Relevant bibliographies by topics / Alpha-ketoglutarate

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Alpha-ketoglutarate'

Author: Grafiati
Published: 4 June 2021
Last updated: 9 February 2022

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Journal articles on the topic "Alpha-ketoglutarate"

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Le Boucher, Jacques, Biol Eng, Colette Coudray-Lucas, et al. "Enteral administration of ornithine alpha-ketoglutarate or arginine alpha-ketoglutarate." Critical Care Medicine 25, no. 2 (1997): 293–98. http://dx.doi.org/10.1097/00003246-199702000-00017.

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Hou, Yongqing. "Alpha-Ketoglutarate and intestinal function." Frontiers in Bioscience 16, no. 1 (2011): 1186. http://dx.doi.org/10.2741/3783.

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REX SHEU, KWAN-FU, and JOHN P. BLASS. "The alpha-Ketoglutarate Dehydrogenase Complex." Annals of the New York Academy of Sciences 893, no. 1 OXIDATIVE/ENE (1999): 61–78. http://dx.doi.org/10.1111/j.1749-6632.1999.tb07818.x.

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Gougoux, A., P. Vinay, and M. Duplain. "Maleate-induced stimulation of glutamine metabolism in the intact dog kidney." American Journal of Physiology-Renal Physiology 248, no. 4 (1985): F585—F593. http://dx.doi.org/10.1152/ajprenal.1985.248.4.f585.

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Studies were performed in anesthetized normal dogs to evaluate the effects of maleate on renal metabolism. Intravenous administration of maleate (50 mg/kg) markedly increased urinary excretion of glutamine, glutamate, alpha-ketoglutarate, alanine, lactate, pyruvate, and citrate. Despite a fourfold rise in renal cortical concentration of alpha-ketoglutarate, glutamine utilization expressed per 100 ml glomerular filtration rate almost doubled following maleate administration, whereas total ammonia production increased threefold, most of this ammonia being diverted into the renal vein. The renal
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Wu, Nan, Mingyao Yang, Uma Gaur, Huailiang Xu, Yongfang Yao, and Diyan Li. "Alpha-Ketoglutarate: Physiological Functions and Applications." Biomolecules & Therapeutics 24, no. 1 (2016): 1–8. http://dx.doi.org/10.4062/biomolther.2015.078.

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Tian, Qiyu, Xiangdong Liu, and Min Du. "Alpha-ketoglutarate for adipose tissue rejuvenation." Aging 12, no. 14 (2020): 13845–46. http://dx.doi.org/10.18632/aging.103853.

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Uzgare, Arti, Kavalukas Sandra, Qiang Zhang, Luc Cynober, and Adrian Barbul. "Ornithine alpha ketoglutarate enhances wound healing." Journal of the American College of Surgeons 209, no. 3 (2009): S72. http://dx.doi.org/10.1016/j.jamcollsurg.2009.06.174.

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Simpson, D. P., N. McSherry, E. Scarbrough, and K. Zweifel. "Substrate responses to acid-base alterations in dog renal proximal tubules." American Journal of Physiology-Renal Physiology 262, no. 6 (1992): F1039—F1046. http://dx.doi.org/10.1152/ajprenal.1992.262.6.f1039.

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Proximal tubules from dog kidney were incubated for 2-6 min with low concentrations of pyruvate, glutamine, and malate. When initial medium citrate was between 0 and 0.5 mM and alpha-ketoglutarate was between 0 and 0.1 mM, concentrations of these two substrates in tubules and media after incubation were lower with 10 than with 40 mM HCO3-. Malate levels in tubules and media changed in the opposite direction. CO2 formation from labeled citrate or alpha-ketoglutarate was greater at low than at high HCO3-. In tubules treated with digitonin to disrupt the cell membrane, differences in citrate and
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Cynober, L. "Metabolic interaction between ornithine and alpha-ketoglutarate as a basis for the action of ornithine alpha-ketoglutarate." Clinical Nutrition 12, no. 1 (1993): 54–56. http://dx.doi.org/10.1016/0261-5614(93)90150-3.

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Donati, L., M. Signorini, and S. Grappolini. "Ornithine alpha-ketoglutarate administration in burn injury." Clinical Nutrition 12, no. 1 (1993): 70–71. http://dx.doi.org/10.1016/0261-5614(93)90159-2.

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Dissertations / Theses on the topic "Alpha-ketoglutarate"

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Omede, Ameh. "Role of alpha-ketoglutarate receptor G-protein coupled receptor 99 (GPR99) in cardiac hypertrophy." Thesis, University of Manchester, 2015. https://www.research.manchester.ac.uk/portal/en/theses/role-of-alphaketoglutarate-receptor-gprotein-coupled-receptor-99-gpr99-in-cardiac-hypertrophy(83b04dba-5bfe-4623-ade1-12c779133b80).html.

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Cardiac hypertrophy and heart failure (HF) remains one of the major health problems in the UK and worldwide. However, advances in their management are limited because the underlying pathological mechanisms are not completely understood. Therefore, it is important to understand novel signalling pathways leading to HF. Myocardial hypertrophy is a crucial pathophysiological process that can lead to the development of HF. Signalling initiated by members of G-protein-coupled receptors (GPCRs) proteins plays an important role in mediating cardiac hypertrophy. One member of this family, the G-protein
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Romano, Manuela. "Stage-specific changes in the Krebs cycle network regulate human erythroid differentiation." Thesis, Montpellier, 2018. http://www.theses.fr/2018MONTT077.

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Le processus conduisant à la prolifération et différenciation des cellules souches hématopoïétiques (CSH) en cellules de toutes les lignées sanguines s’appelle l’hématopoïèse. Bien que l'engagement des CSH soit régi par les cytokines, les facteurs de transcription, les modificateurs épigénétiques et la niche des CSH, notre groupe a constaté que leur engagement vers la lignée érythroïde dépendait aussi du métabolisme de la glutamine. La glutaminolyse contribue à la biosynthèse des nucléotides de novo ainsi qu’à la production de l'alpha-kétoglutarate (αKG), intermédiaire métabolique du cycle TCA
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Yang, Bingquan [Verfasser]. "Effects of alpha-ketoglutarate on cell growth, glucose, glutamine, lactate and ammonia metabolism in C2C12 cells / Bingquan Yang." Ulm : Universität Ulm. Medizinische Fakultät, 2012. http://d-nb.info/1023729164/34.

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Neidig, Michael Lee. "Structure/function correlations in oxygen and substrate activating mononuclear non-heme iron enzymes : alpha-ketoglutarate-dependent dioxygenases and lipoxygenases /." May be available electronically:, 2007. http://proquest.umi.com/login?COPT=REJTPTU1MTUmSU5UPTAmVkVSPTI=&clientId=12498.

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Ferreira, Matias Maria. "Targeting the metabolic environment to modulate T cell effector function." Thesis, Montpellier, 2019. http://www.theses.fr/2019MONTT020.

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L’activation des cellules T est initiée suite à la rencontre avec un antigène spécifique. Les études réalisées pour mieux comprendre ce processus d’activation se sont principalement focalisées sur le rôle des cellules présentatrices d'antigènes et des cytokines. Toutefois, des données récentes soulignent également l'importance du microenvironnement métabolique pour soutenir l’augmentation des besoins énergétiques et biosynthétiques liés à la stimulation antigénique. Cette reprogrammation métabolique est conditionnée par la disponibilité en nutriments et la teneur en oxygène qui peuvent être al
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GonÃalves, Eduardo Silvio Gouveia. "Ornitina alfa-cetoglutarato na isquemia-reperfusÃo intestinal em ratos." Universidade Federal do CearÃ, 2009. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=4687.

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Conselho Nacional de Desenvolvimento CientÃfico e TecnolÃgico<br>Objetivo: Avaliar os efeito da ornitina &#945;-cetoglutarato (OKG) no sangue e tecido intestinal de ratos submetidos à isquemia/reperfusÃo intestinal atravÃs da determinaÃÃo das concentraÃÃes in vivo no sangue e no tecido do intestino delgado, submetido a isquemia/reperfusÃo, de glicose, G 6 PDH, piruvato, acetoacetato, lactato, 3 HBDH, glutationa, T-Bars, mieloperoxidase, CPK e LDH. MÃtodo: Sessenta ratos (Rattus norvergicus albinus, Rodentia Mammalia) foram distribuÃdos aleatoriamente em cinco grupos de 12 animais: Sham 0â (s0â
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Manangi, Megharaja K. "Chicken lysine [alpha]-ketoglutarate reductase (LKR) in different tissues and effects of graded levels of dietary lysine on LKR and lysine oxidation." Morgantown, W. Va. : [West Virginia University Libraries], 2000. http://etd.wvu.edu/templates/showETD.cfm?recnum=1734.

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Thesis (M.S.)--West Virginia University, 2000.<br>Title from document title page. Document formatted into pages; contains viii, 92 p. : ill. Vita. Includes abstract. Includes bibliographical references.
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Tatke, Gorakh Digambar. "Elucidating The Role of MifS-MifR Two-Component System in Regulating Pseudomonas aeruginosa Pathogenicity." FIU Digital Commons, 2016. http://digitalcommons.fiu.edu/etd/3002.

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Pseudomonas aeruginosa is a Gram-negative, metabolically versatile, opportunistic pathogen that exhibits a multitude of virulence factors, and is extraordinarily resistant to a gamut of clinically significant antibiotics. This ability is in part mediated by two-component systems (TCS) that play a crucial role in regulating virulence mechanisms, metabolism and antibiotic resistance. Our sequence analysis of the P. aeruginosa PAO1 genome revealed the presence of two open reading frames, mifS and mifR, which encodes putative TCS proteins, a histidine sensor kinase MifS and a response regulator Mi
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Price, John C. "Mechanistic dissection of taurine [alpha]-ketoglutarate dioxygenase (TauD) a model [alpha]-ketoglutarate dioxygenase /." 2005. http://etda.libraries.psu.edu/theses/approved/WorldWideIndex/ETD-1055/index.html.

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Lu, Wei, and 呂. 維. "Decoupling itaconate production from alpha-ketoglutarate biosynthesis in E.coli." Thesis, 2018. http://ndltd.ncl.edu.tw/handle/3u694t.

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Book chapters on the topic "Alpha-ketoglutarate"

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Bhattacharya, R. "Recent perspectives on alpha-ketoglutarate." In Toxicology of Cyanides and Cyanogens. John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781118628966.ch27.

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Visser, Sam P. "Taurine/Alpha-Ketoglutarate Dioxygenase – Computational Studies." In Encyclopedia of Biophysics. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-16712-6_596.

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de Visser, Sam P. "Taurine/Alpha-Ketoglutarate Dioxygenase: Computational Studies." In Encyclopedia of Biophysics. Springer Berlin Heidelberg, 2021. http://dx.doi.org/10.1007/978-3-642-35943-9_596-1.

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Li, Changhong. "Insulin Secretion and the Glutamine-Glutamate-Alpha-Ketoglutarate Axis." In Glutamine in Clinical Nutrition. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1932-1_19.

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Li, Changhong, Franz M. Matschinsky, and Charles A. Stanley. "Amino Acid-Stimulated Insulin Secretion: The Role of the Glutamine-Glutamate-Alpha-Ketoglutarate Axis." In Monogenic Hyperinsulinemic Hypoglycemia Disorders. KARGER, 2012. http://dx.doi.org/10.1159/000334516.

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Robertson, Tausha. "Ornithine, Ornithine Alpha-Ketoglutarate and Taurine." In Nutritional Ergogenic Aids. CRC Press, 2004. http://dx.doi.org/10.1201/9780203507704.ch12.

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Nulton-Persson, Amy, Luke Szweda, and Kenneth Humphries. "Inactivation and Inhibition of Alpha-Ketoglutarate Dehydrogenase." In Oxidative Stress and Disease. CRC Press, 2008. http://dx.doi.org/10.1201/9781420045390.ch8.

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"Ornithine Alpha-Ketoglutarate Administration in Surgical, Trauma and Cancer-Bearing Patients." In Nutritional Support in Cancer and Transplant Patients. CRC Press, 2001. http://dx.doi.org/10.1201/9781498713320-15.

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Conference papers on the topic "Alpha-ketoglutarate"

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Mondul, Alison, Steven Moore, Joshua Sampson, Stephanie Weinstein, and Demetrius Albanes. "Abstract 936: Serum lipid metabolites and alpha-ketoglutarate are inversely associated with aggressive prostate cancer." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-936.

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"Conformational dynamics in methylated DNA repair by human Fe(II)/alpha-ketoglutarate dependent dioxygenases ALKBH2 and ALKBH3." In Bioinformatics of Genome Regulation and Structure/ Systems Biology. institute of cytology and genetics siberian branch of the russian academy of science, Novosibirsk State University, 2020. http://dx.doi.org/10.18699/bgrs/sb-2020-352.

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"Assessing Nutraceutical Alpha Ketoglutarate for Its Effectiveness: 99m Technetium-Mebrofenin Nuclear Imaging In CCl4 Induced Hepatotoxic Animal Model." In International Conference on Chemical, Food and Environment Engineering. International Academy Of Arts, Science & Technology, 2015. http://dx.doi.org/10.17758/iaast.a0115065.

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