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1

Québec. Ministère de L'éducation. Direction des Politiques et Plans. Reconnaissance des Acquis de Formation: Repères Actuels. Éducation. S.l: s.n, 1985.

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2

Sansregret, Marthe. La reconnaissance des acquis: Portfolio. LaSalle, Qué: Hurtubise HMH, 1988.

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3

Ansart, Sandrine. La VAE [validation des acquis de l'expérience]: Un outil de développement des compétences. Paris: Dunod, 2010.

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4

Centre européen pour le développement de la formation professionnelle. Développement Régional et Formation Professionnelle: Analyse et Animation de la Relation Développement : Formation Professionnelle: Acquis et Perspectives. S.l: s.n, 1986.

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5

Robin, Ginette. Guide en reconnaissance des acquis: Plus qu'un c.v., un portfolio de ses apprentissages. 3rd ed. Boucherville, Qué: Vermette, 1988.

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6

Colloque "La Reconnaissance des acquis" (1988 Moncton, N.-B.). Les actes du Colloque "La Reconnaissance des acquis": 27-28-29 mai 1988. Ottawa, Ont: Réseau national d'action éducation femmes, 1988.

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7

Sansregret, Marthe. La reconnaissance des acquis: Cours sur l'élaboration d'un portfolio. LaSalle, Qué: Hurtubise HMH, 1988.

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8

La reconnaissance des acquis: Fonctions et tâches des administrateurs, conseillers et évaluateurs. Montréal, Qué: Hurtubise HMH, 1988.

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9

Sansregret, Marthe. La reconnaissance des acquis: Fonctions et tâches des administrateurs, conseillers et évaluateurs. Montréal: Hurtubise HMH, 1988.

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10

Alberta Council on Admissions and Transfer. Business plan, 1999-2002. Edmonton: The Council, 1999.

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11

Lucas, Raoul. Etude sur la reconnaissance et la validation des acquis à la Réunion: Étude réalisée par le Département études et récherches du CAFOC (Centre académique de formation continue). Sainte Clotilde [Réunion]: Le Département, 1989.

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12

Pierre, Michard, ed. Le Bilan personnel et professionnel: Instrument de management. Paris: ESF Editeur, 1991.

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13

Ontario. Le curriculum de l'Ontario 11e et 12e année: Orientation et formation au cheminement de carrière. Toronto, Ont: Imprimeur de la Reine, 2000.

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14

Boffo, Vanna, ed. A Glance at Work. Florence: Firenze University Press, 2012. http://dx.doi.org/10.36253/978-88-6655-187-4.

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The topics of work flexibility, precarious jobs, and the relationship between work, the market and production are subjects that are widely debated in the sociological, philosophical, economic and political spheres. Yet these topics are less touched on in the tradition of pedagogical research. The intention of this book is to build a seedbed for reflection on the central position assumed by work in the lives of every woman and man, inhabitants of a planet in which the transformation of work activities is imposing radical changes on lifestyles, community-building and societies. Work is not an abstract concept, but is incorporated into every human person who does it and into the relationships linking them to others. Man, his education and human formation provide the pivot around which to perform a pedagogical survey within the universe of "work", and inside the relationship between the human condition and working/professional life. What sense does work acquire today when going to observe children, young people, adults or migrants? Namely, what sense does it assume when its pivotal viewpoint is shifted off-centre in time and space? The essays intend to spark agile but critical, synchronic and diachronic reflection which, stemming from contextual questions on the meaning of work and on change in the workplace, will proceed to investigate the subjects in their specific lives and existential conditions. Essays by: Vanna Boffo, Pietro Causarano, Giovanna Del Gobbo, Emiliano Macinai, Maria Rita Mancaniello, Stefano Oliviero and Clara Silva.
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15

Farzad, Mehdi, and Saeed Paivandi. Reconnaissance et validation des acquis en formation. Economica, 2000.

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16

Francine, Landry, and Simosko Susan 1942-, eds. La Reconnaissance des acquis: Manuel de l'évaluateur. Mont-Royal, Qué: Modulo, 1989.

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17

L'Europe et la formation professionnelle des jeunes: Transferer les acquis de PETRA dans LEONARDO et Emploi/Youthstart. Diffusion, Documentation francaise, 1995.

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18

L' Europe et la formation professionnelle des jeunes: Transférer les acquis de PETRA dans LEONARDO et Emploi/Youthstart. Paris: Racine éditions, 1995.

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19

Schine, Joan. Service Learning (National Society for the Study of Education Yearbooks). The National Society For The Study Of Ed, 1997.

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20

Vanzo, Alberto. Kant and Abstractionism about Concept Formation. Oxford University Press, 2017. http://dx.doi.org/10.1093/acprof:oso/9780190608040.003.0014.

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This chapter outlines Kant’s account of empirical concept formation and discusses two objections that have been advanced against it. Kant holds that we form empirical concepts, such as color concepts, by comparing sensory representations of individuals, identifying shared features, and abstracting from the differences between them. According to the first objection, we cannot acquire color concepts in this way because there is no feature that all and only the instances of a given color share, and because the boundary between colors is conventional. According to the second objection, assuming that all instances of a given color share certain features, we can identify them only if we already possess a concept of that color. Neither of the objections is convincing as it stands. Kant can offer replies to both objections that are consistent with his views and with empirical evidence concerning the perception and representation of colors.
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21

Takemoto, Hiroyuki, Yoshi Kawamoto, and Takeshi Furuichi. The formation of Congo River and the origin of bonobos: A new hypothesis. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198728511.003.0016.

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The Congo River functions as a strong geographical barrier for many terrestrial mammals in the Congo Basin, separating forest habitat into right and left banks of the river. However, there has been little discussion on the biogeography of the Congo Basin because the history of the river has been obscured. Based on the recent information of the sea-floor sediments near the mouth of the river and the geophysical survey on the continent, this chapter proposes a plausible hypothesis on the Congo River formation and presents a consequent hypothesis on the divergence of bonobos (Pan paniscus) from other Pan populations. The present hypothesis is also helpful for understanding the distribution of other primates and other mammals in the basin. Furthermore, this hypothesis suggests that all hominid clades, including human, chimpanzee and gorilla, except bonobo, evolved in the area north or east of the Congo River. La rivière du Congo a la fonction d’une barrière géographique forte pour plusieurs mammifères dans le bassin du Congo, séparant l’habitat forêt dans les banques gauches et droites de la rivière. Cependant, il y a eu peu de discussions sur la biogégraphie du bassin du Congo, parce que l’histoire de la rivière a été voilée. Récemment, quelque données importantes qui peuvent avoir des liens avec la formation de la rivière du Congo ont été acquise, surtout par la recherche des sédiments du fond marin près de la bouche de la rivière et par l’enquête géographique du continent. À partir de cette nouvelle information, nous avons proposé une hypothèse plausible sur la formation de la rivière du Congo. Nous avons aussi présenté une hypothèse conséquente sur la divergence des bonobos (Pan paniscus) des autres populations Pan (voire Takemoto et al., 2015 pour la publication originale de cette étude). L’hypothèse présente nous aide aussi à comprendre la distribution des autre primates et des autres mammifères dans le bassin. De plus, cette hypothèse suggère que tous les hominidés clades, humains inclus, chimpanzés et gorilla à l’exception du bonobo, ont évolué dans la régions du nord ou de l’est de la rivière du Congo.
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22

Auyoung, Elaine. Enduring Minds in Austen. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190845476.003.0003.

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This chapter draws on social psychological models of impression formation to show how Pride and Prejudice enables readers to acquire uncommonly durable mental models of its characters. This makes it possible to explain why Jane Austen’s readers have sometimes claimed that even flat characters devoid of psychological complexity can seem as lifelike and familiar as actual friends. The chapter also presents a new way to account for another distinctive feature of Austen’s reception history, which is the tendency for fans of Austen’s novels to display a possessive desire for friendship with the author herself. From a sociolinguistic perspective, Austen’s ironic, impersonal, and indirect style of narration permits readers to feel as if they share private rapport with the implied author.
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23

Fischer-Lichte, Erika. Only with Beauty Man Shall Play. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780199651634.003.0003.

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Chapter 1, ‘Only with Beauty Man Shall Play. Goethe’s Production of Ion in Weimar (1802)’, proceeds from Goethe’s and Schiller’s responses to the French Revolution. While Goethe hailed the Bildung of the individual—that is, the development of his potential to the full—as the substitute for a revolution, Schiller believed that it was the aesthetic education of the individual that would finally result in a free state. The production of a Greek tragedy as an autonomous work of art that precluded the formation of empathy in the spectator (contrary to the domestic tragedy) was supposed to offer the spectator the possibility of aesthetic distance and thus enable him to acquire Bildung. To this end, Goethe developed a completely new aesthetics that the majority of spectators rejected—Ion turned out to be a flop.
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24

Aquino, Frederick D. Maximus the Confessor. Edited by William J. Abraham and Frederick D. Aquino. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780199662241.013.18.

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The current landscape of virtue epistemology is ripe with possibilities for theological engagement and appropriation. Constructively speaking, Maximus the Confessor (580–662 ce) is a fitting example of this kind of intersection. In terms of mapping the cognitive economy of the spiritual life, he draws attention to virtuous and contemplative practices that enable the intellect to attain its proper end (divine likeness) and acquire the related epistemic goods. Accordingly, this chapter shows how the virtues, for Maximus, contribute to the formation of a deep and abiding desire for the relevant epistemic goods (e.g. contemplation of God in and through nature, illumination of divine truths, wisdom, and perceptual knowledge of God) as well as playing a supportive role in the pursuit of them. It also offers briefly some concluding reflections concerning Maximus’s pairing of virtue and knowledge, and identifies a few areas of enquiry that warrant further work and development.
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25

Chinca, Mark. Meditating Death in Medieval and Early Modern Devotional Writing. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780198861980.001.0001.

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Meditating about death and the afterlife was one of the most important techniques that Christian societies in medieval and early modern Europe had at their disposal for developing a sense of individual selfhood. Believers who regularly and systematically reflected on the inevitability of death and the certainty of eternal punishment in hell or reward in heaven would acquire an understanding of themselves as unique persons defined by their moral actions; they would also learn to discipline themselves by feeling remorse for their sins, doing penance, and cultivating a permanent vigilance over their future thoughts and deeds. The book covers a crucial period in the formation and transformation of the technique of meditating on death: from the thirteenth century, when a practice that had mainly been the preserve of a monastic elite began to be more widely disseminated among all segments of Christian society, to the sixteenth, when the Protestant Reformation transformed the technique of spiritual exercise into a Bible-based mindfulness that avoided the stigma of works piety. The book discusses the textual instructions for meditation as well as the theories and beliefs and doctrines that lay behind them; the sources are Latin and vernacular and enjoyed widespread circulation in Roman Christian and Protestant Europe during the period under consideration.
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26

Gurukkal, Rajan. History and Theory of Knowledge Production. Oxford University Press, 2019. http://dx.doi.org/10.1093/oso/9780199490363.001.0001.

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This book seeks to provide an introductory outline of the history and theory of knowledge production, notwithstanding the vastness of the subject. It is to try and do a history of intellectual formation or history of ideas. One can see it as a textbook of historical epistemology, which in spatio-temporal terms historicizes knowledge production and contextualizes methodological development. It addresses itself as the historical process of the social constitution of knowledge, that is, the social history of the making of knowledge. Its objective is to make researchers of knowledge knowledgeable about the significant elements that underlie the history of knowledge. These elements constitute contemporary compulsions that make, shape, and regulate knowledge. Understanding what they mean and how they work is essential to prepare researchers as self-consciously realistic about the socio-economic and cultural process of knowledge production. What forces engender knowledge, how certain forms of it acquire precedence over the rest, and why are questions examined. Who decides what knowledge means or what should be recognized as knowledge becomes important here. We confine the discussion of knowledge systems to the broad heads, namely, the non-European, specifically the Indian and the European. Examining the process of the rise of science and new science, the book ends up reviewing speculative thoughts and imagination about the dynamics of subatomic micro-universe as well as the mechanics of the galactic macro-universe.
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27

Badimon, Lina, and Gemma Vilahur. Atherosclerosis and thrombosis. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0040.

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Atherosclerosis is the main underlying cause of heart disease. The continuous exposure to cardiovascular risk factors induces endothelial activation/dysfunction which enhances the permeability of the endothelial layer and the expression of cytokines/chemokines and adhesion molecules. This results in the accumulation of lipids (low-density lipoprotein particles) in the extracellular matrix and the triggering of an inflammatory response. Accumulated low-density lipoprotein particles suffer modifications and become pro-atherogenic, enhancing leucocyte recruitment and further transmigration across the endothelium into the intima. Infiltrated monocytes differentiate into macrophages which acquire a specialized phenotypic polarization (protective or harmful), depending on the stage of the atherosclerosis progression. Once differentiated, macrophages upregulate pattern recognition receptors capable of engulfing modified low-density lipoprotein, leading to foam cell formation. Foam cells release growth factors and cytokines that promote vascular smooth muscle cell migration into the intima, which then internalize low-density lipoprotein via low-density lipoprotein receptor-related protein-1 receptors. As the plaque evolves, the number of vascular smooth muscle cells decline, whereas the presence of fragile/haemorrhagic neovessels increases, promoting plaque destabilization. Disruption of this atherosclerotic lesion exposes thrombogenic surfaces that initiate platelet adhesion, activation, and aggregation, as well as thrombin generation. Both lipid-laden vascular smooth muscle cells and macrophages release the procoagulant tissue factor, contributing to thrombus propagation. Platelets also participate in progenitor cell recruitment and drive the inflammatory response mediating the atherosclerosis progression. Recent data attribute to microparticles a potential modulatory effect in the overall atherothrombotic process. This chapter reviews our current understanding of the pathophysiological mechanisms involved in atherogenesis, highlights platelet contribution to thrombosis and atherosclerosis progression, and provides new insights into how atherothrombosis may be modulated.
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28

Badimon, Lina, and Gemma Vilahur. Atherosclerosis and thrombosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0040_update_001.

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Atherosclerosis is the main underlying cause of heart disease. The continuous exposure to cardiovascular risk factors induces endothelial activation/dysfunction which enhances the permeability of the endothelial layer and the expression of cytokines/chemokines and adhesion molecules. This results in the accumulation of lipids (low-density lipoprotein particles) in the intimal layer and the triggering of an inflammatory response. Accumulated low-density lipoprotein particles attached to the extracellular matrix suffer modifications and become pro-atherogenic, enhancing leucocyte recruitment and further transmigration across the endothelium into the intima. Infiltrated pro-atherogenic monocytes (mainly Mon2) differentiate into macrophages which acquire a specialized phenotypic polarization (protective/M1 or harmful/M2), depending on the stage of the atherosclerosis progression. Once differentiated, macrophages upregulate pattern recognition receptors capable of engulfing modified low-density lipoprotein, leading to foam cell formation. Foam cells release growth factors and cytokines that promote vascular smooth muscle cell migration into the intima, which then internalize low-density lipoproteins via low-density lipoprotein receptor-related protein-1 receptors becoming foam cells. As the plaque evolves, the number of vascular smooth muscle cells decline, whereas the presence of fragile/haemorrhagic neovessels and calcium deposits increases, promoting plaque destabilization. Disruption of this atherosclerotic lesion exposes thrombogenic surfaces rich in tissue factor that initiate platelet adhesion, activation, and aggregation, as well as thrombin generation. Platelets also participate in leucocyte and progenitor cell recruitment are likely to mediate atherosclerosis progression. Recent data attribute to microparticles a modulatory effect in the overall atherothrombotic process and evidence their potential use as systemic biomarkers of thrombus growth. This chapter reviews our current understanding of the pathophysiological mechanisms involved in atherogenesis, highlights platelet contribution to thrombosis and atherosclerosis progression, and provides new insights into how atherothrombosis may be prevented and modulated.
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29

Badimon, Lina, and Gemma Vilahur. Atherosclerosis and thrombosis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0040_update_002.

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Abstract:
Atherosclerosis is the main underlying cause of heart disease. The continuous exposure to cardiovascular risk factors induces endothelial activation/dysfunction which enhances the permeability of the endothelial layer and the expression of cytokines/chemokines and adhesion molecules. This results in the accumulation of lipids (low-density lipoprotein particles) in the intimal layer and the triggering of an inflammatory response. Accumulated low-density lipoprotein particles attached to the extracellular matrix suffer modifications and become pro-atherogenic, enhancing leucocyte recruitment and further transmigration across the endothelium into the intima. Infiltrated pro-atherogenic monocytes (mainly Mon2) differentiate into macrophages which acquire a specialized phenotypic polarization (protective/M1 or harmful/M2), depending on the stage of the atherosclerosis progression. Once differentiated, macrophages upregulate pattern recognition receptors capable of engulfing modified low-density lipoprotein, leading to foam cell formation. Foam cells release growth factors and cytokines that promote vascular smooth muscle cell migration into the intima, which then internalize low-density lipoproteins via low-density lipoprotein receptor-related protein-1 receptors becoming foam cells. As the plaque evolves, the number of vascular smooth muscle cells decline, whereas the presence of fragile/haemorrhagic neovessels and calcium deposits increases, promoting plaque destabilization. Disruption of this atherosclerotic lesion exposes thrombogenic surfaces rich in tissue factor that initiate platelet adhesion, activation, and aggregation, as well as thrombin generation. Platelets also participate in leucocyte and progenitor cell recruitment are likely to mediate atherosclerosis progression. Recent data attribute to microparticles a modulatory effect in the overall atherothrombotic process and evidence their potential use as systemic biomarkers of thrombus growth. This chapter reviews our current understanding of the pathophysiological mechanisms involved in atherogenesis, highlights platelet contribution to thrombosis and atherosclerosis progression, and provides new insights into how atherothrombosis may be prevented and modulated.
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