Relevant bibliographies by topics / Adipose tissue, senescence, aging, oxidative stress

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters

Academic literature on the topic 'Adipose tissue, senescence, aging, oxidative stress'

Author: Grafiati
Published: 11 March 2023
Last updated: 31 July 2025

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Journal articles on the topic "Adipose tissue, senescence, aging, oxidative stress"

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Gorwood, Jennifer, Tina Ejlalmanesh, Christine Bourgeois, et al. "SIV Infection and the HIV Proteins Tat and Nef Induce Senescence in Adipose Tissue and Human Adipose Stem Cells, Resulting in Adipocyte Dysfunction." Cells 9, no. 4 (2020): 854. http://dx.doi.org/10.3390/cells9040854.

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Background: Aging is characterized by adipose tissue senescence, inflammation, and fibrosis, with trunk fat accumulation. Aging HIV-infected patients have a higher risk of trunk fat accumulation than uninfected individuals—suggesting that viral infection has a role in adipose tissue aging. We previously demonstrated that HIV/SIV infection and the Tat and Nef viral proteins were responsible for adipose tissue fibrosis and impaired adipogenesis. We hypothesized that SIV/HIV infection and viral proteins could induce adipose tissue senescence and thus lead to adipocyte dysfunctions. Methods: Featu
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Balasubramanian, Priya, Tamas Kiss, Rafal Gulej, et al. "Accelerated Aging Induced by an Unhealthy High-Fat Diet: Initial Evidence for the Role of Nrf2 Deficiency and Impaired Stress Resilience in Cellular Senescence." Nutrients 16, no. 7 (2024): 952. http://dx.doi.org/10.3390/nu16070952.

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High-fat diets (HFDs) have pervaded modern dietary habits, characterized by their excessive saturated fat content and low nutritional value. Epidemiological studies have compellingly linked HFD consumption to obesity and the development of type 2 diabetes mellitus. Moreover, the synergistic interplay of HFD, obesity, and diabetes expedites the aging process and prematurely fosters age-related diseases. However, the underlying mechanisms driving these associations remain enigmatic. One of the most conspicuous hallmarks of aging is the accumulation of highly inflammatory senescent cells, with mo
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Schosserer, Markus, Johannes Grillari, Christian Wolfrum, and Marcel Scheideler. "Age-Induced Changes in White, Brite, and Brown Adipose Depots: A Mini-Review." Gerontology 64, no. 3 (2017): 229–36. http://dx.doi.org/10.1159/000485183.

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Aging is a time-related process of functional decline at organelle, cellular, tissue, and organismal level that ultimately limits life. Cellular senescence is a state of permanent growth arrest in response to stress and one of the major drivers of aging and age-related disorders. Senescent cells accumulate with age, and removal of these cells delays age-related disorders in different tissues and prolongs healthy lifespan. One of the most studied aging mechanisms is the accumulation of reactive oxygen species damage in cells, organs, and organisms over time. Elevated oxidative stress is also fo
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Caron, Martine, Martine Auclair, Anais Vissian, Corinne Vigouroux, and Jacqueline Capeau. "Contribution of Mitochondrial Dysfunction and Oxidative Stress to Cellular Premature Senescence Induced by Antiretroviral Thymidine Analogues." Antiviral Therapy 13, no. 1 (2008): 27–38. http://dx.doi.org/10.1177/135965350801300103.

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Objectives Treatment of HIV-infected patients is associated with early onset of aging-related comorbidities. Some of the adverse effects of antiretroviral therapy have been attributed to the mitochondrial toxicity of nucleoside reverse transcriptase inhibitors (NRTI), and it is of note that mitochondrial dysfunction and oxidative stress are involved in the aging processes. In this regard, we examined whether NRTIs could accelerate the senescence of cultured cells. Methods Human fibroblasts were exposed to NRTIs from culture passage 1 to 14. Cytochrome c-oxidase (COX) subunits 2 and 4, mitochon
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Nandini Mode and Chaitanya B. "Senescence-Associated Secretory Phenotype (SASP) in Diabetes." Journal of Pharma Insights and Research 3, no. 1 (2025): 205–11. https://doi.org/10.69613/qjzz6g14.

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Cellular senescence represents a critical biological process characterized by permanent cell cycle arrest and distinct metabolic alterations. The senescence-associated secretory phenotype (SASP) has emerged as a fundamental mediator linking cellular aging to various pathological conditions, including diabetes mellitus. Recent investigations have unveiled the intricate relationship between SASP and pancreatic β-cell dysfunction, insulin resistance, and diabetic complications. The accumulation of senescent cells in pancreatic islets correlates with diminished insulin production and secretion, wh
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Russo, Lara, Serena Babboni, Maria Grazia Andreassi, et al. "Treating Metabolic Dysregulation and Senescence by Caloric Restriction: Killing Two Birds with One Stone?" Antioxidants 14, no. 1 (2025): 99. https://doi.org/10.3390/antiox14010099.

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Cellular senescence is a state of permanent cell cycle arrest accompanied by metabolic activity and characteristic phenotypic changes. This process is crucial for developing age-related diseases, where excessive calorie intake accelerates metabolic dysfunction and aging. Overnutrition disturbs key metabolic pathways, including insulin/insulin-like growth factor signaling (IIS), the mammalian target of rapamycin (mTOR), and AMP-activated protein kinase. The dysregulation of these pathways contributes to insulin resistance, impaired autophagy, exacerbated oxidative stress, and mitochondrial dysf
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Kornicka, K., R. Walczak, A. Mucha, and K. Marycz. "Released from ZrO2/SiO2 coating resveratrol inhibits senescence and oxidative stress of human adipose-derived stem cells (ASC)." Open Chemistry 16, no. 1 (2018): 481–95. http://dx.doi.org/10.1515/chem-2018-0039.

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AbstractThe rapid aging of the population results in increased number of metabolic and degenerative disorders, especially in the elderly.Thus, a novel approach in the fields of orthopedic and reconstructive surgery for bone regeneration is strongly desirable. A new perspective in the therapy of bone fractures is tissue engineering which combines living cells with biomaterials to develop modern substitutes that can restore tissue functions. Metallic biomaterials, including stainless steel and pure titanium, have been extensively used for the fabrication of surgical implants over decades. Chemic
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Wang, Xiujuan, Rui Liu, Chan Wei, Meihong Xu, and Yong Li. "Exogenous Nucleotides Improved the Oxidative Stress and Sirt-1 Protein Level of Brown Adipose Tissue on Senescence-Accelerated Mouse Prone-8 (SAMP8) Mice." Nutrients 14, no. 14 (2022): 2796. http://dx.doi.org/10.3390/nu14142796.

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Brown adipose tissue (BAT) is of great importance in rodents for maintaining their core temperature via non-shivering thermogenesis in the mitochondria. BAT′s thermogenic function has been shown to decline with age. The activation of adenosine 5′-monophosphate (AMP)-activated protein kinase/sirtuin-1 (AMPK/Sirt-1) is effective in regulating mitochondrial function. Exogenous nucleotides (NTs) are regulatory factors in many biological processes. Nicotinamide mononucleotide (NMN), which is a derivative of NTs, is widely known as a Sirt-1 activator in liver and muscle, but the effect of NMN and NT
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Kornicka, Katarzyna, Krzysztof Marycz, Krzysztof Andrzej Tomaszewski, Monika Marędziak, and Agnieszka Śmieszek. "The Effect of Age on Osteogenic and Adipogenic Differentiation Potential of Human Adipose Derived Stromal Stem Cells (hASCs) and the Impact of Stress Factors in the Course of the Differentiation Process." Oxidative Medicine and Cellular Longevity 2015 (2015): 1–20. http://dx.doi.org/10.1155/2015/309169.

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Human adipose tissue is a great source of autologous mesenchymal stem cells (hASCs), which are recognized for their vast therapeutic applications. Their ability to self-renew and differentiate into several lineages makes them a promising tool for cell-based therapies in different types of degenerative diseases. Thus it is crucial to evaluate age-related changes in hASCs, as the elderly are a group that will benefit most from their considerable potential. In this study we investigated the effect of donor age on growth kinetics, cellular senescence marker levels, and osteogenic and adipogenic po
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Park, Jeong Seop, Jiyuan Piao, Gabee Park, and Hyun Sook Hong. "Substance-P Restores Cellular Activity of ADSC Impaired by Oxidative Stress." Antioxidants 9, no. 10 (2020): 978. http://dx.doi.org/10.3390/antiox9100978.

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Oxidative stress induces cellular damage, which accelerates aging and promotes the development of serious illnesses. Adipose-derived stem cells (ADSCs) are novel cellular therapeutic tools and have been applied for tissue regeneration. However, ADSCs from aged and diseased individuals may be affected in vivo by the accumulation of free radicals, which can impair their therapeutic efficacy. Substance-P (SP) is a neuropeptide that is known to rescue stem cells from senescence and inflammatory attack, and this study explored the restorative effect of SP on ADSCs under oxidative stress. ADSCs were
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Dissertations / Theses on the topic "Adipose tissue, senescence, aging, oxidative stress"

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TEBON, MAELA, Mauro Zamboni, and E. Zoico. "MODULAZIONE DEI FENOMENI DI SENESCENZA CELLULARE DI ADIPOCITI E PREADIPOCITI IN VITRO." Doctoral thesis, 2019. http://hdl.handle.net/11562/995191.

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L’invecchiamento è accompagnato da una complessa rete di processi biologici caratterizzati da uno stato infiammatorio cronico basale, a sua volta correlato allo sviluppo di patologie età-associate come: aterosclerosi, osteoartriti, Alzheimer, diabete di tipo 2. Una sempre più approfondita analisi del processo di senescenza tessutospecifico potrebbe consentire l’individuazione di target terapeutici utili a modulare il processo complessivo dell’invecchiamento stesso. L’invecchiamento è causa di declino delle funzionalità sistemiche e il tessuto adiposo è uno degli organi maggiormente colpiti in
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Book chapters on the topic "Adipose tissue, senescence, aging, oxidative stress"

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Jay Sarkar, Tapash, Maiko Hermsmeier, Jessica L. Ross, and G. Scott Herron. "Genetic and Epigenetic Influences on Cutaneous Cellular Senescence." In Senescence [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.101152.

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Skin is the largest human organ system, and its protective function is critical to survival. The epithelial, dermal, and subcutaneous compartments are heterogeneous mixtures of cell types, yet they all display age-related skin dysfunction through the accumulation of an altered phenotypic cellular state called senescence. Cellular senescence is triggered by complex and dynamic genetic and epigenetic processes. A senescence steady state is achieved in different cell types under various and overlapping conditions of chronological age, toxic injury, oxidative stress, replicative exhaustion, DNA da
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