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Journal articles on the topic 'Adenomatose'

Author: Grafiati

Published: 4 June 2021

Last updated: 3 February 2022

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1

Cosechen, Mayara Schulze, Adma Silva de Lima Wojcik, Flávio Meingast Piva, Betina Werner, and Sérgio Zuneda Serafini. "Adenomatose erosiva do mamilo." Anais Brasileiros de Dermatologia 86, no. 4 suppl 1 (2011): 17–20. http://dx.doi.org/10.1590/s0365-05962011000700003.

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A adenomatose erosiva do mamilo é uma complexa proliferação benigna mamária que pode ser confundida com neoplasias malignas da mama. A apresentação típica cursa com descarga mamária, eritema, erosão e formação de crostas. O processo é geralmente assintomático e de instalação insidiosa. A adenomatose erosiva do mamilo pode ser confundida com condições benignas, como a dermatite de contato, psoríase e infecções, mas seu principal diagnóstico diferencial é a Doença de Paget. O tratamento é cirúrgico e o prognóstico, excelente

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2

Silva, Alisson Paulo, Lorenna Alves Gomes, Bianca Moreira Souza, et al. "NANISMO EM GATO ASSOCIADO À ADENOMATOSE FOLICULAR TIREOIDIANA." Revista de Ciência Veterinária e Saúde Pública 5, no. 2 (2018): 175. http://dx.doi.org/10.4025/revcivet.v5i2.41850.

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O trabalho descreve os achados anatomo-histopatológicos e radiológicos do esqueleto de um gato com nanismo associado à adenomatose folicular tireoidiana. Uma gata de 40 dias com pequena estatura e pesando 100g foi encaminhada para a necropsia com histórico de crescimento ósseo severamente reduzido e suspeita de nanismo. Na necropsia, os ossos de todo o esqueleto eram curtos e frágeis. No RX, os ossos longos apresentaram epífises radioluscentes. Microscopicamente, foram observadas características de osteoporose e interrupção do crescimento endocondral, intramembranoso e aposicional, caracteriza

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3

Ridder, Dirk, Diane Duret, Marcus-Alexander Wörns та ін. "Entstehung eines hepatozellulären Karzinoms in einem inflammatorischen Adenom mit β-Catenin-Mutation bei einem Patienten mit hepatozellulärer Adenomatose und HNF1α-Keimbahnmutation". Zeitschrift für Gastroenterologie 57, № 01 (2019): 46–51. http://dx.doi.org/10.1055/a-0821-7154.

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ZusammenfassungMutationen des hepatocyte nuclear factor 1 α-(HNF1α) Gens lösen einen MODY3-Diabetes aus und sind mit einer hepatozellulären Adenomatose assoziiert. Eine maligne Transformation von HNF1α-mutierten hepatozellulären Adenomen ist selten. Wir beschreiben hier den Fall eines 28-jährigen Mannes mit heterozygoter HNF1α-Keimbahnmutation mit MODY3-Diabetes und einer hepatozellulären Adenomatose mit multiplen, HNF1α-inaktivierten Adenomen, der ein inflammatorisches Adenom mit β-Catenin-Mutation mit maligner Transformation in ein gut differenziertes hepatozelluläres Karzinom (HCC) entwicke

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4

Driemeier, David, Valéria Moojen, Giani Saraiva Faccini, and Rosemari Teresinha de Oliveira. "Adenomatose pulmonar ("jaagsiekte") em ovino no Rio Grande de Sul." Ciência Rural 28, no. 1 (1998): 147–50. http://dx.doi.org/10.1590/s0103-84781998000100026.

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Descreve-se, no Rio Grande do Sul, Brasil, o primeiro caso de adenomatose pulmonar ("jaagsiekte ") em um ovino da raça Karakul, fêmea de 2 anos, filha de pais importados da Alemanha. O ovino morreu de acidose lática por sobrecarga alimentar e as lesões pulmonares representaram achados incidentais. Macroscopicamente encontraram-se duas áreas esbranquiçadas nodulares com aproximadamente 4 e 6cm de diâmetro respectivamente, com bordos irregulares que, microscopicamente, correspondiam a tumoração epitelial bem diferenciada, formando projeçôes papilares para o interior do lúmen alveolar e bronquiol

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5

Machado, Márcio Martins, Ana Cláudia Ferreira Rosa, Paulo Herman, William Abrão Saad, and Giovanni Guido Cerri. "Adenomatose hepática múltipla: diagnóstico pela ultra-sonografia intra-operatória (USIO). Nota prévia." Radiologia Brasileira 34, no. 6 (2001): 369–72. http://dx.doi.org/10.1590/s0100-39842001000600010.

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Os autores relatam um caso de adenomatose hepática múltipla diagnosticado pela ultra-sonografia intra-operatória (USIO). Neste caso foram identificadas 13 lesões pela USIO. Os exames pré-operatórios (ultra-sonografia abdominal, tomografia computadorizada helicoidal e ressonância magnética) mostraram apenas três lesões, que foram inicialmente consideradas potencialmente ressecáveis. Todos os adenomas apresentavam consistência idêntica à do fígado adjacente, não sendo, portanto, perceptíveis à palpação. A USIO apresenta maior acurácia na identificação de nódulos hepáticos, quando comparada com o

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6

Rabast, U., T. Padel, H. Rammert, E. Helwing, and O. Klinge. "Adenomatose der Gallenwege als seltene Ursache einer Cholestase." DMW - Deutsche Medizinische Wochenschrift 114, no. 22 (2008): 866–70. http://dx.doi.org/10.1055/s-2008-1066686.

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7

Costa, Brunno Augusto José, Lucas de Sena Leme, Bruna Zini de Paula Freitas, et al. "POLIPOSE ADENOMATOSE FAMILIAR E VARIANTE RARO DO GENE MSH6 ‐ RELATO DE CASO." Journal of Coloproctology 38 (October 2018): 37. http://dx.doi.org/10.1016/j.jcol.2018.08.079.

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8

Bulow, S. "Duodenal adenomatosis in familial adenomatous polyposis." Gut 53, no. 3 (2004): 381–86. http://dx.doi.org/10.1136/gut.2003.027771.

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9

B�low, S., T. Alm, O. Fausa, R. Hultcrantz, H. J�rvinen, and H. Vasen. "Duodenal adenomatosis in familial adenomatous polyposis." International Journal of Colorectal Disease 10, no. 1 (1995): 43–46. http://dx.doi.org/10.1007/bf00337586.

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10

Виговська, К. Л. "Диференційна діагностика пухлин молочних залоз у кішок". Вісник Полтавської державної аграрної академії, № 1 (28 березня 2013): 178–82. http://dx.doi.org/10.31210/visnyk2013.01.44.

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Наведені дані, отримані в результаті клінікогістологічних досліджень пухлин молочних залоз кішок. У досліджені брали участь 79 тварин віком від семи місяців до вісімнадцяти років, які поступали на прийом із приводу новоутворень молочної залози. Гістологічні дослідження пухлин молочної залози підтвердили діагноз: доброякісні пухлини: 10 – фіброаденоматози, 4 – кістози, 6 – аденоматози, 7 – змішаної будови, 52 злоякісні пухлини (аденокарциноми). З-поміж злоякісних пухлин виділено зразки інфільтруючого та неінфільтруючого раку, що має тубулярну, папілярну або солідну структуру. Гістологічні дослі

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11

Kadmon, Martina, Andrea Tandara, and Christian Herfarth. "Duodenal adenomatosis in familial adenomatous polyposis coli." International Journal of Colorectal Disease 16, no. 2 (2001): 63–75. http://dx.doi.org/10.1007/s003840100290.

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12

Leal, Raquel Franco, Maria de Lourdes Setsuko Ayrizono, Cláudio Saddy Rodrigues Coy, Francisco Callejas-Neto, João José Fagundes, and Juvenal Ricardo Navarro Góes. "Polipose gastroduodenal em doentes com polipose adenomatosa familiar Pós-Retocolectomia." Arquivos de Gastroenterologia 44, no. 2 (2007): 133–36. http://dx.doi.org/10.1590/s0004-28032007000200009.

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RACIONAL: As manifestações extracólicas, como os pólipos gastroduodenais e o tumor do duodeno, são fatores que influenciam a morbimortalidade dos doentes com polipose adenomatosa familiar no seguimento pós-retocolectomia total. OBJETIVO: Investigar a freqüência destas alterações em doentes com polipose adenomatosa familiar e verificar a eficácia do rastreamento endoscópico. MÉTODO:No período de 1984 a 2005, 62 doentes com polipose adenomatosa familiar pós-retocolectomia foram estudados retrospectivamente pelo Grupo de Coloproctologia da Faculdade de Ciências Médicas da Universidade Estadual de

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13

Maehata, Yuji, Motohiro Esaki, Minako Hirahashi, Takanari Kitazono, and Takayuki Matsumoto. "Duodenal adenomatosis in Japanese patients with familial adenomatous polyposis." Digestive Endoscopy 26 (April 2014): 30–34. http://dx.doi.org/10.1111/den.12255.

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14

Latchford, Andrew, Michelle Gallagher, Douglas Newton, Swain David, Robin Phillips, and Brian Saunders. "Therapeutic Endoscopy for Duodenal Adenomatosis in Familial Adenomatous Polyposis." Gastrointestinal Endoscopy 63, no. 5 (2006): AB176. http://dx.doi.org/10.1016/j.gie.2006.03.382.

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15

Lepistö, A., T. Kiviluoto, J. Halttunen, and H. Järvinen. "Surveillance and treatment of duodenal adenomatosis in familial adenomatous polyposis." Endoscopy 41, no. 06 (2009): 504–9. http://dx.doi.org/10.1055/s-0029-1214719.

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16

Ficari, F., A. Cama, R. Valanzano, et al. "APC gene mutations and colorectal adenomatosis in familial adenomatous polyposis." British Journal of Cancer 82, no. 2 (2000): 348–53. http://dx.doi.org/10.1054/bjoc.1999.0925.

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17

Utsunomiya, J., H. Aioi, T. Yamamura, et al. "Surgical managements and their result of adenomatosis coli(familial adenomatous polyposis)." Nippon Daicho Komonbyo Gakkai Zasshi 40, no. 6 (1987): 721–33. http://dx.doi.org/10.3862/jcoloproctology.40.721.

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18

Skipworth, James R. A., Clare Morkane, Dimitri Aristotle Raptis, et al. "Pancreaticoduodenectomy for advanced duodenal and ampullary adenomatosis in familial adenomatous polyposis." HPB 13, no. 5 (2011): 342–49. http://dx.doi.org/10.1111/j.1477-2574.2011.00292.x.

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19

Silva, Julia Schmidt, Afonso Calil Mury Mallmann, Ruytakashi Koshimizu, Daniela Cerqueira Koppe, Fabíola Elizabete Savaris, and Luciano Pinto de Carvalho. "Adenomas colorretais: fatores de risco associados à displasia de alto grau." Revista Brasileira de Coloproctologia 29, no. 2 (2009): 209–15. http://dx.doi.org/10.1590/s0101-98802009000200008.

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FUNDAMENTOS: O estudo dos pólipos, em especial os adenomatosos, é relevante devido à correlação direta com o carcinoma colorretal. Objetivo: Analisar quais são os fatores de risco para uma displasia de alto grau do pólipo adenomatoso retirados endoscopicamente. MÉTODOS: Avaliamos todas as colonoscopias realizadas pelo Serviço de Coloproctologia no período de janeiro de 2003 a dezembro de 2006. Foram incluídos os pacientes com diagnóstico de pólipos adenomatosos. Analisamos a existência da relação entre adenomas com displasia de alto grau e os fatores associados dos pacientes e pólipos. RESULTA

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20

Brambilla, Eduardo, Marcos Antonio Dal Ponte, Viviane Raquel Buffon, Roberto Taboada Fellini, Alexandre Dal Bosco, and Rafael Schalins May. "Aderência ao controle colonoscópico nos pacientes submetidos à ressecção endocóspica de adenomas." Revista Brasileira de Coloproctologia 26, no. 3 (2006): 249–52. http://dx.doi.org/10.1590/s0101-98802006000300004.

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OBJETIVOS: Analisar a aderência de pacientes ao controle colonoscópico após ressecção endoscópica de pólipos adenomatosos. PACIENTES E MÉTODOS: Foram avaliados 74 pacientes que realizaram colonoscopia e apresentaram pólipos adenomatosos. Estes indivíduos foram orientados a realizar colonoscopia de controle após 1 ano, de acordo com o número de adenomas, diâmetro e classificação histológica. Além da orientação na consulta, os pacientes foram avisados por ligação telefônica no período indicado para colonoscopia de controle. RESULTADOS: No período de outubro de 2000 até abril de 2006 foram acompa

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21

Vasen, H. F., S. Bulow, T. Myrhoj, et al. "Decision analysis in the management of duodenal adenomatosis in familial adenomatous polyposis." Gut 40, no. 6 (1997): 716–19. http://dx.doi.org/10.1136/gut.40.6.716.

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22

Skipworth, J. R. A., S. Bannoo, A. Shankar, S. Clark, G. J. Webster, and S. P. Pereira. "Utility of endoscopic ultrasound in patients with familial adenomatous polyposis and duodenal adenomatosis." Pancreatology 12, no. 3 (2012): e14. http://dx.doi.org/10.1016/j.pan.2012.03.037.

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23

Berkhout, M., H. M. J. Roelofs, R. H. M. te Morsche, et al. "Detoxification enzyme polymorphisms are not involved in duodenal adenomatosis in familial adenomatous polyposis." British Journal of Surgery 95, no. 4 (2007): 499–505. http://dx.doi.org/10.1002/bjs.6027.

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24

Labib, Peter L., George Goodchild, James P. Turbett, et al. "Endoscopic ultrasound in the assessment of advanced duodenal adenomatosis in familial adenomatous polyposis." BMJ Open Gastroenterology 6, no. 1 (2019): e000336. http://dx.doi.org/10.1136/bmjgast-2019-000336.

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ObjectiveCurrent surveillance strategies for duodenal adenomatosis in familial adenomatous polyposis (FAP) miss malignancies and underestimate cancer risk in ampullary disease. This study aimed to evaluate the utility of endoscopic ultrasound (EUS) in the assessment of FAP patients with duodenal and/or ampullary polyposis referred for surgical intervention.DesignA retrospective analysis of FAP patients undergoing index EUS between December 2006 and May 2015 was performed. Follow-up was completed in January 2018, including review of all EUS procedures and surgical interventions (median follow-u

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Cyrany, Jiri, Stanislav Rejchrt, Marcela Kopacova, Vera Tycova, and Jan Bures. "W1602: Duodenal Adenomatosis in Patients With Familial Adenomatous Polyposis - Endoscopic Diagnosis and Therapy." Gastrointestinal Endoscopy 71, no. 5 (2010): AB369—AB370. http://dx.doi.org/10.1016/j.gie.2010.03.1010.

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26

Soons, E., T. M. Bisseling, M. C. A. van Kouwen, G. Möslein, and P. D. Siersema. "Endoscopic management of duodenal adenomatosis in familial adenomatous polyposis—A case‐based review." United European Gastroenterology Journal 9, no. 4 (2021): 461–68. http://dx.doi.org/10.1002/ueg2.12071.

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27

Ferreira Gomes, Nara Maria Ribeiro Soares, Jeany Borges e. Silva Ribeiro, and Djalma Ribeiro Costa. "ASSOCIAÇÃO ENTRE LOCALIZAÇÃO, ASPECTO MORFOLÓGICO E PADRÃO HISTOLÓGICO DE PÓLIPOS COLORRETAIS AVALIADOS NO HOSPITAL UNIVERSITÁRIO DA UNIVERSIDADE FEDERAL DO PIAUÍ." Jornal de Ciências da Saúde do Hospital Universitário da Universidade Federal do Piauí 1, no. 2 (2018): 29. http://dx.doi.org/10.26694/2595-0290.20181229-416995.

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INTRODUÇÃO: A retirada dos pólipos adenomatosos por colonoscopia impede a progressão da seqüência adenoma-carcinoma, favorecendo a prevenção de câncer colorretal. Assim, objetiva-se demonstrar a associação entre a localização do pólipo colorretal com o aspecto morfológico e o padrão histológico em pacientes atendidos no Hospital Universitário da Universidade Federal do Piauí. METODOLOGIA: Estudo transversal de uma série de exames de colonoscopia realizados no período de setembro de 2015 a setembro de 2016. Variáveis demográficas, laudo colonoscópico e descrição anatomopatológica foram analisad

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28

Özkan, Cumali, Serkan Yıldırım, Zübeyir Huyut, and Mustafa Özbek. "Selected tumour biomarker levels in sheep with pulmonary adenomatosis." Journal of Veterinary Research 64, no. 1 (2020): 39–44. http://dx.doi.org/10.2478/jvetres-2020-0017.

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AbstractIntroductionSheep pulmonary adenomatosis (ovine pulmonary adenomatosis, OPA, Jaagsiekte) is a chronic contagious bronchoalveolar carcinoma caused by the Jaagsiekte sheep retrovirus. Since effective treatment and a vaccination procedure are not currently possible, control and eradication of the disease is difficult. It leads to serious economic losses around the world, therefore studies are currently underway in order to design control and eradication programmes. In this study, levels and changes in selected tumour markers (carcinoembryonic antigen (CEA), carbohydrate antigen (CA) 125,

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Matsumoto, Takayuki, Mitsuo Iida, Yoichiro Kobori, Mitsuru Mizuno, Shotaro Nakamura, and Kazuoki Hizawa. "Progressive Duodenal Adenomatosis in a Familial Adenomatous Polyposis Pedigree with APC Mutation at Codon 1556." Diseases of the Colon & Rectum 45, no. 2 (2002): 229–33. http://dx.doi.org/10.1007/s10350-004-6153-9.

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30

Abdulhameed, Tenya, Zheen Jaff, and Nali Maaruf. "Clinicopathological study and immunohistochemical evaluation of cyclin D1 in adenomatous polyps." Zanco Journal of Medical Sciences 25, no. 1 (2021): 456–63. http://dx.doi.org/10.15218/zjms.2021.007.

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Background and objective: There are many histological types of colorectal polyps. Most of these polyps are benign epithelial polyps harboring very low risks of cancerous changes. Adenomatous polyps are the most known cancer precursors. Cyclin D1 gene amplification and or overexpression occurs in many human cancers. Cyclin D1 participates potentially in the multistep process of colorectal carcinogenesis. The present study aimed to assess the clinicopathological features of colorectal polyps and evaluate the significance of immunohistochemistry expression of cyclin D1 in adenomatouse polyp as a

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31

Lee, Ho Sung, Jae Sung Choi, Ki Hyun Seo, et al. "A Case of Congenital Cystic Adenomatoid Malformation of the Lng with Atypical Adenomatous Hyperplasia in Adult." Tuberculosis and Respiratory Diseases 66, no. 5 (2009): 385. http://dx.doi.org/10.4046/trd.2009.66.5.385.

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32

de Vos tot Nederveen Cappel, W. H., H. J. Järvinen, J. Björk, T. Berk, G. Griffioen, and H. F. A. Vasen. "Worldwide survey among polyposis registries of surgical management of severe duodenal adenomatosis in familial adenomatous polyposis." British Journal of Surgery 90, no. 6 (2003): 705–10. http://dx.doi.org/10.1002/bjs.4094.

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33

Alderlieste, Yasser A., Barbara A. Bastiaansen, Elisabeth M. H. Mathus-Vliegen, Dirk J. Gouma, and Evelien Dekker. "“High rate of recurrent adenomatosis during endoscopic surveillance after duodenectomy in patients with familial adenomatous polyposis”." Familial Cancer 12, no. 4 (2013): 699–706. http://dx.doi.org/10.1007/s10689-013-9648-5.

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34

Hossne, Rogério Saad, Mara Fernandes Maranhão, Flávio Augusto de Carvalho, and Fabiana Guandalini Mendes. "Estudo retrospectivo do resultado anatomopatológico de 100 polipectomias colonoscópicas realizadas na FMB-UNESP." Revista Brasileira de Coloproctologia 27, no. 3 (2007): 251–55. http://dx.doi.org/10.1590/s0101-98802007000300001.

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OBJETIVO: Analisar retrospectivamente o resultado do estudo anatomopatológico de polipectomias colonoscópicas realizadas no Setor de Endoscopia da Faculdade de Medicina de Botucatu - UNESP durante os anos de 2002 e 2003. Material e Métodos: Estudamos retrospectivamente, a partir de procedimentos colonoscópicos realizados em nosso serviço, as características dos pólipos retirados: tamanho, localização e distribuição, bem como o motivo da indicação do mesmo e o resultado do estudo anatomopatológico. RESULTADOS: Num total de 100 polipectomias colonoscópicas realizadas em 75 pacientes, observamos

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35

Sulbaran, M., F. Campos, U. Ribeiro, et al. "Risk factors for advanced duodenal and ampullary adenomatosis in familial adenomatous polyposis: a prospective, single-center study." Endoscopy International Open 06, no. 05 (2018): E531—E540. http://dx.doi.org/10.1055/a-0577-2650.

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Abstract Background and study aims To determine the clinical features associated with advanced duodenal and ampullary adenomas in familial adenomatous polyposis. Secondarily, we describe the prevalence and clinical significance of jejunal polyposis. Patients and methods This is a single center, prospective study of 62 patients with familial adenomatous polyposis. Duodenal polyposis was classified according to Spigelman and ampullary adenomas were identified. Patients with Spigelman III and IV duodenal polyposis underwent balloon assisted enteroscopy. Predefined groups according to Spigelman an

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Mathus-Vliegen, Elisabeth M. H., Karam S. Boparai, Evelien Dekker, and Nan van Geloven. "Progression of duodenal adenomatosis in familial adenomatous polyposis: due to ageing of subjects and advances in technology." Familial Cancer 10, no. 3 (2011): 491–99. http://dx.doi.org/10.1007/s10689-011-9433-2.

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Račaitė, Julita, Alina Šumkovskaja, Audronė Arlauskienė, Ingrida Pilypienė, and Elena Landsbergytė-Bukauskienė. "Congenital cystic adenomatous malformation: a case report and a literature review." Acta medica Lituanica 25, no. 2 (2018): 95–100. http://dx.doi.org/10.6001/actamedica.v25i2.3762.

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Background. A congenital cystic adenomatoid malformation (CCAM) is a foetal pulmonary development abnormality caused by airway dysgenesis that is characterized by cystic or adenomatous lesions in the terminal bronchioles. The size of the mass, the degree of the mediastinal shift, and the presence of hydrops and polyhydramnios can all affect the severity of a case. Treatment can be initiated at early stages by applying prenatal and postnatal methods. Because CCAM is a rare pathology that is often only accidentally diagnosed during routine ultrasounds, we would like to share our case report to e

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38

Ramanojam, Shandilya, Mayur Vilas Limbhore, Pallavi Rathi, Vikrant Sane, and Adil Mevawala. "ADENOMATOID ODONTOGENIC TUMOR CASE REPORT." Era's Journal of Medical Research 6, no. 2 (2019): 186–88. http://dx.doi.org/10.24041/ejmr2019.154.

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Parés, David, Miguel Pera, Sara González, Montserrat Pascual Cruz, and Ignacio Blanco. "Poliposis adenomatosa familiar." Gastroenterología y Hepatología 29, no. 10 (2006): 625–35. http://dx.doi.org/10.1157/13095198.

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40

Pox, Christian. "Familiäre adenomatöse Polyposis." Gastroenterologie up2date 9, no. 04 (2013): 239–50. http://dx.doi.org/10.1055/s-0033-1358911.

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41

Rozas Llerena, Rubén. "Hiperplasia adenomatosa del endocervix." Revista Peruana de Ginecología y Obstetricia 37, no. 11 (2015): 51–56. http://dx.doi.org/10.31403/rpgo.v37i1561.

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Se reporta los primeros 50 casos de hiperplasia adenomatosa del endocervix, en 500 biopsias de cuello uterino. La edad promedio fue 47 años. El síntoma principal fue ginecorragia. El diagnóstico clínico predominante fue fibroma y los demás diagnósticos anatomo- patológicos en orden de importancia fueron: Adenomiosis, leiomiomas, endometrio atrófico, endometrio proliferativo, endometrio secretor e hiperplasia adenomatosa del endometrio. Se postula el factor hormonal estrogénico alto, como probable etiopatogenia de esta entidad, en mujeres postmenopaúsicas, multíparas. Al examen microscópico es

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Olmedo Bareiro, Ricardo, Aldo Martinez Lopez, Amanda Fretes Gómez, and Alejandro Caballero Rodrigues. "Familial Adenomatous Polyposis. Assisted videolaparoscopic proctocolectomy. Case report." Cirugía paraguaya 45, no. 1 (2021): 37–38. http://dx.doi.org/10.18004/sopaci.2021.abril.37.

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Ordóñez Daza, Dora Eugenia, and Juan José Herrera Escandón. "Tumor odontogénico adenomatoide en la región mandibular: reporte de un caso / Adenomatoid Odontogenic Tumor in Mandibular Region: Case Report." Universitas Odontologica 34, no. 72 (2015): 27. http://dx.doi.org/10.11144/javeriana.uo34-72.toar.

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Gui, Xianyong, Martin Köbel, Jose GP Ferraz, et al. "Histological and molecular diversity and heterogeneity of precancerous lesions associated with inflammatory bowel diseases." Journal of Clinical Pathology 73, no. 7 (2019): 391–402. http://dx.doi.org/10.1136/jclinpath-2019-206247.

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AimsInflammatory bowel disease (IBD)-associated precancerous lesions may be adenomatous or non-adenomatous with various histomorphologies. We aim to validate the newly proposed classification, to explore the neoplastic nature of the non-adenomatous lesions and to elucidate the molecular mechanisms underlying the different histomorphologies.Methods44 background precursor lesions identified in 53 cases of surgically resected IBD-associated colorectal and ileal carcinomas were reviewed for the histomorphological features (classified into adenomatous, mucinous, sessile serrated adenoma (SSA)-like,

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Bioulac-Sage, Paulette, Christine Sempoux, Laurent Possenti, et al. "Pathological Diagnosis of Hepatocellular Cellular Adenoma according to the Clinical Context." International Journal of Hepatology 2013 (2013): 1–13. http://dx.doi.org/10.1155/2013/253261.

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In Europe and North America, hepatocellular adenomas (HCA) occur, classically, in middle-aged woman taking oral contraceptives. Twenty percent of women, however, are not exposed to oral contraceptives; HCA can more rarely occur in men, children, and women over 65 years. HCA have been observed in many pathological conditions such as glycogenosis, familial adenomatous polyposis, MODY3, after male hormone administration, and in vascular diseases. Obesity is frequent particularly in inflammatory HCA. The background liver is often normal, but steatosis is a frequent finding particularly in inflamma

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Timonera, Elizabeth R., Maria Emilia Paiva, Jose Manuel Lopes, Catarina Eloy, Theodore van der Kwast, and Sylvia L. Asa. "Composite Adenomatoid Tumor and Myelolipoma of Adrenal Gland: Report of 2 Cases." Archives of Pathology & Laboratory Medicine 132, no. 2 (2008): 265–67. http://dx.doi.org/10.5858/2008-132-265-catamo.

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Abstract Adenomatoid tumor and myelolipoma are benign, hormonally inactive tumors that are often incidental findings in the adrenal glands. Myelolipoma is more common than adenomatoid tumor in this location but both are rare, and as yet, the pathogenesis of both remains unclear. We report 2 cases of composite adenomatoid tumor and myelolipoma, incidentally found in the adrenal gland on investigation for other diseases. To our knowledge, composite adenomatoid tumor and myelolipoma of adrenal gland has not been previously reported.

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Imai, Yukihiro, and Eugene J. Mark. "Cystic Adenomatoid Change Is Common to Various Forms of Cystic Lung Diseases of Children." Archives of Pathology & Laboratory Medicine 126, no. 8 (2002): 934–40. http://dx.doi.org/10.5858/2002-126-0934-cacict.

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Abstract Context.—Congenital cystic adenomatoid malformation, intralobar sequestration, extralobar sequestration, bronchopulmonary foregut malformation, bronchial atresia, and lobar emphysema generally are treated as separate entities, but there are commonalities among them. Objective.—To clarify the identity of congenital cystic adenomatoid malformation in relation to other cystic diseases. Design.—We studied 10 consecutive cases of surgically resected cystic lung disease in children. We studied the bronchial and vascular trees by mapping bronchi, pulmonary blood vessels, systemic blood vesse

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Acikalin, Mustafa Fuat, Huseyin Mete Tanir, Sinan Ozalp, Emine Dundar, Evrim Ciftci, and Emel Ozalp. "Diffuse Uterine Adenomatoid Tumor in a Patient With Chronic Hepatitis C Virus Infection." International Journal of Gynecologic Cancer 19, no. 2 (2009): 242–44. http://dx.doi.org/10.1111/igc.0b013e31819bc8f9.

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AbstractUterine adenomatoid tumors are usually solitary lesions. Adenomatoid tumors diffusely infiltrating the entire myometrium have rarely been reported in the literature. A feature common to half of the reported cases of diffuse uterine adenomatoid tumor was an immunocompromised status of the patient caused by the medications for renal transplantation. In this article, we describe an unusual case of diffuse uterine adenomatoid tumor in a patient with chronic hepatitis C virus infection. Pelvic examination showed an irregular and enlarged uterus. Supracervical hysterectomy and bilateral salp

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Rathinagopal, Jenny, Shiby Kuriakose, Shiv Bhanot, et al. "N-myristoyltransferase 2: A novel marker for colorectal cancer screening." Journal of Clinical Oncology 35, no. 15_suppl (2017): e15114-e15114. http://dx.doi.org/10.1200/jco.2017.35.15_suppl.e15114.

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e15114 Background: Colorectal cancer (CRC) is the second most common cause of cancer related deaths in North America. Most CRCs arise from pre-malignant adenomatous polyps with some of these polyps developing into cancer over years providing an ideal opportunity for detection and intervention. However, the unpleasant nature of the current screening methods often leads to non-compliance. N-Myristoyltransferase (NMT) has been reported to be overexpressed in CRC tissues. In this study we show that NMT2, an isoform of NMT, is overexpressed in peripheral blood mononuclear cells (PBMC) of individual

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ÖZDEMİR, Özmert M. A., Hacer ERGİN, Abdullah KARACA, Uğur KOLTUKSUZ, Nevzat KARABULUT, and Nagihan YALÇIN. "Congenital Cystic Adenomatoid Malformation: Case Report." Turkiye Klinikleri Journal of Medical Sciences 30, no. 2 (2010): 798–802. http://dx.doi.org/10.5336/medsci.2008-7564.

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