Relevant bibliographies by topics / Activation threshold hypothesis

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  2. Dissertations / Theses
  3. Conference papers

Academic literature on the topic 'Activation threshold hypothesis'

Author: Grafiati
Published: 4 June 2021
Last updated: 17 February 2022

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Journal articles on the topic "Activation threshold hypothesis"

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Latash, Mark, Mindy Levin, John Scholz, and Gregor Schöner. "Motor control theories and their applications." Medicina 46, no. 6 (2010): 382. http://dx.doi.org/10.3390/medicina46060054.

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We describe several infl uential hypotheses in the field of motor control including the equilibrium-point (referent confi guration) hypothesis, the uncontrolled manifold hypothesis, and the idea of synergies based on the principle of motor abundance. The equilibrium-point hypothesis is based on the idea of control with thresholds for activation of neuronal pools; it provides a framework for analysis of both voluntary and involuntary movements. In particular, control of a single muscle can be adequately described with changes in the threshold of motor unit recruitment during slow muscle stretch
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Qin, Chunhua, Amy G. Aslamkhan, Kara Pearson, et al. "AhR Activation in Pharmaceutical Development: Applying Liver Gene Expression Biomarker Thresholds to Identify Doses Associated With Tumorigenic Risks in Rats." Toxicological Sciences 171, no. 1 (2019): 46–55. http://dx.doi.org/10.1093/toxsci/kfz125.

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Abstract Aryl hydrocarbon receptor (AhR) activation is associated with carcinogenicity of non-genotoxic AhR-activating carcinogens such as 2,3,7,8-tetrachlorodibenzodioxin (TCDD), and is often observed with drug candidate molecules in development and raises safety concerns. As downstream effectors of AhR signaling, the expression and activity of Cyp1a1 and Cyp1a2 genes are commonly monitored as evidence of AhR activation to inform carcinogenic risk of compounds in question. However, many marketed drugs and phytochemicals are reported to induce these Cyps modestly and are not associated with di
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Purmohammad, Mehdi. "Linguistic alignment in L1–L2 dialogue." Language and Dialogue 5, no. 2 (2015): 312–33. http://dx.doi.org/10.1075/ld.5.2.07pur.

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The aim of this paper is to investigate alignment in L1–L2 dialogue. More specifically, I examine to what extent alignment in L1–L2 is different from alignment in L1–L1 dialogue. I investigate different variables that may affect linguistic alignment in the course of L1–L2 dialogue. As more variables, such as differences in language proficiency of interlocutors, affect the linguistic alignment in L1–L2 dialogue, it appears that linguistic alignment in L1–L2 dialogue is different from linguistic alignment in L1–L1 dialogue both quantitatively and quantitatively. I also discuss the mechanisms tha
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Ning, Xue-Han, Shi-Han Chen, Norman E. Buroker та ін. "Short-cycle hypoxia in the intact heart: hypoxia-inducible factor 1α signaling and the relationship to injury threshold". American Journal of Physiology-Heart and Circulatory Physiology 292, № 1 (2007): H333—H341. http://dx.doi.org/10.1152/ajpheart.00078.2006.

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Hypoxia-inducible factor 1α (HIF-1α) transcriptionally activates multiple genes, which regulate metabolic cardioprotective and cross-adaptive mechanisms. Hypoxia and several other stimuli induce the HIF-1α signaling cascade, although little data exist regarding the stress threshold for activation in heart. We tested the hypothesis that relatively mild short-cycle hypoxia, which produces minimal cardiac dysfunction and no sustained or major disruption in energy state, can induce HIF-1α activation. We developed a short-cycle hypoxia protocol in isolated perfused rabbit heart to test this hypothe
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Nagano-Saito, Atsuko, Paul Cisek, Andrea S. Perna, et al. "From anticipation to action, the role of dopamine in perceptual decision making: an fMRI-tyrosine depletion study." Journal of Neurophysiology 108, no. 2 (2012): 501–12. http://dx.doi.org/10.1152/jn.00592.2011.

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During simple sensorimotor decision making, neurons in the parietal cortex extract evidence from sensory information provided by visual areas until a decision is reached. Contextual information can bias parietal activity during the task and change the decision-making parameters. One type of contextual information is the availability of reward for correct decisions. We tested the hypothesis that the frontal lobes and basal ganglia use contextual information to bias decision making to maximize reward. Human volunteers underwent functional MRI while making decisions about the motion of dots on a
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Chua, M., and A. F. Dulhunty. "Noninactivating tension in rat skeletal muscle. Effects of thyroid hormone." Journal of General Physiology 94, no. 1 (1989): 183–203. http://dx.doi.org/10.1085/jgp.94.1.183.

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Inactivation of excitation-contraction coupling was examined in extensor digitorum longus (EDL) and soleus muscle fibers from rats injected daily with tri-iodothyronine (T3, 150 micrograms/kg) for 10-14 d. Steady-state activation and inactivation curves for contraction were obtained from measurements of peak potassium contracture tension at different surface membrane potentials. The experiments tested the hypothesis that noninactivating tension is a "window" tension caused by the overlap of the activation and inactivation curves. Changes in the amplitude and voltage dependence of noninactivati
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Porcellini, Simona, Elisabetta Traggiai, Ursula Schenk, et al. "Regulation of peripheral T cell activation by calreticulin." Journal of Experimental Medicine 203, no. 2 (2006): 461–71. http://dx.doi.org/10.1084/jem.20051519.

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Regulated expression of positive and negative regulatory factors controls the extent and duration of T cell adaptive immune response preserving the organism's integrity. Calreticulin (CRT) is a major Ca2+ buffering chaperone in the lumen of the endoplasmic reticulum. Here we investigated the impact of CRT deficiency on T cell function in immunodeficient mice reconstituted with fetal liver crt−/− hemopoietic progenitors. These chimeric mice displayed severe immunopathological traits, which correlated with a lower threshold of T cell receptor (TCR) activation and exaggerated peripheral T cell re
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Dotan, Raffy, Cameron Mitchell, Rotem Cohen, Panagiota Klentrou, David Gabriel, and Bareket Falk. "Child—Adult Differences in Muscle Activation — A Review." Pediatric Exercise Science 24, no. 1 (2012): 2–21. http://dx.doi.org/10.1123/pes.24.1.2.

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Children differ from adults in many muscular performance attributes such as size-normalized strength and power, endurance, fatigability and the recovery from exhaustive exercise, to name just a few. Metabolic attributes, such as glycolytic capacity, substrate utilization, and VO2 kinetics also differ markedly between children and adults. Various factors, such as dimensionality, intramuscular synchronization, agonist-antagonist coactivation, level of volitional activation, or muscle composition, can explain some, but not all of the observed differences. It is hypothesized that, compared with ad
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Hollowell, D. E., P. R. Bhandary, A. W. Funsten, and P. M. Suratt. "Respiratory-related recruitment of the masseter: response to hypercapnia and loading." Journal of Applied Physiology 70, no. 6 (1991): 2508–13. http://dx.doi.org/10.1152/jappl.1991.70.6.2508.

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To test the hypothesis that a muscle that closes the jaw, the masseter, can be recruited by ventilatory stimuli, we studied the electromyographic activation of the masseter and genioglossus in seven normal awake males who were exposed in random order to progressive hyperoxic hypercapnia, inspiratory threshold loading (-40 cmH2O), and combined hypercapnia and loading. With hypercapnia, the masseter was generally recruited after the genioglossus had been activated. Once recruited, activation of both muscles increased linearly with increasing CO2. Combined hypercapnia and loading produced more ac
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Baranov, Sergei V., Oxana V. Baranova, Svitlana Yablonska, et al. "Mitochondria modulate programmed neuritic retraction." Proceedings of the National Academy of Sciences 116, no. 2 (2018): 650–59. http://dx.doi.org/10.1073/pnas.1811021116.

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Neuritic retraction in the absence of overt neuronal death is a shared feature of normal aging and neurodegenerative disorders, but the intracellular mechanisms modulating this process are not understood. We propose that cumulative distal mitochondrial protein damage results in impaired protein import, leading to mitochondrial dysfunction and focal activation of the canonical apoptosis pathway in neurites. This is a controlled process that may not lead to neuronal death and, thus, we term this phenomenon “neuritosis.” Consistent with our hypothesis, we show that in primary cerebrocortical neur
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Dissertations / Theses on the topic "Activation threshold hypothesis"

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Stölten, Katrin. "The Effects of Age of Onset on VOT in L2 Aquisition and L1 Attrition : A Study of the Speech Production and Perception of Advanced Spanish-Swedish Bilinguals." Doctoral thesis, Stockholms universitet, Centrum för tvåspråkighetsforskning, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-94887.

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This thesis explores the role of age in second language (L2) acquisition and first language (L1) attrition. The focus is on Voice Onset Time (VOT) in the production and categorical perception of word-initial L1 and L2 stops in highly advanced L1 Spanish learners of L2 Swedish. Using as the point of departure a maturational constraints perspective and the Critical Period Hypothesis (CPH), Study I examines the impact of age of onset (AO) of L2 acquisition on the production of L2 Swedish voiceless stops. The results show that there are AO effects even in the speech of highly advanced L2 learners
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Conference papers on the topic "Activation threshold hypothesis"

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Simone, E. R., T. A. Davies, N. A. Zabe, S. M. Greenberg-seperaky, and N. E. Larsen. "EARLY PLATELET-THROMBIN RECEPTORS AND THEIR FUNCTIONS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643730.

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Human platelets possess less than 1000 high affinity [Kd=10-9]and 50-100,000 receptors of lower [Kd=10-7] affinity for o(α-thrombin. The selective derivatization of thrombin with the bifunctional crosslinking agent, DNCO, has enabled us to identify these receptorsvia covalent binding of either active siteinhibited tosyllyslmethylketothrombin (TLCK-T) or active Ctf-thrombin (T).Kinetic studies of the inhibition of the platelet-thrombin response by covalently and noncovalently bound TLCK-T have helped to elucidate the roles of the high and low affinity thrombin receptors. The activation paramete
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