To see the other types of publications on this topic, follow the link: Adhesion of platelets.

Books on the topic 'Adhesion of platelets'

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 21 books for your research on the topic 'Adhesion of platelets.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse books on a wide variety of disciplines and organise your bibliography correctly.

1

Jacek, Hawiger, ed. Platelets: Receptors, adhesion, secretion. Academic, 1989.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
2

Kirton, Christopher Michael. The ability of platelets to promote the adhesion of flowing neutrophils to confluent vascular endothelium. University of Birmingham, 1999.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
3

L, Gordon J., ed. Vascular endothelium: Interactions with circulating cells. Elsevier, 1991.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
4

Eriksson, Andreas. Platelet adhesion to proteins in microplates: Applications in experimental and clinical research. Department of Medical and Health Sciences, Linköping University, 2008.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
5

Platelets, receptors, adhesion, secretion. Academic, 1989.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
6

Abelson, John N., Melvin I. Simon, and Jacek J. Hawiger. Platelets: Receptors, Adhesion, Secretion, Part A. Elsevier Science & Technology Books, 1989.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
7

Platelets: Receptors, adhesion, secretion Part A. Elsevier, 1989. http://dx.doi.org/10.1016/s0076-6879(00)x0378-3.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Abelson, John N., Melvin I. Simon, and Jacek J. Hawiger. Platelets: Receptors, Adhesion, Secretion, Part B. Elsevier Science & Technology Books, 1992.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
9

(Editor), John N. Abelson, Melvin I. Simon (Editor), and Jacek J. Hawiger (Editor), eds. Platelets: Receptors, Adhesion, Secretion, Part A, Volume 169: Volume 169: Platelets Part A (Methods in Enzymology). Academic Press, 1989.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
10

(Editor), John N. Abelson, Melvin I. Simon (Editor), and Jacek J. Hawiger (Editor), eds. Platelets: Receptors, Adhesion, Secretion, Part A, Volume 169: Volume 169: Platelets Part A (Methods in Enzymology). Academic Press, 1989.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
11

(Editor), John N. Abelson, Melvin I. Simon (Editor), and Jacek J. Hawiger (Editor), eds. Platelets: Receptors, Adhesion, Secretion, Part B, Volume 215: Volume 215: Platelets Part B (Methods in Enzymology). Academic Press, 1992.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
12

Curry, Nicola, and Raza Alikhan. Normal platelet function. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0281.

Full text
Abstract:
The platelet is a small (2–4 µm in diameter), discoid, anucleate cell that circulates in the blood. In health, it plays a vital role in haemostasis, and in disease it contributes to disorders of bleeding and thrombosis. Platelets are produced from the surface of megakaryocytes in the bone marrow, under tight homeostatic control regulated by the cytokine thrombopoietin. Platelets have a lifespan of approximately 7–10 days, and usually circulate in the blood stream in a quiescent state. Intact, undamaged vessel walls help to maintain platelets in this inactive state by releasing nitric oxide, wh
APA, Harvard, Vancouver, ISO, and other styles
13

Pruthi, Rajiv K. Coagulation (Hemostasis and Thrombosis). Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0295.

Full text
Abstract:
The coagulation system has 2 essential functions: to maintain hemostasis and to prevent and limit thrombosis. The procoagulant component of the hemostatic system prevents and controls hemorrhage. Vascular injury results in activation of hemostasis, which consists of vasospasm, platelet plug formation (platelet activation, adhesion, and aggregation), and fibrin clot formation (by activation of coagulation factors in the procoagulant system). The anticoagulant system prevents excessive formation of blood clots, and the fibrinolytic system breaks down and remodels blood clots. Quantitative abnorm
APA, Harvard, Vancouver, ISO, and other styles
14

Badimon, Lina, and Gemma Vilahur. Atherosclerosis and thrombosis. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0040.

Full text
Abstract:
Atherosclerosis is the main underlying cause of heart disease. The continuous exposure to cardiovascular risk factors induces endothelial activation/dysfunction which enhances the permeability of the endothelial layer and the expression of cytokines/chemokines and adhesion molecules. This results in the accumulation of lipids (low-density lipoprotein particles) in the extracellular matrix and the triggering of an inflammatory response. Accumulated low-density lipoprotein particles suffer modifications and become pro-atherogenic, enhancing leucocyte recruitment and further transmigration across
APA, Harvard, Vancouver, ISO, and other styles
15

Badimon, Lina, and Gemma Vilahur. Atherosclerosis and thrombosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0040_update_001.

Full text
Abstract:
Atherosclerosis is the main underlying cause of heart disease. The continuous exposure to cardiovascular risk factors induces endothelial activation/dysfunction which enhances the permeability of the endothelial layer and the expression of cytokines/chemokines and adhesion molecules. This results in the accumulation of lipids (low-density lipoprotein particles) in the intimal layer and the triggering of an inflammatory response. Accumulated low-density lipoprotein particles attached to the extracellular matrix suffer modifications and become pro-atherogenic, enhancing leucocyte recruitment and
APA, Harvard, Vancouver, ISO, and other styles
16

Badimon, Lina, and Gemma Vilahur. Atherosclerosis and thrombosis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0040_update_002.

Full text
Abstract:
Atherosclerosis is the main underlying cause of heart disease. The continuous exposure to cardiovascular risk factors induces endothelial activation/dysfunction which enhances the permeability of the endothelial layer and the expression of cytokines/chemokines and adhesion molecules. This results in the accumulation of lipids (low-density lipoprotein particles) in the intimal layer and the triggering of an inflammatory response. Accumulated low-density lipoprotein particles attached to the extracellular matrix suffer modifications and become pro-atherogenic, enhancing leucocyte recruitment and
APA, Harvard, Vancouver, ISO, and other styles
17

Morel, Zeynep. Platelet adhesion to various surfaces studied by on-line acoustic wave sensor. U of Toronto, 1999.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
18

Barros, Rodrigo José Saraiva de, Tereza Cristina de Brito Azevedo, Carla de Castro Sant’Anna, Marianne Rodrigues Fernandes, Leticia Martins Lamarão, and Rommel Mario Rodríguez Burbano. Grupos sanguíneos e anticorpos anti-eritrocitários de importância transfusional. Brazil Publishing, 2020. http://dx.doi.org/10.31012/978-65-5861-112-7.

Full text
Abstract:
Immunohematology is an area dedicated to the study of the interactions of the immune system and blood cells in transfusion practice. Blood transfusion is a therapeutic technique that has been widely used since the 17th century. The transfusion medicine aims to repair the pathological needs of blood components in the living organism, be it red blood cells, plasma, platelets, clotting factors, among others. Despite being a therapeutic means, transfusion of blood components can be considered at risk because it is a biological material and due to the transfusion immunological reactions that can be
APA, Harvard, Vancouver, ISO, and other styles
19

Biologic properties of platelet-derived microparticles: Characterization of mechanisms of generation and adhesion. National Library of Canada, 2001.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
20

Immunocytochemical Aspects of Platelet Membrane Glycoproteins and Adhesive Proteins During Activation (Progress in Histochemistry and Cytochemistry, Vol 30, No 1). Fischer Gustav Verlag GmbH & Co. KG, 1996.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
21

Levi, Marcel, and Tom van der Poll. Coagulation and the endothelium in acute injury in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0307.

Full text
Abstract:
Vascular endothelial cells play a pivotal mediatory role in many responses to systemic inflammation, including the cross-talk between coagulation and inflammation in sepsis. Endothelial cells respond to the cytokines expressed and released by activated leukocytes, but can also release cytokines themselves. Furthermore, endothelial cells are able to express adhesion molecules and growth factors that may not only promote the inflammatory response further, but also affect a myriad of downstream responses. It has recently become clear that, in addition to these mostly indirect effects of the endot
APA, Harvard, Vancouver, ISO, and other styles
We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!