Relevant bibliographies by topics / Androgènes Androgènes Prostate

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Academic literature on the topic 'Androgènes Androgènes Prostate'

Author: Grafiati
Published: 4 June 2021
Last updated: 11 February 2022

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Journal articles on the topic "Androgènes Androgènes Prostate"

1

Balk, Steven P., and Karen E. Knudsen. "AR, the cell cycle, and prostate cancer." Nuclear Receptor Signaling 6, no. 1 (2008): nrs.06001. http://dx.doi.org/10.1621/nrs.06001.

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The androgen receptor (AR) is a critical effector of prostate cancer development and progression. The dependence of this tumor type on AR activity is exploited in treatment of disseminated prostate cancers, wherein ablation of AR function (achieved either through ligand depletion and/or the use of AR antagonists) is the first line of therapeutic intervention. These strategies are initially effective, and induce a mixed response of cell cycle arrest or apoptosis in prostate cancer cells. However, recurrent, incurable tumors ultimately arise as a result of inappropriately restored AR function. B
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You, Zongbing, Ying Dong, Xiangtian Kong, Yi Zhang, Robert L. Vessella, and Jonathan Melamed. "Differential Expression of IL-17RC Isoforms in Androgen-Dependent and Androgen-Independent Prostate Cancers." Neoplasia 9, no. 6 (2007): 464–70. http://dx.doi.org/10.1593/neo.07109.

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Narayanan, Ramesh, Michael L. Mohler, Casey E. Bohl, Duane D. Miller, and James T. Dalton. "Selective androgen receptor modulators in preclinical and clinical development." Nuclear Receptor Signaling 6, no. 1 (2008): nrs.06010. http://dx.doi.org/10.1621/nrs.06010.

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Androgen receptor (AR) plays a critical role in the function of several organs including primary and accessory sexual organs, skeletal muscle, and bone, making it a desirable therapeutic target. Selective androgen receptor modulators (SARMs) bind to the AR and demonstrate osteo- and myo-anabolic activity; however, unlike testosterone and other anabolic steroids, these nonsteroidal agents produce less of a growth effect on prostate and other secondary sexual organs. SARMs provide therapeutic opportunities in a variety of diseases, including muscle wasting associated with burns, cancer, or end-s
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Gong, Yanqing, Dan Wang, Javid A. Dar, et al. "Nuclear Export Signal of Androgen Receptor (NESAR) Regulation of Androgen Receptor Level in Human Prostate Cell Lines via Ubiquitination and Proteasome-Dependent Degradation." Endocrinology 153, no. 12 (2012): 5716–25. http://dx.doi.org/10.1210/en.2012-1841.

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Abstract Androgen receptor (AR) plays a key role in prostate development and carcinogenesis. Increased expression and/or stability of AR is associated with sensitization of prostate cancer cells to low levels of androgens, leading to castration resistance. Hence, understanding the mechanisms regulating AR protein stability is clinically relevant and may lead to new approaches to prevent and/or treat prostate cancer. Using fluorescence microscopy, Western blot, and pulse chase assay, we showed that nuclear export signal (NES)AR, a nuclear export signal in the ligand binding domain (LBD) of AR,
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Abbasova, Daria V., Svetlana B. Polikarpova, Nikolai A. Kozlov, Madina P. Baranova, Irina P. Kovalenko, and Elena I. Ignatova. "Neuroendocrine carcinoma of the prostate (review of the literature)." Journal of Modern Oncology 21, no. 3 (2019): 52–55. http://dx.doi.org/10.26442/18151434.2019.3.190673.

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Neuroendocrine neoplasia (NEC) of the prostate gland is a rather rare extrapulmonary neuroendocrine carcinoma and makes up only 0.5 to 1% of all malignant neoplasms of this localization. NEC of the prostate gland is a tumor of epithelial origin, histologically and immunohistochemically identical to analogues in the lungs and digestive system. When stained with hemotoxylin-eosin, neuroendocrine cells cannot always be visualized; they are best recognized by the immunohistochemical method of investigation using specific markers. Currently, a number of neuroendocrine markers are used, the expressi
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Røe, Kathrine, Therese Seierstad, Alexandr Kristian, et al. "Longitudinal Magnetic Resonance Imaging-Based Assessment of Vascular Changes and Radiation Response in Androgen-Sensitive Prostate Carcinoma Xenografts under Androgen-Exposed and Androgen-Deprived Conditions." Neoplasia 12, no. 10 (2010): 818–25. http://dx.doi.org/10.1593/neo.10484.

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Beliakoff, Jason, and Zijie Sun. "Zimp7 and Zimp10, two novel PIAS-like proteins, function as androgen receptor coregulators." Nuclear Receptor Signaling 4, no. 1 (2006): nrs.04017. http://dx.doi.org/10.1621/nrs.04017.

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The androgen receptor (AR) plays a critical role in male sexual development and in normal and malignant prostate cell growth and survival. It has been shown that AR transcriptional activation is regulated through interactions with a variety of transcriptional co-regulators. The Protein Inhibitors of Activated STATs (PIAS) are transcriptional co-regulators, and have been shown to modulate AR-mediated transcription. In this brief, we summarize our recent studies on two novel PIAS-like proteins, Zimp7 and Zimp10. Particularly, we address the functional interactions between the AR and these two pr
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Oram, Shane W., Junkui Ai, Gina M. Pagani, et al. "Expression, Function of the Human Androgen-Responsive Gene AD11 in Prostate Cancer." Neoplasia 9, no. 8 (2007): 643–51. http://dx.doi.org/10.1593/neo.07415.

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Gernone, Angela, Senia Trabucco, Eliano Cascardi, Leonardo Resta, Franco Silvestris, and Anna Napoli. "Expression of androgen receptor, somatostatin receptor subtypes, aurora kinase A, and interleukin-6 in prostate cancer before androgen ablation." Journal of Clinical Oncology 35, no. 15_suppl (2017): e16508-e16508. http://dx.doi.org/10.1200/jco.2017.35.15_suppl.e16508.

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e16508 Background: Neuroendocrine differentiation (NED) in prostate cancer (PC) can be detected by immunohistochemistry as single cells in conventional adenocarcinoma. NEPC is a poor-recognized late presentation of hormone refractory subtype of PC AR-negative. NEPC correlates with poor prognosis, tumor progression during androgen-deprivation therapy and frequent visceral metastases. Aurora kinase A (AURKA) and Interleukin-6 (IL-6) cooperate to induce NED. The aim of this study was to correlate the expression of somatostatin receptor (SSTR) 1- 2- 3- 4- 5 subtypes, AURKA and IL-6 in primary PC w
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Pernicová, Zuzana, Eva Slabáková, Gvantsa Kharaishvili, et al. "Androgen Depletion Induces Senescence in Prostate Cancer Cells through Down-regulation of Skp2." Neoplasia 13, no. 6 (2011): 526—IN13. http://dx.doi.org/10.1593/neo.11182.

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Books on the topic "Androgènes Androgènes Prostate"

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Alves, Ines Teles, Jan Trapman, and Guido Jenster. Molecular biology of prostate cancer. Edited by James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0059.

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Prostate cancer is a heterogeneous disease that arises through the acquisition of key malignant hallmarks. At the molecular level, prostate tumours are dependent upon the androgen receptor pathway, which affects cell function, growth, and behaviour through downstream androgen-regulated genes. Prostate cancer requires this activity and manipulates the AR pathway to maintain signalling. For example, mutation of the AR (to bind ligands other than androgens) or amplification/duplication of the AR allows signalling to continue in the absence of testosterone. Around 50% of prostate cancers have a ge
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Mukherji, Deborah, Aurelius Omlin, Carmel Pezaro, and Johann De Bono. Novel therapies and emerging strategies for the treatment of patients with castration-resistant prostate cancer. Edited by James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0069.

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Castration-resistant prostate cancer (CRPC) represents a final stage of this malignancy for many men and is defined as the progression of prostate cancer despite castrate levels of testosterone. CRPC may present as a rising PSA, the development of new metastases, or worsening of known metastases. Recent advances have resulted in five new treatments for CRPC: the immunotherapy sipuleucel-T; the cytotoxic cabazitaxel; the androgen biosynthesis inhibitor abiraterone acetate; the radioisotope radium-223; and the antiandrogen enzalutamide. These have all improved overall survival in randomized phas
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Conference papers on the topic "Androgènes Androgènes Prostate"

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Asim, Mohammad, Charlie Massie, Anne Warren, et al. "Abstract LB-003: Androgen-regulated proteome reveals a therapeutically relevant androgen receptor coactivator target in prostate cancer." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-lb-003.

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Du, Panpan, Na Liu, Yuanyuan Liu, Tao Yue, Huayan Pu, and Shaorong Xie. "High-throughput Mechanical Phenotyping of Androgen-Sensitive and Nonsensitive Prostate Cancer Cells Using a Real-time Deformability Cytometry." In 2019 IEEE 14th International Conference on Nano/Micro Engineered and Molecular Systems (NEMS). IEEE, 2019. http://dx.doi.org/10.1109/nems.2019.8915635.

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Takezawa, Yuta, Atsushi Mizokami, Kazuaki Machioka, et al. "Abstract 1581: Crosstalk of androgen sensitive prostate cancer cells and insensitive prostate cancer cells." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-1581.

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Khurana, Namrata, Sudha Talwar, Partha Chandra, Asim B. Abdel-Mageed, Debasis Mondal, and Suresh Sikka. "Abstract 2990: Sulforaphane enhances the anti-cancer efficacy of anti-androgens in prostate cancer cells (LNCaP and C4-2B) by increasing androgen receptor (AR) degradation." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-2990.

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Watson, Philip A., Minna D. Balbas, Zeda Zhang, et al. "Abstract 238: Androgen insensitivity syndrome germline loss-of-function mutations in the androgen receptor that acquire somatic gain-of-function in prostate cancer." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-238.

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Ferrari, Anna C., Hatem Sabaawy, Mark Stein, David Foran, Ying Chen, and Srinivasan Yegnasubramanian. "Abstract 2415: Genome-wide alterations in gene expression of prostate cancer (PC) cells surviving neo-adjuvant androgen deprivation therapy." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-2415.

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7

Yenki, Parvin, Hans Adomat, Chi Wing Cheng, and Christopher Ong. "Abstract 1164: Semaphorin 3C promotes de novo steroidogenesis in androgen-deprived prostate cancer cells." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-1164.

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8

Liao, Chun-Peng, Leng-Ying Chen, Andrea Luethy, Youngsoo Kim, Robert MacLeod, and Mitchell Gross. "Abstract LB-268: Androgen signaling in cancer-associated fibroblasts contributes to progression of prostate cancer." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-lb-268.

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9

Gao, Shuai, Dong Han, Yanfei Gao, et al. "Abstract 1976: Androgen receptor activity is reprogrammed by lysine-specific demethylase 1 in prostate cancer." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-1976.

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Grant, Delores J., Lauren Howard, Emily Wiggins, et al. "Abstract 813: The association of androgen metabolizing enzymes and prostate cancer in a multiethnic study." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-813.

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Reports on the topic "Androgènes Androgènes Prostate"

1

Hu, Guo-fu, Hiroko Kishikawa, and Norie Yoshioka. A New Concept for Androgen Receptor-Independent Growth of Prostate Cancer. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada462435.

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Bhattacharyya, Rumi S. Selective Androgen Receptor Down-Regulators (SARDs): A New Prostate Cancer Therapy. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada475119.

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Hu, Guo-fu, Hiroko Kishikawa, and Norie Yoshioka. A New Concept for Androgen Receptor-Independent Growth of Prostate Cancer. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada477379.

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Zhang, Haitao. New Strategy for Prostate Cancer Prevention Based on Selenium Suppression of Androgen Receptor Signaling. Defense Technical Information Center, 2010. http://dx.doi.org/10.21236/ada534266.

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Zhang, Haitao. New Strategy for Prostate Cancer Prevention Based on Selenium Suppression of Androgen Receptor Signaling. Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada511818.

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Zhang, Haitao. New Strategy for Prostate Cancer Prevention Based on Selenium Suppression of Androgen Receptor Signaling. Defense Technical Information Center, 2010. http://dx.doi.org/10.21236/ada525643.

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Zhang, Haitao. New Strategy for Prostate Cancer Prevention Based on Selenium Suppression of Androgen Receptor Signaling. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada463812.

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Zhang, Haitao. New Strategy for Prostate Cancer Prevention Based on Selenium Suppression of Androgen Receptor Signaling. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada475534.

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9

Sadar, Marianne D. Characterization of a New In Vivo Prostate Tumor Model that Progresses to Androgen-Independence and its Application in Determining Changes in Gene Expression. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada413293.

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