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Dissertations / Theses on the topic 'Acute Respiratry Distress Syndrome'

Author: Grafiati

Published: 9 March 2023

Last updated: 31 July 2025

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1

Dushianthan, Ahilanandan. "Phospholipid kinetics in acute respiratory distress syndrome." Thesis, University of Southampton, 2014. https://eprints.soton.ac.uk/374568/.

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2

Henderson, William Roy. "Expiratory time constant heterogeneity in experimental acute respiratory distress syndrome." Thesis, University of British Columbia, 2016. http://hdl.handle.net/2429/58370.

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Purpose This thesis evaluated regional heterogeneity of pulmonary mechanical values within models of lung injury. To this end four separate studies were completed. I evaluated regional expiratory time constant (τE) heterogeneity and tissue strain (ε) in a lung model using functional respiratory imaging (FRI) (Study 1, Chapter 2), and developed an in vivo porcine model of lung injury (Study 2, Chapter 3). This model was used to assess changes in τE due to manipulations of respiratory gas density (Study 3, Chapter 4) and mechanical ventilation parameters (Study 4, Chapter 5). Methods Study

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3

Camprubí, Rimblas Marta. "Nebulized anti-coagulants as a therapy for acute lung injury and acute respiratory distress syndrome." Doctoral thesis, Universitat Autònoma de Barcelona, 2018. http://hdl.handle.net/10803/663961.

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La síndrome de distrés respiratori agut (ARDS) és una insuficiència respiratòria aguda amb una incidència global a Europa de 17,9 per cada 100.000 persones-any. Tot i els avenços en el tractament de suport dels pacients amb ARDS, la mortalitat continua sent alta (40%) i els pacients que sobreviuen presenten seqüeles persistents. Actualment no existeix un tractament efectiu. La fisiopatologia de l’ARDS es caracteritza per l’activació de la coagulació i la inflamació a nivell pulmonar, juntament amb el trencament de la barrera alveolar-capil·lar. Això comporta la formació d’edema proteic, la in

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4

Peters, Mark John. "The role of platelets in acute inflammation." Thesis, University College London (University of London), 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.271069.

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5

Greene, Michelle Kathleen. "Therapeutic evaluation of an immunomodulatory nanoparticle in acute respiratory distress syndrome." Thesis, Queen's University Belfast, 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.680123.

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Acute Respiratory Distress Syndrome (ARDS) is characterised by dysregulated inflammation within the lungs, which can severely compromise pulmonary architecture and function. Despite intensive efforts to develop therapies for ARDS, there are no curative pharmacological interventions at present. Instead, management of ARDS is primarily supportive and the syndrome is associated with substantial mortality, morbidity and healthcare expenditure. In pursuit of an effective ARDS therapy, this thesis explored the potential application of a novel sialic acid-functionalised nanoparticle (SNP). This nanop

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6

Quinlan, Gregory John. "Oxidative damage to extracellular proteins and lipids during acute lung injury." Thesis, Imperial College London, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.281713.

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7

Aggarwal, Anjna. "Mediators and mechanisms of persistent pulmonary neutrophilia in acute lung injury." Thesis, Imperial College London, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.289880.

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8

Singh, Nanak. "Incidence and pathogenesis of acute lung injury and the acute respiratory distress syndrome in humans." Thesis, University of East Anglia, 2013. https://ueaeprints.uea.ac.uk/48050/.

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Acute lung injury (ALI) and it’s more severe form, acute respiratory distress syndrome (ARDS), are conditions characterised by neutrophilic pulmonary inflammation, refractory hypoxaemia and diffuse alveolar damage. This work reports on the first UK based prospective study of the incidence and longer term mortality of ALI/ARDS in a general intensive care unit. Results reveal significant under recognition of this condition, which occurred in 12.5% of the ICU population (n=344). Hospital and 2 year mortality rates were 50-55% and 58-61% respectively. Neutrophils are central in the pathogenesis of

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9

Jordan, Simon James. "The pathogenesis of lung injury following cardiothoracic surgery." Thesis, Imperial College London, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.249727.

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10

Crag, Thelma R. "Investigation into the mechanisms and treatment of acute living injury and the acute respiratory distress syndrome." Thesis, Queen's University Belfast, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.534709.

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11

Rocksʹen, David. "Acute lung injury : study of pathogenesis and therapeutic interventions /." Umeå : Univ, 2003. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-161.

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12

Baker, Cathy Sue. "Rationale for surfactant replacement therapy in patients with acute lung injury." Thesis, Imperial College London, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.243281.

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13

Jia, Xiaoming M. Eng Massachusetts Institute of Technology. "The effects of mechanical ventilation on the development of Acute Respiratory Distress Syndrome." Thesis, Massachusetts Institute of Technology, 2007. http://hdl.handle.net/1721.1/41629.

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Thesis (M. Eng.)--Massachusetts Institute of Technology, Dept. of Electrical Engineering and Computer Science, 2007.<br>Includes bibliographical references (p. 74-76).<br>Acute Respiratory Distress Syndrome (ARDS) is a severe lung illness characterized by inflammation and fluid accumulation in the respiratory system. Historically, ARDS and other forms of respiratory failure have been treated using mechanical ventilation to help maintain gas exchange in the lungs. However, clinical investigators are beginning to discover the adverse effects of mechanical ventilation if it is not applied properl

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14

BELLANI, GIACOMO. "Imaging of lung metabolic activity by means of positron emission tomography during acute lung injury." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2010. http://hdl.handle.net/10281/7887.

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Neutrophilic inflammation plays a key role in the pathogenesis of acute respiratory distress syndrome (ARDS) and acute lung injury (ALI). Positron emission tomography (PET) with [F]-fluoro-2-deoxy-D-glucose (FDG) can be used to image cellular metabolism that, during lung inflammatory processes, likely reflects neutrophils activity. The aim of this study was to assess the magnitude and regional distribution of inflammatory metabolic activity in the lungs of patients with ALI/ARDS by PET with FDG. Fifteen patients with ALI/ARDS were enrolled, while four spontaneously breathing and two mechanical

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15

Zhai, Ruoyang. "Effects of sevoflurane in the treatment of Acute Respiratory Distress Syndrome : a translational approach." Electronic Thesis or Diss., Université Clermont Auvergne (2021-...), 2023. http://www.theses.fr/2023UCFA0077.

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Le syndrome de détresse respiratoire aiguë est une cause majeure d'insuffisance respiratoire avec un taux de mortalité élevé. Elle se caractérise par des lésions alvéolaires diffuses, un œdème alvéolaire et une défaillance respiratoire hypoxémique qui entraînent de lourds coûts de santé. Actuellement, les traitements disponibles pour le SDRA restent principalement de soutien, et aucune approche pharmacologique n'est traduite avec succès en application clinique. Il existe deux processus majeurs au cours du développement physiopathologique du SDRA qui conduisent à la formation d'un œdème pulmona

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16

Morrison, Thomas. "Human mesenchymal stromal cell regulation of pulmonary macrophage populations in the Acute Respiratory Distress Syndrome." Thesis, Queen's University Belfast, 2017. https://pure.qub.ac.uk/portal/en/theses/human-mesenchymal-stromal-cell-regulation-of-pulmonary-macrophage-populations-in-the-acute-respiratory-distress-syndrome(da547b07-7974-436a-8943-7e2ddf3933cc).html.

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The Acute Respiratory Distress Syndrome (ARDS) is a devastating clinical disorder characterised by excessive inflammation in the alveolar compartment resulting in oedema of the airspaces due to loss of integrity in the alveolar epithelial-endothelial barrier. ARDS is associated with high mortality rates and there are currently no effective pharmacological therapies available. Human Mesenchymal Stromal Cells (hMSCs) are a promising candidate therapy which are currently being investigated in clinical trials for ARDS. However their mechanisms of effects in lung injury are not fully elucidated. A

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17

Chen, Yan Mi. "Regulation of iron mediated signalling processes during endotoxaemia : implications for the acute respiratory distress syndrome." Thesis, Imperial College London, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.395766.

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18

Martin, Collin Armand. "Pathology of complicated Babesia rossi-associated acute lung injury and respiratory distress syndrome in dogs." Diss., University of Pretoria, 2020. http://hdl.handle.net/2263/77412.

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A proportion of Babesia rossi infections in dogs are classified as complicated. One of the most lethal complications is the acute lung injury (ALI) and acute respiratory destress syndrome (ARDS). Patients affected by this complication usually succumb within 24 hours and there are similarities between this disease and malaria associated ALI and ARDS in humans, both rapidly fatal conditions. Both diseases are caused by haemoprotozoal parasites transmitted by insect vectors, namely ticks for babesiosis and mosquitoes for malaria. The pulmonary pathology of complicated babesiosis is poorly describ

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19

Güldner, Andreas, Robert Huhle, Alessandro Beda, et al. "Periodic Fluctuation of Tidal Volumes Further Improves Variable Ventilation in Experimental Acute Respiratory Distress Syndrome." Frontiers Research Foundation, 2018. https://tud.qucosa.de/id/qucosa%3A32492.

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In experimental acute respiratory distress syndrome (ARDS), random variation of tidal volumes (VT ) during volume controlled ventilation improves gas exchange and respiratory system mechanics (so-called stochastic resonance hypothesis). It is unknown whether those positive effects may be further enhanced by periodic VT fluctuation at distinct frequencies, also known as deterministic frequency resonance.We hypothesized that the positive effects of variable ventilation on lung functionmay be further amplified by periodic VT fluctuation at specific frequencies. In anesthetized and mechanically ve

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20

Dancer, Rachel Catherine Anne. "Investigation into a potential role for vitamin D in the pathogenesis of acute respiratory distress syndrome." Thesis, University of Birmingham, 2017. http://etheses.bham.ac.uk//id/eprint/7859/.

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Patients undergoing oesophagectomy are at risk of developing Acute Respiratory Distress Syndrome (ARDS), an immune mediated form of severe respiratory failure. The immunomodulatory properties of Vitamin D are increasingly recognised. We hypothesised that preoperative Vitamin D supplementation would reduce levels of perioperative alveolar oedema in patients undergoing oesophagectomy. Vitamin D deficiency is common in patients with and at risk of ARDS. High dose supplementation with cholecalciferol is a safe and effective method of increasing Vitamin D levels. Supplementation reduces perioperati

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21

Hamid, Umar Imran. "Use of clinically relevant human models to test novel therapies for the acute respiratory distress syndrome." Thesis, Queen's University Belfast, 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.695362.

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In the current era, there are no effective therapies for the treatment of acute respiratory distress syndrome (ARDS) despite numerous clinical trials. Current strategies are aimed at improving pulmonary perfusion, recruitment of atelectatic alveoli and reducing iatrogenic injuries to the lung. The human models of ARDS give important information when testing potential drug therapies and serve as a bridge between experimental studies and phase 11/111 clinical trials. I was able to establish the ex vivo lung perfusion model and studied the effects of aspirin in reducing pulmonary inflammation pro

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22

Sales, Raquel Pinto. "Acute Respiratory Distress Syndrome (ARDS) is an inflammatory disease characterized by pulmonary edema, stiff lungs and hypoxemia." Universidade Federal do CearÃ, 2014. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=12672.

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Acute Respiratory Distress Syndrome (ARDS) is an inflammatory disease characterized by pulmonary edema, stiff lungs and hypoxemia. Patients with ARDS are more susceptible to VILI (ventilator induced lung injury). Under mechanical ventilation, lung stress and strain are the main determinants of VILI and in patients with muscle effort patient-ventilator asynchrony may enhance this phenomenon. Ventilation modes PCV and VCV with auto-flow can minimize patient-ventilator asynchrony, but then can liberate the offer of flow and tidal volume, compromising the protective ventilatory strategy in ARDS. T

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23

Medford, Andrew R. L. "The role of vascular endothelial growth factor (VEGF) in repair and recovery from acute respiratory distress syndrome (ARDS)." Thesis, University of Edinburgh, 2007. http://hdl.handle.net/1842/6661.

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Acute Respiratory Distress Syndrome (ARDS) is the most extreme form of acute lung injury and continues to have a significant morbidity and mortality. Unfortunately, the mechanisms involved in the recovery and repair of the lung following ARDS remain poorly understood. An understanding of these is pivotal to improving outcome from acute lung injury. Several observational studies have suggested a potential relationship between Vascular Endothelial Growth Factor (VEGF) in the lung and the development/outcome of ARDS. In this thesis, three potential mechanisms underlying these observations have be

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24

Tubau, Llopart Isabel. "Biomarkers of oxidative stress in acute respiratory distress syndrome in exhaled breath measured online by mass spectrometry." Doctoral thesis, Universitat Autònoma de Barcelona, 2012. http://hdl.handle.net/10803/96305.

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Introducció: En el context de la síndrome del destret respiratori agut (SDRA), neutròfils activats emigren dels capil·lars pulmonars cap a l’alvèol i a l'espai intersticial, on alliberen radicals lliures d'oxigen anomenats espècies reactives de l’oxigen (ROS) que juguen un paper molt important en la patogènesis del SDRA. La secreció de ROS i d'altres mediadors proinflamatoris culminen en la lesió de la membrana alveolo-capil·lar, causant increment de la permeabilitat i edema pulmonar. Quan ROS interaccionen amb els lípids de les membranes de les cèl·lules pulmonars, té lloc un procés anomenat

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25

Retamal, Montes Jaime. "Aspects on ventilation induced stress and strain on regional and global inflammation in experimental acute respiratory distress syndrome." Doctoral thesis, Uppsala universitet, Anestesiologi och intensivvård, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-296952.

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Mechanical ventilation (MV) is a life-saving therapy in acute respiratory distress syndrome (ARDS), a condition that affects 3000 patients/year in Sweden with a mortality rate of about 40%. However, MV may induce or worsen lung injury causing “ventilator-induced lung injury (VILI)”. From a mechanical perspective strain (deformation, or relative change in lung volume) and stress (tension) have been postulated as main determinants of VILI. High respiratory rate is potentially another factor that may exacerbate VILI by amplifying the total energy transmitted to the lungs during MV. In this thesis

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26

Craven, Thomas Henry John. "Resolving uncertainty in acute respiratory illness using optical molecular imaging." Thesis, University of Edinburgh, 2017. http://hdl.handle.net/1842/29507.

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Ventilator associated pneumonia (VAP) and acute respiratory distress syndrome (ARDS) are two respiratory conditions unique to mechanically ventilated patients. The diagnosis of these conditions, and therefore any subsequent treatment, are befuddled by uncertainty. VAP rates vary considerably according to the diagnostic or surveillance criteria used. The pathogenesis of ARDS is well understood but when the internationally agreed consensus criteria are employed, the histological hallmarks are absent about half the time, indicating a disconnection between the clinical diagnosis and what is known

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27

Tsang, Hing-pang Clement, and 曾慶鵬. "The effectiveness of extracorporeal membrane oxygenation for pandemic influenza A (H1N1) induced acute respiratory distress syndrome in adults." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/193823.

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Given that pandemic swine flu outbreak led to substantial admission in intensive care unit, extracorporeal membrane oxygenation has been increasingly applied to those who suffered from H1N1 infection induced acute respiratory distress syndrome. This review is going to evaluate the effectiveness of using ECMO based on five related observational studies. The result, discussion and policy implication in Hong Kong are discussed. Since the ECMO system has been technological improved in recent years, there are less complications when applying ECMO. In view of evidence of reviewed studies, applicatio

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28

Johansson, Joakim. "Function of granulocytes after burns and trauma, associations with pulmonary vascular permeability, acute respiratory distress syndrome, and immunomodulation." Doctoral thesis, Linköpings universitet, Institutionen för klinisk och experimentell medicin, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-94513.

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Background: Our innate immunesystem protects us from infections but, since its methods is not all specific for microorganisms, may also induce collateral damage. Severe physical injury often proved deadly throughout evolution. Such injuries may induce massive collateral damage. Nowadays we can initiate advanced critical care for affected patients and save them from imminent trauma-related death. We are therefore faced with the fact that the collateral damage from the immune system may pose a major threat to the patient, the pathophysiology of which is not amenable to direct medical treatment a

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29

Robinson, Bryce RH M. D. "Implications of acute resuscitation and mechanical ventilation strategies upon pulmonary complications following injury." University of Cincinnati / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1427882608.

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30

Hirani, Nikhil A. "The regulation of interleukin-8 from macrophages by acute hypoxia and hyperoxia : a role in the pathogenesis of the acute respiratory distress syndrome (ARDS)." Thesis, University of Edinburgh, 2002. http://hdl.handle.net/1842/28236.

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Adequate oxygenation is a critical requirement for normal cellular function in humans. In patients with conditions such as major trauma, sepsis and aspiration injury there is often significant local and widespread tissue hypoxia as a consequence of inadequate oxygen supply or impaired oxygen utilisation. In this thesis, the role of acute hypoxia and hyperoxia were investigated in a novel animal model of acute lung injury. Bronchoscopic instillation of HC1 acid in an anaesthetised, ventilated rabbit resulted in reproducible acute neutrophilic lung injury in the instilled lobe. The contra-latera

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31

Chuang, Jiin-Haur. "Acute respiratory distress syndrome in septic shock : the role of endogenous opiates and the effect of an opioid antagonist." Thesis, McGill University, 1985. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=65382.

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32

Plouffe, Jannell. "Development of an evidence-based clinical practice guideline for prone positioning in acute respiratory distress syndrome for the pediatric patient." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/MQ62825.pdf.

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33

Bassford, Christopher R. "11β-hydroxysteroid dehydrogenase glucocorticoid metabolism within the lung and its influence on macrophage function in the acute respiratory distress syndrome". Thesis, University of Warwick, 2011. http://wrap.warwick.ac.uk/49584/.

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The acute respiratory distress syndrome (ARDS) is an important cause of respiratory failure in critically ill patients characterised by severe inflammation within the lungs. This inflammation is limited by anti-inflammatory glucocorticoid hormones released from the hypothalamus-pituitary-adrenal (HPA) system. This thesis reports a series of investigations into glucocorticoid concentrations and glucocorticoid metabolism within the lungs of patients with ARDS. It also contains an investigation into a potential biomarker for ARDS. Our study of glucocorticoid concentrations in alveolar epithelial

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34

Witte, Esther [Verfasser], and Jörg [Akademischer Betreuer] Reutershan. "Die Bedeutung des Adenosinrezeptors A2B bei der Migration neutrophiler Granulozyten im Acute Respiratory Distress Syndrome / Esther Witte ; Betreuer: Jörg Reutershan." Tübingen : Universitätsbibliothek Tübingen, 2013. http://d-nb.info/1160683530/34.

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35

Campos, Fábio Joly [UNESP]. "Efeito do óxido nítrico inalatório associado à ventilação mecânica protetora na lesão pulmonar aguda induzida experimentalmente." Universidade Estadual Paulista (UNESP), 2011. http://hdl.handle.net/11449/92154.

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Made available in DSpace on 2014-06-11T19:25:36Z (GMT). No. of bitstreams: 0 Previous issue date: 2011-02-09Bitstream added on 2014-06-13T20:53:44Z : No. of bitstreams: 1 campos_fj_me_botfm.pdf: 1625208 bytes, checksum: b0c89648b6d10484c83049fbb2b2478e (MD5)<br>Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)<br>Fundamentação/Objetivos: Síndrome do desconforto respiratório agudo (SDRA) cursa com elevada taxa de mortalidade a despeito do melhor entendimento de sua fisiopatologia e recentes avanços no tratamento. Ventilação mecânica (VM), uma das mais importantes formas de tratame

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36

Gusman, Pablo Braga [UNESP]. "Distribuição regional de gás e tecido na síndrome da angústia respiratória aguda: consequências do efeito da pressão expiratória final positiva." Universidade Estadual Paulista (UNESP), 2007. http://hdl.handle.net/11449/100145.

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Made available in DSpace on 2014-06-11T19:30:29Z (GMT). No. of bitstreams: 0 Previous issue date: 2007-02-02Bitstream added on 2014-06-13T19:40:09Z : No. of bitstreams: 1 gusman_pb_dr_botfm.pdf: 625469 bytes, checksum: 27745b379b0a215dafab05d5182357ac (MD5)<br>Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)<br>Desde sua primeira descrição, muito se investiu no melhor conhecimento da SARA, na identificação de sua etiologia, seus fatores de risco, seus mecanismos e mediadores, escolhendo-se métodos críticos de avaliação clínica que também pudessem definir o prognóstico. Com

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37

Selke, Maren Verena [Verfasser], Michael [Akademischer Betreuer] Quintel, Sven [Akademischer Betreuer] Kantelhardt, and Patricia [Akademischer Betreuer] Virsik-köpp. "Interaktionen zwischen "Acute Intracranial Hypertension" und "Acute Respiratory Distress" Syndrome : Auswirkungen auf den hypoxiesensiblen Hippocampus / Maren Verena Selke. Gutachter: Michael Quintel ; Sven Kantelhardt ; Patricia Virsik-Köpp. Betreuer: Michael Quintel." Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2011. http://d-nb.info/1042733988/34.

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Campos, Fabio Joly. "Efeito do óxido nítrico inalatório associado à ventilação mecânica protetora na lesão pulmonar aguda induzida experimentalmente /." Botucatu : [s.n.], 2011. http://hdl.handle.net/11449/92154.

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Orientador: José Roberto Fioretto<br>Banca: Ana Lúcia dos Anjos Ferreira<br>Banca: Regina Grigolli Cesar<br>Resumo: Fundamentação/Objetivos: Síndrome do desconforto respiratório agudo (SDRA) cursa com elevada taxa de mortalidade a despeito do melhor entendimento de sua fisiopatologia e recentes avanços no tratamento. Ventilação mecânica (VM), uma das mais importantes formas de tratamento, é baseada na utilização de estratégias protetoras com baixo volume corrente (VC) e elevada pressão expiratória final positiva (PEEP). Existem várias terapias adjuvantes, entre as quais o óxido nítrico inalató

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39

Mendler, Marc Robin [Verfasser]. "Einfluss von permissiver Hyperkapnie auf den Gasaustausch, die Lungenschädigung und die Hämodynamik am Versuchstier mit schwerem "Acute Respiratory Distress Syndrome (ARDS)" / Marc Robin Mendler." Ulm : Universität Ulm. Medizinische Fakultät, 2014. http://d-nb.info/1046890174/34.

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40

Doolittle, Lauren May. "The Impact of Alveolar Type II Cell Mitochondrial Damage and Altered Energy Production on Acute Respiratory Distress Syndrome Development During Influenza A Virus Infection." The Ohio State University, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu159224389333959.

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41

Gusman, Pablo Braga. "Distribuição regional de gás e tecido na síndrome da angústia respiratória aguda: consequências do efeito da pressão expiratória final positiva /." Botucatu : [s.n.], 2007. http://hdl.handle.net/11449/100145.

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Orientador: Luiz Antonio Vane<br>Banca: Yara Marcondes Machado Castiglia<br>Banca: José Reinaldo Cerqueira Braz<br>Banca: José Luiz Gomes do Amaral<br>Banca: Sílvia Regina Rios Vieira<br>Resumo: Desde sua primeira descrição, muito se investiu no melhor conhecimento da SARA, na identificação de sua etiologia, seus fatores de risco, seus mecanismos e mediadores, escolhendo-se métodos críticos de avaliação clínica que também pudessem definir o prognóstico. Com o objetivo de verificar se os efeitos da PEEP dependem da morfologia pulmonar, comparando sua resposta em três grupos de pacientes, descri

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42

Kanzler, Stephanie Sarah Verfasser], Stefan [Akademischer Betreuer] Uhlig, and Gabriele [Akademischer Betreuer] [Pradel. "A murine two-hit model to investigate the early time-course of inflammation in acute respiratory distress syndrome / Stephanie Sarah Kanzler ; Stefan Uhlig, Gabriele Pradel." Aachen : Universitätsbibliothek der RWTH Aachen, 2021. http://d-nb.info/1238081312/34.

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43

Boissier, Florence. "Dysfonction vasculaire pulmonaire et ventriculaire droite au cours du SDRA : approche échocardiographique." Thesis, Paris Est, 2014. http://www.theses.fr/2014PEST0057.

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Contexte: Le syndrome de détresse respiratoire aigüe (SDRA) est associé à une dysfonction vasculaire pulmonaire. Objectifs: Préciser le retentissement cardiaque de cette dysfonction vasculaire pulmonaire en recherchant la fréquence et le pronostic du foramen ovale perméable, du passage transpulmonaire de bulles en échographie de contraste, du cœur pulmonaire aigu (CPA), de la dysfonction systolique ventriculaire droite ainsi que de la déformation ventriculaire gauche au cours du SDRA. Nous avons aussi évalué la tolérance hémodynamique de la ventilation en Pression Expiratoire Positive (PEP) él

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44

Rossi, Patrik. "The role of the endothelin system in experimental acute lung injury with special reference to the formation of extra-vascular lung water /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-700-6/.

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Pereira, Marcelo Luís Monteiro. "O papel da heme oxigenase 1 na síndrome do desconforto respiratório agudo associada à malária." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/42/42133/tde-11112016-154538/.

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A malária é uma doença causada pelo parasita do gênero Plasmodium e que foi responsável por cerca de 440.000 mortes em 2015. A síndrome do desconforto respiratório agudo (SDRA) é uma das principais complicações clínicas da malária. O modelo murino DBA/2 reproduz os sinais clínicos da SDRA observados em humanos, quando infectado com o Plasmodium berghei ANKA. Além disso, altos níveis da enzima heme oxigenase 1 (HO-1) foram observados em casos de malária cerebral e em SDRA em humanos. Os nossos dados indicam que os níveis da HO-1 estão aumentados em camundongos que desenvolvem SDRA associada à m

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Huhle, Robert [Verfasser], de Abreu Marcelo [Gutachter] Gama, and Ute [Gutachter] Morgenstern. "Periodic Variable Mechanical Ventilation and Dynamics of Recruitment and De-recruitment in Experimental Acute Respiratory Distress Syndrome / Robert Huhle ; Gutachter: Marcelo Gama de Abreu, Ute Morgenstern." Dresden : Technische Universität Dresden, 2019. http://d-nb.info/1227833407/34.

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Huhle, Robert [Verfasser], Marcelo Gama de [Gutachter] Abreu, and Ute [Gutachter] Morgenstern. "Periodic Variable Mechanical Ventilation and Dynamics of Recruitment and De-recruitment in Experimental Acute Respiratory Distress Syndrome / Robert Huhle ; Gutachter: Marcelo Gama de Abreu, Ute Morgenstern." Dresden : Technische Universität Dresden, 2019. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa2-364976.

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Carpi, Mario Ferreira [UNESP]. "Efeito imediato e prolongado da administração precoce de óxido nítrico inalatório em crianças portadoras de síndrome do desconforto respiratório agudo." Universidade Estadual Paulista (UNESP), 2003. http://hdl.handle.net/11449/104678.

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Made available in DSpace on 2014-06-11T19:33:27Z (GMT). No. of bitstreams: 0 Previous issue date: 2003Bitstream added on 2014-06-13T19:44:14Z : No. of bitstreams: 1 carpi_mf_dr_botfm.pdf: 214359 bytes, checksum: 0134d7f3e72fe3bbaff53ae651e657bf (MD5)<br>Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)<br>A Síndrome do Desconforto Respiratório Agudo (SDRA) é a forma clínica mais grave da lesão pulmonar aguda e, apesar do melhor entendimento de sua fisiopatologia, a taxa de mortalidade permanece elevada. O óxido nítrico inalatório (NOi) é um vasodilatador seletivo de áreas pulmona

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Scaccabarozzi, D. "THE PATHOGENESIS OF MALARIA ACUTE RESPIRATORY DISTRESS SYNDROME (MA-ARDS): MODIFICATION OF THE LIPID PROFILE, ANTIOXIDANT DEFENCES AND CYTOKINE CONTENT IN DIFFERENT TISSUES OF MALARIA INFECTED MICE." Doctoral thesis, Università degli Studi di Milano, 2014. http://hdl.handle.net/2434/231156.

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INTRODUCTION Malaria is a major health problem, with more than 650.000 deaths and 200 million clinical cases each year. Respiratory distress as malaria associated acute respiratory distress syndrome (MA-ARDS) is a common complication. The pathogenesis of MA-ARDS is mainly inflammatory and one of the main observation is the presence of abundant monocytes and macrophages inside the blood capillaries, in the interstitium and also in alveolar spaces. Malaria pigment or haemozoin (Hz) is often seen in these cells reflecting active phagocytosis and leads to the production of cytokines and other infl

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Carpi, Mário Ferreira. "Efeito imediato e prolongado da administração precoce de óxido nítrico inalatório em crianças portadoras de síndrome do desconforto respiratório agudo /." Botucatu : [s.n.], 2003. http://hdl.handle.net/11449/104678.

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Orientador: José Roberto Fioretto<br>Resumo: A Síndrome do Desconforto Respiratório Agudo (SDRA) é a forma clínica mais grave da lesão pulmonar aguda e, apesar do melhor entendimento de sua fisiopatologia, a taxa de mortalidade permanece elevada. O óxido nítrico inalatório (NOi) é um vasodilatador seletivo de áreas pulmonares ventiladas, promovendo a otimização da relação ventilação/perfusão nestas áreas, com melhora da oxigenação e facilitação do esvaziamento do ventrículo direito. Tais efeitos permitiriam a redução de parâmetros ventilatórios, habitualmente elevados na SDRA, diminuindo o ris

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