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Journal articles on the topic 'Alpha-1,3-glucan'

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Published: 4 June 2021
Last updated: 10 March 2023

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1

Koneti, Adi, Michael J. Linke, Elmer Brummer, and David A. Stevens. "Evasion of Innate Immune Responses: Evidence for Mannose Binding Lectin Inhibition of Tumor Necrosis Factor Alpha Production by Macrophages in Response to Blastomyces dermatitidis." Infection and Immunity 76, no. 3 (2007): 994–1002. http://dx.doi.org/10.1128/iai.01185-07.

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ABSTRACT Serum factors, including mannose binding lectins (MBL), influence innate responses to microbes. Little is known about the effects of serum factors or MBL on the interaction of Blastomyces dermatitidis, a pulmonary fungal pathogen, with macrophages or on tumor necrosis factor alpha (TNF-α) production. Since macrophage production of TNF-α is an important innate immune response, we examined a mouse peritoneal macrophage (PM) cell line (RAW) and resident PM from CD-1 mice to study TNF-α production by PM stimulated with heat-killed (HK) or live B. dermatitidis yeast cells. Mouse serum and
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2

Steadman, R., M. M. Petersen, N. Topley, et al. "Differential augmentation by recombinant human tumor necrosis factor-alpha of neutrophil responses to particulate zymosan and glucan." Journal of Immunology 144, no. 7 (1990): 2712–18. http://dx.doi.org/10.4049/jimmunol.144.7.2712.

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Abstract Zymosan (Z) and its major insoluble carbohydrate component beta-linked glucan activate human neutrophils (PMN) through a trypsin-sensitive recognition mechanism. This mechanism is believed to involve the PMN CR3R. Both Z and glucan generated dose and time-dependent release of the secondary lysosomal granule marker vitamin B12 binding protein, leukotriene B4 (LTB4) and superoxide from PMN and were phagocytosed with similar dose-dependent kinetics. The PMN superoxide and LTB4 responses to glucan; however, were consistently greater than those to the same doses of Z. The phagocytosis of b
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3

Koschella, Andreas, Thomas Heinze, Antje Tied, Katja Geitel, Chih-Ying Chien та Tadahisa Iwata. "Comparative Studies on Regioselectivity of α- and β-Linked Glucan Tosylation". Molecules 25, № 22 (2020): 5382. http://dx.doi.org/10.3390/molecules25225382.

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Alpha- and beta-linked 1,3-glucans have been subjected to conversion with p-toluenesulfonic acid (tosyl) chloride and triethylamine under homogeneous reaction conditions in N,N-dimethyl acetamide/LiCl. Samples with a degree of substitution of tosyl groups (DSTs) of up to 1.91 were prepared by applying 5 mol reagent per mole repeating unit. Hence, the reactivity of α-1,3-glucan is comparable with cellulose and starch, while the β-1,3-linked glucan curdlan is less reactive. The samples dissolve in aprotic dipolar media independent of the DSTs and possess a solubility in less polar solvents that
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4

HANADA, Nobuhiro, Yoshihisa YAMASHITA, Hideo MIYAZAKI, and Tadamichi TAKEHARA. "Inhibition of glucan synthesis by antiserum against purified 1,3-.ALPHA.-D-glucan synthase from Streptococcus sobrinus." JOURNAL OF DENTAL HEALTH 39, no. 2 (1989): 211–16. http://dx.doi.org/10.5834/jdh.39.211.

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5

Noble, P. W., P. M. Henson, C. Lucas, M. Mora-Worms, P. C. Carré, and D. W. Riches. "Transforming growth factor-beta primes macrophages to express inflammatory gene products in response to particulate stimuli by an autocrine/paracrine mechanism." Journal of Immunology 151, no. 2 (1993): 979–89. http://dx.doi.org/10.4049/jimmunol.151.2.979.

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Abstract Macrophages maintain an essential role in orchestrating the host inflammatory response by selectively mobilizing portions of their large secretory repertoire in response to phagocytic as well as other stimuli. For example, after exposure to the inflammatory particulate stimulus zymosan (or its derivative beta 1,3-glucan), monocyte/macrophages synthesize and release lysosomal hydrolases, mobilize arachadonic acid, and secrete cytokines such as TNF-alpha and IL-8. However, the mechanisms by which particulate stimuli promote the selective synthesis and release of macrophage-derived infla
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6

Kottom, Theodore J., Deanne M. Hebrink, Paige E. Jenson, Gunnar Gudmundsson та Andrew H. Limper. "Evidence for Proinflammatory β-1,6 Glucans in the Pneumocystis carinii Cell Wall". Infection and Immunity 83, № 7 (2015): 2816–26. http://dx.doi.org/10.1128/iai.00196-15.

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Inflammation is a major cause of respiratory impairment duringPneumocystispneumonia. Studies support a significant role for cell wall β-glucans in stimulating inflammatory responses. Fungal β-glucans are comprised ofd-glucose homopolymers containing β-1,3-linked glucose backbones with β-1,6-linked glucose side chains. Prior studies inPneumocystis cariniihave characterized β-1,3 glucan components of the organism. However, recent investigations in other organisms support important roles for β-1,6 glucans, predominantly in mediating host cellular activation. Accordingly, we sought to characterize
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7

Klimpel, K. R., and W. E. Goldman. "Cell walls from avirulent variants of Histoplasma capsulatum lack alpha-(1,3)-glucan." Infection and Immunity 56, no. 11 (1988): 2997–3000. http://dx.doi.org/10.1128/iai.56.11.2997-3000.1988.

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8

Tintoré, Maria, Jordi Cuñé, Vaclav Vetvicka, and Carlos de Lecea. "Anti-Inflammatory Effects, Protection of Gut Barrier Integrity and Stimulation of Phagocytosis of Postbiotic Combination ABB C1." Nutraceuticals 3, no. 1 (2023): 109–18. http://dx.doi.org/10.3390/nutraceuticals3010009.

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This study evaluated the anti-inflammatory effects, the protection of gut barrier integrity, and the stimulation of phagocytosis in peripheral cells of a nutritional supplement based on a synergistic combination of yeast-based ingredients with a unique 1,3/1,6-glucan complex and a consortium of postbiotic Saccharomyces cerevisiae rich in selenium and zinc. The anti-inflammatory effect in caco-2 cells in the presence and absence of a pro-inflammatory challenge (tumour necrosis factor alpha [TNF-α]/interferon gamma [IFN-ɣ]) showed statistically significant reductions in IFN-ɣ induced protein-10
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9

Murray, P. D., S. L. Swain, and M. P. Kagnoff. "Regulation of the IgM and IgA anti-dextran B1355S response: synergy between IFN-gamma, BCGF II, and IL 2." Journal of Immunology 135, no. 6 (1985): 4015–20. http://dx.doi.org/10.4049/jimmunol.135.6.4015.

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Abstract T cell help is required for the induction of the humoral antibody response to dextran B1355S, a type II thymus-independent bacterial polysaccharide antigen. In the present study we have identified three B cell growth and differentiation factors that can substitute for T cells in the induction of IgM and IgA antibody responses to alpha(1,3) glucan determinants on dextran B1355S. Dextran B1355S stimulated murine B cell cultures supplemented with a combination of murine recombinant interferon-gamma (IFN-gamma) and a late-acting B cell growth and differentiation factor, BCGF II, produced
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10

Adibi, Azin, David Valdesueiro, Jorge Mok, et al. "Sustainable barrier paper coating based on alpha-1,3 glucan and natural rubber latex." Carbohydrate Polymers 282 (April 2022): 119121. http://dx.doi.org/10.1016/j.carbpol.2022.119121.

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11

Lenges, Christian, Natnael Behabtu, Jorge Mok, Ibrahim Sendijarevic, and Aisa Sendijarevic. "Engineered polysaccharide alpha‐1,3 glucan as isocyanate‐reactive component in viscoelastic polyurethane foams." Journal of Applied Polymer Science 138, no. 10 (2020): 49979. http://dx.doi.org/10.1002/app.49979.

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12

Soltys, Jindrich, та Mark T. Quinn. "Modulation of Endotoxin- and Enterotoxin-Induced Cytokine Release by In Vivo Treatment with β-(1,6)-Branched β-(1,3)-Glucan". Infection and Immunity 67, № 1 (1999): 244–52. http://dx.doi.org/10.1128/iai.67.1.244-252.1999.

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ABSTRACT Leukocytes activated by endotoxin or enterotoxins release proinflammatory cytokines, thereby contributing to the cascade of events leading to septic shock. In the present studies, we analyzed the effects of in vivo administration of a soluble immunomodulator, β-(1,6)-branched β-(1,3)-glucan (soluble β-glucan), on toxin-stimulated cytokine production in monocytes and lymphocytes isolated from treated mice. In vitro stimulation of lymphocytes isolated from soluble β-glucan-treated mice with lipopolysaccharide (LPS) resulted in enhanced production of interleukin-6 (IL-6) and suppressed p
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13

Hozová, B., Ľ. Kuniak, P. Moravčíková, and A. Gajdošová. "Determination of water-insoluble β-D-glucan in the whole-grain cereals and pseudocereals." Czech Journal of Food Sciences 25, No. 6 (2008): 316–24. http://dx.doi.org/10.17221/747-cjfs.

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Water-insoluble β-(1,3)-D-glucan (lichenan) was determined in 43 samples of various cereal (oats, barley, wheat, millet) and pseudocereal (buckwheat, amaranth) cultivars using a modified procedure with fungal α-amylase (Fermizyme P 300). The content of water-insoluble β-glucan varied in the dependence on the cereal species and cultivars. The highest content was registered in covered oat cultivars (Cyril and the new breeding cultivar PS-100) ranging from 26.7 to 28.2 g/100 g dry matter (d.m.) followed by less traditional cereals such as millet (<I>Panicum milia
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14

Mok, Jorge Wu, Natnael Behabtu, Christian Lenges, Ibrahim Sendijarevic, and Aisa Sendijarevic. "Evaluation of the engineered polysaccharide alpha‐1,3 glucan in a thermoplastic polyurethane model system." Journal of Applied Polymer Science 138, no. 9 (2020): 49931. http://dx.doi.org/10.1002/app.49931.

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15

Adibi, Azin, James Kim, Jorge Mok, Christian Lenges, Leonardo Simon, and Tizazu H. Mekonnen. "Enzymatic polymerization designed alpha-1,3 glucan particle morphology as reinforcing fillers of dipped and casted rubber films." Carbohydrate Polymers 267 (September 2021): 118234. http://dx.doi.org/10.1016/j.carbpol.2021.118234.

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16

Hogan, L. H., and B. S. Klein. "Altered expression of surface alpha-1,3-glucan in genetically related strains of Blastomyces dermatitidis that differ in virulence." Infection and Immunity 62, no. 8 (1994): 3543–46. http://dx.doi.org/10.1128/iai.62.8.3543-3546.1994.

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17

Lekkala, Madhavi, Ann Marie LeVine, Michael J. Linke, et al. "Effect of Lung Surfactant Collectins on Bronchoalveolar Macrophage Interaction with Blastomyces dermatitidis: Inhibition of Tumor Necrosis Factor Alpha Production by Surfactant Protein D." Infection and Immunity 74, no. 8 (2006): 4549–56. http://dx.doi.org/10.1128/iai.00243-06.

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ABSTRACT Alveolar surfactant modulates the antimicrobial function of bronchoalveolar macrophages (BAM). Little is known about the effect of surfactant-associated proteins in bronchoalveolar lavage fluid (BALF) on the interaction of BAM and Blastomyces dermatitidis. We investigated BALF enhancement or inhibition of TNF-α production by BAM stimulated by B. dermatitidis. BAM from CD-1 mice were stimulated with B. dermatitidis without or with normal BALF, surfactant protein A-deficient (SP-A−/−) or surfactant protein D-deficient (SP-D−/−) BALF, or a mixture of SP-A−/− and SP-D−/− BALF. An enzyme-l
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18

Shen, Hui, Si Min Chen, Wei Liu, et al. "Abolishing Cell Wall Glycosylphosphatidylinositol-Anchored Proteins in Candida albicans Enhances Recognition by Host Dectin-1." Infection and Immunity 83, no. 7 (2015): 2694–704. http://dx.doi.org/10.1128/iai.00097-15.

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Fungi can shield surface pathogen-associated molecular patterns (PAMPs) for evading host immune attack. The most common and opportunistic human pathogen,Candida albicans, can shield β-(1 3)-glucan on the cell wall, one of the major PAMPs, to avoid host phagocyte Dectin-1 recognition. The way to interfere in the shielding process for more effective antifungal defense is not well established. In this study, we found that deletion of theC. albicansGPI7gene, which was responsible for adding ethanolaminephosphate to the second mannose in glycosylphosphatidylinositol (GPI) biosynthesis, could block
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19

MIYANISHI, NOBUMITSU, YOSHIKO IWAMOTO, ETSUO WATANABE, and TATSUYA ODA. "Induction of TNF-.ALPHA. Production from Human Peripheral Blood Monocytes with .BETA.-1,3-Glucan Oligomer Prepared from Laminarin with .BETA.-1,3-Glucanase from Bacillus clausii NM-1." Journal of Bioscience and Bioengineering 95, no. 2 (2003): 192–95. http://dx.doi.org/10.1263/jbb.95.192.

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20

Wang, Yinan, Mengke Wang, Min Li, Te Zhao, and Lin Zhou. "Cinnamaldehyde inhibits the growth of Phytophthora capsici through disturbing metabolic homoeostasis." PeerJ 9 (April 30, 2021): e11339. http://dx.doi.org/10.7717/peerj.11339.

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Background Phytophthora capsici Leonian (P. capsici) can cause wilting and roots rotting on pepper and other cash crops. The new fungicide cinnamaldehyde (CA) has high activity against this pathogen. However, its potential mechanism is still unknown. Methods In order to gain insights into the mechanism, isobaric tags for relative and absolute quantification (iTRAQ)-based quantitative proteomics was used to analyze P. capsici treated with CA. The iTRAQ results were evaluated by parallel reaction monitoring (PRM) analysis and quantitative real-time PCR (qRT-PCR) analysis. Kyoto Encyclopedia of G
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21

Bom, Vinícius Leite Pedro, Patrícia Alves de Castro, Lizziane K. Winkelströter, et al. "The Aspergillus fumigatus sitA Phosphatase Homologue Is Important for Adhesion, Cell Wall Integrity, Biofilm Formation, and Virulence." Eukaryotic Cell 14, no. 8 (2015): 728–44. http://dx.doi.org/10.1128/ec.00008-15.

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ABSTRACTAspergillus fumigatusis an opportunistic pathogenic fungus able to infect immunocompromised patients, eventually causing disseminated infections that are difficult to control and lead to high mortality rates. It is important to understand how the signaling pathways that regulate these factors involved in virulence are orchestrated. Protein phosphatases are central to numerous signal transduction pathways. Here, we characterize theA. fumigatusprotein phosphatase 2A SitA, theSaccharomyces cerevisiaeSit4p homologue. ThesitAgene is not an essential gene, and we were able to construct anA.
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22

Eissenberg, L. G., and W. E. Goldman. "Histoplasma variation and adaptive strategies for parasitism: new perspectives on histoplasmosis." Clinical Microbiology Reviews 4, no. 4 (1991): 411–21. http://dx.doi.org/10.1128/cmr.4.4.411.

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This review summarizes the biology of Histoplasma capsulatum in relation to a wide variety of corresponding pathologies in histoplasmosis. Features of these disease syndromes can be explained in part by natural variations within the fungal population and adaptations made by individual organisms to specific environments. H. capsulatum grows as mycelia and conidia in the soil; once inhaled, the organism undergoes a dramatic morphological and physiological conversion to a yeast form. The yeasts proliferate within the phagolysosomes of macrophages, using a variety of specific strategies for intrac
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23

Zhu, Xiaohan, Arbia Arfaoui, Mohammad Sayari, Lorne R. Adam, and Fouad Daayf. "Gene Expression of Putative Pathogenicity-Related Genes in Verticillium dahliae in Response to Elicitation with Potato Extracts and during Infection Using Quantitative Real-Time PCR." Pathogens 10, no. 5 (2021): 510. http://dx.doi.org/10.3390/pathogens10050510.

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Quantitative real-time PCR was used to monitor the expression of 15 Verticillium dahliae’s genes, putatively involved in pathogenicity, highly (HAV) and weakly aggressive (WAV) V. dahliae isolates after either (i) elicitation with potato leaf, stem, or root extracts, or (ii) inoculation of potato detached petioles. These genes, i.e., coding for Ras-GAP-like protein, serine/threonine protein kinase, Ubiquitin-conjugating enzyme variant-MMS2, NADH-ubiquinone oxidoreductase, Thioredoxin, Pyruvate dehydrogenase E1 VdPDHB, myo-inositol 2-dehydrogenase, and HAD-superfamily hydrolase, showed differen
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24

Sretenovic, Ljiljana, Milan Petrovic, S. Aleksic, et al. "Influence of yeast, probiotics and enzymes in rations on dairy cows performances during transition." Biotehnologija u stocarstvu 24, no. 5-6 (2008): 33–43. http://dx.doi.org/10.2298/bah0806033s.

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The main goal of this paper was to investigate the influence of some additives included into high yielding dairy cows rations to overcome the problems in early lactation. These substances directly affect the productive performances of dairy cows as well as udder health. The commercial name of investigated preparation is 'YEASTURE' and it is composed of live yeast cultures selected from three strains Saccharomyces cerevisisiae in combination with probiotic bacteria and enzymes (Lactobacillus casei, Streptococcus faecium, Aspergillus oryzae, Lactobacillus acidophilus, 1,3-b and 1,6 D-Glucan, hem
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25

Srivastava, Shweta, Neha Shree Maurya, Shikha Kushwah, and Ashutosh Mani. "Current Promising Therapeutic Targets for Aspergillosis Treatment." Journal of Pure and Applied Microbiology 15, no. 2 (2021): 484–99. http://dx.doi.org/10.22207/jpam.15.2.09.

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Aspergillosis is a fungal disease caused by different species of Aspergillus. They live in soil,dust and decomposed material. Number of Aspergillus species found till now is about 300 and more are still to be identified. Only few Aspergillus species can cause human disease and the most common species for human infection is Aspergillus fumigatus, which is a ubiquitous airborne saprophytic fungus. Severity of the disease ranges from an allergic response to life-threatening generalized infection. They grow optimally at 37°C and can grow upto 50°C. The fungal conidia are being constantly inhaled b
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26

Bowden, Timothy J., Igor Kraev, and Sigrun Lange. "Extracellular Vesicles and Post-Translational Protein Deimination Signatures in Mollusca—The Blue Mussel (Mytilus edulis), Soft Shell Clam (Mya arenaria), Eastern Oyster (Crassostrea virginica) and Atlantic Jacknife Clam (Ensis leei)." Biology 9, no. 12 (2020): 416. http://dx.doi.org/10.3390/biology9120416.

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Oysters and clams are important for food security and of commercial value worldwide. They are affected by anthropogenic changes and opportunistic pathogens and can be indicators of changes in ocean environments. Therefore, studies into biomarker discovery are of considerable value. This study aimed at assessing extracellular vesicle (EV) signatures and post-translational protein deimination profiles of hemolymph from four commercially valuable Mollusca species, the blue mussel (Mytilus edulis), soft shell clam (Mya arenaria), Eastern oyster (Crassostrea virginica), and Atlantic jacknife clam (
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27

Stączek, Sylwia, Agnieszka Zdybicka-Barabas, Adrian Wiater, Małgorzata Pleszczyńska та Małgorzata Cytryńska. "Activation of cellular immune response in insect model host Galleria mellonella by fungal α-1,3-glucan". Pathogens and Disease 78, № 9 (2020). http://dx.doi.org/10.1093/femspd/ftaa062.

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Abstract Alpha-1,3-glucan, in addition to β-1,3-glucan, is an important polysaccharide component of fungal cell walls. It is reported for many fungal species, including human pathogenic genera: Aspergillus, Blastomyces, Coccidioides, Cryptococcus, Histoplasma and Pneumocystis, plant pathogens, e.g. Magnaporthe oryzae and entomopathogens, e.g. Metarhizium acridum. In human and plant pathogenic fungi, α-1,3-glucan is considered as a shield for the β-1,3-glucan layer preventing recognition of the pathogen by the host. However, its role in induction of immune response is not clear. In the present
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28

Chen, Tian, Andrew S. Wagner, Robert N. Tams та ін. "Lrg1 Regulates β (1,3)-Glucan Masking in Candida albicans through the Cek1 MAP Kinase Pathway". mBio 10, № 5 (2019). http://dx.doi.org/10.1128/mbio.01767-19.

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ABSTRACT Candida albicans is among the most prevalent opportunistic human fungal pathogens. The ability to mask the immunogenic polysaccharide β (1,3)-glucan from immune detection via a layer of mannosylated proteins is a key virulence factor of C. albicans. We previously reported that hyperactivation of the Cek1 mitogen-activated protein (MAP) kinase pathway promotes β (1,3)-glucan exposure. In this communication, we report a novel upstream regulator of Cek1 activation and characterize the impact of Cek1 activity on fungal virulence. Lrg1 encodes a GTPase-activating protein (GAP) that has bee
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29

Garfoot, Andrew L., Qian Shen, Marcel Wüthrich, Bruce S. Klein та Chad A. Rappleye. "The Eng1 β-Glucanase EnhancesHistoplasmaVirulence by Reducing β-Glucan Exposure". mBio 7, № 2 (2016). http://dx.doi.org/10.1128/mbio.01388-15.

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ABSTRACTThe fungal pathogenHistoplasma capsulatumparasitizes host phagocytes. To avoid antimicrobial immune responses,Histoplasmayeasts must minimize their detection by host receptors while simultaneously interacting with the phagocyte. PathogenicHistoplasmayeast cells, but not avirulent mycelial cells, secrete the Eng1 protein, which is a member of the glycosylhydrolase 81 (GH81) family. We show thatHistoplasmaEng1 is a glucanase that hydrolyzes β-(1,3)-glycosyl linkages but is not required forHistoplasmagrowthin vitroor for cell separation. However,Histoplasmayeasts lacking Eng1 function hav
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30

Guirao-Abad, José Pedro, Ruth Sánchez-Fresneda, Francisco Machado, Juan Carlos Argüelles та María Martínez-Esparza. "Micafungin Enhances the Human Macrophage Response to Candida albicans through β-Glucan Exposure". Antimicrobial Agents and Chemotherapy 62, № 5 (2018). http://dx.doi.org/10.1128/aac.02161-17.

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ABSTRACT Micafungin belongs to the antifungal family of echinocandins, which act as noncompetitive inhibitors of the fungal cell wall β-1,3- d -glucan synthase. Since Candida albicans is the most prevalent pathogenic fungus in humans, we study the involvement of micafungin in the modulation of the inflammatory response developed by human tissue macrophages against C. albicans . The MIC for micafungin was 0.016 μg/ml on the C. albicans SC5314 standard strain. Micafungin induced a drastic reduction in the number of exponential SC5314 viable cells, with the fungicidal effect being dependent on th
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