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Relevant bibliographies by topics / Adrenergic stress

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Academic literature on the topic 'Adrenergic stress'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Adrenergic stress"

1

Wong, Dona L., T. C. Tai, David C. Wong-Faull, Robert Claycomb, and Richard Kvet��ansk��. "Adrenergic Responses to Stress." Annals of the New York Academy of Sciences 1148, no. 1 (2008): 249–56. http://dx.doi.org/10.1196/annals.1410.048.

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Nakamori, T., A. Morimoto, K. Morimoto, N. Tan, and N. Murakami. "Effects of alpha- and beta-adrenergic antagonists on rise in body temperature induced by psychological stress in rats." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 264, no. 1 (1993): R156—R161. http://dx.doi.org/10.1152/ajpregu.1993.264.1.r156.

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We investigated the effects of intraperitoneal injection of alpha- and beta-adrenergic antagonists on psychological stress-induced responses in free-moving rats. Psychological stress was induced by immersion in 2-cm-deep water. The intraperitoneal injection of the alpha-adrenergic blocker, phentolamine (10 mg/kg), attenuated the stress-induced rise in body temperature and hypertension but enhanced tachycardia. In contrast, intraperitoneal injection of the beta-adrenergic blocker, propranolol (1 mg/kg), suppressed tachycardia but had no effect on rise in body temperature and hypertension during

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Haustein, U. F. "Adrenergic urticaria and adrenergic pruritus." Acta Dermato-Venereologica 70, no. 1 (1990): 82–84. http://dx.doi.org/10.2340/00015555708284.

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We report here on 2 patients with adrenergic urticaria and adrenergic pruritus, respectively. The lesions and features developed during phases of stress and during the attacks were associated with an increase in the plasma concentrations of noradrenalin, adrenalin and prolactin. The dopamine plasma level was elevated only in the case of adrenergic urticaria. The symptoms could be reproduced by intradermal injection of adrenalin and noradrenalin and treated successfully with propanolol, a blocker of beta-adrenergic receptors. Adrenergic urticaria is a rare but distinct entity, which has to be s

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Suleymanoglu, Yilmaz, Dimitar Bakalov, Zafer Sabit, Vlayko Vodenicharov, Radka Tafradjiiska-Hadjiolova, and Hristina Nocheva. "The endogenous cannabinoid and the adrenergic systems in modulation of stress-response." Pharmacia 71, no. () (2024): 1–8. https://doi.org/10.3897/pharmacia.71.e115659.

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In our modern, fast-paced society, excessive stress (or distress) is a major risk factor for developing a plethora of diseases. There are several neuromediatory systems in the brain that regulate the response to stress, including the adrenergic and endocannabinoid systems. In our experiments, we study the effects of the endocannabinoid system on the restrain stress-induced analgesia (r-SIA). The experiments were done on male Wistar rats. The animals were confined in special restrainers for a period of one hour. The animals were treated with Clonidine (at 4 mg/kg) – a prototypical α2-agonist; Y

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Bryan, R. M. "Cerebral blood flow and energy metabolism during stress." American Journal of Physiology-Heart and Circulatory Physiology 259, no. 2 (1990): H269—H280. http://dx.doi.org/10.1152/ajpheart.1990.259.2.h269.

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Many, but not all, stressful events are accompanied by increases in cerebral blood flow and/or energy metabolism. The stressful events include pharmacological paralysis, footshock, conditioned fear, hypotension, hypoglycemia, hypoxia, noise, and ethanol withdrawal. These increases are significant because 1) all brain regions are often affected, i.e., certain stressful events have global effects on cerebral blood flow and energy metabolism; and 2) various stressful events appear to have a common adrenergic mechanism for increasing cerebral blood flow and energy metabolism. The adrenergic mechan

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Okajima, Masaki, Masayuki Takamura, Philippe Véquaud, Robert Parent та Michel Lavallée. "β-Adrenergic receptor blockade impairs NO-dependent dilation of large coronary arteries during exercise". American Journal of Physiology-Heart and Circulatory Physiology 284, № 2 (2003): H501—H510. http://dx.doi.org/10.1152/ajpheart.00419.2002.

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Shear stress-dependent nitric oxide (NO) formation prevents immoderate vascular constriction. We examined whether shear stress-dependent NO formation limits exercise-induced coronary artery constriction after β-adrenergic receptor blockade in dogs. Control exercise led to increases ( P < 0.01) in coronary blood flow (CBF) by 38 ± 5 ml/min from 41 ± 5 ml/min and in the external diameter of epicardial coronary arteries (CD) by 0.24 ± 0.03 mm from 3.33 ± 0.20 mm. CD and shear stress were linearly related. After propranolol, CD fell ( P < 0.01) during exercise (0.08 ± 0.03 from 3.23 ± 0.19 m

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Maloyan, Alina, та Michal Horowitz. "β-Adrenergic signaling and thyroid hormones affect HSP72 expression during heat acclimation". Journal of Applied Physiology 93, № 1 (2002): 107–15. http://dx.doi.org/10.1152/japplphysiol.01122.2001.

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Heat acclimation upregulates 72-kDa heat shock protein (HSP72) and predisposes to faster activation of the heat shock response (HSR). This study investigates the role played by β-adrenergic signaling and/or plasma thyroxine level in eliciting these features by using rats undergoing 1) heat acclimation (AC; 34°C, 2 and 30 days); 2) AC with β-adrenergic blockade; 3) AC-maintained euthyroid; 4) hypothyroid; 5) hyperthyroid; and 6) controls. The hsp72 mRNA (RT-PCR) and HSP72 levels (Western blot) were measured before and after heat stress (2 h, 41°C, rectal temperature monitored). β-Adrenergic blo

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Perry, S., S. Reid, and A. Salama. "The effects of repeated physical stress on the b-adrenergic response of the rainbow trout red blood cell." Journal of Experimental Biology 199, no. 3 (1996): 549–62. http://dx.doi.org/10.1242/jeb.199.3.549.

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The effects of a 7-day period of daily physical stress (chasing until exhaustion) on the beta-adrenergic response of the rainbow trout (Oncorhynchus mykiss) red blood cell (rbc) were examined in vitro. Physical stress was associated with pronounced increases in the circulating levels of the catecholamine hormones (adrenaline and noradrenaline) measured on days 1, 3 and 7 of the stress regime. After 7 days, the numbers of high-affinity cell surface beta-adrenoceptors were reduced in the physically stressed fish when measured in vitro under conditions of normoxia (20 % reduction) or hypoxia (30

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Qiao, Guanxi, Mark J. Bucsek, Bonnie L. Hylander, and Elizabeth A. Repasky. "Adrenergic signaling impairs activation of CD8+ T cells by blocking metabolic reprogramming." Journal of Immunology 198, no. 1_Supplement (2017): 76.27. http://dx.doi.org/10.4049/jimmunol.198.supp.76.27.

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Abstract Adrenergic stress promotes tumor progression by several mechanisms. Mice housed at standard housing temperature (ST, 22°C) experience chronic adrenergic cold stress sufficient to elevate norepinephrine levels compared to mice housed at thermoneutrality (TT, 30°C) and we found that tumors grow faster at ST. We have also previously reported that the anti-tumor immune response is suppressed at ST. Tumor infiltrating CD8+ T cells from these mice have reduced expression of activation and effector function markers. These deficits are reversed by housing mice at TT which reduced adrenergic s

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Nocheva, Hristina Hristova, Eleonora Nikolaeva Encheva-Stoykova, and Evgeni Evgeniev Grigorov. "Interaction between endocannabinoids and the adrenergic system before and after stress-exposure." Pharmacia 69, no. 1 (2022): 249–54. http://dx.doi.org/10.3897/pharmacia.69.e80550.

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Cold stress-induced analgesia (c-SIA) has been evaluated in male Wistar rats injected with cannabinoid receptors type 1 and a2-adrenergic receptor agonists and antagonists in different combinations before or after stress exposure. The aim of the study was to evaluate whether the endogenous cannabinoid and the adrenergic systems influenced c-SIA, and the patterns of their potential interaction. Exogenous administration of anandamide and Clonidine together, before or after stress exposure, increased c-SIA even with differences in the time of manifestation of the effect, its duration and the degr

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Dissertations / Theses on the topic "Adrenergic stress"

1

Camp, Robert M. "Effect of Chronic Stress Exposure on Beta-adrenergic Receptor Signaling and Fear- Learning." Kent State University / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=kent1449576849.

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Manion, Sean T. "Amygdala, anxiety & alpha-1 adrenoreceptors : investigations utilizing a rodent model of traumatic stress /." Download the thesis in PDF, 2006. http://www.lrc.usuhs.mil/dissertations/pdf/Manion2006.pdf.

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Dalal, Suman, Cerrone R. Foster, Bhudev C. Das, Mahipal Singh та Krishna Singh. "β-Adrenergic Receptor Stimulation Induces Endoplasmic Reticulum Stress in Adult Cardiac Myocytes: Role in Apoptosis". Digital Commons @ East Tennessee State University, 2012. https://dc.etsu.edu/etsu-works/8572.

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Accumulation of misfolded proteins and alterations in calcium homeostasis induces endoplasmic reticulum (ER) stress, leading to apoptosis. In this study, we tested the hypothesis that β-AR stimulation induces ER stress, and induction of ER stress plays a pro-apoptotic role in cardiac myocytes. Using thapsigargin and brefeldin A, we demonstrate that ER stress induces apoptosis in adult rat ventricular myocytes (ARVMs). β-AR-stimulation (isoproterenol; 3h) significantly increased expression of ER stress proteins, such as GRP-78, Gadd-153, and Gadd-34, while activating caspase-12 in ARVMs. In mos

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Nagabhushan, Sahana. "Neural Mechanisms Underlying Stress-Induced Depression and Its Prevention." Kent State University Honors College / OhioLINK, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=ksuhonors1304462392.

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Hagenmaier, Jacob Andrew. "Investigation into an ongoing dilemma: undefined welfare implications challenging the use of β-adrenergic agonists in beef production". Diss., Kansas State University, 2016. http://hdl.handle.net/2097/35217.

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Doctor of Philosophy<br>Department of Diagnostic Medicine/Pathobiology<br>Daniel U. Thomson<br>Beta-adrenergic agonists (βAA) are administered during the final weeks of the beef production system to improve efficiency and increase meat yield. Welfare concerns linked to the administration of βAA have garnered significant attention in recent years due to anecdotal reports of increased mortality during βAA feeding periods and cattle without obvious disease or injury having difficulty walking at abattoirs being overrepresented in cattle fed βAA. Thomson et al. (2015) reported 2 events where cattle

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Chen, Xiaochuan, Amy C. Kelly, Dustin T. Yates, Antoni R. Macko, Ronald M. Lynch, and Sean W. Limesand. "Islet adaptations in fetal sheep persist following chronic exposure to high norepinephrine." BIOSCIENTIFICA LTD, 2017. http://hdl.handle.net/10150/623222.

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Complications in pregnancy elevate fetal norepinephrine (NE) concentrations. Previous studies in NE-infused sheep fetuses revealed that sustained exposure to high NE resulted in lower expression of α2-adrenergic receptors in islets and increased insulin secretion responsiveness after acutely terminating the NE infusion. In this study, we determined if the compensatory increase in insulin secretion after chronic elevation of NE is independent of hyperglycemia in sheep fetuses and whether it is persistent in conjunction with islet desensitization to NE. After an initial assessment of glucose-sti

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Zhang, Xiaobin. "Generalized anxiety disorder in the elderly : role of bio-environmental factors and genetic vulnerability." Thesis, Montpellier, 2016. http://www.theses.fr/2016MONTT036/document.

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Le trouble anxieux généralisé (TAG) est un problème majeur de santé publique. Il est fréquemment sous ou mal diagnostiqué, notamment chez les personnes âgées. Il est souvent comorbide et source d’incapacité et présente un faible taux de rémission complète. Malgré des conséquences socio-économiques et sanitaires importantes, les recherches sur les déterminants du TAG en population générale âgée restent limitées. L'objectif de cette thèse était d’étudier les caractéristiques cliniques et étiologiques du TAG à partir de l'étude prospective ESPRIT, constituée de 2259 personnes âgées de 65 ans et p

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Reader, Brenda Faye. "Social Stress Induces Immunoenhancement During Allergic Airway Inflammation and Infection." The Ohio State University, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=osu1385475903.

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Calderón, Diego Mauricio Bravo. "Expressão de receptor beta-2 adrenérgico em carcinoma espinocelular de boca e sua associação com a evolução clínica tumoral." Universidade de São Paulo, 2011. http://www.teses.usp.br/teses/disponiveis/25/25150/tde-27072011-095304/.

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Os hormônios produzidos durante o estresse e seus receptores específicos têm sido amplamente envolvidos com a progressão do câncer. O objetivo deste estudo foi avaliar a expressão dos receptores β2 adrenérgicos pelas células malignas de carcinomas espinocelulares de boca (CEC) e sua correlação com as a características clínicas, evolução e o prognóstico dos pacientes. Um total de 106 pacientes portadores de CEC de boca em estádios clínicos II, III e IV, tratados no Departamento de Cirurgia de Cabeça e Pescoço e Otorrinolaringologia do Hospital de Câncer A.C. Camargo, São Paulo, Brasil, no

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Karimigalougahi, Keyvan. "Redox regulation of eNOS and Na+-K+ pump: therapeutic implications in diabetes." Thesis, The University of Sydney, 2014. http://hdl.handle.net/2123/12135.

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Background: Oxidative stress is central in pathobiology of many diseases. The focus of this Thesis is on oxidative mechanisms involved in signaling through angiotensin II (Ang II)- and beta-1 adrenergic receptors (ARs), and their effects on the membrane proteins endothelial nitric oxide synthase (eNOS) and Na+-K+ pump in cardiovascular system. The redox mechanism investigated was glutathionylation, a reversible modification of cysteines, with effects on proteins similar to phosphorylation. As the final aim, we examined the effects of beta-3 AR stimulation on redox pathways and membrane protein

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Books on the topic "Adrenergic stress"

1

Lekas, Michael Chris. The role of adrenergic mechanisms in the regulation of glucose turnover during stress. National Library of Canada, 1995.

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2

E, Beamish Robert, Panagia Vincenzo, Dhalla Naranjan S, and International Symposium on Stress and Heart Disease (1984 : Winnipeg, Man.), eds. Pathogenesis of stress-induced heart disease: Proceedings of the International Symposium on Stress and Heart Disease, June 26-29, 1984, Winnipeg, Canada. Nijhoff, 1985.

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3

Lakhkar, Anand. Adrenergic Receptor Agonists and Antagonists in the Treatment of Post Traumatic Stress Disorder. GRIN Verlag GmbH, 2018.

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4

Frenier, Susan L. Urethral pressure response to alpha adrenergic agonist and antagonist drugs in the normal male cat. 1990.

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(Editor), R. E. Beamish, V. Panagia (Editor), and Naranjan S. Dhalla (Editor), eds. Pathogenesis of Stress-induced Heart Disease (Developments in Cardiovascular Medicine). Springer, 1985.

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6

Jani, Suni, Ryan Herringa, and Derek Hursey. Pharmacologic Treatment of Children with Trauma- and Stressor-Related Disorders. Edited by Frederick J. Stoddard, David M. Benedek, Mohammed R. Milad, and Robert J. Ursano. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190457136.003.0023.

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This chapter reviews the principles of psychopharmacological treatment for trauma and stressor-related disorders in children. It will discuss emerging psychopharmacological treatments and theories of trauma- and stress-related disorders (TSRDs) common to childhood, reactive attachment disorder, and disinhibited social engagement disorder. Although there is limited literature on the role of pharmacotherapy in treating TSRDs in children, it has been recognized that α‎- and β‎-adrenergic blocking agents, novel antipsychotic agents, non-selective serotonin reuptake inhibitor antidepressants such a

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Book chapters on the topic "Adrenergic stress"

1

Frank, S. M., S. N. Raja, L. A. Fleisher, C. Beattie, M. S. Higgins, and M. J. Breslow. "Cardiovascular and Adrenergic Manifestations of Cold Stress." In Thermal Balance in Health and Disease. Birkhäuser Basel, 1994. http://dx.doi.org/10.1007/978-3-0348-7429-8_44.

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Stepanović, S., V. M. Varagić, J. Nikolić, and R. Kvetnansky. "The Significance of the Cholinergic-Adrenergic Interactions in the Response of the Rat to Immobilization Stress." In Neuroendocrine Correlates of Stress. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-8553-0_10.

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Kvetňanský, R., and T. Torda. "Heart Adrenergic System Activity in Rats during Adaptation to Repeated Stress." In Developments in Cardiovascular Medicine. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4613-2589-5_1.

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Kelsey, Robert M. "Beta-adrenergic cardiovascular reactivity and adaptation to stress: The cardiac pre-ejection period as an index of effort." In How motivation affects cardiovascular response: Mechanisms and applications. American Psychological Association, 2012. http://dx.doi.org/10.1037/13090-002.

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"Adrenergic Stress." In The Etiopathogenesis of Coronary Heart Disease. CRC Press, 2004. http://dx.doi.org/10.1201/9781498712811-12.

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HAMNER, M., and G. ARANA. "Beta-Adrenergic Blockers1." In Encyclopedia of Stress. Elsevier, 2007. http://dx.doi.org/10.1016/b978-012373947-6/00054-4.

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Hamner, M. B., and G. W. Arana. "Beta-Adrenergic Blockers." In Encyclopedia of Stress. Elsevier, 2007. http://dx.doi.org/10.1016/b978-012373947-6.00054-4.

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Sevigny, C. P., C. Menuet, A. Y. Fong, J. K. Bassi, A. A. Connelly, and A. M. Allen. "Adrenergic Neurons in the CNS." In Stress: Neuroendocrinology and Neurobiology. Elsevier, 2017. http://dx.doi.org/10.1016/b978-0-12-802175-0.00003-6.

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RAŠKOVÁ, H., L. ČELEDA, Z. URBANOVÁ, J. VANĚČEK, and A. KUBIČEK. "STRESS, ADRENERGIC AGENTS AND INFECTION." In Receptors and Centrally Acting Drugs Pharmacokinetics and Drug Metabolism. Elsevier, 1986. http://dx.doi.org/10.1016/b978-0-08-034191-0.50048-9.

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Myers, Karyn M., Robert Claycomb, T. C. Tai, William A. Carlezon, Richard Kvetnansky, and Dona Lee Wong. "Stress, HIF1a, and Adrenergic Function." In Catecholamine Research in the 21st Century. Elsevier, 2014. http://dx.doi.org/10.1016/b978-0-12-800044-1.00198-7.

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Conference papers on the topic "Adrenergic stress"

1

Le, Caroline P., Matthew A. Pimentel, Corina Kim, et al. "Abstract A113: Chronic stress promotes lymph node metastasis through beta-adrenergic regulation of lymphangiogenesis." In Abstracts: AACR Special Conference on Advances in Breast Cancer Research: Genetics, Biology, and Clinical Applications - October 3-6, 2013; San Diego, CA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1557-3125.advbc-a113.

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Plummer, Howard, Hussein A. N. Al-Wadei, Benjamin Unger, and Hildegard M. Schuller. "Abstract 1023: Social stress modulates beta-adrenergic and potassium channel-mediated signaling in lung adenocarcinoma." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-1023.

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Khan, Danyaal, Christie Richardson, and Martin Forsberg. "Doxazosin Immediate Release as Alternative Treatment for Nightmares in Post-Traumatic Stress Disorder: A Case Report." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. http://dx.doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.34_2024.

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Nightmares and flashbacks are common debilitating symptoms of post-traumatic stress disorder (PTSD) that can disrupt daily functioning in patients. Prazosin, an alpha-1 adrenergic antagonist, has been commonly used off-label for the treatment of PTSD intrusion symptoms, although its short half-life is limiting. Doxazosin, another alpha-1 antagonist, is starting to be investigated in the treatment of PTSD-related nightmares due to its smaller side effect profile and longer half-life. In our case report, we present a case of a patient with PTSD-related nightmares who was successfully treated wit

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Dinca-Panaitescu, S., M. Ciurea, A. Achim, and R. Negoescu. "RR-independent 0.1 Hz power in QT spectrum indexes myocardial adrenergic strain by both emotional and attention stress." In Proceedings of the 19th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. 'Magnificent Milestones and Emerging Opportunities in Medical Engineering'. IEEE, 1997. http://dx.doi.org/10.1109/iembs.1997.754536.

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Bocheva, Adriana I. "Do α2-adrenergic agents modulate the effects of MIF-1 and Tyr-MIF-1 after three models of stress?" У Xth Conference Biologically Active Peptides. Institute of Organic Chemistry and Biochemistry, Academy of Sciences of the Czech Republic, 2007. http://dx.doi.org/10.1135/css200709014.

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Eng, Jason W.-L., Chelsey B. Reed, Kathleen Kokolus та ін. "Abstract B72: Environmental temperature-induced chronic stress drives therapeutic resistance in murine tumor models through β2-adrenergic receptor activation". У Abstracts: AACR Special Conference: Tumor Immunology and Immunotherapy: A New Chapter; December 1-4, 2014; Orlando, FL. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/2326-6074.tumimm14-b72.

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Leigh, Nicholas D., Kathleen M. Kokolus, Jason W.-L. Eng, et al. "Abstract B43: The degree of adrenergic stress signaling regulates the severity of graft versus host disease following allogeneic hematopoietic cell transplantation." In Abstracts: AACR Special Conference: Tumor Immunology and Immunotherapy: A New Chapter; December 1-4, 2014; Orlando, FL. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/2326-6074.tumimm14-b43.

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Hulsurkar, Mohit M., Meixiang Sang, Haiping Song, and Wenliang Li. "Abstract 4181: Chronic stress and beta adrenergic signaling promote angiogenesis and prostate cancer progression through suppressing the expression of Thrombospondin 1." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-4181.

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Moustaka, Kalliopi, Stergios Katsiougiannis, Roxane Tenta, et al. "THU0223 CHRONIC ADRENERGIC STIMULATION OF MINOR SALIVARY GLANDS OF PATIENTSWITH PRIMARY SJÖGREN’S DRIVES ER STRESS AND ACTIVATION OF THE UNFOLDED PROTEIN RESPONSE." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.1438.

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Bult, H., H. Fret, A. G. Herman, F. Jordaens, F. A. M. Peeters, and T. J. Verbeuren. "ENDOTHELIUM CURTAILS THE TRANSFER OF PLATELET SEROTONIN INTO THE BLOOD VESSEL WALL." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643795.

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Blood platelets are the principal source of serotonin (5-hydroxytryptamine, 5HT) in the circulation. As it is assumed that 5HT release by activated platelets can influence vascular tone, we investigated whether platelet derived 5HT can penetrate from the lumen into the vessel wall. Venous blood was collected from pentobarbitone anaesthetized dogs on 5.8 mM EDTA, platelet rich plasma was prepared and incubated with 3H-5HT and 14c-adenine. After washing, a platelet suspension (PS, 108/ml) was made in Ca2+ free Krebs'. A segment of each saphenous vein was removed and de-endothelialized for about

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