Academic literature on the topic 'Amphibian metamorphosis'

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Journal articles on the topic "Amphibian metamorphosis"

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Brown, Donald D., and Liquan Cai. "Amphibian metamorphosis." Developmental Biology 306, no. 1 (June 2007): 20–33. http://dx.doi.org/10.1016/j.ydbio.2007.03.021.

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Kuzmin, Sergius L. "Feeding of amphibians during metamorphosis." Amphibia-Reptilia 18, no. 2 (1997): 121–31. http://dx.doi.org/10.1163/156853897x00017.

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AbstractThe feeding ecology of 28 amphibian species with complete life cycles has been studied from the last pre-metamorphic stages to metamorphosed juveniles. The widespread view that feeding ceases completely during metamorphosis is not confirmed. Generally, however, amphibian feeding rate decreases at metamorphosis. Foraging in Caudata either does not cease (Hynobiidae, rheophilous Salamandridae) or ceases only before the end of transformation, which takes less than one metamorphic stage. The cessation of foraging in Anura coincides with the transformation of the mouth and digestive tract at the beginning of the metamorphic climax. Foraging on small animals starts just after the change from a larval to a post-metamorphic mouth, i.e., before the end of metamorphosis.
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Spaul, E. A. "Iodine and Amphibian Metamorphosis." Proceedings of the Zoological Society of London 95, no. 3 (August 21, 2009): 995–1006. http://dx.doi.org/10.1111/j.1469-7998.1925.tb07113.x.

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Mathiron, Anthony G. E., Jean-Paul Lena, Sarah Baouch, and Mathieu Denoël. "The ‘male escape hypothesis’: sex-biased metamorphosis in response to climatic drivers in a facultatively paedomorphic amphibian." Proceedings of the Royal Society B: Biological Sciences 284, no. 1853 (April 19, 2017): 20170176. http://dx.doi.org/10.1098/rspb.2017.0176.

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Paedomorphosis is a major evolutionary process that bypasses metamorphosis and allows reproduction in larvae. In newts and salamanders, it can be facultative with paedomorphs retaining gills and metamorphs dispersing. The evolution of these developmental processes is thought to have been driven by the costs and benefits of inhabiting aquatic versus terrestrial habitats. In this context, we aimed at testing the hypothesis that climatic drivers affect phenotypic transition and the difference across sexes because sex-ratio is biased in natural populations. Through a replicated laboratory experiment, we showed that paedomorphic palmate newts ( Lissotriton helveticus ) metamorphosed at a higher frequency when water availability decreased and metamorphosed earlier when temperature increased in these conditions. All responses were sex-biased, and males were more prone to change phenotype than females. Our work shows how climatic variables can affect facultative paedomorphosis and support theoretical models predicting life on land instead of in water. Moreover, because males metamorphose and leave water more often and earlier than females, these results, for the first time, give an experimental explanation for the rarity of male paedomorphosis (the ‘male escape hypothesis’) and suggest the importance of sex in the evolution of paedomorphosis versus metamorphosis.
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Newman, Robert A. "Adaptive Plasticity in Amphibian Metamorphosis." BioScience 42, no. 9 (October 1992): 671–78. http://dx.doi.org/10.2307/1312173.

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HAYES, TYRONE B. "Amphibian Metamorphosis: An Integrative Approach." American Zoologist 37, no. 2 (April 1997): 121–23. http://dx.doi.org/10.1093/icb/37.2.121.

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Hoskins, Sally G. "Metamorphosis of the amphibian eye." Journal of Neurobiology 21, no. 7 (October 1990): 970–89. http://dx.doi.org/10.1002/neu.480210704.

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DENT, JAMES NORMAN. "Hormonal Interaction in Amphibian Metamorphosis." American Zoologist 28, no. 2 (May 1988): 297–308. http://dx.doi.org/10.1093/icb/28.2.297.

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Reiss, John O. "The phylogeny of amphibian metamorphosis." Zoology 105, no. 2 (January 2002): 85–96. http://dx.doi.org/10.1078/0944-2006-00059.

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Shi, Yun-Bo. "Molecular biology of amphibian metamorphosis." Trends in Endocrinology & Metabolism 5, no. 1 (January 1994): 14–20. http://dx.doi.org/10.1016/1043-2760(94)90116-3.

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Dissertations / Theses on the topic "Amphibian metamorphosis"

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Shewade, Leena H. "Role of Glucocorticoid Signaling in Regulation of Amphibian Metamorphosis." University of Cincinnati / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1535466761073155.

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Ruthsatz, Katharina [Verfasser], and Kathrin H. [Akademischer Betreuer] Dausmann. "Amphibians in a changing world : an ecophysiological perspective on amphibian metamorphosis / Katharina Ruthsatz ; Betreuer: Kathrin H. Dausmann." Hamburg : Staats- und Universitätsbibliothek Hamburg, 2019. http://d-nb.info/1176702076/34.

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Ruthsatz, Katharina Verfasser], and Kathrin H. [Akademischer Betreuer] [Dausmann. "Amphibians in a changing world : an ecophysiological perspective on amphibian metamorphosis / Katharina Ruthsatz ; Betreuer: Kathrin H. Dausmann." Hamburg : Staats- und Universitätsbibliothek Hamburg, 2019. http://nbn-resolving.de/urn:nbn:de:gbv:18-95339.

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James, Stacy M. "Amphibian metamorphosis and juvenile terrestrial performance following chronic cadmium exposure in the aquatic environment." Diss., Columbia, Mo. : University of Missouri-Columbia, 2005. http://hdl.handle.net/10355/4140-D1763/.

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Thesis (Ph. D.)--University of Missouri-Columbia, 2005.
The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Title from title screen of research.pdf file viewed on (May 24, 2006) Vita. Includes bibliographical references.
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Page, Robert Bryce. "TRANSCRIPTIONAL AND MORPHOLOGICAL CHANGES DURING THYROXINE-INDUCED METAMORPHOSIS OF THE MEXICAN AXOLOTL AND AXOLOTL-TIGER SALAMANDER HYBRIDS." UKnowledge, 2009. http://uknowledge.uky.edu/gradschool_diss/774.

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For nearly a century, amphibian metamorphosis has served as an important model of how thyroid hormones regulate vertebrate development. Consequently metamorphosis has been studied in a number of ways including: morphologically, developmentally, ecologically, and from an endocrine perspective. Over the last two decades, much has been learned about the molecular basis of anuran (frog) metamorphosis. However, very little is known about the molecular underpinnings of urodele (salamander) metamorphosis. Using the axolotl and axolotl hybrids as models, I present some of the first studies on the gene expression changes that occur during urodele metamorphosis. In Chapter 1, the motivation for the research described in the subsequent chapters is presented and the literature is briefly reviewed. In Chapter 2, the first microarray analysis of urodele metamorphosis is presented. This analysis shows that hundreds of genes are differentially expressed during thyroid hormone-induced metamorphic skin remodeling. Chapter 3 extends the analysis presented in Chapter 2 by showing that the transcriptional patterns associated with metamorphic skin remodeling are robust even when the concentration of thyroid hormone used to induce metamorphosis is varied by an order of magnitude. Chapter 4 makes use of the differentially expressed genes identified in Chapters 2 and 3 to articulate the first model of urodele metamorphosis to integrate changes in morphology, gene expression, and histology. In addition, Chapter 4 outlines a novel application for piecewise linear regression. In turn, Chapter 5 makes use of the model presented in Chapter 4 to demonstrate that full siblings segregating profound variation in metamorphic timing begin to diverge in phenotype early during larval development. In Chapter 6 the conclusions drawn from the research are summarized and future directions are suggested.
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Duarte, Guterman Paula. "Cross-Talk Between Estrogen and Thyroid Hormones During Amphibian Development." Thèse, Université d'Ottawa / University of Ottawa, 2011. http://hdl.handle.net/10393/19967.

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It is generally thought that in amphibians, thyroid hormones (THs) regulate metamorphosis, while sex steroids (estrogens and androgens) regulate gonadal differentiation. However, inhibition of TH synthesis in frogs alters gonadal differentiation, suggesting instead that these two endocrine axes interact during development. Specifically, THs may be involved in male development, while estrogens may inhibit tadpole metamorphosis. However, we do not currently know the mechanisms that account for these interactions, let alone how such mechanisms may differ between species. To develop and test new hypotheses on the roles of sex steroids and THs, I first examined transcriptional profiles (mRNA) of enzymes and receptors related to sex steroids and THs during embryogenesis and metamorphosis in Silurana tropicalis. Tadpoles were exposed to either an estrogen synthesis inhibitor (fadrozole) or TH (triiodothyronine, T3) during early larval or tadpole development. Acute exposures of S. tropicalis to fadrozole or T3 during early development resulted in increased expression of androgen- and TH-related genes in whole body larvae, while chronic exposure to fadrozole during metamorphosis affected gonadal differentiation but did not affect tadpole development. On the other hand, acute exposure to T3 during metamorphosis increased the expression of androgen-related transcripts both in the brain and gonad. In S. tropicalis, the results suggested that cross-talk is primarily in one direction (i.e., effect of THs on the reproductive axis) with a strong relationship between TH and androgen status. Lastly, I established developmental transcript profiles and investigated T3 regulation of brain and gonad transcripts in Engystomops pustulosus. I then compared these results with S. tropicalis and an earlier study in Lithobates pipiens. While each species developed with similar profiles, they differed in their response to T3. Exposure to T3 resulted in either an increase in androgen-related genes (S. tropicalis) or a decrease in estrogen-related genes (E. pustulosus and L. pipiens). In conclusion, these data demonstrated that cross-talk mechanisms differ among these three evolutionary separate species, but in all cases, T3 appears to affect the balance of sex steroids, stimulating the androgen system and providing potential mechanisms of the masculinising effects of THs. These results will contribute to understanding the mechanisms of hormone interactions and their evolutionary basis in frogs.
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Puglis, Holly J. "Effects of Terrestrial Buffer Zones on Amphibians in Managed Green Spaces." Miami University / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=miami1280773926.

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King, Marie Kumsher. "Evaluation of the Developmental Effects and Bioaccumulation Potential of Triclosan and Triclocarban Using the South African Clawed Frog, Xenopus Laevis." Thesis, University of North Texas, 2010. https://digital.library.unt.edu/ark:/67531/metadc33178/.

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Triclosan (TCS) and triclocarban (TCC) are antimicrobials found in U.S. surface waters. This dissertation assessed the effects of TCS and TCC on early development and investigated their potential to bioaccumulate using Xenopus laevis as a model. The effects of TCS on metamorphosis were also investigated. For 0-week tadpoles, LC50 values for TCS and TCC were 0.87 mg/L and 4.22 mg/L, respectively, and both compounds caused a significant stunting of growth. For 4-week tadpoles, the LC50 values for TCS and TCC were 0.22 mg/L and 0.066 mg/L; and for 8-week tadpoles, the LC50 values were 0.46 mg/L and 0.13 mg/L. Both compounds accumulated in Xenopus. For TCS, wet weight bioaccumulation factors (BAFs) for 0-, 4- and 8-week old tadpoles were 23.6x, 1350x and 143x, respectively. Lipid weight BAFs were 83.5x, 19792x and 8548x. For TCC, wet weight BAFs for 0-, 4- and 8-week old tadpoles were 23.4x, 1156x and 1310x. Lipid weight BAFs were 101x, 8639x and 20942x. For the time-to-metamorphosis study, TCS showed an increase in weight and snout-vent length in all treatments. Exposed tadpoles metamorphosed approximately 10 days sooner than control tadpoles. For the hind limb study, although there was no difference in weight, snout-vent length, or hind limb length, the highest treatment was more developed compared to the control. There were no differences in tail resorption rates between the treatments and controls. At relevant concentrations, neither TCS nor TCC were lethal to Xenopus prior to metamorphosis. Exposure to relatively high doses of both compounds resulted in stunted growth, which would most likely not be evident at lower concentrations. TCS and TCC accumulated in Xenopus, indicating that the compound has the potential to bioaccumulate through trophic levels. Although TCS may increase the rate of metamorphosis in terms of developmental stage, it did not disrupt thyroid function and metamorphosis in regards to limb development and tail resorption.
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Walsh, Patrick Thomas. "The plasticity of life histories during larval development and metamorphosis, using amphibians as study organisms." Thesis, University of Glasgow, 2008. http://theses.gla.ac.uk/183/.

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The ability of animals to vary growth, development rate and behaviour in response to environmental conditions has been well documented, particularly during the larval phase in animals with complex life cycles. The evolution and maintenance of plasticity in response to environmental conditions is likely to be adaptive in animals that face unpredictable environments. However, there are two aspects of life histories in animals with complex life cycles, which would be expected to favour plasticity, that have received limited attention: traits during metamorphic climax and variation in the life history phase at which temperate species spend the winter. Therefore the aims of this thesis were to consider the environmental factors that are likely to result in plasticity in the timing and duration of metamorphic climax and contribute to variation in the over-wintering life stage, using amphibians as study animals. To assess the ability of animals to respond to environmental conditions during metamorphic climax conditions were manipulated during metamorphosis independent of larval treatment. Accordingly all larvae entered metamorphic climax having experienced the same conditions. The African clawed toad, Xenopus laevis, was used. I examined the influence of environmental temperature, predation risk and starting body size on several traits during the transitional stage (e.g. mass, snout-vent length (SVL), head width, tail morphology, duration and locomotor performance). Morphological measures and the duration of the life stage were shown to vary with temperature and predation risk. As predicted, higher temperatures and the risk of predation resulted in faster development through metamorphosis and smaller sizes on completion. The acceleration of metamorphosis was demonstrated to have potential costs, not in the form of reduced locomotor performance as predicted, but in a reduction in juvenile size as a result of faster metamorphic development. This suggests that, during this potentially vulnerable stage, it would be advantageous to take more time to complete in the absence of predators. Greater body size at the onset of metamorphosis requires a longer time to complete metamorphic climax suggesting that having a greater quantity of tissue to reconfigure during metamorphosis takes more time. Therefore, the conditions experienced during metamorphosis may have important implications for juvenile fitness and should be considered in studies of life history plasticity. In many temperate species with complex life cycles, the life history stage at which a species can survive the winter is generally fixed, imposing time limits on the timing of development. Most of these species must therefore often modify developmental rate to reach the appropriate stage or size at the onset of winter, usually at a cost to other traits. However, variation in the stage or developmental group that some amphibian, fish and insect species spend the winter has been observed, such as in the common frog Rana temporaria in the UK, which can spend the first winter as either a tadpole or as a juvenile frog. To investigate the factors that contribute to this variation in life history, I examined the influence of environmental temperature, food availability and water depth on the rate of larval development and growth. Data on development, growth and environmental temperature of a field population of R. temporaria, which have been observed to over-winter as larvae, were collected to determine how and when the two divergent early life history patterns of development were established. Development rate was slowed by reduced temperatures and food availability and greater water depth during rearing. Temperature and food availability also had a significant impact on the proportion of larvae that over-wintered, but in the field other factors are likely to contribute to the within-population variation in wintering strategy. While a greater water depth did prolong larval development, as predicted, this does not appear to be due to the cost of surfacing to respire acting as a constraint on development, since a similar slowing in development was observed in the lung-less Bufo bufo tadpoles. The results of these studies did not allow a definitive assessment of whether over-wintering as larvae represents an adaptive strategy or occurs as the result of developmental constraints. There is some evidence that over-wintering as larvae might be adaptive, since on completion of metamorphosis individuals that wintered as larvae were larger than those that completed metamorphosis late in the summer. Further work is necessary to identify other factors contributing to the over-wintering of larvae in Rana temporaria and to determine the adaptive significance, if any, of the alternative life history patterns.
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Purrenhage, Jennifer Lyn. "Importance of Habitat Structure for Pond-Breeding Amphibians in Multiple Life Stages." Miami University / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=miami1240957514.

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Books on the topic "Amphibian metamorphosis"

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S, Owen Oliver. Tadpole to frog. Edina, Minn: Abdo & Daughters, 1994.

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Wendy, Pfeffer. From tadpole to frog. New York: HarperCollins, 1994.

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ill, Kubo Hidekazu, ed. The tadpole. Milwaukee: Raintree, 1986.

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Back, Christine. Tadpole and frog. Morristown, N.J: Silver Burdett, 1986.

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undifferentiated, Harold Fox. Amphibian Morphogenesis. Springer, 2011.

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Amphibian Morphogenesis. Humana Press, 2011.

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Test No. 231: Amphibian Metamorphosis Assay. OECD, 2009. http://dx.doi.org/10.1787/9789264076242-en.

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Amphibian Metamorphosis: From Morphology to Molecular Biology. Wiley-Liss, 1999.

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1929-, Gilbert Lawrence I., Tata Jamshed R, and Atkinson Burr G, eds. Metamorphosis: Postembryonic reprogramming of gene expression in amphibian and insect cells. San Diego: Academic Press, 1996.

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Lawrence I. Gilbert (Series Editor), Jamshed R. Tata (Series Editor), and Burr G. Atkinson (Series Editor), eds. Metamorphosis: Postembryonic Reprogramming of Gene Expression in Amphibian and Insect Cells (Cell Biology). Academic Press, 1996.

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Book chapters on the topic "Amphibian metamorphosis"

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Amano, Tosikazu, Liezhen Fu, Atsuko Ishizuya-Oka, and Yun-Bo Shi. "Thyroid Hormone-Induced Apoptosis during Amphibian Metamorphosis." In Molecular Mechanisms of Programmed Cell Death, 9–19. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4757-5890-0_2.

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Tata, Jamshed R. "How Hormones Regulate Programmed Cell Death during Amphibian Metamorphosis." In Programmed Cell Death, 1–11. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-0072-2_1.

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Ishizuya-Oka, Atsuko. "Regulation of Adult Intestinal Stem Cells through Thyroid Hormone-Induced Tissue Interactions during Amphibian Metamorphosis." In Cell and Molecular Biology and Imaging of Stem Cells, 153–72. Hoboken, New Jersey: John Wiley & Sons, Inc, 2014. http://dx.doi.org/10.1002/9781118285602.ch6.

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Endler, P. C., C. Heckmann, E. Lauppert, W. Pongratz, J. Alex, D. Dieterle, C. Lukitsch, et al. "The Metamorphosis of Amphibians and Information of Thyroxin Storage Via the Bipolar Fluid Water and on a Technical Data Carrier; Transference Via an Electronic Amplifier." In Fundamental Research in Ultra High Dilution and Homoeopathy, 155–87. Dordrecht: Springer Netherlands, 1998. http://dx.doi.org/10.1007/978-94-011-5878-7_11.

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KALTENBACH, JANE C. "Endocrinology of Amphibian Metamorphosis." In Metamorphosis, 403–31. Elsevier, 1996. http://dx.doi.org/10.1016/b978-012283245-1/50013-0.

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DENVER, ROBERT J. "Neuroendocrine Control of Amphibian Metamorphosis." In Metamorphosis, 433–64. Elsevier, 1996. http://dx.doi.org/10.1016/b978-012283245-1/50014-2.

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ATKINSON, BURR G., CAREN HELBING, and YUQING CHEN. "Reprogramming of Genes Expressed in Amphibian Liver during Metamorphosis." In Metamorphosis, 539–66. Elsevier, 1996. http://dx.doi.org/10.1016/b978-012283245-1/50017-8.

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YOSHIZATO, KATSUTOSHI. "Cell Death and Histolysis in Amphibian Tail during Metamorphosis." In Metamorphosis, 647–71. Elsevier, 1996. http://dx.doi.org/10.1016/b978-012283245-1/50021-x.

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Denver, Robert J. "Neuroendocrinology of Amphibian Metamorphosis." In Current Topics in Developmental Biology, 195–227. Elsevier, 2013. http://dx.doi.org/10.1016/b978-0-12-385979-2.00007-1.

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TATA, JAMSHED R. "Hormonal Interplay and Thyroid Hormone Receptor Expression during Amphibian Metamorphosis." In Metamorphosis, 465–503. Elsevier, 1996. http://dx.doi.org/10.1016/b978-012283245-1/50015-4.

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