Relevant bibliographies by topics / Actin reorganization

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Actin reorganization'

Author: Grafiati
Published: 4 June 2021
Last updated: 18 February 2022

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Journal articles on the topic "Actin reorganization"

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Hirshman, Carol A., and Charles W. Emala. "Actin reorganization in airway smooth muscle cells involves Gq and Gi-2 activation of Rho." American Journal of Physiology-Lung Cellular and Molecular Physiology 277, no. 3 (1999): L653—L661. http://dx.doi.org/10.1152/ajplung.1999.277.3.l653.

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Extracellular stimuli induce cytoskeleton reorganization (stress-fiber formation) in cells and Ca2+ sensitization in intact smooth muscle preparations by activating signaling pathways that involve Rho proteins, a subfamily of the Ras superfamily of monomeric G proteins. In airway smooth muscle, the agonists responsible for cytoskeletal reorganization via actin polymerization are poorly understood. Carbachol-, lysophosphatidic acid (LPA)-, and endothelin-1-induced increases in filamentous actin staining are indicative of actin reorganization (filamentous-to-globular actin ratios of 2.4 ± 0.3 in
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Bernstein, BW, and JR Bamburg. "Reorganization of actin in depolarized synaptosomes." Journal of Neuroscience 5, no. 10 (1985): 2565–69. http://dx.doi.org/10.1523/jneurosci.05-10-02565.1985.

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Letort, Gaëlle, Hajer Ennomani, Laurène Gressin, Manuel Théry, and Laurent Blanchoin. "Dynamic reorganization of the actin cytoskeleton." F1000Research 4 (October 1, 2015): 940. http://dx.doi.org/10.12688/f1000research.6374.1.

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Cellular processes, including morphogenesis, polarization, and motility, rely on a variety of actin-based structures. Although the biochemical composition and filament organization of these structures are different, they often emerge from a common origin. This is possible because the actin structures are highly dynamic. Indeed, they assemble, grow, and disassemble in a time scale of a second to a minute. Therefore, the reorganization of a given actin structure can promote the formation of another. Here, we discuss such transitions and illustrate them with computer simulations.
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Ivanov, Andrei I., Ingrid C. McCall, Charles A. Parkos, and Asma Nusrat. "Role for Actin Filament Turnover and a Myosin II Motor in Cytoskeleton-driven Disassembly of the Epithelial Apical Junctional Complex." Molecular Biology of the Cell 15, no. 6 (2004): 2639–51. http://dx.doi.org/10.1091/mbc.e04-02-0163.

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Disassembly of the epithelial apical junctional complex (AJC), composed of the tight junction (TJ) and adherens junction (AJ), is important for normal tissue remodeling and pathogen-induced disruption of epithelial barriers. Using a calcium depletion model in T84 epithelial cells, we previously found that disassembly of the AJC results in endocytosis of AJ/TJ proteins. In the present study, we investigated the role of the actin cytoskeleton in disassembly and internalization of the AJC. Calcium depletion induced reorganization of apical F-actin into contractile rings. Internalized AJ/TJ protei
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Narumiya, Shuh, Toshimasa Ishizaki, and Naoki Watanabe. "Rho effectors and reorganization of actin cytoskeleton." FEBS Letters 410, no. 1 (1997): 68–72. http://dx.doi.org/10.1016/s0014-5793(97)00317-7.

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Dasgupta, Swapan K., Anhquyen Le, Qi Da, Miguel Cruz, Rolando E. Rumbaut, and Perumal Thiagarajan. "Wdr1-Dependent Actin Reorganization in Platelet Activation." PLOS ONE 11, no. 9 (2016): e0162897. http://dx.doi.org/10.1371/journal.pone.0162897.

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JANISCH, R. "Actin reorganization during regeneration of paramecium caudatum." Cell Biology International Reports 14 (September 1990): 206. http://dx.doi.org/10.1016/0309-1651(90)90924-n.

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Carton, Iris, Diane Hermans, and Jan Eggermont. "Hypotonicity induces membrane protrusions and actin remodeling via activation of small GTPases Rac and Cdc42 in Rat-1 fibroblasts." American Journal of Physiology-Cell Physiology 285, no. 4 (2003): C935—C944. http://dx.doi.org/10.1152/ajpcell.00069.2003.

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An important consequence of cell swelling is the reorganization of the F-actin cytoskeleton in different cell types. We demonstrate in this study by means of rhodamine-phalloidin labeling and fluorescence microscopy that a drastic reorganization of F-actin occurs in swollen Rat-1 fibroblasts: stress fibers disappear and F-actin patches are formed in peripheral extensions at the cell border. Moreover, we demonstrate that activation of both Rac and Cdc42, members of the family of small Rho GTPases, forms the link between the hypotonic stimulation and F-actin reorganization. Indeed, inhibition of
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Watts, RG, and TH Howard. "Mechanisms for actin reorganization in chemotactic factor-activated polymorphonuclear leukocytes." Blood 81, no. 10 (1993): 2750–57. http://dx.doi.org/10.1182/blood.v81.10.2750.2750.

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Abstract Cytoskeletal structure in polymorphonuclear leukocytes (PMNs) is thought to reflect a simple equilibrium between two actin pools (globular [G]- and filamentous [F] actin). Recent description of two distinct F-actin pools in PMNs (Triton-insoluble [stable] and Triton- soluble [labile] F-actin pools) (Watts and Howard, Cell Motil Cytoskeleton, 21:25, 1992) suggest a tripartite equilibrium between these F-actin pools and G-actin and multiple possible mechanisms for polymerization. To study the contribution of each actin pool to actin dynamics in PMNs, changes in actin content of the Trit
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Watts, RG, and TH Howard. "Mechanisms for actin reorganization in chemotactic factor-activated polymorphonuclear leukocytes." Blood 81, no. 10 (1993): 2750–57. http://dx.doi.org/10.1182/blood.v81.10.2750.bloodjournal81102750.

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Cytoskeletal structure in polymorphonuclear leukocytes (PMNs) is thought to reflect a simple equilibrium between two actin pools (globular [G]- and filamentous [F] actin). Recent description of two distinct F-actin pools in PMNs (Triton-insoluble [stable] and Triton- soluble [labile] F-actin pools) (Watts and Howard, Cell Motil Cytoskeleton, 21:25, 1992) suggest a tripartite equilibrium between these F-actin pools and G-actin and multiple possible mechanisms for polymerization. To study the contribution of each actin pool to actin dynamics in PMNs, changes in actin content of the Triton-solubl
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Dissertations / Theses on the topic "Actin reorganization"

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Stevenson, Victoria A. "Actin Reorganization in Drosophila Syncytial Blastoderm Embryos: a Dissertation." eScholarship@UMMS, 2002. http://escholarship.umassmed.edu/gsbs_diss/308.

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This work addresses the mechanism of cell cycle specific actin reorganization in Drosophila syncytial blastoderm embryos. During mitosis in typical animal cells after chromosome segregation is complete, daughter cells are separated in a process called cytokinesis. Cytokinesis is ordinarily driven by constriction of an actin ring that physically pinches the cell in two. The early Drosophila embryo is a syncytium; nuclei divide in a single cell without intervening cytokinesis. During the later syncytial divisions, nuclei are arranged in a monolayer at the cortex of the embryo. This stage of embr
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Ard, Ryan. "Regulation of RhoA Activation and Actin Reorganization by Diacylglycerol Kinase." Thesis, Université d'Ottawa / University of Ottawa, 2012. http://hdl.handle.net/10393/22669.

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Rho GTPases are critical regulators of actin cytoskeletal dynamics. The three most well characterized Rho GTPases, Rac1, RhoA and Cdc42 share a common inhibitor, RhoGDI. It is only recently becoming clear how upstream signals cause the selective release of individual Rho GTPases from RhoGDI. For example, our laboratory showed that diacylglycerol kinase zeta (DGKz), which converts diacylglycerol (DAG) to phosphatidic acid (PA), activates PAK1-mediated RhoGDI phosphorylation on Ser-101/174, causing selective Rac1 release and activation. Phosphorylation of RhoGDI on Ser-34 by PKCa has recently be
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MacLeod, Matthew Alexander. "LFA-1 outside-in signalling and actin cytoskeleton reorganization in cytotoxic T lymphocytes." Thesis, University of British Columbia, 2006. http://hdl.handle.net/2429/31970.

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Integrins are heterodimeric cell-surface receptors that mediate the adhesion of cells to the extracellular matrix and to other cells. Leukocyte function-associated molecule-1 (LFA-1) is the most prevalent integrin expressed on T cells and is known to be crucial for migration, extravasation and immunological synapse formation. In addition to its role as an adhesion molecule, LFA-1 can induce intracellular signals that influence T cell effector functions, including activation of proteins involved in actin polymerization. However, these studies have used experimental means to activate LFA-1
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Moldovan, Leni. "The role of reactive oxygen species in the actin cytoskeleton reorganization and cellular motility /." The Ohio State University, 1999. http://rave.ohiolink.edu/etdc/view?acc_num=osu1488192960170305.

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Foley, Tanya. "Diacylglycerol Kinase Iota Mediates Actin Cytoskeletal Reorganization by Regulating the Activities of RhoC and Rac1." Thesis, Université d'Ottawa / University of Ottawa, 2015. http://hdl.handle.net/10393/32333.

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Cell migration is required for a number of physiological processes and is implicated in pathologies such as tumor metastasis. Cell motility is dependent upon dynamic actin reorganization, and is regulated by the Rho family of small GTPases. Rho GTPases are molecular switches that cycle between their active and inactive conformations. The best-studied members of this family are Rac1, RhoA, and Cdc42. Each is responsible for the formation of specific actin structures. Diacylglycerol kinases (DGKs) act at the membrane to convert diacylglycerol (DAG) and phosphatidic acid (PA), maintaining the bal
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Noria, Sabrena. "Shear stress-induced morphological adaptations of endothelial cells, reorganization of cell adhesion complexes and the actin cytoskeleton." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp05/NQ63691.pdf.

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Lalonde, Julie Kathleen. "TC10, a mammalian Rho GTPase responsible for actin cytoskeleton reorganization and cardiac hypertrophy in the murine heart." Thesis, University of Ottawa (Canada), 2003. http://hdl.handle.net/10393/26507.

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Rho guanosine triphosphatases (GTPases) act as molecular switches, cycling between two conformational states: an active, GTP-bound state and an inactive, GDP-bound state to control many complex cellular events in eukaryotic cells. Many Rho GTPases, including RhoA, Cdc42 and Rac1, have been extensively characterized and are involved in actin reorganization, activation of MAPK cascades, cell cycle progression, cellular proliferation, invasion, differentiation and apoptosis. TC10 was identified and classified as a Rho GTPase over ten years ago, however the precise role of this protein, which is h
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Fajol, Abul [Verfasser], and Florian [Akademischer Betreuer] Lang. "GSK3-controlled sympathetic activity in FGF23 production and FGF23 gene regulation by actin cytoskeleton reorganization / Abul Fajol ; Betreuer: Florian Lang." Tübingen : Universitätsbibliothek Tübingen, 2016. http://d-nb.info/1197694234/34.

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Degen, Janine [Verfasser], and Stefanie [Gutachter] Kliche. "The ADAP/SKAP55-module regulates F-actin cytoskeleton reorganization through the interaction with Ena/VASP proteins / Janine Degen ; Gutachter: Stefanie Kliche." Magdeburg : Universitätsbibliothek Otto-von-Guericke-Universität, 2020. http://d-nb.info/1226931987/34.

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Kallin, Anders. "Modulation of PDGF Receptor Signaling via the Phosphatase SHP-2 and the Docking Protein Gab1." Doctoral thesis, Uppsala University, Ludwig Institute for Cancer Research, 2003. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-3748.

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<p>x</p><br><p>Platelet-derived growth factors (PDGF), a family of potent mitogens and chemoattractants for cells of mesenchymal origin, elicit their biological effects through the binding of two related receptor tyrosine kinases, denoted α- and β-receptors. The binding of PDGF to the receptors causes receptor dimerization and autophosphorylation on tyrosine residues. Src homology 2 (SH2) domain-containing proteins then bind the phosphorylated receptors, mediating further propagation of the signal. This thesis describes how the interaction between the PDGF receptors and some of their downstrea
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Books on the topic "Actin reorganization"

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Dept, Massachusetts Metropolitan District Commission Metropolitan Police. Plan of action. Metropolitan Police Dept., 1987.

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Case studies of mass tort limited fund class action settlements & bankruptcy reorganizations. Federal Judicial Center, 2000.

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United States. Department of Housing and Urban Development. HUD reinvention: From blueprint to action : summary. U.S. Dept. of Housing and Urban Development, 1995.

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United States. Department of Housing and Urban Development. HUD reinvention: From blueprint to action : summary. U.S. Dept. of Housing and Urban Development, 1995.

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United States. Department of Housing and Urban Development. HUD reinvention: From blueprint to action : summary. U.S. Dept. of Housing and Urban Development, 1995.

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Zilka, Carla. Business restructuring: An action template for reducing cost and growing profit. Wiley, 2010.

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Zilka, Carla. Business restructuring: An action template for reducing cost and growing profit. Wiley, 2010.

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Zilka, Carla. Business restructuring: An action template for reducing cost and growing profit. Wiley, 2010.

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Affairs, United States Congress Senate Committee on Governmental. Organization of federal executive departments and agencies; agencies and functions of the Federal government established, abolished, continued, modified, reorganized, extended, transferred, or changed in name by legislative or executive action during calendar years 1997 and 1998. U.S. G.P.O., 1999.

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Johnson, David E. Medical risk in the Future Force unit of action: Results of the Army Medical Department Transformation Workshop IV. RAND, Arroyo Center and RAND Health, 2005.

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Book chapters on the topic "Actin reorganization"

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Okeyo, Kennedy Omondi, Hiromi Miyoshi, and Taiji Adachi. "Involvement of Mechanical Strain in Actin Network Reorganization." In Frontiers of Biomechanics. Springer Japan, 2014. http://dx.doi.org/10.1007/978-4-431-55163-8_4.

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McGough, Amy, Brian Pope, and Alan Weeds. "The ADF/Cofilin Family: Accelerators of Actin Reorganization." In Results and Problems in Cell Differentiation. Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-540-46560-7_10.

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McGovern, Kathryn W., and Kathryn A. DeFea. "Molecular Mechanisms Underlying Beta-Arrestin-Dependent Chemotaxis and Actin-Cytoskeletal Reorganization." In Arrestins - Pharmacology and Therapeutic Potential. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-41199-1_17.

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Métais, O., S. Yanase, C. Flores, P. Bartello, and M. Lesieur. "Reorganization of Coherent Vortices in Shear Layers under the Action of Solid-Body Rotation." In Turbulent Shear Flows 8. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77674-8_28.

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Rey, Emmanuel, Martine Laprise, and Sophie Lufkin. "Sustainability Issues at the Neighbourhood Scale." In Neighbourhoods in Transition. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-82208-8_5.

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AbstractWe previously identified that urban brownfield regeneration projects are relevant strategies to limit urban sprawl while revitalizing portions of cities, namely mixed-use neighbourhoods. Moreover, these neighbourhoods in transition are opportunities to foster the implementation of sustainability objectives within European metropolitan areas. This chapter explore this subject by deepening the sustainability issues at the neighbourhood scale. To provide the basis for discussion, we first attempt to frame the urban sustainability concept and to explain how the neighbourhood scale is a means of action for cities. Then, we analyse the different sustainability issues according to a wide variety of parameters that must especially be taken into account during sustainable neighbourhood projects, and more precisely urban brownfield regeneration projects. These parameters cover the four pillars of sustainability—the environment, society, economy, and governance—and the polycentric reorganization of European metropolitan areas.
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DeFea, Kathryn A. "Arrestins in Actin Reorganization and Cell Migration." In Progress in Molecular Biology and Translational Science. Elsevier, 2013. http://dx.doi.org/10.1016/b978-0-12-394440-5.00008-5.

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Ridley, Anne J. "[33] Growth factor-induced actin reorganization in Swiss 3T3 cells." In Small GTPases and Their Regulators Part B: Rho Family. Elsevier, 1995. http://dx.doi.org/10.1016/0076-6879(95)56035-1.

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ADACHI, T., and K. SATO. "THRESHOLD FIBER STRAIN THAT INDUCES REORGANIZATION OF CYTOSKELETAL ACTIN STRUCTURE IN OSTEOBLASTIC CELLS." In Biomechanics at Micro- and Nanoscale Levels. WORLD SCIENTIFIC, 2006. http://dx.doi.org/10.1142/9789812773838_0005.

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Aspenström, Pontus. "Chapter 1 Roles of F-BAR/PCH Proteins in the Regulation of Membrane Dynamics and Actin Reorganization." In International Review of Cell and Molecular Biology. Elsevier, 2008. http://dx.doi.org/10.1016/s1937-6448(08)01601-8.

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Paterson, Sarah. "The Rise of Trading." In Corporate Reorganization Law and Forces of Change. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780198860365.003.0004.

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This chapter explores the second of the changes in logic and practice in the field of finance with which the book is concerned: the shift from hold-to-maturity debt investment strategies to trading strategies. The argument is made that the concept of collective action problems in US corporate reorganization law ought to be replaced by different concepts when the reorganization is among sophisticated, strongly adjusting financial creditors and investors and when debt trades in secondary markets. The chapter further argues that the more recent development of modern corporate reorganization law in England and Wales has enabled terms and concepts to emerge which are better adapted to traded debt markets. Overall, however, the chapter emphasizes the relationship between reduced salience of the collective action concept and a secondary market for distressed debt. It does not suggest a new, universal conceptual framework for corporate reorganization law in which the collective action concept is permanently replaced.
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Conference papers on the topic "Actin reorganization"

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Shibatay, N., K. Tanaka, K. Okamoto, and T. Onji. "REORGANIZATION OF ACTIN AND MYOSIN IN THE ACTIVATED PLATELETS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643539.

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This study was done to clarify the intracellular dynamic arrangements of myosin(My) and actin(Ac) in activation process of human platelets (PLs) from unactivated to activated stage (clot retraction) in electron microscopy. The observation of unactivated PLs was done either in the fresh whole blood fixed directly with 0.1 % glutaraldehyde or in PLs isolated by gel filtration of platelet rich plasma(PRP) containing prostaglandin I2 (10 ng/ml). The isolated PLs mounted on a glass cover slip were used as activated PLs (adrerent ones). The contracted PLs were prepared in PRP incubated with thrombin
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Parameswaran, Harikrishnan, and Bela Suki. "Myosin Motor-Induced Reorganization Of The Actin Cytoskeleton Following Stretch." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a1259.

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Brüning, D., P. Drain, and I. Rustenbeck. "Correlation of actin reorganization and granule movement in stimulated primary beta cells." In Diabetes Kongress 2018 – 53. Jahrestagung der DDG. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1641803.

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Liu, Yi, and Juan Ren. "Modeling and Control of Dynamic Cellular Mechanotransduction: Part I — Actin Cytoskeleton Quantification." In ASME 2018 Dynamic Systems and Control Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/dscc2018-9180.

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Living cells respond to external stimuli through the reorganization of the actin cytoskeleton, and the actin cytoskeleton significantly affects the cellular mechanical behavior. However, due to the lack of approaches to actin cytoskeleton quantification, the dynamics of mechanotransduction is still poorly understood. In this study, we propose an image recognition-based quantification (IRQ) approach to actin cytoskeleton quantification. IRQ quantifies the actin cytoskeleton through three parameters: the partial actin-cytoskeletal deviation (PAD), the total actin-cytoskeletal deviation (TAD) and
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Pozhvanov, G. A., E. I. Sharova, and S. S. Medvedev. "Redox-dependent reorganization of the actin cytoskeleton in the root of arabidopsis under stress and regulatory effects." In IX Congress of society physiologists of plants of Russia "Plant physiology is the basis for creating plants of the future". Kazan University Press, 2019. http://dx.doi.org/10.26907/978-5-00130-204-9-2019-357.

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Hsu, Hui-Ju, Andrea Locke, Susan Q. Vanderzyl, and Roland Kaunas. "Stretch-Induced Stress Fiber Remodeling and MAPK Activations Depend on Mechanical Strain Rate." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53464.

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Actin stress fibers (SFs), bundles of actin filaments crosslinked by α-actinin and myosin II in non-muscle cells, are mechanosensitive structural elements that respond to applied stress and strain to regulate cell morphology, signal transduction and cell function. Results from various studies indicate that myosin-generated contraction extends SFs beyond their unloaded lengths and cells maintain fiber strain at an optimal level that depends on actomyosin activity (Lu et al., 2008). Stretching the matrix upon which cells adhere perturbs the cell-matrix traction forces and cells respond by active
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Ohashi, Toshiro, Yusaku Niida, Ryoichi Tanaka, and Masaaki Sato. "Simultaneous Measurement of Morphology and Traction Forces of Endothelial Cells Under Fluid Shear Stress." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80694.

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Under fluid shear stress, vascular endothelial cells (ECs) cultured in a monolayer are known to exhibit marked elongation and orientation to the direction of flow [1]. It is also observed that intracellular F-actin filament distributions changed depending on the amplitude of shear stress and the direction of flow, suggesting morphology of ECs is closely related to cytoskeltal structure [2]. ECs generate contractile forces by the actin-myosin machinery and the forces are transmitted to underlying substrate as cellular traction forces. We hypothesize that reorganization of cytoskeletal structure
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Takahashi, Satoshi, Masahiro Toda, Hikaru Sasaki, and Takeshi Kawase. "Abstract 531: RNA interference-mediated downregulation of uPARAP decreases invasion and migration property in glioma cells through reorganization of the actin cytoskeleton." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-531.

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Joshi, Sagar D., and Lance A. Davidson. "Remote Control of Apical Epithelial Sheet Contraction by Laser Ablation or Nano-Perfusion: Acute Stimulus Triggers Rapid Remodeling of F-Actin Network in Apical Cortex." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-204904.

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Apical contraction is the major tissue movement during remodeling of epithelial sheets in development. During apical contraction, groups of cells narrow their apices to form bottle-shaped structures, driving events such as sea-urchin gastrulation [1], Drosophila ventral-furrow formation, vertebrate neurulation and wound healing [2]. Tissue-folding events such as invagination, ingression and involution involve this tissue movement in which cells actively build “rifts” and “tubes”. Epithelial cells integrate genetic information, mechanical signals, and biochemical gradients to build these struct
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Tondon, Abhishek, Hui-Ju Hsu, and Roland Kaunas. "The Direction of Cyclic Stretch-Induced Stress Fiber Orientation Depends on Matrix Rigidity." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53501.

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Mechanical properties of the cellular environment such as elastic rigidity have been shown to play an important role in the regulation of important cellular processes such as proliferation, differentiation and apoptosis (1–3). Intracellular tension decreases with decreasing matrix rigidity (1). Actin stress fibers (SFs), the major structural element in cells bearing tension, are also less prevalent on soft vs. rigid matrices (4). We have developed a theoretical model of stretch-induced SFs that predicts SFs reorient perpendicular to the direction of cyclic stretch in order to maintain SF tensi
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Reports on the topic "Actin reorganization"

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Stoyanova, Tihomira, Veselina Uzunova, Albena Momchilova, Rumiana Tzoneva, and Iva Ugrinova. The Treatment of Breast Cancer Cells with Erufosine Leads to Actin Cytoskeleton Reorganization, Inhibition of Cell Motility, Cell Cycle Arrest and Apoptosis. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, 2021. http://dx.doi.org/10.7546/crabs.2021.01.11.

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