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Journal articles on the topic 'Acute gastritis'

Author: Grafiati

Published: 28 May 2022

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1

Schultz, Marc J., René W. M. van der Hulst, and Guido N. J. Tytgat. "Acute phlegmonous gastritis." Gastrointestinal Endoscopy 44, no. 1 (July 1996): 80–83. http://dx.doi.org/10.1016/s0016-5107(96)70236-5.

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2

Cortes-Barenque, F., J. C. Salceda-Otero, D. Angulo-Molina, and D. Lozoya-González. "Acute phlegmonous gastritis." Revista de Gastroenterología de México (English Edition) 79, no. 4 (October 2014): 299–301. http://dx.doi.org/10.1016/j.rgmxen.2014.11.001.

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3

Chen, Chi-Hung, Jeng-Shiann Shin, and Jen-Chieh Huang. "Acute phlegmonous gastritis." Advances in Digestive Medicine 4, no. 1 (March 2017): 32–34. http://dx.doi.org/10.1002/aid2.12032.

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4

O'Toole, P. A., and J. A. Morris. "Acute phlegmonous gastritis." Postgraduate Medical Journal 64, no. 750 (April 1, 1988): 315–16. http://dx.doi.org/10.1136/pgmj.64.750.315.

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5

Chamberlain, Craig E. "ACUTE HEMORRHAGIC GASTRITIS." Gastroenterology Clinics of North America 22, no. 4 (December 1993): 843–73. http://dx.doi.org/10.1016/s0889-8553(21)00109-6.

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6

Rahmawati, Rahmawati. "FAKTOR – FAKTOR YANG BERHUBUNGAN DENGAN TIMBULNYA GEJALA GASTRITIS PADA SISWA SEKOLAH MENENGAH ATAS KOTA JAMBI." Jurnal Ilmiah Dikdaya 8, no. 2 (December 6, 2018): 334. http://dx.doi.org/10.33087/dikdaya.v8i2.115.

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Gastritis is an inflammation of the gastric mucosa that can be acute, chronic, diffuse or localized, the stomach is a digestive device that functions mechanically in storing food and drinks, breaking food into small particles, mixing with gastric fluid and gradually entering the intestine . The clinical picture of erosive acute gastritis varies from mild, asymptomatic, to very severe. Factors for gastritis are foods contaminated with bacteria, caffeine-containing drinks, alcohol, drugs such as non-steroidal anti-inflammatory drugs aspirin, acidic foods and drinks, spicy foods, and irregular diets. This research is an analytical study with a cross sectional approach. The sample of this study were students from Jambi City High School who were randomly drawn to get 930 samples. The results obtained from 930 respondents, most (61.3%) had chronic gastritis. And found a significant relationship between diet and the onset of symptoms of acute and chronic gastritis with P-Value = 0,000. Based on the results of this study, the researchers expect health information, especially regarding gastritis by local health workers so as to reduce the incidence of acute and chronic gastritis. Keywords: Acute Gastritis, Chronic Gastritis, Gastritis Factors

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7

Saito, Makoto, Masanobu Morioka, Hiromi Kanno, and Shinya Tanaka. "Acute Phlegmonous Gastritis with Neutropenia." Internal Medicine 51, no. 20 (2012): 2987–88. http://dx.doi.org/10.2169/internalmedicine.51.8537.

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8

King, Andrew J., Toby Eyre, Deepa Rangarajan, Rebecca Sampson, and Henri Grech. "Acute Isolated Transmural Neutropenic Gastritis." Journal of Clinical Oncology 30, no. 1 (January 1, 2012): e1-e2. http://dx.doi.org/10.1200/jco.2011.38.0964.

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9

Bloodworth, L. L., P. E. Stevens, R. F. Bury, J. P. Arm, and D. J. Rainford. "Emphysematous gastritis after acute pancreatitis." Gut 28, no. 7 (July 1, 1987): 900–902. http://dx.doi.org/10.1136/gut.28.7.900.

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10

Villablanca, Pedro A., Shashvat Sukhal, Asimul Ansari, and Dergham Mohammed. "Acute gastritis‐induced Takotsubo's cardiomyopathy." Clinical Case Reports 1, no. 2 (November 22, 2013): 91–95. http://dx.doi.org/10.1002/ccr3.32.

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11

Prajapati, Devendra K., Kapil Rampal, and Jyoti M. Prajapati. "Acute necrotizing gastritis with gangrenous stomach." International Surgery Journal 5, no. 1 (December 26, 2017): 11. http://dx.doi.org/10.18203/2349-2902.isj20175880.

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Acute necrotizing gastritis with gangrene of the stomach wall is a rarely encountered condition. This often a fatal condition if is attributed to vascular occlusion, chemical and mechanical injuries or infectious etiologies. We review the available literature with a case of acute necrotising gastritis with unknown etiology. Case was managed successfully with emergency partial gastrectomy.

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12

Feng, Jiexiong, Yizhen Weng, Jiyan Yuan, Naping Li, Xuefeng Zhou, and Peng Zhang. "Acute phlegmonous gastritis in an infant." Journal of Pediatric Surgery 40, no. 4 (April 2005): 745–47. http://dx.doi.org/10.1016/j.jpedsurg.2005.01.036.

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13

Kobayashi, Masanori, Ou Yamaguchi, Koji Nagata, Kouichi Nonaka, and Shomei Ryozawa. "Acute hemorrhagic gastritis after nivolumab treatment." Gastrointestinal Endoscopy 86, no. 5 (November 2017): 915–16. http://dx.doi.org/10.1016/j.gie.2017.04.033.

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14

Collier, D. StJ, J. Crampton, and W. G. Everett. "ACUTE HAEMORRHAGIC GASTRITIS CONTROLLED BY OMEPRAZOLE." Lancet 333, no. 8641 (April 1989): 776. http://dx.doi.org/10.1016/s0140-6736(89)92589-0.

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15

Sood, Bimal P., Naveen Kalra, and S. Suri. "CT features of acute phlegmonous gastritis." Clinical Imaging 24, no. 5 (September 2000): 287–88. http://dx.doi.org/10.1016/s0899-7071(00)00216-3.

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16

Chia, John K., and Andrew Y. Chia. "Acute Gastritis Associated With Enterovirus Infection." Archives of Pathology & Laboratory Medicine 134, no. 1 (January 1, 2010): 16–17. http://dx.doi.org/10.5858/2009-0018-ler1.1.

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17

Escrich, Víctor, Ángela Martinez, Berta Lapeña, Marta Mayorga, Antonio Cuadrado, Javier Crespo, and Emilio Fábrega. "Varicella-Zoster Gastritis in a Liver Transplant Patient: A Call to Attention." OBM Hepatology and Gastroenterology 05, no. 02 (March 1, 2021): 1. http://dx.doi.org/10.21926/obm.hg.2102057.

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The reactivation of the Varicella-zoster virus (VZV) is a rare cause of acute gastritis in adults. About 30 cases have been reported in the literature, mostly with immunocompromised patients and mainly after bone marrow transplantation or during the development of malignant hematological diseases. Clinically, it is usually accompanied by cutaneous manifestations. Here, we studied a case of VZV gastritis in a liver transplant (LT) patient. We described the main symptoms, endoscopic findings, histologic changes, and treatment of VZV gastritis. Till now, no case of acute gastritis due to the reactivation of VZV after solid organ transplantation had been reported [2–5]. This was the first reported case of acute gastritis by the reactivation of VZV after LT without cutaneous vesicular eruption. Gastrointestinal symptoms usually develop a week before the onset of fever and cutaneous manifestations. However, in some cases, like this one, vesicular rashes may be absent, making the diagnosis quite challenging. In conclusion, through this case, we suggest including VZV gastritis in the differential diagnosis of gastrointestinal symptoms after transplantation and informing about the response of VZV gastritis to treatment with oral acyclovir.

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18

Skvortsov, V. V., L. V. Zaklyakova, B. N. Levitan, M. Yu Bolgova, I. K. Zaklyakov, and E. A. Golieva. "Modern approaches to pharmacotherapy of chronic gastritis." Meditsinskiy sovet = Medical Council, no. 15 (October 19, 2021): 40–47. http://dx.doi.org/10.21518/2079-701x-2021-15-40-47.

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The definition of gastritis is based on the histological features of the gastric mucosa. This is not the erythema observed during gastroscopy, and there are no specific clinical manifestations or symptoms that determine it. The modern classification of gastritis is based on time (acute and chronic), histological features, anatomical distribution and the main pathological mechanisms. Acute gastritis will develop into chronic if left untreated. Helicobacter pylori (H. pylori) is the most common cause of gastritis worldwide. However, from 60 to 70% H. pylori-negative subjects with functional dyspepsia or non-erosive gastroesophageal reflux were also found to have gastritis. H. pylori-negative gastritis is considered when a person meets all four of these criteria: negative triple staining of biopsies of the gastric mucosa, no history of treatment of H. pylori. In these patients, the cause of gastritis may be associated with tobacco smoking, alcohol consumption and / or the use of nonsteroidal anti-inflammatory drugs (NSAIDs) or steroids. Other causes of gastritis include autoimmune gastritis associated with antibodies of serum anti-parietal and anti-internal factor; organisms other than H. pylori, such as Mycobacterium avium intracellulare, Herpes simplex and Cytomegalovirus; gastritis caused by acid reflux; Rare causes of gastritis include collagen gastritis, sarcoidosis, eosinophilic gastritis and lymphocytic gastritis. The clinical picture, laboratory studies, gastroscopy, as well as histological and microbiological examination of tissue biopsies are important for the diagnosis of gastritis and its causes. Treatment of gastritis caused by H. pylori leads to the rapid disappearance of polymorphic-nuclear infiltration and a decrease in chronic inflammatory infiltrate with gradual normalization of the mucous membrane. Other types of gastritis should be treated based on their etiology.

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19

Al Baihaqi, Rohmat. "NURSING CARE FOR ACUTE PAIN RELATED TO GASTRITIS AT ANGGREK ROOM RSI NASHRUL UMMAH LAMONGAN." Journal of Vocational Nursing 2, no. 1 (May 31, 2021): 10. http://dx.doi.org/10.20473/jovin.v2i1.26882.

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Introduction: Gastritis is a problem that many people experience and can occur at various ages. The most common symptom for gastritis sufferers is acute pain. However, acute pain nursing care in gastritis patients is different. This study is to explain Acute Pain Nursing Care for Gastritis Clients at Anggrek Room RSI Nashrul Ummah Lamongan. Methods: This study used case study design. Sampling of this study was one child suffering from gastritis with acute pain symptom. Data of this study were collected by interview, observation, physical examination and documentation. The data that obtained were analyzed with descriptive methods, diagnosis and evaluation. Results: The results showed that there were several gaps between case and theories for the assessment stage. A gap occurs in the assessment of the physical examination of the nasal passages which reveals the presence of nasal lobe breathing while holding pain. The priority of nursing diagnosis was acute pain and the focus of interventions was on pain management. Conclusion: Providing implementation of non-pharmacological techniques (distraction and relaxation techniques) and pharmacological (collaboration providing analgesics), environmental management, touch management, providing knowledge of the causes of pain and efforts to reduce pain, and observation of conditions that are effective in reducing pain in patients

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20

El-Serag, H. B., A. Sonnenberg, M. M. Jamal, J. M. Inadomi, L. A. Crooks, and R. M. Feddersen. "Corpus gastritis is protective against reflux oesophagitis." Gut 45, no. 2 (August 1, 1999): 181–85. http://dx.doi.org/10.1136/gut.45.2.181.

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BACKGROUNDGastric acid is important in the pathogenesis of reflux oesophagitis. Acid production by the gastric corpus is reduced in corpus gastritis.AIMSTo determine whether corpus gastritis protects against reflux oesophagitis.METHODSPatients presenting for elective oesophagogastroduodenoscopy were studied. Two biopsy specimens were taken from the antrum, corpus, and cardia and stained with haematoxylin/eosin and Diff-Quick II stains. The presence and severity of gastritis were graded according to a modified updated Sydney classification.RESULTSOf 302 patients, 154 had endoscopic signs of reflux oesophagitis. There was no difference between patients with and controls without oesophagitis in the overall infection rates with Helicobacter pylori. Acute or chronic corpus gastritis occurred less often in patients with than those without reflux oesophagitis. Compared with controls, corpus gastritis was less severe in patients with reflux oesophagitis. The presence of acute or chronic gastritis in the corpus was significantly correlated with either type of gastritis in other areas of the stomach. In a multivariate logistic regression, age, sex, smoking status, and the presence of chronic corpus gastritis all exerted a significant influence on the presence of reflux oesophagitis. Chronic corpus gastritis was associated with a 54% reduced risk for reflux oesophagitis.CONCLUSIONSWhile infection withH pylori alone may not affect the occurrence of reflux oesophagitis, the development of chronic corpus gastritis seems to be protective.

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21

Yong, Sherri, Houssam Attal, and Gregorio Chejfec. "Pseudomembranous Gastritis." Archives of Pathology & Laboratory Medicine 124, no. 4 (April 1, 2000): 619–24. http://dx.doi.org/10.5858/2000-124-0619-pg.

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Abstract A 36-year-old Hispanic man who had undergone allogeneic bone marrow transplantation, complicated by graft versus host disease, was admitted with acute gastrointestinal symptoms, including severe diarrhea and diffuse abdominal pain. He also had a persistent cough with sputum production. Blood cultures yielded Escherichia coli, and sputum cultures grew Apergillus species. The patient was treated with antifungal agents and broad-spectrum antibiotics. Despite aggressive medical therapy, the patient died 10 days after admission. Postmortem examination disclosed severe, bilateral confluent bronchopneumonia, with numerous septated branching hyphae consistent with Aspergillus species fungal organisms that involved the pulmonary parenchyma and tracheobronchial tree. Although the small and large bowels were only mildly congested, the entire gastric mucosa was covered with a 1.5-cm-thick pseudomembrane that contained numerous Aspergillus organisms. Our report represents the first description, to our knowledge, of a diffuse inflammatory pseudomembrane in the stomach, a complication that to date has only been associated with small and large bowel involvement.

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22

Min, Sun Young. "Acute phlegmonous gastritis complicated by delayed perforation." World Journal of Gastroenterology 20, no. 12 (2014): 3383. http://dx.doi.org/10.3748/wjg.v20.i12.3383.

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23

Perigela, HariCharan, MuraliKrishna Vasamsetty, VaraPrasad Bangi, and Sivaraj Nagabhushigari. "Gastric gangrene due to acute necrotizing gastritis." Journal of Dr. NTR University of Health Sciences 3, no. 1 (2014): 38. http://dx.doi.org/10.4103/2277-8632.128429.

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24

Salmeron, Marcelo, Nicole Desplaces, Anne Lavergne, and Rémi Houdart. "CAMPYLOBACTER-LIKE ORGANISMS AND ACUTE PURULENT GASTRITIS." Lancet 328, no. 8513 (October 1986): 975–76. http://dx.doi.org/10.1016/s0140-6736(86)90625-2.

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25

LIN, CHUN-YEN, CHING-SONG LEE, DENG-YN LIN, CHIEN-FU HONG, YI-YIN JAN, PAUL-Y. LIN, PANG-CHI CHEN, and CHENG-SHYONG WU. "Emphysematous gastritis secondary to acute gastric dilatation." Journal of Gastroenterology and Hepatology 10, no. 5 (October 1995): 612–15. http://dx.doi.org/10.1111/j.1440-1746.1995.tb01356.x.

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26

Coton, Thierry, Claire Mallaret, Catherine Coilliot, Dominique Carré, and Michel Guisset. "Severe acute ulcerated gastritis induced by salt." La Presse Médicale 38, no. 3 (March 2009): 499–500. http://dx.doi.org/10.1016/j.lpm.2008.11.012.

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27

Hegazi, MO, A. Almansour, and K. Taha. "Recurrent diabetic ketoacidosis related to acute gastritis." Practical Diabetes International 27, no. 8 (October 1, 2010): 336–336. http://dx.doi.org/10.1002/pdi.1516.

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28

&NA;. "H. pylori eradication therapy for acute gastritis?" Inpharma Weekly &NA;, no. 1273 (February 2001): 18. http://dx.doi.org/10.2165/00128413-200112730-00042.

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29

Al-Hamoudi, Waleed K., Lesley Alpert, and Andrew Szilagyi. "Acute Symptomatic Gastritis Due to Helicobacter heilmannii." Helicobacter 11, no. 5 (October 2006): 446–50. http://dx.doi.org/10.1111/j.1523-5378.2006.00437.x.

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30

Yang, Hongxin, Zhiqiang Yan, Jiaju Chen, Haitao Xie, Haibin Wang, and Qian Wang. "Diagnosis and treatment of acute phlegmonous gastritis." Medicine 97, no. 18 (May 2018): e0629. http://dx.doi.org/10.1097/md.0000000000010629.

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31

Nakamura, Kiyokuni, Ryo Tamura, Yoshitomi Yasui, and Hideaki Okajima. "Atypical presentation at acute gastritis: significant gastric wall thickening as a presentation of a primary Helicobacter pylori infection in children." BMJ Case Reports 14, no. 7 (July 2021): e243912. http://dx.doi.org/10.1136/bcr-2021-243912.

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Helicobacter pylori infection could cause chronic inflammation in the stomach and induce peptic ulcer disease or even malignant tumour. The initial infection of the organism happens in childhood but most of cases are latent. We had a case of 10-year-old girl who presented with acute epigastric pain and significant thickening of the stomach wall on CT. The finding seemed atypical for acute gastritis so oesophagogastroduodenoscopy and serology examination were added and the primary infection of H. pylori was confirmed with the exclusion of other possible diagnoses like eosinophilic gastritis and IgA vasculitis. Acute gastritis is one of the most common sickness in children, however, it would be worthwhile considering further investigation including H. pylori infection in a case of atypical presentation to prevent negative consequences derived from chronic H. pylori infection.

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32

Yeo, Dahee, Su-Jung Hwang, Ye-Seul Song, and Hyo-Jong Lee. "Humulene Inhibits Acute Gastric Mucosal Injury by Enhancing Mucosal Integrity." Antioxidants 10, no. 5 (May 11, 2021): 761. http://dx.doi.org/10.3390/antiox10050761.

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This study was designed to determine whether α-humulene, a major constituent in many plants used in fragrances, has a protective role against gastric injury in vivo and in vitro. A rat model of hydrochloric acid (HCl)/ethanol-induced gastritis and human mast cells (HMC-1) were used to investigate the mucosal protective effect of α-humulene. α-Humulene significantly inhibited gastric lesions in HCl/ethanol-induced acute gastritis and decreased gastric acid secretion pyloric ligation-induced gastric ulcers in vivo. In addition, α-humulene reduced the amount of reactive oxygen species and malondialdehyde through upregulation of prostaglandin E2 (PGE2) and superoxide dismutase (SOD). In HMC-1 cells, α-humulene decreased intracellular calcium and increased intracellular cyclic adenosine monophosphate (cAMP) levels, resulting in low histamine levels. α-Humulene also reduced the expression levels of cytokine genes such as interleukin (IL)-1β, IL-6, tumor necrosis factor (TNF) by downregulating nuclear factor-κB (NF-κB) nuclear translocation. Finally, α-humulene upregulated the expression levels of mucin 5AC (Muc5ac), Muc6, trefoil factor 1 (Tff1), trefoil factor 2 (Tff2), and polymeric immunoglobulin receptor (pigr). α-Humulene may attenuate HCl/ethanol-induced gastritis by inhibiting histamine release and NF-κB activation and stimulating antioxidants and mucosal protective factors, particularly Muc5ac and Muc6. Therefore, these data suggest that α-humulene is a potential drug candidate for the treatment of stress-induced or alcoholic gastritis.

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33

Muna Aguon, Paul, Tyler Aasen, and Brenda Shinar. "Emphysematous gastritis developing after isolated cardioembolic event." International Journal of Medicine 6, no. 1 (January 18, 2018): 6. http://dx.doi.org/10.14419/ijm.v6i1.8819.

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Emphysematous gastritis is a very rare disease associated with a high mortality rate, making prompt recognition and early treatment of the underlying cause essential. We present a case of emphysematous gastritis developing after a cardio embolic-related acute ischemic injury to the gastric mucosa.

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34

Caletti, G., P. Fusaroli, A. Tucci, M. Fedrigo, G. Bettini, and E. Roda. "Severe acute gastritis associated with Helicobacter pylori infection." Digestive and Liver Disease 32, no. 1 (January 2000): 34–38. http://dx.doi.org/10.1016/s1590-8658(00)80042-0.

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35

Barbosa, A. J., D. M. Queiroz, E. N. Mendes, G. A. Rocha, A. S. Carvalho, and M. L. Roquete. "Campylobacter pylori associated acute gastritis in a child." Journal of Clinical Pathology 42, no. 7 (July 1, 1989): 779. http://dx.doi.org/10.1136/jcp.42.7.779.

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36

ODAI, Tsuyoshi, and Takenori HIBINO. "The Abdominal Ultrasonographic Appearance of Acute Phlegmonous Gastritis." Kansenshogaku Zasshi 90, no. 2 (2016): 113–19. http://dx.doi.org/10.11150/kansenshogakuzasshi.90.113.

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37

Cruz, F. O., P. S. Soffia, P. M. Del Rio, M. P. Fava, and I. G. Duarte. "Acute phlegmonous gastritis with mural abscess: CT diagnosis." American Journal of Roentgenology 159, no. 4 (October 1992): 767–68. http://dx.doi.org/10.2214/ajr.159.4.1529840.

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38

Kagawa, Takao. "Endoscopic examination in patients with acute corrosive gastritis." Journal of Nippon Medical School 57, no. 6 (1990): 547–55. http://dx.doi.org/10.1272/jnms1923.57.547.

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Katsinelos, Panagiotis, Jannis Kountouras, George Paroutoglou, Athanasios Beltsis, Kostas Mimidis, Ioannis Pilpilidis, and Christos Zavos. "Severe acute haemorrhagic gastritis controlled by hydrogen peroxide." European Journal of Gastroenterology & Hepatology 18, no. 1 (January 2006): 107–10. http://dx.doi.org/10.1097/00042737-200601000-00019.

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40

Lavelle, John P., Steve Landas, Frank A. Mitros, and Jeffrey L. Conklin. "Acute gastritis associated with spiral organisms from cats." Digestive Diseases and Sciences 39, no. 4 (April 1994): 744–50. http://dx.doi.org/10.1007/bf02087417.

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41

Taniguchi, Hideaki, Masahito Aimi, Hiroshi Matsushita, and Gaku Shimazaki. "A case of phlegmonous gastritis after acute pharyngitis." Clinical Journal of Gastroenterology 14, no. 2 (February 4, 2021): 500–505. http://dx.doi.org/10.1007/s12328-021-01345-2.

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42

Mihály, Emese, Tamás Micsik, Márk Juhász, László Herszényi, and Zsolt Tulassay. "Gastritis and gastropathy." Orvosi Hetilap 155, no. 2 (January 2014): 43–61. http://dx.doi.org/10.1556/oh.2014.29807.

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Alterations of the stomach mucosa in response to different adverse effects result in various morphological and clinical symptoms. Gastric mucosa alterations can be classified on the bases of diverse viewpoints. It makes this overview difficult, that identical toxic effects may cause different mucosal changes and different toxic agents may produce similar mucosal appearance. The more accurate understanding of the pathological processes which develop in the stomach mucosa needs reconsideration. The authors make an attempt to define gastritis and gastropathy in order to classify and present their features. Gastritis is a histological definition indicating mucosal inflammation. Acute gastritis is caused by infections. The two most important forms of chronic gastritis are metaplastic atrophic gastritis with an autoimmune origin and Helicobacter pylori inflammation. Gastropathy is the name of different structural alterations of the mucosa. Its most important feature is the paucity of inflammatory signs. Gastropathies can be divided into 4 categories based on the nature of the underlying pathological effect, on its morphological appearance and the way of the development. Differential diagnosis is an important pathological and clinical task because different treatment methods and prognosis. Orv. Hetil., 2014, 155(2), 43–61.

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43

Maya Syari, Dina, and Hotna Sari. "EVALUASI PENGGUNAAN OBAT PROTON PUMP INHIBITOR PADA PASIEN RAWAT JALAN DENGAN GANGGUAN LAMBUNG (GASTRITIS) DI RUMAH." JIFI (Jurnal Ilmiah Farmasi Imelda) 5, no. 1 (September 30, 2021): 1–4. http://dx.doi.org/10.52943/jifarmasi.v5i1.623.

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Gastritis is an inflammatory process in the gastric mucosa and gastric mucosa. Gastritis is a state of inflammation or bleeding off the gastric mucosa that is acute, chronic, diffuse, or local. Gastritis or heartburn is an inflammation of the stomach wall, this disease is often found to arise suddenly which is usually characterized by nausea or vomiting, bleeding pain, weakness, decreased appetite or headache. The purpose of this study was to determine the PPI class of drugs most widely used by outpatients with gastric gastritis disorders. Knowing the factors that most often influence of gastric gastritis disorders.To find out the age most often affected by gastric gastritis. This research method used an observational method with the research design used was data collection carried out retrospectively, namely by tracing records at the outpatient installation of Imelda Hospital Medan from January 1 to December 31, 2019. The data obtained at the installation is descriptive and evaluate the use of PPIs with the treatment of gastritis aimed at improving the patient's quality of life, relieving complaints, curing gastritis, preventing recurrences and complications. Besed on the results of the study, the most outpatients with gastritis at the Imleda Hospital Medan were 15 women (17%), 6 men (29%), And the most commonlyused PPI drugs for gastritis patients were lansoprazole in 18 patients (86%), Omeprazole 3 patients (14%).

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44

Yang, Hang, and Bing Hu. "Immunological Perspective: Helicobacter pylori Infection and Gastritis." Mediators of Inflammation 2022 (March 8, 2022): 1–11. http://dx.doi.org/10.1155/2022/2944156.

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Helicobacter pylori is a spiral-shaped gram-negative bacterium. Its infection is mainly transmitted via oral-oral and fecal-oral routes usually during early childhood. It can achieve persistent colonization by manipulating the host immune responses, which also causes mucosal damage and inflammation. H. pylori gastritis is an infectious disease and results in chronic gastritis of different severity in near all patients with infection. It may develop from acute/chronic inflammation, chronic atrophic gastritis, intestinal metaplasia, dysplasia, and intraepithelial neoplasia, eventually to gastric cancer. This review attempts to cover recent studies which provide important insights into how H. pylori causes chronic inflammation and what the characteristic is, which will immunologically explain H. pylori gastritis.

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Sulaimani, Mohammed, Abdulaziz Rashed Alshehri, Mohammed Hmoud, Najat Waggas, and Ghaleb Aboalsamh. "Emphysematous gastritis: a case report." International Journal of Scientific Reports 3, no. 1 (December 29, 2016): 19. http://dx.doi.org/10.18203/issn.2454-2156.intjscirep20164844.

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<p>Emphysematous gastritis (EG) is a rare disease entity related to infection of the gastric wall. It is caused by gas-forming pathogens including <em>Enterobacter</em> species, <em>Clostridium perfringens</em> and others. EG patients clinically present with symptoms similar to acute abdomen and can have a mortality that can reach up to 80%. Alcoholism was identified as a risk factor for such a disease. Different diseases like diabetes mellitus, ulcerative colitis and use of nonsteroidal anti-inflammatory agents were found to be associated with EG. To date, no clear guidelines regarding EG management, however, early detection and management is crucial to prevent related deaths. In the acute setting, the role of surgery is limited. Surgical interventions might be needed if obstruction of gastric outlet or perforation ensues. Fifty six years old male, heavy smoker, alcoholic with a history of ischemic heart disease, presented to Emergency Department with a complaint of severe epigastric abdominal pain for two days. The patient was vitally stable, conscious, oriented, however, looks in severe pain. His lab investigations were within normal ranges. Computed tomography (CT) of the abdomen was consistent with emphysematous gastritis. To our knowledge, this is the first case of emphysematous gastritis reported in Saudi Arabia using only radiological modality.</p>

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Bashirova, D. K., B. Sh Fatkullin, and R. R. Karimov. "Peculiarities of the course of salmonollosis gastroenteritis in the presence of chronic chelicobacteriosis gastritis." Kazan medical journal 77, no. 3 (June 15, 1996): 184–88. http://dx.doi.org/10.17816/kazmj104432.

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The course of salmonollosis gastroenteritis in the presence of chronic chelicjbacteriosis, with more pronounced and prolonged gastritis syndrome, slower stool normalization is revealed. The signs of chronic gastritis acute condition are found in these patients in 20% of the cases in the reconvalescence period, with consequent significant increase of the stomach mucous membrane colonization by chelicobacteria.

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Lupu, V. V., Gabriela Paduraru, Eliza Tighici-Saizu, Claudia Olaru, Nicoleta Gimiga, Angelica Cristina Marin, Cornelia Savescu, Ioana Florea, and Marin Burlea. "GASTRITIS AND HELICOBACTER PYLORI IN CHILDREN – PRIMARY AND SECONDARY ENDOSCOPIC DIAGNOSIS." Romanian Journal of Pediatrics 64, no. 3 (September 30, 2015): 268–71. http://dx.doi.org/10.37897/rjp.2015.3.6.

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The infection with Helicobacter pylori (H pylori) represents an important issue of public health. Aim. Establishing the prevalence of H. pylori infection in children and presenting the cases according to the gastritis type by performing upper digestive endoscopies. Material and method. The retrospective study referred to a period of 5 years and included 1269 children evaluated by upper endoscopy to establish the H. pylori infection rate. Results. The frequency of H. pylori in the case of acute gastritis was significantly more reduced (34.78%) than in the case of chronic gastritis (54.94%). Referring to the 10 types of gastritis, the most frequent ones are purpuric (43.66%), nodular purpuric (25.93%) and nodular antral (15.84%). At the other end, atrophic and hypertrophic gastritis were positioned. The most frequent associations pointed out endoscopically are those with 1st degree esophagitis - 51.6%, duodenitis – 45.07% and purpuric duodenitis – 24.74%.Conclusions. The H. pylori infection is the most frequent etiologic factor for chronic gastritis (54.94%). The early identification of the infection is essential to destroy the bacteria and to prevent the development of various types of gastritis that are later on endoscopically identified.

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Sablina, Anastasiya O., Oleg A. Sablin, Julia V. Andreeva, Gennadii G. Rodionov, I. I. Shantyr, Inna E. Ushal, and Igor A. Samusenko. "H. pylori eradication therapy: impact of gastric mucosa atrophy on transport of amoxicillin to H. pylori colonization area." HERALD of North-Western State Medical University named after I.I. Mechnikov 13, no. 2 (August 30, 2021): 57–66. http://dx.doi.org/10.17816/mechnikov70280.

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AIM: The aim was to assess systemic transport of amoxicillin, the most common antibiotic in H. pylori eradication regimens to the gastric in atrophic gastritis. MATERIALS AND METHODS: Systemic transport of amoxicillin to the gastric lumen of rats was evaluated in washes from the gastric mucosa in the model of atrophic gastritis after intravenous drug infusion. Transport of amoxicillin from bloodstream to the gastric lumen was also assessed in patients with atrophic and non-atrophic gastritis in aspirated via nasogastric probe gastric juice after oral drug administration. Amoxicillin concentration was measured in samples using liquid chromatography-mass spectrometry. RESULTS: In rats with induced atrophic gastritis, hyperemia and acute erosions of the gastric mucosa, as well as microscopic signs of non-active chronic body gastritis and non-active antral atrophic gastritis were found. Amoxicillin concentration in washes from the gastric mucosa was significantly (p 0.01) higher in rats of experimental group than in control group at all time points (30, 60, 120, 240 min after drug infusion). The lowest mean amoxicillin concentration in gastric juice was observed in patients with antral atrophy (p 0.01). The maximum amoxicillin concentration in gastric secretion was found at the 180th min of aspiration in patients with atrophy of gastric mucosa, while in patients of the group of comparison it was found at 30-120th min of aspiration. CONCLUSIONS: Acute gastric mucosa erosions enhance amoxicillin delivery to gastric lumen in rats. Atrophy of antral mucosa more than in the corpus is characterized by decreased amoxicillin transfer from systemic bloodstream to gastric lumen in patients after oral amoxicillin intake. The gastric mucosa atrophy should be taken into consideration while predicting the efficacy of H. pylori eradication therapy in patients with chronic gastritis.

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Wang, Jiwei, Tao Zhang, Xin Zhou, Handong Huang, Maijian Wang, and Ming Xie. "Combination of antibiotics, gastric lavage and nasojejunal feeding—an effective alternative for the management of acute phlegmonous gastritis: a case report." Journal of International Medical Research 49, no. 2 (February 2021): 030006052098574. http://dx.doi.org/10.1177/0300060520985742.

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BackgroundPhlegmonous gastritis is a rare bacterial infection of the gastric wall, characterised by purulent inflammation of the gastric mucosa, submucosa and muscularis layers. Phlegmonous gastritis has a high mortality rate, even with correct diagnosis and antimicrobial therapy.Case presentationA 22-year-old man presented for acute epigastric pain associated with aqueous diarrhoea, vomiting and sustained fever. Abdominal computed tomography showed diffuse oedema and thickened gastric wall, increased number and size of abdominal lymph nodes and the absence of pneumoperitoneum. Fibregastroscopy revealed oedematous, ridged and thickened gastric mucosa with abundant purulent secretion, especially in the antrum, consistent with phlegmonous gastritis, which was confirmed by histological evaluation of gastric biopsies. Cultures of the tissue biopsies and purulent secretion were positive for Enterococcus cecorum. He was treated with sensitive antibiotics according to the antibiogram, and importantly, with continuous gastric lavage and individualised nutritional support therapy. He eventually recovered well and was discharged with no abdominal symptoms.ConclusionsOur case indicates that early diagnosis and immediate treatment are crucial to achieve positive outcomes. The combination of sensitive antibiotics, gastric lavage and early enteral nutrition via nasojejunal feeding might be an effective alternative for the comprehensive treatment of acute phlegmonous gastritis.

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Feyisa, Zelalem Tadese, and Berhanu Teshome Woldeamanuel. "Prevalence and associated risk factors of gastritis among patients visiting Saint Paul Hospital Millennium Medical College, Addis Ababa, Ethiopia." PLOS ONE 16, no. 2 (February 9, 2021): e0246619. http://dx.doi.org/10.1371/journal.pone.0246619.

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Background The health of individuals is not only the absence of disease checked medically, but also encompasses social and psychological aspects. Any departure from the state of physiological, psychological, or social well-being was affected by different factors. However, all contributory factors were not equally responsible for affecting disease. This study was undertaken as a search for the relative effects of sociocultural and individual behavioral factors contributing to acute and chronic gastritis patients visiting Saint Paul Hospital Millennium Medical College (SPHMMC). Methods A cross-sectional study was carried out on 364 patients visited SPHMMC in the study. Primary data were collected through an interview schedule tool with an exit approach by validating questions pertaining to sociocultural and individual behavioral factors. The status of gastritis was measured as whether patients had Helicobacter Pylori infection, signs and symptoms indicated gastritis that occurred, and persisted for less than a month, greater than a month, or none of the signs and symptoms. Descriptive statistics, bivariate analysis, and multivariable ordinal logistic regression model were used to identify the predictors of gastritis severity. P-value ≤ 0.05 was declared as an indicator of statistically significant. Results The prevalence of gastritis in the study area was 78.8%. Specifically, 48.9% and 29.9% had acute and chronic gastritis, respectively. The study found that low income and taking medicinal drugs was slightly significantly contributed to higher gastritis status; however, being younger age was slightly significantly contributed to lower gastritis status. Furthermore, the results indicated that eating spiced foods (Adjusted Odds Ratio (AOR) = 1.508; 95% CI: 1.046, 2.174), lack of physical exercise regularly (AOR = 1.780; 95% CI: 1.001, 3.168), stress (AOR = 2.168; 95% CI: 1.379, 3.4066), and substance use (AOR = 1.478; 95% CI: 1.093, 1.999) were significantly contributed to higher gastritis status. Conclusions The findings suggested that women should take enough rest and sleep well, men refrain from involvement in any risky behaviors, young people and those who earn low income per month should equip with knowledge and understanding on how to practice good health behaviors, eating foods on time, avoiding eating spiced food frequently, doing physical exercise regularly, and taking medicinal drugs according to physician advice are recommended.

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