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Journal articles on the topic "ADH1C"

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Ayuso, Pedro, Elena García-Martín, José A. Cornejo-García, José A. G. Agúndez, and José María Ladero. "Genetic Variants of Alcohol Metabolizing Enzymes and Alcohol-Related Liver Cirrhosis Risk." Journal of Personalized Medicine 11, no. 5 (2021): 409. http://dx.doi.org/10.3390/jpm11050409.

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Alcohol-related liver disease (ARLD) is a major public health issue caused by excessive alcohol consumption. ARLD encompasses a wide range of chronic liver lesions, alcohol-related liver cirrhosis being the most severe and harmful state. Variations in the genes encoding the enzymes, which play an active role in ethanol metabolism, might influence alcohol exposure and hence be considered as risk factors of developing cirrhosis. We conducted a case-control study in which 164 alcohol-related liver cirrhosis patients and 272 healthy controls were genotyped for the following functional single nucleotide variations (SNVs): ADH1B gene, rs1229984, rs1041969, rs6413413, and rs2066702; ADH1C gene, rs35385902, rs283413, rs34195308, rs1693482, and rs35719513; CYP2E1 gene, rs3813867. Furthermore, copy number variations (CNVs) for ADH1A, ADH1B, ADH1C, and CYP2E1 genes were analyzed. A significant protective association with the risk of developing alcohol-related liver cirrhosis was observed between the mutant alleles of SNVs ADH1B rs1229984 (Pc value = 0.037) and ADH1C rs283413 (Pc value = 0.037). We identified CNVs in all genes studied, ADH1A gene deletions being more common in alcohol-related liver cirrhosis patients than in control subjects, although the association lost statistical significance after multivariate analyses. Our findings support that susceptibility to alcohol-related liver cirrhosis is related to variations in alcohol metabolism genes.
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Tóth, Réka, Szilvia Fiatal, Beáta Petrovski, Martin McKee, and Róza Ádány. "Combined Effect of ADH1B RS1229984, RS2066702 and ADH1C RS1693482/ RS698 Alleles on Alcoholism and Chronic Liver Diseases." Disease Markers 31, no. 5 (2011): 267–77. http://dx.doi.org/10.1155/2011/350528.

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The aim of this study was to analyze the combined effect of the most frequent alcohol dehydrogenase polymorphisms (Arg48His and Arg370Cys in ADH1B, Arg272Gln and Ile350Val in ADH1C) on the alcohol use habits, alcohol dependence and chronic liver diseases in Hungary.The study included men, aged 45–64 years. Altogether, 241 cases with chronic liver disease (CLD) and 666 randomly selected controls without CLD were analysed for all four polymorphisms. Associations between the polymorphisms, individually, and in combination, and excessive and problem drinking and CLD, were assessed using logistic regression.In this study we have identified a novel mutation, called ADH1B Arg370His. The ADH1C Arg272Gln and Ile350Val showed almost complete linkage. The 272Gln/350Val allele increased the risk of excessive and problem drinking in homozygous form (OR = 1.582,p= 0.035, CI = 1.034–2.421, OR = 1.780,p= 0.016, CI = 1.113–2.848, respectively). The joint analysis showed that when combined with the wild type ADH1C Arg272/Ile350 allele, the ADH1B 48His is protective against CLD (OR = 0.368,p= 0.019, CI = 0.159–0.851).The results obtained in the study help not only to clarify the effects of different ADH SNPs but to better understand how these polymorphisms modify each other’s effects in the development of alcoholism and related diseases.
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Cleveland, H. Harrington, Gabriel L. Schlomer, David J. Vandenbergh, et al. "Associations between alcohol dehydrogenase genes and alcohol use across early and middle adolescence: Moderation × Preventive intervention." Development and Psychopathology 30, no. 1 (2017): 297–313. http://dx.doi.org/10.1017/s0954579417000633.

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AbstractData from the in-school sample of the PROSPER preventive intervention dissemination trial were used to investigate associations between alcohol dehydrogenase genes and alcohol use across adolescence, and whether substance misuse interventions in the 6th and 7th grades (targeting parenting, family functioning, social norms, youth decision making, and peer group affiliations) modified associations between these genes and adolescent use. Primary analyses were run on a sample of 1,885 individuals and included three steps. First, we estimated unconditional growth curve models with separate slopes for alcohol use from 6th to 9th grade and from 9th to 12th grade, as well as the intercept at Grade 9. Second, we used intervention condition and three alcohol dehydrogenase genes, 1B (ADH1B), 1C (ADH1C), and 4 (ADH4) to predict variance in slopes and intercept. Third, we examined whether genetic influences on model slopes and intercepts were moderated by intervention condition. The results indicated that the increase in alcohol use was greater in early adolescence than in middle adolescence; two of the genes, ADH1B and ADH1C, significantly predicted early adolescent slope and Grade 9 intercept, and associations between ADH1C and both early adolescent slope and intercept were significantly different across control and intervention conditions.
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Mohelnikova-Duchonova, Beatrice, David Vrana, Ivana Holcatova, Miroslav Ryska, Zdenek Smerhovsky, and Pavel Soucek. "CYP2A13, ADH1B, and ADH1C Gene Polymorphisms and Pancreatic Cancer Risk." Pancreas 39, no. 2 (2010): 144–48. http://dx.doi.org/10.1097/mpa.0b013e3181bab6c2.

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Ji, Yong Bae, Seung Hwan Lee, Kyung Rae Kim, et al. "Association between ADH1B and ADH1C polymorphisms and the risk of head and neck squamous cell carcinoma." Tumor Biology 36, no. 6 (2015): 4387–96. http://dx.doi.org/10.1007/s13277-015-3078-y.

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Rebello, André Soares, and Maria da Glória Da Costa Carvalho. "Metodologia para estudo do polimorfismo do gene da enzima álcool desidrogenase." Revista de Ciências Médicas e Biológicas 7, no. 2 (2008): 163. http://dx.doi.org/10.9771/cmbio.v7i2.4445.

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As principais enzimas responsáveis pelo metabolismo do álcool são a álcool desidrogenase (ADH) e aldeído desidrogenase (ALDH). EsSas enzimas estão presentes em várias formas e são codificadas por diferentes genes. Alguns desses genes apresentam polimorfismos, e as enzimas codificadas por eles podem apresentar diferenças quanto à eficiência metabólica em relação ao álcool e ao aldeído acético. EsSa variação tem se mostrado um fator que influencia a quantidade de álcool ingerido e o risco no aumento de abuso e dependência ao álcool. Neste trabalho, nós descrevemos um método que permite estudar o polimorfismo de um dos genes da enzima álcool desidrogenase, o gene ADH1C. O DNA foi isolado de doadores e o polimorfismo foi determinado pela reação em cadeia pela polimerase (PCR). Nossos resultados confirmam a viabilidade da técnica por nós descrita para o estudo do polimorfismo do gene ADH1C.
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Dannenberg, Luke O., Hui-Ju Chen, Huijun Tian, and Howard J. Edenberg. "Differential Regulation of the Alcohol Dehydrogenase 1B (ADH1B) and ADH1C Genes by DNA Methylation and Histone Deacetylation." Alcoholism: Clinical and Experimental Research 30, no. 6 (2006): 928–37. http://dx.doi.org/10.1111/j.1530-0277.2006.00107.x.

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Wang, Jiwen, Jinyu Wei, Xiaoling Xu, et al. "Replication Study of ESCC Susceptibility Genetic Polymorphisms Locating in the ADH1B-ADH1C-ADH7 Cluster Identified by GWAS." PLoS ONE 9, no. 4 (2014): e94096. http://dx.doi.org/10.1371/journal.pone.0094096.

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Mulligan, Connie J., Robert W. Robin, Michael V. Osier, et al. "Allelic variation at alcohol metabolism genes ( ADH1B , ADH1C , ALDH2 ) and alcohol dependence in an American Indian population." Human Genetics 113, no. 4 (2003): 325–36. http://dx.doi.org/10.1007/s00439-003-0971-z.

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van Beek, Jenny H. D. A., Gonneke Willemsen, Marleen H. M. de Moor, Jouke Jan Hottenga, and Dorret I. Boomsma. "Associations Between ADH Gene Variants and Alcohol Phenotypes in Dutch Adults." Twin Research and Human Genetics 13, no. 1 (2010): 30–42. http://dx.doi.org/10.1375/twin.13.1.30.

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AbstractRecently, Macgregor et al. (2009) demonstrated significant associations of ADH polymorphisms with reactions to alcohol and alcohol consumption measures in an Australian sample. The aim of the present study was to replicate these findings in a Dutch sample. Survey data on alcohol phenotypes came from 1,754 unrelated individuals registered with the Netherlands Twin Register. SNPs in the ADH gene cluster located on chromosome 4q (n= 491) were subdivided in seven gene sets: ADH5, ADH4, ADH6, ADH1A, ADH1B, ADH1C and ADH7. Within these sets associations of SNPs with alcohol consumption measures, age at onset variables, reactions to alcohol and problem drinking liability were examined. Of the original 38 SNPs studied by Macgregor et al. (2009), six SNPs were not available in our dataset, because one of them had a minor allele frequency < .01 (rs1229984) and five could not be imputed. The remaining SNP associations with alcohol phenotypes as identified by Macgregor et al. (2009) were not replicated in the Dutch sample, after correcting for multiple genotype and phenotype testing. Significant associations were found however, for reactions to alcohol with a SNP in ADH5 (rs6827292,p= .001) and a SNP just upstream of ADH5 (rs6819724,p= .0007) that is in strong LD with rs6827292. Furthermore, an association between age at onset of regular alcohol use and a SNP just upstream of ADH7 (rs2654849,p= .003) was observed. No significant associations were found for alcohol consumption and problem drinking liability. Although these findings do not replicate the earlier findings at the SNP level, the results confirm the role of the ADH gene cluster in alcohol phenotypes.
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Dissertations / Theses on the topic "ADH1C"

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Wolnitzky, Wenk Javier Humberto. "Polimorfismos en el Gen de la deshidrogenasa alcohólica 1C (ADH1C) aumentan el riesgo de alcoholismo en la población chilena." Tesis, Universidad de Chile, 2008. http://www.repositorio.uchile.cl/handle/2250/105263.

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Memoria para optar al título de Químico Farmacéutico<br>El riesgo individual de alcoholismo tiene un componente hereditario de 40 a 60%. Los únicos factores genéticos bien establecidos para una susceptibilidad diferenciada al alcoholismo son polimorfismos en los genes de las principales enzimas del metabolismo del etanol, la deshidrogenasa alcohólica (ADH) y la deshidrogenasa aldehídica mitocondrial (ALDH2). Las variantes polimórficas de estas enzimas difieren en sus propiedades catalíticas y en sus distribuciones étnicas. El etanol es metabolizado en el hígado a acetaldehído por la ADH y luego a acetato por la ALDH2. En portadores de una ALDH2 inactiva (ALDH2*2) el acetaldehído, en lugar de degradarse, se acumula en el cuerpo produciendo reacciones desagradables que evitan una ingesta excesiva de alcohol. Se postula que los portadores de las variantes rápidas de la ADH (ADH1C1, ADH1B2) también experimentan un alza de acetaldehído corporal luego de consumir etanol, teniendo así una protección genética contra el alcoholismo. En contraste, los portadores de las ADHs lentas (ADH1C2 o ADH1B1) o de la ALDH2 de gran actividad (ALDH2*1) tienen un mayor riesgo de alcoholismo. La variante inactiva de la ALDH2 (ALDH2*2) es exclusiva de la población del este de Asia (40%), que tiene además frecuencias altas de los alelos protectores ADH1C*1 (90%) y ADH1B*2 (70%), mientras que las poblaciones caucásicas tienen frecuencias menores de estos alelos (50% y 5%, respectivamente). Para determinar si los alelos ADH1C*2 o ADH1B*1 de la deshidrogenasa alcohólica (ADHs lentas) aumentan el riesgo de alcoholismo en la población chilena se determinaron los genotipos de la ADH1C y la ADH1B en pacientes alcohólicos (n = 30) y población chilena general (n = 105). Se amplificaron segmentos específicos de estos genes utilizando la reacción en cadena de la polimerasa (PCR) y se analizaron las variaciones codificantes de cambios aminoacídicos mediante digestión con enzimas de restricción, separando los fragmentos resultantes por tamaño mediante electroforesis (RFLP). Se evaluó la significación estadística de las diferencias encontradas entre los grupos en las frecuencias alélicas y genotípicas de los genes ADH1C y ADH1B. La frecuencia del alelo ADH1C*2 es significativamente mayor (p < 0,04) en pacientes alcohólicos (0,40) que en la población general (0,26), aumentando casi al doble el riesgo de alcoholismo (OR = 1,96) respecto del alelo ADH1C*1. La frecuencia genotípica de los homocigotos ADH1C*2/*2 es también mayor (p < 0,05) en pacientes alcohólicos (0,14) que en la población general (0,07), teniendo estos individuos un riesgo de alcoholismo casi cuatro veces mayor (OR = 3,85) que los homocigotos ADH1C*1/*1. Las frecuencias del alelo ADH1B*1 son prácticamente idénticas en ambos grupos (alrededor de 0,95), haciendo imposible determinar si éste aumenta el riesgo de alcoholismo en la población chilena, dado el tamaño muestral empleado. Se analizaron cinco variaciones nucleotídicas adicionales, lo que permitió encontrar en la población chilena una variante del alelo ADH1C*2 exclusiva de la población nativa de América (ADH1C*2Thr351) y describir por primera vez la existencia de dos variantes nuevas del alelo ADH1B*1. La variante ADH1B*1Ser59 incorpora un polimorfismo (A5226T) antes reportado aisladamente y la variante ADH1B*1Arg367 incorpora una mutación encontrada aquí (T15490G). No se encontró evidencia de que estas variantes modifiquen el riesgo de alcoholismo en la población chilena, pero sí se encontró una tendencia hacia un riesgo aumentado de alcoholismo para el alelo ADH1C*2Thr351, al compararlo con los alelos ADH1C*1 y ADH1C*2 en conjunto. A partir de los resultados de esta memoria se pueden emitir tres conclusiones. (I) El alelo ADH1C*2 y el genotipo ADH1C*2/*2 aumentan el riesgo de alcoholismo en la población chilena respecto del alelo ADH1C*1 y el genotipo ADH1C*1/*1. (II) El genotipo de la ADH1C se puede determinar analizando sólo la posición nucleotídica 11939 (aa 271) y no la posición 15115 (aa 349), ya que sus identidades están perfectamente asociadas (11939A / 15115G y 11939G / 15115A) y porque el análisis de la posición nucleotídica 11939 tiene una mayor fortaleza en su diseño experimental por a) tener reacciones positivas para las dos identidades nucleotídicas (A y G) de esta posición y b) determinar un cambio aminoacídico de mayor importancia por encontrarse en el sitio activo de la enzima. (III) El genotipo de la ADH1B puede ser excluido del análisis del aumento del riesgo de alcoholismo conferido por las variantes lentas de la ADH en la población chilena porque su diferencia entre grupos es mínima.
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Kumar, Kavitha. "Intrusion Detection in Mobile Adhoc Networks." Connect to full text in OhioLINK ETD Center, 2009. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=toledo1260232844.

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Thesis (M.S.)--University of Toledo, 2009.<br>Typescript. "Submitted to the Graduate Faculty as partial fulfillment of the requirements for the Master of Science Degree in Engineering." "A thesis entitled"--at head of title. Bibliography: leaves 80-84.
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Jia, Sijun. "An effective solution for bluetooth adhoc networking." Thesis, University of British Columbia, 2009. http://hdl.handle.net/2429/7238.

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Bluetooth operates in the unlicensed ISM frequencies with a spread spectrum and frequency hopping, and it has the features such as robustness, low power consumption and low cost. Therefore, Bluetooth has been supported on the most number of devices of various types. Bluetooth connection is defined as in Piconet which has limits on range and the number of devices, so many researches have been done to connect more devices across longer ranges with Bluetooth, but there has not been an effective solution so far due to the protocol limitation, device versatility and mobility. In this thesis, we designed and implemented such a solution based on Piconet topology, existing Internet and adhoc networking protocols. In our solution, networking is performed on a high and abstract layer, so devices with different hardware or operating systems can join the network by installing a program without low level system change. We adjusted and implemented the standard Adhoc On-demand Distance Vector (AODV) protocol for IEEE 802.11 in our system to support route discovery and maintenance in Bluetooth network. We also used techniques such as pre-warm and redundant routes to improve the performance and robustness of the network. Our system support scalable peer to peer networking without any centralized controls. We tested our solution on real devices and on device emulators in large scale, and the result showed the system can form Bluetooth network efficiently in a scalable way.
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Larafa, Claire Sondès. "Services AAA dans les réseaux adhoc mobiles." Phd thesis, Institut National des Télécommunications, 2011. http://tel.archives-ouvertes.fr/tel-00698490.

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La mobilité est une composante importante de la liberté des personnes. L'évolution des moyens technologiques y contribue au premier chef. Outre la question du transport, celle du maintien du lien entre les individus est en ce sens particulièrement prégnante. Elle a mis à rude épreuve la notion de réseaux de télécommunications puisqu'il s'agit de répondre, pour des individus éparpillés ou concentrés, mais mobiles, au besoin de rester reliés. De l'ère des réseaux analogiques à celle des réseaux numériques, de l'ère des réseaux filaires à celle des réseaux sans fil et mobiles, la technologie n'a cessé d'évoluer. Ces dernières décennies ont vu apparaître des réseaux numériques sans fil, où non seulement il y a mobilité des utilisateurs mais aussi mobilité de l'infrastructure du réseau à laquelle ils contribuent. Ces réseaux se constituent de façon spontanée. Ils se maintiennent de manière autonome. On les désigne par le terme réseaux ad hoc mobiles (en anglais Mobile Ad hoc Networks ou MANET) qui s'oppose naturellement à celui de réseaux à infrastructure. La sécurité est une préoccupation générale des êtres humains. Ils en ressentent aussi le besoin en matière de réseaux. Ce besoin est particulièrement criant lorsque sont échangées des données critiques, financières ou stratégiques. La confidentialité des échanges, l'authentification des sources, l'assurance d'intégrité, la prévention de la récusation sont autant d'objectifs qu'il faut alors atteindre. Diverses solutions de sécurité ont été conçues dans cette optique pour les réseaux filaires puis ont ensuite été adaptées aux réseaux sans-fil et mobiles. Les architectures AAA (Authentication, Authorization, Accounting) en font partie. Elles sont en général utilisées dans un contexte commercial. Tant par leur facilité de déploiement que par la baisse des coûts de mise en œuvre qu'ils engendrent, les réseaux ad hoc mobiles, après avoir bien servi dans le domaine militaire, semblent avoir un avenir dans les applications commerciales. C'est pourquoi, nous nous proposons dans cette thèse de concevoir une architecture AAA adaptée aux spécificités de ces réseaux. Nous étudions d'abord les réseaux ad hoc mobiles et leurs caractéristiques. Ensuite, nous présentons les solutions de sécurité qui existent dans les réseaux à infrastructure. Nous examinons, en particulier, les solutions qui permettent le contrôle d'accès et dont sont engendrées les architectures AAA. Les solutions AAA proposées pour les MANETs sont par la suite analysées et classifiées afin de déterminer les manques et les vulnérabilités. Cette étude approfondie nous amène à proposer une architecture AAA répondant aux attentes identifiées. C'est une architecture distribuée qui répond, en particulier, au besoin d'autonomie des opérations dans les MANETs et où les protocoles exécutés peuvent impliquer simultanément plus de deux parties. Un ensemble de protocoles et de mécanismes d'authentification et d'autorisation s'intégrant avec la suite des protocoles IPv6 a été proposé. Leur sécurité a été discutée. Celle, en particulier du protocole d'authentification a fait l'objet de validation formelle. Contrairement aux protocoles utilisés dans la phase d'autorisation des services AAA proposés, le mode de communication multi-parties et multi-sauts du protocole d'authentification nous a poussé à mener une analyse approfondie de ses performances. Pour cela, nous avons eu recours, dans un premier temps, à la modélisation au moyen de calculs mathématiques explicites ensuite à la simulation. Les résultats obtenus montrent que ce protocole passe à l'échelle d'un MANET comprenant au moins cent nœuds. Dans certaines conditions d'implémentation que nous avons définies, ses performances, tant celle liée à sa probabilité de terminaison avec une issue favorable que celle portant sur son temps d'exécution, atteignent des valeurs optimales.
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Bulut, Gulsah. "Service Discovery Oriented Clustering For Mobile And Adhoc Networks." Master's thesis, METU, 2010. http://etd.lib.metu.edu.tr/upload/2/12611889/index.pdf.

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Adhoc networks do not depend on any fixed infrastructure. The most outstanding features of adhoc networks are non-centralized structure and dynamic topology change due to high mobility. Since mentioned dynamics of mobile adhoc networks complicate reaching the resources in the network, service discovery is significantly an important part of constructing stand-alone and self-configurable mobile adhoc networks. The heterogeneity of the devices and limited resources such as battery are also load up more difficulty to service discovery. Due to the volatile nature of the adhoc networks, service discovery algorithms proposed for mobile and adhoc networks suffer from some problems. Scalability becomes a problem when the service discovery is based on flooding messages over the network. Furthermore, the high traffic which occurs due to the message exchange between network nodes makes the communication almost impossible. Partitioning a network into sub-networks is an efficient way of handling scalability problem. In this thesis, a mobility based service discovery algorithm for clustered MANET is presented. The algorithm has two main parts. First one is for partitioning the MANET into sub-networks, named &ldquo<br>clustering&rdquo<br>. Second part is composed of an efficient discovery of services on overall network. Clustering algorithm used in this study is enhanced version of DMAC (Distributed Mobility Adaptive Clustering, which is one of the golden algorithms of the wireless network clustering area). To be fast and flexible in service discovery layer, a simple and fastresponding algorithm is implemented. Integration of two algorithms enables devices to be mobile in the network
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Yarlagadda, Jyothsna. "Energy-efficient even-parity network coding for adhoc networks." Thesis, Wichita State University, 2012. http://hdl.handle.net/10057/5982.

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The major difference between wired and wireless networks is their broadcast nature, specifically how transmitted data by one node may reach other nodes and vice-versa. This broadcast nature is a curse for wired networks but a blessing for wireless networks. Network coding is a technique where instead of just forwarding packets arrived at relay nodes, the node will collect several packets and then combine them together using an algebraic algorithm for transmissions. Network coding reduces the energy consumption by decreasing the number of transmissions required to communicate a given amount of information across the network. The aim of this thesis is to enhance network-coding strategy in order to improve energy gain, which in turn increases throughput in ad hoc networks. To that end, this thesis also proposes an even parity scheme that reduces processing time and power of nodes by improving coding opportunities. In addition, an even parity scheme allows for the coding of large numbers of packets at a time instead of coding just two packets using normal XOR operation.<br>Thesis (M.S.)--Wichita State University, College of Engineering, Dept. of Electrical Engineering and Computer Science
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Pore, Ghee Lye. "A performance analysis of routing protocols for adhoc networks." Thesis, Monterey, Calif. : Springfield, Va. : Naval Postgraduate School ; Available from National Technical Information Service, 2006. http://library.nps.navy.mil/uhtbin/hyperion/06Mar%5FPore.pdf.

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Thesis (M.S. in Electrical Engineering)--Naval Postgraduate School, March 2006.<br>Thesis Advisor(s): John C. McEachen. "March 2006." Includes bibliographical references (p. 85-89). Also available online.
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Cho, Jinyoun. "Efficient safety message dissemination methods in vehicular adhoc networks." Diss., Georgia Institute of Technology, 2015. http://hdl.handle.net/1853/53390.

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The methods for efficient safety message dissemination in VANETs were proposed. First, the method for using multi-channel was proposed. Using the proposed multi-channel method (divide-and-deliver algorithm), the safety message was delivered to the target device with less delay compared to the traditional single-channel method. This method showed resilient performance even in poor wireless channels compared to the single-channel method. Second, to improve low reliability in low vehicle density situations, the enhanced divide-and-deliver algorithm was proposed. The network coding was a key technique to the enhancement. For the efficient use of network coding, rigorous analysis was conducted and an algorithm was proposed to change the number of network coding packets adaptively by the vehicle densities. Finally, the method for delivering safety messages to multi-direction was proposed. This multi-vehicle selection broadcast (MSB) algorithm avoided the collision between multiple rebroadcasts among vehicles and removed unnecessary packets by using backoff slots. The contributions of this research include reducing delay and increasing reliability for the dissemination of safety messages.
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Tezeren, Serdar U. "Reed-Muller codes in error correction in wireless adhoc networks." Thesis, Monterey, Calif. : Springfield, Va. : Naval Postgraduate School ; Available from National Technical Information Service, 2004. http://library.nps.navy.mil/uhtbin/hyperion/04Mar%5FTezeren.pdf.

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Thesis (M.S. in Electrical Engineering)--Naval Postgraduate School, March 2004.<br>Thesis advisor(s): Murali Tummala, Roberto Cristi. Includes bibliographical references (p. 133-134). Also available online.
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Nguyen-Salamanis, Khanh-Loan. "Adhoc-Verwendung einer elektronischen Tafel unter Verwendung der Jini-Technologie." [S.l. : s.n.], 2000. http://www.bsz-bw.de/cgi-bin/xvms.cgi?SWB8862165.

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Books on the topic "ADH1C"

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Adhuc tempus: Aún hay tiempo : novela. [s.n.], 2008.

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Vasanthi, T. Optimum Reliability Analysis of Mobile Adhoc Networks using Universal Generating Function under Limited Delivery Time and Cost. Edited by Kokula Krishna Hari K and K. Saravanan. Association of Scientists, Developers and Faculties, 2016.

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Penfield. Adhoc Short Takes. 5th ed. Not Avail, 1998.

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Goshgarian. Adhoc Goshgarian Adhoc Reader 5e, and Aaron the Little Brown Essential. 5th ed. Not Avail, 1998.

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Kishlansky. Adhoc Civilization in West CB + Discover West CIV Thru Maps Adhoc Bundle. Not Avail, 1997.

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Kishlansky. Adhoc Sources of World History. Not Avail, 1998.

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Waterman, Robert. La stratégie des équipes adhoc. Maxima, 2000.

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Stamper. Adhoc Essentials of Dats Communications. Not Avail, 1998.

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Randall. Adhoc American Lives Volume I. Not Avail, 1998.

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Lial. Adhoc Intermediate Algebra W/Early Graphs and Functions and Overcoming Math Anxiety Adhoc Bundle. Not Avail, 1997.

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Book chapters on the topic "ADH1C"

1

Wolmeringer, Gottfried. "AdHoc zum ersten Ergebnis." In Profikurs Eclipse 3. Vieweg+Teubner Verlag, 2004. http://dx.doi.org/10.1007/978-3-322-96873-9_3.

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Bharati, Sailesh, and Weihua Zhuang. "Cooperation in ADHOC MAC." In Wireless Networks. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-58721-9_4.

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Bradshaw, John L. "Terra Australis (nobis adhuc) incognita." In Reflections of a Neuropsychologist. Routledge, 2018. http://dx.doi.org/10.4324/9781351060752-15.

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Marcelín-Jiménez, Ricardo. "Locally-Constructed Trees for Adhoc Routing." In Personal Wireless Communications. Springer Berlin Heidelberg, 2006. http://dx.doi.org/10.1007/11872153_17.

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Ghassemi, Fatemeh, and Ali Movaghar. "Modeling Routing Protocols in Adhoc Networks." In Communications in Computer and Information Science. Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-89985-3_52.

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Bruhadeshwar, Bezawada, and Sandeep S. Kulkarni. "User Revocation in Secure Adhoc Networks." In Distributed Computing and Internet Technology. Springer Berlin Heidelberg, 2005. http://dx.doi.org/10.1007/11604655_43.

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Cheng, Chi Kan, Jimmy Ho Man Lee, and Peter J. Stuckey. "Box Constraint Collections for Adhoc Constraints." In Principles and Practice of Constraint Programming – CP 2003. Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-540-45193-8_15.

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Kumari, Parveen, Gaurav Aggarwal, and Sugandha Singh. "Clustering in Mobile Adhoc Network: WCA Algorithm." In International Conference on Intelligent Data Communication Technologies and Internet of Things (ICICI) 2018. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-030-03146-6_58.

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Vidhate, Deepak, Anita Patil, and Supriya Sarkar. "Bandwidth Estimation Scheme for Mobile Adhoc Network." In Communications in Computer and Information Science. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-12214-9_23.

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Vijayan, R., and N. Jeyanthi. "Trust Management Approaches in Mobile Adhoc Networks." In Studies in Big Data. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-030-01566-4_4.

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Conference papers on the topic "ADH1C"

1

Huang, Shiang-Fu, Huei-Tzu Chien, Sou-De Cheng, Chun-Ta Liao, and Hung-Ming Wang. "Abstract 3255: The roles of ALDH2, ADH1B and ADH1C gene polymorphism in predicting upper digestive tract multiple primary malignancies in head and neck cancer patients." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-3255.

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Gajjar, Sachin, and Hari M. Gupta. "Improving performance of adhoc TCP in Mobile Adhoc Networks." In 2008 Annual IEEE India Conference (INDICON). IEEE, 2008. http://dx.doi.org/10.1109/indcon.2008.4768816.

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Bukkawar, S., and N. S. Killarikar. "Hybrid cellular adhoc network." In the International Conference & Workshop. ACM Press, 2011. http://dx.doi.org/10.1145/1980022.1980200.

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Wu, Wei, Lusheng Yan, Benxiong Huang, Yijun Mo, and Hai Hu. "Modeling of Heterogeneous Multidimensional ADHOC." In 2010 6th International Conference on Wireless Communications, Networking and Mobile Computing (WiCOM). IEEE, 2010. http://dx.doi.org/10.1109/wicom.2010.5601184.

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Manjuladevi, V., and R. Jennie Bharathi. "Efficient anonymous geographic adhoc routing." In 2014 International Conference on Electronics and Communication Systems (ICECS). IEEE, 2014. http://dx.doi.org/10.1109/ecs.2014.6892816.

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Mishra, Bharati, Priyadarshini Nayak, Subhashree Behera, and Debasish Jena. "Security in vehicular adhoc networks." In the 2011 International Conference. ACM Press, 2011. http://dx.doi.org/10.1145/1947940.1948063.

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Shamsi, Jawwad, S. Ali Raza, Hassan Farid, Taha Munir, and Abbas Mehdi. "MACNET: Mobile Adhoc Collaborative NETworks." In 10th IEEE International Conference on Collaborative Computing: Networking, Applications and Worksharing. ICST, 2014. http://dx.doi.org/10.4108/icst.collaboratecom.2014.257322.

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Mangrulkar, R. S., and Mohammad Atique. "Trust based secured adhoc On demand Distance Vector Routing protocol for mobile adhoc network." In 2010 Sixth International Conference on Wireless Communication and Sensor Networks (WCSN). IEEE, 2010. http://dx.doi.org/10.1109/wcsn.2010.5712310.

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Belbachir, Redouane, Mekkakia Maaza Zoulikha, Ali Kies, and Bernard Cousin. "Bandwidth reservation in mobile adhoc networks." In 2012 IEEE Wireless Communications and Networking Conference (WCNC). IEEE, 2012. http://dx.doi.org/10.1109/wcnc.2012.6214240.

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Suri, Pushpa, and Sona Rani. "Bluetooth network-the adhoc network concept." In Proceedings 2007 IEEE SoutheastCon. IEEE, 2007. http://dx.doi.org/10.1109/secon.2007.342994.

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Reports on the topic "ADH1C"

1

Freeling, M. A genetic analysis of Adh1 regulation. Office of Scientific and Technical Information (OSTI), 1992. http://dx.doi.org/10.2172/5821489.

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Freeling, M. A genetic analysis of Adh1 regulation. Progress report, June 1991--February 1992. Office of Scientific and Technical Information (OSTI), 1992. http://dx.doi.org/10.2172/10124127.

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