Relevant bibliographies by topics / Adrenergic nerve

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Adrenergic nerve'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Adrenergic nerve"

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Powell, Cynthia C., and Charles L. Martin. "Distribution of cholinergic and adrenergic nerve fibers in the lacrimal glands of dogs." American Journal of Veterinary Research 50, no. 12 (1989): 2084–88. https://doi.org/10.2460/ajvr.1989.50.12.2084.

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SUMMARY The distribution of postganglionic autonomic nerve fibers in the lacrimal gland and gland of the third eyelid of dogs was studied by use of histochemical techniques. Adrenergic nerve distribution was identified by use of the sucrose-potassium phosphate-glyoxylic acid technique. A loose network of adrenergic nerves was found throughout the interstitium around acini and blood vessels and in vessel walls. Acetylcholinesterase staining was used to identify cholinergic nerve fibers. A cholinergic distribution pattern around acini and blood vessels similar to the adrenergic pattern was found
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Noh, Mi, Hee-Seong Jang, Jinu Kim, and Babu Padanilam. "Renal Sympathetic Nerve-Derived Signaling in Acute and Chronic Kidney Diseases." International Journal of Molecular Sciences 21, no. 5 (2020): 1647. http://dx.doi.org/10.3390/ijms21051647.

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The kidney is innervated by afferent sensory and efferent sympathetic nerve fibers. Norepinephrine (NE) is the primary neurotransmitter for post-ganglionic sympathetic adrenergic nerves, and its signaling, regulated through adrenergic receptors (AR), modulates renal function and pathophysiology under disease conditions. Renal sympathetic overactivity and increased NE level are commonly seen in chronic kidney disease (CKD) and are critical factors in the progression of renal disease. Blockade of sympathetic nerve-derived signaling by renal denervation or AR blockade in clinical and experimental
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Pitetti, K. H., G. A. Iwamoto, J. H. Mitchell, and G. A. Ordway. "Stimulating somatic afferent fibers alters coronary arterial resistance." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 256, no. 6 (1989): R1331—R1339. http://dx.doi.org/10.1152/ajpregu.1989.256.6.r1331.

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We used a constant flow preparation to study the changes in left circumflex coronary arterial (LCCA) pressure and resistance evoked by electrical stimulation of branches of muscle, cutaneous, and mixed nerves in the hindlimb of anesthetized dogs. Stimulation (20 Hz) of all three nerve types at 20, 70, 100, and 200 times the voltage threshold that evoked compound action potentials significantly (P less than 0.05) increased LCCA resistance. Stimulation at three and five times threshold had no effect on this same variable. Cooling the nerve to 2-4 degrees C, temperatures that block myelinated ner
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Tangsucharit, Panot, Shingo Takatori, Pengyuan Sun, et al. "Do cholinergic nerves innervating rat mesenteric arteries regulate vascular tone?" American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 303, no. 11 (2012): R1147—R1156. http://dx.doi.org/10.1152/ajpregu.00317.2012.

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Vascular blood vessels have various types of cholinergic acetylcholine receptors (AChR), but the source of ACh has not been confirmed. Perivascular adrenergic nerves and nonadrenergic calcitonin gene-related peptide (CGRP)-containing (CGRPergic) nerves innervate rat mesenteric arteries and regulate vascular tone. However, function of cholinergic innervation remains unknown. The present study investigated cholinergic innervation by examining effects of cholinesterase inhibitor (neostigmine), a muscarinic AChR antagonist (atropine), and a nicotinic AChR antagonist (hexamethonium) on adrenergic n
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Amano, Tatsuro, Naoto Fujii, Yoshimitsu Inoue, and Narihiko Kondo. "Cutaneous adrenergic nerve blockade attenuates sweating during incremental exercise in habitually trained men." Journal of Applied Physiology 125, no. 4 (2018): 1041–50. http://dx.doi.org/10.1152/japplphysiol.00370.2018.

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It remains unknown whether cutaneous adrenergic nerves functionally contribute to sweat production during exercise. This study examined whether cutaneous adrenergic nerve blockade attenuates sweating during incremental exercise, specifically in habitually trained individuals. Accordingly, 10 habitually trained and 10 untrained males (V̇o2max: 56.7 ± 5.4 and 38.9 ± 6.7 ml·kg−1·min−1, respectively; P < 0.001) performed incremental semirecumbent cycling (20 W/min) until exhaustion. Sweat rates (ventilated capsule) were measured at two bilateral forearm skin sites on which either 10 mM bretyliu
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Kubes, P., M. Melinyshyn, K. Nesbitt, S. M. Cain, and C. K. Chapler. "Participation of alpha 2-adrenergic receptors in neural vascular tone of canine skeletal muscle." American Journal of Physiology-Heart and Circulatory Physiology 262, no. 6 (1992): H1705—H1710. http://dx.doi.org/10.1152/ajpheart.1992.262.6.h1705.

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Studies were carried out in anesthetized, paralyzed, and ventilated dogs to determine whether postsynaptic alpha 2-adrenergic receptors participated in neurally mediated vascular tone in skeletal muscle. Hindlimb skeletal muscle resistance (RL) and blood flow (QL) were determined before, during, and after reversible cold block of the sciatic nerve. This sequence of observations was repeated 30 min after blockade of alpha 1-adrenergic receptors with prazosin. Then the alpha 2-adrenergic receptors were blocked with yohimbine, and the nerve cold block was repeated. When the sciatic nerve was cold
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Cohen, RA. "Platelet 5-Hydroxytryptamine and Vascular Adrenergic Nerves." Physiology 3, no. 5 (1988): 185–89. http://dx.doi.org/10.1152/physiologyonline.1988.3.5.185.

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Platelets contain and release large amounts of 5-hydroxytryptamine that can influence the function of vascular adrenergic nerves. The immediate effect on neuronal function of low concentrations of the amine is to inhibit norepinephrine release. 5-Hydroxytryptamine may also be accumulated by the same uptake mechanism that reaccumulates norepinephrine into the adrenergic nerve endings. In diseased blood vessels where platelet aggregation occurs, 5-hydroxytryptamine may therby assume an alternative transmitter role, altering the function of the sympathetic nerves.
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Luo, Min, Gregory D. Fink, Keith J. Lookingland, John A. Morris та James J. Galligan. "Impaired function of α2-adrenergic autoreceptors on sympathetic nerves associated with mesenteric arteries and veins in DOCA-salt hypertension". American Journal of Physiology-Heart and Circulatory Physiology 286, № 4 (2004): H1558—H1564. http://dx.doi.org/10.1152/ajpheart.00592.2003.

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The present study tested the hypothesis that there is impaired function of α2-adrenergic autoreceptors and increased transmitter release from sympathetic nerves associated with mesenteric arteries and veins from DOCA-salt rats. High-performance liquid chromatography was used to measure the overflow of ATP and norepinephrine (NE) from electrically stimulated mesenteric artery and vein preparations in vitro. In sham arteries, nerve stimulation evoked a 1.5-fold increase in NE release, whereas in DOCA-salt arteries there was a 3.9-fold increase in NE release over basal levels ( P < 0.05). In c
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Danser, A. H., R. van den Ende, R. R. Lorenz, N. A. Flavahan, and P. M. Vanhoutte. "Prejunctional beta 1-adrenoceptors inhibit cholinergic transmission in canine bronchi." Journal of Applied Physiology 62, no. 2 (1987): 785–90. http://dx.doi.org/10.1152/jappl.1987.62.2.785.

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The aim of the present study was to determine in canine bronchi the effects produced by norepinephrine (released from adrenergic nerve terminals) on cholinergic neurotransmission. Electrical stimulation of canine bronchi activates cholinergic and adrenergic nerve fibers. The adrenergic neuronal blocker, bretylium tosylate, inhibited the increase in [3H]norepinephrine overflow evoked by electrical stimulation but did not prevent that caused by the indirect sympathomimetic tyramine. During blockade of the exocytotic release of norepinephrine with bretylium, the pharmacological displacement of th
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Nausch, Lydia W. M., Adrian D. Bonev, Thomas J. Heppner, Yvonne Tallini, Michael I. Kotlikoff, and Mark T. Nelson. "Sympathetic nerve stimulation induces local endothelial Ca2+ signals to oppose vasoconstriction of mouse mesenteric arteries." American Journal of Physiology-Heart and Circulatory Physiology 302, no. 3 (2012): H594—H602. http://dx.doi.org/10.1152/ajpheart.00773.2011.

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It is generally accepted that the endothelium regulates vascular tone independent of the activity of the sympathetic nervous system. Here, we tested the hypothesis that the activation of sympathetic nerves engages the endothelium to oppose vasoconstriction. Local inositol 1,4,5-trisphosphate (IP3)-mediated Ca2+ signals (“pulsars”) in or near endothelial projections to vascular smooth muscle (VSM) were measured in an en face mouse mesenteric artery preparation. Electrical field stimulation of sympathetic nerves induced an increase in endothelial cell (EC) Ca2+ pulsars, recruiting new pulsar sit
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Dissertations / Theses on the topic "Adrenergic nerve"

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Yen, Laurene Dao-Pei. "Sympathetic sprouting and changes in nociceptive sensory innervation in the glabrous skin of the rat hind paw following partial peripheral nerve injury." Thesis, McGill University, 2007. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=101873.

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Previous studies have suggested that sympathetic sprouting in the periphery may contribute to the development and persistence of sympathetically-maintained pain in animal models of neuropathic pain. The purpose of this thesis was to examine morphological changes in the cutaneous innervation in rats after chronic constriction injury (CCI) to the sciatic nerve. More specifically, this study addresses the question of whether sympathetic fibres sprout de novo into the upper dermis of the rat hindpaw skin after CCI of the sciatic nerve. We also determined changes in peptidergic sensory innervation
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Welsh, Crystal Marie. "Immunohistochemical Identification of Dopaminergic and Adrenergic Nerves in the Equine Ovary." OpenSIUC, 2010. https://opensiuc.lib.siu.edu/theses/210.

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AN ABSTRACT OF THE THESIS OF Crystal Marie Welsh, for the Masters of Science degree in Equine Reproductive Physiology, presented on October 6, 2009, at Southern Illinois University Carbondale. TITLE: IMMUNOHISTOCHEMICAL IDENTIFICATION OF DOPAMINERGIC AND ADRENERGIC NERVES IN THE EQUINE OVARY MAJOR PROFESSOR: Dr. Sheryl S. King Dopamine (DA) appears to play a role in seasonal reproduction in the mare. We hypothesize that DA is delivered to the ovary by nerves. The objective of this study was to use immunohistochemical (IHC) localization to identify ovarian dopaminergic and adrenergic nerve
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McCarron, John Gerald. "Factors influencing transmitter release from the adrenergic nerves in bovine mesenteric lymphatic vessels." Thesis, University of Ulster, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.328210.

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Belvisi, Maria Gabriela. "Excitatory non-adrenergic non-cholinergic nerves and their modulation in guinea-pig airways." Thesis, Imperial College London, 1990. http://hdl.handle.net/10044/1/47771.

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Monteiro, Edison Daniel Schneider. "Quantificação digital da imunoexpressão de receptores adrenérgicos e terminações nervosas no detrusor de portadores da síndrome de prune belly." Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/5/5153/tde-28052008-143257/.

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INTRODUÇÃO: A síndrome de prune belly (PBS) é caracterizada por uma tríade com flacidez da parede abdominal, criptorquidia bilateral e malformações do trato urinário que compreende bexiga de capacidade aumentada, com complacência elevada, hipossensibilidade, hipocontratilidade, com divertículo ou fístula uracal e resíduo pós-miccional elevado. Alguns autores recomendam tratamento clínico, porém outros propõe correção cirúrgica, com reconstrução da via urinária incluindo ureteroplastia e cistoplastia redutoras, orquidopexia e abdominoplastia. Mesmo após a cirurgia, alguns doentes necessitam de
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Books on the topic "Adrenergic nerve"

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Shali͡apina, V. G. Adrenergicheskai͡a innervat͡sii͡a matki. Nauka, Leningradskoe otd-nie, 1988.

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McCarron, John Gerald. Factors influencing transmitter release from the adrenergic nerves in bovine mesenteric lymphatic vessels. The author], 1987.

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Agarwal, Deepti, Ifeyinwa C. Ifeanyi, and Mercy A. Udoji. Intrathecal Drug Delivery Systems. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190271787.003.0030.

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Intrathecal drug delivery (ITDD), while initially intended for terminal oncology patients suffering from cancer pain, is currently widely used for chronic nonmalignant pain states. Before intrathecal drug delivery device (IDDD) implantation, patients with nonmalignant chronic pain must be screened for psychologic, behavioral, and medical etiologies for their pain, in addition to having a documented failure of maximal medical therapy and a successful intrathecal drug trial. Classes of drugs used for intrathecal therapy include opioids, local anesthetics, adrenergic agonists, and NMDA receptor a
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Book chapters on the topic "Adrenergic nerve"

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Trendelenburg, U. "The Dynamics of Adrenergic Nerve Endings." In Adrenergic System and Ventricular Arrhythmias in Myocardial Infarction. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74317-7_5.

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Magnon, Claire. "The Adrenergic Nerve Network in Cancer." In Advances in Experimental Medicine and Biology. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-73119-9_15.

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Baron, Ralf, and Srinivasa N. Raja. "Role of adrenergic transmitters and receptors in nerve and tissue injury related pain." In Mechanisms and Mediators of Neuropathic Pain. Birkhäuser Basel, 2002. http://dx.doi.org/10.1007/978-3-0348-8129-6_9.

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Youdim, M. B. H. "PC12 cells as a window for the differentiation of neural crest into adrenergic nerve ending and adrenal medulla." In Recent Advances in Neuropharmacology. Springer Vienna, 1991. http://dx.doi.org/10.1007/978-3-7091-9175-0_8.

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Felten, David L., Kimberly Gibson-Berry, and J. H. David Wu. "Innervation of Bone Marrow by Tyrosine Hydroxylase-Immunoreactive Nerve Fibers and Hemopoiesis-Modulating Activity of a β-Adrenergic Agonist in Mouse." In Molecular Biology of Hematopoiesis 5. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-0391-6_76.

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Baker, D. M. "The Influence of Hypothyroidism on the Rat Adrenergic Nerve Plexus: Quantitative Image Analysis of Plexus Density in Auerbach’s Plexus and the Right Atrium." In Histochemistry and Cell Biology of Autonomic Neurons and Paraganglia. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-72749-8_54.

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Langham, Maurice E. "The Sympathetic Nerve Innervation of the Eye and the Actions of the Adrenergic Neuron Transmitter Norepinephrine on Intraocular Pressure and Ocular Blood Flow." In Ischemia and Loss of Vascular Autoregulation in Ocular and Cerebral Diseases. Springer New York, 2009. http://dx.doi.org/10.1007/978-0-387-09716-9_9.

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Larsson, P. A., S. Bööj, K. Lundmark, M. Goldstein, and A. Dahlström. "Cytofluorimetric Scanning Studies on Axonal Transport in Reserpinized Adrenergic Nerves." In Histochemistry and Cell Biology of Autonomic Neurons and Paraganglia. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-72749-8_50.

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Owman, Christer, Jing-Yu Chang, and Jan Erik Hardebo. "Presence of 5-hydroxytryptamine in adrenergic nerves of the brain circulation: its role in sympathetic neurotransmission and regulation of the cerebral vessel wall." In Cardiovascular Pharmacology of 5-Hydroxytryptamine. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-0479-8_16.

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Dell’italia, Lours J., and Jeffrey L. Ardell. "Sympathetic Nervous System in the Evolution of Heart Failure." In Basic and Clinical Neurocardiology. Oxford University PressNew York, NY, 2004. http://dx.doi.org/10.1093/oso/9780195141290.003.0012.

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Abstract In response to a variety of stresses, including exercise, traumatic injury, and blood loss, the need for increased cardiac output is met by a commensurate increase in adrenergic drive. As a result, norepinephrine (NE), the primary neurotransmitter of the adrenergic system, is released from sympathetic nerve endings in the heart. Myocardial adrenergic receptor signaling path­ ways are activated, resulting in increases in heart rate, rate of electrical conduction, and force of contraction. Early sympathetic neuronal activation in cardiac injury and/or hemodynamic overload increases vent
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Conference papers on the topic "Adrenergic nerve"

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Enciu, Valeriu, and Sergiu Didoruc. "The adrenergic and cholinergic nervous components in the fibrous formations of the autopodia in cattle." In Simpozion Ştiinţific Internaţional "50 ani de învăţământ superior medical veterinar din Republica Moldova". Technical University of Moldova, 2025. https://doi.org/10.52326/ismv2024.23.

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Fibrous formations of the autopodium in cattle have a neuro-vascular apparatus of high capacity for adjustment, which included nerve fibers of vegetative origin. Using histochemical methods allows the differentiated study of components whish innervates the bodies and establish functional membership of nerve fibers, by highlighting the catecholamine and acetylcholinesterase activity. Selective detection of nervous conductors in the fibrous formations of the autopodium in cattle, with adrenergic components, was done by histofluorescence method. To outlining the cholinergic component of the veget
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Perry, C., R. Gao, X. Peng, et al. "Adrenergic Nerve-Associated Lung Fibrosis Is Driven by Myeloid Netrin-1." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a2410.

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Lelkes, Peter I., and Brian R. Unsworth. "Cellular Signaling Mechanisms Involved in the 3-Dimensional Assembly and Differentiation of PC12 Pheochromocytoma Cells Under Simulated Microgravity in NASA Rotating Wall Vessel Bioreactors." In ASME 1998 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1998. http://dx.doi.org/10.1115/imece1998-0791.

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Abstract Rotating Wall Vessel (RWV) Bioreactors simulate microgravity and facilitate 3-D tissue-like assembly through spatial co-localization and cell-cell interactions. This unique cell culture venue is well suited to assess the role of micro-environmental cues in the assembly and tissue-specific differentiation of cells in culture. Our long term goal is to use RWV Bioreactors for generating functional neuroendocrine 3-D constructs which may be useful as clinical replacement tissue in treating neurodegenerative diseases. As a model we are using PC12 pheochromocytoma cells, a bipotential rat a
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Renz, Bernhard W., Marina Macchini, Yoku Hayakawa та ін. "Abstract 5111: A novel β 2 adrenergic-nerve growth factor feed forward loop promotes pancreatic cancer". У Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-5111.

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SUKUL, N. C., S. GHOSH, and S. P. SINHA BABU. "HIGH DILUTION EFFECTS OF STRYCHNOS NUXVOMICA L ON HYPOTHALAMIC NEURONS AND ADRENERGIC NERVE ENDINGS OF ALCOHOLIC RATS." In Proceedings of the International School of Biophysics. WORLD SCIENTIFIC, 1998. http://dx.doi.org/10.1142/9789812816887_0022.

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Clemmer, John S., W. Andrew Pruett, and Robert L. Hester. "Simulating Baroreflex Activation Therapy for the Treatment of Heart Failure with Preserved Ejection Fraction." In 2022 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2022. http://dx.doi.org/10.1115/dmd2022-1043.

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Abstract Clinical trials demonstrate baroreflex activation therapy (BAT) reduces LV mass and blood pressure (BP) in hypertensive patients and in patients with hypertensive heart failure with preserved ejection fraction (HFpEF). It is thought that high sympathetic nerve activity (SNA) in the heart plays a role in the disease progression seen in these patients. However, the impact of BAT on hemodynamics, cardiac SNA, and disease progression during HFpEF is unknown. In the present study, we used HumMod, a large physiology model to predict the time-dependent changes of BAT during HFpEF. Our result
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Bult, H., H. Fret, A. G. Herman, F. Jordaens, F. A. M. Peeters, and T. J. Verbeuren. "ENDOTHELIUM CURTAILS THE TRANSFER OF PLATELET SEROTONIN INTO THE BLOOD VESSEL WALL." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643795.

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Blood platelets are the principal source of serotonin (5-hydroxytryptamine, 5HT) in the circulation. As it is assumed that 5HT release by activated platelets can influence vascular tone, we investigated whether platelet derived 5HT can penetrate from the lumen into the vessel wall. Venous blood was collected from pentobarbitone anaesthetized dogs on 5.8 mM EDTA, platelet rich plasma was prepared and incubated with 3H-5HT and 14c-adenine. After washing, a platelet suspension (PS, 108/ml) was made in Ca2+ free Krebs'. A segment of each saphenous vein was removed and de-endothelialized for about
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Zahalka, Ali, Anna Arnal-Estape, Maria Maryanovich, Fumio Nakahara, Cristian Cruz, and Paul S. Frenette. "Abstract 1821: Sympathetic nerves regulate a metabolic switch promoting angiogenesis through adrenergic signaling in prostate cancer." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-1821.

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Thom, S., A. Hughes, G. Martin, P. Goldberg, and P. Server. "VASODILATOR PEPTIDES - CGRP AND VIP." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643720.

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The two peptides calcitonin gene related peptide (C6RP) and vasoactive intestinalpeptide (VIP) are widely distributed in animal species including man and a number ofdiverse actions of the peptides have been described [1,2]. They share vasoactive properties [3,4] and may have important functions as neurotransmitters in a non-adrenergic, non-cholinergic nervous system [5]. VIP has been located in perivascular nerves supplying several tissues and is co-stored with acetylcholine in the parasympathetic system [6]. CGRP has also been widely identified in the nervous system, the cardiovascular system
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