, a promising therapeutic approach for AD treatment is the inhibition of AChE/BChE activities. Common features of an AD brain include low levels of acetylcholine, the presence of amyloid-β peptides deposits and severe oxidative stress triggered by lipid peroxidation and for

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Scheefers-Borchel, U., and G. Muller-Berghaus. "A NEW FIBRIN-SPECIFIC ANTIBODY DISCRIMINATING BETWEEN FIBRIN AND FIBRINOGEN IS DIRECTED AGAINST THE SYNTHETIC PEPTIDE LEU-ILE-ASP-GLY-LYS-MET." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643771.

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To determine soluble fibrin in blood of patients with coagulation disorders we produced monoclonal antibodies which distinct fibrin from fibrinogen and other blood constituents. Fibrin-specific monoclonal antibodies were obtained by immunizing mice with the synthetic hexapeptide Leu-Ile-Asp-Gly-Lys-Met which was covalently linked to KLH via its C-terminus. Several of the monoclonal antibodies which reacted with the hexapeptide also reacted with batroxobin-induced desAA-fibrin and thrombin-induced desAABB-fibrin, but not with fibrinogen. No reaction was observed with plasmin-induced fibrinogeno

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Antonarakis, E. "The Molecular Genetics of Hemophilia A Stylianos." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643980.

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Hemophilia A is a common X linked hereditary disorder of blood coagulation due to deficiency of factor 8. The gene for factor 8 has been cloned and characterized (Nature 312:326-342, 1984). It is divided into 26 exons and 25 introns and spans 186 kb of DNA. The CGNA is 9 kb and codes for 2351 amino acids. The first 19 amino acids comprise the secretory leader peptide and the mature excreted polypeptide consists of 2332 amino acids. The nucleotide sequence of the exons and the exon-intron junctions is known and the complete amino acid sequence has been deducedSeveral laboratories have used clon

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Homes, W. E., H. R. Lijnen, L. Nelles, C. Kluft та D. Collen. "AN ALANINE INSERTION IN α2-ANTIPLASMIN ‘ENSCHEDE’ ABOLISHES ITS PLASM IN INHIBITORY ACTIVITY". У XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642897.

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Congenital deficiencies of the fibrinolytic inhibitor α2antiplasmin (α2AP) may result in bleeding disorders. An abnormal a AP (α2AP‘Enschede’) is known. 2 siblings with 3% functional activity and normal antigen level have parents with 50% activity and normal antigen. The protein interacts normally with the lysine-binding site(s) of plasmin(ogen) but does not inhibit plasmin irreversibly. α2AP Enschede is a plasmin substrate that like the normal protein releases a M 8,000 peptide upon reaction with plasmin. In the present study, Southern blot analysis, using an α2AP cDNA probe showed a restrict

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