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Journal articles on the topic 'Aldara'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 April 2022

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1

Zaslavsky, D. V., I. N. Chuprov, A. A. Sidikov та ін. "Application of a preparation of imiquimod in а dosage form of ointment 5% in therapy of basal cell carcinoma". Vestnik dermatologii i venerologii 91, № 6 (2015): 67–74. http://dx.doi.org/10.25208/0042-4609-2015-91-6-67-74.

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Objective. To estimate efficiency and safety of application of a preparation of Aldara (imiquimod, 5% external application cream) it is independent or in combination with cryotherapy in therapy of the basal cell carcinoma of the skin (BCC). Material and methods. Research included 78 patients (22 women and 56 men) with various forms of a basal cell carcinoma of the skin. All patients were divided into 3 groups: the main group (n = 30) the patients receiving therapy by Aldara (imiquimod, 5% external application cream) made, second (n = 21) - the patients receiving therapy by Aldara’s, and also 1

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2

Buck, H. W. "Imiquimod (Aldara? cream)." Infectious Diseases in Obstetrics and Gynecology 6, no. 2 (1998): 49–51. http://dx.doi.org/10.1002/(sici)1098-0997(1998)6:2<49::aid-idog3>3.0.co;2-2.

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3

Goldstein, Jérémie D., Esen Y. Bassoy, Assunta Caruso, et al. "IL-36 signaling in keratinocytes controls early IL-23 production in psoriasis-like dermatitis." Life Science Alliance 3, no. 6 (2020): e202000688. http://dx.doi.org/10.26508/lsa.202000688.

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IL-36R signaling plays an important role in the pathogenesis of psoriasis. We ought to assess the specific function of IL-36R in keratinocytes for the pathology of Aldara-induced psoriasis-like dermatitis. Il36rΔK mice presenting deletion of IL-36R in keratinocytes were similarly resistant to Aldara-induced ear inflammation as Il36r−/− mice, but acanthosis was only prevented in Il36r−/− mice. FACS analysis revealed that IL-36R signaling in keratinocytes is mandatory for early neutrophil infiltration in Aldara-treated ears. RNASeq and qRT-PCR experiments demonstrated the crucial role of IL-36R

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4

HB, Slade, Owens ML, Tomai MA, and Miller RL. "Imiquimod 5% cream (Aldara™)." Expert Opinion on Investigational Drugs 7, no. 3 (1998): 437–49. http://dx.doi.org/10.1517/13543784.7.3.437.

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5

Lyons, Joseph M., James I. Ito, and Servaas A. Morré. "Efficacy of an Immune Modulator in ExperimentalChlamydia trachomatisInfection of the Female Genital Tract." Infectious Diseases in Obstetrics and Gynecology 2006 (2006): 1–6. http://dx.doi.org/10.1155/idog/2006/61265.

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Objective.The aim of this study was to determine if vaginal application of the immune response modifier imiquimod (Aldara cream, 3M Pharmaceuticals, St Paul, Minn) would alter the course and/or outcome of female genital tract infection with a human isolate ofChlamydia trachomatisin a murine model.Methods.Groups of CF-1 mice were treated with Aldara on three different schedules: (1) ongoing beginning 5 days prior to and continuing through day 5 of infection; (2) a single prophylactic dose 2 hours prior to infection; and (3) therapeutic from day 4 to day 14 of infection. Mice were infected vagin

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6

Hesterberg, Böhlen, and Brand. "Imiquimod in der Behandlung von therapieresistenten Verrucae – eine neue Therapieoption?" Praxis 92, no. 12 (2003): 535–39. http://dx.doi.org/10.1024/0369-8394.92.12.535.

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Les verrues récalcitrantes peuvent poser un problème thérapeutique pour le médecin traitant dans la pratique quotidienne. Nous avons étudié l'efficacité de la crème Imiquimod (Aldara®) chez 22 patient(e)s souffrantes de verrues récalcitrantes. Une rémission complète a pu être constatée dans 41% et une réduction partielle dans 50% des patient(e)s. Dans 9% des cas seulement, Imiquimod ne produisait aucun effet. À part un érythème perilésionnel fréquemment observé et étant souvent associé à une réponse favorable au traitement, il n'y avait pas d'effets secondaires significatifs. Dans le traitemen

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7

Di Fusco, Davide, Carmine Stolfi, Antonio Di Grazia, et al. "Albendazole negatively regulates keratinocyte proliferation." Clinical Science 134, no. 7 (2020): 907–20. http://dx.doi.org/10.1042/cs20191215.

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Abstract Background: Increased keratinocyte proliferation occurs in the skin of psoriatic patients and is supposed to play a role in the pathogenesis of this disorder. Compounds interfering with keratinocyte proliferation could be useful in the management of psoriatic patients. Aim: To investigate whether albendazole, an anti-helmintic drug that regulates epithelial cell function in various systems, inhibits keratinocyte proliferation in models of psoriasis. Methods: Aldara-treated mice received daily topical application of albendazole. Keratinocyte proliferation and keratin (K) 6 and K16 expr

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8

Wendling, Patrice. "Long-Term Use Of Aldara Safe In AK Patients." Skin & Allergy News 36, no. 4 (2005): 12. http://dx.doi.org/10.1016/s0037-6337(05)70086-3.

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9

Campanelli, A., D. Salomon, and J. H. Saurat. "Traitement des kératoses actiniques par imiquimod topique (Aldara®)." Annales de Dermatologie et de Vénéréologie 131, no. 4 (2004): 387–88. http://dx.doi.org/10.1016/s0151-9638(04)93622-8.

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10

Carraway, J. "Using Aldara, copper peptide, and niacinamide for skin care." Aesthetic Surgery Journal 24, no. 1 (2004): 83–84. http://dx.doi.org/10.1016/j.asj.2003.10.009.

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11

&NA;. "FDA Approval for Aldara Cream for Superficial Basal Cell Carcinoma." Oncology Times 26, no. 15 (2004): 20. http://dx.doi.org/10.1097/01.cot.0000293176.77573.96.

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12

Prado, Arturo, Patricio Andrades, and Susana Benitez. "PRELIMINARY RESULTS AFTER FACE PEELS USING ALDARA CREAM 5% (IMIQUIMOD)." Plastic and Reconstructive Surgery 116, no. 5 (2005): 1563–68. http://dx.doi.org/10.1097/01.prs.0000184345.09254.61.

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13

Horváth, S., Kemény, R. Komlódi, A. Perkecz, E. Pintér, and R. Gyulai. "LB1561 Localized Aldara-induced psoriasiform dermatitis model using Finn chambers." Journal of Investigative Dermatology 138, no. 9 (2018): B16. http://dx.doi.org/10.1016/j.jid.2018.06.097.

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14

Vinter, H., L. Iversen, T. Steiniche, K. Kragballe, and C. Johansen. "Aldara®-induced skin inflammation: studies of patients with psoriasis." British Journal of Dermatology 172, no. 2 (2014): 345–53. http://dx.doi.org/10.1111/bjd.13236.

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15

Mrowietz, U. "Aldara-induced skin inflammation in mice: close enough to psoriasis?" British Journal of Dermatology 172, no. 2 (2015): 313. http://dx.doi.org/10.1111/bjd.13602.

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16

Bourgognon, J., M. Suessmilch, A. McColl, and J. Cavanagh. "Abstract #4297 Blood brain barrier breakdown following topical aldara treatment." Brain, Behavior, and Immunity 81 (October 2019): 24–25. http://dx.doi.org/10.1016/j.bbi.2019.08.086.

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17

E Perlman, Sally. "Aldara™ as treatment for condylomata acuminata in the prepubertal child." Journal of Pediatric and Adolescent Gynecology 14, no. 3 (2001): 148–49. http://dx.doi.org/10.1016/s1083-3188(01)00101-2.

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18

Baumann, Leslie S., and Monica L. Halem. "Lip Silicone Granulomatous Foreign Body Reaction Treated With Aldara (Imiquimod 5%)." Dermatologic Surgery 29, no. 4 (2003): 429–32. http://dx.doi.org/10.1046/j.1524-4725.2003.29102.x.

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19

BAUMANN, LESLIE S., and MONICA L. HALEM. "Lip Silicone Granulomatous Foreign Body Reaction Treated With Aldara (Imiquimod 5%)." Dermatologic Surgery 29, no. 4 (2003): 429–32. http://dx.doi.org/10.1097/00042728-200304000-00023.

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20

Xie, Sijing, Zhonglan Su, Bin Zhang, et al. "SIRT1 Activation Ameliorates Aldara-Induced Psoriasiform Phenotype and Histology in Mice." Journal of Investigative Dermatology 135, no. 7 (2015): 1915–18. http://dx.doi.org/10.1038/jid.2015.82.

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21

Horváth, S., Á. Kemény, A. Perkecz, E. Pintér, and R. Gyulai. "434 Development of Aldara-induced chronic (15-day) psoriasiform dermatitis model." Journal of Investigative Dermatology 139, no. 9 (2019): S289. http://dx.doi.org/10.1016/j.jid.2019.07.436.

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22

Shibuya, R., A. Kitoh, and K. Kabashima. "619 The C5a-C5aR1 axis suppresses the Aldara-induced immune activation." Journal of Investigative Dermatology 137, no. 5 (2017): S107. http://dx.doi.org/10.1016/j.jid.2017.02.641.

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23

Maybury, C. M., P. Di Meglio, and C. H. Smith. "Aldara-induced dermatitis is associated with development of liver fibrosis in mice." British Journal of Dermatology 179, no. 1 (2018): 9–10. http://dx.doi.org/10.1111/bjd.16693.

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24

Poirier, Nicolas, Mélanie Chevalier, Caroline Mary, et al. "Selective CD28 antagonist prevents Aldara-induced skin inflammation in non-human primates." Experimental Dermatology 25, no. 3 (2016): 233–34. http://dx.doi.org/10.1111/exd.12891.

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25

Rather, Irfan Ahmad, Vivek K. Bajpai, Jonghun Han, and Gyeong-Jun Nam. "Imiquimod-induced psoriasis-like skin inflammation in mouse model." Bangladesh Journal of Pharmacology 11, no. 4 (2016): 849. http://dx.doi.org/10.3329/bjp.v11i4.28662.

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<p>Psoriasis is a skin disorder characterized by skin inflammation and plaques. Induction of psoriasis in animal model include following steps: a) Selection of animal model, b) Hair removing from the back or ear, c) treatment of skin with Aldara, a cream containing 5% imiquimod and d) Observation. Imiquimod-induced skin inflammation in animal model resembles with psoriasis.</p><p><strong>Video Clip:</strong></p><p><a href="https://youtube.com/v/QY-RESHbH-U">Psoriaisis-like skin:</a> 9 min 10 sec</p>

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26

Atanaskova Mesinkovska, Natasha, Danyelle Dawes, Apra Sood, and Wilma Bergfeld. "Acantholytic Pityriasis Rubra Pilaris Associated with Imiquimod 3.75% Application." Case Reports in Dermatological Medicine 2011 (2011): 1–3. http://dx.doi.org/10.1155/2011/412684.

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Imiquimod is an immunomodulator with both antitumor and antiviral properties. It is currently available in two cream formulations as Aldara (imiquimod 5%) and the newly approved Zyclara (imiquimod 3.75%). Imiquimod has been associated with localized erythema, crusting, and scaling at the site of application. However, more severe generalized skin eruptions including erythema multiforme, psoriasis, and hyperpigmentation have been described. The newly approved imiquimod 3.75% cream is a presumably safer alternative due to its lower concentration. This paper describes the development of generalize

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27

Ryu, Jubin, and F. Clarissa Yang. "A Review of Topical Imiquimod in the Management of Basal Cell Carcinoma, Actinic Keratoses, and Other Skin Lesions." Clinical Medicine. Therapeutics 1 (January 2009): CMT.S1969. http://dx.doi.org/10.4137/cmt.s1969.

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Imiquimod (trade name Aldara™) is a small molecule of the imidazoquinoline family, a group of nucleoside analogs that were first synthesized as potential antiviral agents. It has since been discovered to activate both innate and adaptive immunity, as well as apoptosis. Clinically, it has been approved for three indications thus far: external genital warts, actinic keratosis, and superficial basal cell carcinoma. In addition to these applications, a number of off-label uses have been reported in the literature. In this review, we summarize and discuss the literature describing imiquimod's mecha

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28

Balducci, Mario, Berardino De Bari, Stefania Manfrida, Giuseppe Roberto D'Agostino, and Vincenzo Valentini. "Treatment of Merkel Cell Carcinoma with Radiotherapy and Imiquimod (Aldara): A Case Report." Tumori Journal 96, no. 3 (2010): 508–11. http://dx.doi.org/10.1177/030089161009600324.

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29

Bezdek, Siegfried, Ashref Hdnah, Tanya Sezin, et al. "The genetic difference betweenC57Bl/6JandC57Bl/6Nmice significantly impacts Aldara™-induced psoriasiform dermatitis." Experimental Dermatology 26, no. 4 (2016): 349–51. http://dx.doi.org/10.1111/exd.13131.

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30

Prinz, Immo, та Inga Sandrock. "γδ T cells come to stay: Innate skin memory in the Aldara model". European Journal of Immunology 45, № 11 (2015): 2994–97. http://dx.doi.org/10.1002/eji.201546033.

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31

Neu, Savannah D., Anna Strzepa, Dustin Martin, Mary G. Sorci-Thomas, Kirkwood A. Pritchard, and Bonnie N. Dittel. "Myeloperoxidase Inhibition Ameliorates Plaque Psoriasis in Mice." Antioxidants 10, no. 9 (2021): 1338. http://dx.doi.org/10.3390/antiox10091338.

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Plaque psoriasis is a common inflammatory condition of the skin characterized by red, flaking lesions. Current therapies for plaque psoriasis target many facets of the autoimmune response, but there is an incomplete understanding of how oxidative damage produced by enzymes such as myeloperoxidase contributes to skin pathology. In this study, we used the Aldara (Imiquimod) cream model of plaque psoriasis in mice to assess myeloperoxidase inhibition for treating psoriatic skin lesions. To assess skin inflammation severity, an innovative mouse psoriasis scoring system was developed. We found that

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32

Roukens, Anna H., Ann C. Vossen, Greet J. Boland, Willem Verduyn, Jaap T. van Dissel, and Leo G. Visser. "Intradermal hepatitis B vaccination in non-responders after topical application of imiquimod (Aldara®)." Vaccine 28, no. 26 (2010): 4288–93. http://dx.doi.org/10.1016/j.vaccine.2010.04.029.

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33

Sezin, T., D. Zillikens, and C. Sadik. "Leukotrienes Do Not Modulate the Course of Aldara™-induced Psoriasiform Dermatitis in Mice." Acta Dermato Venereologica 95, no. 3 (2015): 341–42. http://dx.doi.org/10.2340/00015555-1924.

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34

Al-Mayahy, Mohammed Hussain, Maria Marlow, and David J. Scurr. "The Complementary Role of ToF-SIMS in the Assessment of Imiquimod Permeated into the Skin from a Microemulsion Dosage Form." Al Mustansiriyah Journal of Pharmaceutical Sciences 19, no. 4 (2019): 196–210. http://dx.doi.org/10.32947/ajps.v19i4.650.

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The assessment of drug permeation into/across the skin is traditionally accomplished using Franz diffusion cells with subsequent analysis by conventional chromatographic methods such as HPLC and more recently using advanced imaging techniques. In this context, time  of flight-secondary ion mass spectrometry (ToF-SIMS) offers distinctive advantages in mapping drugs within skin with high sensitivity and chemical specificity without the need for fluorescent tags or radiolabels. The work in this paper uses the combination of conventional and advanced methods to evaluate imiquimod perme

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35

Gupta, Aditya K., Melanie Browne, and Robyn Bluhm. "Imiquimod: A Review." Journal of Cutaneous Medicine and Surgery 6, no. 6 (2002): 554–60. http://dx.doi.org/10.1177/120347540200600607.

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Background and Objective: Imiquimod (Aldara) is an immune response modifier used primarily to treat anogenital warts. Imiquimod induces cytokines and enhances cell-mediated cytolytic antiviral activity in vivo. It exhibits antiviral and antitumor effects; however, it does not exhibit direct antiviral effects in vitro. By inducing cytokines, such as interferon A, imiquimod stimulates both the innate immune response and the cellular arm of acquired immunity. Currently, imiquimod is approved for use in the treatment of external genital and perianal warts in adults. Conclusion: Beyond its establis

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36

Owens, M. L., L. Edwards, A. Ferenczy, K. F. Trofatter, T. L. Fox, and M. H. Smith. "Imiquimod 5% cream (aldara) is effective and safe in women with external genital/perianal warts." International Journal of Gynecology & Obstetrics 70 (2000): B132. http://dx.doi.org/10.1016/s0020-7292(00)83127-6.

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37

Owens, Mary L., William E. Bridson, Sheri L. Smith, Jane A. Myers, Terry L. Fox, and Teresa M. Wells. "Percutaneous penetration of Aldara™ cream, 5% during the topical treatment of genital and perianal warts." Primary Care Update for OB/GYNS 5, no. 4 (1998): 151. http://dx.doi.org/10.1016/s1068-607x(98)00034-1.

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38

Kwon, Hyok Bu, Yun Seok Choi, Joon Ho Lee, et al. "Treatment of Verrucous Carcinoma of the Lower Lip with Topical Imiquimod (Aldara®) and Debulking Therapy." Annals of Dermatology 23, Suppl 1 (2011): S68. http://dx.doi.org/10.5021/ad.2011.23.s1.s68.

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39

Vinter, H., K. Kragballe, T. Steiniche, M. Gaestel, L. Iversen та C. Johansen. "Tumour necrosis factor-α plays a significant role in the Aldara-induced skin inflammation in mice". British Journal of Dermatology 174, № 5 (2016): 1011–21. http://dx.doi.org/10.1111/bjd.14320.

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40

Gerlach, Justin. "Short-term climate change and the extinction of the snail Rhachistia aldabrae (Gastropoda: Pulmonata)." Biology Letters 3, no. 5 (2007): 581–85. http://dx.doi.org/10.1098/rsbl.2007.0316.

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The only known population of the Aldabra banded snail Rhachistia aldabrae declined through the late twentieth century, leading to its extinction in the late 1990s. This occurred within a stable habitat and its extinction is attributable to decreasing rainfall on Aldabra atoll, associated with regional changes in rainfall patterns in the late twentieth and early twenty-first century. It is proposed that the extinction of this species is a direct result of decreasing rainfall leading to increased mortality of juvenile snails.

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41

Artola, Koldo. "Gulibarko aldaeraren inguruan (eta 3 – Txulapaingo azpialdaera: Elbarrena)." Fontes Linguae Vasconum, no. 131 (June 30, 2021): 9–46. http://dx.doi.org/10.35462/flv131.1.

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Louis-Lucien Bonaparte printzeak, euskara sailkatzeko bere ahaleginean, nafar Pirinioko bere talaiatik so eginez nonbait, Iruñez haraindiko multzo handi bati aldaera gisako kategoria iritzi eta subdialecto ultra-pamplonés gisako deitura eman zion, honen barruan Gulibarko hau, Oltzako eta Zizurko aldaerak kokatuz, guztiak ere hegoaldeko goi-nafarrera deitu zuen euskalkiaren barruan. Saio honen azpitituluan dakusagunez, printzeak Gulibarko aldaera honen baitan tartekatu eta de Juslapeña izendatu zuen azpialdaera ezagutzeko ekarpena egiten saiatu gara, duela hogeita hamar urte inguru bildu genitu

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42

Stein, Pamela, Karsten Gogoll, Tanja Peters, et al. "Transcutaneous Immunization with a Solid Nanoscopic Imiquimod Suspension Enhances Tumor Rejection." Blood 126, no. 23 (2015): 2224. http://dx.doi.org/10.1182/blood.v126.23.2224.2224.

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Abstract Introduction: Transcutaneous immunization (TCI) is a novel vaccination strategy to induce strong therapeutic cytotoxic T-lymphocyte (CTL) responses by directly targeting skin-resident professional antigen-presenting cells (APC). This vaccination approach is very promising to overcome current limitations of standard vaccination approaches that are mostly effective in prophylaxis, but not in the treatment of diseases. In this context, we have developed a TCI method based on a synthetic TLR7 agonist imiquimod that partial tumor protection in experimental rodent models. In our present wor

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43

Avcilar, Huseyin, and Ahmet Eken. "Could imiquimod (Aldara 5% cream) or other TLR7 agonists be used in the treatment of COVID-19?" Medical Hypotheses 144 (November 2020): 110202. http://dx.doi.org/10.1016/j.mehy.2020.110202.

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44

Leroy, B., S. Ahbib, L. Marot, and P. Vereecken. "P076. Sequential treatment with MAL-PDT and imiquimod (Aldara®) in difficult-to-treat basal cell carcinomas." Melanoma Research 21 (June 2011): e59. http://dx.doi.org/10.1097/01.cmr.0000399537.31086.16.

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45

DeRoberts, Dean, and Joseph A. Molnar. "P44: Intense Pulsed Light Tattoo Removal in Conjunction with Imiquimod (Aldara??): A Double-Blind, Randomized Controlled Study." Plastic and Reconstructive Surgery 118, Supplement (2006): 125–26. http://dx.doi.org/10.1097/00006534-200609151-00141.

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46

Hume, Julian P., and David Martill. "Repeated evolution of flightlessness in Dryolimnas rails (Aves: Rallidae) after extinction and recolonization on Aldabra." Zoological Journal of the Linnean Society 186, no. 3 (2019): 666–72. http://dx.doi.org/10.1093/zoolinnean/zlz018.

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AbstractThe Aldabra rail, Dryolimnas cuvieri subsp. aldabranus, endemic to the Aldabra Atoll, Seychelles, is the last surviving flightless bird in the Indian Ocean. Aldabra has undergone at least one major, total inundation event during an Upper Pleistocene (Tarantian age) sea-level high-stand, resulting in the loss of all terrestrial fauna. A flightless Dryolimnas has been identified from two temporally separated Aldabran fossil localities, deposited before and after the inundation event, providing irrefutable evidence that a member of Rallidae colonized the atoll, most likely from Madagascar

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47

Kemény, Lajos, and Nikoletta Nagy. "New perspective in immunotherapy: local imiquimod treatment." Orvosi Hetilap 151, no. 19 (2010): 774–83. http://dx.doi.org/10.1556/oh.2010.28866.

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Az imikimod, az imidazol-kinolin-aminok családjába tartozó, kis, szintetikus, nukleotidszerű molekula. Ismert immunválasz-módosító, vírusellenes és tumorellenes hatása, amelyet a Toll-like receptorok (TLR7 és TLR8) közvetítenek. Az imikimod főként a TLR7-et kifejező plazmacitoid dendritikus sejteket és a Langerhans-sejteket célozza meg, és csak másodlagosan vesz részt egyéb gyulladásos sejtek aktiválásában. A TLR7 stimulációjakor indukálódó jelátviteli mechanizmusok mind a veleszületett, mind a szerzett immunrendszert aktiválják, különös tekintettel a sejt által közvetített immunreakciókra. Az

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48

Langley, P., P. Salden, and M. Nuijten. "PIN7: PHARMACOECONOMIC EVALUATION OF IMIQUIMOD (ALDARA) FOR THE TREATMENT OF EXTERNAL GENITAL WARTS IN A DUTCH TREATMENT ENVIRONMENT." Value in Health 4, no. 6 (2001): 441. http://dx.doi.org/10.1016/s1098-3015(11)71586-8.

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49

Grine, L., C. Libert та R. E. Vandenbroucke. "Comment on ‘Tumour necrosis factor‐α plays a significant role in the Aldara‐induced skin inflammation in mice’". British Journal of Dermatology 174, № 6 (2016): 1419. http://dx.doi.org/10.1111/bjd.14453.

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50

Sverre, JM, FKO Kristensen, L. Hamel-Gariépy, and T. Albrektsen. "PSN2 COST-EFFECTIVENESS ANALYSIS: ALDARA™ (IMIQUIMOD) CREAM, 5% IN THE TREATMENT OF SUPERFICIAL BASAL CELL CARCINOMA IN NORWAY." Value in Health 8, no. 6 (2005): A143. http://dx.doi.org/10.1016/s1098-3015(10)67587-0.

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