Relevant bibliographies by topics / Alveolar mechanics

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Alveolar mechanics'

Author: Grafiati
Published: 4 June 2021
Last updated: 19 February 2022

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Journal articles on the topic "Alveolar mechanics"

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Prange, Henry D. "LAPLACE’S LAW AND THE ALVEOLUS: A MISCONCEPTION OF ANATOMY AND A MISAPPLICATION OF PHYSICS." Advances in Physiology Education 27, no. 1 (March 2003): 34–40. http://dx.doi.org/10.1152/advan.00024.2002.

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Both the anatomy and the mechanics of inflation of the alveoli, as presented in most textbooks of physiology, have been misunderstood and misrepresented. The typical representation of the acinus as a “bunch of grapes” bears no resemblance to its real anatomy; the alveoli are not independent little balloons. Because of the prevalence of this misconception, Laplace’s law, as it applies to spheres, has been invoked as a mechanical model for the forces of alveolar inflation and as an explanation for the necessity of pulmonary surfactant in the alveolus. Alveoli are prismatic or polygonal in shape,
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Dong, Jun, Yan Qiu, Huimin Lv, Yue Yang, and Yonggang Zhu. "Investigation on Microparticle Transport and Deposition Mechanics in Rhythmically Expanding Alveolar Chip." Micromachines 12, no. 2 (February 12, 2021): 184. http://dx.doi.org/10.3390/mi12020184.

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The transport and deposition of micro/nanoparticles in the lungs under respiration has an important impact on human health. Here, we presented a real-scale alveolar chip with movable alveolar walls based on the microfluidics to experimentally study particle transport in human lung alveoli under rhythmical respiratory. A new method of mixing particles in aqueous solution, instead of air, was proposed for visualization of particle transport in the alveoli. Our novel design can track the particle trajectories under different force conditions for multiple periods. The method proposed in this study
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Bates, Jason H. T. "Understanding Alveolar Mechanics." Critical Care Medicine 41, no. 5 (May 2013): 1374–75. http://dx.doi.org/10.1097/ccm.0b013e31827c02b8.

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LIU, TIANYA, YUXING WANG, XIAOYU LIU, LAN YUAN, DEYU LI, HUITING QIAO, and YUBO FAN. "EFFECTS OF ALVEOLAR MORPHOLOGY ON ALVEOLAR MECHANICS: AN EXPERIMENTAL STUDY OF MOUSE LUNG BASED ON TWO- AND THREE-DIMENSIONAL IMAGING METHODS." Journal of Mechanics in Medicine and Biology 19, no. 04 (June 2019): 1950027. http://dx.doi.org/10.1142/s0219519419500271.

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Understanding alveolar mechanics is important for preventing the possible lung injuries during mechanical ventilation. Alveolar clusters with smaller size are found having lower compliance in two-dimensional studies. But the influence of alveolar shape on compliance is unclear. In order to investigate how alveolar morphology affects their behavior, we tracked subpleural alveoli of isolated mouse lungs during quasi-static ventilation using two- and three-dimensional imaging techniques. Results showed that alveolar clusters with smaller size and more spherical shape had lower compliance. There w
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Sera, Toshihiro, Hideo Yokota, Gaku Tanaka, Kentaro Uesugi, Naoto Yagi, and Robert C. Schroter. "Murine pulmonary acinar mechanics during quasi-static inflation using synchrotron refraction-enhanced computed tomography." Journal of Applied Physiology 115, no. 2 (July 15, 2013): 219–28. http://dx.doi.org/10.1152/japplphysiol.01105.2012.

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We visualized pulmonary acini in the core regions of the mouse lung in situ using synchrotron refraction-enhanced computed tomography (CT) and evaluated their kinematics during quasi-static inflation. This CT system (with a cube voxel of 2.8 μm) allows excellent visualization of not just the conducting airways, but also the alveolar ducts and sacs, and tracking of the acinar shape and its deformation during inflation. The kinematics of individual alveoli and alveolar clusters with a group of terminal alveoli is influenced not only by the connecting alveolar duct and alveoli, but also by the ne
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Roan, Esra, and Christopher M. Waters. "What do we know about mechanical strain in lung alveoli?" American Journal of Physiology-Lung Cellular and Molecular Physiology 301, no. 5 (November 2011): L625—L635. http://dx.doi.org/10.1152/ajplung.00105.2011.

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The pulmonary alveolus, terminal gas-exchange unit of the lung, is composed of alveolar epithelial and endothelial cells separated by a thin basement membrane and interstitial space. These cells participate in the maintenance of a delicate system regulated not only by biological factors but also by the mechanical environment of the lung, which undergoes dynamic deformation during breathing. Clinical and animal studies as well as cell culture studies point toward a strong influence of mechanical forces on lung cells and tissues including effects on growth and repair, surfactant release, injury,
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Perlman, Carrie E. "On modeling edematous alveolar mechanics." Journal of Applied Physiology 117, no. 8 (October 15, 2014): 937. http://dx.doi.org/10.1152/japplphysiol.00696.2014.

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Wilson, Theodore A., Ron C. Anafi, and Rolf D. Hubmayr. "Mechanics of edematous lungs." Journal of Applied Physiology 90, no. 6 (June 1, 2001): 2088–93. http://dx.doi.org/10.1152/jappl.2001.90.6.2088.

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Using the parenchymal marker technique, we measured pressure (P)-volume (P-V) curves of regions with volumes of ∼1 cm3 in the dependent caudal lobes of oleic acid-injured dog lungs, during a very slow inflation from P = 0 to P = 30 cmH2O. The regional P-V curves are strongly sigmoidal. Regional volume, as a fraction of volume at total lung capacity, remains constant at 0.4–0.5 for airway P values from 0 to ∼20 cmH2O and then increases rapidly, but continuously, to 1 at P = ∼25 cmH2O. A model of parenchymal mechanics was modified to include the effects of elevated surface tension and fluid in t
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Wilson, Theodore A. "Parenchymal mechanics, gas mixing, and the slope of phase III." Journal of Applied Physiology 115, no. 1 (July 1, 2013): 64–70. http://dx.doi.org/10.1152/japplphysiol.00112.2013.

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A model of parenchymal mechanics is revisited with the objective of investigating the differences in parenchymal microstructure that underlie the differences in regional compliance that are inferred from gas-mixing studies. The stiffness of the elastic line elements that lie along the free edges of alveoli and form the boundary of the lumen of the alveolar duct is the dominant determinant of parenchymal compliance. Differences in alveolar size cause parallel shifts of the pressure-volume curve, but have little effect on compliance. However, alveolar size also affects the relation between surfa
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McCann, Ulysse G., Henry J. Schiller, Louis A. Gatto, Jay M. Steinberg, David E. Carney, and Gary F. Nieman. "Alveolar mechanics alter hypoxic pulmonary vasoconstriction*." Critical Care Medicine 30, no. 6 (June 2002): 1315–21. http://dx.doi.org/10.1097/00003246-200206000-00028.

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Dissertations / Theses on the topic "Alveolar mechanics"

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Liu, Hui. "The application of alveolar microscope on alveolar mechanics of ventilator-induced lung injury." [S.l. : s.n.], 2008. http://nbn-resolving.de/urn:nbn:de:bsz:25-opus-61847.

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Dash, Shari Anne Ahmed El. "Estudo tomográfico de pressões de colapso alveolar e níveis isogravitacionais em pulmões de pacientes com SDRA e LPA." Universidade de São Paulo, 2009. http://www.teses.usp.br/teses/disponiveis/5/5159/tde-25062009-113611/.

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Estudo clínico prospectivo, em 11 pacientes com SARA ou LPA, avaliando o comportamento regional da densidade do tecido pulmonar e do colapso alveolar ao longo dos três eixos do espaço. Foram realizadas tomografias seriadas, após manobra de recrutamento inicial e após níveis de PEEP progressivamente decrescentes. Regressão linear múltipla (R2=0.83) mostrou importante gradiente no eixo gravitacional (p<0.001) e não no sentido céfalo-caudal (p<0.001), nem da direita para a esquerda (p<0.05). Isto corrobora o conceito do pulmão líquido, em que a resultante das pressões exercidas pelo diafragma, es
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Namati, Eman, and eman@namati com. "Pre-Clinical Multi-Modal Imaging for Assessment of Pulmonary Structure, Function and Pathology." Flinders University. Computer Science, Engineering and Mathematics, 2008. http://catalogue.flinders.edu.au./local/adt/public/adt-SFU20081013.044657.

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In this thesis, we describe several imaging techniques specifically designed and developed for the assessment of pulmonary structure, function and pathology. We then describe the application of this technology within appropriate biological systems, including the identification, tracking and assessment of lung tumors in a mouse model of lung cancer. The design and development of a Large Image Microscope Array (LIMA), an integrated whole organ serial sectioning and imaging system, is described with emphasis on whole lung tissue. This system provides a means for acquiring 3D pathology of fixed w
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Rolle, Trenicka. "Lung Alveolar and Tissue Analysis Under Mechanical Ventilation." VCU Scholars Compass, 2014. http://scholarscompass.vcu.edu/etd/3398.

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Mechanical ventilation has been a major therapy used by physicians in support of surgery as well as for treating patients with reduced lung function. Despite its many positive outcomes and ability to maintain life, in many cases, it has also led to increased injury of the lungs, further exacerbating the diseased state. Numerous studies have investigated the effects of long term ventilation with respect to lungs, however, the connection between the global deformation of the whole organ and the strains reaching the alveolar walls remains unclear. The walls of lung alveoli also called the alveola
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Liao, Pinhu. "Mechanotransduction in alveolar epithelial cells subjected to mechanical strain." Thesis, Imperial College London, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.479153.

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Chen, Shanze [Verfasser], and Silke [Akademischer Betreuer] Meiners. "Molecular mechanism of alveolar macrophage polarization and cell communication with alveolar epithelial cell / Shanze Chen. Betreuer: Silke Meiners." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2015. http://d-nb.info/1080479074/34.

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McKechnie, Stuart R. "The roles of hyperoxia and mechanical deformation in alveolar epithelial injury and repair." Thesis, University of Edinburgh, 2008. http://hdl.handle.net/1842/2691.

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The alveolar epithelium is a key functional component of the air-blood barrier in the lung. Comprised of two morphologically distinct cell types, alveolar epithelial type I (ATI) and type II (ATII) cells, effective repair of the alveolar epithelial barrier following injury appears to be an important determinant of clinical outcome. The prevailing view suggests this repair is achieved by the proliferation of ATII cells and the transdifferentiation of ATII cells into ATI cells. Supplemental oxygen and mechanical ventilation are key therapeutic interventions in the supportive treatment of respira
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Fois, Georgio [Verfasser]. "Response of alveolar type II pneumocytes to mechanical stimulation / Giorgio Fois." Ulm : Universität Ulm. Medizinische Fakultät, 2012. http://d-nb.info/1019167831/34.

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Dickie, A. John. "Mechanisms by which endotoxin-stimulated alveolar macrophages impair lung epithelial sodium transport." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape11/PQDD_0025/MQ51593.pdf.

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Mossadeq, Sayeed. "Kinetics and mechanisms of accumulation for liposomal ciprofloxacin into rat alveolar macrophages." VCU Scholars Compass, 2013. http://scholarscompass.vcu.edu/etd/501.

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The kinetics and mechanism of accumulation for liposomal ciprofloxacin (Lipo-CPFX) into the rat alveolar macrophage NR8383 cells were studied in vitro, in comparison to unformulated ciprofloxacin (CPFX). Upon incubation with CPFX or Lipo-CPFX, cellular drug accumulation was determined from the cell lysates or efflux was from the extracellular media by fluorescence-HPLC. The accumulation for Lipo-CPFX reached the asymptotic values at ≥ 2 hours, which was a result of uptake and efflux. The uptake appeared to be due to liposomes, mediated via cellular energy-independent mechanism like lipid fusio
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Books on the topic "Alveolar mechanics"

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Kreit, John W. Respiratory Mechanics. Edited by John W. Kreit. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190670085.003.0001.

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Ventilation can occur only when the respiratory system expands above and then returns to its resting or equilibrium volume. This is just another way of saying that ventilation depends on our ability to breathe. Although breathing requires very little effort and even less thought, it’s nevertheless a fairly complex process. Respiratory Mechanics reviews the interaction between applied and opposing forces during spontaneous and mechanical ventilation. It discusses elastic recoil, viscous forces, compliance, resistance, and the equation of motion and the time constant of the respiratory system. I
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Muders, Thomas, and Christian Putensen. Pressure-controlled mechanical ventilation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0096.

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Beside reduction in tidal volume limiting peak airway pressure minimizes the risk for ventilator-associated-lung-injury in patients with acute respiratory distress syndrome. Pressure-controlled, time-cycled ventilation (PCV) enables the physician to keep airway pressures under strict limits by presetting inspiratory and expiratory pressures, and cycle times. PCV results in a square-waved airway pressure and a decelerating inspiratory gas flow holding the alveoli inflated for the preset time. Preset pressures and cycle times, and respiratory system mechanics affect alveolar and intrinsic positi
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Martin-Loeches, Ignacio, and Antonio Artigas. Respiratory support with positive end-expiratory pressure. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0094.

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Positive-end-expiratory pressure (PEEP) is the pressure present in the airway (alveolar pressure) above atmospheric pressure that exists at the end of expiration. The term PEEP is defined in two particular settings. Extrinsic PEEP (applied by ventilator) and intrinsic PEEP (PEEP caused by non-complete exhalation causing progressive air trapping). Applied (extrinsic) PEEP—is usually one of the first ventilator settings chosen when mechanical ventilation (MV) is initiated. Applying PEEP increases alveolar pressure and volume. The increased lung volume increases the surface area by reopening and
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MacIntyre, Neil R. Indications for mechanical ventilation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0091.

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Mechanical ventilation is indicated when the patient’s ability to ventilate the lung and/or effect gas transport across the alveolar capillary interface is compromised to point that harm is imminent. In practice, this means addressing one or more of three fundamental pathophysiological processes—loss of proper ventilatory control, ventilatory muscle demand-capability imbalances, and/or loss of alveolar patency. A fourth general indication involves providing a positive pressure assistance to allow tolerance of an artificial airway in the patient unable to maintain a patent and protected airway.
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Ware, Lorraine B. Pathophysiology of acute respiratory distress syndrome. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0108.

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The acute respiratory distress syndrome (ARDS) is a syndrome of acute respiratory failure characterized by the acute onset of non-cardiogenic pulmonary oedema due to increased lung endothelial and alveolar epithelial permeability. Common predisposing clinical conditions include sepsis, pneumonia, severe traumatic injury, and aspiration of gastric contents. Environmental factors, such as alcohol abuse and cigarette smoke exposure may increase the risk of developing ARDS in those at risk. Pathologically, ARDS is characterized by diffuse alveolar damage with neutrophilic alveolitis, haemorrhage,
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Lucangelo, Umberto, and Massimo Ferluga. Pulmonary mechanical dysfunction in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0084.

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In intensive care units practitioners are confronted every day with mechanically-ventilated patients and should be able to sort out from all the data available from modern ventilators to tailored patient ventilatory strategy. Real-time visualization of pressure, flow and tidal volume provide valuable information on the respiratory system, to optimize ventilatory support and avoiding complications associated with mechanical ventilation. Early determination of patient–ventilator asynchrony, air-trapping, and variation in respiratory parameters is important during mechanical ventilation. A correc
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Joynt, Gavin M., and Gordon Y. S. Choi. Blood gas analysis in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0072.

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Arterial blood gases allow the assessment of patient oxygenation, ventilation, and acid-base status. Blood gas machines directly measure pH, and the partial pressures of carbon dioxide (PaCO2) and oxygen (PaO2) dissolved in arterial blood. Oxygenation is assessed by measuring PaO2 and arterial blood oxygen saturation (SaO2) in the context of the inspired oxygen and haemoglobin concentration, and the oxyhaemoglobin dissociation curve. Causes of arterial hypoxaemia may often be elucidated by determining the alveolar–arterial oxygen gradient. Ventilation is assessed by measuring the PaCO2 in the
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Hedenstierna, Göran, and Hans Ulrich Rothen. Physiology of positive-pressure ventilation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0088.

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During positive pressure ventilation the lung volume is reduced because of loss of respiratory muscle tone. This promotes airway closure that occurs in dependent lung regions. Gas absorption behind the closed airway results sooner or later in atelectasis depending on the inspired oxygen concentration. The elevated airway and alveolar pressures squeeze blood flow down the lung so that a ventilation/perfusion mismatch ensues with more ventilation going to the upper lung regions and more perfusion going to the lower, dependent lung. Positive pressure ventilation may impede the return of venous bl
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Cuartero, Mireia, and Niall D. Ferguson. High-frequency ventilation and oscillation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0098.

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High-frequency oscillatory ventilation (HFOV) is a key member of the family of modes called high-frequency ventilation and achieves adequate alveolar ventilation despite using very low tidal volumes, often below the dead space volume, at frequencies significantly above normal physiological values. It has been proposed as a potential protective ventilatory strategy, delivering minimal alveolar tidal stretch, while also providing continuous lung recruitment. HFOV has been successfully used in neonatal and paediatric intensive care units over the last 25 years. Since the late 1990s adults with ac
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Lumb, Andrew B., and Natalie Drury. Respiratory physiology in anaesthetic practice. Edited by Jonathan G. Hardman. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0002.

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Moving away from the structure of traditional texts, this chapter follows the journey of oxygen molecules as they move from inspired air to their point of use in mitochondria, with some digressions along the way to cover other relevant aspects of respiratory physiology. The chapter encompasses all the key aspects of respiratory physiology and also highlights physiological alterations that occur under both general and regional anaesthesia, moving the physiological principles discussed into daily anaesthetic practice. The chapter explores relevant anatomy of the airways, lungs, and pleura. The h
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Book chapters on the topic "Alveolar mechanics"

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Romero, P. V. "Alveolar micromechanics." In Basics of Respiratory Mechanics and Artificial Ventilation, 119–31. Milano: Springer Milan, 1999. http://dx.doi.org/10.1007/978-88-470-2273-7_10.

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Albert, S., B. Kubiak, and G. Nieman. "Protective Mechanical Ventilation: Lessons Learned From Alveolar Mechanics." In Yearbook of Intensive Care and Emergency Medicine, 245–55. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-77290-3_23.

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Stenqvist, O., and H. Odenstedt. "Alveolar Pressure/volume Curves Reflect Regional Lung Mechanics." In Yearbook of Intensive Care and Emergency Medicine, 407–14. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-49433-1_37.

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Tanne, Kazuo. "Biomechanical Responses of Craniofacial and Alveolar Bones to Mechanical Forces in Orthodontics." In Interfaces in Medicine and Mechanics—2, 299–308. Dordrecht: Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3852-9_31.

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Meissner, S., L. Knels, T. Koch, E. Koch, S. Adami, X. Y. Hu, and N. A. Adams. "Experimental and Numerical Investigation on the Flow-Induced Stresses on the Alveolar-Epithelial-Surfactant-Air Interface." In Notes on Numerical Fluid Mechanics and Multidisciplinary Design, 67–80. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-20326-8_4.

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Scharnagl, Hubert, Winfried März, Markus Böhm, Thomas A. Luger, Federico Fracassi, Alessia Diana, Thomas Frieling, et al. "Alveolar Proteinosis." In Encyclopedia of Molecular Mechanisms of Disease, 69. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_6651.

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Scharnagl, Hubert, Winfried März, Markus Böhm, Thomas A. Luger, Federico Fracassi, Alessia Diana, Thomas Frieling, et al. "Alveolar Lipoproteinosis." In Encyclopedia of Molecular Mechanisms of Disease, 69. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_6652.

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Scharnagl, Hubert, Winfried März, Markus Böhm, Thomas A. Luger, Federico Fracassi, Alessia Diana, Thomas Frieling, et al. "Alveolar Phospholipidosis." In Encyclopedia of Molecular Mechanisms of Disease, 69. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_6653.

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Braun-Falco, Markus, Henry J. Mankin, Sharon L. Wenger, Markus Braun-Falco, Stephan DiSean Kendall, Gerard C. Blobe, Christoph K. Weber, et al. "Pulmonary Alveolar Phospholipoproteinosis." In Encyclopedia of Molecular Mechanisms of Disease, 1754–55. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_6655.

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Braun-Falco, Markus, Henry J. Mankin, Sharon L. Wenger, Markus Braun-Falco, Stephan DiSean Kendall, Gerard C. Blobe, Christoph K. Weber, et al. "Pulmonary Alveolar Proteinosis." In Encyclopedia of Molecular Mechanisms of Disease, 1755–56. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_1491.

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Conference papers on the topic "Alveolar mechanics"

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Dong, Jun, Huimin Lv, Yue Yang, and Yonggang Zhu. "Mixing in deformable alveolar cavity." In 22nd Australasian Fluid Mechanics Conference AFMC2020. Brisbane, Australia: The University of Queensland, 2020. http://dx.doi.org/10.14264/5c5ed86.

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Oeckler, RA, BJ Walters, RW Stroetz, and RD Hubmayr. "Osmotic Pressure Alters Alveolar Epithelial Cell Plasma Membrane Mechanics Via PIP2 and Cytoskeletal Rearrangement." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a2499.

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Merrikh, A. A., and J. L. Lage. "Time-Dependent Diffusion in the Alveolar Region of the Lungs: Effect of Moving Red Blood Cells." In ASME 2002 International Mechanical Engineering Congress and Exposition. ASMEDC, 2002. http://dx.doi.org/10.1115/imece2002-39530.

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Results from a preliminary numerical simulation of alveolar gas diffusion with moving capillary red blood cells (RBCs) are presented. The alveolar region is modeled with four basic constituents, namely the alveolus (or gas region), the tissue (a region lumping the alveolar and capillary membranes, and the interstitial fluid), the blood plasma (a liquid region) and the RBCs. A single, straight capillary with equally spaced RBCs moving together with the blood plasma is considered in this preliminary study. The numerical simulation attempts also to mimic the time-varying gas concentration in the
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Wallbank, A. M., S. Niemiec, C. Zgheib, E. Nozik, K. Liechty, and B. J. Smith. "The Relationship Between Alveolar Leak and Lung Mechanics in Endotoxin-Induced Acute Lung Injury with CNP-miR146a Treatment." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a4659.

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Krueger, Alexander, Lilla Knels, Sven Meissner, Martina Wendel, Axel R. Heller, Thomas Lambeck, Thea Koch, and Edmund Koch. "Three-dimensional Fourier-domain optical coherence tomography of alveolar mechanics in stepwise inflated and deflated isolated and perfused rabbit lungs." In European Conference on Biomedical Optics. Washington, D.C.: OSA, 2007. http://dx.doi.org/10.1364/ecbo.2007.6627_6.

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Krueger, Alexander, Lilla Knels, Sven Meissner, Martina Wendel, Axel R. Heller, Thomas Lambeck, Thea Koch, and Edmund Koch. "Three-dimensional Fourier-domain optical coherence tomography of alveolar mechanics in stepwise inflated and deflated isolated and perfused rabbit lungs." In European Conference on Biomedical Optics, edited by Peter E. Andersen and Zhongping Chen. SPIE, 2007. http://dx.doi.org/10.1117/12.727891.

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Parameswaran, Harikrishnan, Ascanio D. Araújo, and Béla Suki. "Estimating Mechanical Forces In The Alveolar Walls." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a3653.

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Schneider, D., K. Smith, J. Speth, C. Wilke, D. Lyons, L. R. K. Penke, A. Lauring, B. B. Moore, and M. Peters-Golden. "Mechanisms of Alveolar Macrophage Derived Extracellular Vesicle Defense Against Influenza Infection of Alveolar Epithelial Cells." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a7425.

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Wall, Wolfgang A., Andrew Comerford, Lena Wiechert, and Sophie Rausch. "Coupled and Multi-Scale Building Blocks for a Comprehensive Computational Lung Model." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206407.

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Abstract:
Mechanical ventilation is a vital supportive therapy for critical care patients suffering from Acute Respiratory Distress syndrome (ARDS) or Acute Lung Injury (ALI) in view of oxygen supply. However, a number of associated complications often occur, which are collectively termed ventilator induced lung injuries (VILI) [1]. Biologically, these diseases manifest themselves at the alveolar level and are characterized by inflammation of the lung parenchyma following local overdistension or high shear stresses induced by frequent alveolar recruitment and derecruitment. Despite the more recent adopt
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McGee, Maria, and Henry Rothberger. "MECHANISMS OF PROCOAGULANT GENERATION BY ALVEOLAR MACROPHAGES DURING MATURATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643168.

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Abstract:
During maturation in vivo and in vitro alveolar macrophages generate procoagulant(s) capable of activating the extrinsic pathway. It is generally agreed that at least part of the activity is due to TF (tissue factor). However, whether or not macrophages also generate functional factor VII or X is controversial. To characterize procoagulant activity increases, we measured kinetic parameters defining interactions between components of the TF-VII complex on membranes of alveolar macrophages either freshly isolated or cultured in serum free medium. In incubation mixtures with fixed concentrations
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