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Books on the topic 'Adipokin'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Preedy, Victor R., and Ross J. Hunter. Adipokines. Boca Raton, FL: CRC Press, 2011.

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2

Fantuzzi, Giamila, and Carol Braunschweig, eds. Adipose Tissue and Adipokines in Health and Disease. Totowa, NJ: Humana Press, 2014. http://dx.doi.org/10.1007/978-1-62703-770-9.

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3

Fantuzzi, Giamila, and Theodore Mazzone, eds. Adipose Tissue and Adipokines in Health and Disease. Totowa, NJ: Humana Press, 2007. http://dx.doi.org/10.1007/978-1-59745-370-7.

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4

Preedy, Victor R., and Ross J. Hunter. Adipokines. Taylor & Francis Group, 2016.

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5

Preedy, Victor R., and Ross J. Hunter. Adipokines. Taylor & Francis Group, 2016.

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6

Adipokines. Taylor & Francis Group, 2017.

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7

Adipokines 2.0. MDPI, 2020. http://dx.doi.org/10.3390/books978-3-03928-587-7.

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8

Neumann, Elena, Klaus Frommer, and Ulf Müller-Ladner. Acute-phase responses and adipocytokines. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0058.

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Adipokines, also called adipocytokines, are highly bioactive substances mainly expressed by adipose tissue. In addition to adipocytes, different cell types resident in various tissues produce adipokines under pathophysiological conditions. Adipokines include a growing number of pluripotent molecules such as adiponectin, resistin, leptin, and visfatin. Since distinct effects of adipokines on inflammation have been described, their influence on the (innate) immune system has been investigated in rheumatology, gastroenterology, and endocrinology. This review gives an overview on the current knowledge about the influence which adipokines have on the immune system and chronic inflammation in rheumatic diseases.

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9

Felice, Valentina Di, Dario Coletti, and Marilia Seelaender, eds. Myokines, Adipokines, Cytokines in Muscle Pathophysiology. Frontiers Media SA, 2020. http://dx.doi.org/10.3389/978-2-88966-272-2.

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10

Fantuzzi, Giamila, and Carol Braunschweig. Adipose Tissue and Adipokines in Health and Disease. Humana, 2016.

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11

Fantuzzi, Giamila, Theodore Mazzone, A. P. Goldberg, and S. K. Fried. Adipose Tissue and Adipokines in Health and Disease. Humana, 2011.

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12

Fantuzzi, Giamila, and Carol Braunschweig. Adipose Tissue and Adipokines in Health and Disease. Humana, 2014.

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13

Fantuzzi, Giamila, and Theodore Mazzone. Adipose Tissue And Adipokines In Health And Disease. Humana P.,U.S., 2006.

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14

Fantuzzi, Giamila, and Theodore Mazzone. Adipose Tissue and Adipokines in Health and Disease. Springer, 2008.

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15

(Editor), Giamila Fantuzzi, and Theodore Mazzone (Editor), eds. Adipose Tissue and Adipokines in Health and Disease (Nutrition and Health). Humana Press, 2007.

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16

B. Bayraktar* and E. Tekce. Effect of probiotics supplementation on adipokine profile (visfatin, adiponectin and chemerin), intestinal (citrulline) and thyroid function in Japanese quail subjected to heat stress. Verlag Eugen Ulmer, 2020. http://dx.doi.org/10.1399/eps.2020.309.

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17

Yang, Zhihong, and Xiu-Fen Ming. Adventitia and perivascular adipose tissue—the integral unit in vascular disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0020.

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Obesity and obesity-associated metabolic disorders are highly associated with cardiovascular disease. Abnormal ectopic deposition and accumulation of adipose tissue in organs, including perivascular space (perivascular adipose tissue, PVAT) in obesity are emerging to contribute to vascular disease development through pathological paracrine and/or endocrine secretion of cytokines, namely adipokines, which are vasoactive factors including vascular relaxing and contracting factors, smooth muscle growth promoting and inhibiting factors, and pro- and anti-inflammatory factors. In obesity, production of these factors from PVAT is altered and in imbalance which favours vascular contraction, pathological remodelling, and inflammation. In cross-talk with the endothelium, the functional changes of adventitia and PVAT are detrimental and importantly contribute to the acceleration of vascular atherosclerosis and complications associated with obesity and metabolic disorders

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18

Deveza, Leticia A., Changhai Ding, Xingzhong Jin, Xia Wang, Zhaohua Zhu, and David J. Hunter. Laboratory tests. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0019.

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In clinical practice, laboratory tests play a major role in the investigation of underlying diseases predisposing to osteoarthritis (OA) (e.g. metabolic and genetic causes) and in the differential diagnosis of other joint disorders, particularly autoimmune and crystal-related arthropathies. In terms of diagnosis and management of OA, there is still no recommendation in guidelines for the use of laboratory tests. However, an increasing number of potential applications of laboratory tests in OA are emerging. In this regard, research investigating the role of vitamin D and other substances such as inflammatory markers, cytokines, and adipokines in the OA process is helping to reveal novel pathophysiologic mechanisms and, consequently, new possible therapeutic approaches. Furthermore, biochemical markers of cartilage, bone, and synovium metabolism is a growing field in OA and may also contribute to the diagnosis of early OA stages, assessment of prognosis, and management, once properly validated.

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