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Journal articles on the topic 'Aging of the lens'

Author: Grafiati
Published: 8 September 2021
Last updated: 8 February 2022

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1

Glasser, Adrian. "Lens Dysfunction through Aging." Ophthalmology 114, no. 3 (March 2007): 618.e1–618.e2. http://dx.doi.org/10.1016/j.ophtha.2006.12.004.

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2

Hood, Brian D., Brett Garner, and Roger J. W. Truscott. "Human Lens Coloration and Aging." Journal of Biological Chemistry 274, no. 46 (November 12, 1999): 32547–50. http://dx.doi.org/10.1074/jbc.274.46.32547.

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3

Pokorny, Joel, Vivianne C. Smith, and Margaret Lutze. "Aging of the human lens." Applied Optics 26, no. 8 (April 15, 1987): 1437. http://dx.doi.org/10.1364/ao.26.001437.

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4

Mohanty, Bimal Prasanna, Soma Bhattacharjee, Prasenjit Paria, Arabinda Mahanty, and Anil Prakash Sharma. "Lipid Biomarkers of Lens Aging." Applied Biochemistry and Biotechnology 169, no. 1 (November 21, 2012): 192–200. http://dx.doi.org/10.1007/s12010-012-9963-6.

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5

Sharma, K. Krishna, and Puttur Santhoshkumar. "Lens aging: Effects of crystallins." Biochimica et Biophysica Acta (BBA) - General Subjects 1790, no. 10 (October 2009): 1095–108. http://dx.doi.org/10.1016/j.bbagen.2009.05.008.

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6

Smith, Christine. "Aging Through a Queer Lens." Gerontologist 59, no. 1 (January 9, 2019): 192–93. http://dx.doi.org/10.1093/geront/gny163.

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7

McMahon, Mary, Craig Forester, and Rochelle Buffenstein. "Aging through an epitranscriptomic lens." Nature Aging 1, no. 4 (April 2021): 335–46. http://dx.doi.org/10.1038/s43587-021-00058-y.

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8

Faranda, Adam P., Mahbubul H. Shihan, Yan Wang, and Melinda K. Duncan. "The aging mouse lens transcriptome." Experimental Eye Research 209 (August 2021): 108663. http://dx.doi.org/10.1016/j.exer.2021.108663.

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9

Croft, Mary Ann, Paul L. Kaufman, Kathryn S. Crawford, Michael W. Neider, Adrian Glasser, and Laszlo Z. Bito. "Accommodation dynamics in aging rhesus monkeys." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 275, no. 6 (December 1, 1998): R1885—R1897. http://dx.doi.org/10.1152/ajpregu.1998.275.6.r1885.

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Accommodation, the mechanism by which the eye focuses on near objects, is lost with increasing age in humans and monkeys. This pathophysiology, called presbyopia, is poorly understood. We studied aging-related changes in the dynamics of accommodation in rhesus monkeys aged 4–24 yr after total iridectomy and midbrain implantation of an electrode to permit visualization and stimulation, respectively, of the eye’s accommodative apparatus. Real-time video techniques were used to capture and quantify images of the ciliary body and lens. During accommodation in youth, ciliary body movement was biphasic, lens movement was monophasic, and both slowed as the structures approached their new steady-state positions. Disaccommodation occurred more rapidly for both ciliary body and lens, but with longer latent period, and slowed near the end point. With increasing age, the amplitude of lens and ciliary body movement during accommodation declined, as did their velocities. The latent period of lens and ciliary body movements increased, and ciliary body movement became monophasic. The latent period of lens and ciliary body movement during disaccommodation was not significantly correlated with age, but their velocity declined significantly. The age-dependent decline in amplitude and velocity of ciliary body movements during accommodation suggests that ciliary body dysfunction plays a role in presbyopia. The age changes in lens movement could be a consequence of increasing inelasticity or hardening of the lens, or of age changes in ciliary body motility.
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10

Chhunchha, Bhavana, Eri Kubo, and Dhirendra P. Singh. "Clock Protein Bmal1 and Nrf2 Cooperatively Control Aging or Oxidative Response and Redox Homeostasis by Regulating Rhythmic Expression of Prdx6." Cells 9, no. 8 (August 8, 2020): 1861. http://dx.doi.org/10.3390/cells9081861.

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Many disorders of aging, including blinding-diseases, are associated with deficiency of brain and muscle arnt-like protein 1 (Bmal1) and, thereby, dysregulation of antioxidant-defense pathway. However, knowledge is limited regarding the role of Bmal1 regulation of antioxidant-pathway in the eye lens/lens epithelial cells (LECs) at the molecular level. We found that, in aging human (h)LECs, a progressive decline of nuclear factor erythroid 2-related factor 2 (Nrf2)/ARE (antioxidant response element)-mediated antioxidant genes was connected to Bmal1-deficiency, leading to accumulation of reactive oxygen species (ROS) and cell-death. Bmal1-depletion disrupted Nrf2 and expression of its target antioxidant genes, like Peroxiredoxin 6 (Prdx6). DNA binding and transcription assays showed that Bmal1 controlled expression by direct binding to E-Box in Prdx6 promoter to regulate its transcription. Mutation at E-Box or ARE reduced promoter activity, while disruption of both sites diminished the activity, suggesting that both sites were required for peak Prdx6-transcription. As in aging hLECs, ROS accumulation was increased in Bmal1-deficient cells and the cells were vulnerable to death. Intriguingly, Bmal1/Nrf2/Prdx6 and PhaseII antioxidants showed rhythmic expression in mouse lenses in vivo and were reciprocally linked to ROS levels. We propose that Bmal1 is pivotal for regulating oxidative responses. Findings also reveal a circadian control of antioxidant-pathway, which is important in combating lens/LECs damage induced by aging or oxidative stress.
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11

Alió, Jorge L., Patricia Schimchak, Herminio P. Negri, and Robert Montés-Micó. "Crystalline Lens Optical Dysfunction through Aging." Ophthalmology 112, no. 11 (November 2005): 2022–29. http://dx.doi.org/10.1016/j.ophtha.2005.04.034.

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12

Claman, Emily, and Caitlin Condell. "Aging Through a Different Lens (527)." Journal of Pain and Symptom Management 41, no. 1 (January 2011): 269. http://dx.doi.org/10.1016/j.jpainsymman.2010.10.175.

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13

Hardy, Joseph L., Christina M. Frederick, Paul Kay, and John S. Werner. "Color Naming, Lens Aging, and Grue." Psychological Science 16, no. 4 (April 2005): 321–27. http://dx.doi.org/10.1111/j.0956-7976.2005.01534.x.

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Many languages without separate terms for green and blue are or were spoken in locations receiving above-average exposure to ultraviolet-B (UV-B) radiation. It has been proposed that this correlation is caused by premature lens aging. This conclusion was supported by an experiment in which younger observers used the term “blue” less often when they described simulated paint chips filtered through the equivalent of an older observer's lens—removing much short-wavelength light—than when they described the unfiltered versions of the same paint chips. Some stimuli that were called “blue” without simulated aging were called “green” when filtered. However, in the experiment reported here, we found that the proportion of “blue” color-name responses did not differ between younger subjects and older observers with known ocular media optical densities. Color naming for stimuli that were nominally green, blue-green, or blue was virtually identical for older and younger observers who viewed the same (unfiltered) stimuli. Our results are inconsistent with the lens-brunescence hypothesis.
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14

Wishart, Tayler F. L., Mary Flokis, Daisy Y. Shu, Shannon J. Das, and Frank J. Lovicu. "Hallmarks of lens aging and cataractogenesis." Experimental Eye Research 210 (September 2021): 108709. http://dx.doi.org/10.1016/j.exer.2021.108709.

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15

Tipperman, R. "Straylight Effects with Aging and Lens Extraction." Yearbook of Ophthalmology 2008 (January 2008): 15–16. http://dx.doi.org/10.1016/s0084-392x(08)79188-3.

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16

Lamb, Sarah. "Interrogating Healthy/Successful Aging: An Anthropologist's Lens." General Anthropology 26, no. 2 (September 2019): 1–9. http://dx.doi.org/10.1111/gena.12059.

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17

Glasser, A. "Presbyopia and aging in the crystalline lens." Journal of Vision 3, no. 12 (March 28, 2010): 22. http://dx.doi.org/10.1167/3.12.22.

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18

Van Den Berg, Thomas J. T. P., L. J. (René) Van Rijn, Ralph Michael, Christian Heine, Tanja Coeckelbergh, Christian Nischler, Helmuth Wilhelm, et al. "Straylight Effects with Aging and Lens Extraction." American Journal of Ophthalmology 144, no. 3 (September 2007): 358–63. http://dx.doi.org/10.1016/j.ajo.2007.05.037.

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19

Wei, Zongbo, Caili Hao, Jingru Huangfu, Ramkumar Srinivasagan, Xiang Zhang, and Xingjun Fan. "Aging lens epithelium is susceptible to ferroptosis." Free Radical Biology and Medicine 167 (May 2021): 94–108. http://dx.doi.org/10.1016/j.freeradbiomed.2021.02.010.

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20

Minaeva, Olga, Srikant Sarangi, Danielle M. Ledoux, Juliet A. Moncaster, Douglas S. Parsons, Kevin J. Washicosky, Caitlin A. Black, et al. "In Vivo Quasi-Elastic Light Scattering Eye Scanner Detects Molecular Aging in Humans." Journals of Gerontology: Series A 75, no. 9 (June 9, 2020): e53-e62. http://dx.doi.org/10.1093/gerona/glaa121.

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Abstract The absence of clinical tools to evaluate individual variation in the pace of aging represents a major impediment to understanding aging and maximizing health throughout life. The human lens is an ideal tissue for quantitative assessment of molecular aging in vivo. Long-lived proteins in lens fiber cells are expressed during fetal life, do not undergo turnover, accumulate molecular alterations throughout life, and are optically accessible in vivo. We used quasi-elastic light scattering (QLS) to measure age-dependent signals in lenses of healthy human subjects. Age-dependent QLS signal changes detected in vivo recapitulated time-dependent changes in hydrodynamic radius, protein polydispersity, and supramolecular order of human lens proteins during long-term incubation (~1 year) and in response to sustained oxidation (~2.5 months) in vitro. Our findings demonstrate that QLS analysis of human lens proteins provides a practical technique for noninvasive assessment of molecular aging in vivo.
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21

Costagliola, C., G. Iuliano, E. Rinaldi, A. Trapanese, V. Russo, A. Camera, and G. Scibelli. "In vivo Measurement of Human Lens Aging using the Lens Opacity Meter." Ophthalmologica 199, no. 4 (1989): 158–61. http://dx.doi.org/10.1159/000310034.

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22

Yan, Yu, Haiyang Yu, Liyao Sun, Hanruo Liu, Chao Wang, Xi Wei, Fanqian Song, et al. "Laminin α4 overexpression in the anterior lens capsule may contribute to the senescence of human lens epithelial cells in age-related cataract." Aging 11, no. 9 (May 10, 2019): 2699–723. http://dx.doi.org/10.18632/aging.101943.

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23

Fernández, Joaquín, Manuel Rodríguez-Vallejo, Javier Martínez, Ana Tauste, and David P. Piñero. "From Presbyopia to Cataracts: A Critical Review on Dysfunctional Lens Syndrome." Journal of Ophthalmology 2018 (June 27, 2018): 1–10. http://dx.doi.org/10.1155/2018/4318405.

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Dysfunctional lens syndrome (DLS) is a term coined to describe the natural aging changes in the crystalline lens. Different alterations in the refractive properties and transparency of the lens are produced during the development of presbyopia and cataract, such as changes in internal high order aberrations or an increase in ocular forward scattering, with a potentially significant impact on clinical measures, including visual acuity and contrast sensitivity. Objective technologies have emerged to solve the limits of current methods for the grading of the lens aging, which have been linked to the DLS term. However, there is still not a gold standard or evidence-based clinical guidelines around these new technologies despite multiple research studies have correlated their results with conventional methods such as visual acuity or the lens opacification system (LOCS), with more scientific background around the ocular scattering index (OSI) and Scheimpflug densitometry. In either case, DLS is not a new evidence-based concept that leads to new knowledge about crystalline lens aging but it is a nomenclature change of two existing terms, presbyopia and cataracts. Therefore, this term should be used with caution in the scientific peer-reviewed literature.
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24

Koretz, Jane F., Christopher A. Cook, and Paul L. Kaufman. "Aging of the human lens: changes in lens shape at zero-diopter accommodation." Journal of the Optical Society of America A 18, no. 2 (February 1, 2001): 265. http://dx.doi.org/10.1364/josaa.18.000265.

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25

Peterson, James, Gary Radke, and Larry Takemoto. "Interaction of lens alpha and gamma crystallins during aging of the bovine lens." Experimental Eye Research 81, no. 6 (December 2005): 680–89. http://dx.doi.org/10.1016/j.exer.2005.04.006.

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26

SULOCHANA, K. N., S. RAMAKRISHNAN, and R. PUNITHAM. "Increased lens dipeptidase activity in aging and cataract." British Journal of Ophthalmology 83, no. 7 (July 1, 1999): 885. http://dx.doi.org/10.1136/bjo.83.7.885.

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27

Siik, Seppo, P. Juhani Airaksinen, and Anja Tuulonen. "Light scatter in aging and cataractous human lens." Acta Ophthalmologica 70, no. 3 (May 27, 2009): 383–88. http://dx.doi.org/10.1111/j.1755-3768.1992.tb08584.x.

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28

Glasser, Adrian, Mary Ann Croft, and Paul L. Kaufman. "Aging of the Human Crystalline Lens and Presbyopia." International Ophthalmology Clinics 41, no. 2 (2001): 1–15. http://dx.doi.org/10.1097/00004397-200104000-00003.

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29

Tregillus, Katherine E. M., John S. Werner, and Michael A. Webster. "Adjusting to a sudden “aging” of the lens." Journal of the Optical Society of America A 33, no. 3 (February 3, 2016): A129. http://dx.doi.org/10.1364/josaa.33.00a129.

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30

Torres, S. "ACTIVE AGING POLICIES AND THE SOCIAL EXCLUSION LENS." Innovation in Aging 1, suppl_1 (June 30, 2017): 1361–62. http://dx.doi.org/10.1093/geroni/igx004.5006.

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31

Mandal, Krishnagopal, and Sidney Lerman. "Stability of Normal and Aging Lens Gamma Crystallins." Ophthalmic Research 25, no. 5 (1993): 295–301. http://dx.doi.org/10.1159/000267328.

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32

Fiske, B. J., Marsha Beach, and Bradley Coffey. "CA/C RATIOS, AGING, AND NEARPOINT LENS WEAR." Optometry and Vision Science 72, SUPPLEMENT (December 1995): 83. http://dx.doi.org/10.1097/00006324-199512001-00130.

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33

de Vries, A. C. J., M. A. Vermeer, A. L. A. M. Hendriks, H. Bloemendal, and L. H. Cohen. "Biosynthetic capacity of the human lens upon aging." Experimental Eye Research 53, no. 4 (October 1991): 519–24. http://dx.doi.org/10.1016/0014-4835(91)90169-f.

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34

Manoogian, Margaret. "Adopting an Intersectionality Lens Within an Undergraduate Gerontology Curriculum." Innovation in Aging 4, Supplement_1 (December 1, 2020): 534. http://dx.doi.org/10.1093/geroni/igaa057.1727.

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Abstract Students who plan geriatric/gerontology careers typically learn the biopsychosocial domains of aging. Using intersectionality to understand older adults and family experiences (Calasanti & Kiecolt, 2012), however, offers students a deeper understanding of how aging adults may face interconnected oppressions and inequalities based on race/ethnicity, gender, age, sexual orientation, socioeconomic status, health, and other aspects of social location within micro and macro contexts. Through systematic assessment of student learning outcomes, a planned programmatic approach to integrating intersectionality was adopted within an undergraduate gerontology program. This multifaceted approach will be highlighted including new course development, course case studies, community member voices, practicum applied practices, and research activities. Calasanti, T. & Kiecolt, K. J. (2012) Intersectionality and aging families. In Blieszner, R., & Bedford, V. H. (Eds.), Handbook of families and aging (pp. 263-286). Santa Barbara, CA: Praeger. Part of a symposium sponsored by Age-Friendly University (AFU) Interest Group.
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35

Gendron, Christi M., Tuhin S. Chakraborty, Brian Y. Chung, Zachary M. Harvanek, Kristina J. Holme, Jacob C. Johnson, Yang Lyu, Allyson S. Munneke, and Scott D. Pletcher. "Neuronal Mechanisms that Drive Organismal Aging Through the Lens of Perception." Annual Review of Physiology 82, no. 1 (February 10, 2020): 227–49. http://dx.doi.org/10.1146/annurev-physiol-021119-034440.

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Sensory neurons provide organisms with data about the world in which they live, for the purpose of successfully exploiting their environment. The consequences of sensory perception are not simply limited to decision-making behaviors; evidence suggests that sensory perception directly influences physiology and aging, a phenomenon that has been observed in animals across taxa. Therefore, understanding the neural mechanisms by which sensory input influences aging may uncover novel therapeutic targets for aging-related physiologies. In this review, we examine different perceptive experiences that have been most clearly linked to aging or age-related disease: food perception, social perception, time perception, and threat perception. For each, the sensory cues, receptors, and/or pathways that influence aging as well as the individual or groups of neurons involved, if known, are discussed. We conclude with general thoughts about the potential impact of this line of research on human health and aging.
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36

Stein, Daniel, Amir Mizrahi, Anastasia Golova, Adam Saretzky, Alfredo Garcia Venzor, Zeev Slobodnik, Shai Kaluski, Monica Einav, Ekaterina Khrameeva, and Debra Toiber. "Aging and pathological aging signatures of the brain: through the focusing lens of SIRT6." Aging 13, no. 5 (March 9, 2021): 6420–41. http://dx.doi.org/10.18632/aging.202755.

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37

Zhou, Hai-Yan, Wei-Jia Yan, and Xin-Chuan Wang. "Ultrasound elastography for evaluating stiffness of the human lens nucleus with aging: a feasibility study." International Journal of Ophthalmology 14, no. 2 (February 18, 2021): 240–44. http://dx.doi.org/10.18240/ijo.2021.02.09.

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AIM: To investigate the significance of ultrasound elastography for evaluating stiffness of the human lens nucleus in volunteers with different ages. METHODS: A total of 90 volunteers (lens transparency, uncorrected visual acuity ≥0.5, intraocular pressure: 14-19 mm Hg) were divided into 3 groups according to age: Group A (30 people, median age: 82±3.5y, mean axial lengths 23.7±0.5 mm); Group B (30 people, median age: 46±2.1y, mean axial lengths 23.9±0.4 mm); and Group C (30 people, median age: 22±3.5y, mean axial lengths 24.0±0.4 mm). Lens nuclear stiffness was measured by Free-hand qualitative elastography by independent operators. Strain gray scale and color-coded elastography maps were recorded. In each case, three consecutive detections were performed and strain ratio was used for statistical analysis. RESULTS: Elastography analysis showed excellent diagnostic performance for lens sclerosis. Lens strain ratio was lowest (0.03±0.01)% in Group A and highest (2.03±0.43)% in Group C. Lens strain ratio was moderate (0.64±0.10)% in Group B. There were significant differences between these three groups (P<0.05). The lens nucleus strain rate changes with age. With aging, the lens nucleus strain rate and resilience decrease, demonstrating harder texture. CONCLUSION: The relationship between human lens stiffness and age is demonstrated by ultrasound elastography. Older age is associated with lower strain ratio and less resilience of the lens.
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38

Cheng, Catherine, Justin Parreno, Roberta B. Nowak, Sondip K. Biswas, Kehao Wang, Masato Hoshino, Kentaro Uesugi, et al. "Age-related changes in eye lens biomechanics, morphology, refractive index and transparency." Aging 11, no. 24 (December 16, 2019): 12497–531. http://dx.doi.org/10.18632/aging.102584.

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39

DUNCAN, G., I. M. WORMSTONE, and P. D. DAVIES. "The aging human lens: structure, growth, and physiological behaviour." British Journal of Ophthalmology 81, no. 10 (October 1, 1997): 818–23. http://dx.doi.org/10.1136/bjo.81.10.818.

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40

Besner, Sebastien, Giuliano Scarcelli, Roberto Pineda, and Seok-Hyun Yun. "In Vivo Brillouin Analysis of the Aging Crystalline Lens." Investigative Opthalmology & Visual Science 57, no. 13 (October 3, 2016): 5093. http://dx.doi.org/10.1167/iovs.16-20143.

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41

Bettelheim, Frederick A., Martin J. Lizak, and J. Samuel Zigler. "Syneretic Response of Aging Normal Human Lens to Pressure." Investigative Opthalmology & Visual Science 44, no. 1 (January 1, 2003): 258. http://dx.doi.org/10.1167/iovs.02-0422.

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42

Occhipinti, Joseph R., Marjorie A. Mosier, and Neal L. Burstein. "Autofluorescence and Light Transmission in the Aging Crystalline Lens." Ophthalmologica 192, no. 4 (1986): 203–9. http://dx.doi.org/10.1159/000309647.

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43

Geeraets, Walter J., Wade Harrel, DuPont Guerry III, William T. Ham, and Harold A. Mueller. "AGING, ANOMALIES AND RADIATION EFFECT OF THE RABBIT LENS." Acta Ophthalmologica 43, no. 1 (May 27, 2009): 3–21. http://dx.doi.org/10.1111/j.1755-3768.1965.tb06364.x.

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44

Stefek, Milan, and Cimen Karasu. "Eye Lens in Aging and Diabetes: Effect of Quercetin." Rejuvenation Research 14, no. 5 (October 2011): 525–34. http://dx.doi.org/10.1089/rej.2011.1170.

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45

Cook, Christopher A., Jane F. Koretz, Arnold Pfahnl, John Hyun, and Paul L. Kaufman. "Aging of the human crystalline lens and anterior segment." Vision Research 34, no. 22 (November 1994): 2945–54. http://dx.doi.org/10.1016/0042-6989(94)90266-6.

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46

Dawczynski, J., and J. Strobel. "„The aging lens“ – neue Konzepte zum Alterungsprozess der Linse." Der Ophthalmologe 103, no. 9 (September 2006): 759–64. http://dx.doi.org/10.1007/s00347-006-1410-z.

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47

Shield, Renee. "Real World Anthropology in two Settings: A Nursing Home and a Health Care Policy Project." Practicing Anthropology 20, no. 2 (April 1, 1998): 11–13. http://dx.doi.org/10.17730/praa.20.2.353008jw77684143.

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Anthropologists have a unique opportunity to address real-world issues of aging in our own country. In graduate school I decided to redirect an ethnomusicological interest in ritual performance into an anthropological perspective on the subject of age. For one thing, as a new parent, I realized I was aging. Performance based questions in ethnomusicological suggested a lens through which to view aging. I did my dissertation on an American nursing home, applying concepts of rites of passage, performance and reciprocity to understand the behavior and the perspectives of nursing home participants. In subsequent years I have had two more opportunities to use the anthropologic lens to focus on aging in Rhode Island, first, as an educator in a nursing home and, second, as a participant in a health care reform project for the elderly of Rhode Island.
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48

Siezen, Roland J. "Eye lens aging in the spiny dogfish (Squalus acanthias) I. Age determination from lens weight." Current Eye Research 8, no. 7 (January 1989): 707–12. http://dx.doi.org/10.3109/02713688909025805.

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49

Dubbelman, M., and G. L. Van der Heijde. "The shape of the aging human lens: curvature, equivalent refractive index and the lens paradox." Vision Research 41, no. 14 (June 2001): 1867–77. http://dx.doi.org/10.1016/s0042-6989(01)00057-8.

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50

Koretz, Jane F., Christopher A. Cook, and Paul L. Kaufman. "Aging of the human lens: changes in lens shape upon accommodation and with accommodative loss." Journal of the Optical Society of America A 19, no. 1 (January 1, 2002): 144. http://dx.doi.org/10.1364/josaa.19.000144.

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