Journal articles: 'Agnosie'

1

TANAKA, MISATO. "Auditory agnosia (akustische agnosie)." Japan Journal of Logopedics and Phoniatrics 27, no. 2 (1986): 190–92. http://dx.doi.org/10.5112/jjlp.27.190.

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2

Landis, T., and M. Regard. "Hemianopsie und Agnosie." Klinische Monatsblätter für Augenheilkunde 192, no. 05 (May 1988): 525–28. http://dx.doi.org/10.1055/s-2008-1050172.

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3

Fechtelpeter, A., S. Göddenhenrich, W. Huber, and L. Springer. "Ansätze zur Therapie von auditiver Agnosie." Folia Phoniatrica et Logopaedica 42, no. 2 (1990): 83–97. http://dx.doi.org/10.1159/000266050.

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4

Borruat, F. X. "Conduite à tenir… devant une agnosie visuelle." Revue Neurologique 170 (April 2014): A183—A184. http://dx.doi.org/10.1016/j.neurol.2014.01.038.

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Platel, H., B. Lechevalier, J. Lambert, and F. Eustache. "Agnosie uditive e sindromi affini: valutazione clinica, cognitiva e psicopatologica." EMC - Neurologia 9, no. 4 (January 2009): 1–11. http://dx.doi.org/10.1016/s1634-7072(09)70505-7.

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Platel, H., B. Lechevalier, C. Descat, J. Lambert, and F. Eustache. "Agnosie uditive e sindromi correlate: studio clinico, cognitivo e psicopatologico." EMC - Neurologia 17, no. 2 (April 2017): 1–11. http://dx.doi.org/10.1016/s1634-7072(17)83852-6.

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Diesfeldta, H. F. A. "Associatieve visuele agnosie. De minder zichtbare gevolgen van een herseninfarct." Tijdschrift voor Gerontologie en Geriatrie 42, no. 1 (February 2011): 17–28. http://dx.doi.org/10.1007/s12439-011-0003-8.

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Hauw, F., M. Pernon, C. Lucas, H. Chabriat, and E. Cognat. "Agnosie auditive secondaire à un infarctus temporal bilatéral en 2 temps." Pratique Neurologique - FMC 8, no. 4 (December 2017): 216–20. http://dx.doi.org/10.1016/j.praneu.2017.06.015.

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9

Charnallet, A. "Agnosie et deficits categorie-specifiques: confrontation des modeles symboliques et episodiques." Neurocase 4, no. 4 (August 1, 1998): 399m—427. http://dx.doi.org/10.1093/neucas/4.4.399-m.

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Ackermann, H., and K. Mathiak. "Symptomatologie, pathologischanatomische Grundlaqen und Pathomechanismen zentraler Hörstörungen (reine Worttaubheit, auditive Agnosie, Rindentaubheit)." Fortschritte der Neurologie · Psychiatrie 67, no. 11 (November 1999): 509–23. http://dx.doi.org/10.1055/s-2007-995228.

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11

Bühler, Marco M., Brigitte Rüegger-Frey, Annette Ciurea-Löchel, and Sacha Beck. "Wenn eine neue Brille nichts nützt – ausgeprägte Sehstörung trotz normalem Visus." Praxis 105, no. 15 (July 2016): 905–8. http://dx.doi.org/10.1024/1661-8157/a002427.

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Zusammenfassung. Wir stellen den Fall einer 65-jährigen Frau vor mit seit wenigen Jahren bestehenden Sehstörungen bei unauffälligem ophthalmologischem Befund. Die neuropsychologische Untersuchung ergab eine komplexe Agnosie mit deutlichen Einschränkungen in visuell-konstruktiven und visuell-perzeptiven Leistungen bei nur leichten Defiziten in der Mnestik. Die Befunde passen zu einer posterioren kortikalen Atrophie. Der Störung liegt eine Neurodegeneration zugrunde, meist als Folge einer Alzheimerkrankheit. Da die Patienten über ein ausgeprägtes Störungsbewusstsein verfügen, sind eine gute Aufklärung sowie das Entwickeln geeigneter Kompensationsstrategien sehr wichtig.

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Bayen, E., A. Leger, S. Deltour, S. Crozier, and Y. Samson. "G - 6 Héminégligence et agnosie visuelle post-AIC de l’artère cérébrale postérieure gauche." Revue Neurologique 163, no. 4 (April 2007): 14. http://dx.doi.org/10.1016/s0035-3787(07)90512-3.

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Maurer, R., and V. Descloux. "Agnosie des repères : je n’ai peut-être pas oublié, mais je reconnais trop." Neurophysiologie Clinique/Clinical Neurophysiology 44, no. 1 (January 2014): 147–48. http://dx.doi.org/10.1016/j.neucli.2013.10.117.

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Merkenschlager, Andreas, and Christian Mühe. "Einfluss interiktaler EEG-Entladungen auf die Kognition." Kinder- und Jugendmedizin 03, no. 02 (2003): 59–63. http://dx.doi.org/10.1055/s-0037-1617777.

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ZsuammenfassungInteriktale epilepsietypische Potenziale werden als eine der möglichen Gründe für die häufiger ungünstige kognitive Performance von Kindern mit Epilepsie diskutiert. Es werden dabei punktuelle kognitive Defizite, TCI (»transitory cognitive impairment«), von längerfristigen Störungen unterschieden. Letztere können sich bei der Rolando-Epilepsie z. T. durch Aufmerksamkeitsstörungen und Verhaltensveränderungen zeigen, beim Landau-Kleffner-Syndrom durch eine auditive Agnosie und durch einen globalen kognitiven Abbau beim CSWS (kontinuierliche Spike-wave-Aktivität). Entsprechende pathophysiologische Erklärungsmodelle werden aufgezeigt. Therapieempfehlungen zur EEG-Sanierung bestehen beim Landau-Kleffner-Syndrom und CSWS, Therapieversuche zur EEG-Behandlung anfallsfreier Kinder mit weniger auffälligen kognitiven Einbußen zeigen uneinheitliche Ergebnisse. Diese bedürfen derzeit einer Einzelfallentscheidung mit sorgfältiger Dokumentation.

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15

Mineau, Suzanne, and Laurent Mottron. "Étude de cas : Intervention thérapeuthique utilisant les « intérêts particuliers » chez un enfant autiste avec agnosie visuelle." Santé mentale au Québec 23, no. 1 (September 11, 2007): 67–84. http://dx.doi.org/10.7202/032437ar.

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RÉSUMÉ Bien que l'éducation spécialisée sous ses divers modes soit consensuellement reconnue comme l'abord privilégié des troubles envahissants du développement, il n'existe à ce jour que peu de travaux sur l'intervention rééducative chez les personnes autistes d'intelligence normale. Cet article présente l'intervention psychoéducative faite avec un jeune garçon autiste de trois ans dans un centre de jour pédopsychiatrique. L'intérêt de cette observation est double. Tout d'abord, cet enfant était porteur, en plus de son autisme, d'une affection de caractère exceptionnel, l'agnosie visuelle. D'autre part, ses « intérêts restreints » autistiques, c'est-à-dire les comportements d'attirance perceptive et de questionnement pour une classe d'objets particuliers ou un paramètre particulier de ces objets, ont été utilisés comme levier pour l'amener à améliorer ses capacités de communication et d'interactions sociales.

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16

Forster. "Neuropsychological aspects in assessment of dementia." Therapeutische Umschau 56, no. 2 (February 1, 1999): 83–87. http://dx.doi.org/10.1024/0040-5930.56.2.83.

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Die Neuropsychologie kommt in der interdisziplinären Demenzdiagnostik in der Früherkennung, in der Differentialdiagnostik und in der Verlaufsdiagnostik zum Einsatz. Mittels standardisierter neuropsychologischer Verfahren wird eine differenzierte Abklärung über Art und Ausmaß von kognitiven Funktionsstörungen, insbesondere von Gedächtnisstörungen, Aphasie, Apraxie, Agnosie und Störungen der exekutiven Funktionen durchgeführt. In Patienten mit Alzheimerkrankheit (AD) sind oft episodische und semantische Gedächtnisleistungen und bestimmte Sprachfunktionen bereits im Frühstadium betroffen. Apraktische und agnostische Störungen werden häufig erst im späteren Verlauf beobachtet. Patienten mit einer vaskulären Demenz werden je nach Läsionsart und -ort neben den Gedächtnisstörungen eher hemisphärisch-lateralisierte Befunde (mit z.B. aphasischen, apraktischen oder räumlich-konstruktiven Störungen) aufweisen. In verschiedenen Demenzformen aus dem frontotemporalen Kreis sind in erster Linie die exekutiven Funktionen und/oder die Persönlichkeit betroffen.

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17

Benke, Thomas. "Early concepts of tactile object recognition: An historical synopsis and appraisal of Josef Gerstmann's Reine taktile Agnosie (1918)." Cognitive Neuropsychology 18, no. 3 (May 2001): 263–66. http://dx.doi.org/10.1080/02643290042000125.

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18

Gutzmann, Hans, Karl-Heinz Schmidt, and Annett Petermann. "Das Bálint-Holmes-Syndrom bei posteriorer kortikaler Atrophie." Zeitschrift für Gerontopsychologie & -psychiatrie 21, no. 2 (June 2008): 137–45. http://dx.doi.org/10.1024/1011-6877.21.2.137.

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Die Kasuistik beschreibt eine 76jährige Patientin, deren kognitive Störungen nach eigen- und fremdanamnestischen Angaben erstmals im Alter von ca. 70 Jahren aufgefallen waren. Neben leichten bis mäßigen mnestischen Defiziten traten im Verlauf der Krankheitsgeschichte sehr früh auch visuelle Störungen und Störungen in der räumlichen Orientierung auf. Aufgrund eines fremdaggressiven Verhaltens und eines Delirs wurde sie stationär gerontopsychiatrisch behandelt. Im Rahmen der zu diesem Zeitpunkt durchgeführten umfassenden klinischen und paraklinischen Diagnostik wurde das Vollbild eines Bálint-Holmes-Syndroms mit okulärer Apraxie, optischer Agnosie, gravierenden räumlichen Orientierungsstörungen und einer dorsalen wie ventralen Simultanagnosie diagnostiziert. Es waren die diagnostischen Kriterien einer Demenz vom Alzheimer-Typ erfüllt. Die Ergebnisse der bildgebenden Diagnostik legen differenzialdiagnostisch eine posteriore kortikale Atrophie (PCA) als fokale Variante einer Demenz vom Alzheimer-Typ nahe. In der Diskussion zu praktischen Schlussfolgerungen wird auf die Notwendigkeiten einer differenzierten neuropsychologischen Diagnostik sowie der genauen Aufklärung der die Patientin betreuenden Pflegepersonen über die Spezifik des Krankheitsbildes hingewiesen.

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19

Vecera, Shaun P., and Kendra S. Gilds. "What Processing Is Impaired in Apperceptive Agnosia? Evidence from Normal Subjects." Journal of Cognitive Neuroscience 10, no. 5 (September 1998): 568–80. http://dx.doi.org/10.1162/089892998562979.

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Visual agnosia is a neuropsychological syndrome characterized by a failure of object identification. Apperceptive agnosia, an object identification deficit caused by damage to early perceptual processes, has been explained by appealing to both damaged early sensory processes and to damaged preattentive grouping processes. Which of these two explanations best accounts for the behavior of these patients? We present results from two experiments designed to distinguish rival theoretical accounts of apperceptive agnosia. In our studies, we attempted to simulate apperceptive agnosia in neurologically intact subjects. Sensory-deficit accounts of the syndrome predict that degrading visual processing would make normal subjects perform like patients; grouping-deficit accounts predict that removing perceptual organization cues from visual displays would make normal subjects perform like patients. We were able to simulate the behavior of an apperceptive agnosic patient by removing perceptual organization cues, consistent with a grouping-deficit account of this syndrome. The implications for understanding both apperceptive agnosia and normal visual functioning are discussed.

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20

Hiraoka, Kotaro, Kyoko Suzuki, Kazumi Hirayama, and Etsuro Mori. "Visual Agnosia for Line Drawings and Silhouettes without Apparent Impairment of Real-Object Recognition: A Case Report." Behavioural Neurology 21, no. 3-4 (2009): 187–92. http://dx.doi.org/10.1155/2009/429762.

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We report on a patient with visual agnosia for line drawings and silhouette pictures following cerebral infarction in the region of the right posterior cerebral artery. The patient retained the ability to recognize real objects and their photographs, and could precisely copy line drawings of objects that she could not name. This case report highlights the importance of clinicians and researchers paying special attention to avoid overlooking agnosia in such cases. The factors that lead to problems in the identification of stimuli other than real objects in agnosic cases are discussed.

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21

Riddoch, M. Jane, Glyn W. Humphreys, Nabeela Akhtar, Harriet Allen, R. Martyn Bracewell, and Andrew J. Schofield. "A tale of two agnosias: Distinctions between form and integrative agnosia." Cognitive Neuropsychology 25, no. 1 (February 2008): 56–92. http://dx.doi.org/10.1080/02643290701848901.

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22

TIKHOMIROV, G. V., and V. N. GRIGORYEVA. "Visual object agnosia and novel landmarks agnosia in patients with acute supratentorial stroke." Practical medicine 18, no. 5 (2020): 163–68. http://dx.doi.org/10.32000/2072-1757-2020-5-163-168.

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The purpose — to estimate the frequency of visual general objects agnosia and novel landmarks agnosia in patients with acute ischemic stroke and to describe the clinical manifestations of these phenomena and association between them. Materil and methods. 75 patients with ischemic stroke were enrolled. Computed tomography and/or magnetic resonance scan to confirm the ischemic lesion, neurological, ophthalmological and neuropsychological examination were performed in all patients. Visual perception skills (gnosis) were measured by Birmingham Object Recognition Battery tests and authors’ Novel landmarks recognition test. Results. 8 out of 75 patients (10,7%) had visual agnosia documented by the neuropsychological testing, of these 3 patients had question-induced complaints («clinically evident» agnosia, group 1) and 5 patients had no complaints («clinically blurred» agnosia, group 2). 67 patients (89,3%) were not agnostic (group 3). The patients of the 1st and 2nd groups performed perception (gnosis) test significantly worse compared with those of the 3rd group while group 1 demonstrated significantly worse test results than group 2. A significant positive correlation was found between the results of Birmingham Object Recognition Battery tests and the Novel landmarks recognition test. Conclusion. General objects and landmarks recognition is impaired in 10,7% patients with acute ischemic hemispheric stroke. As a rule, general object agnosia is associated with landmark agnosia. Visual general objects and landmarks agnosia may be «clinically evident» verified by the patients’ question-induced complaints as well as by the results of neuropsychological testing, or «clinically blurred», verified only by testing. The test results were statistically significantly worse in «clinically evident» than in «clinically blurred» visual agnosia.

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Riddoch, M. J., G. W. Humphreys, and M. Bracewell. "A tale of two agnosias: Functional differences between integrative and visual form agnosia." Journal of Vision 6, no. 6 (March 24, 2010): 814. http://dx.doi.org/10.1167/6.6.814.

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Gómez Caffarena, José. "La «agnosia» del creyente." Arbor 171, no. 676 (April 30, 2002): 833–39. http://dx.doi.org/10.3989/arbor.2002.i676.1062.

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Flores Sierra, Ernesto Bayardo. "LAS AGNOSIAS Y SU RELACIÓN CON LA FORMACIÓN HISTÓRICO- CULTURAL DEL PSIQUISMO." Revista Cognosis. ISSN 2588-0578 1, no. 3 (August 26, 2016): 17. http://dx.doi.org/10.33936/cognosis.v1i3.248.

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El presente artículo analiza la formación de la percepción como un fenómeno social, estableciendo un diálogo entre las tesis de la escuela de la Gestalt, los estudios respecto al Test Gestaltico Visomotor y los aportes de la escuela de la psicología histórico- cultural soviética. Propone además el estudio de las agnosias desde la mencionada perspectiva, leyendo en las mismas alteraciones a los fenómenos culturales de la vida psíquica. PALABRAS CLAVE: Percepción; agnosias; psicología; histórico-cultural; Gestalt. ABSTRACT This article analyzes the formation of perception as a social phenomenon, establishing a dialogue between the Gestalt school, the studies regarding the Visomotor Gestaltic Test and the contributions of the school of Soviet historical- cultural psychology. It also proposes the study of agnosia from this perspective, reading in the same alterations the cultural phenomena of psychic life. KEYWORDS: Perception; agnosias; historical-cultural; psychology; Gestalt.

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Gaffan, David, and Charles A. Heywood. "A Spurious Category-Specific Visual Agnosia for Living Things in Normal Human and Nonhuman Primates." Journal of Cognitive Neuroscience 5, no. 1 (January 1993): 118–28. http://dx.doi.org/10.1162/jocn.1993.5.1.118.

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Patients with visual associative agnosia have a particular difficulty in identifying visually presented living things (plants and animals) as opposed to nonliving things. It has been claimed that this effect cannot be explained by differences in the inherent visual discriminability of living and nonliving things. To test this claim further, we performed two experiments with normal subjects. In Experiment 1 normal human observers were asked to identify objects in tachistoscopically presented line drawings. They made more errors with living things than with nonliving things. In Experiment 2 normal monkeys learned to discriminate among the same line drawings for food reward. They made many more errors in discriminating among living things than nonliving things. Agnosic patients' responses to the same line drawings were made available to us for correlative analysis with the subjects' responses to these drawings in Experiments 1 and 2. We conclude that a category-specific visual agnosia for living things can arise as a consequence of a modality-specific but not category-specific impairment in visual representation, since living things are more similar to each other visually than nonliving things are.

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27

Li, Mingshuang, Wei Tang, Chang Liu, Yun Nan, Wenjing Wang, and Qi Dong. "Vowel and Tone Identification for Mandarin Congenital Amusics: Effects of Vowel Type and Semantic Content." Journal of Speech, Language, and Hearing Research 62, no. 12 (December 18, 2019): 4300–4308. http://dx.doi.org/10.1044/2019_jslhr-s-18-0440.

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Purpose This study aimed to explore the effects of Mandarin congenital amusia with or without lexical tone deficit (i.e., tone agnosia and pure amusia) on Mandarin vowel and tone identification in different types of vowels (e.g., monophthong, diphthongs, and triphthongs) embedded in consonant–vowel contexts with and without semantic content. Method Thirteen pure amusics (i.e., amusics with normal lexical processing), 5 tone agnosics (i.e., with lexical tone deficit), and 12 controls were screened with Montreal Battery of Evaluation of Amusia and lexical tone tests ( Nan et al., 2010 ; Peretz et al., 2003 ). Vowel-plus-tone identification tasks with the factors of vowel type and syllables with and without semantic content (e.g., real and nonsense words) were examined among the 3 groups, and identification scores were calculated in 3 formats: vowel-plus-tone identification, vowel identification, and tone identification. Results Tone agnosics showed significantly poorer performances on identifications of vowel, tone, and vowel plus tone across monophthongs, diphthongs, and triphthongs in both real and nonsense words compared to pure amusics and controls. Their deficits were similar across the 3 types of vowels, while the deficit on vowel-plus-tone identification was more severe in nonsense words than in real words. On the other hand, pure amusics performed similarly with controls across all these conditions. Conclusions Tone agnosia might affect both musical pitch and phonological processing, resulting in deficits in lexical tone and vowel perception. On the contrary, pure amusics's effect is primarily on musical pitch perception but not on lexical tone or phonemic deficit. Vowel type did not affect speech deficits for tone agnosics, while they relied more on semantic content as a compensation.

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VIGNOLO, LUIGI A. "Music Agnosia and Auditory Agnosia." Annals of the New York Academy of Sciences 999, no. 1 (November 2003): 50–57. http://dx.doi.org/10.1196/annals.1284.005.

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Suzuki, Kyoko. "Agnosia." Higher Brain Function Research 29, no. 2 (2009): 216–21. http://dx.doi.org/10.2496/hbfr.29.216.

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Baelz, Peter. "Agnosis." Theology 100, no. 798 (November 1997): 448. http://dx.doi.org/10.1177/0040571x9710000615.

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Greenwald, Margaret, and Judy Mikola. "Agnosia." Perspectives on Neurophysiology and Neurogenic Speech and Language Disorders 12, no. 1 (April 2002): 25–30. http://dx.doi.org/10.1044/nnsld12.1.25.

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Farah, Martha J. "Agnosia." Current Opinion in Neurobiology 2, no. 2 (April 1992): 162–64. http://dx.doi.org/10.1016/0959-4388(92)90005-6.

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Farah, Martha J. "Agnosia." Current Biology 2, no. 5 (May 1992): 235. http://dx.doi.org/10.1016/0960-9822(92)90353-c.

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Kocyba, C., and B. Choukroun. "Syndrome de Capgras… à partir d’un cas clinique." European Psychiatry 29, S3 (November 2014): 569. http://dx.doi.org/10.1016/j.eurpsy.2014.09.252.

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« Curieux petit syndrome », le syndrome de Capgras consiste en un trouble de l’identification des personnes. Il porte atteinte au sentiment de familiarité, renvoyant un inconfortable vécu d’étrangeté. Cela donne une véritable particularité clinique dans la relation et la prise en charge du patient. Partant de la situation de Mme. A., nous entamons une revue de la littérature des caractéristiques sémiologiques et des considérations étiologiques du syndrome de Capgras. Initialement dénommé « illusion des sosies », ce syndrome renvoie à la conviction délirante du patient qu’un ou des proches ont été substitués par un ou des sosies. Ce dernier renvoie un sentiment de persécution d’intensité variable selon les patients. Capgras résume sa description clinique en parlant d’« agnosie d’identification », car « partout le patient saisit la ressemblance et partout il méconnaît l’identité ». La clinique de Mme. A., par ses intrications psychiatriques, biologiques, cognitives et neurologiques, souligne la nécessité d’une approche multifactorielle et intégrative dans la compréhension du syndrome de Capgras. À côté des hypothèses psychodynamiques, deux modèles issus des sciences cognitives se dégagent dans la littérature : le modèle séquentiel de reconnaissance des visages par Bruce et Young et le modèle « à deux voies ». Des modèles ultérieurs tentent ensuite de faire le lien avec les données de neuro-imagerie, notamment fonctionnelle. Issus de ces démarches intégratives, des approches globales du syndrome de Capgras semblent se développer dans les travaux les plus récents de la littérature. Avec une clinique à la rencontre de la psychiatrie et de la neurologie, les questions autour de l’organicité du syndrome de Capgras persistent sans être entièrement tranchées. Ce syndrome demeure néanmoins d’une grande richesse pour le clinicien, tant sur le plan théorique que dans les liens avec le patient et son entourage.

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Bosch, Daniel. "Agnosis: Cambridge." Annals of Internal Medicine 154, no. 4 (February 15, 2011): 226. http://dx.doi.org/10.7326/0003-4819-154-4-201102150-00002.

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Small, D. M., N. Bernasconi, A. Bernasconi, V. Sziklas, and M. Jones-Gotman. "Gustatory agnosia." Neurology 64, no. 2 (January 24, 2005): 311–17. http://dx.doi.org/10.1212/01.wnl.0000149515.77718.35.

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Rothi, L. J., L. Mack, and K. M. Heilman. "Pantomime agnosia." Journal of Neurology, Neurosurgery & Psychiatry 49, no. 4 (April 1, 1986): 451–54. http://dx.doi.org/10.1136/jnnp.49.4.451.

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Reed, Catherine L., Richard J. Caselli, and Martha J. Farah. "Tactile agnosia." Brain 119, no. 3 (1996): 875–88. http://dx.doi.org/10.1093/brain/119.3.875.

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Platz, T. "Tactile agnosia." Brain 119, no. 5 (1996): 1565–74. http://dx.doi.org/10.1093/brain/119.5.1565.

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Riddoch, M. Jane, and Glyn W. Humphreys. "Visual agnosia." Neurologic Clinics 21, no. 2 (May 2003): 501–20. http://dx.doi.org/10.1016/s0733-8619(02)00095-6.

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Anonymous and A. Basu. "Visual agnosia." BMJ 345, no. 09 2 (November 9, 2012): e7342-e7342. http://dx.doi.org/10.1136/bmj.e7342.

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Álvarez, R., and J. Masjuan. "Visual agnosia." Revista Clínica Española (English Edition) 216, no. 2 (March 2016): 85–91. http://dx.doi.org/10.1016/j.rceng.2015.10.002.

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Barton, Jason J. S. "Visual Agnosia." American Journal of Ophthalmology 112, no. 3 (September 1991): 361. http://dx.doi.org/10.1016/s0002-9394(14)76760-1.

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Ramachandran, V. S., E. L. Altschuler, and S. Hillyer. "Mirror agnosia." Proceedings of the Royal Society of London. Series B: Biological Sciences 264, no. 1382 (May 22, 1997): 645–47. http://dx.doi.org/10.1098/rspb.1997.0091.

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Travers, Paul. "Immunological agnosia." Nature 363, no. 6425 (May 1993): 117–18. http://dx.doi.org/10.1038/363117a0.

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Biran, I., and H. B. Coslett. "Visual agnosia." Current Neurology and Neuroscience Reports 3, no. 6 (November 2003): 508–12. http://dx.doi.org/10.1007/s11910-003-0055-4.

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47

TAMAI, AKIRA. "Agnosia of color." Higher Brain Function Research 7, no. 3 (1987): 216–21. http://dx.doi.org/10.2496/apr.7.216.

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VANZANDVOORT, M., T. NIJBOER, and E. DEHAAN. "Developmental Colour Agnosia." Cortex 43, no. 6 (2007): 750–57. http://dx.doi.org/10.1016/s0010-9452(08)70503-3.

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Mazzucchi, A., L. Posteraro, G. Nuzzi, and M. Parma. "Unilateral Visual Agnosia." Cortex 21, no. 2 (June 1985): 309–16. http://dx.doi.org/10.1016/s0010-9452(85)80036-8.

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Chandra, Sadanandavalli Retnaswami, and Thomas Gregor Issac. "Mirror Image Agnosia." Indian Journal of Psychological Medicine 36, no. 4 (October 2014): 400–403. http://dx.doi.org/10.4103/0253-7176.140726.

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Journal articles on the topic 'Agnosie'

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