Relevant bibliographies by topics / Ancrod

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Ancrod'

Author: Grafiati
Published: 4 June 2021
Last updated: 6 February 2022

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Journal articles on the topic "Ancrod"

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Cole, C. Wm, and Ianis Bormanis. "ANCROD." Journal of Trauma: Injury, Infection, and Critical Care 35, no. 1 (July 1993): 175. http://dx.doi.org/10.1097/00005373-199307000-00095.

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Dempfle, Carl-Erik, Sotiria Argiriou, Klaus Kucher, H. Müller-Peltzer, Klaus Rübsamen, and Dieter L. Heene. "Analysis of fibrin formation and proteolysis during intravenous administration of ancrod." Blood 96, no. 8 (October 15, 2000): 2793–802. http://dx.doi.org/10.1182/blood.v96.8.2793.

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Abstract Ancrod is a purified fraction of venom from the Malayan pit viper, Calloselasma rhodostoma, currently under investigation for treatment of acute ischemic stroke. Treatment with ancrod leads to fibrinogen depletion. The present study investigated the mechanisms leading to the reduction of plasma fibrinogen concentration. Twelve healthy volunteers received an intravenous infusion of 0.17 U/kg body weight of ancrod for 6 hours. Blood samples were drawn and analyzed before and at various time points until 72 hours after start of infusion. Ancrod releases fibrinopeptide A from fibrinogen, leading to the formation of desAA-fibrin monomer. In addition, a considerable proportion of desA-profibrin is formed. Production of desA-profibrin is highest at low concentrations of ancrod, whereas desA-profibrin is rapidly converted to desAA-fibrin at higher concentrations of ancrod. Both desA-profibrin and desAA-fibrin monomers form fibrin complexes. A certain proportion of complexes carries exposed fibrin polymerization sites EA, indicating that the terminal component of the protofibril is a desAA-fibrin monomer unit. Soluble fibrin complexes potentiate tissue-type plasminogen activator-induced plasminogen activation. Significant amounts of plasmin are formed when soluble fibrin in plasma reaches a threshold concentration, leading to the proteolytic degradation of fibrinogen and fibrin. In the present setting, high concentrations of soluble fibrin are detected after 1 hour of ancrod infusion, whereas a rise in fibrinogen and fibrin degradation products, and plasmin-α2–plasmin inhibitor complex levels is first detected after 2 hours of ancrod infusion. Ancrod treatment also results in the appearance of cross-inked fibrin degradation productd-dimer in plasma.
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Dempfle, Carl-Erik, Sotiria Argiriou, Klaus Kucher, H. Müller-Peltzer, Klaus Rübsamen, and Dieter L. Heene. "Analysis of fibrin formation and proteolysis during intravenous administration of ancrod." Blood 96, no. 8 (October 15, 2000): 2793–802. http://dx.doi.org/10.1182/blood.v96.8.2793.h8002793_2793_2802.

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Ancrod is a purified fraction of venom from the Malayan pit viper, Calloselasma rhodostoma, currently under investigation for treatment of acute ischemic stroke. Treatment with ancrod leads to fibrinogen depletion. The present study investigated the mechanisms leading to the reduction of plasma fibrinogen concentration. Twelve healthy volunteers received an intravenous infusion of 0.17 U/kg body weight of ancrod for 6 hours. Blood samples were drawn and analyzed before and at various time points until 72 hours after start of infusion. Ancrod releases fibrinopeptide A from fibrinogen, leading to the formation of desAA-fibrin monomer. In addition, a considerable proportion of desA-profibrin is formed. Production of desA-profibrin is highest at low concentrations of ancrod, whereas desA-profibrin is rapidly converted to desAA-fibrin at higher concentrations of ancrod. Both desA-profibrin and desAA-fibrin monomers form fibrin complexes. A certain proportion of complexes carries exposed fibrin polymerization sites EA, indicating that the terminal component of the protofibril is a desAA-fibrin monomer unit. Soluble fibrin complexes potentiate tissue-type plasminogen activator-induced plasminogen activation. Significant amounts of plasmin are formed when soluble fibrin in plasma reaches a threshold concentration, leading to the proteolytic degradation of fibrinogen and fibrin. In the present setting, high concentrations of soluble fibrin are detected after 1 hour of ancrod infusion, whereas a rise in fibrinogen and fibrin degradation products, and plasmin-α2–plasmin inhibitor complex levels is first detected after 2 hours of ancrod infusion. Ancrod treatment also results in the appearance of cross-inked fibrin degradation productd-dimer in plasma.
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Demers, C., JS Ginsberg, P. Brill-Edwards, A. Panju, TE Warkentin, DR Anderson, C. Turner, and JG Kelton. "Rapid anticoagulation using ancrod for heparin-induced thrombocytopenia [see comments]." Blood 78, no. 9 (November 1, 1991): 2194–97. http://dx.doi.org/10.1182/blood.v78.9.2194.2194.

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Abstract In order to determine the efficacy and safety of ancrod, a rapid acting defibrinogenating drug, for patients with heparin-induced thrombocytopenia, 11 consecutive patients who required anticoagulant therapy because of venous thromboembolism and who developed acute heparin-induced thrombocytopenia or had a history of heparin-induced thrombocytopenia were treated with ancrod. Heparin therapy was discontinued (in patients receiving heparin) and ancrod started at a dose of 1 to 2 U/kg every 24 hours with subsequent daily doses adjusted to maintain fibrinogen levels between 0.5 and 1.0 g/L. Ancrod was continued until warfarin had become effective. The platelet count increased to more than 150 x 10(9)/L within 2 to 10 days in all thrombocytopenic patients. Two patients with a history of heparin- induced thrombocytopenia maintained normal platelet counts while receiving ancrod. Two patients had recurrent venous thrombosis while receiving warfarin, 10 days after ancrod was discontinued: one of these patients had metastatic pancreatic carcinoma and developed phlegmasia cerulea dolens and the other patient developed a venographically proven extension of her deep venous thrombosis. One patient suffered a bleeding episode into the thigh with a 16-g/L decrease in her hemoglobin level while receiving ancrod therapy. No other side effects were noted. Our experience indicates that ancrod therapy is a reasonable approach for patients with heparin-induced thrombocytopenia who require anticoagulant therapy.
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Demers, C., JS Ginsberg, P. Brill-Edwards, A. Panju, TE Warkentin, DR Anderson, C. Turner, and JG Kelton. "Rapid anticoagulation using ancrod for heparin-induced thrombocytopenia [see comments]." Blood 78, no. 9 (November 1, 1991): 2194–97. http://dx.doi.org/10.1182/blood.v78.9.2194.bloodjournal7892194.

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In order to determine the efficacy and safety of ancrod, a rapid acting defibrinogenating drug, for patients with heparin-induced thrombocytopenia, 11 consecutive patients who required anticoagulant therapy because of venous thromboembolism and who developed acute heparin-induced thrombocytopenia or had a history of heparin-induced thrombocytopenia were treated with ancrod. Heparin therapy was discontinued (in patients receiving heparin) and ancrod started at a dose of 1 to 2 U/kg every 24 hours with subsequent daily doses adjusted to maintain fibrinogen levels between 0.5 and 1.0 g/L. Ancrod was continued until warfarin had become effective. The platelet count increased to more than 150 x 10(9)/L within 2 to 10 days in all thrombocytopenic patients. Two patients with a history of heparin- induced thrombocytopenia maintained normal platelet counts while receiving ancrod. Two patients had recurrent venous thrombosis while receiving warfarin, 10 days after ancrod was discontinued: one of these patients had metastatic pancreatic carcinoma and developed phlegmasia cerulea dolens and the other patient developed a venographically proven extension of her deep venous thrombosis. One patient suffered a bleeding episode into the thigh with a 16-g/L decrease in her hemoglobin level while receiving ancrod therapy. No other side effects were noted. Our experience indicates that ancrod therapy is a reasonable approach for patients with heparin-induced thrombocytopenia who require anticoagulant therapy.
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Wiles, P. G., S. R. Nelson, K. K. Hampton, B. Casali, M. Boothby, and C. R. M. Prentice. "Therapeutic defibrinogenation by ancrod." Blood Coagulation & Fibrinolysis 1, no. 5 (October 1990): 385–88. http://dx.doi.org/10.1097/00001721-199010000-00005.

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Liu, Shuo, Victor J. Marder, David E. Levy, Shur-Jen Wang, Fan Yang, Annlia Paganini-Hill, and Mark J. Fisher. "Ancrod and Fibrin Formation." Stroke 42, no. 11 (November 2011): 3277–80. http://dx.doi.org/10.1161/strokeaha.111.622753.

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Soutar, R. L., and J. S. Ginsberg. "Anticoagulant therapy with ancrod." Critical Reviews in Oncology/Hematology 15, no. 1 (August 1993): 23–33. http://dx.doi.org/10.1016/1040-8428(93)90018-y.

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Krishnamurti, C., C. Bolan, CA Colleton, TM Reilly, and BM Alving. "Role of plasminogen activator inhibitor-1 in promoting fibrin deposition in rabbits infused with ancrod or thrombin." Blood 82, no. 12 (December 15, 1993): 3631–36. http://dx.doi.org/10.1182/blood.v82.12.3631.3631.

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Abstract The role of defective fibrinolysis caused by elevated activity of plasminogen activator inhibitor-1 (PAI-1) in promoting fibrin deposition in vivo has not been well established. The present study compared the efficacy of thrombin or ancrod, a venom-derived enzyme that clots fibrinogen, to induce fibrin formation in rabbits with elevated PAI-1 levels. One set of male New Zealand rabbits received intravenous endotoxin to increase endogenous PAI-1 activity followed by a 1-hour infusion of ancrod or thrombin; another set of normal rabbits received intravenous human recombinant PAI-1 (rPAI-1) during an infusion of ancrod or thrombin. Thirty minutes after the end of the infusion, renal fibrin deposition was assessed by histopathology. Animals receiving endotoxin, rPAI-1, ancrod, or thrombin alone did not develop renal thrombi. All endotoxin-treated rabbits developed fibrin deposition when infused with ancrod (n = 4) or thrombin (n = 6). Fibrin deposition occurred in 7 of 7 rabbits receiving both rPAI-1 and ancrod and in only 1 of 6 receiving rPAI-1 and thrombin (P “ .01). In vitro, thrombin but not ancrod was inactivated by normal rabbit plasma and by purified antithrombin III or thrombomodulin. The data indicate that elevated levels of PAI-1 promote fibrin deposition in rabbits infused with ancrod but not with thrombin. In endotoxin-treated rabbits, fibrin deposition that occurs with thrombin infusion may be caused by decreased inhibition of procoagulant activity and not increased PAI-1 activity.
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Krishnamurti, C., C. Bolan, CA Colleton, TM Reilly, and BM Alving. "Role of plasminogen activator inhibitor-1 in promoting fibrin deposition in rabbits infused with ancrod or thrombin." Blood 82, no. 12 (December 15, 1993): 3631–36. http://dx.doi.org/10.1182/blood.v82.12.3631.bloodjournal82123631.

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The role of defective fibrinolysis caused by elevated activity of plasminogen activator inhibitor-1 (PAI-1) in promoting fibrin deposition in vivo has not been well established. The present study compared the efficacy of thrombin or ancrod, a venom-derived enzyme that clots fibrinogen, to induce fibrin formation in rabbits with elevated PAI-1 levels. One set of male New Zealand rabbits received intravenous endotoxin to increase endogenous PAI-1 activity followed by a 1-hour infusion of ancrod or thrombin; another set of normal rabbits received intravenous human recombinant PAI-1 (rPAI-1) during an infusion of ancrod or thrombin. Thirty minutes after the end of the infusion, renal fibrin deposition was assessed by histopathology. Animals receiving endotoxin, rPAI-1, ancrod, or thrombin alone did not develop renal thrombi. All endotoxin-treated rabbits developed fibrin deposition when infused with ancrod (n = 4) or thrombin (n = 6). Fibrin deposition occurred in 7 of 7 rabbits receiving both rPAI-1 and ancrod and in only 1 of 6 receiving rPAI-1 and thrombin (P “ .01). In vitro, thrombin but not ancrod was inactivated by normal rabbit plasma and by purified antithrombin III or thrombomodulin. The data indicate that elevated levels of PAI-1 promote fibrin deposition in rabbits infused with ancrod but not with thrombin. In endotoxin-treated rabbits, fibrin deposition that occurs with thrombin infusion may be caused by decreased inhibition of procoagulant activity and not increased PAI-1 activity.
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Dissertations / Theses on the topic "Ancrod"

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Peterson, Jonathan Darryl. "Career Anchor Associations and the Boundaryless Career : a comparative Analysis between France and the United States." Thesis, Aix-Marseille, 2012. http://www.theses.fr/2012AIXM1037.

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Le concept de carrière, autrefois considéré comme étant un chemin linéaire au sein d'une même entreprise dont les perspectives d'évolution étaient tenues pour acquis, semble aujourd'hui évoluer vers de nouveaux concepts de carrière. La rhétorique organisationnelle depuis la fin des années 1980 a progressivement évolué a d'un concept d'emploi long terme et permanent vers une nécessité claire de prise de responsabilité individuelle en vue d'assurer sa propre progression et son propre développement de carrière (Bolton & Gold, 1994; Baruch, 2006). En outre, les différences générationnelles et les avancées technologiques ont considérablement modifié l'environnement et le marché du travail, facilitant ainsi les échanges au-delà des frontières géographiques et introduisant la nécessité de gérer les différences culturelles (Crepeau et al., 2006). Nous analysons deux concepts de carrière afin de comprendre la vision contemporaine de carrière : en premier lieu, nous avons utilisé la théorie d'ancres de carrière développée par Schein (1978) qui a donné lieu à une compréhension plus large des différences individuelles ainsi que de la culture organisationnelle selon une perspective interdisciplinaire. Schein a défendu le point de vue que l'individu développe un « concept de soi » associé à sa carrière, qui devient une ancre stabilisant et influençant ses décisions liées à sa carrière. Dans une nouvelle conceptualisation de cette analyse, Feldman & Bolino (1996) défendent le point de vue que les ancres de carrière ont des associations les unes avec les autres. Ils proposent un modèle de compatibilité ou d'opposition basé sur le concept des huit ancres de carrière de Schein
Careers that were once conceptualized as life-long linear paths with frequent upward mobility within one organization have increasingly given way to new models of how careers unfold. The organizational rhetoric since the late 1980's has progressively changed from offering long-term and life-long employment, to clear messages concerning the imperative of individual career responsibility for one's progression and development (Bolton & Gold, 1994; Baruch, 2006). In addition, generational differences and technological advances have greatly changed the face of the work place, easily allowing the crossing of borders and the necessity to deal with cultural differences (Crepeau et. al., 2006). To understand how contemporary workers navigate in their careers, we mobilized two main career concepts. First, we used the theory of career anchors developed Schein (1978), which gave rise to wider understanding of both individual differences and organizational culture from an interdisciplinary perspective. Schein postulated that individuals develop a career “self-concept” which becomes an “anchor”, stabilizing and directing individual career decision-making. A re-conceptualization of his work by Feldman & Bolino (1996) postulated that career anchors could be associated with each other, proposing a model of compatibility or opposition in the relationships between the eight career anchors of Schein. Second, we used the boundaryless career concept, which suggested a change from the traditional career ideal of one single employer for the entirety of one's professional life (Arthur & Rousseau, 1996; Sullivan & Arthur, 2006)
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Phoenix, David Andrew. "The amphiphilic #alpha# - helical anchor." Thesis, University of Liverpool, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.317213.

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Lundin, Reid. "Introduction to concrete anchor rods." Kansas State University, 2012. http://hdl.handle.net/2097/15040.

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Master of Science
Department of Architectural Engineering
Don Phillippi
Concrete anchors represent an important transition for both the design and construction of a project. Anchors are produced in two main categories: cast-in-place and post-installed. For designers, anchors are used to attach steel members to supporting concrete members. The anchors are designed using the provisions outlined in Building Code Requirements for Structural Concrete, ACI 318-11, Appendix D. These anchors are used to transmit tension and shear forces by using an individual anchor or a multiple anchor group. For contractors, anchor installation marks the transition between concrete and steel construction. Various types of anchors are produced by manufacturers, requiring contractors to be familiar with many installation methods. Careful planning and coordination is necessary to layout and place anchors into their correct location. Once anchors are installed, they must be protected from damage resulting from moving machinery and material. The purpose of this report is to introduce the basics to design concrete anchors by outlining the provisions in ACI 318-11 Appendix D and demonstrating these provisions with design examples. Anchor types, applications and common construction issues important to the structural engineer are also discussed.
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Dias, Cristiana de Oliveira. "Sonae SGPS: retail the anchor." Master's thesis, NSBE - UNL, 2010. http://hdl.handle.net/10362/10342.

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Ewertz, Correa Benjamín. "Mall de Ancud." Tesis, Universidad de Chile, 2016. http://repositorio.uchile.cl/handle/2250/143446.

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Memoria para optar al título de Arquitecto
Hace ya 30 años que se instauró la figura del Mall en Chile, convirtiéndose en un fenómeno que no ha dejado a nadie indiferente, pues si bien su desarrollo ha sido prácticamente imperceptible para el común de la sociedad, no es menos cierto que ha cambiado, de algún u otro modo, la idiosincrasia del ciudadano chileno. En la actualidad, es una realidad latente e innegable que el Mall es considerado un elemento básico e indispensable para el Chile contemporáneo, llegando al punto de constituir una parte fundamental en el “imaginario de la ciudad”, es decir, a percibirlo como pieza clave que forma parte del contexto social. A pesar de ello, para nuestra profesión es aún un tema tabú, poco discutido, llegando a ser considerado como un espacio no arquitectónico, una estructura “horrorosa” que se empecina en destruir el entorno de la ciudad. Sin embargo, hoy en día, este tema ha generado gran controversia con la llegada del Mall en Castro,Chiloé, construcción que no dejó a nadie exento de opiniones, principalmente por la puesta en peligro de la imagen del patrimonio local dada su imponente morfología. Pese a ello y sumándole las graves falencias urbanas, paisajísticas y, principalmente, legales, el Mall tuvo un gran apoyo por la comunidad chilota, quienes lo consideran como un efecto necesario e imprescindible para el desarrollo local. Es por lo antes mencionado que la empresa que proyectó el Mall de Castro, consideró los deseos y la gran aceptación que el proyecto tuvo entre los habitantes de la Isla, llevándolo a adquirir nuevos terrenos en la zona norte del archipiélago, específicamente en Ancud, con el objeto de construir un inmueble de características similares; no obstante, la construcción tiene como principal falencia, la poca comunicación arquitectónica con la ciudad, pues es un recinto hermético, un espacio indiferente con el contexto chilote. Es en este mismo terreno de Ancud donde ubico el proyecto de título, pues es una oportunidad directa de intervenir en la ciudad, como también es una posibilidad de lograr la articulación entre lo público y lo privado, lo local con lo foráneo, consolidando a la vez la zona comercial, turística, social y urbana más importante de la zona norte de la Isla de Chiloé. Por todo lo anterior es necesario entender que el Mall es un efecto consolidado y que no debemos caer en su rechazo, sino que por el contrario, es indispensable interiorizar y reflexionar sobre este tema; por ello es preciso preguntarse ¿cómo se puede articular un centro comercial de esta tipología en una ciudad con alto carácter como Ancud?
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Ahlinder, Kristina. "Development of a Folding Boat Anchor." Thesis, KTH, Maskinkonstruktion (Inst.), 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-141185.

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This report is the result from a Master of Science Thesis work at the Royal Institute of Technology, and is executed by technology student Kristina Ahlinder for the behalf of Design Company Top Notch Design AB. Top Notch Design wants to expand their range of marine products and look in to the market of boat anchors. The aim of the project is to develop an anchor for small leisure boats for the Swedish market with focus on usability and safety. A detailed informational study was executed at the beginning of the project. To collect information regarding how potential users experience current anchors on the market, a user test and a digital questionnaire was done. Through the questionnaire, the user test, requirements from the company and the informational study a criteria specification was made. Some of the criteria were that it should be an easy to stow day anchor designed for leisure boats up to 27 feet and have a maximal weight of 8 kg. Other important criteria were good usability and innovative and interesting design. The ideation stage of the project was mainly based upon brainstorming sessions. 11 concepts were sketched and prototyped in paper and wood. Ten of the concepts were manufactured in small-scale and welded together in steel, in order to evaluate the functionality. The anchors were dragged in a sandbox, and the drag-force was measured with a nanometer. The anchor that performed best in the drag test was a kind of foldable plow anchor. This is the concept that was chosen to be developed.The anchor was further developed and tested before being modeled in the CAD-program SolidWorks. The final design consists of a fluke and a shaft connected by a joint, and a handle for usability, ease of recovery and for getting it in the right angle on the seafloor. The anchor has an automatic locking mechanism in the joint, and rubber details. The anchor’s material is mainly aluminum and weighs 3.8 kilos and has a length of 48 centimeters.The anchor fulfills the criteria specification and most of the preferences and according to calculations the joint will hold for forces higher than 2000 N. The anchor will, according to calculations, have an approximate selling price of slightly over 500 SEK. According to feedback given by potential users, 40 % has a good impression of the anchor and 26 % could imagine buying it. The outcome is a new and innovation type of anchor that suits the modern small-boat owner. The anchor has to be tested in full scale with the correct materials to fully be evaluated. The anchor would contribute to the conservative marine market. The anchor would be a brave and interesting addition to the current anchor market, as well as to TND’s current marine products.
Denna rapport redovisar resultatet av ett examensarbete vid Kungliga Tekniska Högskolan, utfört av teknologstudent Kristina Ahlinder för företaget Top Notch Design AB. Top Notch Design vill utöka sitt sortiment av marina produkter och undersöka marknaden för båtankare. Syftet med projektet är att utveckla ett ankare för små fritidsbåtar för den svenska marknaden, med fokus på användarvänlighet och säkerhet. En detaljerad informationssökning genomfördes i början av projektet. För att samla in information om hur potentiella användare upplever nuvarande ankare på marknaden utfördes ett användartest och en enkätundersökning. Genom resultat från enkäten, användartestet, krav från företaget samt informationssökningen kunde en kravspecifikation tas fram. Några av kriterierna var att det skulle vara ett stuvbart dagsankare avsett för fritidsbåtar upp till 27 fot och har en maximal vikt på 8 kg. Andra viktiga kriterier gällde avändbarhet och design. Den idégenerering som utfördes i projektet var huvudsakligen baserad på brainstorming. 11 koncept modellerades i papper och trä. Tio av de koncepten tillverkades som skalmodeller i stål för att utvärdera funktionaliteten. Modellerna drogs sedan i en byggd sandlåda och dragkraften mättes med en nanometer. Ankaret som visade sig bäst i dragtestet var ett slags hopfällbart plogankare. Detta var det koncept som valdes för vidareutveckling. Ankaret vidareutvecklades och testades vidare innan det slutgiltiga konceptet modellerades i CAD-programmet SolidWorks. Den slutliga konstruktionen består av ett fly och ett skaft förbunden med en led, samt ett handtag/peke. Ankaret har en automatisk låsmekanism i leden samt gummidetaljer för att förbättra användarvänligheten. Ankaret är huvudsakligen i aluminium och väger 3,8 kilo och har en längd av 48 centimeter.Ankaret uppfyller kravspecifikationen och de flesta av de önskemål som ställts upp. Enligt beräkningar utförda kommer ledens axel att klara krafter högre än 2000 N. Ankaret kommer, enligt beräkningar, ha ett ungefärligt försäljningspris på drygt 500 kronor. Enligt feedback från potentiella användare har 40 % ett bra intryck av ankaret och 26 % kan tänka sig att köpa det. Resultatet är ett nytt och innovativt ankare som passar den moderna småbåtsägaren. Ankaret är ett modigt och intressant komplement till det nuvarande ankarutbudet, liksom till TND nuvarande marina produkter.
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Gericke, Ludwig. "Mindful mediations at Three Anchor Bay." Master's thesis, University of Cape Town, 2011. http://hdl.handle.net/11427/13096.

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This project is a synthesis of, on the one hand, the interventionist architect curiously and deliberately plotting form and visualising construction and, on the other hand, the human being often wilfully retreated and joyfully observing the uninterrupted and the conflicting. It is this dialectic - rather than immovable theoretical principles - that has informed not only my process, but also my design. In this sense this project represents what I believe to be the most important feature of my architectural education: the inexplicable joy in the constant re-evaluation of the imprecise nexus between the deliberately mediated and the uninterrupted. This impulse is also what (perhaps unknowingly at the time) attracted me to Three Anchor Bay - a site of untameable swells, impenetrable rhythms, ebb and flow. It is a site that necessitates decisiveness in a counterintuitive form; boundaries. Any frontier, however versatile and accommodating, requires commitment (few are capable of confidently kayaking beyond an otherwise parameter-defining promenade). Drawing a line is not only the problem of the architect, but the human being. Although this paper is largely a personal essay instead of a coherent treatise (I reserve the right to remain sceptical of every decision), it is important to make a few general observations. The first is supremely personal: I am decidedly fallible. Although harsh introspection is generally more valuable and courageous than the resolute defence of personal conviction, I often found myself passionately defending lines I have drawn (especially ones that I have spent a lot of time re- drawing and erasing). Redrawing can be a counterintuitive struggle and it has often been difficult to regard it as a necessary and unpredictable process rather than as emblematic of some sort of failure. Although common sense urges us to "learn from our mistakes", it is never quite that simple. This project has, in short, caused me to constantly mediate between conviction and perpetual self-criticism. Secondly, these ideas are by no means new and have been repeated (and often ignored) in various contexts. Karl Popper, for instance, believed that "any idea of Utopia is necessarily closed owing to the fact that it chokes on its own refutations. The simple notion of a good model for society that cannot be left open for falsification is totalitarian” (Taleb, 2004, p.128-129). The same is true of architecture - particularly those projects that are resolutely planted in a pre-determined style, ideology or “balance”.
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Yorgancioglu, Kaan. "Using Anchor Nodes for Link Prediction." Case Western Reserve University School of Graduate Studies / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1578499802599777.

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Mucolli, Gent. "Fuzzy modeling of suction anchor behavior based on cyclic model tests data." Digital WPI, 2016. https://digitalcommons.wpi.edu/etd-theses/1313.

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This paper proposes a novel model that can predict the displacement of suction caisson anchors under monotonic and cyclic loading. Failure is assumed to occur when the accumulative monotonic and cyclic displacement along the load attachment point is over 60% of the diameter of the anchor. The anchors will go through lateral failure when the accumulative monotonic and cyclic displacement along the loading direction at the load attachment point is over 30% of the diameter. Hence, it is important to predict this displacement and therefore determine the expected failure of the anchor. However, it is difficult to predict displacement using the modern software without knowing the material properties of the soil and piles. Hence a new model that relies only on the normalized static load (Fa/Ff), normalized cyclic load (Fcy/Ff ), loading angle (Θ), and the number of cycles (N) is proposed. The inputs for training of the proposed model are (Fa/Ff), (Fcy/Ff), (Θ), (α) and (N). The output of the model will be the displacement normalized by the diameter of the anchor. To generalize the trained model, unused sets of data are used to validate the model. Furthermore, a comparative study is performed to evaluate the effectiveness of the proposed model. It is shown from extensive simulation that the model can accurately predict the normalized displacement of suction caisson anchors.
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Lindquist, Lisa. "Corrosion of steel bridge Girder anchor bolts." Thesis, Georgia Institute of Technology, 2008. http://hdl.handle.net/1853/24649.

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The research objectives for this project were to explicitly define the anchor bolt corrosion problem in the state of Georgia and recommend action to the Georgia Department of Transportation. The bearing assembly of concern is the plate bearing assembly, in which carbon steel and/or bronze plates are anchored by either carbon steel or stainless steel anchor bolts. Inspection report data revealed that anchor bolt corrosion was ubiquitous for all environments in Georgia; the problem was reported for 27% of the steel girder bridges throughout the state. Based on a synthesis of the field investigations, bolt failure analyses, laboratory experimental testing, and review of GDOT inspection report surveys, the corrosion of carbon steel anchor bolts is caused universally by concentration cell corrosion. Other corrosion mechanisms of concern are galvanic and crevice corrosion, which are both enhanced by the current bearing design. Corrosion protection provided through zinc galvanization cannot sufficiently protect the carbon steel bolt for its entire service life. Corrosion potential and cyclic polarization data confirmed that ASTM Type 304, Type 316, Type 2101, and Type 2205 were protected from concentration cell and localized corrosion in the simulated bearing environment. Therefore, it is recommended that the stainless steel anchor bolts of these types be use in future designs and that the bolts should be electrically separated from all dissimilar metals using a Nylon or Teflon washer to prevent preferential corrosion of carbon steel. It is further recommended that the bronze lube plate should be eliminated entirely and that the bearing type should be a reinforced elastomeric bearing. Maintenance of existing sliding plate bearings should include regular cleaning by brushing away debris from the bearing surfaces, and bridges with carbon steel anchor bolts should be retrofitted to provide additional lateral restraint according to current maintenance procedures.
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Books on the topic "Ancrod"

1

Stenson, James B. Anchor. New York, NY: Scepter Publishing, 2003.

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León, Alberto Ajón. Ancora. La Habana, Cuba: Editorial Letras Cubanas, 2003.

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Das, M. K. Anchor Cochin. Edited by Cochin Port Trust. Cochin: Cochin Port Trust, 2008.

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Cerchiamoci ancora. Napoli: tullio pironti editore, 2012.

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Rebora, Roberto. Non ancora. Milano: Libri Scheiwiller, 1989.

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Hancox, Michael. Anchor Handling. Ledbury: Oilfield Publications, 1994.

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Amare ancora. [Milano]: Sperling, 1993.

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Piero, Alessandro Del. Giochiamo ancora. Milano: Mondadori, 2012.

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Nannetti, Remo. Ancora grammatica. Leamington Spa: Language Centre Publications, 1998.

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Bianchi, Bianca. Vivrò ancora. Firenze: Morgana, 1997.

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Book chapters on the topic "Ancrod"

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Mauckner, A., R. Schneider, W. Sturm, W. Doering, S. Wolff, H. Brückmann, K. Willmes, M. Schlenker, and E. B. Ringelstein. "Enzymatische Defibrinogenierung mit ANCROD bei subkortikaler arteriosklerotischer Enzephalopathie." In Verhandlungen der Deutschen Gesellschaft für Neurologie, 1076–80. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-83771-5_258.

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Mauri, J. M., R. Poveda, M. T. González, M. Carrera, J. Carreras, C. Díaz, and P. Ferrer. "Defibrination with Ancrod in Crescentic Glomerular Lesions: Preliminary Observation." In Current Therapy in Nephrology, 28–30. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0865-2_6.

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Shekhar, Shashi, and Hui Xiong. "Anchor Points." In Encyclopedia of GIS, 20. Boston, MA: Springer US, 2008. http://dx.doi.org/10.1007/978-0-387-35973-1_55.

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Regardt, Olle, Lars Rönnbäck, Maria Bergholtz, Paul Johannesson, and Petia Wohed. "Anchor Modeling." In Conceptual Modeling - ER 2009, 234–50. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-04840-1_19.

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Mehlhorn, Heinz. "Raushitaenia ancora." In Encyclopedia of Parasitology, 1. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27769-6_5051-1.

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Plachouras, Vassilis. "Anchor Text." In Encyclopedia of Database Systems, 102–3. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4614-8265-9_939.

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Dobhal, D. P. "Anchor Ice." In Encyclopedia of Earth Sciences Series, 40. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-90-481-2642-2_19.

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Plachouras, Vassilis. "Anchor Text." In Encyclopedia of Database Systems, 84. Boston, MA: Springer US, 2009. http://dx.doi.org/10.1007/978-0-387-39940-9_939.

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Jia, Junbo. "Anchor Piles." In Soil Dynamics and Foundation Modeling, 641–54. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-40358-8_25.

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Luo, Yong. "Mooring Anchor." In Encyclopedia of Ocean Engineering, 1–7. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-10-6963-5_151-1.

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Conference papers on the topic "Ancrod"

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Reddy, K. N. N., B. Cercek, and W. Ganz. "ENHANCEMENT OF PLASMINOGEN ACTIVATION BY TISSUE-TYPE PLASMINOGEN ACTIVATOR IN THE PRESENCE OF ANCROD AND FIBRINOGEN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644405.

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Ancrod, a thrombin-like enzyme from Malayan pit viper venom, when infused into experimental animals or man converts fibrinogen into fibrin micro-clots. In a recent study we have found that in dogs pretreatment with ancrod markedly enhanced thrombolysis by recombinant tissue-type plasminogen activator (rt-PA), presumably by depleting fibrinogen and preventing new fibrin uptake by the thrombus during lysis. The rapid clearance of large amounts of fibrinogen from the circulation and the appearance of fibrin degradation products during ancrod treatment is indicative of high fibrinolytic activity. In this study we found that ancrod enhances activation of plasminogen by rt-PA indirectly via plasmin mediated digestion of fibrin. To a reaction mixture containing lys-plasminogen, human fibrinogen and val-leu-lys-pNA (S-2251), ancrod was added followed by rt-PA. At time intervals plasminogen activation was followed by measuring amidolytic activity on S-2251 at 405 nm. Ancrod or fibrinogen alone had no significant effect on the activation of plasminogen by rt-PA. However, the amidolytic activity increased with time in reaction mixture containing ancrod and fibrinogen. When a small amount of alpha-2-antiplasmin was added to the reaction mixture prior to the addition of rt-PA (the inhibitor level was sufficient to inhibit only a fraction of plasmin generated during the assay period) the activation rate was very much reduced. Thus, a small amount of plasmin initiates digestion of fibrin which may result in the exposure of new sites on fibrin that enhance the rate of activation of plasminogen by rt-PA. The rapid clearance of fibrinogen during ancrod infusion may not be due to increased susceptibility of micro-clots to lysis but to increased rate of plasmin formation. These results further confirm the observations of other investigators about the role of plasmin in the activation of plasminogen by tissue plasminogen activator.
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Reber, G., Ph de Moerloose, M. Sinclair, A. Schweizer, J. P. Gardaz, and C. A. Bouvier. "LOW MOLECULAR WEIGHT HEPARIN, STANDARD HEPARIN AND ANCR0D AS ANTICOAGULANT FOR EXTRAC0RP0REAL MEMBRANE LUNG CO2 REMOVAL IN DOGS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643086.

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A low molecular weight heparin (LMWH, Sandoz), a standard heparin (SH, LiqueminR) and ancrod (Arvin^)have been compared in an extracorporeal veno-venous bypass for CO2 removal using a membrane lung in dogs. Four animals received 150 anti-FXa U/kg in bolus followed 90 min later by 40 anti-FXa U/kg/h of LMWH and four other 300 IU/kg followed by 100 IU/kg/h of SH. Six dogs received 1 U/Kg of ancrod intravenously during 2 h before bypass started.Mean FXa inhibition was 49% in LMWH group and 29% in SH group, mean FI la inhibition 31% and 49% respectively. No statistically significant differences were found between LMWH and SH groups for any of the parameters measured (fibrinogen, FV, antithrombin III, plasminogen, α 2-anti piasmin, platelet counts). At the end of bypass 5000 U protamine abolished both anti-FXa and anti-FIIa activities in SH group, but failed to neutralize more than half of anti-FXa in LMWH group. In ancrod group no clottable fibrinogen was detectable. A dramatic fall of α2-antiplasmin was observed after ancrod infusion and, during bypass, sharp decreases in FV and platelet counts without significant antithrombin III and plasminogen consumption were noted.No bleeding occurred in any group throughout the seven hours of bypass. The main finding was fibrin deposition in the tubing in three out of four dogs receiving LMWH, whereas no fibrin deposition occurred either in SH or in ancrod group.These results suggest that in this model which involves blood contact with artificial surfaces (1) SH and ancrod are efficient to prevent clotting, but not the LMWH used at the dosage reported (2) high anti-FIIa activities are required to prevent fibrin formation (3) ancrod defibrination does not protect against severe haemostatic disturbances and (4) the use of LMWH may raise problems when emergency neutralisation procedures are required.
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Pretice, C. R. M., and H. A. Townsend. "ANCROD PROPHYLAXIS AFTER SURGERY FOR FRACTURED NECK OF FEMUR: A STUDYOF FATAL PULMONARY EMBOLISM." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643685.

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A pilot multi-centre randomized controlledtrial was carried out to compare ancrod (Arvin, Knoll) versus no medical treatment in 453 patients having surgery for fractured neck of femur to assess prevention of fatal post-operative pulmonary embolism (PE).Ancrod was given subcutaneously by 5 daily injections starting immediately post-operatively; initially 4u/kg bw were given followed by 4 injections of 1u/kg bw, to reduce fibrinogen levels to 80mg/dl. The primary objective of the study was to record mortality due to PE, as shown by the DeathCertificate, within 3 months after surgery.Death Certificates were analysed by 2 medical assessors, unaware of the patient treatment group. Of 239 control patients, not given ancrod, there were 5 deaths due to PE and 2 deaths where PE may have been contributory. Total deaths were 30 (12.5%). In 214 ancrod treated patients there were 2 deaths due to PE and a further 3 where it may have been contributory. Total deaths were 31 (14.5%), not significantly different from the control group. Deaths from PE occurred between16 and 66 days after surgery. Although in this study there was abeneficial tendency for ancrod to reduce fatal PE it is likely that at least 6,000 patients would be needed to demonstrate that any drug significantly reduces by 50% the incidence of fatal PE compared to the control group. Wound infection was recorded in 16 patients in both groups. Wound haematomas were seen in 26 ancrod patients compared to 6 controls (p<0.02) but were not sufficiently serious to warrant re-exploration or prolonged hospital stay. The low mortality due to PE in our patients with fractured neck of femur (2%) is contrasted with the figures of Sevitt & Gallagher, 1959(8%). The low incidence of fatal PE in thehigh risk group studied here should be taken into account when assessing future antithrombotic prophylaxis after surgery. Advances in anaesthetics, surgery and rehabilitation may have contributed to the decline in fatal post-operative PE. Effective assessment of drugs for prophylaxis against post-operative venous thrombosis is best carried out by large scale simple controlled trials using fatal PE a the primary end point. Collaborative Centres were located in Portsmouth, Cape Town, Glasgow, Belfast and Leeds.
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Lowe, O. DG. "RHEOLOGY AND VENOUS THROMBOEMBOLISM." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643990.

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Changes in the composition of the blood, venous stasis, and interaction of the blood with the vessel wall (Virchow's triad) all have rheological aspects which may promote venous thrombogenesis.Blood composition and rheology. Increasing levels of venous haematocrit and fibrinogen increase bulk blood viscosity, especially at low shear rates such as are encountered in veins, when red cell aggregation occurs. Static blood requires a minimum shear stress for flow (yield stress), which is also strongly dependent on haematocrit and fibrinogen levels. Increases in haematocrit and fibrinogen also promote platelet adhesion and aggregation. Polycythaemia carries an increased risk of venous thromboembolism, which can be reduced by lowering the haematocrit; conversely, anaemic patients (renal failure, pernicious anaemia) have a subnormal prevalence of pulmonary embolism at autopsy. Increased preoperative levels of haematocrit, fibrinogen and blood viscosity predicted postoperative deep vein thrombosis in some studies, but not in others: they have complex relationships to other risk factors and illnesses. Postoperative changes in haematocrit, fibrinogen and blood viscosity may also be relevant to thrombogenesis, as may haemoconcentration in leg veins.Venous flow disturbance and rheology. The flow behaviour of particles and cells in venous valve pockets has been studied by Karino: particles and cells were observed to leave mainstream flow and circulate in paired vortices in low-shear areas within the valve pockets. A cell-poor hypoxic area at the apex of the valve pocket may favour thrombogenesis. Valve pockets might therefore act as in vivo aggregometers, with optimal conditions for activated cells or coagulation products to promote platelet and red cell aggregation, which might be facilitated by increases in haematocrit or fibrinogen. Sevitt has observed cellular aggregates in valve pockets at autopsy, which might act as a nidus for thrombus initiation. Successive layers of thrombus will disturb flow steamlines, as well as generating procoagulant activity: hence a series of "aggregometers" might result in successive bursts of thrombosis and the layered structure of venous thrombi observed by Sevitt. Variations in haematocrit, fibrinogen and red cell aggregation may influence stasis of blood following venous occlusion by thrombus, and hence affect thrombotic extension; they may also influence residual lung perfusion following pulmonary embolism.Therapeutic aspects of rheology. Leg stockings and other physical methods of preventing deep vein thrombosis may improve flow disturbance in valve pockets, as well as in axial veins. The efficacy of perioperative dextran in prevention of venous thromboembolism may partly reflect haemodilution and its rheological consequences. Likewise, postoperative defibrination with ancrod reduced the incidence and extent of deep vein thrombosis after hip surgery, which may partly reflect reductions in plasma viscosity and red cell aggregation. Defibrination with ancrod reduced the haemodynamic disturbance, and the mortality, of experimental pulmonary embolism in dogs, possibly by increasing residual perfusion.. Similarly, improved perfusion after thrombolytic therapy of pulmonary embolism in man may reflect the rheological consequences of fibrinogen depletion, as well as thrombolysis.
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Farny, Jacob, Matthew Jennex, Rebekah Olsen, and Melissa Rodriguez. "Anchor." In the 2012 ACM annual conference extended abstracts. New York, New York, USA: ACM Press, 2012. http://dx.doi.org/10.1145/2212776.2212438.

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Miura, Motoki, Taro Sugihara, and Susumu Kunifuji. "Anchor garden." In the 14th annual ACM SIGCSE conference. New York, New York, USA: ACM Press, 2009. http://dx.doi.org/10.1145/1562877.1562925.

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Barbarino, Andrea, and Daniele Tomatis. "ANCore Analysis." In SNA + MC 2013 - Joint International Conference on Supercomputing in Nuclear Applications + Monte Carlo, edited by D. Caruge, C. Calvin, C. M. Diop, F. Malvagi, and J. C. Trama. Les Ulis, France: EDP Sciences, 2014. http://dx.doi.org/10.1051/snamc/201402208.

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Bakke, Monika, and Geir Magne Berg. "Rolling Anchor System." In SPE/ICoTA Coiled Tubing and Well Intervention Conference and Exhibition. Society of Petroleum Engineers, 2006. http://dx.doi.org/10.2118/99708-ms.

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Taraldsen, A. "Anchor Chain Fractures." In Offshore Technology Conference. Offshore Technology Conference, 1985. http://dx.doi.org/10.4043/5059-ms.

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Braginsky, Anastasia, Alex Kogan, and Erez Petrank. "Drop the anchor." In SPAA '13: 25th ACM Symposium on Parallelism in Algorithms and Architectures. New York, NY, USA: ACM, 2013. http://dx.doi.org/10.1145/2486159.2486184.

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Reports on the topic "Ancrod"

1

Housley, R., S. Ashmore, and C. Wallace. Trust Anchor Format. RFC Editor, June 2010. http://dx.doi.org/10.17487/rfc5914.

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Reddy, R., and C. Wallace. Trust Anchor Management Requirements. RFC Editor, October 2010. http://dx.doi.org/10.17487/rfc6024.

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Housley, R., S. Ashmore, and C. Wallace. Trust Anchor Management Protocol (TAMP). RFC Editor, August 2010. http://dx.doi.org/10.17487/rfc5934.

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Zephyr Feryok, Zephyr Feryok. Mountain Rescue Anchor Strength Testing. Experiment, December 2014. http://dx.doi.org/10.18258/4299.

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Petrik, W. A. Aquifers and hydrology at Anchor Point, Alaska. Alaska Division of Geological & Geophysical Surveys, 1993. http://dx.doi.org/10.14509/1609.

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Piperias, P. Fatigue Testing of a Floor Anchor Specimen. Fort Belvoir, VA: Defense Technical Information Center, March 1992. http://dx.doi.org/10.21236/ada251674.

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Taylor, Nathan L., and Robert Strauch. Suture Anchor Arthroplasty for Thumb Carpometacarpal Osteoarthritis. Fort Belvoir, VA: Defense Technical Information Center, July 2004. http://dx.doi.org/10.21236/ada424777.

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Humphrey, Neil F. Quantifying Fluvial Sediment Transport by Anchor Ice. Fort Belvoir, VA: Defense Technical Information Center, September 1999. http://dx.doi.org/10.21236/ada630378.

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Huston, G., S. Weiler, G. Michaelson, and S. Kent. Resource Public Key Infrastructure (RPKI) Trust Anchor Locator. RFC Editor, February 2012. http://dx.doi.org/10.17487/rfc6490.

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Ashmore, S., and C. Wallace. Using Trust Anchor Constraints during Certification Path Processing. RFC Editor, August 2010. http://dx.doi.org/10.17487/rfc5937.

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