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1

Guttenberg, Gregor [Verfasser], and Manfred [Akademischer Betreuer] Jung. "Clostridiale Glukosylierende Toxine: Untersuchungen zur Autoprozessierung von Clostridium sordellii Letalem Toxin und Clostridium novyi alpha-Toxin sowie funktionelle Charakterisierung von Clostridium perfringens TpeL-Toxin." Freiburg : Universität, 2012. http://d-nb.info/1123467994/34.

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Eaton, Julian Timothy. "Structural studies of Clostridium perfringens alpha toxin." Thesis, Birkbeck (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.417896.

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Justin, Neil. "Structural studies of clostridium perfringens alpha toxin." Thesis, Birkbeck (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.392355.

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4

Reutemann, Mathias. "ICAM-1 abhängige Akkumulation neutrophiler Granulozyten und Leukotrien-vermittelte Kardiodepression in Staphylococcus aureus [alpha]-Toxin-perfundierten [Alpha-Toxin-perfundierten] Rattenherzen." [S.l.] : [s.n.], 2002. http://deposit.ddb.de/cgi-bin/dokserv?idn=965811611.

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5

Carnegie, Andrew Mark. "Effects of C.perfringens alpha toxin on cell signalling." Thesis, Birkbeck (University of London), 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.416052.

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6

Wegner, Judith. "Exotoxin-Schock, ausgelöst durch Staphylococcus-aureus-[alpha]-Toxin [Staphylococcus-aureus-alpha-Toxin], am Tiermodell Ratte Auswirkungen auf Gefässendothel, Leukozytenakkumulation und Thrombozytenaggregation /." [S.l.] : [s.n.], 2005. http://deposit.ddb.de/cgi-bin/dokserv?idn=97606538X.

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7

Sawicki, Maria. "Immunological studies on staphylococcal alpha toxin and its fragments." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0020/MQ52656.pdf.

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8

Penha, Marcelo De Luca. "Detecção dos genes das toxinas alfa, beta e épsilon de Clostridium perfringens isolados a partir de amostras clínicas de bovinos pela reação em cadeia da polimerase." Universidade de São Paulo, 2004. http://www.teses.usp.br/teses/disponiveis/10/10134/tde-06072005-101119/.

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O Clostridium perfringens é um microrganismo anaeróbio que está presente no solo e no trato intestinal dos mamíferos. Provoca intoxicação alimentar nos seres humanos, doenças enterotoxêmicas nos animais domésticos e gangrena gasosa em ambos os grupos. O C. perfringens é classificado em cinco tipos (A, B, C, D e E) mediante a produção de quatro toxinas principais (alfa, beta, épsilon e iota). Neste trabalho foi possível padronizar a técnica de PCR para detectar a presença dos genes cpa, cpb e etx a partir de culturas de C. perfringens. A sensibilidade analítica da técnica de PCR a partir de cul
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9

Leslie, Dario Lyall. "Genetic analysis of alpha toxin (phospholipase C) from Clostridium perfringens." Thesis, University of Newcastle Upon Tyne, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.346420.

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10

Sylvester, Ian David. "The characterisation and conjugation of the fungal toxin #alpha#-sarcin." Thesis, University of Warwick, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.308083.

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11

Bullifent, Helen Lisa. "The regulation of the alpha-toxin gene of Clostridium perfringens." Thesis, University of Sheffield, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.296729.

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12

Eger, Frank. "Durch Staphylococcus-aureus-[alpha]-Toxin [Staphylococcus-aureus-Alpha-Toxin] permeabilisierte, mit Simian Virus 40 infizierte CV1-Zellen als Modellsystem zum Studium der DNA-Replikation höherer Zellen." Tübingen, Stäudach 107 : F. Eger, 2001. http://deposit.ddb.de/cgi-bin/dokserv?idn=963193643.

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13

Thompson, James Russell. "Imaging the assembly of the Staphylococcal pore-forming toxin alpha-Hemolysin." Thesis, University of Oxford, 2009. http://ora.ox.ac.uk/objects/uuid:e320004a-6118-4dac-af2a-eca6e90be7ac.

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Alpha-hemolysin is a pore-forming toxin secreted by pathogenic Staphylococcus aureus. Its spontaneous oligomerization and assembly into a trans-bilayer beta-barrel pore is a model for the assembly of many other pore-forming toxins. It is studied here in vitro as a means to probe general membrane protein oligomerization and lipid bilayer insertion. This thesis details the results of experiments to develop and implement a novel in vitro lipid bilayer system, Droplet-on-Hydrogel Bilayers (DHBs) for the single-molecule imaging of alpha-hemolysin assembly. Chapter 2 describes the development of DHB
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14

Berger, Katharina [Verfasser]. "Integritätsstörung endothelialer Junktionsproteine durch Staphylococcus aureus Alpha-Toxin-Stabilisierung durch Adrenomedullin / Katharina Berger." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2010. http://d-nb.info/1024784266/34.

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15

Miyashiro, Simone. "Caracterização de isolados de Clostridium perfringens de ruminantes." Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/10/10134/tde-16092014-155341/.

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C. perfringens é uma bactéria anaeróbia presente no intestino delgado do homem e animais em equilíbrio e, sob a ação de alguns fatores predisponentes como mudança brusca de alimentação ou super alimentação, stress no manejo ou alto parasitismo intestinal, há a proliferação do microrganismo com a consequente produção de potentes toxinas que provocam a morte do animal. Dentre as toxinas principais destaca-se a toxina alfa, importante fator de virulência, produzida por todos os tipos de C. perfringens, sendo os pertencentes ao tipo A os maiores produtores. A fim de caracterizar o microrganismo em
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16

Russo, Michael J. (Michael Joseph). "Controlled poration of the cell membrane using alpha-toxin with a metal-actuated switch." Thesis, Massachusetts Institute of Technology, 1995. http://hdl.handle.net/1721.1/11491.

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Thesis (M.S.)--Massachusetts Institute of Technology, Dept. of Mechanical Engineering, 1995.<br>On t.p., "[alpha]" appears as the lower-case Greek letter.<br>Includes bibliographical references (leaves 73-80).<br>by Michael J. Russo.<br>M.S.
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17

Simon, Melanie Eva Maria [Verfasser]. "α-Toxin [Alpha-Toxin] von Staphylococcus aureus induziert ein Nierenversagen durch Aktivierung der intrarenalen Thromboxansynthese im Modell der isolierten Rattenniere / eingereicht von Melanie Eva Maria Simon". Giessen : VVB Laufersweiler, 2010. http://d-nb.info/1008284920/34.

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18

Hoven, Gisela von [Verfasser]. "Induktion von Pro-Autophagie-Signalen durch einen extra- oder intrazellulären Alpha-Toxin-Angriff / Gisela von Hoven." Mainz : Universitätsbibliothek Mainz, 2013. http://d-nb.info/1032940409/34.

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19

Cargnelutti, Marilisa [Verfasser]. "Sequencing of alpha- and beta2- toxin-genes from C. perfringens strains isolated from rabbits / Marilisa Cargnelutti." Berlin : Freie Universität Berlin, 2009. http://d-nb.info/102362172X/34.

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20

Zhang, Guangtao. "Design, synthesis, and evaluation of cholera toxin inhibitors and [alpha]-helix mimetics of dormancy survival regulator /." Thesis, Connect to this title online; UW restricted, 2006. http://hdl.handle.net/1773/8485.

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21

Ghosh, Gargi. "A WHOLE CELL BASED BIOSENSOR FOR MONITORING PHYSIOLOGICAL TOXINS AND EARLY SCREENING OF CANCER." UKnowledge, 2008. http://uknowledge.uky.edu/gradschool_diss/578.

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Recently a whole cell based biosensor has been developed in our laboratory that consists of a monolayer of human umbilical vein endothelial cells (HUVECs) on the asymmetric cellulose triacetate (CTA) membrane of an ion selective electrode (ISE). When a confluent cell monolayer is formed across the membrane, response from the sensor is inhibited due to inhibited ion transport across the membrane. When the cell based biosensor is exposed to permeability modifying agents, the permeability across the cell monolayer is altered facilitating more ion transport and as a result the response from the se
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22

Rogers, Tara Marie. "Investigation of Alpha-Toxin Secretions in Biofilm Conditioned Medium as a Potential Pro-Inflammatory Disruptor to Macrophages." Kent State University Honors College / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ksuhonors1557145132511741.

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23

O'Brien, David Kenneth. "The Interactions of Clostridium Perfringens With Phagocytic Cells." Diss., Virginia Tech, 2003. http://hdl.handle.net/10919/27164.

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Clostridium perfringens is the most common cause of gas gangrene (clostridial myonecrosis), a disease that begins when ischemic tissues become contaminated with C. perfringens. C. perfringens quickly multiplies in ischemic tissues and spreads to healthy areas, leading to high levels of morbidity and mortality. As a species, the bacterium can synthesize thirteen different toxins. The alpha toxin (PLC) and perfringolysin O (PFO) are thought to be important virulence factors in gangrene. We wished to understand how C. perfringens is capable of avoiding killing by the host immune system, and d
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24

Eger, Frank [Verfasser]. "Durch Staphylococcus-aureus-α-Toxin [Staphylococcus-aureus-Alpha-Toxin] permeabilisierte, mit Simian Virus 40 infizierte CV1-Zellen als Modellsystem zum Studium der DNA-Replikation höherer Zellen / vorgelegt von Frank Eger". Tübingen, Stäudach 107 : F. Eger, 2001. http://d-nb.info/963193643/34.

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25

Gatsos, Xenia, and xgatsos@optusnet com au. "The development of live vectored vaccines targeting the alpha-toxin of Clostridium perfringens for the prevention of necrotic enteritis in poultry." RMIT University. Applied Sciences, 2007. http://adt.lib.rmit.edu.au/adt/public/adt-VIT20080212.142403.

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The Ą-toxin of Clostridium perfringens is a toxin involved in numerous diseases of humans and agriculturally important animals. One of these diseases is necrotic enteritis (NE), a sporadic enteric disease which affects avian species world-wide. This study involved the inactivation of alpha-toxin (Ą-toxin) for use as a potential vaccine candidate to combat NE in chickens, and other diseases caused by C. perfringens type A. During the course of this research a number of Ą-toxin recombinant proteins were developed through molecular inactivation of the Ą-toxin gene, plc. Proteins plc3
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26

Johansson, David. "Bacterial toxins for cancer treatment." Doctoral thesis, Umeå universitet, Medicinsk biovetenskap, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1637.

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Even though anti‐cancer chemotherapy has been continuously improved during the last decades. problems with adverse effects and drug resistance still constitutes a considerable obstacle and sets a demand for new effective treatment options. Tissue homeostasis in multi‐cellular organisms is maintained through intrinsic cell death, apoptosis, which removes unwanted or damaged cells. Disrupted apoptosis is an important factor in tumorgenesis and drug resistance, therefore induction or restoration of apoptotic pathways is also important for the treatment of cancer. Several naturally occurring bacte
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27

Kostova, Vesela. "Shiga toxin targeted strategy for chemotherapy and cancer immunotherapy application using copper-free « Click » chemistry." Thesis, Sorbonne Paris Cité, 2015. http://www.theses.fr/2015USPCB144.

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Pas de résumé<br>Recently targeted therapies appeared as attractive alternatives to classical antitumoral treatments. The approach, developed on the concept of targeting drug to cancer cells, aims to spear normal tissues and decrease the side effects. This doctoral dissertation focuses on developing new anticancer targeted treatments in the field of chemotherapy and cancer immunotherapy by exploiting an original targeting moiety, the B subunit of Shiga toxin (STxB). Its specific properties, such as, recognition with its receptor Gb3 overexpressed in cancer cells or in antigen-presenting cells,
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28

Möller, Nils [Verfasser], Jan-Peter [Akademischer Betreuer] Hildebrandt, Jan-Peter [Gutachter] Hildebrandt, and Wolf-Michael [Gutachter] Weber. "Determinanten der Sensitivität von humanen Atemwegsepithelzellen gegenüber dem alpha-Toxin von Staphylococcus aureus und die Prozessierung des Toxins nach der Porenbildung / Nils Möller ; Gutachter: Jan-Peter Hildebrandt, Wolf-Michael Weber ; Betreuer: Jan-Peter Hildebrandt." Greifswald : Universität Greifswald, 2021. http://d-nb.info/1238233279/34.

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29

Clelland, Lyndsay Jacquelyn. "Role of ROK and PKC in Permeabilized Rabbit Femoral Artery." VCU Scholars Compass, 2007. http://hdl.handle.net/10156/1581.

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30

Félix, Mellanie Karoline do Carmo. "Antígeno inativado de Clostridium Novyi tipo B em emulsão W/O: uma prova de conceito em camundongos Swiss visando o controle de necrose hepática de ruminantes." Universidade Federal do Tocantins, 2018. http://hdl.handle.net/11612/965.

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A bovinocultura brasileira possui grande ênfase no mercado nacional. Doenças que acometem rebanhos comprometem o mercado além de gerarem grandes prejuízos econômicos. O Clostridium novyi tipo B provoca necrose hepática em bovinos através da produção da alfa toxina, uma potente exotoxina que reduz a produtividade através de alterações como hemoglobinúria, redução do apetite, febre, letargia, diminuição da produção de leite e sangue nas fezes. Conter o microrganismo causador torna-se uma busca necessária tanto do ponto de vista econômico quanto social. Entretanto, o controle da doença ainda é re
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31

Jansen, Katja. "Methodische Untersuchungen zu Eigenschaften, Nachweis, Reinigung und Antigenität des a-Toxins [Alpha-Toxins] von Clostridium septicum." [S.l.] : [s.n.], 2000. http://deposit.ddb.de/cgi-bin/dokserv?idn=961183098.

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32

Sullivan, Derek J. "Regulation of #alpha#-haemolysin gene expression in Staphylococcus aureus." Thesis, University of Newcastle Upon Tyne, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.287423.

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33

GUILLOUARD, ISABELLE. "Organisation structurale et fonctionnelle de la toxine alpha de clostridium perfringens." Paris 7, 1997. http://www.theses.fr/1997PA077115.

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La toxine-alpha de clostridium perfringens, principal facteur de virulence produit par les souches pathogenes, joue un role preponderant dans le developpement de la gangrene gazeuse. Phospholipase c, elle peut hydrolyser la phosphatidylcholine (lecithine) et la sphingomyeline, acides gras majeurs des membranes des cellules eucaryotes. Elle est aussi letale, necrosante et hemolytique. Bien caracterisee biochimiquement, les relations existant entre la structure et le mode d'action de la toxine alpha sont encore imprecises. Grace a la remarquable similitude existant entre les 2/3 n-terminaux de l
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Maïga, Arhamatoulaye. "Caractérisation de l'interaction entre la toxine peptidique AdTx1 et le récepteur α1A adrénergique". Paris 6, 2011. http://www.theses.fr/2011PA066037.

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AdTx1 est une toxine à trois doigts de 65 acides aminés découverte dans notre laboratoire à partir du venin du mamba vert (Dendroaspis angusticeps). Elle a une haute affinité et sélectivité pour le récepteur adrénergique α1A-AR par rapport aux autres récepteurs adrénergiques α1. Ces différentes caractéristiques font de cette toxine un outil original pour étudier le récepteur et également une molécule thérapeutique potentielle dans le traitement de l’hypertrophie bénigne de la prostate. Le but de ma thèse est de caractériser le profil pharmacologique de cette toxine et de comprendre l'origine m
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35

Thet, Naing Tun. "Modified tethered bilayer lipid membranes for detection of pathogenic bacterial toxins and characterization of ion channels." Thesis, University of Bath, 2010. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.530158.

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Pathogenic bacteria secrete various virulence factors as their biochemical weapons to gain access to and destroy the target cells. They can directly interact with the outer lipid bilayer membrane of eukaryotic cells, inducing the premature cell death by either apoptosis or necrosis. Such virulence factors account for much of the toxic actions associated with bacterial infection; therefore the detection of such proteins could provide a methodology for sensing/detection of pathogenic bacteria in, for example, food or human tissue. Detection and identification of pathogenic bacteria by convention
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36

TENETTE, CATHERINE. "Modelisation du site de combinaison d'un anticorps libre et lie a son antigene, la toxine alpha." Paris 11, 1996. http://www.theses.fr/1996PA112141.

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Ce travail porte sur la prediction de la structure tridimensionnelle du complexe entre un anticorps monoclonal et son antigene, la toxine alpha. Cet anticorps est capable de neutraliser les toxines curarisantes courtes de serpent et de reconnaitre une partie du site fonctionnel de ces toxines, c'est-a-dire que son epitope sur les toxines recouvre en partie le site toxique reconnu par le recepteur nicotinique a l'acetylcholine. Pour cela, le fragment variable de l'anticorps a ete modelise a partir de sa sequence en acides amines. La modelisation par homologie a ete utilisee pour construire la r
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37

Thiersé, Danièle. "Perméabilisation des cellules chromaffines par la toxine alpha : Rôle des protéines G dans le mécanismes d'exocytose." Université Louis Pasteur (Strasbourg) (1971-2008), 1991. http://www.theses.fr/1991STR1A005.

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38

Pan-Montojo, Francisco, Mathias Schwarz, Clemens Winkler, Mike Arnhold, Sullivan Gregory A. O', Arun Pal, Jonas Said, et al. "Environmental toxins trigger PD-like progression via increased alpha-synuclein release from enteric neurons in mice." Nature Publishing Group, 2012. https://tud.qucosa.de/id/qucosa%3A28923.

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Pathological studies on Parkinson's disease (PD) patients suggest that PD pathology progresses from the enteric nervous system (ENS) and the olfactory bulb into the central nervous system. We have previously shown that environmental toxins acting locally on the ENS mimic this PD-like pathology progression pattern in mice. Here, we show for the first time that the resection of the autonomic nerves stops this progression. Moreover, our results show that an environmental toxin (i.e. rotenone) promotes the release of alpha-synuclein by enteric neurons and that released enteric alpha-synuclein is u
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39

Pan-Montojo, Francisco, Mathias Schwarz, Clemens Winkler, Mike Arnhold, Sullivan Gregory A. O', Arun Pal, Jonas Said, et al. "Environmental toxins trigger PD-like progression via increased alpha-synuclein release from enteric neurons in mice." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2015. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-180702.

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Pathological studies on Parkinson's disease (PD) patients suggest that PD pathology progresses from the enteric nervous system (ENS) and the olfactory bulb into the central nervous system. We have previously shown that environmental toxins acting locally on the ENS mimic this PD-like pathology progression pattern in mice. Here, we show for the first time that the resection of the autonomic nerves stops this progression. Moreover, our results show that an environmental toxin (i.e. rotenone) promotes the release of alpha-synuclein by enteric neurons and that released enteric alpha-synuclein is u
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40

Miles, Jr George Emmett. "On the structure and assembly of staphylococcal leukocidin: a study of the molecular architecture of beta-barrel pore-forming toxins." Texas A&M University, 2003. http://hdl.handle.net/1969.1/3952.

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Staphylococcal leukocidin pores are formed by the obligatory interaction of two distinct polypeptides, one of class F and one of class S, making them unique in the family of &#946;-barrel pore-forming toxins (&#946;-PFTs). By contrast, other &#946;-PFTs form homooligomeric pores. For example, the staphylococcal &#945;- hemolysin is a homoheptamer. Limited and controversial data exist on the assembly and molecular architecture of the leukocidin pore. In this work, biochemical and biophysical methods were used to characterize the leukocidin pore produced by the LukF (HlgB) and LukS (HlgC) compon
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Benoit, Evelyne. "Électrophysiologie et pharmacologie du courant sodium de la fibre nerveuse myélinisée de grenouille : contributions à la mise en évidence de trois formes interconvertibles du canal sodium." Paris 11, 1986. http://www.theses.fr/1986PA112131.

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Cette thèse présente une analyse du courant sodium de la fibre nerveuse myélinisée de grenouille à l'aide de la technique du potentiel imposé. Le courant sodium de la membrane nodale est activé par des impulsions dépolarisantes. Ce courant montre une activation et une inactivation dépendantes du potentiel. La cinétique d'inactivation du courant sodium est bien décrite par la somme de deux phases exponentielles et une phase constante. Ces trois phases présentent des propriétés cinétiques (courbes courant-potentiel, courbes inactivation stationnaire-potentiel, potentiels d'inversion, levées de l
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Gross, Grégori. "Modification des voies de repliement d'une petite protéine riche en ponts disulfure : la toxine alpha de Naja nigricollis." Phd thesis, Museum national d'histoire naturelle - MNHN PARIS, 2008. http://tel.archives-ouvertes.fr/tel-00364212.

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Le repliement des protéines, dernière étape du processus d'expression de l'information génétique, demeure imparfaitement expliqué. Pendant ma thèse, j'ai étudié le repliement oxydant d'une petite protéine riche en ponts disulfure, la toxine a de Naja nigricollis. J'ai pu montrer que l'addition d'un acide aminé dans une boucle de la structure entraîne un ralentissement global du repliement in vitro causé par une permutation des intermédiaires productifs. L'étude en RMN des intermédiaires de repliement a permis de proposer une explication structurale à cette modification de comportement cinétiqu
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Gross, Grégori. "Modification des voies de repliement d’une petite protéine riche en ponts disulfure : la toxine alpha de Naja nigricollis." Paris, Muséum national d'histoire naturelle, 2008. http://www.theses.fr/2008MNHN0010.

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Le repliement des protéines, dernière étape du processus d’expression de l’information génétique, demeure imparfaitement expliqué. Pendant ma thèse, j’ai étudié le repliement oxydant d’une petite protéine riche en ponts disulfure, la toxine  de Naja nigricollis. J’ai pu montrer que l’addition d’un acide aminé dans une boucle de la structure entraîne un ralentissement global du repliement in vitro causé par une permutation des intermédiaires productifs. L’étude en RMN des intermédiaires de repliement a permis de proposer une explication structurale à cette modification de comportement cinétiqu
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Teixeira-Clerc, Fatima. "Etude de l'interaction entre le récepteur nicotinique de l'acetylcholine et la toxine alpha de Naja nigricollis par ingénierie chimique du ligand." Paris 6, 2003. http://www.theses.fr/2003PA066316.

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45

Gilles, Nicolas. "Effets pharmacologiques des toxines de type alpha de scorpion sur les canaux sodiques de l'insecte et du mammifère." Paris 5, 2000. http://www.theses.fr/2000PA05P601.

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46

Petitcolin, Marie-Anne. "Vieillissement artériel et sensibilité au calcium de la contraction : couplage entre récepteurs [alpha]1-adrénergiques et protéines G[indice i/o]." Nancy 1, 2000. http://www.theses.fr/2000NAN12012.

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Thèse de doctorat en sciences du médicament. Ce travail de thèse a eu pour objectif : 1] l'étude des mécanismes intracellulaires de régulation de la sensibilité au calcium intracellulaire ([Ca2+][indice i]) de la réponse contractile des cellules musculaires lisses, lors d'une stimulation par un agoniste, la noradrénaline, 2] la comparaison de ces mécanismes dans une artère musculaire de faible diamètre, l'artère caudale, et une artère de transfert, l'aorte, chez le rat, et 3] l'analyse des perturbations de ces mécanismes lors du vieillissement vasculaire. L'originalité de ce travail repose sur
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Huang, Mengying [Verfasser], and Martin [Akademischer Betreuer] Borggrefe. "Alpha 1-adrenoceptor signaling contributes to toxic effects of catecholamine on electrical properties in human-induced stem cell-derived cardiomyocytes / Mengying Huang ; Betreuer: Martin Borggrefe." Heidelberg : Universitätsbibliothek Heidelberg, 2021. http://d-nb.info/1228539898/34.

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[Verfasser], Gunnaporn Veerachato. "On the cellular stress response to Staphylococcus aureus alpha-toxin / Gunnaporn Veerachato." 2007. http://d-nb.info/982704682/34.

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Reutemann, Mathias [Verfasser]. "ICAM-1 abhängige Akkumulation neutrophiler Granulozyten und Leukotrien-vermittelte Kardiodepression in Staphylococcus aureus α-Toxin-perfundierten [Alpha-Toxin-perfundierten] Rattenherzen / vorgelegt von Mathias Reutemann". 2002. http://d-nb.info/965811611/34.

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Wegner, Judith [Verfasser]. "Exotoxin-Schock, ausgelöst durch Staphylococcus-aureus-α-Toxin [Staphylococcus-aureus-alpha-Toxin], am Tiermodell Ratte : Auswirkungen auf Gefäßendothel, Leukozytenakkumulation und Thrombozytenaggregation / eingereicht von Judith Wegner". 2005. http://d-nb.info/97606538X/34.

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