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Books on the topic 'Alcoholic Fatty Liver'

Author: Grafiati

Published: 4 June 2021

Last updated: 12 February 2022

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1

Farrell, Geoffrey C., Arthur J. McCullough, and Christopher P. Day, eds. Non-Alcoholic Fatty Liver Disease. Oxford, UK: Wiley-Blackwell, 2013. http://dx.doi.org/10.1002/9781118556153.

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2

Bugianesi, Elisabetta, ed. Non-Alcoholic Fatty Liver Disease. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-95828-6.

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3

Chalasani, Naga, and Gyongyi Szabo, eds. Alcoholic and Non-Alcoholic Fatty Liver Disease. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-20538-0.

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4

Farrell, Geoffrey C., Arthur J. McCullough, and Christopher Paul Day. Non-alcoholic fatty liver disease: A practical guide. Chichester, West Sussex: John Wiley & Sons, 2013.

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5

Taylor-Robinson, Simon D., and Roger Williams. Clinical dilemmas in non-alcoholic fatty liver disease. Chichester, West Sussex: John Wiley & Sons Inc., 2016.

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6

Williams, Roger, and Simon D. Taylor-Robinson, eds. Clinical Dilemmas in Non-Alcoholic Fatty Liver Disease. Chichester, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781118924938.

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7

Buko, V. U. Prostaglandiny pri alkogolʹnom porazhenii pecheni. Minsk: "Navuka i tėkhnika", 1991.

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8

U, Leuschner, James O. F. W, and Dancygier Henryk, eds. Steatohepatitis: NASH and ASH. Dordrecht: Kluwer Academic, 2001.

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9

Fitzpatrick, Emer. Non-alcoholic fatty liver disease. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198759928.003.0061.

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The chapter on non-alcoholic fatty liver disease includes the risk factors for this ever-increasing condition, the pathophysiology, as well as the differential of hepatic steatosis. Finally it includes the most current principles of its management.

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10

Beattie, R. Mark, Anil Dhawan, and John W.L. Puntis. Fatty liver disease in children. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0055.

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Demographics 406Pathophysiology 406Differential diagnoses 407Presenting features 407Investigation 408Management 409Fatty liver disease is now increasingly recognized in children, particularly in the setting of obesity.The term non-alcoholic steatohepatitis (NASH) was first coined in 1980 by Ludwig to describe a pattern of liver injury in adults in which the liver histology was consistent with alcoholic hepatitis, but in whom significant alcohol consumption was denied. NASH can be considered as part of a broader spectrum of non-alcoholic fatty liver disease that extends from simple steatosis through steatohepatitis that is characterized by the potential to progress to fibrosis, cirrhosis and subsequent end stage liver disease....

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11

Keshav, Satish, and Alexandra Kent. Alcoholic liver disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0211.

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Alcoholic liver disease develops in excessive drinkers and can manifest in three forms: alcoholic fatty liver (steatosis; >80%), alcoholic hepatitis (10%–35%), and cirrhosis (10%). The more alcohol consumed, the greater the risk of alcoholic liver disease, although other factors may also be involved. Alcohol can cause significant damage without producing any symptoms, and many patients will only have liver dysfunction detected on routine blood tests. Many patients report non-specific symptoms, such as anorexia, morning nausea, diarrhoea, and vague right upper quadrant abdominal pain. The underlying pathogenesis of alcohol-induced injury is not fully understood but is thought to involve various mechanisms. This chapter discusses alcoholic liver disease, focusing on its etiology, symptoms, demographics, natural history, complications, diagnosis, prognosis, and treatment.

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12

Day, Christopher P., Geoffrey C. Farrell, and Arthur J. McCullough. Non-Alcoholic Fatty Liver Disease: A Practical Guide. Wiley & Sons, Incorporated, John, 2013.

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13

undifferentiated, Roger Williams, and Simon D. Taylor-Robinson. Clinical Dilemmas in Non-Alcoholic Fatty Liver Disease. Wiley & Sons, Incorporated, John, 2016.

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14

undifferentiated, Roger Williams, and Simon D. Taylor-Robinson. Clinical Dilemmas in Non-Alcoholic Fatty Liver Disease. Wiley & Sons, Incorporated, John, 2016.

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15

Non-Alcoholic Fatty Liver Disease Research 2016. MDPI, 2018. http://dx.doi.org/10.3390/books978-3-03897-601-1.

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16

Chalasani, Naga, and Gyongyi Szabo. Alcoholic and Non-Alcoholic Fatty Liver Disease: Bench to Bedside. Springer, 2019.

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17

Chalasani, Naga, and Gyongyi Szabo. Alcoholic and Non-Alcoholic Fatty Liver Disease: Bench to Bedside. Springer, 2015.

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18

Bugianesi, Elisabetta. Non-Alcoholic Fatty Liver Disease: A 360-degree Overview. Springer, 2020.

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19

Valenzuela, Rodrigo, ed. Non-Alcoholic Fatty Liver Disease - Molecular Bases, Prevention and Treatment. InTech, 2018. http://dx.doi.org/10.5772/68045.

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20

Keshav, Satish, and Palak Trivedi. Miscellaneous liver diseases. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0216.

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This chapter discusses miscellaneous liver diseases, including non-alcoholic fatty liver disease, liver abscess, and nodular regenerative hyperplasia. It explores definitions of the diseases, their etiologies, typical symptoms, uncommon symptoms, demographics, natural history, complications, diagnostic approach, other diagnoses that should be considered, prognosis, and treatment.

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21

Nutritional Intake and the Risk for Non-alcoholic Fatty Liver Disease (NAFLD). MDPI, 2019. http://dx.doi.org/10.3390/books978-3-03897-599-1.

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22

Avand, Ghazal. Metabolic syndrome in non-alcoholic fatty liver disease: A comparison between simple steatosis and non-alcoholic steatohepatitis. 2006.

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23

Mancell, Sara, and Deepa Kamat. Nutritional management of liver disease. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198759928.003.0068.

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The chapter on nutrition and liver disease describes the pathophysiology of the malnutrition that frequently accompanies chronic liver disease in children and then discusses the suggested management as well as ways of nutritional assessment and monitoring of the children. There is also a section of specific suggestions for various conditions such as biliary atresia, Alagille syndrome, Wilson disease, non-alcoholic fatty liver disease, as well as an overview of the various special feeds and supplements used in these conditions.

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24

Mohammed, Saira Saddia. Obesity is not associated with non-alcoholic fatty liver disease (NAFLD) in HIV-positive gay males. 2006.

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25

Carton, James. Hepatobiliary pathology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759584.003.0008.

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This chapter discusses hepatobiliary pathology and covers acute viral hepatitis, chronic viral hepatitis, alcoholic liver disease, non-alcoholic fatty liver disease, autoimmune hepatitis (AIH), primary biliary cholangitis (PBC), primary sclerosing cholangitis (PSC), Wilson’s disease, hereditary haemochromatosis, cirrhosis, benign liver lesions, hepatocellular carcinoma (liver cell cancer), intrahepatic cholangiocarcinoma, cholecystitis, and extrahepatic bile duct carcinoma.

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26

Carton, James. Hepatobiliary pathology. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199591633.003.0007.

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Acute viral hepatitis 116Chronic viral hepatitis 117Alcoholic liver disease 118Non-alcoholic fatty liver disease 119Autoimmune hepatitis 120Primary biliary cirrhosis 121Primary sclerosing cholangitis 122Wilson's disease 123Hereditary haemochromatosis 124Cirrhosis 125Benign liver lesions 126Hepatocellular carcinoma 127Intrahepatic cholangiocarcinoma ...

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27

Jolly, Elaine, Andrew Fry, and Afzal Chaudhry, eds. Gastroenterology and hepatology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199230457.003.0009.

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Chapter 9 covers the basic science and clinical topics relating to gastroenterology and hepatology which trainees are required to learn as part of their basic training and demonstrate in the MRCP. It covers basic science, gastrointestinal investigation, malabsorption and malnutrition, inflammatory bowel disease, acute upper gastrointestinal haemorrhage, lower gastrointestinal bleeding and related disorders, gastrointestinal infections, gastrointestinal cancer, miscellaneous gastrointestinal problems, normal liver and biliary function, variceal disease, hepatic tumours, acute (fulminant) liver failure, haemochromatosis, Wilson disease (hepatolenticular degeneration), Alpha-1 antitrypsin deficiency, alcohol-induced liver disease, hepatitis, biliary diseases, and non-alcoholic fatty liver disease.

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28

Keshav, Satish, and Alexandra Kent. Prevention of gastrointestinal disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0346.

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Disease prevention is usually directed where there is considerable morbidity or mortality, and etiological factors that can be controlled, treated, or reduced. The greatest morbidity and mortality from gastrointestinal disease is related to infectious diarrhoea and gastrointestinal cancer, both of which can be prevented. Smoking has been closely associated with oesophageal, gastric, and liver cancer and also has a significant effect in inflammatory bowel disease. In addition, alcohol consumption and viral hepatitis are preventable causes of liver disease, liver failure, and hepatic cancer. This chapter addresses the prevention of gastrointestinal disease, focusing on alcohol, smoking, peptic ulcer disease, colorectal cancer, oesophageal adenocarcinoma, hepatitis B, hepatitis C, non-alcoholic fatty liver disease, and gastrointestinal infection.

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29

(Editor), U. Leuschner, O.F.W. James (Editor), and H. Dancygier (Editor), eds. Steatohepatitis (NASH and ASH) (Falk Symposium). Springer, 2001.

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30

Plebani, Mario, Monica Maria Mion, and Martina Zaninotto. Biomarkers of renal and hepatic failure. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0039.

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In the last few years, major advances have been achieved in the understanding of the molecular and pathophysiological mechanisms which underlie the complex interactions between the heart and the kidney, as well as between the heart and the liver. According to these new insights, new biomarkers have been proposed for better evaluating and monitoring patients affected by cardiovascular diseases. In addition, some biomarkers should be used as risk factors and for an early identification and treatment of these severe diseases. This chapter reviews the most important biomarkers for evaluating the ‘cardiorenal syndrome’, in particular, the measurement of serum creatinine and its use for calculating the glomerular filtration rate which, with the new and more efficient equation, namely Chronic Kidney Disease Epidemiology Collaboration, still remains the most widely used biomarker. The role of newer biomarkers will be explored. The measurement of cystatin C, representing additional information, particularly in paediatric age groups and in the early phase of kidney disease, plays an increasing role. Neutrophil gelatinase-associated lipocalin is a recently developed and very promising new biomarker for the diagnosis of acute kidney injury, while the well-known albumin/creatinine ratio has been re-evaluated as a simple and useful tool for an early identification of kidney disease. Regarding liver diseases, a growing body of evidence demonstrates the usefulness of non-invasive makers of hepatic fibrosis that may avoid the need for a liver biopsy in most patients. A promising field of research is represented by the role of non-alcoholic fatty liver disease in the pathogenesis of cardiovascular disease.

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31

Thomas London, W., Jessica L. Petrick, and Katherine A. McGlynn. Liver Cancer. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0033.

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Primary liver cancer is the sixth most frequently occurring cancer in the world and the second most common in terms of cancer deaths. The global burden of liver cancer is borne principally by countries in East Asia and Africa, where 80% of liver cancer arises. Incidence rates of liver cancer, however, have begun to decline in Asia, while rates are increasing in low-rate areas such as Europe and North America. The dominant histology of liver cancer in almost all countries is hepatocellular carcinoma (HCC). The major risk factors for HCC—chronic infection with either hepatitis B virus (HBV) or hepatitis C virus (HCV), aflatoxin B1 (AFB1) contamination of foodstuffs, excessive alcohol consumption, and diabetes/obesity/fatty liver disease—all result in chronic inflammation in the liver. HBV infection is preventable by immunization, and HCV infection is largely preventable by public health measures and now is curable with new antiviral therapies.

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