Relevant bibliographies by topics / Adhesion Complex

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Adhesion Complex'

Author: Grafiati
Published: 4 June 2021
Last updated: 7 February 2022

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Journal articles on the topic "Adhesion Complex"

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Young, Katherine A., Laura Biggins, and Hayley J. Sharpe. "Protein tyrosine phosphatases in cell adhesion." Biochemical Journal 478, no. 5 (2021): 1061–83. http://dx.doi.org/10.1042/bcj20200511.

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Adhesive structures between cells and with the surrounding matrix are essential for the development of multicellular organisms. In addition to providing mechanical integrity, they are key signalling centres providing feedback on the extracellular environment to the cell interior, and vice versa. During development, mitosis and repair, cell adhesions must undergo extensive remodelling. Post-translational modifications of proteins within these complexes serve as switches for activity. Tyrosine phosphorylation is an important modification in cell adhesion that is dynamically regulated by the prot
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Sebé-Pedrós, Arnau, and Iñaki Ruiz-Trillo. "Integrin-mediated adhesion complex." Communicative & Integrative Biology 3, no. 5 (2010): 475–77. http://dx.doi.org/10.4161/cib.3.5.12603.

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Mistry, Shiv A., Avi M. Desai, Peidong He, et al. "In vitro investigation of chitosan-polygalacturonic acid polyelectrolyte complex (PEC) biomaterials as anti-adhesive substrates for preventing adhesion formation." TECHNOLOGY 06, no. 03n04 (2018): 110–17. http://dx.doi.org/10.1142/s2339547818500073.

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Adhesions are an abnormal union of membranous surfaces. They are a painful and expensive consequence of abdominal surgeries, specifically in the peritoneal cavity. This complication requires a second surgery to remove the problem, known as adhesiolysis, which we are trying to avoid. Current solutions to adhesion formation either lack efficacy or induce an inflammatory response in the peritoneum. Our focus is to develop a post-surgical adhesion prevention polyelectrolyte complex (PEC) film, composed of an optimal ratio of chitosan (Chi) and polygalacturonic acid (PgA) to prevent adhesion format
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Bach, Cuc T. T., Sarah Creed, Jessie Zhong, et al. "Tropomyosin Isoform Expression Regulates the Transition of Adhesions To Determine Cell Speed and Direction." Molecular and Cellular Biology 29, no. 6 (2009): 1506–14. http://dx.doi.org/10.1128/mcb.00857-08.

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ABSTRACT The balance of transition between distinct adhesion types contributes to the regulation of mesenchymal cell migration, and the characteristic association of adhesions with actin filaments led us to question the role of actin filament-associating proteins in the transition between adhesive states. Tropomyosin isoform association with actin filaments imparts distinct filament structures, and we have thus investigated the role for tropomyosins in determining the formation of distinct adhesion structures. Using combinations of overexpression, knockdown, and knockout approaches, we establi
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Katrín Hákonardóttir, Guðlaug, Pablo López-Ceballos, Alejandra Donají Herrera-Reyes, Raibatak Das, Daniel Coombs, and Guy Tanentzapf. "In vivo quantitative analysis of Talin turnover in response to force." Molecular Biology of the Cell 26, no. 22 (2015): 4149–62. http://dx.doi.org/10.1091/mbc.e15-05-0304.

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Cell adhesion to the extracellular matrix (ECM) allows cells to form and maintain three-dimensional tissue architecture. Cell–ECM adhesions are stabilized upon exposure to mechanical force. In this study, we used quantitative imaging and mathematical modeling to gain mechanistic insight into how integrin-based adhesions respond to increased and decreased mechanical forces. A critical means of regulating integrin-based adhesion is provided by modulating the turnover of integrin and its adhesion complex (integrin adhesion complex [IAC]). The turnover of the IAC component Talin, a known mechanose
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Lotz, M. M., C. A. Burdsal, H. P. Erickson, and D. R. McClay. "Cell adhesion to fibronectin and tenascin: quantitative measurements of initial binding and subsequent strengthening response." Journal of Cell Biology 109, no. 4 (1989): 1795–805. http://dx.doi.org/10.1083/jcb.109.4.1795.

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Cell-substratum adhesion strengths have been quantified using fibroblasts and glioma cells binding to two extracellular matrix proteins, fibronectin and tenascin. A centrifugal force-based adhesion assay was used for the adhesive strength measurements, and the corresponding morphology of the adhesions was visualized by interference reflection microscopy. The initial adhesions as measured at 4 degrees C were on the order of 10(-5)dynes/cell and did not involve the cytoskeleton. Adhesion to fibronectin after 15 min at 37 degrees C were more than an order of magnitude stronger; the strengthening
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Greenwood, Jeffrey A., Anne B. Theibert, Glenn D. Prestwich, and Joanne E. Murphy-Ullrich. "Restructuring of Focal Adhesion Plaques by Pi 3-Kinase." Journal of Cell Biology 150, no. 3 (2000): 627–42. http://dx.doi.org/10.1083/jcb.150.3.627.

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Focal adhesions are an elaborate network of interconnecting proteins linking actin stress fibers to the extracellular matrix substrate. Modulation of the focal adhesion plaque provides a mechanism for the regulation of cellular adhesive strength. Using interference reflection microscopy, we found that activation of phosphoinositide 3-kinase (PI 3-kinase) by PDGF induces the dissipation of focal adhesions. Loss of this close apposition between the cell membrane and the extracellular matrix coincided with a redistribution of α-actinin and vinculin from the focal adhesion complex to the Triton X-
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Israeli, Sharon, Kurt Amsler, Nadezhda Zheleznova, and Patricia D. Wilson. "Abnormalities in focal adhesion complex formation, regulation, and function in human autosomal recessive polycystic kidney disease epithelial cells." American Journal of Physiology-Cell Physiology 298, no. 4 (2010): C831—C846. http://dx.doi.org/10.1152/ajpcell.00032.2009.

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Integrin-associated focal adhesion complex formation and turnover plays an essential role in directing interactions between epithelial cells and the extracellular matrix during organogenesis, leading to appropriate cell spreading, cell-matrix adhesion, and migration. Autosomal recessive polycystic kidney disease (ARPKD) is associated with loss of function of PKHD1-encoded protein fibrocystin-1 and is characterized by cystic dilation of renal collecting tubules (CT) in utero and loss of renal function in patients if they survive the perinatal period. Normal polycystin-1 (PC-1)/focal adhesion co
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Westhoff, M. A., B. Serrels, V. J. Fincham, M. C. Frame, and N. O. Carragher. "Src-Mediated Phosphorylation of Focal Adhesion Kinase Couples Actin and Adhesion Dynamics to Survival Signaling." Molecular and Cellular Biology 24, no. 18 (2004): 8113–33. http://dx.doi.org/10.1128/mcb.24.18.8113-8133.2004.

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ABSTRACT Integrin-associated focal adhesions not only provide adhesive links between cellular actin and extracellular matrix but also are sites of signal transmission into the cell interior. Many cell responses signal through focal adhesion kinase (FAK), often by integrin-induced autophosphorylation of FAK or phosphorylation by Src family kinases. Here, we used an interfering FAK mutant (4-9F-FAK) to show that Src-dependent FAK phosphorylation is required for focal adhesion turnover and cell migration, by controlling assembly of a calpain 2/FAK/Src/p42ERK complex, calpain activation, and prote
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González-Tarragó, Víctor, Alberto Elosegui-Artola, Elsa Bazellières, Roger Oria, Carlos Pérez-González та Pere Roca-Cusachs. "Binding of ZO-1 to α5β1 integrins regulates the mechanical properties of α5β1–fibronectin links". Molecular Biology of the Cell 28, № 14 (2017): 1847–52. http://dx.doi.org/10.1091/mbc.e17-01-0006.

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Fundamental processes in cell adhesion, motility, and rigidity adaptation are regulated by integrin-mediated adhesion to the extracellular matrix (ECM). The link between the ECM component fibronectin (fn) and integrin α5β1 forms a complex with ZO-1 in cells at the edge of migrating monolayers, regulating cell migration. However, how this complex affects the α5β1-fn link is unknown. Here we show that the α5β1/ZO-1 complex decreases the resistance to force of α5β1–fn adhesions located at the edge of migrating cell monolayers while also increasing α5β1 recruitment. Consistently with a molecular c
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Dissertations / Theses on the topic "Adhesion Complex"

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Horton, Edward. "Systems-level analyses of the adhesion nexus." Thesis, University of Manchester, 2015. https://www.research.manchester.ac.uk/portal/en/theses/systemslevel-analyses-of-the-adhesion-nexus(5483903f-35cd-4c90-9e9f-495456b1a371).html.

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Cell adhesion to the extracellular matrix is mediated by the integrin family of adhesion receptors. Integrin receptor engagement initiates the formation of multimolecular protein complexes, termed integrin adhesion complexes (IACs), at the cell membrane. IACs are complex signalling hubs that are enriched in tyrosine-based phosphorylation events and form a mechanochemical connection between integrin receptors and the actin cytoskeleton. Dysregulation of individual IAC components has been reported to influence a wide range of biological processes that contribute to disease. Literature-curated an
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Woodfield, Richard John. "Investigation of the association of P13K with the cadherin-catenin adhesion complex." Thesis, University of Birmingham, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.368435.

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Ng, Daniel. "Investigating the dynamics of adhesion complex turnover by mass spectrometry based proteomics." Thesis, University of Manchester, 2013. https://www.research.manchester.ac.uk/portal/en/theses/investigating-the-dynamics-of-adhesion-complex-turnover-by-mass-spectrometry-based-proteomics(4e6d3051-c007-4715-a290-9acfd45d38a7).html.

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Adhesion complexes (ACs) are large macromolecular complexes of integrins and associated proteins that connect the actin cytoskeleton to the extracellular matrix. In migrating cells, ACs are highly dynamic -- forming and maturing at the cell front and disassembling at the cell rear. The turnover of ACs enables and localises the necessary traction forces required for cell migration. There is evidence for the spatiotemporal recruitment of specific proteins during AC maturation or disassembly; however, a holistic understanding of the compositional changes to ACs during these states is lacking. To
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Hanzlik, Josa A. "Effects of complex vessel geometrics on neutrophil margination and adhesion in post-capillary vessels /." Online version of thesis, 2008. http://hdl.handle.net/1850/9990.

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Teixeira, Pedro Antonio Castelo. "Estudo da virulência, adesão e características fenotípicas de isolados do complexo Sporothrix." Universidade do Estado do Rio de Janeiro, 2011. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=3438.

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Conselho Nacional de Desenvolvimento Científico e Tecnológico<br>A esporotricose é uma doença micótica, infecciosa e crônica, que envolve o tecido cutâneo e subcutâneo, e que pode afetar seres humanos e animais. Esta micose sempre foi atribuída a um único patógeno, o Sporothrix schenckii, um fungo termodimórfico, que cresce como levedura a 37 C e como micélio à temperatura ambiente. No entanto, nos últimos anos, foi demonstrado que isolados identificados como S. schenckii apresentavam grande variabilidade genética, sugerindo que este táxon consiste em um complexo de espécies. Esta doença é cau
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Borja, da rocha Hudson. "Collective effects in muscle contraction and cellular adhesion." Thesis, Université Paris-Saclay (ComUE), 2018. http://www.theses.fr/2018SACLX072/document.

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Deux systèmes biologiques distincts, les muscles squelettiques et les sites d'adhésion de cellules kératocytes en mouvement, sont considérés dans un même cadre en raison de la similitude profonde de leur structure et de leur fonctionnalité. La réponse passive de l'un et de l'autre peut être modélisée à l'aide d'un grand nombre d'unités multi-stables couplées par des interactions à longue portée, et exposées à un désordre spatial fixé et un bruit thermique/mécanique. Les interactions à longue portée dans de tels systèmes conduisent à une synchronisation malgré les fluctuations temporelles et sp
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Fallah, Mohammad Amin [Verfasser], and Achim [Akademischer Betreuer] Wixforth. "Thrombocyte and Melanoma Cell Adhesion under Various Complex Flow Conditions / Mohammad Amin Fallah. Betreuer: Achim Wixforth." Augsburg : Universität Augsburg, 2011. http://d-nb.info/1077700415/34.

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Mills, Fergil. "The role of beta-catenin and the cadherin adhesion complex in synaptic plasticity, learning and addiction." Thesis, University of British Columbia, 2015. http://hdl.handle.net/2429/52342.

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A fundamental property of synapses is their ability to change in response to activity, termed ‘synaptic plasticity’. Synaptic activity can cause long-lasting increases in the strength of synapses (long-term potentiation, or ‘LTP’), as well as decreases in synapse strength (long-term depression or ‘LTD’), both of which are believed to be important for learning and memory. The synaptic adhesion molecules ‘cadherins’ and their intracellular binding partner β-catenin have been identified as key mediators of plasticity at synapses. The cadherin adhesion complex is important for maintaining the stre
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Nathwani, Bhavik Bharat. "Developing luminescent nanoprobes for labeling focal adhesion complex proteins and performing combined AFM-TIRF imaging of these conjugates." Texas A&M University, 2008. http://hdl.handle.net/1969.1/85904.

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Recent progress in the field of semiconductor nanocrystals or Quantum Dots (QDs) has seen them find wider acceptance as a tool in biomedical research labs. As produced, high quality QDs synthesized by high temperature organometallic synthesis, are coated with a hydrophobic ligand. Therefore, they must be further processed to be soluble in water and made biocompatible. A process to coat the QDs with silk fibroin, a fibrous protein derived from the Bombyx mori silk worm, is described. Following the coating process, the characterization of size, optical properties and biocompatibility profile of
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Lee, Seung Eun Ph D. Massachusetts Institute of Technology. "Mechanotransduction by talin : a molecular dynamics study of force-induced recruitment of vinculin to a focal adhesion complex." Thesis, Massachusetts Institute of Technology, 2007. http://hdl.handle.net/1721.1/42290.

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Thesis (Ph. D.)--Massachusetts Institute of Technology, Dept. of Mechanical Engineering, 2007.<br>Includes bibliographical references (p. 112-123).<br>It is now well established that cells can sense mechanical force, but the mechanisms by which force is transduced into a biochemical signal remain poorly understood. One example is the recruitment of vinculin to reinforce initial contacts between a cell and the extracellular matrix due to tensile force. Talin, an essential structural protein in the adhesion, contains the N-terminal five-helix bundle in the rod domain with a known cryptic vinculi
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Books on the topic "Adhesion Complex"

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Steinhoff, Gustav. Cell adhesion molecules in organ transplantation. 2nd ed. Landes Bioscience, 1998.

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Steinhoff, Gustav. Cell adhesion molecules in human organ transplants. R.G. Landes, 1993.

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Woodfield, Richard John. Investigation of the association of P13K with the cadherin-catenin adhesion complex. University of Birmingham, 2000.

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Abbott, Helen. Louis Vierne. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198794691.003.0007.

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Organiste-titulaire of Notre-Dame de Paris for nearly forty years, Louis Vierne composed over sixty songs, including a set of five Baudelaire songs, Cinq Poèmes de Baudelaire, published in 1921. This analysis covers: (a) the context of composition; (b) the connections established between selected poems; (c) the statistical data generated from the adhesion strength tests; and (d) how the data shape an evaluation of Vierne’s settings of Baudelaire. Findings reveal how the poetic line is minimally disrupted in these songs, as the vocal line remains very independent of the piano. As a result, the
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Gregory, Bock, Clark Sarah, and Ciba Foundation, eds. Junctional complexes of epithelial cells. Wiley, 1987.

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Tsai, Ching-Wei, Sanjeev Noel, and Hamid Rabb. Pathophysiology of Acute Kidney Injury, Repair, and Regeneration. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0030.

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Acute kidney injury (AKI), regardless of its aetiology, can elicit persistent or permanent kidney tissue changes that are associated with progression to end-stage renal disease and a greater risk of chronic kidney disease (CKD). In other cases, AKI may result in complete repair and restoration of normal kidney function. The pathophysiological mechanisms of renal injury and repair include vascular, tubular, and inflammatory factors. The initial injury phase is characterized by rarefaction of peritubular vessels and engagement of the immune response via Toll-like receptor binding, activation of
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Book chapters on the topic "Adhesion Complex"

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Ringwald, Martin. "The Uvomorulin—Catenin Complex." In Cellular Adhesion. Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2466-3_5.

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Doolittle, Lynda K., Michael K. Rosen, and Shae B. Padrick. "Purification of Arp2/3 Complex from Saccharomyces cerevisiae." In Adhesion Protein Protocols. Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-538-5_15.

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Byron, Adam. "Proteomic Profiling of Integrin Adhesion Complex Assembly." In Protein Complex Assembly. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7759-8_13.

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Doolittle, Lynda K., Michael K. Rosen, and Shae B. Padrick. "Purification of Native Arp2/3 Complex from Bovine Thymus." In Adhesion Protein Protocols. Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-538-5_14.

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Calvete, Juan José. "Elements for a Structural/Functional Model of Human Platelet Plasma Membrane Fibrinogen Receptor, the Glycoprotein IIb/IIIa Complex (Integrin αIIb/β3)." In Cell Adhesion Molecules. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2830-2_6.

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Irimura, Tatsuro, Masayuki Ota, Yutaka Kawamura, and Yoko Nemoto-Sasaki. "Carbohydrate-Mediated Adhesion of Human Colon Carcinoma Cells to Human Liver Sections." In The Molecular Immunology of Complex Carbohydrates —2. Springer US, 2001. http://dx.doi.org/10.1007/978-1-4615-1267-7_25.

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Kannagi, Reiji. "Transcriptional Regulation of Expression of Carbohydrate Ligands for Cell Adhesion Molecules in the Selectin Family." In The Molecular Immunology of Complex Carbohydrates —2. Springer US, 2001. http://dx.doi.org/10.1007/978-1-4615-1267-7_18.

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Menke, Andre, and Klaudia Giehl. "Regulation of the E-Cadherin Adhesion Complex in Tumor Cell Migration and Invasion." In Cell Migration: Signalling and Mechanisms. KARGER, 2009. http://dx.doi.org/10.1159/000274480.

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Xue, Gi, Qinpin Dai, Gaoquan Shi, and Peiyie Wu. "Complex-Induced Coupling Effect: Adhesion of Polymers to Metal Promoted by Azole Compound." In Controlled Interphases in Composite Materials. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-011-7816-7_23.

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Amatsu, Sho, and Yukako Fujinaga. "Botulinum Hemagglutinin: Critical Protein for Adhesion and Absorption of Neurotoxin Complex in Host Intestine." In Methods in Molecular Biology. Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0430-4_19.

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Conference papers on the topic "Adhesion Complex"

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Lindhorst, Thisbe K. "MIMICKING COMPLEX CARBOHYDRATES TO INHIBIT MICROBIAL ADHESION." In XXIst International Carbohydrate Symposium 2002. TheScientificWorld Ltd, 2002. http://dx.doi.org/10.1100/tsw.2002.372.

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Buckingham, John, Reyda Gonzalez-Nieves, and Mary L. Cutler. "Abstract 5140: Rsu1 and the IPP adhesion complex can regulate adhesion and migration in glioma cell lines." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-5140.

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Kumar, Aloke, David Karig, Suresh Neethirajan, et al. "Adhesion and Formation of Microbial Biofilms in Complex Microfluidic Devices." In ASME 2012 Third International Conference on Micro/Nanoscale Heat and Mass Transfer. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/mnhmt2012-75207.

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Shewanella oneidensis is a metal reducing bacterium, which is of interest for bioremediation and clean energy applications. S. oneidensis biofilms play a critical role in several situations such as in microbial energy harvesting devices. Here, we use a microfluidic device to quantify the effects of hydrodynamics on the biofilm morphology of S. oneidensis. For different rates of fluid flow through a complex microfluidic device, we studied the spatiotemporal dynamics of biofilms, and we quantified several morphological features such as spatial distribution, cluster formation and surface coverage
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Slater, John H., Jordan S. Miller, Shann S. Yu, and Jennifer L. West. "Multifaceted Nano- and Micropatterned Surfaces for Cell Adhesion Manipulation." In ASME 2010 First Global Congress on NanoEngineering for Medicine and Biology. ASMEDC, 2010. http://dx.doi.org/10.1115/nemb2010-13177.

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Surfaces displaying nano- and micropatterned cell adhesive ligands have led to numerous discoveries in cell biology. Soft lithography techniques such as microcontact printing are well suited for creating surfaces displaying micropatterns of one ligand type in a single arrangement but are difficult to implement for the creation of multifaceted surfaces that present multiple ligand types with each ligand confined to their own pattern. To better understand the influence of extracellular matrix (ECM) composition on adhesion site formation and gross cell behavior (motility, proliferation, different
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Tuszynski, George P., Vicki L. Rothman, Andrew Murphy, et al. "Thranbospondin Promotes Cell-and Platelet-Substratum Adhesion." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643820.

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Thrombospondin (TSP), isolated from human platelets, promotes the in vitro, calcium-specific adhesion of a variety of cells, including platelets, melanoma cells, muscle cells, endothelial cells, fibroblasts, and epithelial cells. The cell adhesion-promoting activity of TSP is species independent since human, bovine, pig, rat and mouse cells all adhered to TSP. Furthermore, the cell adhesion-promoting activity of TSP is specific and not due to a nonspecific protein effect or to contamination by fibronectin, vitronectin, or laminin. That is, neither bovine serum albumin nor TSP preparations trea
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Imai, Yohsuke, Hitoshi Kondo, Young Ho Kang, Takuji Ishikawa, Chwee Teck Lim, and Takami Yamaguchi. "A Numerical Model of Adhesion Property of Malaria Infected Red Blood Cells in Micro Scale Blood Flows." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206456.

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Infection by malaria parasite changes mechanical properties of red blood cells (RBCs). Infected red blood cells (IRBCs) lose the deformability but also develop the ability to cytoadhere and rosetting. These outcomes can lead to microvascular blockage [1]. The stiffness of IRBCs [2] and its effects on the flow in micro channels [3] were studied with recent experimental techniques. The cytoadherence and rosetting properties of IRBCs have also been studied experimentally. The cytoadherence is mediated by the interaction of the parasite protein PfEMP1 with several endothelial adhesion molecules, s
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Verma, Devendra, Kalpana Katti, and Dinesh Katti. "Biopolymer Polyelectrolyte Complex Nanocomposites for Bone Tissue Engineering." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206390.

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Recent studies have shown that its necessary for synthetic matrix to provide similar mechanical response as cell’s host environment in the body for desired tissue growth [1,2]. Osteoblast cells reside in the rigid matrix made of collagen fibers and hydroxyapatite crystals. Therefore for the growth of proper functional bone tissue, its utmost necessary that scaffolds provide mechanical response similar to bone matrix. Bone also serves as a mechanical support to the body. Thus scaffolds used for bone tissue engineering should also provide adequate mechanical support to prevent collapse of the ne
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Hanzlik, Josa, Ellen Cretekos, and Kathleen A. Lemkin-Kennard. "Hydrodynamics and Leukocyte Adhesion in Microvessels: State of the Art." In ASME 2008 6th International Conference on Nanochannels, Microchannels, and Minichannels. ASMEDC, 2008. http://dx.doi.org/10.1115/icnmm2008-62067.

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Leukocyte rolling and adhesion are complex physiological processes that have received a great deal of attention over the past decade. Significant increases in the knowledge base related to how leukocytes adhere in shear flows have occurred as a result of the development of novel experimental and computational techniques. Micro- and nano-fabrication techniques have enabled the development of novel flow devices for studying leukocyte adhesion in simple and complex geometries. Improvements in computer technology have enabled simulations of complex flow processes to be developed. As a result of th
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Sakariassen, S. K., E. Fressinaud, D. Meyer, J. J. Sixma, and R. H. Baumgartner. "RHEOLOGY AND PLATELET-SURFACE ADHESION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643987.

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The process of platelet adhesion to sites of vascular injury is pivotal for the arrest of bleeding. The same process may, on the other hand, lead to formation of mural thrombi and may play a role in atherogenesis through the release of platelet-derived growth factor. The events of platelet-surface adhesion may be divided into initial attachment and the subsequent spreading on the surface. These interactions are mediated by a variety of factors, including glycoproteins (GP) in the platelet membrane, von Willebrand factor (vWF) in plasma, and the composition of the surface.However, in most insta
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Lewis, J. C., R. R. Hantgan, N. Kieffer, A. Nurden, and J. Breton-Gorius. "DISTRIBUTION OF GLYCOPROTEINS (GP) lib AND Ilia AND THEIR COMPLEX ON ADHERENT/ACTIVATED PLATELETS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643707.

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Abstract:
Redistribution of GP Ilbllla has recentlybeen demonstrated for platelets activatedin suspension in the presence of either fibrinogen or specific monoclonal antibodies, and it has been suggested that redistribution is important for normal platelet function. Reported here is the distribution of GPIIb an GPIIIa following adhesion, and event focal to the hemostatic process. Human platelets isolated by centrifugation from heparinized blood and washed in Hank’s Salts, pH 6.5 with and without EDTA (3%) were activated by adhesion to carbon-stabilized formvar grids. Subsequent to activation/adhesion, G
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Reports on the topic "Adhesion Complex"

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Furbert-Harris, Paulette M. Eosinophil Cell Lines in a Tri-Cell Multicellular Tumor Spheroid (MTS)/Endothelium Complex: Down Regulation of Adhesion and Integrin Molecules-Implications of Metastasis Inhibition. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada442676.

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