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Relevant bibliographies by topics / American Haibun

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Journal articles

Academic literature on the topic 'American Haibun'

Author: Grafiati

Published: 4 June 2021

Last updated: 30 July 2024

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Journal articles on the topic "American Haibun"

1

Cole, Terry. "A Review of: “Franklyn S. Haiman, Religious Expression and the American Constitution”." Southern Communication Journal 71, no. 3 (2006): 309–11. http://dx.doi.org/10.1080/10417940600942851.

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2

Ghosh, Sweta, Rajbir Singh, Zachary M. Vanwinkle, et al. "Abstract 3250: Microbial metabolite, Urolithin A acts as chemo-sensitizing adjuvant in conventional drug therapies via modulation of drug transporters and EMT markers." Cancer Research 82, no. 12_Supplement (2022): 3250. http://dx.doi.org/10.1158/1538-7445.am2022-3250.

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Abstract Colon cancer is the third leading cause in cancer related deaths in United States of America. Chemoresistance (drug resistance) of tumors is the primary reason for the failure of chemotherapy and a major cause of mortality in colon cancer. The molecular mechanisms involved in chemoresistance or methods to chemosensitization of cancer cells to chemotherapy remain elusive. Recent studies suggest that gut microbiota and microbial metabolites play a crucial role in the development and progression of colon cancer. Urolithin A (UroA) is a gut microbial metabolite derived from ellagitannin/e

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3

Azuaje-Alamo, Manuel. "“(In)comparable Poetries and Transpacific Networks of Translation”." Journal of World Literature 4, no. 4 (2019): 581–604. http://dx.doi.org/10.1163/24056480-00404007.

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Abstract In 1957, writing in Spanish, the Mexican poet Octavio Paz (1914–1998) published the first complete translation into a Western language of the famous travel diary Oku no Hosomichi (The Narrow Road to the Deep North, 1702) by Matsuo Bashō (1644–1694). A thoroughly revised second edition followed in 1970, which included freer translations of Bashō’s haikus. In this definite edition, Paz attempted to synthesize the poetic effect of Bashō’s work for a Latin American readership. By using a textual and archival-based approach, this article analyses Paz’s two Spanish versions against the Japa

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4

Zhou, Haibin, Dimin Wu, Longchuan Bai, et al. "Abstract 4519: Discovery of highly potent, selective and efficacious STAT3 PROTAC degraders capable of achieving complete tumor regression." Cancer Research 84, no. 6_Supplement (2024): 4519. http://dx.doi.org/10.1158/1538-7445.am2024-4519.

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Abstract STAT3 (signal transducer and activator of transcription 3) is a transcription factor and a promising therapeutic targets for cancer and other human diseases. We have previously reported the discovery of SD-36 and SD-91 as potent, selective and highly efficacious STAT3 degraders. In the present study, we report the discovery and extensive evaluation of highly potent, selective and efficacious new STAT3 degraders designed using novel cereblon ligands and novel STAT3 ligands. Our most potent STAT3 degraders are &gt;10-times more potent than SD-36 in inducing STAT3 degradation in cell

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5

Acharyya, Ranjan K., Longchuan Bai, Haibin Zhao, et al. "Abstract 4509: Discovery of potent and highly efficacious STAT3 PROTAC degraders capable of achieving long-lasting tumor regression." Cancer Research 84, no. 6_Supplement (2024): 4509. http://dx.doi.org/10.1158/1538-7445.am2024-4509.

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Abstract STAT3 (signal transducer and activator of transcription 3) is a transcription factor and a promising therapeutic targets for cancer and other human diseases. We have previously reported the discovery of SD-36 and SD-91 as potent, selective and highly efficacious STAT3 degraders. In the present study, we report the discovery of highly potent, selective and efficacious new STAT3 degraders. These STAT3 degraders were designed using our high-affinity STAT3 ligands and high-affinity VHL-1 ligands, with UM-STAT3-3100 being the best. In direct comparison, UM-STAT3-3100 is &gt;10-times mo

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6

Zhao, Xiaobei, Jie Zhu, Yaqiong Zhou, et al. "Abstract 1886: BRY812, an anti-LIV-1 antibody drug conjugate with novel conjugation method for cancer therapeutics." Cancer Research 84, no. 6_Supplement (2024): 1886. http://dx.doi.org/10.1158/1538-7445.am2024-1886.

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Abstract LIV-1, also known as SLC39A6 or ZIP6, belongs to the zinc transporter family and was initially identified as an estrogen-inducible gene in breast cancer. Immunohistochemical (IHC) analysis have revealed escalated LIV-1 expression in estrogen receptor-positive (ER+), hormone-treated tumors (both primary and metastatic sites) as well as ER-/PR-/Her2- (triple-negative) breast cancers. Notably, healthy human tissues exhibit limited LIV-1 expression, primarily in hormone regulated organs (prostate, uterus, and breast). The escalated expression of LIV-1 in breast and prostate cancer, as wel

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7

Zhou, Haibin, Dimin Wu, Longchuan Bai, et al. "Abstract 3881: Discovery of highly potent, selective and efficacious STAT3 PROTAC degraders capable of achieving long-lasting tumor regression." Cancer Research 84, no. 6_Supplement (2024): 3881. http://dx.doi.org/10.1158/1538-7445.am2024-3881.

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Abstract STAT3 (signal transducer and activator of transcription 3) is a transcription factor and a promising therapeutic targets for cancer and other human diseases. We have previously reported the discovery of SD-36 and SD-91 as potent, selective and highly efficacious STAT3 degraders. In the present study, we report the discovery and extensive evaluation of new, highly potent, selective and efficacious new STAT3 degraders. In direct comparison, these compounds are &gt;50-times more potent than SD-36 in inducing STAT3 degradation in cells and demonstrates &gt;500-fold degradation sel

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8

Zhang, Xiaoting, Na Qin, Fenfen Ji, et al. "Abstract 4597: RNA m1A methyltransferase TRMT61A induces MAPK/ERK signaling and is a therapeutic target in colorectal cancer." Cancer Research 84, no. 6_Supplement (2024): 4597. http://dx.doi.org/10.1158/1538-7445.am2024-4597.

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Abstract The widespread presence of N1-methyladenosine (m1A) modifications controls RNA metabolism and is pivotal to fundamental biological processes. However, the understanding of RNA m1A involved in cancer is still minimal. Here we reported the oncogenic role and the therapeutic targeting of RNA m1A methyltransferase TRMT61A in colorectal cancer (CRC). We observed consistent elevation of TRMT61A expression and RNA m1A levels in primary CRC tissues, which was significantly associated with poor patient survival in two independent cohorts (both P&lt;.001). CRISPR/Cas9 screenings revealed th

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9

Gaudio, Rudolf P. "John Haiman, Talk is cheap: Sarcasm, alienation, and the evolution of language. Oxford & New York: Oxford University Press, 1998. Pp. ix, 220. Hb $40.00, pb $18.95." Language in Society 29, no. 1 (2000): 117–20. http://dx.doi.org/10.1017/s0047404500211032.

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In this “idiosyncratic personal essay,” Haiman applies his formidable erudition and powers of social observation to questions that North American linguists in general are, unfortunately, content to ignore: the evolutionary origins and historical development of metalanguage, i.e. the property of language that allows us to say “that which is not,” including something other than what we “really” mean. The interdisciplinary nature of this project – engaging evolutionary biology, anthropology, and psychology, in addition to virtually all the traditional subfields of formal/theoretical linguistics –

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10

Xiao, Haihua, Huangyu Jiang, Kunxian Feng, et al. "Abstract 5345: The application of CDK16 inhibitors in the treatment of triple-negative breast cancer." Cancer Research 83, no. 7_Supplement (2023): 5345. http://dx.doi.org/10.1158/1538-7445.am2023-5345.

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Abstract Triple-negative breast cancer (TNBC) with estrogen receptor (ER), progesterone receptor (PR) and HER2 (c-erbB-2, neu) being negatively expressed in patient cancer tissues displayed its unique clinical pathological difference from other types of breast cancers. TNBC is more common in younger patients and more likely to have a family history of breast cancer. TNBCs more likely have poor prognosis and postsurgical recurrence, with strong invasiveness and metastasis. However, there is lack of effective and TNBC-targeting drugs. The non-typical CDK16 was found highly expressed in TNBCs and

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