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Relevant bibliographies by topics / Alveolar type cell

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Academic literature on the topic 'Alveolar type cell'

Author: Grafiati

Published: 1 April 2023

Last updated: 31 July 2025

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Journal articles on the topic "Alveolar type cell"

1

Sutherland, Leanne M., Yasmin S. Edwards, and Andrew W. Murray. "Alveolar type II cell apoptosis." Comparative Biochemistry and Physiology Part A: Molecular & Integrative Physiology 129, no. 1 (2001): 267–85. http://dx.doi.org/10.1016/s1095-6433(01)00323-3.

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Dormans, J. A. M. A. "The alveolar type III cell." Lung 163, no. 1 (1985): 327–35. http://dx.doi.org/10.1007/bf02713833.

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Griffin, M., R. Bhandari, G. Hamilton, Y. C. Chan, and J. T. Powell. "Alveolar type II cell-fibroblast interactions, synthesis and secretion of surfactant and type I collagen." Journal of Cell Science 105, no. 2 (1993): 423–32. http://dx.doi.org/10.1242/jcs.105.2.423.

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During alveolar development and alveolar repair close contacts are established between fibroblasts and lung epithelial cells through gaps in the basement membrane. Using co-culture systems we have investigated whether these close contacts influence synthesis and secretion of the principal surfactant apoprotein (SP-A) by cultured rat lung alveolar type II cells and the synthesis and secretion of type I collagen by fibroblasts. The alveolar type II cells remained cuboidal and grew in colonies on fibroblast feeder layers and on Matrigel-coated cell culture inserts but were progressively more flat

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Paine, R., P. Christensen, G. B. Toews, and R. H. Simon. "Regulation of alveolar epithelial cell ICAM-1 expression by cell shape and cell-cell interactions." American Journal of Physiology-Lung Cellular and Molecular Physiology 266, no. 4 (1994): L476—L484. http://dx.doi.org/10.1152/ajplung.1994.266.4.l476.

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In normal lung, intercellular adhesion molecule 1 (ICAM-1) is expressed at high levels on thin type I alveolar epithelial cells, but is minimally expressed on cuboidal type II cells. ICAM-1 is induced in primary culture on tissue culture-treated plastic as type II cells undergo transition toward a type I cell-like phenotype. We hypothesized that alveolar epithelial cell expression of ICAM-1 might be regulated in part by signals that influence the state of differentiation of these cells. We found that rat type II cells that were cultured as aggregates of cuboidal cells on a hydrated basement me

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Planus, E., S. Galiacy, M. Matthay, et al. "Role of collagenase in mediating in vitro alveolar epithelial wound repair." Journal of Cell Science 112, no. 2 (1999): 243–52. http://dx.doi.org/10.1242/jcs.112.2.243.

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Type II pneumocytes are essential for repair of the injured alveolar epithelium. The effect of two MMP collagenases, MMP-1 and MMP-13 on alveolar epithelial repair was studied in vitro. The A549 alveolar epithelial cell line and primary rat alveolar epithelial cell cultures were used. Cell adhesion and cell migration were measured with and without exogenous MMP-1. Wound healing of a cell monolayer of rat alveolar epithelial cell after a mechanical injury was evaluated by time lapse video analysis. Cell adhesion on type I collagen, as well as cytoskeleton stiffness, was decreased in the presenc

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Alder, Jonathan K., Christina E. Barkauskas, Nathachit Limjunyawong, et al. "Telomere dysfunction causes alveolar stem cell failure." Proceedings of the National Academy of Sciences 112, no. 16 (2015): 5099–104. http://dx.doi.org/10.1073/pnas.1504780112.

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Telomere syndromes have their most common manifestation in lung disease that is recognized as idiopathic pulmonary fibrosis and emphysema. In both conditions, there is loss of alveolar integrity, but the underlying mechanisms are not known. We tested the capacity of alveolar epithelial and stromal cells from mice with short telomeres to support alveolar organoid colony formation and found that type 2 alveolar epithelial cells (AEC2s), the stem cell-containing population, were limiting. When telomere dysfunction was induced in adult AEC2s by conditional deletion of the shelterin component telom

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Ashino, Yugo, Xiaoyou Ying, Leland G. Dobbs, and Jahar Bhattacharya. "[Ca2+]i oscillations regulate type II cell exocytosis in the pulmonary alveolus." American Journal of Physiology-Lung Cellular and Molecular Physiology 279, no. 1 (2000): L5—L13. http://dx.doi.org/10.1152/ajplung.2000.279.1.l5.

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Pulmonary surfactant, a critical determinant of alveolar stability, is secreted by alveolar type II cells by exocytosis of lamellar bodies (LBs). To determine exocytosis mechanisms in situ, we imaged single alveolar cells from the isolated blood-perfused rat lung. We quantified cytosolic Ca2+ concentration ([Ca2+]i) by the fura 2 method and LB exocytosis as the loss of cell fluorescence of LysoTracker Green. We identified alveolar cell type by immunofluorescence in situ. A 15-s lung expansion induced synchronous [Ca2+]i oscillations in all alveolar cells and LB exocytosis in type II cells. The

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Hammerschmidt, Stefan, Hartmut Kuhn, Christian Gessner, Hans-Jurgen Seyfarth, and Hubert Wirtz. "Stretch-Induced Alveolar Type II Cell Apoptosis." American Journal of Respiratory Cell and Molecular Biology 37, no. 6 (2007): 699–705. http://dx.doi.org/10.1165/rcmb.2006-0429oc.

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Patel, Namrata B., and Jason D. Christie. "Alveolar Type 2 Cell Transplantation in IPF." Chest 150, no. 3 (2016): 481–82. http://dx.doi.org/10.1016/j.chest.2016.05.036.

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Wang, P. M., E. Fujita, and J. Bhattacharya. "Vascular regulation of type II cell exocytosis." American Journal of Physiology-Lung Cellular and Molecular Physiology 282, no. 5 (2002): L912—L916. http://dx.doi.org/10.1152/ajplung.00303.2001.

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To determine whether lung capillary pressure regulates surfactant secretion, we viewed alveoli of the constantly inflated, isolated blood-perfused rat lung by fluorescence microscopy. By alveolar micropuncture we infused fura 2 and lamellar body (LB)-localizing dyes for fluorescence detection of, respectively, the alveolar cytosolic Ca2+concentration ([Ca2+]i) and type II cell exocytosis. Increasing left atrial pressure (Pla) from 5 to 10 cmH2O increased septal capillary diameter by 26% and induced marked alveolar [Ca2+]i oscillations that abated on relief of pressure elevation. The rate of lo

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Dissertations / Theses on the topic "Alveolar type cell"

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Bhandari, R. N. B. "Characterization of a cell adhesion receptor on rat lung alveolar type 2 cells." Thesis, Imperial College London, 1988. http://hdl.handle.net/10044/1/46962.

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Hofer, Christian Carlisle. "Effects of Influenza Infection on Murine Alveolar Type II Cell Function." The Ohio State University, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=osu1406201295.

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Hasegawa, Kouichi. "Fraction of MHCII and EpCAM expression characterizes distal lung epithelial cells for alveolar type 2 cell isolation." Kyoto University, 2018. http://hdl.handle.net/2433/232118.

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Downs, Charles. "Cigarette Smoke Extract-Induced Injury in Alveolar Cells in Model Systems." Diss., The University of Arizona, 2011. http://hdl.handle.net/10150/201510.

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Cigarette smoke contributes to many diseases. The actions of second and third hand smoke, which have implications for non-smokers and the very young, are just beginning to be appreciated. The overarching hypothesis of this project is that cigarette smoke has different injurious actions on alveolar cells based on chronological age. The purpose here was to learn more about the susceptibility of alveolar cells to cigarette smoke extract (CSE)- induced injury by performing studies on pulmonary alveolar and endothelial cells derived from neonatal, young, and old rats. The aims involved: 1. Develo

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Downs, Charles A., Abdel A. Alli, Nicholle M. Johnson, and My N. Helms. "Cigarette smoke extract is a Nox agonist and regulates ENaC in alveolar type 2 cells." AMER INST MATHEMATICAL SCIENCES-AIMS, 2016. http://hdl.handle.net/10150/621494.

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There is considerable evidence that cigarette smoking is the primary etiology of chronic obstructive pulmonary disease (COPD), and that oxidative stress occurs in COPD with the family of tissue nicotinamide adenine dinucleotide phosphate (NADPH) oxidase (Nox) enzymes playing a significant role in lung pathogenesis. The purpose of this study was to determine the effects of cigarette smoke extract (CSE) on Nox signaling to epithelial sodium channels (ENaCs). Pre-treatment with diphenyleneiodonium (DPI), a pan-Nox inhibitor, prevented stimulatory effects of CSE on ENaC activity; open probability

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Clegg, Gareth Roger. "Co-expression of lung alveolar epithelial type I and II cell-selective proteins in response to injury." Thesis, University of Edinburgh, 2007. http://hdl.handle.net/1842/29066.

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This study used a novel combination of ATI and ATII cell-selective antibodies to investigate the phenotype of the alveolar epithelium following Staphylococcus aureus-induced ‘direct’ lung injury. Following distal airway instillation of S. aureus, the alveolar epithelium was covered with ATII cells (MMC4/RTII70-positive cells) and ATI cells (RTI40-positive cells) as seen in control lungs. However, the surface area covered by ATII cells was significantly increased, while the surface area covered by ATI cells was significantly decreased, in comparison with controls. The alveolar wall of S. aure

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Korogi, Yohei. "In Vitro Disease Modeling of Hermansky-Pudlak Syndrome Type 2 Using Human Induced Pluripotent Stem Cell-Derived Alveolar Organoids." Kyoto University, 2019. http://hdl.handle.net/2433/243303.

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Dysart, Marilyn Markowski. "Remodeling of the pulmonary microenvironment controls transforming growth factor-beta activation and alveolar type II epithelial to mesenchymal transition." Diss., Georgia Institute of Technology, 2014. http://hdl.handle.net/1853/53421.

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Pulmonary fibrosis is a potentially deadly pathology characterized by excessive deposition of extracellular matrix (ECM), increased tissue stiffness, and loss of tissue structure and function. Recent evidence has suggested epithelial to mesenchymal transition (EMT), the transdifferentiation of an epithelial cell into a mesenchymal fibroblast, is one mechanism that results in the accumulation of myofibroblasts and excessive deposition of ECM. EMT is a highly orchestrated process involving the integration of biochemical signals from specific integrin mediated interactions with ECM proteins and s

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Doolittle, Lauren May. "The Impact of Alveolar Type II Cell Mitochondrial Damage and Altered Energy Production on Acute Respiratory Distress Syndrome Development During Influenza A Virus Infection." The Ohio State University, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu159224389333959.

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Assis, Adriano Freitas de. "Desenvolvimento do fenótipo osteoblástico em células derivadas de osso alveolar humano cultivadas sobre titânio revestido com colágeno tipo I." Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/58/58136/tde-30062008-140042/.

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Os eventos celulares e extracelulares que ocorrem durante o processo de osseointegração do titânio (Ti) são bastante influenciados por suas propriedades de superfície, como morfologia, topografia e composição química. A modificação bioquímica da superfície do Ti consiste em imobilizar proteínas ou peptídeos nessa superfície com a finalidade de induzir respostas celulares e teciduais específicas na interface osso-implante que acelerem ou aumentem a osseointegração. O objetivo deste estudo foi avaliar o desenvolvimento do fenótipo osteoblástico em culturas de células crescidas sobre Ti revestido

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Books on the topic "Alveolar type cell"

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Verfasser, Schließmann Stephan J., Kirschbaum Andreas Verfasser, Plönes Till 1976 Verfasser, et al., eds. Roflumilast-N-oxide induces surfactant protein expression in human alveolar epithelial cells type II. Universität, 2012.

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Gandhi, Shephali G. The effect of pulmonary edema fluid on ion transport by adult alveolar type II epithelial cells. 2007.

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Book chapters on the topic "Alveolar type cell"

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Kauffman, Shirley L., and Tamiko Sato. "Alveolar Type II Cell Adenoma, Lung, Mouse." In Respiratory System. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-96846-4_16.

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Panos, Ralph J. "Cytokines and Alveolar Type II Cells." In Cytokines of the Lung. CRC Press, 2022. http://dx.doi.org/10.1201/9781003066927-17.

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Lwebuga-Mukasa, J. S. "Isolation of type Il alveolar epithelial cells." In Methods in Pulmonary Research. Birkhäuser Basel, 1998. http://dx.doi.org/10.1007/978-3-0348-8855-4_15.

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Liu, Yuru. "Type II Cells as Progenitors in Alveolar Repair." In Lung Stem Cells in the Epithelium and Vasculature. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-16232-4_2.

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Kosmider, Beata, Robert J. Mason, and Karim Bahmed. "Isolation and Characterization of Human Alveolar Type II Cells." In Methods in Molecular Biology. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8570-8_7.

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Jansing, Nicole L., Jazalle McClendon, Hidenori Kage, et al. "Isolation of Rat and Mouse Alveolar Type II Epithelial Cells." In Methods in Molecular Biology. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8570-8_6.

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Burkhardt, R., P. von Wichert, J. J. Batenburg, and L. M. G. van Golde. "Regulation of Phosphatidylcholine Synthesis in Type II Alveolar Epithelial Cells." In Surfactant Replacement Therapy. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-73305-5_43.

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Thiedemann, K. U., I. Paulini, N. Lüthe, et al. "Regeneration, Differentiation, and Neoplastic Transformation of Type II Alveolar Epithelial Cells." In Advances in Controlled Clinical Inhalation Studies. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77176-7_18.

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Schließmann, Stephan J., K. Höhne, A. Charra, et al. "Differences in form stability between human non-tumorous alveolar epithelial cells type 2 and alveolar carcinoma cells under biaxial stretching." In IFMBE Proceedings. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-89208-3_483.

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Gonzalez, Robert F., and Leland G. Dobbs. "Isolation and Culture of Alveolar Epithelial Type I and Type II Cells from Rat Lungs." In Methods in Molecular Biology. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-62703-125-7_10.

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Conference papers on the topic "Alveolar type cell"

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Wugk, Sophia, and Suzanne M. Cloonan. "Iron-loaded alveolar macrophages alter alveolar type 2 epithelial cell function in COPD." In ERS Lung Science Conference 2025 abstracts. European Respiratory Society, 2025. https://doi.org/10.1183/23120541.lsc-2025.tp237.

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Li, C., N. Peinado, S. M. Smith, et al. "Wnt5a Promotes Alveolar Type 1 Cell Differentiation During Lung Development." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a5480.

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Phan, Thi Thanh Huyen, Manu M. Kanti, Silvia Schauer, et al. "Lipid hydrolysis fuels alveolar type 2 cell-mediated lung regeneration." In ERS Lung Science Conference 2025 abstracts. European Respiratory Society, 2025. https://doi.org/10.1183/23120541.lsc-2025.tp107.

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Hirayama, Y., K. Hasegawa, S. Ashino, et al. "Analysis of Functional Dynamics of Healthy Type 2 Alveolar Epithelial Cells as Progenitor Cells in Response to Type 1 Alveolar Epithelial Cell Specific Lung Injury." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a2574.

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Kulish, Vladimir V., José L. Lage, Connie C. W. Hsia, and Robert L. Johnson. "Red Blood Cell Distribution Effect on Lung Diffusing Capacity: A Macroscopic Analysis." In ASME 2000 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2000. http://dx.doi.org/10.1115/imece2000-2228.

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Abstract A novel mathematical model derived from fundamental engineering principles for simulating the spatial and temporal gas diffusion process within the alveolar region of the lung was presented recently by Koulich et al. [1]. The model depends on a physical property of the alveolar region termed effective diffusivity, function of the diffusivity, solubility, and interface geometry of each alveolar constituent. Unfortunately, the direct determination of the effective diffusivity of the alveolar region is impractical because of the difficulty in describing the internal geometry of each alve

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Driscoll, Barbara, Jooeun Lee, Raghava Reddy, et al. "Distal Lung Regeneration Following Targeted Alveolar Epithelial Type 2 Cell Depletion." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4206.

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Zhang, D., L. A. Vickers, N. Gao, A. A. Sathe, C. Xing, and C. K. Garcia. "Single Cell Transcriptomic Profiling of Alveolar Epithelial Type 2 Cells in Sftpa1 G231V Mice." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a6144.

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Lehmann, M., Q. Hu, Y. Hu, et al. "Inflammaging and Impaired Progenitor Cell Function in Aged Alveolar Epithelial Type (AT) II Cells." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a4205.

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Korogi, Y., S. Gotoh, S. Ikeo, et al. "Alveolar Epithelial Type 2 Cell Dysfunction in Hermansky-Pudlak Syndrome Type 2 Patient-Derived Induced Pluripotent Stem Cells." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5268.

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Allawzi, A., R. Roy, N. Burns, et al. "Alveolar Type 2 Cell Dependent Lactate Resolves Alveolar Inflammation via Crosstalk with Resident Alveolar Macrophages in Two Models of Acute Lung Injury." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a4500.

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Reports on the topic "Alveolar type cell"

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Lin, Hongwei, Yanjun Gao, Kang Sun, and Faguang Jin. Association between PM2.5 pollution and outpatient visits for respiratory diseases in China: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.5.0144.

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Review question / Objective: Previous epidemiological studies on the association between PM2.5 pollution and outpatient visits for respiratory diseases in China were mostly limited to one region， and the different papers have no coherent results. Our objective is to perform a systematic review and meta-analysis of the relevant literature in order to summarize the association between PM2.5 pollution and outpatient visits for respiratory diseases in multiple cities in China. Condition being studied: As an important component of air pollutants, particulate matter 2.5 (PM2.5) can float in the atmo

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Incidence of Lung Cancer Types. National Center for Chronic Disease Prevention and Health Promotion (U.S.). Division of Cancer Prevention and Control., 2024. https://doi.org/10.15620/cdc/174572.

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More lung cancers, including small cell carcinomas, adenocarcinomas, and squamous cell carcinomas, are being found at an early stage when they are more treatable. Screening can help find lung cancers of all types before they start spreading. Lung cancer begins in the airways of the lungs. These include the bronchi (the tubes leading to the lungs), the bronchioles (tiny branches of air tubes in the lungs), and the alveoli (the tiny air sacs at the end of the bronchioles). Types of lung cancer are named after the cells found in the cancer. Doctors use special tests to determine what type of cell

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