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Journal articles on the topic 'Alcoholic Neuropathy'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Koike, Haruki, and Gen Sobue. "Alcoholic neuropathy." Current Opinion in Neurology 19, no. 5 (2006): 481–86. http://dx.doi.org/10.1097/01.wco.0000245371.89941.eb.

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2

Behl, Tapan, Harlokesh N. Yadav, and Pyare L. Sharma. "Alcoholic Neuropathy: Involvement of Multifaceted Signalling Mechanisms." Current Molecular Pharmacology 14, no. 1 (2020): 2–10. http://dx.doi.org/10.2174/1874467213666200512114943.

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Background: Alcoholic neuropathy is a chronic disorder caused by excessive consumption of alcohol. Damage to the nerves results in unusual sensations in the limbs, decrease mobility and loss of some body functions. Objective: Alcohol is considered a major villain for exclusively creating the debilitating condition of the neuropathic state. This review critically examines the key mediators involved in the pathogenesis of alcoholic neuropathy and the targets which upon selective inhibition alleviates the progression of alcoholic neuropathy. Method: A thorough study of research and review article

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3

Thejaswini Karanth, Someswar Deb, Pranathi R, and Kasthuri P. "An efficient approach on alcohol dependence syndrome." International Journal of Research in Phytochemistry and Pharmacology 8, no. 3 (2018): 24–27. http://dx.doi.org/10.26452/ijrpp.v8i3.1391.

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Alcohol consumption results in multiple end-organ damages. Predominantly the nervous system is affected in the form of Wernicke encephalopathy, Korsakoff psychosis, cerebellar ataxia and peripheral neuropathy. Neuropathy due to alcohol consumption depends on the period and extent of total lifetime of alcohol consumption. Cultural and racial factors also play a role in the development of alcohol neuropathy. Alcohol consumption results in multiple end-organ damages. Alcoholic neuropathy is the most common harmful effects of excessive alcohol intake. The incidence of alcoholic neuropathy in the s

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4

Niyati, Gosai, Goswami Dinkar, Mundra Shashi, Makwana Arun, and Desai Gaurav. "Clinical Profile of Patients of Alcoholic Neuropathy at Tertiary Care Hospital, Gujarat, India." International Journal of Pharmaceutical and Clinical Research 15, no. 1 (2023): 1031–36. https://doi.org/10.5281/zenodo.13153163.

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<strong>Background:</strong> Alcohol is one of the most commonly used substances in the world. After ingestion, alcohol distributes throughout body tissues and rapidly crosses the blood-brain barrier. According to DSM-IV, studies involving clinical and electrodiagnostic criteria prove that neuropathy is present in 25-66% of chronic alcoholics. Alcohol consumption results in multiple end-organ damages. <strong>Objectives: </strong>To study the clinical profile of alcoholic neuropathy patients. <strong>Materials and Methods</strong>: This cross-sectional study was carried out

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5

Behan, Caragh, Abbie Lane, and Mary Clarke. "Disulfiram induced peripheral neuropathy: between the devil and the deep blue sea." Irish Journal of Psychological Medicine 24, no. 3 (2007): 115–16. http://dx.doi.org/10.1017/s0790966700010454.

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AbstractDisulfiram is widely used for the aversive treatment of alcoholism. It is well tolerated in most patients; however 1:15,000 will develop peripheral neuropathy, which is frequently misdiagnosed as alcoholic neuropathy. Most cases present with a motor-sensory neuropathy of the lower limbs, which tends to improve when administration ceases, though some cases may be left with permanent sequelae. This adverse reaction can be avoided by using the lowest effective dose. A literature review using the Medline revealed 66 citations relating disulfiram to peripheral neuropathy, and the number of

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6

NISHIYAMA, Kazutoshi, and Manabu SAKUTA. "Mexiletine for Painful Alcoholic Neuropathy." Internal Medicine 34, no. 6 (1995): 577–79. http://dx.doi.org/10.2169/internalmedicine.34.577.

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7

Koike, Haruki, Masahiro Iijima, Makoto Sugiura, et al. "Alcoholic neuropathy is clinicopathologically distinct from thiamine-deficiency neuropathy." Annals of Neurology 54, no. 1 (2003): 19–29. http://dx.doi.org/10.1002/ana.10550.

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8

B. Limaye, Shraddha, and Harshada R. Patil. "The Effect of Otago Exercise Program for Fall Prevention in Alcoholic Neuropathy Patients." International Journal of Health Sciences and Research 12, no. 6 (2022): 168–73. http://dx.doi.org/10.52403/ijhsr.20220622.

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Introduction: Otago Exercise Program (OEP) consists of lower limb strengthening, balance and endurance exercises designed to prevent risk of fall. Alcoholic Neuropathy [AN] patients frequently complaints of muscle pain, difficult in gait, reduced muscle strength, falls and cramps thus they are at a high risk of fall. The aim of study is to find the effect of OEP for fall prevention in alcoholic neuropathy patients. Method: Study consists of 34 male patients aged 45 to 64 years diagnosed with AN. Patients at moderate high and risk of fall on Tinetti Performance Oriented Mobility Assessment [TPO

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9

Arvind, Kumar, K. Nandmer A, K. Nandmer V, and Dube Simmi. "Study of Pattern of Neuropathy in Chronic Liver Diseases in aTertiary Care Center of Bhopal, Madhya Pradesh." International Journal of Pharmaceutical and Clinical Research 16, no. 2 (2024): 331–37. https://doi.org/10.5281/zenodo.11067710.

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<strong>Background:</strong> Chronic liver disease (CLD) is a leading cause of death worldwide, especially in developing nations. CLD is increasing in prevalence, as has been established recently. The most common causes of CLD include chronic viral hepatitis, alcoholic liver disease, non-alcoholic fatty liver disease, and hemochromatosis. <strong>Aims and Objectives:</strong> To study the pattern of neuropathy in patients with CLD. <strong>Materials and Methods: </strong>Present cross-sectional study was performed on 100 consenting patients with cirrhosis of any etiolo

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10

L. D'Amour, Monique, Julie Bruneau, and Roger F. Butterworth. "Abnormalities of Peripheral Nerve Conduction in Relation to Thiamine Status in Alcoholic Patients." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 18, no. 2 (1991): 126–28. http://dx.doi.org/10.1017/s0317167100031565.

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ABSTRACT:Thiamine status was evaluated using the erythrocyte transketolase activation assay in 20 alcoholic patients admitted on a voluntary basis to a Detoxification Unit. Electromyographic evaluation revealed significant reductions of motor and sensory conduction velocities in the alcoholic group. 38% of alcoholic patients had at least one abnormal peripheral nerve conduction parameter. Findings were consistent with an axonal type of neuropathy. 30% of alcoholic patients showed significant erythrocyte transketolase activation deficits indicative of severe thiamine deficiency. In the case of

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11

Juntunen, Juhani, Esko Matikainen, Juha Nickels, Reino Ylikahri, and Maija Sarviharju. "ALCOHOLIC NEUROPATHY AND HEPATOPATHY IN MICE." Acta Pathologica Microbiologica Scandinavica Series A :Pathology 91A, no. 1-6 (2009): 137–44. http://dx.doi.org/10.1111/j.1699-0463.1983.tb02738.x.

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12

Barter, F., and A. R. Tanner. "Autonomic neuropathy in an alcoholic population." Postgraduate Medical Journal 63, no. 746 (1987): 1033–36. http://dx.doi.org/10.1136/pgmj.63.746.1033.

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13

Shashidhar, Mehta, Kumar Chanchal, Parvez Suhel, and Bhatnagar Suhasini. "Alcohol-Induced Neuropathy: Exploring the Impact on Molecular Channels and Pain Mechanisms." International Journal Of Ayurvedic And Herbal Medicine 15, no. 02 (2025): 4845–60. https://doi.org/10.5281/zenodo.15235359.

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<strong>ABSTRACT: </strong> Neuropathic pain is a severe form of pain that is brought on by dysfunction in the brain and is connected with a number of degenerative alterations in the nervous system. Hyperalgesia and allodynia are two of the main symptoms of pain. Many neurological diseases seen in chronic alcoholics are linked to dietary deficiencies, especially vitamin deficits that are crucial for healthy brain function. The brain may suffer from severe functional impairment and tissue damage, mostly neuronal and vascular, as a result of specific vitamin and nutritional shortages that d

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14

Dudek, Iga, Danuta Hajduga, Cezary Sieńko, et al. "Alcohol-Induced Neuropathy in Chronic Alcoholism: Causes, Pathophysiology, Diagnosis, and Treatment Options." Current Pathobiology Reports 8, no. 4 (2020): 87–97. http://dx.doi.org/10.1007/s40139-020-00214-w.

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Abstract Purpose of the Review Alcohol abuse causes a wide range of disorders that affect the nervous system. These include confusion, cerebellar ataxia, peripheral neuropathy, and cognitive impairment. Chronic and excessive alcohol consumption is the primary cause of peripheral neuropathy. It is worth noting that peripheral neuropathy has no reliable treatment due to the poor understanding of its pathology. Recent Findings Coasting is a major feature of alcoholic neuropathy, largely due to chronic alcohol abuse. Its major features are hyperalgesia, allodynia, and burning pain. Even though muc

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15

Abrorova, Durdona, Gulnora Togaeva, and Aziza Davranova. "CLINICAL AND FUNCTIONAL FEATURES OF THE COURSE IN PATIENTS WITH TYPE 2 DIABETES MELLITUS WITH AUTONOMIC NEUROPATHY." UZBEK MEDICAL JOURNAL 2, no. 3 (2021): 11–15. http://dx.doi.org/10.26739/2181-0664-2021-3-2.

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Diabetic neuropathy (DN) is one of the most common complications of diabetes mellitus (DM), which leads to severalconditions that reduce performance and threaten thepatient's life. Neuropathy against the background of diabetes takes the topplace, accounting for 40% of all neuropathies, ahead of alcoholic neuropathy [2]. Diabetic neuropathy is a combination of syndromes of damage to the nervous system with a predominant lesion of its various parts (sensorimotor or autonomic).Keywords:Diabetes mellitus,diabetic autonomic cardiac neuropathy, circadian index, heart denervation, painless ischemia

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16

Mittal, Mahim, Pavan Kumar Singh, and Sonu Kurian. "Study of prevalence and pattern of peripheral neuropathy in patients with liver cirrhosis." International Journal of Advances in Medicine 4, no. 4 (2017): 1041. http://dx.doi.org/10.18203/2349-3933.ijam20173228.

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Background: Peripheral neuropathy is not an uncommon complication of liver cirrhosis but it is not given much clinical relevance. The pattern and prevalence of peripheral neuropathy has also not been studied extensively. Alcohol use, coexisting diseases and nutritional deficiency have been postulated as factors responsible for neuropathy. The aim of this study was to define the pattern and prevalence of neuropathy in cirrhosis and to determine any etiologic correlation.Methods: 85 patients of cirrhosis of liver were studied. Apart from routine investigations, serum vitamin B12 estimation was a

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17

Genç, Hakan, Barış Nacır, Aynur Karagöz, and Hatice Rana Erdem. "Alcoholic Neuropathy with Superimposed Focal Entrapment Neuropathies." Meandros Medical and Dental Journal 18, no. 3 (2017): 222–25. http://dx.doi.org/10.4274/meandros.2227.

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18

Tessitore, Maria Eduarda, Laís da Silva Pereira-Rufino, Carlos Eduardo Panfilio, et al. "Alcoholic neuropathy associated with chronic alcohol intake." IBRO Neuroscience Reports 13 (December 2022): 177–86. http://dx.doi.org/10.1016/j.ibneur.2022.08.004.

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19

Hawley, Rollin J., John F. Kurtzke, Vernon W. Armbrustmacher, Nirmal Saini, and Herbert Manz. "The course of alcoholic-nutritional peripheral neuropathy." Acta Neurologica Scandinavica 66, no. 5 (2009): 582–89. http://dx.doi.org/10.1111/j.1600-0404.1982.tb03146.x.

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20

Dina, Olayinka A., Sachia G. Khasar, Nicole Alessandri-Haber, Paul G. Green, Robert O. Messing, and Jon D. Levine. "Alcohol-induced stress in painful alcoholic neuropathy." European Journal of Neuroscience 27, no. 1 (2007): 83–92. http://dx.doi.org/10.1111/j.1460-9568.2007.05987.x.

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21

Gane, Edward, Rachael Bergman, and David Hutchinson. "Resolution of alcoholic neuropathy following liver transplantation." Liver Transplantation 10, no. 12 (2004): 1545–48. http://dx.doi.org/10.1002/lt.20282.

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22

Mehta, Shashi Dhar, Umesh Kumar, Anil Kumar Mavi, Vinod Kumar, Suhel Parvez, and Suhasini Bhatnagar. "Effect of Allium Sativum on the Alcohol Induced Neuropathic Pain in Wistar Rats." Defence Life Science Journal 7, no. 4 (2022): 248–56. http://dx.doi.org/10.14429/dlsj.7.17774.

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Neuropathic pain is a severe pain condition associated with pathological changes which have deleterious effects like dysfunction of the brain. Alcohol abuse is known to be the most common reason at present for developing neuropathic pain. Alcohol-induced neuropathic pain can be assessed by ache studies such as hyperalgesia and allodynia which characterizes the response severity of induced pain. In the current research, we used the Allium Sativum (Garlic) bulb extract to study the effect on alcohol-administered rodents. The behavioral and biochemical studies have been done. It has been reported

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23

Bethiun, S. "Study of prevalence and determinants of peripheral neuropathy in patients with type 2 diabetes mellitus." MedPulse International Journal of Physiology 18, no. 3 (2021): 10–14. http://dx.doi.org/10.26611/1031832.

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Background: India is witnessing a depressing situation due to escalating incidence and prevalence of type 2 diabetes mellitus (T2DM) and its inevitable outcomes of cardiovascular diseases (CVD), diabetic neuropathy, nephropathy and retinopathy. Present study was aimed to investigate peripheral neuropathy in adult patients with type 2 diabetes. Material and Methods: Present study was hospital based, prospective, observational study conducted in subjects recruited from the diabetes OPD of both gender ≥ 30 years, known case of type 2 diabetes mellitus. The screening for DPN was conducted using th

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24

Vujisic, Slavica, Ljiljana Radulovic, Sladjana Knezevic-Apostolski, Stevan Petkovic, Filip Vukmirovic, and Slobodan Apostolski. "Disulfiram-induced polyneurophaty." Vojnosanitetski pregled 69, no. 5 (2012): 453–75. http://dx.doi.org/10.2298/vsp1205453v.

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Introduction. Disulfiram is used in the treatment of chronic alcoholism, because of the unpleasant symptoms produced after ethanol intake. Although it is well tolerated in most patients, one in 15,000 patients will develop peripheral neuropathy every year, which is frequently misdiagnosed as alcoholic neuropathy. Case report. We report clinical, laboratory, electrophysiological and histopathological features in a 19-year-old patient who developed an acute distal sensorymotor neuropathy during the treatment of alcoholism. At the end of 4-month treatment with disulfiram 250 mg/day, the patient c

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25

Chopra, Kanwaljit, and Vinod Tiwari. "Alcoholic neuropathy: possible mechanisms and future treatment possibilities." British Journal of Clinical Pharmacology 73, no. 3 (2012): 348–62. http://dx.doi.org/10.1111/j.1365-2125.2011.04111.x.

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26

Paparrigopoulos, Thomas, Elias Tzavellas, Dimitris Karaiskos, and Ioannis Liappas. "Alcoholic Optic Neuropathy: Another Complication of Alcohol Abuse." Journal of Neuropsychiatry and Clinical Neurosciences 20, no. 3 (2008): 368–69. http://dx.doi.org/10.1176/jnp.2008.20.3.368a.

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27

Hendrickse, M. T. "QT interval, autonomic neuropathy, and alcoholic liver disease." Lancet 342, no. 8862 (1993): 61. http://dx.doi.org/10.1016/0140-6736(93)91933-d.

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28

Gonzalez-Reimers, E., M. Alonso-Socas, F. Santolaria-Fernandez, J. Hernandez-Peña, A. Conde-Martel, and F. Rodriguez-Moreno. "Autonomic and peripheral neuropathy in chronic alcoholic liver disease." Drug and Alcohol Dependence 27, no. 3 (1991): 219–22. http://dx.doi.org/10.1016/0376-8716(91)90004-i.

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29

Tabaraud, F., J. M. Vallat, J. Hugon, H. Ramiandrisoa, M. Dumas, and J. L. Signoret. "Acute or subacute alcoholic neuropathy mimicking Guillain-Barré syndrome." Journal of the Neurological Sciences 97, no. 2-3 (1990): 195–205. http://dx.doi.org/10.1016/0022-510x(90)90218-c.

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30

MENEZO ROZALEN, JL, JA AVINO-MARTINEZ, C. PERIS-MARTINEZ, E. ESPANA-GREGORI, and FJ ROMERO. "Effect of different antioxidants in experimental alcoholic optic neuropathy." Acta Ophthalmologica 86 (September 4, 2008): 0. http://dx.doi.org/10.1111/j.1755-3768.2008.423.x.

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31

Anand, G. Joshi, and N. Patil Shrirang. "Electrophysiological Evaluation of Peripheral Neuropathy in Chronic Alcoholic Patients." Biomedical and Pharmacology Journal 13, no. 1 (2020): 475–80. http://dx.doi.org/10.13005/bpj/1908.

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32

Tan, E. T. H., D. G. Lambie, R. H. Johnson, B. J. Robinson, and E. A. Whiteside. "Sleep apnoea in alcoholic patients after withdrawal." Clinical Science 69, no. 6 (1985): 655–61. http://dx.doi.org/10.1042/cs0690655.

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1. Respiration during sleep was studied in 16 withdrawn alcoholic patients and in 12 control subjects. 2. The alcoholic patients had increased numbers of central (P < 0.01) and of obstructive (P < 0.05) apnoea and of hypopnoea episodes (P < 0.01) as compared with controls. 3. Significant positive associations were found between the frequencies of central apnoea (P < 0.05) or hypopnoea (P < 0.01) and clinical evidence of central nervous system damage in the alcoholic patients. Hypopnoea also showed a significant association with vagal neuropathy (P < 0.05), assessed by tests o

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33

Golovacheva, V. A. "Early diagnosis and treatment of diabetic and alcoholic polyneuropathy in outpatient practice." Neurology, Neuropsychiatry, Psychosomatics 12, no. 6 (2020): 96–103. http://dx.doi.org/10.14412/2074-2711-2020-6-96-103.

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Diabetes mellitus (DM) and chronic alcoholism (CA) are diseases that damage many organs and systems of the body and, in particular, lead to peripheral neuropathies. The pathogenesis of peripheral neuropathies caused by diabetes mellitus (DM) and CA is complex and diverse. Different types of peripheral neuropathies develop according to the leading pathogenetic mechanism. The most common type of peripheral neuropathy in DM is diabetic distal symmetric polyneuropathy (DSPN) and that in CA is alcoholic polyneuropathy (APN). The principles of diagnosis and treatment of DSPN and APN are considered.

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34

Koryagina, Anna D., Andrei V. Murashko, and Ekaterina V. Popova. "Peripheral neuropathy occurring during pregnancy (literature review)." V.F.Snegirev Archives of Obstetrics and Gynecology 10, no. 3 (2023): 187–93. http://dx.doi.org/10.17816/2313-8726-2023-10-3-187-193.

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Peripheral neuropathy is a general scientific term used to describe disorders of the peripheral nerves of any etiology. Along with postinfectious, diabetic, paraneoplastic, and alcoholic neuropathy, neuropathy of pregnancy represents one of the forms of this pathology. In this study, the authors conducted an analysis of domestic and foreign literature from open sources over the past 20 years. The study of available data revealed that the most common peripheral neuropathies observed during pregnancy are carpal tunnel syndrome, Bells palsy, and neuropathies affecting the lower extremities. Despi

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35

Pradana, Genta, and Drajad Priyono. "Intoksikasi Alkohol Akibat Minuman Keras Oplosan." Jurnal Kesehatan Andalas 10, no. 1 (2021): 65. http://dx.doi.org/10.25077/jka.v10i1.1689.

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The incidence of alcoholic liquor poisoning (alcoholic liquor) in Indonesia shows a high number. One of the causes of alcohol poisoning is methanol. Methanol poisoning in Indonesia usually occurs as a result of drinking mixed alcohol liquor. It has been reported a male 23 years old with decreased consciousness after drinking oplosan alcohol suspected to contain methanol. The examination reveals that patients has metabolic acidosis, toxic optic neuropathy (TON) and erosive gastritis. Treatment was acidosis correction, prevent formation of metabolites, hemodialysis with supportive and symptomati

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36

Kempler, P., K. Keresztes, I. Istenes, É. Kádár, K. Buzási, and Zs Hermányi. "Autonomic neuropathy and QT-interval prolongation in nonalcoholic diabetics, nondiabetic alcoholics and in alcoholic diabetic patients." Diabetes Research and Clinical Practice 50 (September 2000): 269–70. http://dx.doi.org/10.1016/s0168-8227(00)80917-1.

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37

Kashkin, Vladimir A. "Analgesic effect of cardiotonic steroids in rats with alcoholic neuropathy." Integrative Physiology 1, no. 2 (2020): 133–39. http://dx.doi.org/10.33910/2687-1270-2020-1-2-133-139.

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38

Ningshen, Robinson, SA Rajeev, RK Bikramjit Singh, O. Punyabati, and SBhagyabati Devi. "Evaluation of autonomic neuropathy in alcoholic liver disease in Manipur." Journal of Medical Society 31, no. 3 (2017): 152. http://dx.doi.org/10.4103/jms.jms_31_14.

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39

Sachedina, Shafina, and Cory Toth. "Progression in idiopathic, diabetic, paraproteinemic, alcoholic, and B12 deficiency neuropathy." Journal of the Peripheral Nervous System 18, no. 3 (2013): 247–55. http://dx.doi.org/10.1111/jns5.12042.

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40

Paladin, F., and Russo Perez. "The Haematic Thiamine Level in the Course of Alcoholic Neuropathy." European Neurology 26, no. 3 (1987): 129–33. http://dx.doi.org/10.1159/000116324.

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41

Lee, Kwang-woo, Sung-Hyun Lee, Han-bo Lee, and Seong-ho Park. "PS-22-3 Clinical and electrophysiological characteristics of alcoholic neuropathy." Electroencephalography and Clinical Neurophysiology/Electromyography and Motor Control 97, no. 4 (1995): S138. http://dx.doi.org/10.1016/0924-980x(95)92932-c.

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42

Decaux, G., P. Cauchie, A. Soupart, M. Kruger, and F. Delwiche. "Role of vagal neuropathy in the hyponatraemia of alcoholic cirrhosis." BMJ 293, no. 6561 (1986): 1534–36. http://dx.doi.org/10.1136/bmj.293.6561.1534.

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43

Navarro, Xavier, Ramon Miralles, Josep M. Espadaler, and Juan Rubiés-Prat. "Comparison of sympathetic sudomotor and skin responses in alcoholic neuropathy." Muscle & Nerve 16, no. 4 (1993): 404–7. http://dx.doi.org/10.1002/mus.880160410.

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44

Donnadieu-Rigole, Hélène, Vincent Daien, Dominique Blanc, et al. "The Prevalence of Optic Neuropathy in Alcoholic Patients-A Pilot Study." Alcoholism: Clinical and Experimental Research 38, no. 7 (2014): 2034–38. http://dx.doi.org/10.1111/acer.12468.

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45

Haridas, V. T., A. B. Taly, M. Pratima, and S. Rao. "PO31-FR-05 Sympathetic skin response [SSR] – inferences from alcoholic neuropathy." Journal of the Neurological Sciences 285 (October 2009): S321. http://dx.doi.org/10.1016/s0022-510x(09)71223-7.

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46

Paneyala, Shasthara, Nemichandra S. C., Harsha Sundaramurthy, and Vimala Christina Colaco K. "Neuropathy in the setting of alcoholism-an entity less thought of." International Journal of Research in Medical Sciences 8, no. 12 (2020): 4518. http://dx.doi.org/10.18203/2320-6012.ijrms20205336.

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Disulfiram is a commonly used adjunctive treatment in the management of alcohol dependency. It has been noted that disulfiram can induce peripheral neuropathy, the mechanism of which has not been clearly determined. A 35-year-old patient, reformed alcoholic, on disulfiram presented with complaints of painful distal dysesthesias and foot drop. Clinical examination revealed bilateral foot drop without any objective sensory loss. Patient was evaluated for the same and routine blood investigations including vitamin B-12, inflammatory and virological markers were found to be normal. Nerve conductio

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47

Pratim Dowarah, Bhatri. "Alcoholic Neuropathy and Its Impact among the Tea Garden Workers in the Golaghat District of Assam." International Journal of Science and Research (IJSR) 12, no. 1 (2023): 483–86. http://dx.doi.org/10.21275/sr23110135329.

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48

Yakovlev, A., S. Karasev, and V. Yakovleva. "434 SPINAL CORD STIMULATION FOR TREATMENT OF ALCOHOLIC NEUROPATHY; A CASE REPORT." European Journal of Pain Supplements 4, S1 (2010): 123. http://dx.doi.org/10.1016/s1754-3207(10)70439-9.

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49

D'Amour, Monique L., and Roger F. Butterworth. "Pathogenesis of alcoholic peripheral neuropathy: Direct effect of ethanol or nutritional deficit?" Metabolic Brain Disease 9, no. 2 (1994): 133–42. http://dx.doi.org/10.1007/bf01999766.

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50

Hendrickse, MT, and DR Triger. "Autonomic and peripheral neuropathy in alcoholic liver disease and primary biliary cirrhosis." Journal of Hepatology 13 (January 1991): S130. http://dx.doi.org/10.1016/0168-8278(91)91495-3.

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