Relevant bibliographies by topics / Acute myocardial infarction; ST segment resolution

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  1. Journal articles
  2. Dissertations / Theses
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  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Acute myocardial infarction; ST segment resolution'

Author: Grafiati
Published: 5 June 2025
Last updated: 16 July 2025

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Journal articles on the topic "Acute myocardial infarction; ST segment resolution"

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Islam, Syed Aminul, Md Faruque, Fazlur Rahman, Harisul Hoque, and Nilufar Fatema. "Clinical Impacts of ST- Segment Non-Resolution after Thrombolysis for Myocardial Infarction." University Heart Journal 15, no. 1 (2019): 3–7. http://dx.doi.org/10.3329/uhj.v15i1.41439.

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Streptokinase therapy for acute myocardial infarction reduces early mortality and improves outcomes. Failure of reperfusion after streptokinase therapy for acute myocardial infarction is common and indicates a poor prognosis. We investigated the clinical consequences of non-resolution of the ST segment after thrombolytic therapy for acute ST-elevation myocardial infarction, in 80 consecutive patients admitted to a coronary care unit. Failed thrombolysis was defined as <50% ST-segment resolution at 90 minutes after the start of thrombolytic treatment. Outcomes were measured in terms of in hospital adverse events and mortality at 6 weeks. Thrombolysis was successful, in terms of ST-segment resolution, in 59 patients (73.75%). After adjustment for other factors, ST resolution was the only independent predictor of an uncomplicated recovery in hospital. ST-segment resolution is a useful marker of successful thrombolysis and relates to clinical outcome. Average hospital stay was 2 days greater in non resolved ST-segment group than in ST-segment resolved group. At 6 weeks overall early mortality was much lower in the ST segment resolution group (1.7% versus 57.1% with P value of <0.001). So, non resolution of ST-segment in electrocardiogram following thrombolytic therapy in acute STEMI has paramount importance. If assessed routinely it might assist, along with other clinical markers, in the identification of high risk patients.
 University Heart Journal Vol. 15, No. 1, Jan 2019; 3-7
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Ahmad, Munir, Muhammad Yasir, and Asif Rahmat. "ACUTE ST ELEVATION MYOCARDIAL INFARCTION." Professional Medical Journal 25, no. 05 (2018): 777–83. http://dx.doi.org/10.29309/tpmj/18.4763.

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Uddin, Md Faruque, and AK Fazlul Hoque. "Impact of Diabetic Mellitus on the Effect of Streptokinase in Acute Myocardial Infarction Patients." Medicine Today 24, no. 1 (2013): 16–19. http://dx.doi.org/10.3329/medtoday.v24i1.14108.

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A prospective interventional study was carried out to compare the thrombolytic effect of streptokinase between diabetic and non-diabetic myocardial infarction patients. Out of 187 study subjects with acute ST segment elevation myocardial infarction (STEMI), admitted at coronary care unit, 126 patients were non-diabetic and 61 patients were diabetic. Streptokinase was administered to all patients. Resolution (reduction) of elevated ST segment was evaluated after 90 min of streptokinase administration. Successful reperfusion (=70% ST-resolution) was significantly higher in non-diabetic than diabetic (p<0.001), while failed reperfusion (<30% ST resolution) was significantly higher in diabetic patients (p<0.001). It may be concluded that diabetes mellitus might affect the thrombolytic outcome of acute myocardial infarction patients with diabetes mellitus. DOI: http://dx.doi.org/10.3329/medtoday.v24i1.14108 Medicine TODAY Vol.24(1) 2012 pp.16-19
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Ahmad, Zeeshan, Noor Ul Sabah Shah, Abdul Latif, and Anas Shakir. "Frequency of Successful Resolution of ST T Changes after Thrombolysis with Streptokinase in Patients of Acute St Segment Elevated Myocardial Infarction." Pakistan Journal of Medical and Health Sciences 16, no. 3 (2022): 790–92. http://dx.doi.org/10.53350/pjmhs22163790.

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Objective: To determine the frequency of successful resolution of ST T changes after thrombolysis with streptokinase in patients of acute ST Segment elevated myocardial infarction Design of the Study: Cross sectional study Study Settings: The study was conducted at Cardiology Department, Lady Reading Hospital, Peshawar from 22/10/2019 to 22/4/2020. Material and Methods: Over the course of the investigation, researchers kept tabs on 165 patients. FBC, ECG (cardio fax), and echocardiography (Siemens' Acuson cv-70) are all included in the evaluation and investigations. Every patient had an ECG done 90 minutes following the initiation of SK injection to see if the ST segment abnormalities had gone down. Results of the Study: 35 patients (20%) were between the ages of 30 and 45, while 130 patients (79%), were between the ages of 46 and 60. The average age was 57, with a standard deviation of 12.11. There were 72% male patients and 28% female participants in the study. Seventy-three percent of patients had successful resolution of ST T alterations, while 27 percent of patients did not. Conclusion: Our study concludes that the frequency of successful resolution of ST T changes after thrombolysis with streptokinase was 73% in patients of acute ST Segment elevated myocardial infarction Keywords: successful, resolution of ST T changes, thrombolysis, streptokinase, acute ST segment elevated myocardial infarction
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Aslan, Burhan, and Mehmet Zülküf Karahan. "The Relationship Between ST-Segment Depression in Lead aVR and Coronary Microvascular Function in Acute Inferior Myocardial Infarction." Acta Medica 53, no. 1 (2022): 53–58. http://dx.doi.org/10.32552/2022.actamedica.681.

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Aim: The aim of this study was to investigate the relationship between ST-segment depression in the aVR lead and coronary microvascular function in acute inferior myocardial infarction undergoing primary percutaneous intervention.
 Methods: 287 patients with inferior myocardial infarction confirmed by coronary angiography were divided into two groups with and without ST-segment depression in lead aVR ≥ 0.1 mV on the 12 lead ECG. Electrocardiographic recordings were made for the evaluation of ST-segment resolution before and after primary PCI. Angiographic assessment in the infarct-related artery was performed by using the myocardial blush grade and thrombolysis in myocardial infarction flow. 
 Results: Overall, 51 of 287 patients had ST-segment depression in lead aVR. The number of patients with RCA-induced infarction was higher in the group with ST-segment depression in lead aVR. RCA involvement was present in 44 patients. Peak troponin was higher in the group with ST-segment depression in lead aVR compare to the other group (P <0.001). The MBG was more impaired, and the STR was less regressed in patients with ST depression in lead aVR (p<0,001). The ejection fraction of patients with ST-segment depression in lead aVR was lower.
 Conclusion: We found that ST-segment depression in lead aVR was associated with impaired myocardial perfusion in patients with inferior myocardial infarction.
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Ahmad, Aamir, Syed Muhammad Adnan Shah, Raza Ullah, Syed Muhammad Salman Shah, Syed Mujeeb Ur Rahman, and Muhammad Zuhaid. "Post Thrombolytic St-Segment Resolution Outcome in Acute Myocardial Infarction Patients." Journal of Gandhara Medical and Dental Science 9, no. 2 (2022): 38–42. http://dx.doi.org/10.37762/jgmds.9-2.177.

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OBJECTIVES: The main objective of this study was to assess post thrombolytic resolution of ST-segment and its outcome in patients with acute myocardial infarction. METHODOLOGY: This Prospective Comparative Study was carried out at the Cardiology Unit of Ayub Teaching Hospital, Abbottabad. All patients irrespective of gender and age with ST-Segment elevation myocardial infarction (STEMI), having no immediate access to angioplasty and thrombolysed with streptokinase, were included in this study. ECG was taken at the beginning and 90 minutes after the administration of streptokinase. Based on ST-segment resolution on ECG taken at 90 minutes these patients were classified into group A and B. Group A included patients with ST-segment resolution while group B showed no resolution of ST-segment after streptokinase administration. These patients were followed during their hospital stay for complications such as arrhythmias, cardiogenic shock, acquired ventricular septal defects (VSD) aneurysm and death. RESULTS: Among 115 patients, 94 were male and 21 female. Group A included 102 (89%) patients and group B included 13 (11%). In group A, only 1 (0.98 %) patient developed complications and in group B, 13 patients (100%) developed complications. Arrhythmias were the most common complication among MI patients in group A while cardiogenic shock was the commonest complication in group B. CONCLUSION: ST-segment resolution is a practical and applicable indicator of successful thrombolysis and has a significant correlation with clinical outcome in acute myocardial patients after thrombolysis with streptokinase.
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AHMAD, SHIEKH NADEEM, SYED SAUD HASAN, and MUHAMMAD YOUSUF SALAT. "MYOCARDIAL INFARCTION." Professional Medical Journal 18, no. 04 (2011): 671–77. http://dx.doi.org/10.29309/tpmj/2011.18.04.2667.

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Thrombolytic therapy for Acute Myocardial Infarction has been one of the most potent treatment ever developed for condition that kill more patients worldwide than any other. Objective: To evaluate the benefit and efficacy or observational studies of streptokinase therapy on ST-segment elevation resolution in different types of myocardial infarction that focus especially on the younger age group less than forty years. Study design: To observe the streptokinase therapy, in ST-segment elevation resolution, in age less than 40 years and in different types of myocardial infarction. Place & duration of study: The study was conducted at national institute of cardiovascular diseases (NICVD) of Pakistan, Karachi. Subject and Methods: All patients fulfilling the inclusion criteria for thrombolytic therapy were included. Baseline ECG recorded before streptokinase infusion and repeated at completion of infusion i.e. 90 minutes, day 1 and day 2. Results: Streptokinase therapy on blood pressure, CKMB, and ST-segment resolution at 90 minutes, day 1, and Day2 in less than 40-year of age patient. The mean systolic blood pressure was 124+ 3.32 and 112+3.00 pre and post SK therapy reflecting a percentage decrease of 6.67 and highly significant (P<0.001). The Diastolic blood pressure was decrease to 6.25% with a mean value of 76.80+ 2.70 and 72+1.91 before and after the Streptokinase therapy’s, segment resolution at 90 minutes was decreased to 52.01 percent from the baseline and continued to decrease at Day-1 and Day-2 with a percentage reduction of 70.65 and 83.69 % respectively. The P values were highly significant (P<0.001). Conclusions: Thrombolysis improves survival when given within 12 hours of the onset of symptoms. The magnitude of benefit is greatest when reperfusion is established early. Age itself should not be considered a contraindication for fibronolysis
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Gandhi, Viral, and Vicky Gandhi. "Prognostic Value of ST-segment Deviation in Lead aVR in Electrocardiogram in Acute Myocardial Infarction." Journal of the Practice of Cardiovascular Sciences 10, no. 2 (2024): 88–92. http://dx.doi.org/10.4103/jpcs.jpcs_35_24.

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Background: Acute myocardial infarction (AMI) remains a significant cause of morbidity and mortality worldwide, necessitating effective risk stratification tools for improved management. ST-segment deviation in lead augmented vector right (aVR) has emerged as a potential prognostic marker in AMI, but its clinical relevance in the Indian context requires further investigation. Materials and Methods: We conducted a single-center prospective observational study among patients admitted with AMI to assess the prognostic value of ST-segment deviation in lead aVR. Patients were categorized based on the presence or absence of ST-segment deviation in lead aVR, and demographic, clinical, and angiographic characteristics were compared between groups. Follow-up evaluations were performed to assess left ventricular ejection fraction (LVEF) and the incidence of ventricular tachycardia (VT). Results: The study enrolled 110 patients, categorizing them by ST-segment deviation in lead aVR. Patients with ST elevation in lead aVR showed higher moderate LVEF dysfunction (31%–40%) than those with isoelectric ST segments (P = 0.0058). Conversely, patients with ST depression in lead aVR had increased preserved ejection fraction compared to those with an isoelectric ST segment (P = 0.0414). <50% ST-segment resolution postpercutaneous coronary intervention (PCI) was more common among non-ST-segment elevation myocardial infarction (NSTEMI) patients. The incidence of VT was higher in ST elevation in lead aVR (P = 0.0045). Significant differences in ST-segment resolution after PCI (P = 0.0117) between STEMI and NSTEMI patients were noted. Conclusion: ST-segment deviation in lead aVR serves as a valuable prognostic marker in AMI, correlating with adverse outcomes such as moderate LVEF dysfunction and increased risk of VT. Furthermore, its association with ST-segment resolution post-PCI highlights its role in assessing reperfusion success and guiding management strategies. These findings underscore the importance of ST-segment deviation in lead aVR for risk stratification and personalized management in AMI patients.
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Mukesh, Shandilya, and Prasad Ravivishnu. "Assessing Resolution of ST-Elevation on Treatment with Streptokinase and Also to Predict Short Term Outcome." International Journal of Current Pharmaceutical Review and Research 16, no. 2 (2024): 276–80. https://doi.org/10.5281/zenodo.12739744.

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Aim: The aim of the present study was to assess the efficacy of thrombolysis in Acute STEMI patients, withrespect to resolution of ST-elevation on treatment with streptokinase and also to predictshort term outcome duringhospital stay in terms of adverse events and mortality.Methods: This study was done by analysing the ECG of patients with diagnosis of acute ST segment elevationbefore and after thrombolysis with Streptokinase, admitted to Department of Cardiology, IGIMS, PATNA, Bihar,India for the period of 2 years. 150 Acute STEMI patients who had received thrombolytic therapy withstreptokinase were studied in three groups namely Category A, Category B and Category C based on ST segmentresolution after administration of thrombolytic therapy.Results: In the present study, the minimum age of the patient was 30years, maximum age was 75 years. Maximumnumbers of patients in between 40-59 years constitute 55%. Mean age of present study was 52.8±9.6. Male wassignificantly increased (P<0.000) when compared with female patients. In this study chest pain was the mostcommon mode of presentation, present in 116 (96.66%) patients associated withsweating in 104 (86%) patients,breathlessness seen in 30 (25%) patients. Syncope wasseen in 12 (10%) patients and palpitation in 6 (5%) patients.In this study anterior wall Myocardial infarction was not significant compared with inferior wall myocardialinfarction. Thrombolysis time of <3 hours, 3-5 hours and more than5 hours was noted in a, b and c categoriespatients. B and c categories patients were significantly increased when compared with categories of patients.Conclusion: In this present study we conclude that the efficacy of IV streptokinase for thrombolysis in acuteSTEMI and patients with no ST segment resolution at 90 minutes following thrombolysis were associated withmore frequent adverse events and increased mortality compared to partial and complete resolution group.Percentage of resolution of ST segment following 90 minutes of thrombolysis as a diagnostic test helps in riskstratification of patients.
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Ahmad, Munir, Muhammad Yasir, and Asif Rahmat. "ACUTE ST ELEVATION MYOCARDIAL INFARCTION." Professional Medical Journal 25, no. 05 (2018): 777–83. http://dx.doi.org/10.29309/tpmj/2018.25.05.325.

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Objective: To determine the frequency of in-hospital outcomes in patients ofacute ST elevation myocardial infarction (STEMI) within five days of hospitalization with .70ST segment resolution 90 minutes post thrombolysis. Study Design: Case series. Place andDuration of Study: Department of Cardiology, Faisalabad Institute of Cardiology, Faisalabad,from April, 2016 to October, 2016. Methodology: In 370 patients fulfilling the inclusion andexclusion criteria a baseline 12 lead electrocardiogram was recorded before initiation ofthrombolysis and at 90 minutes thereafter. Conventional contraindications to thrombolysis wereobserved and streptokinase 1.5 mu was administered by intravenous infusion over 60 minutes.Successful thrombolysis was taken as 70% or more ST elevation resolution at 90 minutes frombaseline electrocardiogram measured 80ms from J-point. Patients with successful thrombolysiswere observed for in-hospital clinical outcomes of recurrent angina, congestive cardiac failure,ventricular arrhythmia and death within five days of hospitalization. Results: Out of 370 cases,51.35 %( n=190) were male while 48.65 %( n=180) were female, 25.14 %( n=93) were between30-50 years of age while 74.86 %( n=277) were between 51-65 years of age, the mean agewas 54.98+5.96 years. Frequency of in-hospital outcome was recorded as 10.67 %( n=38) forcongestive cardiac failure, 14.59 %( n=54) for ventricular arrhythmia, 5.40 %( n=20) for mortalitywhile no case had recurrent angina. Conclusion: In-hospital outcome is better in patients of.70% ST resolution at 90 minutes post thrombolysis .This might assist in identification of lowrisk patients who can be discharged early and should not be considered for early invasivestrategy.
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Dissertations / Theses on the topic "Acute myocardial infarction; ST segment resolution"

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Dong, Jun. "Extent of early ST-segment elevation resolution correlates with myocardial salvage assessed by Tc 99m sestamibi scintigraphy in patients with acute myocardial infarction after mechanical or thrombolytic reperfusion therapy." [S.l.] : [s.n.], 2003. http://deposit.ddb.de/cgi-bin/dokserv?idn=967546621.

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Nakatsuma, Kenji. "Inter-Facility Transfer vs. Direct Admission of Patients With ST-Segment Elevation Acute Myocardial Infarction Undergoing Primary Percutaneous Coronary Intervention." 京都大学 (Kyoto University), 2017. http://hdl.handle.net/2433/225453.

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Cannistraci, Carlo Vittorio, Tuomo Nieminen, Masahiro Nishi, et al. ""Summer Shift": A Potential Effect of Sunshine on the Time Onset of ST‐Elevation Acute Myocardial Infarction." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2018. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-235086.

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Background: ST-elevation acute myocardial infarction (STEMI) represents one of the leading causes of death. The time of STEMI onset has a circadian rhythm with a peak during diurnal hours, and the occurrence of STEMI follows a seasonal pattern with a salient peak of cases in the winter months and a marked reduction of cases in the summer months. Scholars investigated the reason behind the winter peak, suggesting that environmental and climatic factors concur in STEMI pathogenesis, but no studies have investigated whether the circadian rhythm is modified with the seasonal pattern, in particular during the summer reduction in STEMI occurrence. Methods and Results: Here, we provide a multiethnic and multination epidemiological study (from both hemispheres at different latitudes, n=2270 cases) that investigates whether the circadian variation of STEMI onset is altered in the summer season. The main finding is that the difference between numbers of diurnal (6:00 to 18:00) and nocturnal (18:00 to 6:00) STEMI is markedly decreased in the summer season, and this is a prodrome of a complex mechanism according to which the circadian rhythm of STEMI time onset seems season dependent. Conclusions: The “summer shift” of STEMI to the nocturnal interval is consistent across different populations, and the sunshine duration (a measure related to cloudiness and solar irradiance) underpins this season-dependent circadian perturbation. Vitamin D, which in our results seems correlated with this summer shift, is also primarily regulated by the sunshine duration, and future studies should investigate their joint role in the mechanisms of STEMI etiogenesis.
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Khan, Jamal Nasir. "Cardiovascular Magnetic Resonance Imaging in the assessment of the management of multivessel coronary artery disease in acute ST-segment elevation myocardial infarction." Thesis, University of Leicester, 2016. http://hdl.handle.net/2381/37963.

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Background: Cardiovascular Magnetic Resonance (CMR) comprehensively assesses myocardial injury in ST-segment elevation myocardial infarction (STEMI). Complete revascularization (CR) may improve outcomes compared to an infarct-related artery (IRA)-only strategy in patients with multivessel disease at primary percutaneous coronary intervention (PPCI). However, CR could cause additional non-IRA infarcts. Objectives: To determine optimal techniques for quantifying infarct characteristics and myocardial strain in STEMI. To assess whether in-hospital CR was associated with increased myocardial injury compared to an IRA-only strategy in the CvLPRIT-CMR substudy. To investigate differences in myocardial injury associated with staged and immediate in-hospital CR. To assess CMR predictors of segmental myocardial functional recovery post-STEMI. Methods: Multicentre PROBE-design trial in STEMI patients with multivessel disease and ≤12 hours symptom duration. Patients were randomized to IRA-only PCI or in-hospital CR. Contrast-enhanced CMR was performed at 3 days post-PPCI and stress CMR at 9 months. The pre-specified primary endpoint was infarct size (IS) on acute CMR. Accuracy, feasibility and observer variability for semi-automated CMR methods of quantifying infarct size and area-at-risk (AAR) were assessed. Strain quantification using Feature Tracking and tagging was assessed. Functional recovery in dysfunctional segments was assessed at follow-up CMR on wall-motion scoring. Results: 205 of 296 patients in the main trial participated in CvLPRIT-CMR and 203 (105 IRA, 98 CR) completed acute CMR. There was a strong trend towards reduced AAR in the CR group (p=0.06). Total IS was similar with IRA-only PCI: 13.5% (6.2-21.9%) and CR: 12.6% (7.2-22.6) of LV mass, p=0.57. The CR group had an increased incidence of non-IRA MI at acute CMR (22/98 vs. 11/105, P=0.02). There was no difference in total IS or ischemic burden between the groups at follow-up CMR. Full-width half-maximum, Otsu's Automated Thresholding and Feature Tracking were used for IS, AAR and strain analysis. Immediate CR was associated with reduced IS. Conclusions: In-hospital CR for multivessel disease in STEMI leads to a small increase in CMR non-IRA MI but total IS was not different from an lRA-only PCI strategy. The comparable ischaemic burden in the groups suggests that the similarly improved medium-term clinical outcomes seen in the CvLPRIT, PRAMI and DANAMI-3- PRIMULTI studies are unlikely to be ischaemia-driven and instead may result from stabilization of unstable plaques and improved collateral flow to the ischaemic AAR.
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Watanabe, Hiroki. "Chronic total occlusion in non-infarct-related artery is closely associated with increased five-year mortality in patients with ST-segment elevation acute myocardial infarction undergoing primary percutaneous coronary intervention (From the CREDO-Kyoto AMI registry)." 京都大学 (Kyoto University), 2017. http://hdl.handle.net/2433/225504.

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Ribas, Barquet Núria. "Influència pronòstica del codi infart en els pacients amb infart agut de miocardi amb elevació del segment st anàlisi del control dels factors de risc cardiovascular." Doctoral thesis, Universitat Autònoma de Barcelona, 2018. http://hdl.handle.net/10803/665722.

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Els últims anys s’ha reduït la mortalitat intrahospitalària de l’infart agut de miocardi, en part, gràcies als tractaments de reperfusió, la fibrinòlisi i posteriorment l’intervencionisme coronari percutani. Recentment s’ha invertit de forma significativa en el tractament de la fase aguda de l’infart amb elevació del segment ST amb la creació de xarxes assistencials que prioritzen l’angioplàstia primària com a teràpia de reperfusió com el codi IAM a Catalunya. L’objectiu del codi IAM és que tots els pacients amb IAMEST rebin el tractament de reperfusió més indicat (d’elecció intervencionisme coronari percutani) en els intervals de temps adequats. El codi IAM va entrar en funcionament al juny del 2009. La implementació del codi IAM es va correlacionar amb un augment del percentatge de IAMEST tractats amb teràpia de reperfusió que va passar del 64% al 89%, mitjançant l’ús generalitzat de l’angioplàstia primària (99% respecte 44%, p < 0,001). El codi IAM també es va correlacionar amb un descens significatiu de la mortalitat intrahospitalària (del 7,2% al 2,5%, p < 0,001) si bé no es van apreciar diferències significatives en la mortalitat a llarg termini. Pel que fa a la mortalitat intrahospitalària, es va objectivar com el descens de la mortalitat es va anar produint al llarg del temps de forma progressiva. Per aquest motiu es va afegir aquesta tendència en el temps a l’anàlisi multivariat. El descens de la mortalitat intrahospitalària objectivat en l’era post-Codi IAM va quedar neutralitzat en afegir al model el tractament mèdic òptim (post-Codi IAM: OR 1,14, IC 95% 0,32 – 4,08, p = 0,840), suggerint que el tractament mèdic pot ser tan important com la reperfusió per disminuir la mortalitat intrahospitalària. Després de la implementació del codi IAM es va apreciar com només un 62% dels pacients tenien un control adequat de la pressió arterial, un 29% tenien el cLDL per sota dels nivells desitjats, un 60% dels fumadors havien deixat de fumar i un 36% dels diabètics tenien la hemoglobina glicosilada dins els marges de referència a l’any del seguiment. Dels malalts que van sobreviure als 6 mesos de l’IAMEST, un 6% van morir i 11% van reingressar per causa cardiovascular després de 20 mesos. L’absència de determinació de cLDL i cHDL en el seguiment es va associar amb un pitjor pronòstic a llarg termini. D’altra banda, també es va avaluar el control de la dislipèmia en la primera analítica de seguiment després d’una síndrome coronària aguda i la utilitat d’una eina clínica de fàcil aplicabilitat (les taules de Masana) per millorar el control de la dislipèmia. Als quatre mesos d’una síndrome coronària aguda, un 45% dels pacients va assolir l’objectiu de cLDL, essent aquest percentatge major quan el tractament fou planificat segons les recomanacions de Masana (56% respecte 30%, p < 0,001). En l’anàlisi multivariant, el gènere masculí (p < 0,001), l’absència de dislipèmia prèvia (p < 0,001) i l’aplicació de les taules de Masana (p = 0,007) foren predictors independents per assolir el cLDL objectiu. Les troballes d’aquests treballs ens haurien de fer reconsiderar l’actitud i el tractament dels pacients amb IAMEST: mentre la implementació del codi IAM s’ha acompanyat de millores en el maneig i pronòstic en la fase aguda, el potencial benefici pronòstic a llarg termini podria estar interferit per un control inadequat dels factors de risc cardiovascular. Els nostres resultats suggereixen que caldria unir esforços per promoure la implementació adequada de les mesures de prevenció secundària. En aquest context, l’ús d’eines de fàcil aplicabilitat com les taules de Masana per ajustar la teràpia hipolipemiant pot millorar el control de la dislipèmia en aquests pacients que, a dia d’avui, és clarament insuficient.<br>In recent years, the in-hospital mortality of ST-elevation acute myocardial infarction (STEMI) has been decreased, in large part, mostly due to reperfusion therapy, initially fibrinolytic treatment and later, primary percutaneous coronary intervention (PPCI). Recently, the treatment of the acute phase of the STEMI has been significantly invested with the creation of healthcare reperfusion networks that prioritize PPCI, such as codi IAM (STEMI ntework) in Catalonia. The purpose of the STEMI network is to ensure that every patient with STEMI receives the most appropriate reperfusion therapy with the appropriate time intervals through a territorial sectorisation of the flow of patients. STEMI network became operative in June 2009. The implementation of STEMI network was correlated with an increase in the percentage of STEMI patients treated with reperfusion therapy that went from 64% to 89% due to the widespread use of PPCI (99% with respect to 44%, p < 0.001). STEMI network was also associated with a significant decrease in in-hospital mortality (from 7.2% to 2.5%, p < 0.001) although no significant differences were appreciated in long-term mortality. Regarding in-hospital mortality, there was a progressive decrease over the years, this tendency was added to multivariate analysis. The decrease in in-hospital mortality observed after implementation of STEMI network was neutralized when incorporating optimal medical treatment to the model (post STEMI code: OR 1.14, 95% CI 0.32 - 4.08, p = 0.840), suggesting that optimal medical treatment can be as important as reperfusion to decrease in-hospital mortality. After the implementation of STEMI network, only 62% of the patients had blood pressure under control, 29% had LDL cholesterol below the desired levels, 60% of smokers had quitted smoking and 36% of diabetic patients had glycosylated haemoglobin within the therapeutic objective after one-year follow-up. The accumulated mortality of 6-months survivors was 6% and 11% of patients were readmitted by cardiovascular disease at 20 months of clinical follow-up. Additionally, an inadequate assessment of LDL cholesterol and HDL cholesterol levels was associated with less favourable long-term cardiovascular outcome after STEMI. On the other hand, usefulness of an easy clinical applicability tool (like Masana tables) to improve the control of dyslipidaemia after an acute coronary syndrome was assessed. After 4 months of follow-up, 45% of patients achieved the objective of LDL cholesterol, being this percentage highest when treatment was planned according to Masana recommendations (56% with respect to 30%, p < 0,001). In multivariate analysis, male gender (p < 0.001), the absence of previous dyslipidaemia (p < 0.001) and the application of Masana tables (p = 0.007) were independent predictors to achieve LDL cholesterol objective. Our findings should make us reconsider the current therapeutic yield of urgent myocardial reperfusion strategies in the setting of a STEMI: while the implementation of assistance networks (such as the STEMI code program) has improved the acute management of STEMI, the potentially beneficial impact of such strategies may be limited by a suboptimal long-term implementation of the secondary prevention strategies. In this sense, usefulness of an easy clinical applicability tool (like Masana tables) can help clinicians achieve dyslipidaemia control which nowadays is clearly insufficient.
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Dong, Jun [Verfasser]. "Extent of early ST-segment elevation resolution correlates with myocardial salvage assessed by Tc 99m sestamibi scintigraphy in patients with acute myocardial infarction after mechanical or thrombolytic reperfusion therapy / Jun Dong." 2003. http://d-nb.info/967546621/34.

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Thongsna, Snong. "The significance of epicardial ST-segment depression in acute myocardial infarction." Thesis, 2000. https://eprints.utas.edu.au/22052/1/whole_ThongsnaSnong2000_thesis.pdf.

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Owusu, Yaw Boahene. "Incident coronary atherosclerosis, unstable angina, non-ST-segment elevation myocardial infarction or ST-segment elevation myocardial infarction in type 2 diabetes : is mean glycated hemoglobin a good predictor?" Thesis, 2010. http://hdl.handle.net/2152/ETD-UT-2010-12-2067.

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Background: Glycated hemoglobin is the indicator of long-term diabetes control and a value below 7 percent is recommended by the American Diabetes Association (ADA) to reduce cardiovascular complications. Diabetic patients have a two- to four-fold risk of cardiovascular disease and approximately two-thirds of diabetic patients die as a result of cardiovascular complications. Three large prospective randomized controlled long-term trials within the last decade reported no significant reduction in cardiovascular complications in type 2 diabetic patients by intensive glycemic control. To the author's knowledge, no known retrospective studies have examined the association between mean serial glycated hemoglobin and coronary atherosclerosis (CA) or acute coronary syndromes (ACS). Objective: This study was designed to determine the association between mean serial glycated hemoglobin with incident CA or ACS in type 2 diabetic patients after controlling for age, gender, hypertension, low density lipoprotein cholesterol (LDL-C), microalbuminuria, aspirin use, statin use, insulin use, tobacco use, and body mass index (BMI). Methods: The study was a retrospective cohort database analysis using the Austin Travis County CommUnityCare[trademark] clinics' electronic medical record for the time period between October 1, 2004 and September 30, 2009. The primary outcome of the study was the incidence of CA or ACS and the primary independent variable was glycated hemoglobin (<7% vs. [greater than or equal to]7%). The study subjects included type 2 diabetic patients aged 30 to 80 years with at least one glycated hemoglobin value per year for a minimum of two consecutive years. Study subjects were excluded if CA or ACS occurred within six months of the index date (i.e., first glycated hemoglobin). Logistic regression analysis was used to address the study objective. Results: Overall, 3069 subjects met the study inclusion criteria with a mean follow-up period of approximately two years. Two percent (N=62) of the subjects had incident CA or ACS. After controlling for age, gender, hypertension diagnosis, LDL-C, microalbuminuria, aspirin use, statin use, insulin use, tobacco use and BMI, there was no significant association (OR=1.026, 95% CI=0.589-1.785, p=0.9289) between mean serial glycated hemoglobin and the incident diagnosis of CA or ACS. Increasing age (OR=1.051, 95% CI=1.025-1.077, p<0.0001), male gender (OR=1.855, 95% CI=1.105-3.115, p=0.0195) and normal weight (normal or underweight compared to obese: OR=0.122, 95% CI=0.017-0.895, p=0.0438) were significantly associated with incident CA or ACS. Conclusions: Mean serial glycated hemoglobin (comparing [greater than or equal to]7% to <7%) was not significantly associated with CA or ACS over a mean follow-up period of approximately two years. Until more evidence becomes available, clinicians and diabetic patients should target glycated hemoglobin level below or close to 7 percent as recommended by the ADA soon after diagnosis while concomitantly controlling nonglycemic risk factors of cardiovascular disease (statin use, aspirin use, blood pressure control, smoking cessation and life style modification), to reduce their long-term risk of incident CA or ACS.<br>text
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Chen, Yen-Hsun, and 陳彥勳. "Characteristics of patients early discharged with acute ST segment elevation myocardial infarction undergoing successful primary percutaneous coronary intervention." Thesis, 2012. http://ndltd.ncl.edu.tw/handle/88344143810466819176.

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Books on the topic "Acute myocardial infarction; ST segment resolution"

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Veldkamp, Rolf Frederik. Continuous digital 12-lead ST-segment monitoring in Acute Myocardial Infarction =: Continue digitale 12-afleidingen ST-segment bewaking tijdens het acute myocard infarct : proefschrift. The Author, 1995.

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Cheong, Adrian, Gabriel Steg, and Stefan K. James. ST-segment elevation myocardial infarction. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0043.

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Acute myocardial infarction with ST-segment elevation is a common and dramatic manifestation of coronary artery disease. It is caused by the rupture of an atherosclerotic plaque in a coronary artery, leading to its total thrombotic occlusion and resultant ischaemia and necrosis of downstream myocardium. The diagnosis of ST-segment elevation myocardial infarction is based on a syndrome of ischaemic chest pain symptoms, associated with typical ST-segment elevation on the electrocardiogram and an eventual rise in biomarkers of myocardial necrosis. The treatment of ST-segment elevation myocardial infarction is focused on re-establishing blood flow in the coronary artery involved, preferably by percutaneous coronary intervention, or by pharmacological thrombolysis in the case of expected lengthy time delays or lack of availability of facilities. Early mortality from ST-segment elevation myocardial infarction can be attributed to the sequelae or complications of myocardial ischaemia, or complications related to therapy. The former include arrhythmias (such as ventricular tachycardia or fibrillation), mechanical complications (such as ventricular free wall, septal, and mitral chordal rupture), and pump failure leading to cardiogenic shock. The latter includes haemorrhagic complications and coronary stent thrombosis. Given that myocardial necrosis is a critically time-dependent process, the organization of an ST-segment elevation myocardial infarction care system and adherence to the latest clinical trial evidence and guidelines are crucial to ensure that patients are treated in an optimal manner.
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Bueno, Héctor, and José A. Barrabés. Non-ST-segment elevation acute coronary syndromes. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0046.

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Non-ST-segment elevation acute coronary syndromes are life-threatening disorders, usually caused by acute coronary thrombosis and subsequent myocardial ischaemia, presenting without persistent ST-segment elevation in the initial electrocardiogram. According to the occurrence of myocardial necrosis, non-ST-segment elevation acute coronary syndromes are divided into non-ST-segment myocardial infarction or unstable angina. The management of non-ST-segment elevation acute coronary syndromes requires an early diagnosis and risk stratification, urgent hospitalization, monitoring, and medical treatment, including antithrombotic therapy with dual antiplatelet therapy (aspirin plus one P2Y12 inhibitor) and parenteral anticoagulation, anti-ischaemic treatment, and preventative therapies. After the initial medical therapy is established, an invasive strategy, consisting of coronary angiography with coronary revascularization (either percutaneous coronary intervention or coronary bypass graft surgery), as appropriate, should be decided. The timing of the invasive strategy should be adjusted, according to the patient’s risk. Given the high event rate of patients with non-ST-segment elevation acute coronary syndromes after hospital discharge, an aggressive long-term preventative therapy should be put in place to improve prognosis.
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Bueno, Héctor, and José A. Barrabés. Non-ST-segment elevation acute coronary syndromes. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199687039.003.0046_update_001.

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Non-ST-segment elevation acute coronary syndromes are life-threatening disorders, usually caused by acute coronary thrombosis and subsequent myocardial ischaemia, presenting without persistent ST-segment elevation in the initial electrocardiogram. According to the occurrence of myocardial necrosis, non-ST-segment elevation acute coronary syndromes are divided into non-ST-segment myocardial infarction or unstable angina. The management of non-ST-segment elevation acute coronary syndromes requires an early diagnosis and risk stratification, urgent hospitalization, monitoring, and medical treatment, including antithrombotic therapy with dual antiplatelet therapy (aspirin plus one P2Y12 inhibitor) and parenteral anticoagulation, anti-ischaemic treatment, and preventative therapies. After the initial medical therapy is established, an invasive strategy, consisting of coronary angiography with coronary revascularization (either percutaneous coronary intervention or coronary bypass graft surgery), as appropriate, should be decided. The timing of the invasive strategy should be adjusted, according to the patient’s risk. Given the high event rate of patients with non-ST-segment elevation acute coronary syndromes after hospital discharge, an aggressive long-term preventative therapy should be put in place to improve prognosis.
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Bueno, Héctor, and José A. Barrabés. Non-ST-segment elevation acute coronary syndromes. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0046_update_002.

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Non-ST-segment elevation acute coronary syndromes are life-threatening disorders, usually caused by acute coronary thrombosis and subsequent myocardial ischaemia, presenting without persistent ST-segment elevation in the initial electrocardiogram. According to the occurrence of myocardial necrosis, non-ST-segment elevation acute coronary syndromes are divided into non-ST-segment myocardial infarction or unstable angina. The management of non-ST-segment elevation acute coronary syndromes requires an early diagnosis and risk stratification, urgent hospitalization, monitoring, and medical treatment, including antithrombotic therapy with dual antiplatelet therapy (aspirin plus one P2Y12 inhibitor) and parenteral anticoagulation, anti-ischaemic treatment, and preventative therapies. After the initial medical therapy is established, an invasive strategy, consisting of coronary angiography with coronary revascularization (either percutaneous coronary intervention or coronary bypass graft surgery), as appropriate, should be decided. The timing of the invasive strategy should be adjusted, according to the patient’s risk. Given the high event rate of patients with non-ST-segment elevation acute coronary syndromes after hospital discharge, an aggressive long-term preventative therapy should be put in place to improve prognosis.
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Cheong, Adrian P., Gabriel Steg, and Stefan K. James. ST-segment elevation MI. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0043_update_001.

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Acute myocardial infarction with ST-segment elevation is a common and dramatic manifestation of coronary artery disease. It is caused by the rupture of an atherosclerotic plaque in a coronary artery, leading to its total thrombotic occlusion and resultant ischaemia and necrosis of downstream myocardium. The diagnosis of ST-segment elevation myocardial infarction is based on a syndrome of ischaemic chest pain symptoms, associated with typical ST-segment elevation on the electrocardiogram and an eventual rise in biomarkers of myocardial necrosis. The treatment of ST-segment elevation myocardial infarction is focused on re-establishing blood flow in the coronary artery involved, preferably by percutaneous coronary intervention, or by pharmacological thrombolysis in the case of expected lengthy time delays or lack of availability of facilities. Early mortality from ST-segment elevation myocardial infarction can be attributed to the sequelae or complications of myocardial ischaemia, or complications related to therapy. The former include arrhythmias (such as ventricular tachycardia or fibrillation), mechanical complications (such as ventricular free wall, septal, and mitral chordal rupture), and pump failure leading to cardiogenic shock. The latter includes haemorrhagic complications and coronary stent thrombosis. Given that myocardial necrosis is a critically time-dependent process, the organization of an ST-segment elevation myocardial infarction care system and adherence to the latest clinical trial evidence and guidelines are crucial to ensure that patients are treated in an optimal manner.
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Erlinge, David, and Göran Olivecrona. Diagnosis and management of ST-elevation of myocardial infarction. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0147.

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ST-elevation myocardial infarction (STEMI) is generally caused by a ruptured plaque that triggers local thrombus formation, which occludes the coronary artery. STEMI should be diagnosed rapidly, based on the combination of ST-segment elevation and symptoms of acute myocardial infarction. The main treatment objective is myocardial tissue reperfusion as quickly as possible. The preferred method of reperfusion is primary percutaneous coronary interventionif transport time is below 2 hours, and thrombolysis if longer STEMI patients with acute onset cardiogenic shock should be evaluated by echocardiography to exclude mechanical complications, such as flail mitral insufficiency, ventricular septal defect or tamponade. Secondary prevention includes aspirin, adenosine diphosphate receptor antagonists, statins, beta-blockers, angiotensin-converting enzymeinhibitors, and lifestyle changes.
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Kočka, Viktor, Steen Dalby Kristensen, William Wijns, Petr Toušek, and Petr Widimský. Percutaneous coronary interventions in acute coronary syndromes. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0047.

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Three different guidelines of the European Society of Cardiology cover the field of percutaneous coronary interventions. Their main recommendations are the following:All patients with an ST-segment elevation myocardial infarction should undergo immediate coronary angiography and percutaneous coronary intervention as soon as possible after the first medical contact. Thrombolysis can be used as an alternative reperfusion therapy if the time delay to primary percutaneous coronary intervention is more than 2 hoursPatients with very high-risk non-ST-segment elevation acute coronary syndromes (recurrent or ongoing chest pain, profound or dynamic electrocardiogram changes, major arrhythmias, or haemodynamic instability) should undergo urgent coronary angiography within less than 2 hours after the initial hospital admissionAll moderate- to high-risk (GRACE score &gt;140 or at least one primary high-risk criterion) non-ST-segment elevation acute coronary syndromes patients should undergo coronary angiography before discharge; the ideal timing is within 24 hours after admission for high-risk groups, and within 72 hours for moderate-risk groupsOther patients with recurrent symptoms or at least one high-risk criterion should undergo coronary angiography within 72 hours of first presentationLow-risk non-ST-segment elevation acute coronary syndromes may be treated conservatively, and the indication for an invasive evaluation can be done, based on the evidence of ischaemia during exercise stress testingStents should be used during all percutaneous coronary intervention procedures, whenever technically feasible. Second-generation drug-eluting stents do not increase stent thrombosis and can be safely used in the ST-segment elevation myocardial infarction and non-ST-segment elevation acute coronary syndrome settingsTriple pharmacotherapy, consisting of aspirin, thienopyridine antiplatelet agent, and anticoagulation with heparin or bivalirudin, should be used in all percutaneous coronary intervention procedures, with glycoprotein IIb/IIIa inhibitors added in patients with a high thrombus burden and low bleeding risk
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Kočka, Viktor, Steen Dalby Kristensen, William Wijns, Petr Toušek, and Petr Widimský. Percutaneous coronary interventions in acute coronary syndromes. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199687039.003.0047_update_001.

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Three different guidelines of the European Society of Cardiology cover the field of percutaneous coronary interventions. Their main recommendations are the following:All patients with an ST-segment elevation myocardial infarction should undergo immediate coronary angiography and percutaneous coronary intervention as soon as possible after the first medical contact. Thrombolysis can be used as an alternative reperfusion therapy if the time delay to primary percutaneous coronary intervention is more than 2 hoursPatients with very high-risk non-ST-segment elevation acute coronary syndromes (recurrent or ongoing chest pain, profound or dynamic electrocardiogram changes, major arrhythmias, or haemodynamic instability) should undergo urgent coronary angiography within less than 2 hours after the initial hospital admissionAll moderate- to high-risk (GRACE score &gt;140 or at least one primary high-risk criterion) non-ST-segment elevation acute coronary syndromes patients should undergo coronary angiography before discharge; the ideal timing is within 24 hours after admission for high-risk groups, and within 72 hours for moderate-risk groupsOther patients with recurrent symptoms or at least one high-risk criterion should undergo coronary angiography within 72 hours of first presentationLow-risk non-ST-segment elevation acute coronary syndromes may be treated conservatively, and the indication for an invasive evaluation can be done, based on the evidence of ischaemia during exercise stress testingStents should be used during all percutaneous coronary intervention procedures, whenever technically feasible. Second-generation drug-eluting stents do not increase stent thrombosis and can be safely used in the ST-segment elevation myocardial infarction and non-ST-segment elevation acute coronary syndrome settingsTriple pharmacotherapy, consisting of aspirin, thienopyridine antiplatelet agent, and anticoagulation with heparin or bivalirudin, should be used in all percutaneous coronary intervention procedures, with glycoprotein IIb/IIIa inhibitors added in patients with a high thrombus burden and low bleeding risk
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Kočka, Viktor, Steen Dalby Kristensen, William Wijns, Petr Toušek, and Petr Widimský. Percutaneous coronary interventions in acute coronary syndromes. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0047_update_002.

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Abstract:
Three different guidelines of the European Society of Cardiology cover the field of percutaneous coronary interventions. Their main recommendations are the following: All patients with an ST-segment elevation myocardial infarction should undergo immediate coronary angiography and percutaneous coronary intervention as soon as possible after the first medical contact. Thrombolysis can be used as an alternative reperfusion therapy if the time delay to primary percutaneous coronary intervention is more than 2 hours. Patients with very high-risk non-ST-segment elevation acute coronary syndromes (recurrent or ongoing chest pain, profound or dynamic electrocardiogram changes, major arrhythmias, or haemodynamic instability) should undergo urgent coronary angiography within less than 2 hours after the initial hospital admissionAll moderate- to high-risk (GRACE score &gt;140 or at least one primary high-risk criterion) non-ST-segment elevation acute coronary syndromes patients should undergo coronary angiography before discharge; the ideal timing is within 24 hours after admission for high-risk groups, and within 72 hours for moderate-risk groups. Other patients with recurrent symptoms or at least one high-risk criterion should undergo coronary angiography within 72 hours of first presentation. Low-risk non-ST-segment elevation acute coronary syndromes may be treated conservatively, and the indication for an invasive evaluation can be done, based on the evidence of ischaemia during exercise stress testing. Stents should be used during all percutaneous coronary intervention procedures, whenever technically feasible. Second-generation drug-eluting stents do not increase stent thrombosis and can be safely used in the ST-segment elevation myocardial infarction and non-ST-segment elevation acute coronary syndrome settings. Triple pharmacotherapy, consisting of aspirin, thienopyridine antiplatelet agent, and anticoagulation with heparin or bivalirudin, should be used in all percutaneous coronary intervention procedures, with glycoprotein IIb/IIIa inhibitors added in patients with a high thrombus burden and low bleeding risk.
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Book chapters on the topic "Acute myocardial infarction; ST segment resolution"

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Gelfand, Eli V., and Christopher P. Cannon. "ST-Segment-Elevation Myocardial Infarction." In Management of Acute Coronary Syndromes. John Wiley & Sons, Ltd, 2009. http://dx.doi.org/10.1002/9780470745465.ch4.

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Nguyen, James, Marko Noc, Thach N. Nguyen, et al. "Acute ST-Segment Elevation Myocardial Infarction." In Practical Handbook of Advanced Interventional Cardiology. Blackwell Publishing Ltd., 2013. http://dx.doi.org/10.1002/9781118592380.ch12.

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Krucoff, Mitchell W., Lowell F. Satler, Curtis E. Green, Charles E. Rackley, and Kenneth M. Kent. "St Segment Changes during Early Myocardial Infarction." In Acute Coronary Care 1987. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-2337-2_9.

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Lee, Justin, Felix Reyes, and Adam S. Budzikowski. "Acute ST-Segment Elevation Myocardial Infarction (STEMI)." In Cardiology Consult Manual. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-89725-7_7.

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Sawh, Chris, Shabnam Rashid, James Palmer, and Ever D. Grech. "Acute coronary syndrome: Acute ST-segment elevation myocardial infarction." In Practical Interventional Cardiology. CRC Press, 2017. http://dx.doi.org/10.1201/9781315113753-20.

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Daka, Matthew, Emad Aziz, Robert Leber, and Mun K. Hong. "Diagnosis and Treatment of ST-Segment Elevation Myocardial Infarction." In Acute Coronary Syndrome. Springer London, 2008. http://dx.doi.org/10.1007/978-1-84628-869-2_6.

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Pollack, Charles V., Richard M. Cantor, and Victoria G. Riese. "Acute Coronary Syndrome: ST-Segment Elevation Myocardial Infarction." In Differential Diagnosis of Cardiopulmonary Disease. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-63895-9_3.

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Tamis-Holland, Jacqueline E., Sandeep Joshi, Angela Palazzo, and Sripal Bangalore. "Diagnosis and Treatment of Non-ST-Segment Elevation Myocardial Infarction." In Acute Coronary Syndrome. Springer London, 2008. http://dx.doi.org/10.1007/978-1-84628-869-2_7.

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Galla, John M., and Arman T. Askari. "Antithrombin Therapy for Acute ST-Segment Elevation Myocardial Infarction." In Antithrombotic Drug Therapy in Cardiovascular Disease. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-235-3_12.

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Winiger, Amy, and George P. Rodgers. "Acute Coronary Syndrome (ACS) ST Segment Elevation Myocardial Infarction." In Cardiovascular Manual for the Advanced Practice Provider. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-35819-7_3.

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Conference papers on the topic "Acute myocardial infarction; ST segment resolution"

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ZORKUN, CAFER, KRZYSZTOF ZMUDKA, MIECZYSLAW PASOWICZ, and WIESLAWA TRACZ. "COMBINATION OF ST SEGMENT RESOLUTION AND BASELINE TROPONIN I LEVEL PREDICTS FATAL EVENTS AT 1 YEAR IN ACUTE MYOCARDIAL INFARCTION." In Proceedings of the 31st International Congress on Electrocardiology. WORLD SCIENTIFIC, 2005. http://dx.doi.org/10.1142/9789812702234_0100.

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Joseph, T., B. Kim, C. Fox, et al. "Acute Severe EBV Myo-Pericarditis Presentation as ST Segment Elevation Myocardial Infarction." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a3528.

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Kohring, A. S., J. R. Bank, C. Burke, and H. Azar. "Acute ST-Segment Elevation Myocardial Infarction in a Young Postinfectious COVID-19 Patient." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a1676.

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Machado Filho, Delfino da Costa, and Thelma da Costa. "Acute pulmonary edema associated with acute coronary syndrome - a case report." In II INTERNATIONAL SEVEN MULTIDISCIPLINARY CONGRESS. Seven Congress, 2023. http://dx.doi.org/10.56238/homeinternationalanais-001.

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Abstract Objective: To report manifestation of acute myocardial infarction with supradenive lament in evolved TS, associated with acute hypertensive pulmonary edema in an adult and smoker patient. Detailing of the case: A 55-year-old smoker (60 years/pack), with a history of myocardial infarction for 15 days, was admitted to the cardiac emergency on 18/07/2022, in severe acute pulmonary edema associated with arterial hypertension. The electrocardiogram (ECG) showed extensive anterior ST-segment unevenness. After measures to stabilize the hemodynamic condition, he underwent cardiac catheterization and angioplasty to the artery responsible for the cardiac event. He underwent a control echocardiogram that showed segmental alterations and an important reduction in the ventricular ejection fraction. Conclusions: Acute myocardial infarction is one of the main causes of morbidity and mortality in Brazil, however, if this emergency is conducted appropriately, within the time limit stipulated by the ACLS protocol, many lives will be saved.
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Firoozabadi, Reza, Richard Gregg, and Saeed Babaeizadeh. "Modeling and Classification of the ST Segment Morphology for Enhanced Detection of Acute Myocardial Infarction." In 2019 Computing in Cardiology Conference. Computing in Cardiology, 2019. http://dx.doi.org/10.22489/cinc.2019.005.

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Kordenat, K., and J. Leasure. "PROTECTIVE EFFECT OF CARNITINE (ST-261, SIGMA-TAU) IN ACUTE MYOCARDIAL INFARCTION IN DOGS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643012.

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Limitation of infarct size (IS), using ST-261, was evaluated in a group (I) of eight dogs, during acute MI. Another group (II) served as the control group. The protocol for both groups was the same except that each dog in the treated group was ST-261 as a single bolus (25 mg/kg, in 20ml normal saline), before inducing an occluding thrombus in the mid-LAD, using a closed-chest model, under x-ray visualization. Percentages of total (gms) myocardium at jeopardy (TMJW) and myocardial necrosis (TMNW), delineated by fluoroscein and TTC dyes, respectively, were calculated and compared to the total ventricular myocardial weight (TVMU), by computer technique for both groups at 3 Hrs post-occlusion of the LAD. Mean serum total CPK (CPK-t) and isozymes (mb-band) were measured before and up to 3 Hrs post-occlusion, as were various hemodynamic and mean precordial (21 lead) ST-segment and T-wave amplitudes. There was 14% less TMJU (p&lt;0.05) and 41% less TMNW (p&lt;0.01) in Group I compared to Group II. The mean % of CPK-mb/CPK-t decreased in I and increased in II over the 3 Hrs of observation. Mean HR decreased (p&lt;0.01) in I compared to II at 3 Hrs postocclusion. The sum of the mean T-wave amplitudes from the precordial electrode sites was less in I at 3 Hrs. It is felt that ST-261 had a protective effect on the myocardium during acute myocardial infarction.
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Resende, L. O., E. S. Resende, and A. O. Andrade. "Assessment of the ST segment deviation area as a potential physiological marker of the acute myocardial infarction." In 2012 34th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2012. http://dx.doi.org/10.1109/embc.2012.6346020.

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Kassim, Aya, Michael Arzt, Henrik Fox, Clemens Wiest, Stefan Buchner, and Stefan Stadler. "Resolution of ST deviation in patients with and without sleep disordered breathing early after acute myocardial infarction." In ERS International Congress 2023 abstracts. European Respiratory Society, 2023. http://dx.doi.org/10.1183/13993003.congress-2023.pa2997.

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Figueras, J., J. Cortadellas, and Y. Monasterio. "REDUCTION OF EARLY MORTALITY AND OF CARDIAC RUPTURE IN ACUTE TRANSMURAL MYOCARDIAL INFARCTION BY INTRAVENOUS STREPTOKINASE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642990.

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Patients (Pts)≤ 70 years old with a first transmural AMI of ≤ 4h (164±55 min) were randomized to receive (Group I,GI n=105) or not (GII, n=102) i.v. streptokinase (SK, 840.000U in lh).Contrc ST segment elevation and at lh and 24h after admission were comparable in both groups. Coronary arteriography performed within 15 days showed a recanalization rate of 64% in GI and of 27% in GII (p&lt;0.001)but an incidence of severe stenosis (≥90&lt;100%) higher in GI (46 vs 22%, p&lt;0.01).Recanalized Pts presented an earlier peak of MB creatin kinase in GI (12 vs 16h p&lt;0.01) as well as in GII (15 vs 21h, p&lt;0.002). The incidence of pericarditis was lower in GI (14 vs 35%, p&lt;0.001). Although hospital mortality was comparable in the 2 groups (GI,8% vs GII,11%), early mortality, &lt;5 days, was lower in GI (2 vs 10%, p&lt;0.02). Sudden electromechanical dissociation was the mechanism of death in 12% of patients from GI and in 77% of those from GII and it was associated with left ventricular free wall rupture in each of the 5 autopsied cases but in none of the 5 autopsied cases who died without electromechanical dissociation During a follow-up of 20±11 months (1-36) , mortality an incidence of angina was similar in both groups but reinfarction rate was higher in GI (16 vs 1%, p&lt;0.05).It is concluded that: 1) In contrast with the changes in ST .segment, an early MB creatin kinase peak is a reliable marker of reperfusion; 2) i.v. SK lowers the incidence of pericarditis and of early mortality reducing the incidence of cardiac rupture; and 3) It is conceivable that early treatment of critical residual stenosis will reduce in hospital mortality and reinfarction in these Pts.
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Steele, Lloyd, James Palmer, Amelia Lloyd, James Fotheringham, Javaid Iqbal, and Ever Grech. "17 Socioeconomic status and its influence on survival after acute ST-segment myocardial infarction treated with primary percutaneous coronary intervention." In British Cardiovascular Society Annual Conference ‘High Performing Teams’, 4–6 June 2018, Manchester, UK. BMJ Publishing Group Ltd and British Cardiovascular Society, 2018. http://dx.doi.org/10.1136/heartjnl-2018-bcs.17.

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