Relevant bibliographies by topics / AGEs-inhibitors

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Academic literature on the topic 'AGEs-inhibitors'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "AGEs-inhibitors"

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Ortega-Castro, J., M. Adrover, J. Frau, J. Donoso, and F. Muñoz. "Cu2+ complexes of some AGEs inhibitors." Chemical Physics Letters 475, no. 4-6 (2009): 277–84. http://dx.doi.org/10.1016/j.cplett.2009.05.074.

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Abudukadeer, Kuerban* Said Salama Moselhy Yaaser Q. Almulaiky Syed Shoeb Razvi Mohammed Nihal Hasan Khalid Omar Abulnaja Taha A. Kumosani Abdulrahman L.-AL-Malki. "NATURAL COMPOUNDS THAT INHIBIT PROTEIN GLYCATION: A REVIEW FOR RECENT FINDINGS." Indo American Journal of Pharmaceutical Sciences 04, no. 11 (2017): 4027–42. https://doi.org/10.5281/zenodo.1045214.

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Diabetes Mellitus is a chronic, lifelong disease currently impacting people throughout the world. A fundamental cause of Diabetes Mellitus: Non-enzymatic protein glycosylation (glycation) contributes to a group of metabolic diseases, including diabetes-associated late complications, atherosclerosis, chronic renal failure, Alzheimer’s disease and inflammatory arthritis. It has been hypothesized that inhibition of glycation may prevent the diseases associated with diabetes. Inhibitors of glycation have been explored for several decades, chiefly using in vitro models, which resulted in discovery
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Zaki, Muthanna K., Mohammed N. Abed, and Fawaz A. Alassaf. "Antidiabetic Agents and Bone Quality: A Focus on Glycation End Products and Incretin Pathway Modulations." Journal of Bone Metabolism 31, no. 3 (2024): 169–81. http://dx.doi.org/10.11005/jbm.2024.31.3.169.

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Diabetes mellitus is associated with inadequate bone health and quality and heightened susceptibility to fractures, even in patients with normal or elevated bone mineral density. Elevated advanced glycation end-products (AGEs) and a suppressed incretin pathway are among the mechanisms through which diabetes affects the bone. Accordingly, the present review aimed to investigate the effects of antidiabetic medications on bone quality, primarily through AGEs and the incretin pathway. Google Scholar, Cochrane Library, and PubMed were used to examine related studies until February 2024. Antidiabeti
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Nabi, Rabia, Sahir Sultan Alvi, Mohd Saeed, Saheem Ahmad, and Mohammad Salman Khan. "Glycation and HMG-CoA Reductase Inhibitors: Implication in Diabetes and Associated Complications." Current Diabetes Reviews 15, no. 3 (2019): 213–23. http://dx.doi.org/10.2174/1573399814666180924113442.

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Introduction: Diabetes Mellitus (DM) acts as an absolute mediator of cardiovascular risk, prompting the prolonged occurrence, size and intricacy of atherosclerotic plaques via enhanced Advanced Glycation Endproducts (AGEs) formation. Moreover, hyperglycemia is associated with enhanced glyco-oxidized and oxidized Low-Density Lipoprotein (LDL) possessing greater atherogenicity and decreased the ability to regulate HMG-CoA reductase (HMG-R). Although aminoguanidine (AG) prevents the AGE-induced protein cross-linking due to its anti-glycation potential, it exerts several unusual pharmaco-toxicolog
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S. Rahbar, Bentham Science Publisher, and Bentham Science Publisher J.L. Figarola. "Inhibitors and Breakers of Advanced Glycation Endproducts (AGEs): A Review." Current Medicinal Chemistry-Immunology, Endocrine & Metabolic Agents 2, no. 2 (2002): 135–61. http://dx.doi.org/10.2174/1568013023358889.

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Reddy, V. Prakash. "Oxidative Stress in Health and Disease." Biomedicines 11, no. 11 (2023): 2925. http://dx.doi.org/10.3390/biomedicines11112925.

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Oxidative stress, resulting from the excessive intracellular accumulation of reactive oxygen species (ROS), reactive nitrogen species (RNS), and other free radical species, contributes to the onset and progression of various diseases, including diabetes, obesity, diabetic nephropathy, diabetic neuropathy, and neurological diseases, such as Alzheimer’s disease (AD), amyotrophic lateral sclerosis (ALS), and Parkinson’s disease (PD). Oxidative stress is also implicated in cardiovascular disease and cancer. Exacerbated oxidative stress leads to the accelerated formation of advanced glycation end p
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Thomas, M., J. Baynes, S. Thorpe, and M. Cooper. "The Role of AGEs and AGE Inhibitors in Diabetic Cardiovascular Disease." Current Drug Targets 6, no. 4 (2005): 453–74. http://dx.doi.org/10.2174/1389450054021873.

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Furukawa, Masako, Tomohito Gohda, Mitsuo Tanimoto, and Yasuhiko Tomino. "Pathogenesis and Novel Treatment from the Mouse Model of Type 2 Diabetic Nephropathy." Scientific World Journal 2013 (2013): 1–8. http://dx.doi.org/10.1155/2013/928197.

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Diabetic nephropathy (DN) is the leading cause of end-stage kidney disease worldwide. However, current treatments remain suboptimal. Many factors, such as genetic and nongenetic promoters, hypertension, hyperglycemia, the accumulation of advanced glycation end products (AGEs), dyslipidemia, and albuminuria/proteinuria itself, influence the progression of this disease. It is important to determine the molecular mechanisms and treatment of this disease. The development of diabetes results in the formation of AGEs, oxidative stress, and the activation of the renin-angiotensin-aldosterone system (
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Lasker, George F., Jason H. Maley, and Philip J. Kadowitz. "A Review of the Pathophysiology and Novel Treatments for Erectile Dysfunction." Advances in Pharmacological Sciences 2010 (2010): 1–10. http://dx.doi.org/10.1155/2010/730861.

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Erectile dysfunction (ED) affects up to 50% of men between the ages of 40 and 70. Treatment with PDE-5 inhibitors is effective in the majority of men with ED. However, PDE-5 inhibitors are not effective when levels of nitric oxide (NO), the principle mediator of erection, are low. The pharmacologic actions of three new potential treatments for ED are discussed in this paper: (1) sGC stimulators/activators, (2) Rho-kinase inhibitors, and (3) sodium nitrite.
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Giancarlo, Aldini. "High resolution mass spectrometric strategies for studying AGEs inhibitors and RAGE antagonists." Free Radical Biology and Medicine 65 (September 2013): S15. http://dx.doi.org/10.1016/j.freeradbiomed.2013.08.141.

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Book chapters on the topic "AGEs-inhibitors"

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Kageyama, Hakuto. "Biological Activities of MAAs and their Applications 4: Anti-glycative Properties." In An Introduction to Mycosporine-Like Amino Acids. BENTHAM SCIENCE PUBLISHERS, 2023. http://dx.doi.org/10.2174/9789815136081123010010.

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Advanced glycation end products (AGEs) are formed by a series of chemical reactions initiated by non-enzymatic glycation reactions. In this process, the reducing sugar binds to the free amino group of the protein. The formation of AGEs that accompany the aging process is thought to be associated with various diseases such as diabetes and Alzheimer's disease. A number of inhibitors derived from synthetic compounds and natural products have been developed and evaluated to prevent the formation of AGEs. Compared to synthetic compounds, natural products are considered to be relatively safe for hum
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Khan, Rujman, Xin Yee Ooi, Matthew Parvus, Laura Valdez, and Andrew Tsin. "Advanced Glycation End Products: Formation, Role in Diabetic Complications, and Potential in Clinical Applications." In The Eye and Foot in Diabetes. IntechOpen, 2020. http://dx.doi.org/10.5772/intechopen.89408.

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Hyperglycemic conditions and disruptions to glucose-regulating pathways lead to increased formation of highly reactive aldehydes, methylglyoxal and glyoxal, which react with certain arginine and lysine residues in proteins to form advanced glycation end products (AGEs). These AGEs damage the integrity of the retinal vasculature predominantly through two mechanisms: non-receptor-mediated damage, which pertains to the interaction with extracellular matrix and its functional properties, and receptor-mediated damage through AGE interactions with their receptors (RAGE) on pericytes and Muller cells
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GHAFOOR, NAILA, ZAINAB TARIQ, RABIA AZMAT, et al. "Polycystic Ovarian Syndrome (PCOS), Underlying Factors and Potential Treatments." In Zoology: Advancements and Research Trends. FahumSci, 2024. http://dx.doi.org/10.61748/zool.2024/01.

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Polycystic ovary syndrome (PCOS) is one of the most prevalent reproductive disorders in females during their reproductive age. Every one woman out of six is diagnosed with PCOS at her reproductive stage in USA. This chapter will assess the underlying reasons as well as potential treatment options against PCOS. There are several factors underlying the development as well as the progression of PCOS. These factors include hyperandrogenism, advanced glycation end products (AGEs), insulin resistance, obesity, and oxidative stress. Oxidative stress is considered one of the fundamental factors which
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Kumar, Amrendra, and Swati Agarwal. "Microbial Products and Their Role in Soil Health and Sustainable Agriculture." In Advances in Environmental Engineering and Green Technologies. IGI Global, 2021. http://dx.doi.org/10.4018/978-1-7998-7062-3.ch007.

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Microbial products are being used from ages in known as well as unknown forms. Some common products harvested from microbes include proteins, amino acids, antibiotics, antibodies, secondary metabolites, organic acids, lipids, and so on. It also includes antivirals, polymers, surfactants, enzyme inhibitors, nutraceuticals, and many industrial and agricultural products. Moreover, sometimes the whole single celled microbes are harvested as a rich source of protein called single cell proteins. In a nutshell, all these products cover almost every economic sector like food, feed, agriculture, health
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Chaudhary, Priyanka, Sanu Diwakar, and Divya Joshi. "Addressing Glycation in Aging." In Drug Discovery and Antiaging Approaches for Human Longevity. IGI Global, 2025. https://doi.org/10.4018/979-8-3693-9730-5.ch012.

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Protein glycation, or simply glycation, is the process by which sugar binds to amino acids, producing reactive oxygen species (ROS) and free radicals. These chemicals generated by glycation play a role in oxidative stress and the formation of Advanced Glycation End Products (AGEs), further leading to disorders such as diabetes and cardiovascular disease. Synthetic substances have been studied for their ability to reverse glycation. However, the adverse effects they cause lead to the search for natural glycation inhibitors. Medicinal plants and compounds derived from them, including polyphenols
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Paley, Elena L. "Discovery of Gut Bacteria Specific to Alzheimer’s Associated Diseases Is a Clue to Understanding Disease Etiology: Meta-Analysis of Population-Based Data on Human Gut Metagenomics and Metabolomics." In Advances in Alzheimer’s Disease. IOS Press, 2022. http://dx.doi.org/10.3233/aiad220003.

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Alzheimer’s disease (AD)-associated sequence (ADAS) of cultured fecal bacteria was discovered in human gut targeted screening. This study provides important information to expand our current understanding of the structure/activity relationship of ADAS and putative inhibitors/activators that are potentially involved in ADAS appearance/disappearance. The NCBI database analysis revealed that ADAS presents at a large proportion in American Indian Oklahoman (C&A) with a high prevalence of obesity/diabetes and in colorectal cancer (CRC) patients from the US and China. An Oklahoman non-native gro
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Vormoor, H. Josef, Tobias F. Menne, and Anthony V. Moorman. "Acute lymphoblastic leukaemia." In Oxford Textbook of Medicine, edited by Chris Hatton and Deborah Hay. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0523.

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Acute lymphoblastic leukaemia (ALL) is a malignant proliferation of lymphoid blasts, most commonly of B-lineage origin. The clinical symptoms and signs are either a consequence of bone marrow failure (infections, bruising, petechiae, pallor, and tiredness) or a consequence of the uncontrolled proliferation of the blasts (lymphadenopathy, hepatosplenomegaly, and cranial nerve palsies). Its peak incidence is in young children but ALL occurs at all ages. More than 80% of all affected children are cured with modern chemotherapy, but unfortunately the outcome of adults is much worse despite some im
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Becker, Richard C., and Frederick A. Spencer. "Plaque-Stabilizing Therapies." In Fibrinolytic and Antithrombotic Therapy. Oxford University Press, 2006. http://dx.doi.org/10.1093/oso/9780195155648.003.0028.

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The association between elevated low density lipoprotein (LDL) cholesterol and atherothrombotic vascular disease is well established. Lipid-lowering therapies reduce cardiovascular events; however, the mechanism of benefit attributable to HMG CoA reductase inhibitors likely transcends lipids alone by directly or indirectly affecting inflammatory response, endothelial cell resistance, apoptosis, and progenitor cell behavior. Although collectively in the same class of drugs, statins have unique pharmacokinetic profiles. They may also differ in their ability to impact inflammatory responses, endo
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Conference papers on the topic "AGEs-inhibitors"

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Hartmann, A., M. Orfanoudaki, P. Blanchard, et al. "Secondary metabolites from marine sources as inhibitors of advanced glycation end products (AGEs) and collagenase." In 67th International Congress and Annual Meeting of the Society for Medicinal Plant and Natural Product Research (GA) in cooperation with the French Society of Pharmacognosy AFERP. © Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-3400086.

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Jakhotia, Sneha, Branislav Nastasijević, Gordana Joksić, Sreenivasa Reddy, and G. Bhanuprakash Reddy. "Gentiana lutea AND DIABETIC COMPLICATIONS." In 8th Workshop Food and Drug Safety and Qualit. Vinča Institute of Nuclear Sciences - National Institute of the Republic of Serbia, 2024. http://dx.doi.org/10.46793/8fdsq.ild1sj.

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Chronic hyperglycemia in diabetes leads to macro and microvascular complications. An increase in the aldose reductase (ALR2) activity, accumulation of sorbitol, accumulation of advanced glycation end-products (AGEs), and inflammation lead to cellular damage, ultimately resulting in complications. ALR2 inhibition has been a consideration to treat/manage diabetic complications, but most of the ALR2 inhibitors are not free of side effects. As a part of our work to find better ALR2 inhibitors, we found that extracts of Gentiana lutea exhibited ALR2 inhibitory potential in vitro. We have also disco
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Andrew, M., B. A. Paes, R. A. Milner, P. J. Powers, M. Johnston, and V. Castle. "THE POSTNATAL DEVELOPMENT OF THE COAGULATION SYSTEM IN THE PREMATURE INFANT." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643606.

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A cohort study was performed to determine the postnatal development of the coagulation system in the “healthy” premature infant. Mothers were approached for consent and a total of 132 premature infants were entered into the study. The group consisted of 64 infants with gestational ages of 34-36 weeks (prem 1) and 68 infants whose gestational age was 33 weeks or less (prem 2). Demographic information and a 2 ml blood sample were obtained on days 1, 5, 30, 90, and 180. Plasma was fractionated and stored at −70°C for batch assaying of the following tests: screening tests, PT, APTT; factor assays
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