Academic literature on the topic 'Anesthesia-induced rhabdomyolysis'

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Journal articles on the topic "Anesthesia-induced rhabdomyolysis"

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Barrons, Robert William, and Liem T. Nguyen. "Succinylcholine-Induced Rhabdomyolysis in Adults: Case Report and Review of the Literature." Journal of Pharmacy Practice 33, no. 1 (2018): 102–7. http://dx.doi.org/10.1177/0897190018795983.

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Purpose: A case of succinylcholine (SCh) and sevoflurane as a probable cause of rhabdomyolysis in an adult is presented, along with a review of the relevant literature and strategies for prevention. Summary: A nondiabetic, morbidly obese 32-year-old female developed rhabdomyolysis after administration of SCh and sevoflurane for diagnostic procedures of 30 minutes’ duration. Thirty-three hours following anesthesia, the patient developed diffuse muscle tenderness and progressive weakness with a creatinine kinase (CK) of 4319 U/L. Urinalysis findings indicated contamination, a white blood cells o
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Tang, Thomas T., Herbert W. Oechler, Daniel Siker, Annette D. Segura, and Ralph A. Franciosi. "Anesthesia-induced rhabdomyolysis in infants with unsuspected Duchenne dystrophy." Acta Paediatrica 81, no. 9 (1992): 716–19. http://dx.doi.org/10.1111/j.1651-2227.1992.tb12344.x.

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Patel, Anuradha. "Anesthesia-Induced Rhabdomyolysis During Corrective Spine Surgery: A Case Report." British Journal of Medicine and Medical Research 3, no. 4 (2013): 1302–7. http://dx.doi.org/10.9734/bjmmr/2013/3165.

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Gray, Rebecca M. "Anesthesia-induced rhabdomyolysis or malignant hyperthermia: is defining the crisis important?" Pediatric Anesthesia 27, no. 5 (2017): 490–93. http://dx.doi.org/10.1111/pan.13130.

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Lochhead, K. M., E. D. Kharasch, and R. A. Zager. "Anesthetic effects on the glycerol model of rhabdomyolysis-induced acute renal failure in rats." Journal of the American Society of Nephrology 9, no. 2 (1998): 305–9. http://dx.doi.org/10.1681/asn.v92305.

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Isoflurane, the most widely used inhalational anesthetic, releases inorganic fluoride during its metabolism by the cytochrome P450 system. Recent experimental data indicate that when cultured proximal tubular cells are exposed to inorganic fluoride, they become relatively resistant to myoglobin- and ATP depletion-mediated attack. The present study was undertaken to assess whether isoflurane anesthesia might confer in vivo cytoprotection, possibly by causing renal tubular inorganic fluoride exposure, thereby mitigating a combined myoglobin/ATP depletion model of acute renal failure (glycerol-in
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Wappler, Frank, Marko Fiege, Markus Steinfath, et al. "Evidence for Susceptibility to Malignant Hyperthermia in Patients with Exercise-induced Rhabdomyolysis." Anesthesiology 94, no. 1 (2001): 95–100. http://dx.doi.org/10.1097/00000542-200101000-00019.

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Background Malignant hyperthermia (MH), heat stroke, and exercise-induced rhabdomyolysis (ER) were suspected to be related syndromes. However, it is not known whether individuals with history of ER have an increased incidence of susceptibility to MH. To establish an association between ER and susceptibility to MH, the authors determined the MH status in patients with a history of MH-like episodes induced by physical stress. Methods Twelve unrelated patients with ER, 18 patients with anesthesia-induced MH, and 28 controls were investigated with the in vitro contracture test (IVCT) according to
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Leo, Anne-Marie, Mark J. McVey, Megumi Iizuka, and Michael D. Richards. "A suspected case of anesthesia-induced rhabdomyolysis in a child undergoing strabismus surgery." Journal of American Association for Pediatric Ophthalmology and Strabismus 23, no. 3 (2019): 167–69. http://dx.doi.org/10.1016/j.jaapos.2019.01.006.

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Carsana, Antonella. "Exercise-Induced Rhabdomyolysis and Stress-Induced Malignant Hyperthermia Events, Association with Malignant Hyperthermia Susceptibility, andRYR1Gene Sequence Variations." Scientific World Journal 2013 (2013): 1–6. http://dx.doi.org/10.1155/2013/531465.

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Exertional rhabdomyolysis (ER) and stress-induced malignant hyperthermia (MH) events are syndromes that primarily afflict military recruits in basic training and athletes. Events similar to those occurring in ER and in stress-induced MH events are triggered after exposure to anesthetic agents in MH-susceptible (MHS) patients. MH is an autosomal dominant hypermetabolic condition that occurs in genetically predisposed subjects during general anesthesia, induced by commonly used volatile anesthetics and/or the neuromuscular blocking agent succinylcholine. Triggering agents cause an altered intrac
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Suzuki, Akiko, Eiji Hashiba, Akinori Matsui, Takeshi Kubota, Hironori Ishihara, and Akitomo Matsuki. "Repeated total intravenous anesthesia for a patient with a history of enflurane-induced rhabdomyolysis." Journal of Anesthesia 11, no. 3 (1997): 231–33. http://dx.doi.org/10.1007/bf02480044.

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Timmins, Matthew A., Henry Rosenberg, Marilyn Green Larach, Carly Sterling, Natalia Kraeva, and Sheila Riazi. "Malignant Hyperthermia Testing in Probands without Adverse Anesthetic Reaction." Anesthesiology 123, no. 3 (2015): 548–56. http://dx.doi.org/10.1097/aln.0000000000000732.

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Abstract Background: Malignant hyperthermia (MH) is triggered by reactions to anesthetics. Reports link nonanesthetic-induced MH-like reactions to a variety of disorders. The objective of the authors was to retrospectively investigate the reasons for referrals for MH testing in nonanesthetic cases and assess their phenotype. In addition, the response to the administration of oral dantrolene in nonanesthetic probands with positive caffeine–halothane contracture test (CHCT) was investigated. Methods: Following institutional research ethics board approval, probands without reaction to anesthesia,
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Book chapters on the topic "Anesthesia-induced rhabdomyolysis"

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Sinton, Jamie W. "Musculoskeletal Disease." In Advanced Anesthesia Review, edited by Alaa Abd-Elsayed. Oxford University PressNew York, 2023. http://dx.doi.org/10.1093/med/9780197584521.003.0259.

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Abstract The spectrum of musculoskeletal disease varies widely. Ambiguity in diagnosis or presentation to the operating room without a definitive diagnosis provides a common challenge to the anesthesiologist. Children with musculoskeletal disease may be uniquely vulnerable to malignant hyperthermia, anesthesia-induced rhabdomyolysis, covert cardiac dysfunction, or respiratory embarrassment. Children with skeletal disorders present unique airway and ventilatory challenges. Postoperative disposition may require escalation in respiratory care with multispecialty input. Finally, analgesia in any c
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