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1

N, Wickramasinghe S., ed. Megaloblastic anaemia. Baillière Tindall, 1995.

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2

C, Gordon-Smith E., ed. Aplastic anaemia. Baillie re, 1989.

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3

Campbell, Ken. Acquired aplastic anaemia. NT Books/Emap Healthcare, 2000.

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4

Executive, NHS Management. Sickle cell anaemia. Health Publications Unit, 1994.

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5

Planque, Maria M. de. Severe aplastic anaemia. Rijksuniv., 1988.

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6

Santos, Claudia Chimisso Dos. Molecular analysis of Fanconi anaemia. National Library of Canada, 1993.

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7

Marsh, Judith Christine Watson. Characterisation of the haemopoietic defect in aplastic anaemia. University of Birmingham, 1990.

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8

Davies, Jill. Anaemia: Over 100 recipes for overcoming iron-deficiency. Thorsons, 1995.

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9

Akinola, Norah Olubunmi. Rheology of vascular occlusion in sickle cell anaemia. University of Birmingham, 1992.

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10

Lewis, S. M. Bench aids for the morphological diagnosis of anaemia. World Health Organization, 2001.

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11

Silverberg, Donald S. (1937- ). Autor, ed. Cardio-renal anaemia syndrome: Basic and clinical aspects. Publicationes Latinoamericanas SRL, 2008.

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12

A, Schad G., and Stott G. J, eds. Hookworm infection and anaemia: Approaches to prevention and control. World Health Organization, 1991.

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13

Boehringer Mannheim UK (Pharmaceuticals) Ltd., ed. Recormon: Epoetin beta : flexible erythropoietin replacement in renal anaemia. Boehringer Mannheim, 1994.

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14

Morrow, Dympna. Is tumour induced anaemia caused by tumour derived cytokines?. The Author], 1995.

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15

S, Mharakurwa. Malaria and anaemia in pregnancy in Gokwe communities, Zimbabwe. Blair Research Institute, Ministry of Health, 1991.

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16

Reddy, M. Ramakrishna. Prevalence of iron deficiency anaemia and malnuitrition in India. Institute for Social and Economic Change, 2004.

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17

Boehringer Mannheim UK (Pharmaceuticals) Ltd., ed. Recormon: Epoetin beta : flexible erythropoietin replacement in renal anaemia. Boehringer Mannheim, 1994.

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18

Dyson, Simon. Sickle-cell anaemia: Current carrier and community awareness in Leicestershire. De Montfort University, 1994.

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19

Mawhinney, Karen Ann. Partial characterization of chicken anaemia virus protein expressed by recombinant baculovirus. The Author], 1994.

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20

Dyson, Simon Martin. Community and professional knowledge of sickle cell anaemia and beta-thalassaemia. De Montfort University, 1997.

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21

Leicestershire Organisation for Sickle-cell Anaemia Research., Leicestershire Thalassaemia Society, and Leicestershire (England). Health Education Department., eds. Sickle-cell anaemia and thalassaemia: Report of the first community conference organised by Leicestershire Organisationfor Sickle-Cell Anaemia Research, Leicestershire Thalassaemia Society & Leicestershire Health Promotion Department. 2nd ed. Leicestershire Health Promotion Department, 1989.

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22

Hyder, S. M. Ziauddin. Prevalence of anaemia and intestinal parasites in a rural community of Bangladesh. Research and Evaluation Division, BRAC, 1998.

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23

1957-, Hadley Andrew, and Soothill Peter 1957-, eds. Alloimmune disorders of pregnancy: Anaemia, thrombocytopenia, and neutropenia in the fetus and newborn. Cambridge University Press, 2002.

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24

Monitoring the Situation of Children and Women Project (Bangladesh) and UNICEF, eds. Report on Anaemia prevalence survey of urban Bangladesh and rural Chittagong Hill Tracts 2003. Monitoring the Situation of Children and Women Project, Bangladesh Bureau of Statistics with assistant from UNICEF, 2004.

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25

World Health Organization. Regional Office for South-East Asia. Prevention of iron deficiency anaemia in adolescents: Role of weekly iron and folic acid supplementation. World Health Organization, Regional Office for South-East Asia, 2011.

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26

Campbell, Ken. Hodgkin's disease: Hodgkin's disease & other lymphomas myeloma, myelodysplasia, aplastic anaemia, the myeloproiferative disorders, the leukaemias. Leukaemia research Foundation, 2001.

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27

Bunch, Chris. Anaemia. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0037.

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Anaemia denotes a reduction in the circulating haemoglobin level or red-cell count below that which is normal for the individual’s age and sex. Anaemia is common and may be a primary problem or a feature of a wide variety of other conditions. This chapter covers the approach to diagnosis, diagnostic tests, therapies, prognosis, and dealing with uncertainty in the initial diagnosis.
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28

Bunch, Chris. Haemolytic anaemia. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0280.

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Haemolytic anaemias occur when the rate of red-cell breakdown is increased and exceeds the marrow’s capacity to generate new cells. Increased red-cell destruction, or haemolysis, may reflect a broad range of disorders. Some involve intrinsic defects in the red cell itself; in others, the red cells are normal but are subjected to external factors which lead to premature destruction. Many of the intrinsic defects are due to inherited disorders affecting the red-cell membrane, its enzymes, or haemoglobin. The marrow can normally compensate for moderate haemolysis by increasing red-cell production
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29

Gordon-Smith. Aplastic Anaemia. Elsevier, 1988.

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30

Pernicious Anaemia. Hammersmith Health Books, 2012.

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31

Trivedi, SS, and Manju Puri. Anaemia in Pregnancy. Jaypee Brothers Medical Publishers (P) Ltd., 2008. http://dx.doi.org/10.5005/jp/books/10044.

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32

Bassand, Jean-Pierre, François Schiele, and Nicolas Meneveau. Anaemia and transfusion. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0071.

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Anaemia, irrespective of the cause-whether linked to, or worsened by, bleeding or phlebotomy, has an unfavourable impact on outcome, in terms of death and myocardial infarction, in acute coronary syndromes. In addition, it is an independent predictor of the risk of bleeding. The treatment of anaemia includes a search for the cause and its mechanism, blood transfusion, and iron therapy. Erythropoietin-stimulating agents are contraindicated. Blood transfusion should be considered with caution. It is indicated in cases of haemodynamic or ischaemic instability. However, in stable patients, blood t
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33

Anaemia in Cancer. Elsevier Science Pub Co, 2001.

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34

Acquired Aplastic Anaemia. Nursing Times Books, Emap Healthcare, 2000.

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35

Okeke, Peter Ubah. Anaemia in Pregnancy. Lulu Press, Inc., 2013.

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36

Tom, Smith. Coping with anaemia. Sheldon Press, 2012.

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37

World Health Organization (WHO). Hookworm Infection and Anaemia. Stationery Office Books, 1991.

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38

Provan, Drew, Trevor Baglin, Inderjeet Dokal, and Johannes de Vos. Red cell disorders. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199683307.003.0002.

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The peripheral blood film in anaemias - Anaemia in renal disease - Anaemia in endocrine disease - Anaemia in joint disease - Anaemia in gastrointestinal disease - Anaemia in liver disease - Iron (Fe) deficiency anaemia - Vitamin B12 deficiency - Folate deficiency - Other causes of megaloblastic anaemia - Anaemia in other deficiency states - Haemolytic syndromes - Genetic control of haemoglobin production - Sickling disorders - HbS—sickle-modifying therapies - Sickle cell trait (HbAS) - Other sickling disorders - Other haemoglobinopathies - Unstable haemoglobins - Thalassaemias - α thalassaemia
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39

Provan, Drew, Trevor Baglin, Inderjeet Dokal, Johannes de Vos, Banu Kaya, and Angela Theodoulou. Red cell disorders. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199683307.003.0002_update_001.

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The peripheral blood film in anaemias - Anaemia in renal disease - Anaemia in endocrine disease - Anaemia in joint disease - Anaemia in gastrointestinal disease - Anaemia in liver disease - Iron (Fe) deficiency anaemia - Vitamin B12 deficiency - Folate deficiency - Other causes of megaloblastic anaemia - Anaemia in other deficiency states - Haemolytic syndromes - Genetic control of haemoglobin production - Sickling disorders - HbS—sickle-modifying therapies - Sickle cell trait (HbAS) - Other sickling disorders - Other haemoglobinopathies - Unstable haemoglobins - Thalassaemias - α‎ thalassaemi
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40

Macdougall, Iain C. Iron management in renal anaemia. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0126.

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Although erythropoiesis-stimulating agent therapy is the mainstay of renal anaemia management, maintenance of an adequate iron supply to the bone marrow is also pivotal in the process of erythropoiesis. Thus, it is important to be able to detect iron insufficiency, and to treat this appropriately. Iron deficiency may be absolute (when the total body iron stores are exhausted) or functional (when the total body iron stores are normal or increased, but there is an inability to release iron from the stores rapidly enough to provide a ready supply of iron to the bone marrow). Several markers of ir
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41

How to Cope with Anaemia. Sheldon Press, 1998.

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42

Macdougall, Iain C. Pocket Reference to Renal Anaemia. Science Press Inc., 2003.

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43

William, Gardner. Case of Progressive Pernicious Anaemia. Franklin Classics, 2018.

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44

Wickramasinghe, S. N. Megaloblastic Anaemia (Bailliere's Clinical Haematology). Elsevier, 1995.

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45

City and Hackney Community Health Council., ed. Sickle cell anaemia and thalassaemia. City and Hackney Community Health Council, 1988.

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46

Prashar, Usha, Elizabeth N. Anionwu, and M. Brozovic. Sickle Cell Anaemia: Who Cares? Runnymede Trust, 1985.

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47

Case of Progressive Pernicious Anaemia. Franklin Classics, 2018.

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48

JOHNSON, Thomas. Cbd Oil for Sickle Cell Anaemia: The Natural Therapeutic Aid for Sickle Cell Anaemia. Independently Published, 2019.

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49

Haymann, Jean-Philippe, and Francois Lionnet. The patient with sickle cell anaemia. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0167.

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In sickle cell anaemia (SCA) a single mutation in the haemoglobin beta-globin gene is responsible for a pleomorphic phenotype leading to acute and chronic life-threatening complications. Healthcare management programmes, patient and family education, infection prophylaxis (especially in childhood), and long-term treatment for some patients (such as hydroxyurea) have significantly improved survival, giving rise to some new long-term issues.Sickle cell-associated nephropathy (SCAN) leads in some cases to chronic renal failure with a significant impact on survival. SCAN is characterized by an inc
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50

Breymann, Christian. Anaemia in pregnancy and the Puerperium. UNI-MED Verlag AG, 2006.

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