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Relevant bibliographies by topics / ACVR2A

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Academic literature on the topic 'ACVR2A'

Author: Grafiati

Published: 4 June 2025

Last updated: 1 August 2025

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Journal articles on the topic "ACVR2A"

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Yamamoto, Y., Y. Kobayashi, Y. Yoshimoto, and K. Okuda. "104 EXPRESSION OF ACTIVIN A AS A LOCAL REGULATOR IN THE BOVINE OVIDUCT." Reproduction, Fertility and Development 27, no. 1 (2015): 145. http://dx.doi.org/10.1071/rdv27n1ab104.

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Activin (ACV) is known as a local regulator of several reproductive functions including follicular development and implantation in mammals. ACVA is a glycopeptide belonging to the transforming growth factor β superfamily, and is a homodimer of inhibin ßA (INHBA) subunits. Follistatin (FST), an ACV-specific binding protein, inhibits ligand-receptor binding. ACV receptor (ACVR) is a hetero-tetramer consisting of 2 kinds of protein, ACVR1 or ACVR1B and ACVR2A or ACVR2B. The oviduct provides an optimal environment for sperm capacitation, fertilization, and early embryonic development. Previous rep

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Szilágyi, Szabina Szófia, Wiktor Burdzinski, Jerome Jatzlau, Marcelo Ehrlich, Petra Knaus, and Yoav I. Henis. "The Activation of the Fibrodysplasia Ossificans Progressiva-Inducing ALK2-R206H Mutant Depends on the Distinct Homo-Oligomerization Patterns of ACVR2B and ACVR2A." Cells 13, no. 3 (2024): 221. http://dx.doi.org/10.3390/cells13030221.

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Mutations in activin-like kinase 2 (ALK2), e.g., ALK2-R206H, induce aberrant signaling to SMAD1/5/8, leading to Fibrodysplasia Ossificans Progressiva (FOP). In spite of extensive studies, the underlying mechanism is still unclear. Here, we quantified the homomeric and heteromeric interactions of ACVR2A, ACVR2B, ALK2-WT, and ALK2-R206H by combining IgG-mediated immobilization of one receptor with fluorescence recovery after photobleaching (FRAP) measurements on the lateral diffusion of a co-expressed receptor. ACVR2B formed stable homomeric complexes that were enhanced by Activin A (ActA), whil

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Yuza, Kizuki, Masayuki Nagahashi, Hiroshi Ichikawa, et al. "Association of activin type II receptor mutation with microsatellite instability in gastric cancer." Journal of Clinical Oncology 35, no. 15_suppl (2017): e23191-e23191. http://dx.doi.org/10.1200/jco.2017.35.15_suppl.e23191.

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e23191 Background: Microsatellite instability-high status (MSI-H) and alterations in the DNA mismatch repair pathway associate with the efficacy of 5-FU and immune checkpoint inhibitors in patients with gastrointestinal cancers. The activin type II receptor (ACVR2) that binds to the transforming growth factor beta superfamily of ligands is frequently mutated in MSI-H colorectal cancer. However, the incidence of ACVR2 mutations in gastric cancer patients remains unclear. The aim of this study is to reveal the incidence and to examine the association between the MSI-H and ACVR2A mutations in gas

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Jia, Yongning, Hongling Yuan, Yaqun Xin, et al. "Abstract 5752: Loss-of-function mutations in ACVR2A are correlated with microsatellite instability in gastric and colorectal cancer." Cancer Research 82, no. 12_Supplement (2022): 5752. http://dx.doi.org/10.1158/1538-7445.am2022-5752.

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Abstract Background: The activin type II receptor (ACVR2), which is a member of the transforming growth factor beta (TGF-β) receptor family, is frequently mutated in colorectal cancer (CRC) and gastric cancer (GC). However, the incidence of ACVR2 mutations in MSI-H patients remains unclear. The aim of this study is to evaluate ACVR2A mutation characteristics and to examine its associations between the high microsatellite instability (MSI-H) and ACVR2A mutations in CRC and GC. Materials and Methods: We analyzed data from two cohorts: a Chinese cohort and the TCGA cohort. The Chinese cohort cons

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Xu, Zhixiao, and Chengshui Chen. "Abnormal Expression and Prognostic Significance of Bone Morphogenetic Proteins and Their Receptors in Lung Adenocarcinoma." BioMed Research International 2021 (May 7, 2021): 1–23. http://dx.doi.org/10.1155/2021/6663990.

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Background. Lung adenocarcinoma (LUAD) is one of the most life-threatening malignancies. The crucial role of bone morphogenetic protein (BMP)/BMP receptors reveals the significance of exploring BMP protein-related prognostic predictors in LUAD. Methods. The mRNA expression of BMPs/BMP receptors was investigated in LUAD and normal lung tissues. Gene Ontology and the Kyoto Encyclopedia of Genes and Genomes pathway analyses were performed, and the prognostic values were assessed by Kaplan-Meier Plotter. Univariate and multivariate Cox regression analyses were executed to ascertain the correlation

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Zhuo, Changhua, Dan Hu, Xiandong Lin, Ying Chen, Xiongwei Zheng, and Chunkang Yang. "Down expression of activin A receptor type 2A in relation to metastatic potential and prognosis of patients with colon cancer." Journal of Clinical Oncology 36, no. 4_suppl (2018): 712. http://dx.doi.org/10.1200/jco.2018.36.4_suppl.712.

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712 Background: The ACVR2A (activin A receptor type 2A) is a membrane receptor in TGF-β signal pathway, which is involved in the regulation of cell proliferation, migration and apoptosis. The expression profiles and biological functions of ACVR2A in colon cancer is largely unknown so far. Methods: ACVR2A expression was investigated using GSE39582 database and two validation cohorts. The in vitro study of the cell proliferation and migration of human colon cell lines was also performed. Results: In GSE39582 database (n = 497), lower mRNA expression of ACVR2A was identified as an inferior progno

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Ongaro, Luisina, Xiang Zhou, Yiming Cui, Ulrich Boehm, and Daniel J. Bernard. "Gonadotrope-specific deletion of the BMP type 2 receptor does not affect reproductive physiology in mice†‡." Biology of Reproduction 102, no. 3 (2019): 639–46. http://dx.doi.org/10.1093/biolre/ioz206.

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Abstract Activins selectively stimulate follicle-stimulating hormone (FSH) secretion by pituitary gonadotrope cells. More recently, other members of the TGFbeta superfamily, the bone morphogenetic proteins (BMPs), were reported to regulate FSH synthesis. Activins and BMPs independently and synergistically stimulate transcription of the FSHbeta subunit (Fshb) gene in immortalized gonadotrope-like cells. Both ligands can signal via the activin receptor type IIA (ACVR2A) to regulate FSH synthesis in vitro. In vivo, global Acvr2a knockout mice exhibit a 60% reduction in circulating FSH relative to

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Li, Cheng-Lin, Zhi-Bin Xu, Xing-Liang Fan, et al. "MicroRNA-21 Mediates the Protective Effects of Mesenchymal Stem Cells Derived from iPSCs to Human Bronchial Epithelial Cell Injury Under Hypoxia." Cell Transplantation 27, no. 3 (2018): 571–83. http://dx.doi.org/10.1177/0963689718767159.

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Airway epithelial cell injury is a key triggering event to activate allergic airway inflammation, such as asthma. We previously reported that administration of mesenchymal stem cells (MSCs) significantly alleviated allergic inflammation in a mouse model of asthma, and the mmu-miR-21/ACVR2A axis may be involved. However, whether MSCs protect against bronchial epithelial cell injury induced by hypoxia, and the underlying mechanism, remain unknown. In our study, the human bronchial epithelial cell line BEAS-2B was induced to undergo apoptosis with a hypoxia mimic of cobalt chloride (CoCl2) damage

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Ashouri, Karam, Yan Yang, Joshua Millstein, et al. "Myostatin/activin pathway gene expression and single nucleotide polymorphisms (SNPs) in metastatic colorectal cancer (mCRC)." Journal of Clinical Oncology 42, no. 3_suppl (2024): 188. http://dx.doi.org/10.1200/jco.2024.42.3_suppl.188.

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188 Background: Cancer cachexia leads to reduced overall survival (OS) in mCRC. Novel therapeutics targeting the myostatin/activin pathway can reverse cachexia. Here we investigate the effect of myostatin/activin pathway gene expression and SNPs in mCRC patients (pts). Methods: Blood samples from 836 pts enrolled in 3 randomized first-line trials: TRIBE (FOLFIRI bevacizumab [bev]; FOLFOXIRI bev), FIRE-3 (FOLFIRI bev; FOLFIRI cetuximab [cet]) and MAVERICC (FOLFIRI bev; FOLFOX bev) were genotyped by Illumina OncoArray. The impact on outcome of 13 SNPs from 5 myostatin/activin genes ( ACVR1B, ACV

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Ma, Sen, Dejun Ji, Xiaolong Wang, Yuxin Yang, Yinghua Shi, and Yulin Chen. "Transcriptomic Analysis Reveals Candidate Ligand-Receptor Pairs and Signaling Networks Mediating Intercellular Communication between Hair Matrix Cells and Dermal Papilla Cells from Cashmere Goats." Cells 12, no. 12 (2023): 1645. http://dx.doi.org/10.3390/cells12121645.

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Hair fiber growth is determined by the spatiotemporally controlled proliferation, differentiation, and apoptosis of hair matrix cells (HMCs) inside the hair follicle (HF); however, dermal papilla cells (DPCs), the cell population surrounded by HMCs, manipulate the above processes via intercellular crosstalk with HMCs. Therefore, exploring how the mutual commutations between the cells are molecularly achieved is vital to understanding the mechanisms underlying hair growth. Here, based on our previous successes in cultivating HMCs and DPCs from cashmere goats, we combined a series of techniques,

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Dissertations / Theses on the topic "ACVR2A"

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Ballikaya, Seda [Verfasser], and Jürgen [Akademischer Betreuer] Kopitz. "Activin Receptor Type 2 A (ACVR2A)-dependent Proteomic and Glycomic Alterations in a Microsatellite Unstable (MSI) Colorectal Cancer Cell Line Model System / Seda Ballikaya ; Betreuer: Jürgen Kopitz." Heidelberg : Universitätsbibliothek Heidelberg, 2014. http://d-nb.info/118003208X/34.

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Silva, Vanessa Gonçalves. "Implementação de um método de validação do controlo de qualidade em amostras de DNA armazenadas no Biobanco-IMM." Master's thesis, Escola Superior de Saúde Egas Moniz, 2014. http://hdl.handle.net/10400.26/6313.

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Dissertação para obtenção do grau de Mestre em Biologia Molecular em Saúde<br>No Biobanco-IMM as amostras são colhidas e armazenadas por longos períodos de tempo sendo de extrema importância que estas sejam de elevada qualidade. Para cumprir este requisito, os procedimentos do biobanco efetuam-se de acordo com diretrizes internacionais para a avaliação da qualidade das amostras que armazena.<br>No Biobanco-IMM, as amostras de DNA são extraídas e avaliadas em relação à sua concentração, pureza (razão A260/A280) e integridade. Por vezes, uma pequena percentagem das amostras pode não cumprir este

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Hino, Kyosuke. "Neofunction of ACVR1 in fibrodysplasia ossificans progressiva." Kyoto University, 2016. http://hdl.handle.net/2433/215941.

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Rouhaud, Lydie. "Organisation, expression et polymorphismes des gènes ACVR2B et FST, intervenant dans la voie de signalisation de la myostatine (GDF-8)." Limoges, 2004. http://aurore.unilim.fr/theses/nxfile/default/806b4b50-8a4d-4d0b-a690-044312fbb961/blobholder:0/2004LIMO0039.pdf.

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La croissance du muscle squelettique est un processus complexe, impliquant de nombreux facteurs moléculaires. La myostatine (GDF-8) est un puissant régulateur négatif du développement musculaire, et l’absence d’une protéine fonctionnelle provoque une incroyable augmentation de la masse musculaire, chez la souris et le bovin. Des mutations dans le gène codant la myostatine ont été associées à l’extrême musculature observée chez le bovin. Cependant, des individus présentant différents degrés d’hypertrophie musculaire ont également été observés, sans qu’aucune mutation identifiée dans le gène cod

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Lunardi, Cláudia Cruz. "Associação entre os polimorfismos dos genes candidatos FTO, TRHR e ACVR1b com a composição corporal e força muscular em idosas brasileiras." reponame:Repositório Institucional da UnB, 2013. http://repositorio.unb.br/handle/10482/13409.

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Tese (doutorado)—Universidade de Brasília, Faculdade de Ciências da Saúde, Programa de Pós-Graduação em Ciências da Saúde, 2013.<br>Submitted by Alaíde Gonçalves dos Santos (alaide@unb.br) on 2013-06-25T13:02:46Z No. of bitstreams: 1 2013_ClaudiaCruzLunardi.pdf: 2104527 bytes, checksum: 869f3995f555f48eaf76b6782428405f (MD5)<br>Approved for entry into archive by Guimaraes Jacqueline(jacqueline.guimaraes@bce.unb.br) on 2013-06-25T14:21:38Z (GMT) No. of bitstreams: 1 2013_ClaudiaCruzLunardi.pdf: 2104527 bytes, checksum: 869f3995f555f48eaf76b6782428405f (MD5)<br>Made available in DSpace on 2013-0

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Maekawa, Hirotsugu. "Prophylactic treatment of rapamycin ameliorates naturally developing and episode -induced heterotopic ossification in mice expressing human mutant ACVR1." Doctoral thesis, Kyoto University, 2021. http://hdl.handle.net/2433/263519.

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Junior, Irineu Renzi. "Expressão dos microRNAs miR-1 e miR-133b e dos genes ACVR1C, CCL18, VGLL3, ASPN, OGDHL, BTC em meningiomas com e sem deleção do cromossomo 22q." Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/17/17137/tde-23012016-113912/.

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Introdução: Dentre os tumores primários do SNC, o meningioma é o tipo mais frequentemente diagnosticado, sendo responsável por 35,5% dos casos, considerando-se todas as faixas etárias. A gênese dos meningiomas é um processo complexo que envolve o acúmulo de alterações genéticas, sendo o evento mais conhecido a deleção no braço longo do cromossomo 22. O entendimento da iniciação e do crescimento dos meningiomas em nível molecular pode ajudar a definir novos alvos de terapia e, neste contexto, tem se destacado na última década o estudo dos microRNAs (miRNAs). Os miRNAs são uma classe de pequenos

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Panchulidze, Irakli [Verfasser], Wolfgang [Akademischer Betreuer] Engel, Rainer [Akademischer Betreuer] Laskawi, and Thomas [Akademischer Betreuer] Crozier. "Zur molekulargenetischen Charakterisierung der Mutationen in den Endoglin-und ACVRL1-Genen bei den Morbus-Osler-Patienten / Irakli Panchulidze. Gutachter: Rainer Laskawi ; Thomas Crozier. Betreuer: Wolfgang Engel." Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2011. http://d-nb.info/1044045469/34.

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Truffaux, Nathalene. "Nouvelles cibles thérapeutiques dans les gliomes infiltrants du tronc cérébral de l'enfant." Thesis, Paris 11, 2014. http://www.theses.fr/2014PA11T022/document.

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Le gliome infiltrant du tronc cérébral est une tumeur rare, non opérable et inéluctablement fatale. En raison du manque de ressource biologique disponible, aucun progrès dans la compréhension de la biologie de ces tumeurs n’a été fait jusqu’à ces dernières années, laissant la radiothérapie pour seul traitement efficace, et seulement transitoirement. Enfin, grâce à la mise en place de collecte d’échantillons de gliomes infiltrant du tronc cérébral au diagnostic ou à l’autopsie, un nombre sans précédent d’analyses biologiques et génomiques a pu être mené et améliorer la connaissance de ces tumeu

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Schwartze, Julian Tristan [Verfasser]. "Glucocorticoids recruit Tgfbr3 and Smad1 to shift transforming growth factor-beta signaling from the Tgfbr1/Smad2/3 axis to the Acvrl1/Smad1 axis in lung fibroblasts / Julian Tristan Schwartze." Gießen : Universitätsbibliothek, 2015. http://d-nb.info/1076005810/34.

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Books on the topic "ACVR2A"

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Association des communautés villageoises responsables (Togo). Plan d'action de l'Association des communautés villageoises responsables (ACVR): Période 2003 à 2007. Association des communautés villageoises responsables, 2003.

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Adufu, Kossi Séna. Rapport du diagnostic organisationnel de l'Association des communautés villageoises responsables (ACVR): Diagnostic réalisé du 20 au 24 janvier 2003. INADES Formation Togo, Bureau national, 2003.

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Hrushovski, Ehud, and François Loeser. Specializations and ACV2F. Princeton University Press, 2017. http://dx.doi.org/10.23943/princeton/9780691161686.003.0009.

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This chapter introduces the theory ACV²F of iterated places and describes some algebraic criteria for v- and g-continuity. It considers the theory ACV²F of triples (K₂,K₁,K₀) of fields with surjective, non-injective places rᵢⱼ : Kᵢ → Kⱼ for i > j, r₂₀ = r₁₀ ° r₂₁, such that K₂ is algebraically closed. The chapter shows that the family of g-open sets is definable in definable families. It also presents some applications of the continuity criteria and concludes by proving that for each definable set of definable functions V → Wsuperscript Number Sign the subset of those that are g-continuous

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ACVR: Évolution institutionelle de 1990 à 2000. ACVR, 2000.

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2006 IVRA/ACVR/ECVDI Joint Annual Scientific Conference: August 7-11. 2006, Westin Bayshore Hotel, Vancouver, British Columbia. s.n., 2006.

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Book chapters on the topic "ACVR2A"

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Maruyama, Rika, and Toshifumi Yokota. "Morpholino-Mediated Exon Skipping Targeting Human ACVR1/ALK2 for Fibrodysplasia Ossificans Progressiva." In Methods in Molecular Biology. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8651-4_32.

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"ACVR2." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-46875-3_100103.

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"ACVR1." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-46875-3_100102.

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"9. Specializations and ACV2F." In Non-Archimedean Tame Topology and Stably Dominated Types. Princeton University Press, 2016. http://dx.doi.org/10.1515/9781400881222-009.

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"Appendix C: ACVAA Monitoring Guidelines." In Errors in Veterinary Anesthesia. John Wiley & Sons, Inc., 2016. http://dx.doi.org/10.1002/9781119259749.app3.

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Kaplan, Frederick S., O. W. Towler, and Eileen M. Shore. "ACVR1 and Fibrodysplasia Ossificans Progressiva." In Epstein's Inborn Errors of Development. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199934522.003.0056.

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Ware, Stephanie M., and John W. Belmont. "ZIC3, CFC1, ACVR2B, and LEFTY2 and the Visceral Heterotaxies." In Inborn Errors Of Development. Oxford University PressNew York, NY, 2008. http://dx.doi.org/10.1093/oso/9780195306910.003.0033.

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Abstract Heterotaxy is a clinical phenotype resulting from a failure to correctly establish left/right (L/R) patterning during embryogenesis. Multiple congenital anomalies may result from discordant positioning of normally asymmetrical internal organs and vasculature. Midline defects and anomalies of paired organs can occur, presumably owing to a failure to correctly segregate left and right. Mutations in three genes that function in the transforming growth factor-β (TGF-β) signaling pathway, Activin receptor type IIB (ACVR2B), the epidermal growth factor-cripto/FRL1/cryptic (EGF-CFC) family m

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Eyries, Mélanie, Barbara Girerd, David Montani, David-Alexandre Tregouët, Marc Humbert, and Florent Soubrier. "Pulmonary hypertension genes as major diagnostic tools." In ESC CardioMed, edited by Marc Humbert. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0577.

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A few genes have been shown to be major predisposing factors for pulmonary hypertension and are responsible for heritable forms of the disease. However, for nearly all genes described, not all mutation carriers develop the disease (autosomal transmission with incomplete penetrance) explaining the presence of genetic mutations in apparently sporadic cases. Beside mutations in major genes (BMPR2 for pulmonary arterial hypertension and EIF2AK4 for recessive heritable pulmonary veno-occlusive disease), other genes have been involved in a very limited number of cases (KCNK3, CAV1, and Smad8). Gene

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"Appendix D: ACVAA Guidelines for Anesthesia in Horses." In Errors in Veterinary Anesthesia. John Wiley & Sons, Inc., 2016. http://dx.doi.org/10.1002/9781119259749.app4.

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"AIP-1, Acvri1, Acvrinp1, Acvrip1, Magi-2, S-SCAM, mKIAA0705." In Encyclopedia of Signaling Molecules. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_100176.

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Conference papers on the topic "ACVR2A"

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Wang, X., and Q. Zhou. "Circular RNA ACVR2A Inhibits NSCLC Progression by Acting as a Molecular Sponge of miR-483-3p to Modulate Chemerin Expression." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a6187.

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Rosado-Mercado, S. M., D. M. Albors-Agullo, E. Rivera Jimenez, and W. De Jesus-Rojas. "Novel ACVRL1 Genetic Mutation Linked with Hereditary Hemorrhagic Telangiectasia." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a3550.

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Montani, D., B. Girerd, F. Coulet, et al. "Clinical Outcomes of Pulmonary Arterial Hypertension in Carriers of ACVRL1 (ALK1) Mutation." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a2659.

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Rosado, S., W. De Jesus-Rojas, and E. Rivera. "ENG and ACVRL1 Genetic Variants Linked to Hereditary Hemorrhagic Telangiectasia in Puerto Rico." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a2372.

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Rusche, Johanna R., Li Xiang-Tischhauser, Michael Bette, et al. "Overexpression of ACVRL1 inhibits the tube formation ability of HMEC-1 endothelial cells." In 95th Annual Meeting German Society of Oto-Rhino-Laryngology, Head and Neck Surgery e. V., Bonn. Georg Thieme Verlag KG, 2024. http://dx.doi.org/10.1055/s-0044-1785118.

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Loomans, Holli A., Laura L. Quast, and Claudia A. Andl. "Abstract 5088: ACVRIB-dependent regulation of extracellular matrix protein in squamous cell carcinoma progression." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-5088.

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Mandic, Robert, Michael Bette, R. Johanna Rusche, et al. "Generation of an ACVRL1 knockout iPS cell line for the in vitro study of HHT type 2 associated angiogenesis." In 94th Annual Meeting German Society of Oto-Rhino-Laryngology, Head and Neck Surgery e.V., Bonn. Georg Thieme Verlag, 2023. http://dx.doi.org/10.1055/s-0043-1767073.

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Buczkowicz, Pawel, Christine Hoeman, Patricia Rakopoulos, et al. "Abstract A18: Uncovering molecular subgroups and a novel cancer driver, ACVR1, in diffuse intrinsic pontine gliomas." In Abstracts: AACR Special Conference: Pediatric Cancer at the Crossroads: Translating Discovery into Improved Outcomes; November 3-6, 2013; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.pedcan-a18.

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Busmachiu, Galina, Victoria Nistreanu, Vladimir Turcan, and Oxana Munjiu. "Date noi privind diversitatea faunei ecosistemelor acva-paluste ale lacului Cahul." In International symposium ”Functional ecology of animals” dedicated to the 70th anniversary from the birth of academician Ion Toderas. Institute of Zoology, Republic of Moldova, 2019. http://dx.doi.org/10.53937/9789975315975.32.

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de Medeiros, Francisco Petrônio Alencar, and Alex Sandro Gomes. "Uma Abordagem baseada em Análise de Redes Sociais para Melhorar a Presença Social em Ambientes Colaborativos de Aprendizagem." In Anais Estendidos do Simpósio Brasileiro de Sistemas Colaborativos. Sociedade Brasileira de Computação - SBC, 2023. http://dx.doi.org/10.5753/sbsc_estendido.2023.229415.

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Abstract:

Acompanhar as interações sociais em Ambientes Colaborativos Virtuais de Aprendizagem (ACVAs) com uma quantidade cada vez maior de ferramentas colaborativas, que muitas vezes estende os limites do ACVA, oferecendo o suporte adequado e em tempo real aos aprendizes é um dos desafios mais intensos e exaustivos que os professores online enfrentam. O objetivo geral deste trabalho foi a investigação de como uma abordagem de monitoramento abrangente das interações sociais dos aprendizes no Amadeus-SIMM impactou positivamente na percepção e mediação docente de forma a promover uma melhoria da presença

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