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Journal articles on the topic 'Acrylamide Genotoxicity'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Salimi, Ahmad, Elahe Baghal, Hassan Ghobadi, Niloufar Hashemidanesh, Farzad Khodaparast, and Enayatollah Seydi. "Mitochondrial, lysosomal and DNA damages induced by acrylamide attenuate by ellagic acid in human lymphocyte." PLOS ONE 16, no. 2 (2021): e0247776. http://dx.doi.org/10.1371/journal.pone.0247776.

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Acrylamide (AA), is an important contaminant formed during food processing under high temperature. Due to its potential neurotoxicity, reproductive toxicity, hepatotoxicity, immunotoxicity, genotoxicity and carcinogenicity effects, this food contaminant has been recognized as a human health concern. Previous studies showed that acrylamide-induced toxicity is associated with active metabolite of acrylamide by cytochrome P450 enzyme, oxidative stress, mitochondrial dysfunction and DNA damage. In the current study, we investigated the role of oxidative stress in acrylamide’s genotoxicity and ther

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2

Besaratinia, A., and G. P. Pfeifer. "Genotoxicity of Acrylamide and Glycidamide." JNCI Journal of the National Cancer Institute 96, no. 13 (2004): 1023–29. http://dx.doi.org/10.1093/jnci/djh186.

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3

Klaunig, James, and Jerry Rice. "Genotoxicity and carcinogenicity of acrylamide." Toxicology Letters 189 (September 2009): S41. http://dx.doi.org/10.1016/j.toxlet.2009.06.130.

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4

Blasiak, Janusz, Ewa Gloc, Katarzyna Wozniak, and Agnieszka Czechowska. "Genotoxicity of acrylamide in human lymphocytes." Chemico-Biological Interactions 149, no. 2-3 (2004): 137–49. http://dx.doi.org/10.1016/j.cbi.2004.08.002.

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5

Abdelaziz, E. Ibrahim, A. El Kareem Rania, and A. Sheir Marwa. "Elucidation of Acrylamide Genotoxicity, Neurotoxicity, and the Protective Role of Gallic Acid and Green Tea." Global Animal Science Journal 1, no. 4 (2014): 69–77. https://doi.org/10.5281/zenodo.25849.

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Acrylamide is a chemical compound used in many technological applications and can be formed naturally when foods, especially those are rich in sugars and low in protein cooked at high temperatures during  (e.g., frying, grilling , baking or toasting ) . It has several harmful health effects including neurotoxicity, carcinogenicity, reproductive toxicity, genotoxicity, and mutagenicity. Humans have chronic contact with acrylamide through eating, e.g., fried potato chips and/or French fries; cereal products, including bread, breakfast cereals, cakes and biscuits; as well as, roasted coffee

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6

Iyer, Aditya Manivannan, Vedika Dadlani, and Harshal Ashok Pawar. "Review on Acrylamide: A Hidden Hazard in Fried Carbohydrate-rich Food." Current Nutrition & Food Science 18, no. 3 (2022): 274–86. http://dx.doi.org/10.2174/1573401318666220104124753.

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Abstract: Acrylamide is classified as a hazard whose formation in carbohydrate-rich food cooked at a high temperature has created much interest in the scientific community. The review attempts to comprehend the chemistry and mechanisms of formation of acrylamide and its levels in popular foods. A detailed study of the toxicokinetics and biochemistry, carcinogenicity, neurotoxicity, genotoxicity, interaction with biomolecules, and its effects on reproductive health has been presented. The review outlines the various novel and low-cost conventional as well as newer analytical techniques for the

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7

Sarion, Cristina, Georgiana Gabriela Codină, and Adriana Dabija. "Acrylamide in Bakery Products: A Review on Health Risks, Legal Regulations and Strategies to Reduce Its Formation." International Journal of Environmental Research and Public Health 18, no. 8 (2021): 4332. http://dx.doi.org/10.3390/ijerph18084332.

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Acrylamide is a contaminant as defined in Council Regulation (EEC) No 315/93 and as such, it is considered a chemical hazard in the food chain. The toxicity of acrylamide has been acknowledged since 2002, among its toxicological effects on humans being neurotoxicity, genotoxicity, carcinogenicity, and reproductive toxicity. Acrylamide has been classified as carcinogenic in the 2A group, with human exposure leading to progressive degeneration of the peripheral and central nervous systems characterized by cognitive and motor abnormalities. Bakery products (bread, crispbread, cakes, batter, break

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8

Venkatasubbaiah. "Analysis of Micronuclei Formation is an Indicator of Genotoxicity Assessment to Acrylamide in Rats and Developing Chick Embryos." Biolife 1, no. 2 (2022): 35–44. https://doi.org/10.5281/zenodo.7185294.

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<strong>ABSTRACT</strong>   The formation of micronuclei (MN) is a widely used and accepted endpoint of genotoxicity testing. This assay provides a simple and direct measure of the induction of structural or numerical aberrations to chromosomes. In this study we describe about acrylamide treated rats and chick eggs with different doses like 0.1, 0.2 and 0.3mgs to eggs and 16, 32, 48 mgs to rats for 24,48 and 72hrs, for the detection of micronucleus formation in reticulocytes. The Acrylamide treatment to rats and chick embryo caused damage not only to peripheral blood cells and also to ret

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9

Erkekoğlu, Pınar, and Terken Baydar. "Toxicity of acrylamide and evaluation of its exposure in baby foods." Nutrition Research Reviews 23, no. 2 (2010): 323–33. http://dx.doi.org/10.1017/s0954422410000211.

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Contaminants are a vast subject area of food safety and quality and can be present in our food chain from raw materials to finished products. Acrylamide, an α,β-unsaturated (conjugated) reactive molecule, can be detected as a contaminant in several foodstuffs including baby foods and infant formulas. It is anticipated that children will generally have intakes that are two to three times those of adults when expressed on a body-weight basis. Though exposure to acrylamide is inevitable, it is necessary to protect infant and children from high exposure. The present review focuses on the several a

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10

Jiang, Liping, Jun Cao, Yu An, et al. "Genotoxicity of acrylamide in human hepatoma G2 (HepG2) cells." Toxicology in Vitro 21, no. 8 (2007): 1486–92. http://dx.doi.org/10.1016/j.tiv.2007.06.011.

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11

Dobrovolsky, Vasily N., M. Monserrat Pacheco-Martinez, L. Patrice McDaniel, Mason G. Pearce, and Wei Ding. "In vivo genotoxicity assessment of acrylamide and glycidyl methacrylate." Food and Chemical Toxicology 87 (January 2016): 120–27. http://dx.doi.org/10.1016/j.fct.2015.12.006.

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12

Rajeh, Nesreen A., Nick Plant, Samar M. Al Saggaf, Hamdy A. Aly, Nasra N. Ayuob, and Sufian M. ElAssouli. "Acrylamide-mediated subacute testicular and genotoxicity, is it reversible?" Egyptian Journal of Histology 35, no. 3 (2012): 424–36. http://dx.doi.org/10.1097/01.ehx.0000418020.81995.dc.

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13

Koyama, Naoki, Manabu Yasui, Yoshimitsu Oda, et al. "Genotoxicity of acrylamide in vitro: Acrylamide is not metabolically activated in standard in vitro systems." Environmental and Molecular Mutagenesis 52, no. 1 (2011): 11–19. http://dx.doi.org/10.1002/em.20560.

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14

Koyama, N., M. Yasui, A. Kimura, et al. "Acrylamide genotoxicity in young versus adult gpt delta male rats." Mutagenesis 26, no. 4 (2011): 545–49. http://dx.doi.org/10.1093/mutage/ger014.

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15

Koyama, Naoki, Hiroko Sakamoto, Mayumi Sakuraba, et al. "Genotoxicity of acrylamide and glycidamide in human lymphoblastoid TK6 cells." Mutation Research/Genetic Toxicology and Environmental Mutagenesis 603, no. 2 (2006): 151–58. http://dx.doi.org/10.1016/j.mrgentox.2005.11.006.

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16

Dearfield, Kerry L., Charles O. Abernathy, Myron S. Ottley, John H. Brantner, and Paul F. Hayes. "Acrylamide: its metabolism, developmental and reproductive effects, genotoxicity, and carcinogenicity." Mutation Research/Reviews in Genetic Toxicology 195, no. 1 (1988): 45–77. http://dx.doi.org/10.1016/0165-1110(88)90015-2.

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17

Mei, Nan, Lea P. McDaniel, Vasily N. Dobrovolsky, et al. "The Genotoxicity of Acrylamide and Glycidamide in Big Blue Rats." Toxicological Sciences 115, no. 2 (2010): 412–21. http://dx.doi.org/10.1093/toxsci/kfq069.

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18

Dasari, Sreenivasulu, Muni Swamy Ganjayi, and Balaji Meriga. "Glutathione S-transferase is a good biomarker in acrylamide induced neurotoxicity and genotoxicity." Interdisciplinary Toxicology 11, no. 2 (2018): 115–21. http://dx.doi.org/10.2478/intox-2018-0007.

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Abstract Glutathione S-transferases (GSTs) are major defence enzymes of the antioxidant enzymatic system. Cytosolic GSTs are more involved in the detoxification than mitochondrial and microsomal GSTs. GSTs are localized in the cerebellum and hippocampus of the rat brain. Acrylamide (AC) is a well assessed neurotoxin of both animals and humans and it produces skeletal muscle weakness and ataxia. AC is extensively used in several industries such as cosmetic, paper, textile, in ore processing, as soil conditioners, flocculants for waste water treatment and it is present in daily consumed food pro

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19

Durling, Louise J. K., and Lilianne Abramsson-Zetterberg. "A comparison of genotoxicity between three common heterocyclic amines and acrylamide." Mutation Research/Genetic Toxicology and Environmental Mutagenesis 580, no. 1-2 (2005): 103–10. http://dx.doi.org/10.1016/j.mrgentox.2004.09.009.

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20

Maher, Agnieszka, Karolina Miśkiewicz, Justyna Rosicka-Kaczmarek, and Adriana Nowak. "Detoxification of Acrylamide by Potentially Probiotic Strains of Lactic Acid Bacteria and Yeast." Molecules 29, no. 20 (2024): 4922. http://dx.doi.org/10.3390/molecules29204922.

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Some potentially probiotic strains of lactic acid bacteria (LAB) and yeast that inhabit the digestive tract of humans are known to detoxify xenobiotics, including acrylamide (AA). The objective of the subsequent research was to evaluate the AA-detoxification capability of LAB and yeast isolated from various sources. Namely, the effect of AA was tested on the growth of LAB and yeast strains, as well in the 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT) assay. Subsequently, the AA-binding ability of LAB and yeast was investigated in various environments, including the pH, inc

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21

Yang, Hye Jin, Sang Hyun Lee, Yong Jin, Jin Hyang Choi, Chang Hoon Han, and Mun Han Lee. "Genotoxicity and toxicological effects of acrylamide on reproductive system in male rats." Journal of Veterinary Science 6, no. 2 (2005): 103. http://dx.doi.org/10.4142/jvs.2005.6.2.103.

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22

Russo, Antonella, Gigliola Gabbani, and Barbara Simoncini. "Weak genotoxicity of acrylamide on premeiotic and somatic cells on the mouse." Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis 309, no. 2 (1994): 263–72. http://dx.doi.org/10.1016/0027-5107(94)90101-5.

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23

Cao, Jun, Yong Liu, Li Jia, et al. "Curcumin Attenuates Acrylamide-Induced Cytotoxicity and Genotoxicity in HepG2 Cells by ROS Scavenging." Journal of Agricultural and Food Chemistry 56, no. 24 (2008): 12059–63. http://dx.doi.org/10.1021/jf8026827.

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24

Dearfield, Kerry L., George R. Douglas, Udo H. Ehling, Martha M. Moore, Gary A. Sega, and David J. Brusick. "Acrylamide: a review of its genotoxicity and an assessment of heritable genetic risk." Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis 330, no. 1-2 (1995): 71–99. http://dx.doi.org/10.1016/0027-5107(95)00037-j.

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25

Yang, Hong-Yuan, Hui-Na Luo, Zai-Mei Wang, Dan-Dan Jin, and Zeng-Ming Yang. "Effects of Acrylamide on Mouse Implantation and Decidualization." International Journal of Molecular Sciences 26, no. 9 (2025): 4129. https://doi.org/10.3390/ijms26094129.

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Acrylamide is a class 2A carcinogen with neurotoxicity and genotoxicity. In addition to industrial production, it is ubiquitous in high-temperature heated high-carbohydrate foods. Numerous studies have confirmed the toxicity of ACR on reproduction. Implantation and decidualization are crucial processes during the establishment of pregnancy in rodents and humans. However, its effect on uterine implantation and decidualization remains poorly understood. The objective of this study is to elucidate the mechanism by which ACR affects implantation and decidualization in mice. ACR is exposed in the d

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26

Rifai, Lubna, and Fatima A. Saleh. "A Review on Acrylamide in Food: Occurrence, Toxicity, and Mitigation Strategies." International Journal of Toxicology 39, no. 2 (2020): 93–102. http://dx.doi.org/10.1177/1091581820902405.

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Acrylamide (AA) is a food contaminant present in a wide range of frequently consumed foods, which makes human exposure to this toxicant unfortunately unavoidable. However, efforts to reduce the formation of AA in food have resulted in some success. This review aims to summarize the occurrence of AA and the potential mitigation strategies of its formation in foods. Formation of AA in foods is mainly linked to Maillard reaction, which is the first feasible route that can be manipulated to reduce AA formation. Furthermore, manipulating processing conditions such as time and temperature of the hea

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27

Gouda, Sahar G., Mahmoud S. Khalil, and Magda M. Naim. "Curcumin protects against testicular damage and genotoxicity induced by acrylamide in male albino mice." Egyptian Journal of Histology 34, no. 2 (2011): 333–45. http://dx.doi.org/10.1097/01.ehx.0000397089.34830.bc.

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28

Zhang, Xiaomei, Liping Jiang, Chenyan Geng, Hiroyuki Yoshimura, and Laifu Zhong. "Inhibition of acrylamide genotoxicity in human liver-derived HepG2 cells by the antioxidant hydroxytyrosol." Chemico-Biological Interactions 176, no. 2-3 (2008): 173–78. http://dx.doi.org/10.1016/j.cbi.2008.08.002.

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29

Larguinho, Miguel, Pedro M. Costa, Gonçalo Sousa, Maria H. Costa, Mário S. Diniz, and Pedro V. Baptista. "Histopathological findings onCarassius auratushepatopancreas upon exposure to acrylamide: correlation with genotoxicity and metabolic alterations." Journal of Applied Toxicology 34, no. 12 (2013): 1293–302. http://dx.doi.org/10.1002/jat.2936.

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30

Lin, Wei-De, Chu-Chyn Ou, Shih-Hao Hsiao, et al. "Effects of Acrylamide-Induced Vasorelaxation and Neuromuscular Blockage: A Rodent Study." Toxics 9, no. 6 (2021): 117. http://dx.doi.org/10.3390/toxics9060117.

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Acrylamide (ACR), which is formed during the Maillard reaction, is used in various industrial processes. ACR accumulation in humans and laboratory animals results in genotoxicity, carcinogenicity, neurotoxicity, and reproductive toxicity. In this study, we investigated the mechanisms by which ACR may induce vasorelaxation and neuromuscular toxicity. Vasorelaxation was studied using an isolated rat aortic ring model. The aortic rings were divided into the following groups: with or without endothelium, with nitric oxide synthase (NOS) inhibition, with acetylcholine receptor inhibition, and with

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31

Kurien, Biji T. "Comment on Curcumin Attenuates Acrylamide-Induced Cytotoxicity and Genotoxicity in HepG2 Cells by ROS Scavenging." Journal of Agricultural and Food Chemistry 57, no. 12 (2009): 5644–46. http://dx.doi.org/10.1021/jf900846n.

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32

Kaur, Navneet, and Nigel G. Halford. "Reducing the Risk of Acrylamide and Other Processing Contaminant Formation in Wheat Products." Foods 12, no. 17 (2023): 3264. http://dx.doi.org/10.3390/foods12173264.

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Wheat is a staple crop, consumed worldwide as a major source of starch and protein. Global intake of wheat has increased in recent years, and overall, wheat is considered to be a healthy food, particularly when products are made from whole grains. However, wheat is almost invariably processed before it is consumed, usually via baking and/or toasting, and this can lead to the formation of toxic processing contaminants, including acrylamide, 5-hydroxymethylfurfural (HMF) and polycyclic aromatic hydrocarbons (PAHs). Acrylamide is principally formed from free (soluble, non-protein) asparagine and

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33

Krishnan, ManonmaniHaravey, Ranjini Ankaiah, and NawneetKumar Kurrey. "The positive intervention effects of resveratrol on acrylamide -induced cyto-/Genotoxicity in primary lymphocytes of rat." Pharmacognosy Magazine 14, no. 59 (2018): 643. http://dx.doi.org/10.4103/pm.pm_378_18.

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34

Keskin, Burcu, Banu Orta-Yilmaz, and Yasemin Aydin. "The role of luteolin in modulation of acrylamide-induced genotoxicity and apoptosis in embryonic fibroblast cells." Mutation Research - Genetic Toxicology and Environmental Mutagenesis 902 (February 2025): 503853. https://doi.org/10.1016/j.mrgentox.2025.503853.

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35

Ghanayem, B. I., K. L. Witt, G. E. Kissling, R. R. Tice, and L. Recio. "Absence of acrylamide-induced genotoxicity in CYP2E1-null mice: Evidence consistent with a glycidamide-mediated effect." Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis 578, no. 1-2 (2005): 284–97. http://dx.doi.org/10.1016/j.mrfmmm.2005.05.004.

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36

Eisenbrand, Gerhard. "Revisiting the evidence for genotoxicity of acrylamide (AA), key to risk assessment of dietary AA exposure." Archives of Toxicology 94, no. 9 (2020): 2939–50. http://dx.doi.org/10.1007/s00204-020-02794-3.

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Abstract The weight of evidence pro/contra classifying the process-related food contaminant (PRC) acrylamide (AA) as a genotoxic carcinogen is reviewed. Current dietary AA exposure estimates reflect margins of exposure (MOEs) < 500. Several arguments support the view that AA may not act as a genotoxic carcinogen, especially not at consumer-relevant exposure levels: Biotransformation of AA into genotoxic glycidamide (GA) in primary rat hepatocytes is markedly slower than detoxifying coupling to glutathione (GS). Repeated feeding of rats with AA containing foods, bringing about uptake of 100

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37

El-Sheikh, Marwa, Ahmed Atef Mesalam, Ayman Mesalam, and Il-Keun Kong. "Acrylamide and Its Metabolite Glycidamide Induce Reproductive Toxicity During In Vitro Maturation of Bovine Oocytes." Toxics 13, no. 3 (2025): 223. https://doi.org/10.3390/toxics13030223.

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Acrylamide (ACR) and its metabolite glycidamide (GLY) are contaminants with known toxic effects, especially in reproductive systems. However, the mechanisms underlying their embryotoxic effects remain inadequately understood. In the current study, we investigated the effects of ACR and GLY exposure on oocyte and embryo developmental competence, focusing on DNA damage, apoptosis, autophagy, and epigenetic regulation. Oocytes were exposed to varying concentrations of ACR and GLY during in vitro maturation. The results demonstrated that both ACR and GLY significantly reduced cleavage and blastocy

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38

Shimamura, Yuko, Misako Iio, Tomoko Urahira, and Shuichi Masuda. "Inhibitory effects of Japanese horseradish (Wasabia japonica) on the formation and genotoxicity of a potent carcinogen, acrylamide." Journal of the Science of Food and Agriculture 97, no. 8 (2016): 2419–25. http://dx.doi.org/10.1002/jsfa.8055.

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39

Ao, Lin, and Jia Cao. "Genotoxicity of Acrylamide and Glycidamide: A Review of the Studies by HPRT Gene and TK Gene Mutation Assays." Genes and Environment 34, no. 1 (2012): 1–8. http://dx.doi.org/10.3123/jemsge.34.1.

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40

Friedman, Marvin A., Errol Zeiger, Dennis E. Marroni, and Dale W. Sickles. "Inhibition of Rat Testicular Nuclear Kinesins (krp2; KIFC5A) by Acrylamide as a Basis for Establishing a Genotoxicity Threshold." Journal of Agricultural and Food Chemistry 56, no. 15 (2008): 6024–30. http://dx.doi.org/10.1021/jf703746f.

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41

Hobbs, Cheryl A., Jeffrey Davis, Kim Shepard, et al. "Differential genotoxicity of acrylamide in the micronucleus andPig-a gene mutation assays in F344 rats and B6C3F1 mice." Mutagenesis 31, no. 6 (2016): 617–26. http://dx.doi.org/10.1093/mutage/gew028.

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42

Lamy, Evelyn, Yvonne Völkel, Peter H. Roos, Fekadu Kassie, and Volker Mersch-Sundermann. "Ethanol enhanced the genotoxicity of acrylamide in human, metabolically competent HepG2 cells by CYP2E1 induction and glutathione depletion." International Journal of Hygiene and Environmental Health 211, no. 1-2 (2008): 74–81. http://dx.doi.org/10.1016/j.ijheh.2007.04.004.

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43

Eisenbrand, Gerhard. "Correction to: Revisiting the evidence for genotoxicity of acrylamide (AA), key to risk assessment of dietary AA exposure." Archives of Toxicology 94, no. 11 (2020): 3935. http://dx.doi.org/10.1007/s00204-020-02893-1.

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44

Manjanatha, Mugimane G., Anane Aidoo, Sharon D. Shelton, et al. "Genotoxicity of acrylamide and its metabolite glycidamide administered in drinking water to male and female Big Blue mice." Environmental and Molecular Mutagenesis 47, no. 1 (2006): 6–17. http://dx.doi.org/10.1002/em.20157.

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45

Crudo, Francesco, Chenyifan Hong, Elisabeth Varga, Giorgia Del Favero, and Doris Marko. "Genotoxic and Mutagenic Effects of the Alternaria Mycotoxin Alternariol in Combination with the Process Contaminant Acrylamide." Toxins 15, no. 12 (2023): 670. http://dx.doi.org/10.3390/toxins15120670.

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Humans are constantly exposed to mixtures of different xenobiotics through their diet. One emerging concern is the Alternaria mycotoxin alternariol (AOH), which can occur in foods typically contaminated by the process contaminant acrylamide (AA). AA is a byproduct of the Maillard reaction produced in carbohydrate-rich foods during thermal processing. Given the genotoxic properties of AOH and AA as single compounds, as well as their potential co-occurrence in food, this study aimed to assess the cytotoxic, genotoxic, and mutagenic effects of these compounds in combination. Genotoxicity was asse

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46

Syberg, Kristian, Mona-Lise Binderup, Nina Cedergreen, and Jette Rank. "Mixture Genotoxicity of 2,4-Dichlorophenoxyacetic Acid, Acrylamide, and Maleic Hydrazide on Human Caco-2 Cells Assessed with Comet Assay." Journal of Toxicology and Environmental Health, Part A 78, no. 6 (2015): 369–80. http://dx.doi.org/10.1080/15287394.2014.983626.

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47

Abdurazakova, Khadizhat Nurmagomedovna, Patimat Shuapandievna Gitinova, and Arats Magomedkhanovna Abakarova. "The modern state of the problem potential mutagenic and carcinogenic activity of food products." Sanitarnyj vrač (Sanitary Doctor), no. 10 (August 7, 2021): 25–36. http://dx.doi.org/10.33920/med-08-2110-02.

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The last few decades have been characterized by increased attention on the part of scientists, international organizations and the public to the problem of reducing adverse external influences on the human body and timely detection and prevention of various diseases. Food — one of the main channels of interaction between the body and the environment-can be a source of a large number of potentially dangerous chemical and biological substances for human health. The harmful effects of foreign substances that enter the human body with food are characterized by a significant variety: from damage to

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48

Allen, Bruce, Errol Zeiger, Greg Lawrence, Marvin Friedman, and Annette Shipp. "Dose–response modeling of in vivo genotoxicity data for use in risk assessment: some approaches illustrated by an analysis of acrylamide." Regulatory Toxicology and Pharmacology 41, no. 1 (2005): 6–27. http://dx.doi.org/10.1016/j.yrtph.2004.09.006.

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49

Ghorbel, Imen, Sameh Maktouf, Nesrine Fendri, et al. "Co-exposure to aluminum and acrylamide disturbs expression of metallothionein, proinflammatory cytokines and induces genotoxicity: Biochemical and histopathological changes in the kidney of adult rats." Environmental Toxicology 31, no. 9 (2015): 1044–58. http://dx.doi.org/10.1002/tox.22114.

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50

Pingarilho, Marta, Nuno G. Oliveira, Célia Martins, et al. "Induction of sister chromatid exchange by acrylamide and glycidamide in human lymphocytes: Role of polymorphisms in detoxification and DNA-repair genes in the genotoxicity of glycidamide." Mutation Research/Genetic Toxicology and Environmental Mutagenesis 752, no. 1-2 (2013): 1–7. http://dx.doi.org/10.1016/j.mrgentox.2012.12.013.

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