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Books on the topic 'Anemia syndrome'

Author: Grafiati

Published: 28 May 2022

Last updated: 30 July 2025

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1

Uzoegwu, Peter Nzeemdu. Families' guide against sickle cell syndrome. Sickle Cell Laboratory, University of Nigeria, 1995.

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2

Shahidi, Nasrollah T., ed. Aplastic Anemia and Other Bone Marrow Failure Syndromes. Springer New York, 1990. http://dx.doi.org/10.1007/978-1-4612-3254-4.

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3

T, Shahidi Nasrollah, ed. Aplastic anemia and other bone marrow failure syndromes. Springer-Verlag, 1990.

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4

Cherry, Anne Blanche Cresswell. Reprogramming Pediatric Genetic Disorders: Pearson Syndrome, Ring 14 Syndrome, and Fanconi Anemia. 2014.

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5

Sybert, Virginia P. Disorders of Pigmentation. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780195397666.003.0004.

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Hyperpigmentation – Carney Complex – Dowling-Degos Disease – Dyskeratosis Congenita – Fanconi Anemia – Hemochromatosis – Incontinentia Pigmenti – LEOPARD Syndrome – Linear and Whorled Nevoid Hypermelanosis – McCune-Albright Syndrome – Naegeli Syndrome – Neurofibromatosis – Nevus Phakomatosis Pigmentovascularis – Peutz-Jeghers Syndrome – Universal Melanosis – Hypopigmentation – Albinisms – Albinism with Deafness – Hermansky-Pudlak Syndrome – Oculocutaneous Albinism Tyrosinase Negative – Oculocutaneous Albinism Tyrosinase Positive – Yellow Mutant Albinism – Cross Syndrome – Hypomelanosis of Ito

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6

Sybert, Virginia P. Disorders of Pigmentation. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190276478.003.0004.

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Chapter 4 covers Hyperpigmentation (including Carney Complex, Dowling-Degos Disease, Dyskeratosis Congenita, Fanconi Anemia, H Syndrome, Hemochromatosis, Incontinentia Pigmenti, LEOPARD Syndrome, Linear and Whorled Nevoid Hypermelanosis, McCune-Albright Syndrome, Naegeli Syndrome, Neurofibromatosis, Nevus Phakomatosis Pigmentovascularis, Peutz-Jeghers Syndrome, and Universal Melanosis) and Hypopigmentation ( Albinisms, Albinism with Deafness, Hermansky-Pudlak Syndrome, Oculocutaneous Albinism Tyrosinase Negative, Oculocutaneous Albinism Tyrosinase Positive, Yellow Mutant Albinism, Cross Syndro

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7

Fridbinstons, Pit Alex. Causes of Thrombocytopenia: Enlarged Spleen, Anemia, Leukemia, Pregnancy, Heavy Alcohol Consumption, Viral Infections, Chemotherapy Medications, Hemolytic Uremic Syndrome, Thrombotic Thrombocytopenic Purpura. Independently Published, 2021.

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8

Publications, ICON Health. HELLP Syndrome - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References. ICON Health Publications, 2004.

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9

Ng, Dominic S. Familial Lecithin Cholesterol Acyl Transferase Deficiency Syndromes. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0034.

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Lecithin cholesterol ester transferase (LCAT) is the sole enzyme in the circulation that mediates the esterification of free cholesterol (FC) to cholesterol ester (CE) in lipoproteins. Mutations in the LCAT gene result in one of two clinical syndromes: complete LCAT deficiency syndrome, and “fish eye disease.” The former is characterized by a broad spectrum of clinical features, including profound high-density lipoprotein (HDL) deficiency, hypertriglyceridemia, corneal opacities, anemia, neuropathies, and nephropathy. In contrast, fish eye disease patients develop severe HDL deficiency and sev

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10

Ng, Ann, and Erin S. Williams. Sickle Cell Disease. Edited by Erin S. Williams, Olutoyin A. Olutoye, Catherine P. Seipel, and Titilopemi A. O. Aina. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190678333.003.0033.

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Sickle cell anemia (sickle cell disease) is a common hemoglobinopathy with anywhere from 90,000 to 100,000 Americans affected. This chronic condition has a predominance in populations of African descent, occurring in approximately 1 out of 365 African American births, compared to 1 out of 16,300 Hispanic births. The sickle cell trait can be detected in 1 of 13 African American births. One of the most common complications associated with sickle cell anemia, vaso-occlusive crises by sickled cells, results in severe pain. Other issues associated with this condition include acute chest syndrome, l

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11

Dr. Susan Lark's Chronic fatigue self help book: Effective solutions for conditions associated with chronic fatigue syndrome, candida, allergies, PMS, menopause, anemia, low thyroid, and depression. Celestial Arts, 1995.

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12

Shaibani, Aziz. Fatigability. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190661304.003.0025.

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Undue fatigability is common in neuromuscular clinics, but nonneuromuscular causes are much more common than neuromuscular causes. Generalized fatigue is commonly caused by anemia, hypothyroidism, obstructive sleep apnea, depression, chronic fatigue syndrome (CFS), uremia, chronic obstructive pulmonary disease (COPD), and other diseases. Physiological fatigue is accentuated by neuromuscular disorders. Most strikingly, myasthenia gravis (MG) causes undue fatigue of the ocular, chewing, swallowing, and breathing muscles. However, amyotrophic lateral sclerosis (ALS), myopathies, and motor neuropa

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13

Shaibani, Aziz. Fatigability. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199898152.003.0025.

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Undue fatigability is common in neuromuscular clinics but non-neuromuscular causes are much more common than neuromuscular causes. Generalized fatigue is commonly caused by anemia, hypothyroidism, obstructive sleep apnea, depression, chronic fatigue syndrome, uremia, COPD, etc. Physiological fatigue is accentuated by neuromuscular disorders. Most strikingly, myasthenia gravis causes undue fatigue of the ocular, chewing, swallowing, and breathing muscles. However, ALS, myopathies, and motor neuropathies are also associated with abnormal fatigue. Myasthenia rarely causes isolated fatigue. Examin

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14

Shahidi, Nasrollah T., and F. Schier. Aplastic Anemia and Other Bone Marrow Failure Syndromes. Springer, 2011.

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15

Shahidi, Nasrollah T. Aplastic Anemia and Other Bone Marrow Failure Syndromes. Island Press, 1989.

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16

Shahidi, Nasrollah T. Aplastic Anemia and Other Bone Marrow Failure Syndromes. Springer London, Limited, 2012.

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