Relevant bibliographies by topics / ANCA glomerulonephritis vasculitis

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  2. Dissertations / Theses
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Academic literature on the topic 'ANCA glomerulonephritis vasculitis'

Author: Grafiati
Published: 3 June 2025
Last updated: 1 August 2025

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Journal articles on the topic "ANCA glomerulonephritis vasculitis"

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Radwan, Yasser, Sarah Berini, Floranne Ernste, and Ashima Makol. "Proteinase 3 (PR3)-antineutrophil cytoplasmic antibody (ANCA)-associated vasculitic neuropathy in diffuse cutaneous systemic sclerosis: a rare duo." BMJ Case Reports 12, no. 11 (2019): e232987. http://dx.doi.org/10.1136/bcr-2019-232987.

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Systemic sclerosis (SSc) is characterised by non-inflammatory vasculopathy, autoimmunity and widespread fibrosis. While the presence of antineutrophil cytoplasmic antibodies (ANCAs) has been reported in SSc, their association with ANCA-associated vasculitis is exceedingly rare. Myeloperoxidase ANCA is more common than proteinase-3 ANCA, and glomerulonephritis is the most common clinical presentation of ANCA-associated vasculitis in SSc. ANCAs have been associated with the adverse disease outcomes in SSc, including higher mortality per recent reports. A 65-year-old man with diffuse cutaneous SS
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Savenkova, N. D. "Treatment strategy ANCA-associated renal vasculitides in children and adolescents." Nephrology (Saint-Petersburg) 23, no. 5 (2019): 107–15. http://dx.doi.org/10.24884/1561-6274-2019-23-5-107-115.

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The article presents the terminology and classification in accordance with the International Сhapel Hill Сonsensus Сonference nomenclature of vasculitides (2012), clinical, immunological and morphological manifestations, therapy strategy and outcome of Antineutrophil cytoplasmic antibody vasculitides (ANCA) renal associated vasculitis (microscopic polyanghiitis, granulomatosis with Wegener's polyangiitis, eosinophilic granulomatosis with polyangiitis Churg-Strauss) in children and adolescents. IgG class antibodies to MPO and PR3, histopathological changes in renal biopsy specimens are consider
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Jennette, J. Charles, and Patrick H. Nachman. "ANCA Glomerulonephritis and Vasculitis." Clinical Journal of the American Society of Nephrology 12, no. 10 (2017): 1680–91. http://dx.doi.org/10.2215/cjn.02500317.

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Trimeche Ajmi, Sihem, Rim Braham, Sarra Toumi, et al. "Benzylthiouracil-Induced Glomerulonephritis." Case Reports in Medicine 2009 (2009): 1–4. http://dx.doi.org/10.1155/2009/687285.

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Vasculitis is a rare complication of antithyroid drugs (ATDs). It was first described with Propylthiouracil (PTU). We report a new case of antineutrophil cytoplasmic antibody (ANCA) vasculitis with glomerulonephritis induced by Benzylthiouracile (BTU). A 50-year-old man with Graves disease treated with BTU developed general malaise and haematuria without skin rash or respiratory involvement. Laboratory data revealed acute renal failure with proteinuria and haematuria. An indirect immunofluorescence test for ANCA was positive, showing a perinuclear pattern with specificity antimyeloperoxidase (
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Drouzas, Konstantinos, Petros Kalogeropoulos, George Liapis, and Sofia Lionaki. "Current Understanding of the Pathogenesis of ANCA-Associated Vasculitis and Novel Treatment Options Targeting Complement Activation." Life 15, no. 5 (2025): 756. https://doi.org/10.3390/life15050756.

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Anti-neutrophil cytoplasmic antibody (ANCA)-associated vasculitis (ANCA-vasculitis) is an autoimmune disease characterized by inflammation and necrosis of small or medium vessels. In the past, the role of the complement in the pathogenesis of ANCA-vasculitis has been underestimated, due to the paucity of the complement at sites of injured glomeruli. Following evidence from animal models of the major role of the complement in pathogenesis of ANCA-vasculitis, the complement has again attracted interest. Immunohistology analysis of pauci-immune glomerulonephritis—ANCA glomerulonephritis (ANCA-GN)
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Preto, Clara, Armandina Silva, Sandra Alves, et al. "The Diagnosing Challenge of a Positive ANCA Vasculitis in the Paediatric Age." Case Reports in Pediatrics 2017 (2017): 1–5. http://dx.doi.org/10.1155/2017/2962794.

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ANCA-positive systemic vasculitides, rare in paediatric age, present multiorganic involvement. A female teenager presented with a history of subglottic stenosis diagnosed at the age of 12. From the investigation carried out, we highlight hematoproteinuria and negative ANCAs. At 15 years old, she was admitted for gastrointestinal symptoms and respiratory distress. She presented poor peripheral perfusion, pulmonary haemorrhage, respiratory failure, and severe renal insufficiency. She was started mechanical ventilation and emergency haemodialysis. The immunological study revealed ANCA MPO positiv
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Kao, Lily, and Cornelia Weyand. "Vasculitis in Systemic Sclerosis." International Journal of Rheumatology 2010 (2010): 1–9. http://dx.doi.org/10.1155/2010/385938.

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Systemic sclerosis (SSc) is a multiorgan connective tissue disease characterized by autoantibody production and fibroproliferative stenosis of the microvasculature. The vascoluopathy associated with SSc is considered to be noninflammatory, yet frank vasculitis can complicate SSc, posing diagnostic and therapeutic challenges. Here, we have reviewed the literature for reports of small-, medium-, and large-vessel vasculitis occurring in SSc. Amongst 88 reported cases of vasculitis in SSc, patients with ANCA-associated vasculitis appear to present a unique subclass in that they combined typical fe
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Van Haare Heijmeijer, Sophie, Dunja Wilmes, Selda Aydin, Caroline Clerckx, and Laura Labriola. "Necrotizing ANCA-Positive Glomerulonephritis Secondary to Culture-Negative Endocarditis." Case Reports in Nephrology 2015 (2015): 1–5. http://dx.doi.org/10.1155/2015/649763.

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Infective endocarditis (IE) and small-vessel vasculitis may have similar clinical features, including glomerulonephritis. Furthermore the association between IE and ANCA positivity is well documented, making differential diagnosis between IE- and ANCA-associated vasculitis particularly difficult, especially in case of culture-negative IE. We report on one patient with glomerulonephritis secondary to culture-negative IE caused byBartonella henselaewhich illustrates this diagnostic difficulty.
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Lamprecht, P., W. H. Schmitt, and W. L. Gross. "Mixed cryoglobulinemia, glomerulonephritis, and ANCA: essential cryoglobulinaemic vasculitis or ANCA-associated vasculitis?" Nephrology Dialysis Transplantation 13, no. 1 (1998): 213–21. http://dx.doi.org/10.1093/ndt/13.1.213.

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Endo, Akiko, Junichi Hoshino, Tatsuya Suwabe, et al. "Significance of Small Renal Artery Lesions in Patients with Antineutrophil Cytoplasmic Antibody-associated Glomerulonephritis." Journal of Rheumatology 41, no. 6 (2014): 1140–46. http://dx.doi.org/10.3899/jrheum.130657.

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Objective.Antineutrophil cytoplasmic antibody (ANCA)-associated glomerulonephritis is a vasculitis affecting the glomerular capillaries and small renal arteries. Although crescent formation has been reported to be characteristic of this condition, the significance of coexisting vasculitis affecting the small renal arteries has not been investigated.Methods.Fifty patients with ANCA-positive rapidly progressive glomerulonephritis whose renal biopsy specimens contained arterioles and/or interlobular arteries were retrospectively evaluated. Cellular crescents and/or necrotizing glomerulonephritis
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Dissertations / Theses on the topic "ANCA glomerulonephritis vasculitis"

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Hamour, Sally. "The role of IL-17 in the pathogenesis of glomerulonephritis and ANCA-associated vasculitis." Thesis, Imperial College London, 2013. http://hdl.handle.net/10044/1/14413.

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Immune-mediated glomerulonephritis and vasculitis accounts for over 10% of end-stage renal failure in the UK. The anti-neutrophil cytoplasm antibody(ANCA)-associated vasculitides (AAV) are a specific subset, characterised by the production of ANCA, and are idiopathic multi-system vasculitides affecting mainly small-calibre vessels and renal glomeruli. Extensive human and animal work by our group and others has attempted to dissect the mechanisms which underlie the pathogenesis of these conditions. Recently, an important development in our understanding of cellular immunity has been the identif
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Pepper, R. J. "The role of calprotectin (S100A8/A9) in the pathogenesis of glomerulonephritis and ANCA-associated vasculitis." Thesis, University College London (University of London), 2013. http://discovery.ucl.ac.uk/1400216/.

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Glomerulonephritis is a common cause of end-stage renal failure and a feature of ANCA-associated vasculitis (AAV). AAV is an example of a small vessel vasculitis, characterised by inflammation of the endothelium and glomeruli in which the interaction between leukocytes and endothelial cells play a crucial role. Macrophages have been demonstrated to have a critical role during the initiation and progression of glomerulonephritis. Calprotectin (also termed S100A8/A9, mrp8/14), is a complex of 2 small calcium binding proteins that is abundantly expressed in neutrophils and monocytes as well as ea
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Delbet, Jean-Daniel. "Étude de preuve de concept thérapeutique évaluant l'efficacité d'un anticorps monoclonal dans le traitement des glomérulonéphrites à croissant ciblant CLDN1 dans les cellules épithéliales pariétales glomérulaires." Electronic Thesis or Diss., Université Paris Cité, 2024. http://www.theses.fr/2024UNIP5227.

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Introduction : La glomérulonéphrite à croissant (cGN) est une forme avancée de glomérulopathie commune à plusieurs maladies rénales d'origine immunitaire. La cGN se caractérise par une prolifération des cellules épithéliales pariétales glomérulaires (PEC), formant des croissants dans la chambre urinaire et conduisant en l'absence de traitement à l'insuffisance rénale terminale. Dans le cadre de ma thèse, j'ai particulièrement étudié la claudine-1 (CLDN1), une protéine transmembranaire impliquée dans les jonctions serrées, qui peut être exposée en dehors de ces jonctions serrées et activer des
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Andrade, Andreia Cristina Cruz. "Glomerulonefrite associada a ANCA - papel do Rituximab." Master's thesis, 2021. http://hdl.handle.net/10316/98452.

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Trabalho Final do Mestrado Integrado em Medicina apresentado à Faculdade de Medicina<br>Introdução: Apesar da eficácia global da terapêutica existente para as vasculites associadas a anticorpos anticitoplasma de neutrófilos (ANCA), tem havido uma procura constante no sentido de reduzir o risco dos seus efeitos secundários. O rituximab tem demonstrado eficácia semelhante ou superior à ciclofosfamida, sem aumento do número total de efeitos adversos. Com este estudo pretende-se comparar o tratamento com rituximab e ciclofosfamida nos doentes com glomerulonefrite associada a ANCA. Métodos: Realizá
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Hoffmann, Johanna Charlotte. "Untersuchung von Biomarkern bei Patienten mit Anca-assoziierter Vaskulitis." Doctoral thesis, 2018. http://hdl.handle.net/11858/00-1735-0000-002E-E40F-5.

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Books on the topic "ANCA glomerulonephritis vasculitis"

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Gross, Wolfgang L., and Julia U. Holle. Clinical features of ANCA-associated vasculitis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0131.

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The primary ANCA-associated vasculitides are granulomatosis with polyangiitis (Wegener's, GPA), microscopic polyangiitis (MPA), and eosinophilic granulomatosis with polyangiitis (EGPA, Churg-Strauss syndrome, CSS). They predominantly affect small (and medium-sized) vessels and share a variable association with ANCA (anti-neutrophil cytoplasm antibody) directed against neutrophil proteinase 3 (PR3, mainly in GPA) and myeloperoxidase (MPO, mainly in MPA and CSS). Crescentic necrotizing glomerulonephritis and alveolar haemorrhage due to pulmonary capillaritis represent classical (vasculitic) orga
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Tombetti, Enrico, and Justin C. Mason. Pathophysiology of vasculitis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0017.

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Vasculitis represents a spectrum of disorders that are often divided on the basis of the predominant vessel size affected into large-, medium- and small-vessel vasculitides. This chapter will focus on the pathogenesis of the anti-neutrophil cytoplasmic antibody (ANCA)-associated medium- and small-vessel vasculitides (AAV), and large-vessel vasculitis, Takayasu arteritis, and giant cell arteritis. Underlying pathogenic mechanisms in vasculitis remain to be fully understood. In particular, the initiating event(s) are not known. A combination of infectious or other environmental triggers on a sus
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Rodriguez-Iturbe, Bernardo, and Mark Haas. Glomerulonephritis associated with endocarditis, deep-seated infections, and shunt nephritis. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0079_update_001.

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Endocarditis is a cause of glomerulonephritis. Healthcare interventions (prosthetic valves, indwelling catheters, pacemaker wires) and intravenous drug abuse are presently the most common causes of endocarditis and Staphylococcus aureus is frequently the infecting bacteria. Shunt nephritis is a form of glomerulonephritis associated with infection of ventriculoatrial shunts implanted to relieve hydrocephalus and, typically, are caused by prolonged infections of low-pathogenicity microorganisms. This complication led to the replacement of the technique by ventriculoperitoneal shunts. Deep-seated
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Cui, Zhao, Neil Turner, and Ming-hui Zhao. Antiglomerular basement membrane disease. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0073_update_001.

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Cyclophosphamide and plasma exchange are the standard of care in rapidly progressive glomerulonephritis or lung haemorrhage caused by antiglomerular basement membrane (anti-GBM) disease, and it is unusual to encounter patients at earlier stages. Steroids are universally used in addition. There is some evidence that plasma exchange may not be a critical part of treatment at an earlier stage. There is no more than anecdotal evidence for other therapies. Slower-onset therapies such as antibodies to B cells are rarely appropriate. If untreated, patients with severe anti-GBM disease will not recove
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Book chapters on the topic "ANCA glomerulonephritis vasculitis"

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Nachman, Patrick H., and Shannon L. Murphy. "ANCA-Associated Vasculitis, Adult." In Glomerulonephritis. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-49379-4_17.

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Gibson, Keisha, and Dorey Glenn. "ANCA-Associated Vasculitis, Pediatric." In Glomerulonephritis. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-49379-4_18.

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Nachman, Patrick H., and Shannon L. Murphy. "ANCA-Associated Vasculitis, Adult." In Glomerulonephritis. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-27334-1_17-1.

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Nachman, Patrick H., and Shannon L. Murphy. "ANCA-Associated Vasculitis, Adult." In Glomerulonephritis. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-27334-1_17-2.

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Gibson, Keisha, and Dorey Glenn. "ANCA-Associated Vasculitis, Pediatric." In Glomerulonephritis. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-27334-1_18-1.

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Free, Meghan E., Dominic J. Ciavatta, J. Charles Jennette, and Ronald J. Falk. "The Pathogenesis and Consequences of Myeloperoxidase-Dependent ANCA Vasculitis and Glomerulonephritis." In Mammalian Heme Peroxidases. CRC Press, 2021. http://dx.doi.org/10.1201/9781003212287-20.

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Grotz, Wolfgang, Ekkehard Röther, and Peter Schollmeyer. "C-ANCA and p-ANCA Positive Vasculitis with Crescentic Glomerulonephritis: The Same Clinical Feature but Different Immunological Pathways ?" In Advances in Experimental Medicine and Biology. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4757-9182-2_82.

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Teeraratkul, Phataraporn, John A. Thorson, John D. Kemp, and James A. Goeken. "Indentification of Apparent Dual ANCA Specificities in a Subset of Patients with Systemic Vasculitis and Crescentic Glomerulonephritis." In Advances in Experimental Medicine and Biology. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4757-9182-2_81.

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Kallenberg, Cees G. M., and Jan W. Cohen Tervaert. "Anti-Neutrophil Cytoplasmic Antibodies (ANCA): New Tools in the Diagnosis and Follow-Up of Necrotizing Glomerulonephritis and Vasculitis." In International Yearbook of Nephrology 1992. Springer London, 1991. http://dx.doi.org/10.1007/978-1-4471-1892-3_15.

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Gross, Wolfgang L., and Julia U. Holle. "Clinical features of ANCA-associated vasculitis." In Oxford Textbook of Rheumatology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0131_update_001.

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The primary ANCA-associated vasculitides are granulomatosis with polyangiitis (Wegener’s, GPA), microscopic polyangiitis (MPA), and eosinophilic granulomatosis with polyangiitis (EGPA, Churg-Strauss syndrome, CSS). They predominantly affect small (and medium-sized) vessels and share a variable association with ANCA (anti-neutrophil cytoplasm antibody) directed against neutrophil proteinase 3 (PR3, mainly in GPA) and myeloperoxidase (MPO, mainly in MPA and CSS). Crescentic necrotizing glomerulonephritis and alveolar haemorrhage due to pulmonary capillaritis represent classical (vasculitic) orga
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Conference papers on the topic "ANCA glomerulonephritis vasculitis"

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Souza, Yasmin Nóbrega e., Maria Eduarda Pereira Florênço, Guilherme Oliveira Lobo, et al. "SYSTEMIC SCLEROSIS ASSOCIATED WITH ANCA-POSITIVE VASCULITIS AND RAPIDLY PROGRESSIVE GLOMERULONEPHRITIS: CASE REPO." In XLI Congresso Brasileiro de Reumatologia. Sociedade Brasileira de Reumatologia, 2024. https://doi.org/10.47660/cbr.2024.2668.

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Modi, P., S. Falde, and U. Specks. "Avacopan for Treatment of MPO-ANCA Associated Vasculitis Presenting as Diffuse Alveolar Hemorrhage and Glomerulonephritis." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a5782.

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Inácio, A., I. Pinto, A. Piedade, P. Domingues, B. Mendes, and K. Soto. "AB1266 RISK FACTORS FOR END-STAGE KIDNEY DISEASE AND DEATH IN ANCA-ASSOCIATED VASCULITIS WITH GLOMERULONEPHRITIS." In EULAR 2024 European Congress of Rheumatology, 12-15 June. Vienna, Austria. BMJ Publishing Group Ltd and European League Against Rheumatism, 2024. http://dx.doi.org/10.1136/annrheumdis-2024-eular.4383.

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Merino-Vico, A., Y. Vegting, A. Jongejan, et al. "POS1006 PROFILING B AND PLASMA CELLS IN KIDNEY BIOPSIES FROM ANCA-ASSOCIATED VASCULITIS PATIENTS WITH GLOMERULONEPHRITIS AT SINGLE-CELL RESOLUTION." In EULAR 2024 European Congress of Rheumatology, 12-15 June. Vienna, Austria. BMJ Publishing Group Ltd and European League Against Rheumatism, 2024. http://dx.doi.org/10.1136/annrheumdis-2024-eular.5716.

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