Relevant bibliographies by topics / Androgen deprivation

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Androgen deprivation'

Author: Grafiati
Published: 4 June 2021
Last updated: 9 March 2023

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Journal articles on the topic "Androgen deprivation"

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Hoon Lee, Jung, Haibiao Gong, Shaheen Khadem, et al. "Androgen Deprivation by Activating the Liver X Receptor." Endocrinology 149, no. 8 (2008): 3778–88. http://dx.doi.org/10.1210/en.2007-1605.

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Prostate cancer is the most commonly diagnosed and the second leading cause of cancer death in men. The androgens-androgen receptor signaling plays an important role in normal prostate development, as well as in prostatic diseases, such as benign hyperplasia and prostate cancer. Accordingly, androgen ablation has been the most effective endocrine therapy for hormone-dependent prostate cancer. Here, we report a novel nuclear receptor-mediated mechanism of androgen deprivation. Genetic or pharmacological activation of the liver X receptor (LXR) in vivo lowered androgenic activity by inducing the
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Rozhivanov, Roman V., Elena N. Andreeva, Galina A. Melnichenko, and Natalya G. Mokrysheva. "Androgens and Antiandrogens influence on COVID-19 disease in men." Problems of Endocrinology 66, no. 4 (2020): 77–81. http://dx.doi.org/10.14341/probl12500.

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The WHO has declared a SARS-CoV-2 pandemic. During a pandemic, the researches aimed at finding the new treatments for SARS-CoV-2 become relevant. The review focuses on studies of androgens and antiandrogens in this disease. Since the beginning of the COVID-19 epidemic, it has been noted that men have more severe forms of infection and higher mortality. The main cause of both the severity of the disease and the high mortality of men from COVID-19 are associated with androgens. It was found that patients receiving androgen deprivation are less likely to become infected and easily tolerate COVID-
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Gamat, Melissa, and Douglas G. McNeel. "Androgen deprivation and immunotherapy for the treatment of prostate cancer." Endocrine-Related Cancer 24, no. 12 (2017): T297—T310. http://dx.doi.org/10.1530/erc-17-0145.

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Prostate cancer is the most common newly diagnosed malignancy in men, and the second most common cause of cancer-related death in the United States. The primary treatment for recurrent prostate cancer is androgen deprivation, and this therapy is typically continued lifelong for patients with metastatic prostate cancer. Androgens and androgen deprivation have profound effects on the immune system, a finding that has become more appreciated in an era where immune-based treatments for cancer are being increasingly explored. Preclinical studies suggest that androgen deprivation could potentially p
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Kamalov, A. A., D. A. Ohobotov, O. Yu Nesterova, A. A. Strigunov, and A. S. Tivtikyan. "Androgen deprivation therapy and hormonal status in men with COVID-19." Urology Herald 10, no. 4 (2022): 141–54. http://dx.doi.org/10.21886/2308-6424-2022-10-4-141-154.

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Severe course of COVID-19 among men compared to the female led to a detailed study of the hormonal status of men with COVID-19. The earliest works about this focused on the incidence and severity of COVID-19 depending on the intake of androgen deprivation therapy. At the same time, different classes of androgen deprivation therapy have different effects on androgen concentration that was not always considered in the analysis. In this regard, we conducted a review of the available literature data with a targeted study of works that included androgen deprivation therapy with a unidirectional eff
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McCarty, Mark F., Jalal Hejazi, and Reza Rastmanesh. "Beyond Androgen Deprivation." Integrative Cancer Therapies 13, no. 5 (2014): 386–95. http://dx.doi.org/10.1177/1534735414534728.

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Mendiratta, Prateek, and Jorge Garcia. "Androgen Deprivation Fortified." International Journal of Radiation Oncology*Biology*Physics 100, no. 5 (2018): 1098. http://dx.doi.org/10.1016/j.ijrobp.2018.01.103.

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Dawson, Nancy A. "Intermittent androgen deprivation." Current Oncology Reports 2, no. 5 (2000): 409–16. http://dx.doi.org/10.1007/s11912-000-0060-6.

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Hussain, Maha, and Mario Eisenberger. "Intermittent Androgen Deprivation." JAMA Oncology 2, no. 12 (2016): 1533. http://dx.doi.org/10.1001/jamaoncol.2016.2650.

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Chen, Jia-Feng, Pei-Wen Lin, Yi-Ru Tsai, Yi-Chien Yang, and Hong-Yo Kang. "Androgens and Androgen Receptor Actions on Bone Health and Disease: From Androgen Deficiency to Androgen Therapy." Cells 8, no. 11 (2019): 1318. http://dx.doi.org/10.3390/cells8111318.

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Androgens are not only essential for bone development but for the maintenance of bone mass. Therefore, conditions with androgen deficiency, such as male hypogonadism, androgen-insensitive syndromes, and prostate cancer with androgen deprivation therapy are strongly associated with bone loss and increased fracture risk. Here we summarize the skeletal effects of androgens—androgen receptors (AR) actions based on in vitro and in vivo studies from animals and humans, and discuss bone loss due to androgens/AR deficiency to clarify the molecular basis for the anabolic action of androgens and AR in b
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Sountoulides, Petros, and Thomas Rountos. "Adverse Effects of Androgen Deprivation Therapy for Prostate Cancer: Prevention and Management." ISRN Urology 2013 (July 25, 2013): 1–8. http://dx.doi.org/10.1155/2013/240108.

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The prostate is an androgen-dependent organ. The increase, growth, homeostasis, and function of the prostate largely depend upon the intraprostatic and serum concentrations of androgens. Therefore, androgens are essential for the physiologic growth of prostatic epithelium. Prostate cancer, the second leading cause of death for men, is also androgen dependent, and androgen suppression is the mainstay of treatment for advanced and metastatic disease. In the state of metastatic disease, androgen suppression is a palliative treatment leading to a median progression-free survival of 18–20 months an
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Dissertations / Theses on the topic "Androgen deprivation"

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Lorch, Robert A. "MicroRNA regulation of prostate cancer desensitization to androgen receptor antagonist drugs during androgen deprivation therapy." Honors in the Major Thesis, University of Central Florida, 2011. http://digital.library.ucf.edu/cdm/ref/collection/ETH/id/462.

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The current standard treatment of prostate cancer by androgen deprivation therapy involves using drugs such as bicalutamide (Casodex) to antagonistically block androgen receptors that are normally present within prostate cells. Usually, the therapy is successful in the short run at limiting the growth of prostate cancer. However, in virtually all cases tumors begin to grow aggressively again after several months of treatment and new therapies must be started. The mechanism by which these prostate cells transform from androgen sensitive to androgen independent and anti-androgen resistant is unc
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Galvão, Daniel Abido. "Resistance exercise in men receiving androgen deprivation therapy for prostate cancer." Connect to thesis, 2006. http://portal.ecu.edu.au/adt-public/adt-ECU2006.0046.html.

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Gilbert, Stephen E. "Cardiovascular health in men on androgen deprivation therapy for prostate cancer." Thesis, Sheffield Hallam University, 2013. http://shura.shu.ac.uk/20685/.

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Androgen deprivation therapy (ADT) is a cornerstone treatment option for men with metastatic or locally advanced prostate cancer, however, treatment with ADT has been associated with increased incidence of adverse cardiovascular events. Strategies to investigate and monitor cardiovascular risk, as well as to reduce such treatment-related morbidity are urgently required in this population. Study 1 of this thesis investigated the differences in endothelial function between men with advanced prostate cancer treated with ADT and matched controls using a case-control design. Flow-mediated dilatatio
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Giannantoni-Ibelli, Gina. "Association Between Androgen Deprivation Therapy for Prostate Cancer and Alzheimer's Disease." ScholarWorks, 2018. https://scholarworks.waldenu.edu/dissertations/6206.

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Alzheimer's disease (AD) is the most common progressive, neurodegenerative disease and form of dementia. The hallmarks of AD are extracellular accumulation of amyloid beta protein, resulting in neuritic, senile plaques and intracellular accumulation of tau protein. AD mainly arises from imbalance of amyloid beta protein production and its clearance in the brain. Testosterone modulates production of amyloid beta protein by decreasing its accumulation. Prostate cancer remains a substantial public health challenge in the United States. While androgen deprivation therapy (ADT) is an effective trea
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Galvao, Daniel A. "Resistance exercise in men receiving androgen deprivation therapy for prostate cancer." Thesis, Edith Cowan University, Research Online, Perth, Western Australia, 2006. https://ro.ecu.edu.au/theses/64.

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This thesis encompasses two literature reviews (chapter 2 & 3) and two experimental chapters (4 and 5) examining the available literature on exercise and cancer, resistance training and its anabolic responses in older men and women, the side effects of Androgen Deprivation Therapy (ADT) for prostate cancer and finally, the role of resistance exercise as a clinical intervention to counteract such changes as an adjuvant therapy.
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Joshee, Paras. "Cognition in prostate cancer patients before undergoing androgen deprivation therapy and elderly males." Thesis, University of Birmingham, 2018. http://etheses.bham.ac.uk//id/eprint/8853/.

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Deleterious cognitive effects of testosterone deprivation in prostate cancer (PC) patients undergoing androgen deprivation therapy have been reported. However, due to methodological limitations of past research, there is mixed consensus of the cognitive domains affected. The current study therefore aimed to assess cognition before ADT through a comprehensive battery of cognitive and neuroimaging investigations which previous undertakings have lacked. A cross sectional study of 30 ageing PC patients before ADT and 29 age and intelligence matched healthy controls underwent neuropsychological and
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Sarkar, Phoebe Lorraine. "Characterizing the role of insulin signalling in advanced prostate cancer." Thesis, Queensland University of Technology, 2017. https://eprints.qut.edu.au/110524/2/Phoebe_Lorraine_Sarkar_Thesis.pdf.

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The majority of prostate cancer patients receiving hormone therapy become insulin resistant and studies show this metabolic dysfunction is associated with more rapid treatment failure, yet the effect of insulin on prostate cancer is not fully known. This thesis discovered the mechanisms by which insulin increases the propensity of prostate cancer cells to migrate which is required for cancer cells to disseminate and metastasise, giving a possible explanation for the more aggressive disease in prostate cancer patients with insulin resistance. The results provide a strong rationale for specifica
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Mennen-Winchell, Lori J. "Self-reported exercise and risk of osteoporosis in prostate cancer patients receiving androgen deprivation therapy." Thesis, Loma Linda University, 2015. http://pqdtopen.proquest.com/#viewpdf?dispub=3721215.

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<p> Prostate cancer is stimulated to grow in response to testosterone. Androgen deprivation therapy (ADT) leads to chemical castration and suppression of prostate cancer cell production. Testosterone levels less then 300ng/ml decreases bone mineral density and could result in osteoporosis. Studies have shown that during the first year of ADT, fracture risk, mainly in hips and spine increases about 50%. In men, 40% of hip fractures result in death. Exercise may reduce the risk of osteoporosis and thus contribute to the prevention of hip and other fractures. There is limited data regarding whet
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Shah, Syed Imran Ali. "Assessment of systemic effects of sex hormone alterations in androgen deprivation therapy for prostate cancer." Thesis, Imperial College London, 2015. http://hdl.handle.net/10044/1/42883.

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Sex hormonal variations cause bodily changes reflecting their systemic functions. This project was designed to define the influence of sex hormones on systemic function in men with prostate cancer (PC) receiving contemporary androgen deprivation therapy (ADT) with luteinising hormone-releasing hormone agonists (LHRHa). LHRHa induced suppression of sex hormones results in multiple serious toxicities of which the cognitive, skeletal and psychosocial aspects were addressed in this project. Acute castration appears to cause cognitive problems in some men with PC. In order to identify the neuropath
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Wiens, Kristin Patricia. "Dietary risk factors for bone loss among men undergoing androgen deprivation therapy for the treatment of prostate cancer." Thesis, University of British Columbia, 2009. http://hdl.handle.net/2429/21737.

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Androgen deprivation therapy (ADT) is the preferred mode of treatment for patients with recurrence of prostate cancer (PC) following definitive treatment and locally advanced disease. With more men commencing ADT earlier in the treatment trajectory and for longer duration, the side effects of ADT are becoming more prevalent and of greater concern to clinicians. ADT can have serious adverse effects on bone mineral density (BMD) and metabolism, leading to the development of osteopenia or osteoporosis. This study was a cross-sectional investigation of dietary risk factors for bone loss, particula
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Books on the topic "Androgen deprivation"

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Saborowski, Karl-Johannes. Konservative Therapie mit Cyproteronacetat und Estradiolundecylat beim Fortgeschrittenen Prostatacarcinom: Eine 5-Jahres-Studie. Ruhr-University Bochum (Ruhr-Universität Bochum), 1987.

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Wilke, Derek R. Preferences for short- versus long term androgen deprivation in prostate cancer survivors. 2005.

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Wilke, Derek R. Preferences for short- versus long-term androgen deprivation in prostate cancer survivors. 2005.

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Walker, Lauren M. (Lauren Marie), 1985- author and Robinson, John W. (John Wellesley), 1956- author, eds. Androgen deprivation therapy: An essential guide for prostate cancer patients and their loved ones. 2014.

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PhD, Richard J. Wassersug, Lauren Walker PhD, and John Robinson PhD R Psych. Androgen Deprivation Therapy: An Essential Guide for Prostate Cancer Patients and Their Loved Ones. Demos Health, 2018.

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Hodgkiss, Andrew. Psychiatric consequences of cancer treatments: hormone and cytokine treatments. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198759911.003.0007.

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The antidepressant and neuroprotective effects of oestradiol are described. Psychiatric consequences of oophorectomy, and treatment with tamoxifen and aromatase inhibitors, are then discussed. Androgen-deprivation therapy has temporary effects on cognitive function and mood that reflect the distribution of androgen receptors in the brain. The rapid-onset adverse psychiatric effects of high-dose glucocorticoids are presented (including ‘steroid psychosis’) and a novel, non-genomic molecular mechanism highlighted. In contrast, the depressive effect of chronic glucocorticoid use is then considere
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Book chapters on the topic "Androgen deprivation"

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Sharifi, Nima. "Androgen Deprivation Therapy." In Drug Management of Prostate Cancer. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-60327-829-4_9.

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Reyes, Charlene, Carla Groshel, and Robert Given. "Androgen Deprivation Therapy." In Chemotherapy and Immunotherapy in Urologic Oncology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-52021-2_7.

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Rohani, Atooshe. "Androgen Deprivation Therapy Cardiotoxicity." In Clinical Cases in Cardiology. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-71155-9_11.

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Morgans, Alicia K., and Matthew R. Smith. "Pitfalls of Androgen Deprivation Therapy." In Management of Prostate Cancer. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-60761-259-9_23.

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Altwein, J. E. "Partial Versus Complete Androgen Deprivation." In Progress in Diagnostics and Therapy of Prostatic Cancer. Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-79947-1_6.

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Pagliarulo, Vincenzo. "Androgen Deprivation Therapy for Prostate Cancer." In Molecular & Diagnostic Imaging in Prostate Cancer. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-99286-0_1.

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Di Silverio, F. "Adjuvant Androgen Deprivation: Basis and Justification." In Incidental Carcinoma of the Prostate. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-76129-4_35.

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Houts, Frederick W., Inna Taller, Douglas E. Tucker, and Fred S. Berlin. "Androgen Deprivation Treatment of Sexual Behavior." In Sexual Dysfunction: Beyond the Brain-Body Connection. KARGER, 2011. http://dx.doi.org/10.1159/000330196.

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Hammerer, Peter, and Lukas Manka. "Androgen Deprivation Therapy for Advanced Prostate Cancer." In Urologic Oncology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-42603-7_77-1.

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Hammerer, Peter, and Lukas Manka. "Androgen Deprivation Therapy for Advanced Prostate Cancer." In Urologic Oncology. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-42623-5_77.

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Conference papers on the topic "Androgen deprivation"

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Shao, Joni Y., Dirk F. Moore, Weichung Shih, Yong Lin, and Grace L. Lu-Yao. "Abstract B11: Risk of fracture and androgen deprivation therapy." In Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Nov 7-10, 2010; Philadelphia, PA. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1940-6207.prev-10-b11.

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Ottman, Richard J., Robert Lorch, Camha Nguyen, and Ratna Chakrabarti. "Abstract 1950: Signature MicroRNAs involved In transition of androgen sensitive prostate cancer cells to androgen-insensitive ones during androgen deprivation therapy." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-1950.

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Santer, Frédéric R., Holger H. H. Erb, Su Jung Oh, et al. "Abstract 3: Mechanistic rationale for MCL1 inhibition during androgen deprivation therapy." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-3.

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Gatalica, Zoran, Phillip Stafford, Elma Contreras, Jeff Swensen, and Rebecca Feldman. "Abstract A035: Therapeutic targets in androgen deprivation therapy-resistant prostatic carcinoma." In Abstracts: AACR-NCI-EORTC International Conference on Molecular Targets and Cancer Therapeutics; October 26-30, 2019; Boston, MA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1535-7163.targ-19-a035.

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Botta, Ginevra, Judit Jane-Valbuena, Terence Wong, John Doench, and Levi A. Garraway. "Abstract 3316: Characterizing mechanisms of resistance to androgen deprivation in prostate cancer." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-3316.

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Nead, Kevin Thomas, Greg Gaskin, Cariad Chester, et al. "Abstract 4307: Influence of age on androgen deprivation therapy-associated Alzheimer's disease." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-4307.

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Clark, Ashlee K., Mitchell Lawrence, Hieu Nim, et al. "Abstract 5234: Transcriptome profiling of single prostate cancer cells following androgen deprivation." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-5234.

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Yin, Yi, and Philip E. Thorpe. "Abstract 621: Targeting phosphatidylserine to improve androgen deprivation therapy of prostate cancer." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-621.

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Brady, Renee, and Heiko Enderling. "Abstract B04: Using PSA dynamics to forecast individual responses to intermittent androgen deprivation." In Abstracts: AACR Special Conference on Advances in Liquid Biopsies; January 13-16, 2020; Miami, FL. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1557-3265.liqbiop20-b04.

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Sha, Kai, Lingxiang Jiang, Chawnshang Chang, Dean Tang, Kent L. Nastiuk, and John J. Krolewski. "Abstract B043: Cancer stem cell selection drives adverse response to androgen-deprivation therapy." In Abstracts: AACR Special Conference: Prostate Cancer: Advances in Basic, Translational, and Clinical Research; December 2-5, 2017; Orlando, Florida. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.prca2017-b043.

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Reports on the topic "Androgen deprivation"

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Chiechi, Antonella. Muscle Dysfunction in Androgen Deprivation: Role of Ryanodine Receptor. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada615939.

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Botta, Ginevra. Characterizing Mechanisms of Resistance to Androgen Deprivation in Prostate Cancer. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada610505.

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Janowsky, Jeri. Markers and Time Course of Neurodegenerative Risk With Androgen Deprivation Therapy. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada548985.

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Hsieh, Chen-Lin. Androgen Deprivation Enhances PLZF-Repressed Cistrome that Promotes the Castration-Resistant Phenotype. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada613273.

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Padalecki, Susan S. Prostate Cancer Metastases to Bone: Role of High Bone Turnover Induced by Androgen Deprivation. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada407345.

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Chen, Jiuzhou, Yong Xin, Yan Yuan, Miao Fang, and Youqi Zhu. Androgen deprivation therapy and radiotherapy in intermediate-risk prostate cancer:A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.8.0095.

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Pollack, Alan. Sensitization of Prostate Cancer Cells to Androgen Deprivation and Radiation via Manipulation of the MDM2 Pathway. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada426171.

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Kohrt, Wendy, L. M. Glode, Robert S. Schwartz, and Daniel W. Barry. Exercise to Counteract Loss of Bone and Muscle During Androgen Deprivation Therapy in Men with Prostate Cancer. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada484506.

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Johnson, Lisa. Modulation of the Immune Response to Androgen Deprivation and Radiation Therapy for the Treatment of Prostate Cancer. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada607786.

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Kohrt, Wendy M., L. M. Glode, Robert S. Schwartz, and Daniel W. Barry. Exercise to Counteract Loss of Bone and Muscle During Androgen Deprivation Therapy in Men with Prostate Cancer. Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada509743.

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