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Dissertations / Theses on the topic 'Adipose tissue inflammation'

Author: Grafiati
Published: 4 June 2021
Last updated: 26 July 2025

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1

Ghaith, Mazen Mohammedsaeed. "Inflammation of white adipose tissue in obesity." Thesis, University of Nottingham, 2016. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.716840.

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Obesity is now recognized as an inflammatory disease, in which white adipose tissue (WAT) is an active site and major contributor to this manifestation. The work in this thesis was directed towards the notion of adipose tissue inflammation in obesity and examined whether low oxygen tension or “hypoxia” may underpin this phenomenon. All experiments in this thesis looked at aspects related to adipose tissue biology, function, regulation and inflammation from the molecular level to the cellular and/or physiological level; with either human adipose biopsies or primary culture of cells isolated fro
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2

Fulham, Melissa A. "Characterization of Adipose Tissue Inflammation in Alcoholic Liver Disease." eScholarship@UMMS, 2011. http://escholarship.umassmed.edu/gsbs_diss/940.

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Adipose tissue inflammation has an impact on liver health and it has been demonstrated that chronic alcohol consumption leads to the expression of pro-inflammatory markers in the adipose tissue. A thorough characterization of alcohol-induced adipose inflammation is lacking, and is important to understand in order to identify immune-related mechanisms that drive this phenomenon. Current therapeutic regimens for alcoholic liver disease are ineffective. It is critical to understand how other organs influence liver injury in this disease when developing novel and effective therapies in the future.
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Fulham, Melissa A. "Characterization of Adipose Tissue Inflammation in Alcoholic Liver Disease." eScholarship@UMMS, 2017. https://escholarship.umassmed.edu/gsbs_diss/940.

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Adipose tissue inflammation has an impact on liver health and it has been demonstrated that chronic alcohol consumption leads to the expression of pro-inflammatory markers in the adipose tissue. A thorough characterization of alcohol-induced adipose inflammation is lacking, and is important to understand in order to identify immune-related mechanisms that drive this phenomenon. Current therapeutic regimens for alcoholic liver disease are ineffective. It is critical to understand how other organs influence liver injury in this disease when developing novel and effective therapies in the future.
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4

Polaki, Venkata Sai Usha Sri. "Role of exercise in macrophage polarization of perivascular adipose tissue and adipose tissue inflammation in hypertensive mice model." Wright State University / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=wright1598890403047938.

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5

Rossetti, Maria Agustina. "The Influence of Oxytocin on Adipose Tissue, Inflammation and Atherosclerosis." Scholarly Repository, 2011. http://scholarlyrepository.miami.edu/oa_theses/295.

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Purpose: The present study investigates the potential anti-inflammatory effects of in vivo oxytocin (OT) infusion on adipose tissue inflammation in the Watanabe Heritable Hyperlipidimic Rabbits (WHHL). Methods: Twenty-eight 3-month-old WHHL were surgically implanted with osmotic minipumps containing OT (n = 14, infusion rate 250 ng/kg/hr) or vehicle (n = 14). Blood samples were taken at baseline, midpoint, and endpoint for lipids and C-reactive protein (CRP). After 16 weeks, animals were sacrificed and samples of adipose tissue (epididiymal, retroperitoneal, mesenteric, pericardial, and subcut
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6

Kusminski, C. M. "Adipose tissue derived factors in obesity, inflammation & energy homeostasis." Thesis, University of Warwick, 2006. http://wrap.warwick.ac.uk/39992/.

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Obesity is the foremost contributory factor in the progression to type 2 diabetes mellitus (T2DM). Moreover, chronic inflammation, through activation of innate immunity is proposed to link obesity, insulin resistance and T2DM. Adipose tissue, traditionally considered a storage compartment for triglycerides, also functions as an active endocrine organ. Adipocyte-secreted products, termed adipokines, may link obesity-associated inflammation and insulin resistance. Adipokines exert multiple effects on insulin sensitisation, glucose homeostasis, inflammatory processes or central systems mediating
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7

Kosicka, Anna. "Role of adipose tissue as an endocrine organ in systemic inflammation." Thesis, University of Westminster, 2014. https://westminsterresearch.westminster.ac.uk/item/964qw/role-of-adipose-tissue-as-an-endocrineorgan-in-systemic-inflammation.

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Introduction: The escalating public health problem represented by obesity has spurred multidisciplinary research into adipose tissue and importantly, the molecular biology of the adipocyte. The concept of adipose tissue as an endocrine organ, in addition to an energy storage compartment, is now pivotal in linking excess adiposity to disease states. Recent studies suggest that obesity related metabolic disorders are characterised by mild chronic inflammation as a result of adipocytokine production from fat tissue leading to dysregulation in the pro/anti-inflammatory systemic balance. Adipokines
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8

Nakazeki, Fumiko. "Loss of periostin ameliorates adipose tissue inflammation and fibrosis in vivo." Kyoto University, 2019. http://hdl.handle.net/2433/242372.

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9

Sardi, C. "DIET AND INFLAMMATION: FOCUS ON THE SMALL INTESTINE AND ADIPOSE TISSUE." Doctoral thesis, Università degli Studi di Milano, 2015. http://hdl.handle.net/2434/252813.

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ABSTRACT Extensive scientific evidence indicating that non-optimal diet and sedentary lifestyle constitute some of the behaviors /risk factors associated with the onset of many diseases with significant societal impact, such as inflammatory bowel disease (IBD) and obesity. Diet is considered one of the main factors contributing to the diversity of gut microbiota. As TLRs/NLRs recognize bacterial products, there is a possibility that changes in bacterial composition induced by diet can promote a different response by TLRs/NLRs and thus may increase susceptibility to intestinal inflammation. A
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10

Ceppo, Franck. "Rôle de la kinase inflammatoire Tpl2 dans l'inflammation du tissu adipeux lors de l'obésité." Thesis, Nice, 2014. http://www.theses.fr/2014NICE4110/document.

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Lors de l’obésité un état inflammatoire chronique de bas grade se développe dans le tissu adipeux et contribue à son dysfonctionnement participant ainsi au développement de l’insulino-résistance. Cette inflammation est due à une infiltration et une activation de différentes cellules immunitaires dans le tissu adipeux, dont des macrophages. Les acides gras saturés et le LPS participent à l’activation des cellules immunitaires conduisant ainsi à la production de cytokines inflammatoires, tous ces facteurs étant impliqués dans le dysfonctionnement du tissu adipeux et dans l’insulino-résistance. L
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11

Cole, Kathryn Ann Beck Melinda A. "Adipose tissue inflammation is associated with immune dysfunction during influenza virus infection." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2007. http://dc.lib.unc.edu/u?/etd,1703.

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Thesis (Ph. D.)--University of North Carolina at Chapel Hill, 2008.<br>Title from electronic title page (viewed Sep. 16, 2008). "... in partial fulfillment of the requirements for the degree of Doctor of Philosophy in the Department of Nutrition." Discipline: Nutrition; Department/School: Public Health.
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12

Li, Dan. "Identification of xanthones to ameliorate metabolic disorders through targeting adipose tissue inflammation." Thesis, University of Macau, 2018. http://umaclib3.umac.mo/record=b3952496.

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13

Ceppo, Franck. "Rôle de la kinase inflammatoire Tpl2 dans l'inflammation du tissu adipeux lors de l'obésité." Electronic Thesis or Diss., Nice, 2014. http://www.theses.fr/2014NICE4110.

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Lors de l’obésité un état inflammatoire chronique de bas grade se développe dans le tissu adipeux et contribue à son dysfonctionnement participant ainsi au développement de l’insulino-résistance. Cette inflammation est due à une infiltration et une activation de différentes cellules immunitaires dans le tissu adipeux, dont des macrophages. Les acides gras saturés et le LPS participent à l’activation des cellules immunitaires conduisant ainsi à la production de cytokines inflammatoires, tous ces facteurs étant impliqués dans le dysfonctionnement du tissu adipeux et dans l’insulino-résistance. L
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14

Travers, Rebecca. "Metabolic and immune system cross-talk in human adipose tissue." Thesis, University of Bath, 2015. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.665387.

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The overall aim of the work presented in this thesis was to further characterise aspects of metabolic and immune system cross-talk in human subcutaneous adipose tissue, with a particular emphasis on the potential role of T-lymphocytes in adipose tissue dysfunction and insulin resistance. Chapter 3 characterised macrophage and T-lymphocyte populations residing in adipose tissue from lean through to class I obese men. This work demonstrated that T-lymphocytes display increased activation with increased adiposity and that potential compensatory mechanisms may be present to help counteract adipose
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15

Méndez, Lara Karen Alejandra. "Nicotinamide effects on adiposity, energy metabolism, inflammation and atherosclerosis in mice." Doctoral thesis, Universitat Autònoma de Barcelona, 2019. http://hdl.handle.net/10803/669348.

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Antecedentes e hipótesis La disfunción del tejido adiposo se encuentra frecuentemente asociada con alteraciones en la homeostasis metabólica, cardiovascular e inflamatoria crónica leve de la obesidad. El tejido adiposo es considerado como diana terapéutica para prevenir la obesidad. Precursores de la nicotinamida adenina dinucleotido (NAD)+, tales como el ribósido de nicotinamida y mononucleótido de nicotinamida promueven el metabolismo energético y previenen la ganancia de peso corporal en modelos animales. Sin embargo, en estos trabajos no se abordó directamente su posible efecto sobre las
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16

Battle, Jenny Helen. "11β-Hydroxysteroid Dehydrogenase Type 1 in adipose tissue macrophages and inflammation in obesity". Thesis, University of Edinburgh, 2014. http://hdl.handle.net/1842/10056.

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17

Thapar, Divya. "Osteopontin knockout abates high fat diet-induced insulin resistance and adipose tissue inflammation." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2008. http://wwwlib.umi.com/cr/ucsd/fullcit?p1459910.

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Thesis (M.S.)--University of California, San Diego, 2008.<br>Title from first page of PDF file (viewed January 5, 2009). Available via ProQuest Digital Dissertations. Includes bibliographical references (p. 43-46).
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18

DI, VINCENZO ANGELICA. "Morphology of visceral and subcutaneous adipose tissue of obese subjects." Doctoral thesis, Università Politecnica delle Marche, 2021. http://hdl.handle.net/11566/289620.

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Nei periodi di bilancio energetico positivo si verifica un progressivo rimodellamento patologico dei tessuti adiposi: gli adipociti aumentano il loro volume portando ad un conseguente aumento della massa grassa. L'ipertrofia delle cellule adipose è associata ad anomalie cellulari e ad una massiccia infiltrazione di macrofagi che inducono ad uno stato infiammatorio cronico di basso grado che porta nel tempo all'insulino-resistenza ed infine al diabete mellito di tipo 2. Un altro segno distintivo dell'obesità è il rimodellamento della matrice extracellulare con un eccessivo accumulo di fibrosi c
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19

Zieger, Konstanze, Juliane Weiner, Anne Kunath, et al. "Ablation of kallikrein 7 (KLK7) in adipose tissue ameliorates metabolic consequences of high fat diet-induced obesity by counteracting adipose tissue inflammation in vivo." Springer, 2018. https://ul.qucosa.de/id/qucosa%3A33207.

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Vaspin is an adipokine which improves glucose metabolism and insulin sensitivity in obesity. Kallikrein 7 (KLK7) is the first known protease target inhibited by vaspin and a potential target for the treatment of metabolic disorders. Here, we tested the hypothesis that inhibition of KLK7 in adipose tissue may beneficially affect glucose metabolism and adipose tissue function. Therefore, we have inactivated the Klk7 gene in adipose tissue using conditional gene-targeting strategies in mice. Klk7-deficient mice (ATKlk7 −/−) exhibited less weight gain, predominant expansion of subcutaneous adipose
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20

Kogan, Sophia. "Role of Inflammation in Diet-Induced Obesity: A Dissertation." eScholarship@UMMS, 2013. http://escholarship.umassmed.edu/gsbs_diss/647.

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Obesity results from expansion of white adipose tissue. The inability of white adipose tissue to adequately store lipids leads to ectopic deposition of lipids in non-adipose tissue that can lead to systemic insulin resistance. It is well known that insulin resistance correlates with inflammation of adipose tissue in obese animals and humans. Decreasing inflammation in the adipose tissue has been proven as a therapeutic strategy for improvement of insulin sensitivity in vivo. Numerous factors secreted by immune cells, including macrophages, have been suggested as regulating adipose tissue insul
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21

Guri, Amir Joseph. "Abscisic acid ameliorates glucose tolerance and obesity-induced inflammation." Diss., Virginia Tech, 2007. http://hdl.handle.net/10919/29433.

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Obesity is a disease characterized by chronic inflammation and the progressive loss in systemic insulin sensitivity. One of the more effective medications in the treatment of insulin resistance have been the thiazolidinediones (TZDs), which act through the nuclear receptor peroxisome proliferator-activated receptor gamma (PPARgamma ). Due to the many side-effects of TZDs, our laboratory sought out a natural phytochemical, abscisic acid (ABA), with chemical similarities to TZDs. Our first study demonstrated that ABA activates PPARgamma in vitro and significantly ameliorates white adipose tis
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22

Garcia, Jéssica Leite. "Impacto da ingestão de carboidratos simples e gordura sobre parâmetros metabólicos, inflamatórios e pró-oxidantes no plasma e no tecido adiposo independente de obesidade." Universidade Estadual Paulista (UNESP), 2018. http://hdl.handle.net/11449/153008.

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Submitted by JÉSSICA LEITE GARCIA null (jessleitegarcia@gmail.com) on 2018-03-13T15:03:34Z No. of bitstreams: 1 DISSERTAÇÃO VERSÃO FINAL - Jéssica Leite Garcia.pdf: 1374409 bytes, checksum: d478be5f838f5ea5c7253f241ca08405 (MD5)<br>Approved for entry into archive by Luciana Pizzani null (luciana@btu.unesp.br) on 2018-03-13T20:18:59Z (GMT) No. of bitstreams: 1 garcia_jl_me_bot.pdf: 1374409 bytes, checksum: d478be5f838f5ea5c7253f241ca08405 (MD5)<br>Made available in DSpace on 2018-03-13T20:18:59Z (GMT). No. of bitstreams: 1 garcia_jl_me_bot.pdf: 1374409 bytes, checksum: d478be5f838f5ea5c7253f241
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23

Keuper, Michaela [Verfasser]. "The role of fat cell apoptosis during obesity-associated adipose tissue inflammation / Michaela Keuper." Ulm : Universität Ulm. Fakultät für Naturwissenschaften, 2011. http://d-nb.info/1017543380/34.

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24

Damouche, Abderaouf. "Infection par le VIH et tissu adipeux." Thesis, Université Paris-Saclay (ComUE), 2017. http://www.theses.fr/2017SACLS124/document.

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Les traitements antirétroviraux (TARV) actuels ne parviennent pas à éradiquer totalement le virus de l'immunodéficience humaine (VIH) de l’organisme, le virus reste présent dans des sites anatomiques ou cellulaires appelés "réservoirs". Cette persistance virale se traduit aussi par une inflammation chronique à bas-bruit, responsable de la majorité des comorbidités associées au VIH.L'objectif de mon travail de thèse est d'étudier le rôle du tissu adipeux infecté en tant que facteur d'inflammation et site de persistance virale. Le tissu adipeux est un organe endocrine, doué de propriétés immunol
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25

Caër, Charles. "Inflammation du tissu adipeux au cours de l'obésité humaine : implication des lymphocytes Th17." Thesis, Paris 6, 2016. http://www.theses.fr/2016PA066105/document.

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Une série d’études récentes chez l’homme et dans les modèles murins a conduit à la mise en évidence d’une réponse immune, qui met en jeu des cellules de l’immunité innée et de l’immunité adaptative dans le tissu adipeux (TA) obèse. Nous avons mis en évidence un dialogue pro-inflammatoire entre les macrophages et les LT CD4+ dans le TA humain obèse mettant en jeu l’IL-1β, l’IL-17 et l’IL-22. Le pourcentage de Th17 est positivement corrélé au % d’HbA1c. De plus, la sécrétion des cytokines impliquées dans cette boucle pro-inflammatoire diminuent après une perte de poids induite par la chirurgie b
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26

MATACCHIONE, GIULIA. "Senescent macrophages in adipose tissue: ex vivo and in vitro characterization." Doctoral thesis, Università Politecnica delle Marche, 2020. http://hdl.handle.net/11566/274586.

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L'obesità è una delle maggiori sfide sanitarie a livello mondiale, considerata fattore di rischio chiave per una serie di patologie legate all'età, incluso il diabete. Recenti studi hanno suggerito che le cellule senescenti (senescent cells, SCs) si accumulano nel tessuto adiposo dell’obeso. Tuttavia, il fenotipo e il ruolo delle SCs nell'infiammazione obesità-dipendente non sono stati chiaramente descritti. Campioni di tessuto adiposo addominale viscerale (visceral adipose tissue, VAT) e sottocutaneo (subcutaneous adipose tissue, SAT) ottenuti da 17 pazienti obesi (BMI≥ 38 kg / m2) sono stat
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27

Amano, Shinya U. "Local Macrophage Proliferation in Adipose Tissue Is a Characteristic of Obesity-Associated Inflammation: A Dissertation." eScholarship@UMMS, 2013. https://escholarship.umassmed.edu/gsbs_diss/652.

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Obesity and diabetes are major public health problems facing the world today. Extending our understanding of adipose tissue biology, and how it changes in obesity, will hopefully better equip our society in dealing with the obesity epidemic. Macrophages and other immune cells accumulate in the adipose tissue in obesity and secrete cytokines that can promote insulin resistance. Adipose tissue macrophages (ATMs) are thought to originate from bone marrow-derived monocytes, which infiltrate the tissue from the circulation. Much work has been done to demonstrate that inhibition of monocyte recruitm
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Amano, Shinya U. "Local Macrophage Proliferation in Adipose Tissue Is a Characteristic of Obesity-Associated Inflammation: A Dissertation." eScholarship@UMMS, 2003. http://escholarship.umassmed.edu/gsbs_diss/652.

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Obesity and diabetes are major public health problems facing the world today. Extending our understanding of adipose tissue biology, and how it changes in obesity, will hopefully better equip our society in dealing with the obesity epidemic. Macrophages and other immune cells accumulate in the adipose tissue in obesity and secrete cytokines that can promote insulin resistance. Adipose tissue macrophages (ATMs) are thought to originate from bone marrow-derived monocytes, which infiltrate the tissue from the circulation. Much work has been done to demonstrate that inhibition of monocyte recruitm
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29

Jalilvand, Anahita D. "Changes in Adipose Tissue Inflammation following Surgical Weight Loss in Patients with Obesity: The Relationship between the Adipose Tissue Immune Microenvironment and Clinical Outcomes after Bariatric Surgery." The Ohio State University, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1576924692384911.

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30

Malandrino, Maria Ida. "Study of carnitine palmitoyltransferase 1A (CPT1A) in adipose tissue. Effects on obesity, inflammation and insulin resistance." Doctoral thesis, Universitat de Barcelona, 2013. http://hdl.handle.net/10803/123966.

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Current lifestyle with high-energy diets and little exercise is triggering an alarming growth in obesity. Excess of adiposity is leading to severe increase in associated pathologies, such as insulin resistance, type 2 diabetes (T2DM), atherosclerosis, cancer, arthritis, asthma, and hypertension. This, together with the lack of efficient anticobesity drugs, is the driving force behind much research. Obesity is associated with adipocyte dysfunction, macrophage infiltration, inflammation and decreased fatty acid oxidation (FAO) levels. Recent results suggest that enhan
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31

Kaaman, Maria. "Role of inflammatory and mitochondria genes in adipose tissue and obesity /." Stockholm, 2007. http://diss.kib.ki.se/2007/978-91-7357-331-3/.

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32

Jones, Danielle Alice. "Adipose tissue as a mediator of inflammation and oxidative cellular damage in obesity and type 2 diabetes." Thesis, Swansea University, 2013. https://cronfa.swan.ac.uk/Record/cronfa42244.

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In the past 30 years the prevalence of obesity has almost trebled resulting in an increased incidence of type 2 diabetes mellitus (T2DM) and other co-morbidities. Visceral adipose tissue is believed to play a vital role in these conditions, but underlying mechanisms remain unclear. A close association exists between obesity, diabetes and oxidative stress, resulting in increased reactive oxygen species formation. The experiments in this thesis address this by searching for possible biochemical changes which may be specific for the onset of obesity related T2DM, as well as looking for genetic al
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33

Riccardi, Daniela Mendes dos Reis. "Perfil lipídico e inflamação sistêmica na caquexia associada ao câncer." Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/42/42134/tde-25022016-103812/.

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A caquexia associada ao câncer, cujo o sintoma mais notável é a severa e rápida perda de peso, afeta cerca de 80% dos pacientes com câncer avaçado e constitui causa direta de morte em 22 a 40% dos casos. Estudos recentes têm mostrado que os mediadores inflamatórios têm um papel importante no desenvolvimento de caquexia, hoje em dia considerada como uma doença inflamatória crónica. Um possível contribuinte é o tecido adiposo branco (TAB) que sofre diversas alterações, como por exemplo, reorganização morfológica e aumento da lipólise, liberando assim ácidos graxos livres (AGL), que por sua vez,
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Kelly, Benjamin M. "Non-pharmacological interventions for the treatment and prevention of cardio-metabolic disease." Thesis, Loughborough University, 2015. https://dspace.lboro.ac.uk/2134/17118.

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In recent years there has been a surge in interest concerning high intensity intermittent exercise training (HIT) due to its ability to confer rapid notable cardio-metabolic health benefits. Specifically, HIT has been shown to improve insulin sensitivity and glycaemic control as well as other cardiovascular health factors after just 2 weeks of training (typically 6 training sessions). This thesis investigated the potential therapeutic role of HIT training within obese cohorts specifically addressing metabolic health, inclusive of inflammatory profiles and glycaemic control.
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Axelsson, Jonas. "Fat tissue, adipokines and clinical complications of chronic kidney disease /." Stockholm : Department of Clinical Science, Intervention and Technology, Divisions of Renal Medicine and Baxter Novum, Karolinska institutet, 2006. http://diss.kib.ki.se/2006/91-7140-653-0/.

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36

Teixeira, Pryscila Dryelle Sousa. "Restrição dietética de magnésio em ratos alimentados com rações hiperlipídicas: implicações sobre o status de ferro e a inflamação no tecido adiposo visceral." Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/9/9132/tde-17052015-140117/.

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A deficiência dietética de magnésio (Mg), muitas vezes observada na população, em obesos, é condição que pode contribuir para a inflamação que, por sua vez, influencia o metabolismo do ferro. Com essa perspectiva, o objetivo deste trabalho foi avaliar a influência da deficiência de magnésio no processo inflamatório do tecido adiposo de ratos alimentados com rações hiperlipídicas e a repercussão desses efeitos na homeostase de ferro (Fe). Dois ensaios, com duração de 8 semanas, foram conduzidos, com ratos Wistar, machos, com peso inicial entre 180 e 200 g. No primeiro ensaio (Ensaio 1), os anim
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Dalmas, Elise. "Adipose tissue inflammation in obesity and during surgery induced-weight loss : emerging paracrine dialogue between macrophages and lymphocytes." Paris 6, 2012. http://www.theses.fr/2012PA066173.

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L’obésité est un état inflammatoire chronique dit de « bas grade », associé à l’augmentation de facteurs pro-inflammatoires dans la circulation et le tissu adipeux. Chez les sujets obèses, nous montrons qu’une série de facteurs inflammatoires sériques évoluent selon une régulation bi-phasique au cours de la perte de poids induite par chirurgie bariatrique, avec une diminution drastique à 3 mois suivie d’un rebond et d’une stabilisation à 1 an. Le statut nutritionnel des patients est un déterminant majeur de ce profile inattendu. Chez ces sujets, nous mettons en évidence un enrichissement en mo
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38

Neves, Rodrigo Xavier das. "Inflamação e alteração metabólica na caquexia: papel dos adipócitos, do fígado e da modulação oferecida pela microbiota intestinal." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/42/42134/tde-06092016-154205/.

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Objetivo do estudo foi estudar a participação dos adipócitos e do fígado na inflamação e o papel da microbiota ao longo da progressão da caquexia. Para verificar o comportamento dos adipócitos e do fígado utilizamos ratos Wistar macho de 8 semanas, divididos em dois grupos: i) controle; ii) tumor. Este último foi subdividido em 2 grupos: a) 7º. e b) 14º. dia após a inoculação das células tumorais. Para avaliar o comportamento da microbiota durante o quadro de caquexia utilizamos camundongos C57Bl/6 convencional e germ free de 8-10 semanas, divididos em quatro grupos: i) Convencional controle;
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Donatto, Felipe Fedrizzi. "Efeitos do treinamento de força e da suplementação de tributirina sobre os parâmetros da caquexia em ratos inoculados com tumor de Walker 256." Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/42/42134/tde-26062014-200642/.

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A caquexia é um síndrome para neoplástica que interfere na morfologia e fisiologia do tecido adiposo. O objetivo do presente estudo foi avaliar os efeitos do treinamento de força e da suplementação de tributirina sobre os parâmetros da caquexia em animais inoculados com células do tumor de Walker 256. Ratos Wistar (n=7) foram randomizados em grupos experimentais: Controle (CT), Tumor (TB), Tumor tratado com tributirina (TBTrib), Treinado com Tumor (TFTB) e Tumor Treinado e suplementado com tributirina (TFTBTrib). Os parâmetros: peso tumoral, peso total carregado, quantidades de glicogênio, per
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Nixon, Mark. "Interactions between glucocorticoid metabolism and inflammation in obesity and insulin resistance." Thesis, University of Edinburgh, 2011. http://hdl.handle.net/1842/5593.

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Inflammation plays a key role in the underlying pathogenesis of obesity and its associated health risks, with increased markers of inflammation evident in both liver and adipose tissue. In parallel, there is dysregulation of glucocorticoid metabolism in obesity, with increased adipose levels of the glucocorticoid-regenerating enzyme 11β-hydroxysteroid dehydrogenase type 1 (11βHSD1) and increased hepatic levels of 5α-reductase type 1 (5αR1), which catalyses the reduction of glucocorticoids. Both the mechanisms and consequences of this glucocorticoid metabolism dysregulation remain unclear, howe
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41

Saroha, Vivek. "Developmental programming of the cell stress response and metabolic inflammation in liver and adipose tissue in an ovine model." Thesis, University of Nottingham, 2017. http://eprints.nottingham.ac.uk/47527/.

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A state of chronic metabolic inflammation and activation of the cell stress response in organs such as liver and adipose tissue are important pathogenic adaptations with the onset of obesity and the metabolic syndrome. The extent to which these processes are modulated by the early life nutritional experience is not well established, especially in large animal models. The overall aim of this thesis was to identify whether nutritional programming during prenatal and postnatal development enhances metabolic inflammation and cell stress response of obesity. A nutritional model of fetal growth rest
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42

Davis, Jeremy E. "Development of obesity-induced inflammation and insulin resistance the role of adipose tissue, fatty acids, and toll-like receptors /." [Ames, Iowa : Iowa State University], 2008.

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43

Awada, Rana. "Inflammation du tissu adipeux et vulnérabilité du système nerveux central." Phd thesis, Université de la Réunion, 2011. http://tel.archives-ouvertes.fr/tel-00783611.

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L'obésité est depuis quelques années reconnue comme un des fléaux du XXIe siècle en affectant environ 10% de la population adulte mondiale. Le passage de l'obésité au stade de pandémie est le résultat de multiples facteurs dont une mauvaise hygiène alimentaire et une sédentarité croissante. La découverte récente de l'existence d'un état inflammatoire chronique au cours de l'obésité pouvant intervenir dans la physiopathologie de la maladie et de ses nombreuses complications fait aujourd'hui l'objet d'intenses investigations. Or, le tissu adipeux (TA) qui, parallèlement à son activité métaboliqu
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Ljung, Lotta. "Aspects on inflammation and cardiovascular comorbidity in rheumatoid arthritis." Doctoral thesis, Umeå universitet, Reumatologi, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-57702.

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There is an increased risk for cardiovascular (CV) comorbidity among patients with rheumatoid arthritis (RA), with premature atherosclerosis, and a higher incidence of CV events, compared with the general population. Disease related factors add to the CV risk, and interact with the traditional CV risk factors. The underlying mechanism for this is not completely understood. In active RA there is a loss of muscle mass and an increase in body fat content. Production of cytokines, i.e., adipokines, in the adipose tissue could link the inflammation with the CV system. Control of the inflammation ha
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Damouche, Abderaouf. "Infection par le VIH et tissu adipeux." Electronic Thesis or Diss., Université Paris-Saclay (ComUE), 2017. http://www.theses.fr/2017SACLS124.

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Les traitements antirétroviraux (TARV) actuels ne parviennent pas à éradiquer totalement le virus de l'immunodéficience humaine (VIH) de l’organisme, le virus reste présent dans des sites anatomiques ou cellulaires appelés "réservoirs". Cette persistance virale se traduit aussi par une inflammation chronique à bas-bruit, responsable de la majorité des comorbidités associées au VIH.L'objectif de mon travail de thèse est d'étudier le rôle du tissu adipeux infecté en tant que facteur d'inflammation et site de persistance virale. Le tissu adipeux est un organe endocrine, doué de propriétés immunol
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46

Belpiede, Luciana Tocci. "A expressão do GLUT4 no tecido adiposo de camundongos varia de acordo com a sensibilidade à insulina durante o desenvolvimento da obesidade." Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/42/42137/tde-19022016-120232/.

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Obesidade envolve redução da expressão do transportador de glicose GLUT4 e do gene codificador SLC2A4 em tecido adiposo, junto com aumento na produção de TNFA, que regula negativamente o SLC2A4. Objetivo: avaliar regulação de Slc2a4/GLUT4 em tecido adiposo perigonadal de camundongos controles (C-tratados com salina) e obesos (MSG-tratados com MSG), submetidos a dieta normolipídica (DN) ou hiperlipídica (DH) por 4, 8, 12 e 16 semanas. Em 4 semanas MSG-DN apresentaram aumento na na sensibilidade insulínica e no GLUT4. A partir de 8 semanas, esse quadro se inverteu, com redução de Slc2a4/GLUT4 e
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Sharkey, Don. "The impact of pre- and postnatal nutrition on cellular stress and inflammation in the renal and perirenal adipose tissue of sheep." Thesis, University of Nottingham, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.523579.

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NERI, I. GIULINI. "BODY COMPOSITION, METABOLIC PARAMETERS, ABDOMINAL ADIPOSE TISSUE, ADIPOCYTOKINES AND MARKERS OF INFLAMMATION IN PATIENTS IN EVOLUTIVE AGE TREATED WITH KETOGENIC DIET." Doctoral thesis, Università degli Studi di Milano, 2014. http://hdl.handle.net/2434/232646.

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The ketogenic diet (KD) is a high-fat, low-carbohydrate, and normal-protein diet which increases the production of the ketone bodies providing an alternative fuel source for the brain, used worldwide for children with intractable epilepsy. Only few studies were performed to study the effect of KD on nutritional status; literature data show that growth may be impaired in children treated with KD. It has been observed an increment in adipose tissue and a change in the glucose tolerance in animals fed with KD, possibly resulting from insulin resistance. No data are available on VAT assessment and
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Hyatt, Tanya C. "Ethnic differences in markers of inflammation with weight loss." Thesis, Birmingham, Ala. : University of Alabama at Birmingham, 2007. https://www.mhsl.uab.edu/dt/2008r/hyatt.pdf.

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Rosa, Neto José Cesar [UNIFESP]. "Efeito do exercício exaustivo sobre aspectos inflamatórios no músculo esquelético e tecido adiposo de ratos." Universidade Federal de São Paulo (UNIFESP), 2010. http://repositorio.unifesp.br/handle/11600/9371.

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Made available in DSpace on 2015-07-22T20:49:55Z (GMT). No. of bitstreams: 0 Previous issue date: 2010-04-28<br>Objetivo: Verificar o efeito inflamatório do exercício agudo exaustivo sobre diferentes depósitos de tecido adiposo branco (mesentérico e retroperitoneal), assim como, sobre músculos esqueléticos (sóleo: fibra muscular tipo I; EDL: fibra muscular tipo II). Métodos: os animais (ratos Wistars) foram submetidos a uma sessão exaustiva de exercício, após período de adaptação. Os depósitos de tecido adiposo branco, retroperitoneal e mesentérico, como, os músculos EDL e sóleo, foram reti
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