Academic literature on the topic 'And Alt'

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Journal articles on the topic "And Alt"

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Morgan, Saretta. "Alt/Nature, and: Alt/Nature." Colorado Review 47, no. 2 (2020): 73–74. http://dx.doi.org/10.1353/col.2020.0040.

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Lotzgeselle, Michael. "Alt ist nicht gleich alt." physiopraxis 4, no. 07/08 (July 2006): 38–40. http://dx.doi.org/10.1055/s-0032-1308013.

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Kramer, Jane C. "ALT." Academic Therapy 23, no. 1 (September 1987): 7–15. http://dx.doi.org/10.1177/105345128702300102.

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Bin Kingombe, Cesar I., Jean-Yves D'Aoust, Geert Huys, Lisa Hofmann, Mary Rao, and Judy Kwan. "Multiplex PCR Method for Detection of Three Aeromonas Enterotoxin Genes." Applied and Environmental Microbiology 76, no. 2 (November 20, 2009): 425–33. http://dx.doi.org/10.1128/aem.01357-09.

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ABSTRACT A novel multiplex PCR method using three sets of specific primers was developed for the detection of the cytotoxic (act), heat-labile (alt), and heat-stable (ast) enterotoxin genes in Aeromonas spp. This assay was used to characterize 35 reference strains as well as 537 food-borne isolates. A total of seven gene pattern combinations were encountered, including act, alt, act/alt, act/alt/ast, act/alt/148-bp amplicon, alt/ast, and alt/148-bp amplicon. The alt gene was detected with 34 reference strains (97%) and occurred singly in 14% of these strains. The frequency of occurrence of the act/alt, act/alt/ast, and alt/ast gene patterns in reference strains was 14 (40%), 2 (6%), and 2 (6%), respectively. An unpredicted amplicon was detected in 11 reference strains (31%). Characterization of this amplicon showed that its size was 148 bp, as generated by the AHLF and AHLR primers, and that it uniquely aligned with the Aeromonas salmonicida A449 genome sequence (GenBank accession number CP000644). This amplicon was named Aeromonas salmonicida subsp. salmonicida hypothetical protein amplicon (AssHPA). In the 537 food-borne isolates, the act and alt genes were most dominant and were detected in 349 (65%) and 452 (84%) isolates, respectively, either alone or in combinations. The act and alt genes occurred singly in 30 (6%) and 128 (24%) of these strains, respectively. The act/alt gene pattern occurred in 315 isolates (59%), whereas the ast gene was always linked to strains exhibiting the act/alt/ast and alt/ast gene combinations in 4 (0.7%) and 5 (0.9%) isolates, respectively. The uniplex amplification of three enterotoxin genes separately confirms the specificity of the unique selected primers. This multiplex PCR is rapid and simple and can detect the presence of three Aeromonas enterotoxin genes in a single assay.
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Jasser, Greta, and Agnes Wankmüller. "Alt-Right, Alt-Tech, Alt-Internet? Rechte Online Plattformen und ihre Funktion." Forschungsjournal Soziale Bewegungen 33, no. 2 (September 11, 2020): 506–12. http://dx.doi.org/10.1515/fjsb-2020-0042.

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Reeder, Laura. "ALT/space." Teaching Artist Journal 9, no. 1 (January 2011): 47–55. http://dx.doi.org/10.1080/15411796.2011.531648.

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Reeder, Laura. "ALT/space." Teaching Artist Journal 9, no. 2 (March 31, 2011): 116–24. http://dx.doi.org/10.1080/15411796.2011.556568.

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Reeder, Laura. "ALT/space." Teaching Artist Journal 9, no. 3 (June 28, 2011): 183–92. http://dx.doi.org/10.1080/15411796.2011.585899.

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Reeder, Laura. "ALT/space." Teaching Artist Journal 9, no. 4 (October 11, 2011): 243–48. http://dx.doi.org/10.1080/15411796.2011.604626.

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Rosenfeld, Malke. "ALT/space." Teaching Artist Journal 10, no. 1 (January 2012): 43–53. http://dx.doi.org/10.1080/15411796.2012.630639.

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Dissertations / Theses on the topic "And Alt"

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James, Jessica. "CTRL-ALT-DELETE." Thesis, California State University, Long Beach, 2013. http://pqdtopen.proquest.com/#viewpdf?dispub=1523196.

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This thesis examines concepts of control, alternation, and deletion (CTRL, ALT, DELETE) through the poetic process. By examining some of the specific poems presented here, one can see the effects of literary and social critics including Michel Foucault, Hart Crane, and Adrienne Rich on my poetry. Thematically, structurally, and linguistically, the poems in this thesis address contemporary concerns and ask the reader to face the challenges of postmillennial life with creativity, empathy, and humor.

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Becker, Hans-Joachim. "Das Feld um "alt" /." Heidelberg : C. Winter, 1991. http://catalogue.bnf.fr/ark:/12148/cb371607491.

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Haas, Anna-Lena [Verfasser], Bernd [Akademischer Betreuer] [Gutachter] Alt-Epping, and Julie [Gutachter] Schanz. "Symptomkontrolle und Lebensqualität als primärer Endpunkt klinischer Studien - ein "Systematic Review" / Anna-Lena Haas ; Gutachter: Bernd Alt-Epping, Julie Schanz ; Betreuer: Bernd Alt-Epping." Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2017. http://d-nb.info/1145292208/34.

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Costa, Badia Xavier. "Paisatges monàstics. El monacat alt-medieval als comtats catalans (segles IX-X)." Doctoral thesis, Universitat de Barcelona, 2019. http://hdl.handle.net/10803/668151.

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La present tesi doctoral proposa una aproximació innovadora al paisatge monàstic dels comtats catalans dels segles IX i X a través de dos nivells de detall ben diferenciats. En primer lloc, es planteja un estudi de caràcter més general que, partint de la identificació de tots els monestirs que s’hi poden documentar, crea una cartografia completa del fenomen monàstic sobre la qual analitzar la seva distribució espacial, els paral·lels que s’hi detecten i els diversos models de comportament que s’hi poden distingir. En segon lloc, es proposa un estudi minuciós de tres paisatges concrets, tots ells amb una elevada concentració de monestirs, per tal de precisar com aquests centres es relacionaren amb el seu entorn físic i social, transformant-lo de manera evident, però, alhora, veient-se profundament condicionats per ell. Així s’aconsegueix donar una visió holística i integral del fet monàstic que permet entendre millor el perquè de la ràpida proliferació de cenobis al llarg dels segles IX i X, així com el complex rol que jugaren en l’articulació social, política, econòmica i religiosa dels comtats catalans.
This doctoral thesis proposes an innovative approach to the monastic landscape of the Catalan counties in the 9th and 10th centuries using two clearly-differentiated levels of detail. Firstly, a study of a more general nature is undertaken, starting by identifying all the monasteries that can be documented, to create a comprehensive cartography of the monastic phenomenon with which to analyse its spatial distribution, the parallels to be detected and the different models of behaviour that can be distinguished. Secondly, a meticulous study of three specific landscapes with a high concentration of monasteries is undertaken, all with the aim of establishing just how these centres interacted with their physical and social settings, clearly transforming them but at the same time being profoundly conditioned by them. This gives a comprehensive, holistic view of the monastic phenomenon to allow a better understanding of the reasons for the rapid proliferation of sites in the 9th and 10th centuries, as well as the complex role
La presente tesis doctoral propone una aproximación innovadora al paisaje monástico de los condados catalanes de los siglos IX y X mediante dos niveles de detalle bien diferenciados. En primer lugar, se plantea un estudio de carácter más general que, partiendo de la identificación de todos los monasterios que se pueden documentar en ese territorio, crea una cartografía completa del fenómeno monástico sobre la cual analizar su distribución espacial, los paralelos que se detectan en ella y los diversos modelos de comportamiento que se pueden distinguir. En segundo lugar, se propone un estudio más minucioso de tres paisajes concretos, todos ellos con una elevada concentración de monasterios, para precisar cómo estos centros se relacionaron con su entorno físico y social, transformándolo de forma evidente, pero, a su vez, viéndose profundamente condicionados por él. De esta manera se consigue dar una visión holística e integral del monacato que permite entender mejor el porqué de la rápida proliferación de cenobios a lo largo de los siglos IX y X, así como el papel tan complejo que jugaron en la articulación social, política, económica y religiosa de los condados catalanes.
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Becker, Claudia. "Steuerung Alt Entfernen / Re-boot Science." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2013. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-101879.

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Am Ende der Dresden Summer School 2012 haben die Teilnehmerinnen und Teilnehmer eigene Ideen und Impulse zur Zukunft der Vernetzung von Kultur- und Wissenschaftseinrichtungen vorgestellt. Claudia Becker, wissenschaftliche Mitarbeiterin am Vilém Flusser Archiv der Universität der Künste Berlin, ist an neuen Wegen der Wissens- und Kulturvermittlung mit digitalen Technologien interessiert. Wissen, Wissenssammlungen und Wissensordnungen haben sich im Laufe der Jahre verändert, ebenso wie die Wissensproduktion, die Schaffung neuen Wissens, die Wissenschaft selbst. Der Baum des Wissens, „arbor porphyriana“ oder auch „arbor scientiae“ war seit der Antike eine gültige Metapher und das Klassifikationsschema für die Struktur des Wissen, die epistemologische Ordnung. So lehnte auch Denis Diderot die Ordnung seiner berühmten Enzyklopädie an die Baumstruktur des Wissens von Francis Bacon an. Wohl wissend, dass Wissen Macht ist, widmeten Diderot und seine Enzyklopädisten einen großen Teil ihrer Lebenszeit, um das Wissen aus allen Bereichen der Welt zu sammeln und aller Welt zugänglich zu machen. Diderot nutzte somit damals schon die Intelligenz des Schwarmes, seine Enzyklopädie ist ein Produkt des „Crowd Sourcing“, eines kollektiven Verbundes mehrerer Autoren, die gemeinsam an einem Werk schreiben, um Wissen im Namen der Aufklärung den Herrschenden zu entreißen und möglichst vielen zugänglich zu machen. Die Parallelen zu einem der heutigen größten und bedeutendsten Wissensprojekte – der Internet-Enzyklopädie Wikipedia – sind unverkennbar. [...]
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Nüesch, Rebecca [Verfasser], Rainer [Akademischer Betreuer] Alt, Rainer [Gutachter] Alt, and Hubert [Gutachter] Österle. "Hybride Kundeninteraktion : Fallstudien und Gestaltungsempfehlungen aus der Bankindustrie / Rebecca Nüesch ; Gutachter: Rainer Alt, Hubert Österle ; Betreuer: Rainer Alt." Leipzig : Universitätsbibliothek Leipzig, 2016. http://d-nb.info/124069539X/34.

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Marzec, Paulina. "NR2C/F telomeric association drives telomere-genome rearrangements in ALT cells." Thesis, Montpellier 2, 2013. http://www.theses.fr/2013MON20179.

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L'immortalité cellulaire est toujours accompagnée par l'activation du mécanisme de maintien des télomères. Dans la plupart des cancers humains, ce rôle est assuré par l'enzyme télomérase. Cependant, dans 15 % des tumeurs, la télomérase n'est pas activée et les télomères sont maintenus par l'allongement alternatif des télomères (ALT), voie qui implique la recombinaison des télomères. ALT est plus fréquent dans les tumeurs provenant de tissus mésenchymateux (sarcomes), representant 40-60 % des cas, que dans les tumeurs épithéliales. Comprendre le mécanisme ALT est primordial dans les thérapies anti-cancéreuses puisque certaines drogues inhibant la télomérase conduisent souvent à l'activation de l'ALT.La voie ALT est définie par de caractéristiques typiques des télomères. Dans les cellules ALT, les recombinaisons aberrantes d'ADN ne se limitent pas aux télomères puisque les génomes sont souvent fortement réarrangés. Les liens de ces caractéristiques génomiques anormales et la maintenance des télomères atypique ne sont pas connues, mais l'instabilité du génome contribue certainement à la transformation. Notre équipe a montré que les récepteurs orphelins appartenant aux familles NR2C/F ont été trouvés enrichies dans les télomères des lignées cellulaires ALT. Nous avons proposé que ces facteurs puissent être recrutés aux télomères par liaison directe à la séquence répétée GGGTCA, un site de liaison à haute affinité pour ces protéines. Mon projet vise à comprendre (i) leur mécanisme de liaison et (ii) leur rôle, dans le processus d'ALT.Dans cette étude nous montrons que dans les sarcomes primaires humains, les télomères d'ALT sont souvent liés par des récepteurs nucléaires orphelins des sous-familles NR2C/F, en particulier dans les tumeurs au stade avancé. Ceci suggère un rôle actif de ces facteurs dans la progression tumorale ALT. En utilisant la technique de ChIP-sequencing, nous avons montré que les protéines NR2C/F se lient à une répétition directe amplifiée (DR0) aux télomères, et pas de manière significative à toute autre combinaison de motif GGGTCA. Nous avons également analysé la distribution sur tout le génome de NR2C2/F2 et TRF2, une protéine de liaison des télomères, dans des cellules ALT (-) et ALT (+). Bien qu'il n'y ait que peu de sites génomiques liés par TRF2 dans les cellules ALT (-), nous avons été surpris d'identifier plusieurs centaines de régions liées par TRF2 dans les cellules ALT (+). Plus surprenant, la grande majorité de ces régions spécifiques TRF2 ALT chevauche des sites endogènes de NR2C2/F2. Étant donné que ces sites ne contiennent généralement pas les répétitions des télomères, TRF2 est probablement recruté de façon indirecte. Conformément à cette interprétation, nous montrons que les facteurs NR2C/F entrainent un rapprochement des loci et sont responsables du regroupement atypique des télomeres dans ALT. De plus, un sous-ensemble de ces régions génomiques uniques a des additions hétérogènes des séquences télomeriques ALT, suggérant un rôle dans le recrutement des télomères par des protéines NR2C/F mais aussi une fonction de ciblage de recombinaison génomique. Systématiquement, nous trouvons que ces réarrangements des télomères/génome sont situés à proximité des motifs GGGTCA endogènes. Le caryotype spectral des lignées cellulaires ATL montre que les sites télomériques interstitielles sont fréquemment localisés aux niveaux des sites de translocations/réarrangements entre deux ou plusieurs chromosomes, ce qui est également observé dans les données de ChIPseq. Ces résultats suggèrent que les réarrangements entres les télomères et le génome pourraient participer à la formation d'un caryotype complexe ce qui caractérise environ 50% des sarcomes. De plus, l'addition de sites télomériques interstitielles dans le génome est spécifique des cellules ALT et est favorisée par les dommages de l'ADN
Cellular immortality is always accompanied by the activation of telomere maintenance mechanism. In most human cancers this role is fulfilled by the telomerase enzyme. However in 15% of tumors, telomerase is not activated and telomeres are maintained by an Alternative Lengthening of Telomeres (ALT) pathway that involves telomere-telomere recombination. Interestingly ALT is more prevalent in tumors originating from mesenchymal tissues (sarcomas), where it is present in 40-60% of cases, than in epithelial tumors. Understanding ALT maintenance is critical since inhibiting telomerase in tumors leads to the activation of ALT. The ALT pathway is operationally defined by typical telomere hallmarks. In ALT cells, aberrant DNA transactions are not restricted to telomeres since genomes are often highly rearranged. Whether these abnormal genomic features are linked to atypical telomere maintenance is not known, but genome instability is certainly contributing to transformation. We have previously shown that orphan receptors of the NR2C/F families were enriched at telomeres in ALT cell lines. We proposed that these factors could be recruited to telomeres through direct binding to the GGGTCA variant repeat, a high affinity binding site for these proteins. My project is aimed at understanding (i) their mechanism of binding and (ii) their role, if any, in the ALT process.We show that in human primary sarcomas, ALT telomeres are often bound by orphan nuclear receptors of the NR2C/F subfamilies, particularly in more advanced-stage tumors. This suggests an active role for these factors in ALT tumor progression. Using ChIP-sequencing, we show that NR2C/F proteins bind to an amplified direct repeat (DR0) at telomeres, and not significantly to any other GGGTCA motif combination. We also analyzed the genome wide distribution of NR2C2/F2 and TRF2, a telomere binding protein, in ALT(-) and in ALT(+) cells. While there are only few genomic sites bound by TRF2 in ALT(-) cells, we were surprised to identify several hundred regions bound by TRF2 in ALT(+) cells. More surprisingly, the great majority of these ALT specific TRF2 regions overlap with endogenous NR2C2/F2 sites. Since these sites usually do not contain telomere repeats, TRF2 is likely indirectly recruited. Consistent with this interpretation, we show that NR2C/F factors drive locus proximity. Moreover, a subset of these unique genomic regions harbor heterogeneous ALT telomere sequence additions, not only suggesting a telomere recruitment role for NR2C/F proteins but also a recombination targeting function in the genome. Consistently, we find these telomere/genome rearrangements are located close to endogenous GGGTCA motifs. Next, we wanted to evaluate a role of these rearrangements in formation of complex karyotype which characterize approximately 50% of sarcomas. We found by spectral karyotyping that interstitial telomeric sites are frequently located at translocation/ rearrangements sites between two or more chromosomes, which we could also observe in our ChIPseq data. Furthermore, we demonstrate that addition of interstitial telomeric sites to the genome is enhanced by DNA damage and specific for ALT genome. Therefore we conclude that NR2C/F factors target telomere proximity to defined NR2C/F regions which enables telomere-genome rearrangements under DNA damage condition. This contributes not only to efficient telomere recombination, but also it drives further genomic instability at selected NR2C/F sites.We believe we identified a new mechanism of telomere dysfunction potentially driving targeted genome instability and mediated by NR2C/F proteins in ALT cells which probably underlie complexity of sarcomas genome. Understanding the ALT mechanism allows designing NR2C/F-targeted therapies in treatment of ALT tumors and therapies for patients treated with anti-telomerase drugs to prevent ALT appearance
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Alt, Katharina [Verfasser]. "Muster gesellschaftlicher Teilhabe in Mainz / Katharina Alt." Mainz : Universitätsbibliothek Mainz, 2019. http://d-nb.info/119193215X/34.

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Stagno, d'Alcontres Martina. "Deconstructing the ALT-associated promyelocytic leukemia bodies." Thesis, University of Leicester, 2007. http://hdl.handle.net/2381/30783.

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Cells in which Alternative lengthening of telomeres (ALT) is activated have been reported to contain a novel PML nuclear body (PML-NB) known as ALT-associated PML body (APB). These large, donut-shaped nuclear structures are unique to telomerase-negative cells and contain, among others, PML, telomeric DNA, the telomere binding proteins TRF1 and TRF2, MRE11 and NBS1. APBs can be correlated with ALT in that they appear when the telomere length pattern becomes characteristic of ALT positive cells and are not found in telomerase positive cell lines. The exact role of these bodies in telomere maintenance and/or protection is as yet unclear as there have been few studies carried out to determine the functional involvement of the specific components. Our study aims at the identification of the essential components of this structure and, ultimately, at studying the effect of APB alterations on telomere length and genomic stability. We began by determining the percentage of APB-positive cells within our chosen cell lines, and found that there were a higher proportion of APB positive cells than reported in the literature. In order to determine the role of PML in formation of APB, we employed RNA interference oligonucleotides directed against nuclear PML. Interestingly, decreased speckled distribution of MRE11 and TRF1 was observed in PML-depleted cells. In contrast, TRF2 was not affected, and its down-regulation has no clear effect on MRE11 or TRF1 localization. TRF2 silencing results in telomere erosion, thus suggesting that in ALT cells TRF2 regulates telomere length. In addition, TRF2 depletion causes inhibition of colony formation, increased cell size, p21 upregulation and induction of genomic instability. Although in ALT cells active ATM is found at APBs, it is only following TRF2 silencing that the ATM target p53 becomes phosphorylated and activated. This study sheds new light on the role of the APB components PML and TRF2 in ALT cells.
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Johnson, David Ward. "New applications for poly(ethylene-alt-maleic anhydride)." Thesis, Durham University, 2010. http://etheses.dur.ac.uk/886/.

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The project throughout is an iterative development of an understanding of poly(ethylene-alt-maleic anhydride) in terms of both its physical behaviour and chemistry. Chapter one involves a thorough and rigorous characterisation of the polymer, and three principle things are established: the polymer is structurally uniform, reacts in a predictable and quantitative manner, closely analogous to that of small molecule anhydrides. It was with this knowledge that further applications could then be developed. In chapter two the polymer is reacted with a series of different hydrophobic and hydrophilic amines in order to create a series of polymers which allowed the mechanism of ice re-crystallisation to be probed. The findings show that hydrophobic volume appears to play an important role in the mechanism of ice re-crystallisation. This finding allowed for tentative developments to be made on the existing theories surrounding antifreeze glycol protein mimicking polymers. Chapter three involved the reaction of the polymer with varying amounts of hydrophobic and hydrophilic amines in order to induce their self assembly in water. The results demonstrated that when the concentration of hydrophobic amines is low that the polymer aggregates can disperse as a small molecule surfactant might. In addition, to this the hydrophobically modified polymers were able to self assemble intra-molecularly at concentration below the critical aggregation concentration providing an opportunity to create complex encapsulation systems. In addition, it was shown that the introduction of small amounts of hydrophilic amines onto the polymer backbone resulted in aggregates forming wherein the remaining anhydride units form the core, consequently there was shown to be a limited period of time in which further reactions could be undertaken such as cross-linking or the immobilisation of nucleophilic compounds. This presents the opportunity for easily manufactured, multi –purpose carrier nano-aggregates for useful compounds, examples may include dispersing or solubilising a hydrophobic drug. Chapter four deals with the production of hydrophobic surfaces by a trivial and easily replicable mechanism, namely by creating a phase separated polymer film and etching away of one of the polymer phases by dissolving it in a solvent selective for one polymer only. While this did not result in a super-hydrophobic surface the contact angle was observed to increase markedly over analogous flat thin films and presents ample opportunity for future optimisation.
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Books on the topic "And Alt"

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Hannover, Germany) Hanns-Lilje-Forum (2004. Alt gegen Jung?: Jung gegen Alt? Hannover: LVH, Lutherisches Verlagshaus, 2005.

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Breemen, Peter G. Van. Alt werden als geistlicher Weg. Wurzburg: Echter, 2004.

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Akipek, Şebnem. Alt vekâlet. Ankara: Yetkin Yayınları, 2003.

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Malița, Liviu. Alt Rebreanu. Cluj: Editura Cartimpex, 2000.

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Salzmann, Diegfried. Otmar Alt. Dusseldorf: Galerie Vomel, 1989.

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Malița, Liviu. Alt Rebreanu. Cluj: Editura Cartimpex, 2000.

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Willy, Athenstädt, Heuer Carl-Heinz, and Von der Heydt-Museum, eds. Otmar Alt. Heidelberg: Edition Braus, 1994.

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Alt ed. New York: G.P. Putnam's Sons, 2003.

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Brauer, Kai, and Wolfgang Clemens, eds. Zu alt? Wiesbaden: VS Verlag für Sozialwissenschaften, 2010. http://dx.doi.org/10.1007/978-3-531-91941-6.

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Edinger, Claudio. Alt-Havanna. Zürich: Edition Stemmle, 1998.

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Book chapters on the topic "And Alt"

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Kampfer, Angelika. "Zum Buch." In ALT, 11. Wien: Böhlau Verlag, 2013. http://dx.doi.org/10.7767/boehlau.9783205792550.11.

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Bayer, Eleonore. "Gemeinsam das Alter meistern." In ALT, 141–53. Wien: Böhlau Verlag, 2013. http://dx.doi.org/10.7767/boehlau.9783205792550.141.

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Houschyar, Khosrow Siamak, and Marcus Lehnhardt. "Lappenplastik: ALT." In Orthopädie und Unfallchirurgie, 1–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-642-54673-0_56-1.

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Jenal, Corinna. "(Alt-)Industrielandschaften." In RaumFragen: Stadt – Region – Landschaft, 831–41. Wiesbaden: Springer Fachmedien Wiesbaden, 2019. http://dx.doi.org/10.1007/978-3-658-25746-0_67.

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Clemens, Wolfgang. "Ageismus und Altersdiskriminierung auf Arbeitsmärkten – eine Einleitung." In Zu alt?, 7–20. Wiesbaden: VS Verlag für Sozialwissenschaften, 2010. http://dx.doi.org/10.1007/978-3-531-91941-6_1.

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Roßnagel, Christian Stamov. "Was Hänschen nicht lernt…? Von (falschen) Altersstereotypen zum (echten) Lernkompetenzmangel." In Zu alt?, 187–204. Wiesbaden: VS Verlag für Sozialwissenschaften, 2010. http://dx.doi.org/10.1007/978-3-531-91941-6_10.

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Schimkat, Heike. "„Alter ist bei uns in der Tat (k)ein Thema!“? Fallanalyse betrieblicher Einstellungsprozesse." In Zu alt?, 205–30. Wiesbaden: VS Verlag für Sozialwissenschaften, 2010. http://dx.doi.org/10.1007/978-3-531-91941-6_11.

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Tenckhoff, Jürgen. "Alter(n) und Altersakzeptanz in Unternehmen." In Zu alt?, 231–50. Wiesbaden: VS Verlag für Sozialwissenschaften, 2010. http://dx.doi.org/10.1007/978-3-531-91941-6_12.

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Kolland, Franz. "Altersbilder und ihre normative Wirkung im Wandel der Erwerbsarbeit." In Zu alt?, 61–80. Wiesbaden: VS Verlag für Sozialwissenschaften, 2010. http://dx.doi.org/10.1007/978-3-531-91941-6_3.

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Amrhein, Ludwig. "Altersintegration als Rezept gegen Ageism? Anmerkungen zum Konzept der „Age Integration“ von Matilda W. Riley." In Zu alt?, 81–96. Wiesbaden: VS Verlag für Sozialwissenschaften, 2010. http://dx.doi.org/10.1007/978-3-531-91941-6_4.

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Conference papers on the topic "And Alt"

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Nambi, Akshay Uttama, Ishit Mehta, Anurag Ghosh, Vijay Lingam, and Venkata N. Padmanabhan. "ALT." In SenSys '19: The 17th ACM Conference on Embedded Networked Sensor Systems. New York, NY, USA: ACM, 2019. http://dx.doi.org/10.1145/3356250.3360037.

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Han, Xue-bing, Jing-xu Zhang, Jin-yu Zhao, Yong-zhi Zhao, Xiao-xia Wu, and Zhi-chen Wang. "Orientation error analysis of an alt-alt photoelectric telescope." In 4th International Symposium on Advanced Optical Manufacturing and testing Technologies: Large Mirrors and Telescopes, edited by Wenhan Jiang, Roland Geyl, Myung K. Cho, and Fan Wu. SPIE, 2009. http://dx.doi.org/10.1117/12.831451.

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Denning, Tamara, Adam Lerner, Adam Shostack, and Tadayoshi Kohno. "Control-Alt-Hack." In the 2013 ACM SIGSAC conference. New York, New York, USA: ACM Press, 2013. http://dx.doi.org/10.1145/2508859.2516753.

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Akera, Atsushi, and Franz Alt. "Franz Alt interview." In ACM Oral History interviews. New York, New York, USA: ACM Press, 2006. http://dx.doi.org/10.1145/1141880.1141881.

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Wu, Shaomei, Jeffrey Wieland, Omid Farivar, and Julie Schiller. "Automatic Alt-text." In CSCW '17: Computer Supported Cooperative Work and Social Computing. New York, NY, USA: ACM, 2017. http://dx.doi.org/10.1145/2998181.2998364.

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Riva, M., S. Basso, R. Canestrari, P. Conconi, D. Fugazza, M. Ghigo, M. Landoni, et al. "An innovative alt-alt telescope for small observatories and amateur astronomers." In SPIE Astronomical Telescopes + Instrumentation, edited by Larry M. Stepp, Roberto Gilmozzi, and Helen J. Hall. SPIE, 2012. http://dx.doi.org/10.1117/12.924977.

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Denning, Tamara, Tadayoshi Kohno, and Adam Shostack. "Control-Alt-Hack™." In Proceeding of the 44th ACM technical symposium. New York, New York, USA: ACM Press, 2013. http://dx.doi.org/10.1145/2445196.2445408.

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Morstatter, Fred, Yunqiu Shao, Aram Galstyan, and Shanika Karunasekera. "FromAlt-Right to Alt-Rechts." In Companion of the The Web Conference 2018. New York, New York, USA: ACM Press, 2018. http://dx.doi.org/10.1145/3184558.3188733.

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McEwan, Tom, and Ben Weerts. "ALT Text and Basic Accessibility." In Proceedings of HCI 2007 The 21st British HCI Group Annual Conference University of Lancaster, UK. BCS Learning & Development, 2007. http://dx.doi.org/10.14236/ewic/hci2007.64.

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Kotzinian, Aram. "Double spin azimuthal asymmetries ALT and ALL in semi-inclusive DIS." In Proceedings of the 17th International Spin Physics Symposium. AIP, 2007. http://dx.doi.org/10.1063/1.2750826.

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Reports on the topic "And Alt"

1

Thuan Nguyen. ALT-GTS-Stem Fabrication. Office of Scientific and Technical Information (OSTI), September 2008. http://dx.doi.org/10.2172/952458.

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Goebel, D. M., R. W. Conn, G. A. Campbell, W. K. Leung, K. H. Dippel, K. H. Finken, G. H. Wolf, G. J. Thomas, A. E. Pontau, and W. Hsu. ALT-I pump limiter experiments. Office of Scientific and Technical Information (OSTI), September 1987. http://dx.doi.org/10.2172/5929099.

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Conn, R. W., Y. Hirooka, B. LaBombard, R. Moyer, D. M. Goebel, W. K. Leung, R. E. Nygren, et al. PISCES and ALT-II: Juelich PSI papers. Office of Scientific and Technical Information (OSTI), August 1988. http://dx.doi.org/10.2172/6823048.

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Daniels, Vonceil. 2017-04-28_W88 ALT 370 Program Overview(OUO). Office of Scientific and Technical Information (OSTI), April 2017. http://dx.doi.org/10.2172/1365527.

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Fuller, V., D. Farinacci, D. Meyer, and D. Lewis. Locator/ID Separation Protocol Alternative Logical Topology (LISP+ALT). RFC Editor, January 2013. http://dx.doi.org/10.17487/rfc6836.

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Provost, Scott, William G. Kalman, Jane M. Kline, Christopher R. Taylor, and Beverly A. Gable. B-1B Alternative Launch Acceptability Region (ALT LAR) Study. Fort Belvoir, VA: Defense Technical Information Center, June 2000. http://dx.doi.org/10.21236/ada387452.

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Lovejoy, Courtney A., and Titia de Lange. Understanding and Targeting the ALT Pathway in Human Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, September 2013. http://dx.doi.org/10.21236/ada601808.

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Casas, Josep, Josep M. Nolla, and Marc Prat. El jaciment altmedieval de la plaça dels Maristes (Pontós, Alt Empordà). Edicions de la Universitat de Lleida, 2017. http://dx.doi.org/10.21001/rap.2017.27.9.

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Goebel, D. M., G. A. Campbell, R. W. Conn, W. K. Leung, K. H. Dippel, K. H. Finken, G. J. Thomas, and A. E. Pontau. Langmuir probe measurements in the TEXTOR tokamak during ALT-I pump limiter experiments. Office of Scientific and Technical Information (OSTI), April 1986. http://dx.doi.org/10.2172/5224011.

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DEPARTMENT OF THE ARMY WASHINGTON DC. Army ALT & The AMC-Fast Activity: Improving Army Capabilities. January - February 2003. Fort Belvoir, VA: Defense Technical Information Center, February 2003. http://dx.doi.org/10.21236/ada410531.

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