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1

Lurbe, Empar, and Elke Wühl, eds. Hypertension in Children and Adolescents. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-18167-3.

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2

Hohn, Arno R. Guidebook for pediatric hypertension. Mount Kisco, N.Y: Futura Pub. Co., 1994.

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3

Dansk hypertensions selskab. Scientific Meeting. Blood pressure in childhood and adolescence: Proceedings of the XXXIst Scientific Meeting, Danish Society of Hypertension, Glostrup Hospital, Copenhagen, February 5, 1988. Oxford: Published for Medisinsk fysiologisk forenings forlag, Oslo by Blackwell Scientific Publications, 1989.

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4

Prenatal nutrition: Clinical guidelines for nurses. White Plains, N.Y: March of Dimes Birth Defects Foundation, 1988.

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5

Loggie, Jennifer M. H. Pediatric and Adolescent Hypertension. Blackwell Science Inc, 1992.

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6

Loggie, Jennifer M. H., 1936-, ed. Pediatric and adolescent hypertension. Boston: Blackwell Scientific Publications, 1991.

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7

Cristina, Moratinos, and Cárdenas Bruno, eds. Hypertension, heredity and stroke. New York: Nova Biomedical Books, 2009.

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8

Cristina, Moratinos, and Cárdenas Bruno, eds. Hypertension, heredity, and stroke. Hauppauge, NY: Nova Science Publishers, 2009.

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9

Cristina, Moratinos, and Cárdenas Bruno, eds. Hypertension, heredity, and stroke. Hauppauge, NY: Nova Science Publishers, 2009.

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10

Gluckman, Sir Peter, Mark Hanson, Chong Yap Seng, and Anne Bardsley. Effects of maternal age on pregnancy outcomes. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198722700.003.0034.

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Maternal age on both ends of the reproductive spectrum (teenage and 35+) is associated with increased risk of adverse pregnancy outcomes, as compared with the age range from 20–34 years old. Some of the increase in pregnancy complications in older mothers is caused by underlying age-related health issues such as hypertension and diabetes, the prevalence of which increases linearly with age. The risks associated with young maternal age are more related to nutritional deficits and the fact that pregnant adolescents may still be growing themselves. Poor fetal growth often seen in adolescent pregnancies possibly results from competition for nutrients. Maternal bone loss is also a concern, as adolescent diets are commonly low in calcium and vitamin D. Pregnant adolescents may benefit from calcium supplementation to compensate for the increased need for their own bone growth and should at minimum receive vitamin D supplements, as recommended for all pregnant women.
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11

Lurbe, Empar, and Elke Wühl. Hypertension in Children and Adolescents: New Perspectives. Springer, 2019.

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12

Rascher, Wolfgang. Treatment of hypertension in children. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0219_update_001.

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Management of hypertension is dependent on the underlying cause and the magnitude of the blood pressure abnormality. Healthy behavioural changes are the primary management tool for treating primary hypertension in adolescents and other cardiovascular risk factors and obesity. In children and adolescents with renal hypertension, high blood pressure requires pharmacological treatment. There is randomized controlled trial evidence to support a blood pressure target for those with proteinuria of not higher than the 50th centile for age. The use of angiotensin-converting enzyme inhibitors is safe in patients with proteinuria, and assumed to be equally beneficial. For those without proteinuria, less stringent targets may be acceptable. Often a combination of two or three drugs is required to lower arterial blood pressure to the target blood pressures. In children and adolescents at or near end-stage renal failure, fluid removal by dialysis may be necessary to control hypertension.
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13

Jacoby, Anne Catherine. CARDIOVASCULAR CIRCADIAN RHYTHMS IN NORMOTENSIVE AND HYPERTENSIVE ADOLESCENTS. 1988.

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14

Rascher, Wolfgang. The hypertensive child. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0218_update_001.

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Arterial hypertension is a well-recognized manifestation of various forms of renal disease both in adults and children. In the paediatric age group, standards for normal blood pressure are different from adults and have now been satisfactorily defined as have standards for measuring blood pressure. The epidemic of overweight and obesity in youth is increasing the prevalence of hypertension among children and adolescents. Measurement of blood pressure requires a technique specific for different age groups of the paediatric population, is more complex and requires particular expertise. Reference values in children requires adaptation to the age and size of the child and interpretation must be related to normative values specific for age, sex, and height. Evaluation for causes of secondary hypertension and for end-organ damage is basically similar in children as in adults. This chapter discusses measuring blood pressure, blood pressure standards, definition, classification, clinical presentation, and diagnostic approach to hypertension in children.
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15

National High Blood Pressure Education Program. and National High Blood Pressure Education Program. Working Group on Hypertension Control in Children and Adolescents., eds. Update on the task force report (1987) on high blood pressure in children and adolescents: A working group report from the National High Blood Pressure Education Program. [Bethesda, Md.?]: National Institutes of Health, National Heart, Lung, and Blood Institute, 1996.

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16

J, Filer Lloyd, Lauer Ronald M. 1930-, Luepker Russell V, and International Conference on the Prevention of Atherosclerosis and Hypertension Beginning in Youth (2nd : 1992 : Orlando, Fla.), eds. Prevention of atherosclerosis and hypertension beginning in youth. Philadelphia: Lea & Febiger, 1994.

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17

Levy, David. Adolescence and emerging adulthood. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198766452.003.0009.

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Adolescence and emerging childhood forms an increasing proportion of the lifespan of urbanized individuals. Glycaemic control worsens during adolescence; physiology and psychology contribute. A1C levels peak around 9% (75 mmol/mol) before declining from late teens onwards. However, unchanging glycaemia (tracking) is common. Glycaemia has generally improved in the past 10–15 years, but significant differences between and within countries persist. Microvascular complications are prevalent at this stage, but have probably also decreased with time. During this important period, the stage can be set for premature macrovascular disease (early onset hypertension, arterial stiffening, dyslipidaemia, and smoking). Exercise reduces the risk of microvascular complications. Smoking is as common in young Type 1 patients than in the general population. Efforts at smoking cessation need reinforcing. Glycaemic control during university does not improve. Transition from paediatric to adult diabetes services is often unsatisfactory; clinics should implement simple procedures focusing on accessibility, flexibility, and improved communications.
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18

National Heart, Lung, and Blood Institute, ed. Update on the task force report (1987) on high blood pressure in children and adolescents: A working group paper from the National High Blood Pressure Education Program. [Bethesda, Md.?]: National Institutes of Health, National Heart, Lung, and Blood Institute, 1996.

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