Academic literature on the topic 'ADAMTS 1 e 4'

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Journal articles on the topic "ADAMTS 1 e 4"

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TURNER, SHARON L., DAVID MANGNALL, NIGEL C. BIRD, ROWENA A. D. BUNNING, and MARIA E. BLAIR-ZAJDEL. "Expression of ADAMTS-1, ADAMTS-4, ADAMTS-5 and TIMP3 by hepatocellular carcinoma cell lines." International Journal of Oncology 41, no. 3 (2012): 1043–49. http://dx.doi.org/10.3892/ijo.2012.1525.

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Lim, Ngee H., Masahide Kashiwagi, Robert Visse, et al. "Reactive-site mutants of N-TIMP-3 that selectively inhibit ADAMTS-4 and ADAMTS-5: biological and structural implications." Biochemical Journal 431, no. 1 (2010): 113–22. http://dx.doi.org/10.1042/bj20100725.

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We have reported previously that reactive-site mutants of N-TIMP-3 [N-terminal inhibitory domain of TIMP-3 (tissue inhibitor of metalloproteinases 3)] modified at the N-terminus, selectively inhibited ADAM17 (a disintegrin and metalloproteinase 17) over the MMPs (matrix metalloproteinases). The primary aggrecanases ADAMTS (ADAM with thrombospondin motifs) -4 and -5 are ADAM17-related metalloproteinases which are similarly inhibited by TIMP-3, but are poorly inhibited by other TIMPs. Using a newly developed recombinant protein substrate based on the IGD (interglobular domain) of aggrecan, gst-I
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Haddock, G., A. K. Cross, J. Plumb, et al. "Expression of ADAMTS-1, -4, -5 and TIMP-3 in normal and multiple sclerosis CNS white matter." Multiple Sclerosis Journal 12, no. 4 (2006): 386–96. http://dx.doi.org/10.1191/135248506ms1300oa.

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ADAMTS (a disintegrin and metalloproteinase with thrombospondin motifs) -1, -4 and -5 proteases have been identified in the CNS at the mRNA level. These glutamyl endopeptidases, inhibited by tissue inhibitor of metalloproteinases (TIMP)-3, are key enzymes in the degradation of the aggregating chondroitin sulphate proteoglycans (CSPGs), and may therefore play a role in CNS extracellular matrix (ECM) changes in multiple sclerosis (MS). We have investigated ADAMTS and TIMP-3 expression in normal and MS CNS white matter by real-time RT-PCR, western blotting and immunohistochemistry. We report for
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Tajima, Takuya, Nami Yamaguchi, Takuji Yokoe, Etsuo Chosa, and Yudai Morita. "A possible issue of articular cartilage degeneration in acute phase after anterior cruciate ligament reconstruction." Orthopaedic Journal of Sports Medicine 9, no. 7_suppl4 (2021): 2325967121S0021. http://dx.doi.org/10.1177/2325967121s00218.

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Objectives: Intra-articular hematoma are caused by articular injury such as ligament rupture or intra-articular fracture and following knee surgery. It has been reported that intra-articular hematoma leads to cartilage degeneration. Exposure of articular cartilage to low concentrations of blood for a period as short as 2 days has been confirmed to induce the irreversible cartilage damage. Anterior cruciate ligament (ACL) injury is increased risk for developing posttraumatic osteoarthritis (OA). Despite improvement in ACL reconstruction (ACLR), the incidence of posttraumatic OA has remained rel
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Yoshida, Koji, Yasuyuki Suzuki, Akio Saito, Kanji Fukuda, Chiaki Hamanishi та Hiroshi Munakata. "Aggrecanase-1 (ADAMTS-4) interacts with α1-antitrypsin". Biochimica et Biophysica Acta (BBA) - General Subjects 1725, № 2 (2005): 152–59. http://dx.doi.org/10.1016/j.bbagen.2005.06.009.

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Tortorella, Micky D., Elizabeth C. Arner, Robert Hills, et al. "ADAMTS-4 (aggrecanase-1): N-Terminal activation mechanisms." Archives of Biochemistry and Biophysics 444, no. 1 (2005): 34–44. http://dx.doi.org/10.1016/j.abb.2005.09.018.

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Mao, Guping, Peihui Wu, Ziji Zhang та ін. "MicroRNA-92a-3p Regulates Aggrecanase-1 and Aggrecanase-2 Expression in Chondrogenesis and IL-1β-Induced Catabolism in Human Articular Chondrocytes". Cellular Physiology and Biochemistry 44, № 1 (2017): 38–52. http://dx.doi.org/10.1159/000484579.

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Background/Aims: Aggrecanase-1 (ADAMTS-4) and aggrecanase-2 (ADAMTS-5) are secreted enzymes belonging to the ADAMTS (a disintegrin and metalloproteinase with thrombospondin motifs) family that play significant roles in the progression of osteoarthritis (OA). Here, we aimed to determine whether the expression of ADAMTS-4/5 in chondrogenesis and inflammation is regulated by microRNA-92a-3p (miR-92a-3p). Methods: MiR-92a-3p and ADAMTS-4/5 expressions were determined by quantitative polymerase chain reaction (qPCR). To investigate the repressive effect of miR-92a-3p on ADAMTS-4/5 expression, chond
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Lee, So-Young, Hyang-Sin Lee, Minchan Gil, et al. "Differential Expression Patterns of a Disintegrin and Metalloproteinase With Thrombospondin Motifs (ADAMTS) -1, -4, -5, and -14 in Human Placenta and Gestational Trophoblastic Diseases." Archives of Pathology & Laboratory Medicine 138, no. 5 (2014): 643–50. http://dx.doi.org/10.5858/arpa.2012-0227-oa.

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Context.—The ability of intermediate trophoblasts to invade maternal tissue during placentation depends on how well they can degrade the extracellular matrix. Invasion into the extracellular matrix requires many complex proteases. A disintegrin and metalloproteinase with thrombospondin motifs (ADAMTS) is a novel family of secreted metalloproteinases. The ADAMTS-1, -4, -5, and -14 subtypes are known to be expressed in human placenta, but little is understood about their expression patterns. Objective.—To examine the expression patterns of ADAMTS-1, -4, -5, and -14 in specific human placenta cel
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Ren, Pingping, Lin Zhang, Gaiping Xu, et al. "ADAMTS-1 and ADAMTS-4 Levels Are Elevated in Thoracic Aortic Aneurysms and Dissections." Annals of Thoracic Surgery 95, no. 2 (2013): 570–77. http://dx.doi.org/10.1016/j.athoracsur.2012.10.084.

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Santamaria, Salvatore, and Rens de Groot. "ADAMTS proteases in cardiovascular physiology and disease." Open Biology 10, no. 12 (2020): 200333. http://dx.doi.org/10.1098/rsob.200333.

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The a disintegrin-like and metalloproteinase with thrombospondin motif (ADAMTS) family comprises 19 proteases that regulate the structure and function of extracellular proteins in the extracellular matrix and blood. The best characterized cardiovascular role is that of ADAMTS-13 in blood. Moderately low ADAMTS-13 levels increase the risk of ischeamic stroke and very low levels (less than 10%) can cause thrombotic thrombocytopenic purpura (TTP). Recombinant ADAMTS-13 is currently in clinical trials for treatment of TTP. Recently, new cardiovascular roles for ADAMTS proteases have been discovere
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Dissertations / Theses on the topic "ADAMTS 1 e 4"

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Hsu, Yi-Ping. "The role of ADAMTS-4 and ADAMTS-1 in angiogenesis." Thesis, University of Sheffield, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.577642.

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Endothelial cells (ECs) form the innermost layer of all blood vessels, and their function and behaviour are important in angiogenesis. VEGF is the most potent angiogenic factor stimulating EC proliferation, migration, and differentiation as well as survival. Vascular endothelial growth factor receptor 2 (VEGFR2) is the major VEGF receptor which mediates VEGF-induced angiogenesis on ECs. Neuropilin 1 (NRPI) is a VEGF co-receptor that enhances VEGF-stimulated VEGFR2 angiogenic signals. A disintegrin and metalloproteinase with thrombospondin repeats (ADAMTS)-I, -2, -5, -8, and -9 have been shown
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Turner, Sharon Louise. "Investigation of ADAM17 and ADAMTS-1, -4 and -5 in liver carcinoma." Thesis, Sheffield Hallam University, 2009. http://shura.shu.ac.uk/20827/.

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Background: Proteolytic enzymes are important mediators of cellular proliferation, angiogenesis and remodelling of the extracellular matrix (ECM); all processes required for tumour growth and metastasis. However, the studies of proteolytic enzymes in hepatic tumours, both primary and metastatic, have largely been limited to specific matrix metalloproteinases e.g. MMP-2, -7 and -9, and urokinase-type plasminogen activator. ADAM17 (a disintegrin and metalloproteinase 17), a membrane-bound sheddase, releases membrane-bound proteins including growth factors, which could contribute to liver tumour
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Gibrel, Gehan G. F. "The role of ADAMTS-1, -4 and -5 in multiple sclerosis." Thesis, Sheffield Hallam University, 2012. http://shura.shu.ac.uk/20683/.

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ADAMTS (a disintegrin and metalloproteinase with thrombospondin motifs)-1, -4 and -5 are secreted enzymes which are members of the glutamyl endopeptidases (GEPs) group of ADAMTSs. These enzymes break down chondroitin sulphate proteoglycans (CSPGs) which are key components of brain extracellular matrix (ECM). In multiple sclerosis (MS), CSPG breakdown by ADAMTSs may enable axonal regeneration or conversely it may lead to alterations of the ECM, allowing influx of inflammatory cells promoting tissue damage. ADAMTS-1, -4 and -5 mRNA expression was studied by quantitative real-time PCR (qRT-PCR) u
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Owen, Kathryn. "Characterisation of ADAMTS-4 endocytosis by chondrosarcoma cells and chondrocytes." Thesis, Imperial College London, 2009. http://hdl.handle.net/10044/1/5575.

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Aggrecan is one of the most abundant components of the articular cartilage matrix and its degradation by aggrecanases is considered to be a key early event in the development of osteoarthritis. This study investigates whether the extracellular level of ADAMTS-4 (a disintegrin and metalloproteinase with thrombospondin type 1 motifs-4), one of the most studied aggrecanases, can be regulated by receptor-mediated endocytosis. Exogenously-added ADAMTS-4 disappeared from the medium of HTB94 cells and TCA-soluble degradation products of ADAMTS-4 increased in the medium over time. Fluorescent-labeled
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Lim, Ngee Han. "TIMP-3 interaction with aggrecanase 1 (ADAMTS-4) and aggrecanse 2 (ADAMTS-5)investigated by site-directed mutagenesis." Thesis, Imperial College London, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.506803.

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Degradation of the proteoglycan aggrecan, one ofthe major components ofthe cartilage matrix, is thought to be a critical step in the development of osteoarthritis (OA). Both matrix metalloproteinases (MMPs) and aggrecanases [members of the ADAMTS (a disintegrin and metalloprotease with thrombospondin motifs) family have been shown to degrade aggrecan in vitro. However, there is debate as to their relative roles in vivo. The tissue inhibitor of metalloproteinases 3 (TIMP-3), which inhibits the MMPs, is the most potent tissue inhibitor of the aggrecanases, ADAMTS-4 and ADAMTS-S. The purpose of t
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Xie, Zhe. "Wnt signaling in human cartilage degeneration and chondrocytes de-differentiation." Thesis, Université de Lorraine, 2016. http://www.theses.fr/2016LORR0111/document.

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La dérégulation de la signalisation Wnt est impliquée dans les anomalies du développement et dans la pathogenèse de nombreuses maladies, y compris l'arthrose. Au cours de ce travail, nous avons étudié l'effet de Wnt-3a sur l’ADAMTS-4 dans les explants de cartilage et les chondrocytes primaires humains. Nous avons observé que Wnt-3a régule négativement l'expression de cette agrecanase et avons démontré que cette inhibition est médiée par Frizzled-8 via l’activation de la voie canonique et inhibition de l'activité de NFκB. En outre, nous avons montré que Wnt-3a est capable de s’opposer à l'induc
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Lima, Maíra de Assis. "Influência dos hormônios esteroidais na migração, invasão e expressão das proteases ADAMTS 1 e 4 em células derivadas de tumores de ovários." Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/42/42134/tde-05102015-190307/.

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O câncer de ovário é uma neoplasia ginecológica e alternâncias hormonais poderiam ter um papel na manifestação da doença. As ADAMTS´s são proteases secretadas dependentes de Zn2+/Ca2+. Nosso objetivo foi avaliar se há Influência de hormônios sexuais nos níveis de expressão de mRNA, proteínas e distribuição de ADAMTS 1 e 4 e alteração na migração e invasão em células tumorais humanas de ovário. As linhagens foram tratadas com progesterona, estrógeno ou testosterona e o controle não recebeu tratamento. O estrógeno e a testosterona induziram uma menor e a progesterona uma maior expressão gênica d
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Gendron, Christi Marie. "Investigation of the role of ADAMTS-4 and ADAMTS-5 in cartilage aggrecan degradation." Thesis, Imperial College London, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.415906.

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Jones, Gavin Carl. "Molecular interactions and activities of ADAMTS-4 and -5." Thesis, University of Sheffield, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.412783.

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Le, Behot Audrey. "Nouvelles cibles thérapeutiques de l'accident vasculaire cérébral : GpIbα et ADAMTS-4". Caen, 2013. http://www.theses.fr/2013CAEN3164.

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Le seul traitement aigu de l’AVC ischémique est la thrombolyse par l’activateur tissulaire du plasminogène recombinant (rtPA). Néanmoins, l’efficacité de la thrombolyse est influencée par la composition du thrombus : lorsqu’il est riche en plaquettes, il est résistant au rtPA. En modélisant in vivo 2 étiologies (embolique et thrombose in situ) de l’AVC ischémique, l’objectif de ce travail a été d’étudier l’efficacité de la thrombolyse et les mécanismes de la thrombose. Dans les conditions ischémiques, la limitation des dommages cérébraux dépend du rétablissement rapide de la perfusion sanguine
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Books on the topic "ADAMTS 1 e 4"

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Apte, Suneel S., ed. ADAMTS Proteases. Springer New York, 2020. http://dx.doi.org/10.1007/978-1-4939-9698-8.

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Age Concern, England. European Resource Unit. 1:4 Media Project. 1:4. [European Resource Unit, Age Concern England], 1996.

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U.S. DEPT. OF THE ARMY. Organizational maintenance manual: Truck, cargo : 1 1/4 ton, 4 x 4, M715 ... truck, ambulance : 1 1/4 ton, 4 x 4, M725 ... truck, maintenance : 1 1/4 ton, 4 x 4, M726. Reproduced and distributed by Portrayal Press, 2007.

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Kagaku Busshitsu Hyōka Kenkyū Kikō and Shin Enerugī Sangyō Gijutsu Sōgō Kaihatsu Kikō (Japan), eds. 4-biniru-1-shikurohekisen: 4-vinyl-1-cyclohexene. Seihin Hyōka Gijutsu Kiban Kikō Kagaku Busshitsu Hyōka Kenkyū Kikō, 2009.

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William, Eggleston. 2 1/4. Twin Palms Publishers, 1999.

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Katrin, Meder, and Kunstverein Braunschweig, eds. 4 1/2. Kunstverein Braunschweig, 2009.

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s, De me tre s. De me triade. Katalogoi 1-4. Agra, 1986.

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Akreyî, Nafiʻ. 4 + 1 = Şanoname. s.n.], 1987.

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Tănsănică, George. 4 în 1. Sakura, 2001.

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Téllez, Javier. 4 1/2. Edited by Meder Katrin and Kunstverein Braunschweig. Kunstverein Braunschweig, 2009.

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Book chapters on the topic "ADAMTS 1 e 4"

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Fowkes, Milan M., and Ngee H. Lim. "Purification and Activity Determination of ADAMTS-4 and ADAMTS-5 and Their Domain Deleted Mutants." In Methods in Molecular Biology. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9698-8_7.

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Hubmacher, Dirk. "Cell-Based Interaction Analysis of ADAMTS Proteases and ADAMTS-Like Proteins with Fibrillin Microfibrils." In Methods in Molecular Biology. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9698-8_16.

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Apte, Suneel S. "ADAMTS Proteins: Concepts, Challenges, and Prospects." In Methods in Molecular Biology. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9698-8_1.

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Jones, Gavin C., Mireille N. Vankemmelbeke, and David J. Buttle. "Expression of Recombinant ADAMTS in Insect Cells." In Methods in Molecular Biology. Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-299-5_5.

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Lambert, Jordi, and Dylan R. Edwards. "Analysis of ADAMTS Effects on Cell Adhesion and Migration." In Methods in Molecular Biology. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9698-8_15.

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Zhang, Rong-Mo, Heena Kumra, and Dieter P. Reinhardt. "Quantification of Extracellular Matrix Fiber Systems Related to ADAMTS Proteins." In Methods in Molecular Biology. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9698-8_19.

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Colige, Alain C. "Challenges and Solutions for Purification of ADAMTS Proteases: An Overview." In Methods in Molecular Biology. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9698-8_4.

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Kohno, Takao, Himari Ogino, Yuko Yamakage, and Mitsuharu Hattori. "Expression and Preparation of Recombinant Reelin and ADAMTS-3 Proteins." In Methods in Molecular Biology. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9698-8_8.

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"ADAMTS-4." In Springer Reference Medizin. Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_300024.

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"ADAMTS." In Encyclopedia of Metalloproteins. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-1533-6_100031.

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Conference papers on the topic "ADAMTS 1 e 4"

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Chen, C. T., S. Park, M. Bhargava, and P. A. Torzilli. "Inhibitory Effect of Mechanical Load on IL-1 Induced Cartilage Degradation Is Mediated by Interferon-Gamma and IL-1 Receptor 1." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193230.

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Matrix remodeling in articular cartilage is regulated by the elevation and activation of aggrecanases (ADAMTS-4 and ADAMTS-5) and matrix metalloproteinases (MMPs) [2–4, 7–9, 10]. Several recent studies from our and other groups have shown that mechanical loading can counteract interleukin 1 (IL-1) induced pro-inflammatory and catabolic events by down-regulating aggrecanases, MMPs, and pro-inflammatory genes [1, 3, 5, 6], but the molecular mechanism is not clear. Many previous studies have shown that the regulation of pro-inflammatory effect of IL-1 come from several aspects: anti-inflammatory
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Killian, Megan L., Barbara Zielinska, and Tammy L. Haut Donahue. "Role of IL-1 on Aggrecanase and COX-2 Gene Expression of Meniscal Explants Following Dynamic Compression." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19110.

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The menisci within the knee likely respond to adverse loading conditions, leading to aggravated cartilage damage and fissuring [1]. Upregulation of catabolic molecules such as interleukin-1α (IL-1α), metalloproteinases (MMPs), aggrecanases (ADAMTS-4 and -5), and cyclooxygenase-2 (COX-2), as well as release of proteoglycans [2], have been shown in vitro for meniscal explants following dynamic loading [3]. A crucial event in matrix degradation is the loss of aggrecan, caused by the ADAMTS family [4]. In osteoarthritic cartilage, IL-1 has been shown to influence COX-2 activity, leading to increas
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DiTullio, A. E., S. Park, P. A. Torzilli, and C. T. Chen. "Cyclic Compression of Chondrocytes Counteracts Pro-Inflammatory Tissue Remodeling Induced by Interleukin-1." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193027.

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Chondrocytes in tissue engineered constructs face challenging environments when first transplanted into a synovial joint, including high levels of compression/shear and pro-inflammatory cytokines. The joint level of interleukin 1 (IL-1) after trauma injury and in a repaired joint is acutely elevated. Matrix remodeling in tissue engineered constructs can be easily affected by the elevation and activation of aggrecanases (ADAMTS-4 and ADAMTS-5) and matrix metalloproteinases (MMPs) [2–4, 7–9, 11]. Several recent studies suggest that tensile loading and unconfined compression of chondrocytes has s
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Wolfe, Valerie M., Seonghun Park, Marjana Tomic, Peter A. Torzilli, and C. T. Christopher Chen. "Load Down-Regulates TNF-Alpha Induced Cartilage Degradation in Part Through NF-KB and P38 Pathways." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176541.

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Pro-inflammatory cytokines, such as interleukin-1 (IL-1) and tumor necrosis factor (TNF), can induce cartilage degradation after acute injury or in inflammatory diseases [1,2,3,7]. The degradative events are coordinated through the elevation and activation of two classes of enzymes, namely matrix metalloproteinases (MMPs) and aggrecanases (ADAMTS-4 and −5) [1,6]. Prior studies suggested that pro-inflammatory responses induced by IL-1β can be inhibited by tensile load [2] and more recently by cyclic compression [8]. It is, however, not clear whether load affects other cytokines, such as TNF-α.
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Mishra, Hemant K., Wu Jianming, Nabendu Pore, and Bruce Walcheck. "Abstract B57: Effect of ADAM17 inhibition on the release of IFN-y during natural killer cell-mediated antibody dependent cellular cytotoxicity in cancer." In Abstracts: AACR Special Conference on Tumor Immunology and Immunotherapy; October 1-4, 2017; Boston, MA. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/2326-6074.tumimm17-b57.

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Tang, Huiyuan, Manfai Lee, Eun Ho Kim, Daniel Bishop, and George M. Rodgers. "Abstract 1816: Endogenous ADAMTS-13 regulates angiogenesis in cultured human endothelial cells." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-1816.

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Yu, Huangsheng, Jia Tong, and Dianhua Wu. "New Optimal (14p, {4, 5}, 1, (3/4, 1/4))-OOCs." In 2015 Seventh International Workshop on Signal Design and its Applications in Communications (IWSDA). IEEE, 2015. http://dx.doi.org/10.1109/iwsda.2015.7458389.

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Benoit, Heather D., Jace D. Kelley, Joel D. White, and Brian A. Garner. "Designing a Therapeutic Mechanical Horse." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19581.

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Hippotherapy is a strategy that uses the movement of a horse to treat individuals with any of a variety of neuro-musculoskeletal disabilities including cerebral palsy, paralysis, traumatic brain injury, stroke and others. The patients’ overall treatment program includes time riding on a therapeutic horse under the direction of a physical therapist. The premise behind hippotherapy is that the movement of the horse drives the patients’ body, and particularly the hips, in a pattern that is similar to natural human movements such as healthy human gait [1][2][3][4]. The rhythmic motion of the ridin
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Li, Bin, Xiaobo Yang, Ankang Jin, Yunqing Zhang, and James Yang. "In-Plane Flexible Ring Tire Model Validation Through ADAMS FTire Model Virtual Tests." In ASME 2015 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2015. http://dx.doi.org/10.1115/detc2015-46630.

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This paper presents the validation for the newly developed in-plane flexible ring tire model by using ADAMS FTire model simulation. The developed in-plane model is unique in two aspects: (1) the neighboring belt segments are connected through normal and tangential directions by springs and dampers, each belt segment is a rigid body and its mass is accumulated at its geometric center. Each belt segment is always perpendicular to the line formed by the wheel center and the belt geometric center, thus there is no rotational constrains between the neighboring belt segments; (2) the representation
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Luo, Yifei, Gang Chen, and Kuan Zhou. "60Gb/s low jitter 4:1 Mux and 1:4 DeMux." In 2008 51st IEEE International Midwest Symposium on Circuits and Systems (MWSCAS). IEEE, 2008. http://dx.doi.org/10.1109/mwscas.2008.4616868.

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Reports on the topic "ADAMTS 1 e 4"

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Demkowicz, Paul Andrew, Jason M. Harp, Philip L. Winston, Scott A. Ploger, and Isabella J. van Rooyen. AGR-1 Compact 4-1-1 Post-Irradiation Examination Results. Office of Scientific and Technical Information (OSTI), 2016. http://dx.doi.org/10.2172/1260880.

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Lewis, Bonnie F. Navy Comptroller. Volume 4, Issue 1. Defense Technical Information Center, 1993. http://dx.doi.org/10.21236/ada269432.

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Briuer, Elke. Dredging Research, Volume 1, Number 4. Defense Technical Information Center, 1998. http://dx.doi.org/10.21236/ada360537.

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Hunn, John, Fred Montgomery, Darren Skitt, and Grant Helmreich. Radial Deconsolidation and Leach-Burn-Leach of AGR-3/4 Compact 1-4 and 10-4. Office of Scientific and Technical Information (OSTI), 2020. http://dx.doi.org/10.2172/1709104.

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Mcdonald R.J., D. A. Landis, R. M. Maier, B. S. Rude, and G. J. Wozniak. Quad fourfold (4 X 4) logic unit (LBL No. 21X6421 P-1). Office of Scientific and Technical Information (OSTI), 1987. http://dx.doi.org/10.2172/6760149.

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Mitchell, Mark V. Module 4 - Part 1 - Hand Calculation Methods. Office of Scientific and Technical Information (OSTI), 2014. http://dx.doi.org/10.2172/1122909.

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Garrett, R. L., and I. K. Paik. Supplementary safety system 1/4 scale testing. Office of Scientific and Technical Information (OSTI), 1993. http://dx.doi.org/10.2172/10107406.

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Stancil, J. The Energy Messenger, Number 1, Volume 4. Office of Scientific and Technical Information (OSTI), 1995. http://dx.doi.org/10.2172/10117385.

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Punjabi, V. Polarization experiments with hadronic and electromagnetic probes. [2. 1 and 4. 4 GeV]. Office of Scientific and Technical Information (OSTI), 1993. http://dx.doi.org/10.2172/6588086.

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Evosevich, S. Hanford Inventory Program Systems Operating Manual: Volumes 1--4. Revision 1. Office of Scientific and Technical Information (OSTI), 1994. http://dx.doi.org/10.2172/10181888.

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