Relevant bibliographies by topics / Aneurysmal dilatation

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Aneurysmal dilatation'

Author: Grafiati
Published: 7 June 2025
Last updated: 16 July 2025

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Journal articles on the topic "Aneurysmal dilatation"

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Kim, Su Wan, Jonggeun Lee, Seogjae Lee, Jee Won Chang, and Chang Lim Hyun. "Pathologic change of an arterialized giant venous aneurysm of a brachiocephalic arteriovenous fistula." Journal of Medicine and Life Science 20, no. 4 (2023): 178–82. http://dx.doi.org/10.22730/jmls.2023.20.4.178.

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Aneurysmal venous dilatation is a frequent complication of arterio venous fistulas (AVFs) created for hemodialysis. Venous aneurysm rupture can lead to lethal hemorrhage. A 49-year-old male patient presented with a giant aneurysmal dilatation of his AVF 10 years after its creation. The patient had complaints of pulsating pain and discomfort due to swelling of the left forearm. We performed an aneurysm resection and revised the overlying dermal lesion through a brachial plexus block. Here, we describe the pathological features of the arterialized venous aneurysm compared to simple venous aneury
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Tang, Kuan, Yuanyou Li, Shuang Luo, and Jin Chen. "The risk factors of chronic ventricular dilatation after aneurysmal subarachnoid haemorrhage." Neurology Asia 28, no. 1 (2023): 105–11. http://dx.doi.org/10.54029/2023umv.

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Objectives: The purpose of this study was to explore the independent risk factors of chronic ventricular dilatation after aneurysmal subarachnoid haemorrhage. Methods: A retrospective study was carried out in patients with aneurysmal subarachnoid haemorrhage and admitted to the Second Affiliated Hospital of Chongqing Medical University from July 2017 to February 2021. The patients were grouped according to whether they had chronic ventricular dilatation. The patients’ demographic, clinical, and imaging datas including gender, age, hypertension, Hunt and Hess grade, Fisher grade, intraventricul
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Shah, Ravish, Tushar J. Vachharajani, and Anil K. Agarwal. "Aneurysmal Dilatation of Dialysis Arteriovenous Access." Open Urology & Nephrology Journal 6, no. 1 (2013): 1–5. http://dx.doi.org/10.2174/1874303x01306010001.

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Arteriovenous fistula is considered the “ideal vascular access” in patients with end stage renal disease (ESRD); however, its creation is not without complications. Aneurysmal dilatation is a poorly defined complication which, can potentially lead to access loss and life threatening hemorrhage. Increased venous pressure due to proximal venous stenosis along with repeated cannulations at the same site, are thought to play a significant role in pathogenesis. Given risk of substantial bleeding, it is recommended to avoid cannulation of aneurysmal area; however, as seen in our patient significant
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Lee, Hung‐Chang, Yhu‐Chering Yang, Shin‐Lin Shih, and Hsein‐Jar Chiang. "Aneurysmal Dilatation of the Portal Vein." Journal of Pediatric Gastroenterology and Nutrition 8, no. 3 (1989): 387–89. http://dx.doi.org/10.1002/j.1536-4801.1989.tb09772.x.

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SUMMARYIn the following study, a case involving an aneurysmal dilatation of the portal vein is reported. The 5‐year‐old boy had a 2 month history of intermittent abdominal pain. A segmental dilatation of the portal vein was noted in the sonographic examination. The portal vein aneurysm diagnosis was later confirmed by superior mesenteric angiography and computed tomography. In our review of the literature, less than 30 cases of portal venous aneurysm have been reported. To our knowledge, this is the youngest case with such an anomaly.
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Tomasi, Jacques, Reda Belhaj Soulami, Marion Rolland, and Jean-Philippe Verhoye. "Endovascular Repair of a Dacron Pseudoaneurysm in an Ascending-to-Descending Aortic Bypass." AORTA 08, no. 04 (2020): 104–6. http://dx.doi.org/10.1055/s-0040-1715087.

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AbstractIn the setting of postcoarctation aortic repair, Dacron graft dilatation and late aneurysms are not uncommon. Reintervention usually involves redo open surgery and replacement of the aneurysmal graft or the pseudoaneurysmal suture line. The present case describes the endovascular repair of a Dacron anastomotic false aneurysm in an extra-anatomic ascending-to-descending aortic bypass, 19 years after surgical correction of aortic recoarctation.
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Yu, Yong peng, Hong qin Zhao, Wei feng Ren, and Xiang lin Chi. "Giant aneurysm of the basilar artery in an 86 year old woman." F1000Research 2 (April 18, 2013): 112. http://dx.doi.org/10.12688/f1000research.2-112.v1.

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In this article we present an 80 year old female patient with an unruptured giant aneurysm of the basilar artery presenting with posterior circulation ischemic symptoms. Angiography and CT revealed giant basilar aneurysmal dilatation with severe and wide intracranial arteriosclerosis. We described the uniqueness of this case. Giant basilar aneurysm is associated with various complications particularly brain stem infarction. It is emphasized that arteriosclerosis plays an important role in the formation of giant basilar aneurysms.
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Hopsu, Erkki, Jussi Tarkkanen, Seija I. Vento, and Anne Pitkäranta. "Acquired Jugular Vein Aneurysm." International Journal of Otolaryngology 2009 (2009): 1–4. http://dx.doi.org/10.1155/2009/535617.

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Venous malformations of the jugular veins are rare findings. Aneurysms and phlebectasias are the lesions most often reported. We report on an adult patient with an abruptly appearing large tumorous mass on the left side of the neck identified as a jugular vein aneurysm. Upon clinical examination with ultrasound, a lateral neck cyst was primarily suspected. Surgery revealed a saccular aneurysm in intimate connection with the internal jugular vein. Histology showed an organized hematoma inside the aneurysmal sac, which had a focally thinned muscular layer. The terminology and the treatment guide
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Nunes, Magda Lahorgue, Ana Paula Silveira Pinho, and Ana Sfoggia. "Cerebral aneurysmal dilatation in an infant with perinatally acquired HIV infection and HSV encephalitis." Arquivos de Neuro-Psiquiatria 59, no. 1 (2001): 116–18. http://dx.doi.org/10.1590/s0004-282x2001000100024.

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Although most children with human immunodeficiency virus (HIV) infection have neurological dysfunction, in childhood the incidence of symptomatic cerebrovascular disease is low. Cerebral aneurysmal arteriopathy in childhood AIDS has been reported in the past and considered to have a relatively long latency following the primary infection. We report a 1 month-old infant with congenitally acquired HIV infection, and herpes encephalitis; she presented a sudden cardiorespiratory arrest followed by coma and was found to have a giant saccular aneurysm of the left basilar artery. Literature review sh
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Hirai, S., S. Mine, E. Kobayashi, I. Yamakami, and A. Yamaura. "Angioarchitecture Predicting Hemorrhage in Cerebral Arteriovenous Malformations." Interventional Neuroradiology 5, no. 1_suppl (1999): 157–60. http://dx.doi.org/10.1177/15910199990050s128.

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To find out lesions responsible for hemorrhage in arteriovenous malformations (AVMs), a retrospective study of angioarchitecture around the nidus was conducted in 27 patients who underwent conservative treatment. Comparison of angiograms revealed disappearance of an intranidal aneurysmal dilatation after the hemorrhagic events in two cases. The hematomas were adjacent to the dilatation, and no subarachnoid hemorrhage was evident. Obstruction of venous drainage, noticed in a case of spontaneous regression of AVM, was not demonstrated in the cases of hemorrhage. The intranidal aneurysmal dilatat
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Vieira, Eduardo, Igor V. Faquini, Jose L. Silva, et al. "Subarachnoid neurocysticercosis and an intracranial infectious aneurysm: case report." Neurosurgical Focus 47, no. 2 (2019): E16. http://dx.doi.org/10.3171/2019.5.focus19280.

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Infectious intracranial aneurysms (IIAs) represent 2%–6% of all intracranial aneurysms and, classically, have been associated with bacterial or fungal agents. The authors report the case of a 42-year-old woman who presented with a typical history of subarachnoid hemorrhage. Digital subtraction angiography (DSA) showed an aneurysmal dilatation on the frontal M2 segment of the left middle cerebral artery (MCA). The patient was treated surgically, and multiple cysts were found in the left carotid and sylvian cisterns, associated with a dense inflammatory exudate that involved the MCA. The cysts w
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Dissertations / Theses on the topic "Aneurysmal dilatation"

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Hofmann, Sigrun R., Antje Heilmann, Hans J. Häusler, Ingo Dähnert, Gabriele Kamin, and Robert Lachmann. "Congenital Idiopathic Dilatation of the Right Atrium: Antenatal Appearance, Postnatal Management, Long-Term Follow-Up and Possible Pathomechanism." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-137447.

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Introduction: Idiopathic dilatation of the right atrium (IDRA) is a rare abnormality usually detected by chance at any time between antenatal and adult life. It is defined as isolated enlargement of the right atrium in the absence of other cardiac lesions causing right atrial dilatation. IDRA can be associated with atrial arrhythmia and systemic embolism. The clinical presentation shows high variability ranging from the lack of any symptoms up to cardiac failure. Methods/Results: We describe 2 children with antenatally diagnosed IDRA, the intrauterine course in 1 case, the postnatal management
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Hofmann, Sigrun R., Antje Heilmann, Hans J. Häusler, Ingo Dähnert, Gabriele Kamin, and Robert Lachmann. "Congenital Idiopathic Dilatation of the Right Atrium: Antenatal Appearance, Postnatal Management, Long-Term Follow-Up and Possible Pathomechanism." Karger, 2012. https://tud.qucosa.de/id/qucosa%3A27734.

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Introduction: Idiopathic dilatation of the right atrium (IDRA) is a rare abnormality usually detected by chance at any time between antenatal and adult life. It is defined as isolated enlargement of the right atrium in the absence of other cardiac lesions causing right atrial dilatation. IDRA can be associated with atrial arrhythmia and systemic embolism. The clinical presentation shows high variability ranging from the lack of any symptoms up to cardiac failure. Methods/Results: We describe 2 children with antenatally diagnosed IDRA, the intrauterine course in 1 case, the postnatal management
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Silva, Junior José Elias da [UNESP]. "Aneurisma de aorta infrarenal tratado por via endovascular em pacientes assintomáticos versus sintomáticos: avaliação da medida do saco aneurismático após um ano de seguimento." Universidade Estadual Paulista (UNESP), 2017. http://hdl.handle.net/11449/152531.

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Silva, Júnior José Elias da. "“Aneurisma de aorta infrarenal tratado por via endovascular em pacientes assintomáticos versus sintomáticos. Avaliação da medida do saco aneurismático após um ano de seguimento.”." Universidade Estadual Paulista (UNESP), 2017. http://hdl.handle.net/11449/152890.

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Submitted by José Elias da Silva Júnior null (jedsjunior@hotmail.com) on 2018-02-28T17:41:26Z No. of bitstreams: 1 TESE FINAL CORRIGIDA.pdf: 992178 bytes, checksum: 6cf7663fc6a01c288850e2c6ea550472 (MD5)<br>Approved for entry into archive by ROSANGELA APARECIDA LOBO null (rosangelalobo@btu.unesp.br) on 2018-03-05T17:48:49Z (GMT) No. of bitstreams: 1 silvajunior_je_me_bot.pdf: 992178 bytes, checksum: 6cf7663fc6a01c288850e2c6ea550472 (MD5)<br>Made available in DSpace on 2018-03-05T17:48:49Z (GMT). No. of bitstreams: 1 silvajunior_je_me_bot.pdf: 992178 bytes, checksum: 6cf7663fc6a01c288850e2c
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Books on the topic "Aneurysmal dilatation"

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Perkins, Jeremy. Peripheral arterial disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0104.

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Peripheral arterial disease is defined as an alteration to the blood supply to a limb, caused by an occlusion or stenosis in the arteries supplying that limb. The acuteness of the arterial compromise, and its severity and extent, will determine the symptoms experienced by the patient. Aneurysmal disease is defined as a localized dilatation of an artery and is most commonly seen in the infrarenal abdominal aorta. An infrarenal abdominal aorta is defined as being aneurysmal if its maximum anteroposterior diameter is 3 cm or greater.
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Bahr, Sabine. Häufigkeit der Entstehung von Rezidiven und Aneurysmen nach Aortenisthmusstenosen: Dilatation bei Erwachsenen. 1994.

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Elliott, Perry, Kristina H. Haugaa, Pio Caso, and Maja Cikes. Restrictive cardiomyopathy and arrhythmogenic right ventricular cardiomyopathy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0044.

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Restrictive cardiomyopathy is a heart muscle disorder characterized by increased myocardial stiffness that results in an abnormally steep rise in intraventricular pressure with small increases in volume in the presence of normal or decreased diastolic left ventricular volumes and normal ventricular wall thickness. The disease may be caused by mutations in a number of genes or myocardial infiltration. Arrhythmogenic right ventricular cardiomyopathy is an inherited cardiac muscle disease associated with sudden cardiac death, ventricular arrhythmias, and cardiac failure. It is most frequently cau
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López-Sendón, José, and Esteban López de Sá. Mechanical complications of myocardial infarction. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0045.

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Mechanical complications after an acute infarction include different forms of heart rupture, including free wall rupture, interventricular septal rupture, and papillary muscle rupture. Its incidence decreased dramatically with the widespread use of reperfusion therapies but may occur in 2–3% of ST-elevation myocardial infarction patients, and mortality is very high if not properly diagnosed, as surgery is the only effective treatment. Echocardiography is the most important tool for diagnosis that should be suspected in patients with hypotension, heart failure, or recurrent chest pain. Awarenes
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López-Sendón, José, and Esteban López de Sá. Mechanical complications of myocardial infarction. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199687039.003.0045_update_001.

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Mechanical complications after an acute infarction include different forms of heart rupture, including free wall rupture, interventricular septal rupture, and papillary muscle rupture. Its incidence decreased dramatically with the widespread use of reperfusion therapies but may occur in 2–3% of ST-elevation myocardial infarction patients, and mortality is very high if not properly diagnosed, as surgery is the only effective treatment. Echocardiography is the most important tool for diagnosis that should be suspected in patients with hypotension, heart failure, or recurrent chest pain. Awarenes
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López-Sendón, José, and Esteban López de Sá. Mechanical complications of myocardial infarction. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0045_update_002.

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Mechanical complications after an acute infarction include different forms of heart rupture, including free wall rupture, interventricular septal rupture, and papillary muscle rupture. Its incidence decreased dramatically with the widespread use of reperfusion therapies but may occur in 2–3% of ST-elevation myocardial infarction patients, and mortality is very high if not properly diagnosed, as surgery is the only effective treatment. Echocardiography is the most important tool for diagnosis that should be suspected in patients with hypotension, heart failure, or recurrent chest pain. Awarenes
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López-Sendón, José, and Esteban López de Sá. Mechanical complications of myocardial infarction. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0045_update_003.

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Mechanical complications after an acute infarction involve different forms of heart rupture, including free wall rupture, interventricular septal rupture, and papillary muscle rupture. Its incidence decreased dramatically with the widespread use of reperfusion therapies occurring in &lt;1% of ST-elevation myocardial infarction patients, and mortality is very high if not properly diagnosed, as surgery is the only effective treatment (Ibanez et al, 2017). Echocardiography is the most important tool for diagnosis that should be suspected in patients with hypotension, heart failure, or recurrent c
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Book chapters on the topic "Aneurysmal dilatation"

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Perloff, Joseph K., Charles W. Urschell, William C. Roberts, and Walter H. Caulfield. "Aneurysmal Dilatation of the Coronary Arteries in Cyanotic Congenital Cardiac Disease*." In Case Reports in Cardiology. CRC Press, 2023. http://dx.doi.org/10.1201/9781003409342-10.

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Zhang, Shaojie, Joan D. Laubrie, S. Jamaleddin Mousavi, Sabrina Ben Ahmed, and Stéphane Avril. "Patient-Specific Finite Element Modeling of Aneurysmal Dilatation After Chronic Type B Aortic Dissection." In Computational Biomechanics for Medicine. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-09327-2_2.

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Zeitler, E., and F. H. W. Heuck. "Aneurysmen und dilatative Angiopathie." In Arterien und Venen. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60381-5_12.

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Ibrahim, Michael, and Nimesh D. Desai. "Thoracic Aortic Dilatation, Aneurysm and Dissection." In Cardiovascular Genetics and Genomics. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-66114-8_21.

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Gupta, Vipul. "Very Small (Less Than 2 mm) Aneurysm with Severe Vasospasm: Pretreatment Dilatation." In 100 Interesting Case Studies in Neurointervention: Tips and Tricks. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-1346-2_54.

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Lylyk, Ivan, Carlos Bleise, Rene Viso, Esteban Scrivano, and Pedro Lylyk. "Paraophthalmic Internal Carotid Artery Aneurysm: Spontaneous Subarachnoid Hemorrhage Caused by the Rupture of a Paraophthalmic Aneurysm, Treated with Coils and Complicated by Severe Vasospasm, Treated with Pharmaceutical Vessel Dilatation and Proximal Balloon Angioplasty; Diffuse Distal Vasospasm Treated with the NeuroFlo Device with Good Clinical Outcome." In The Aneurysm Casebook. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-77827-3_119.

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Lylyk, Ivan, Carlos Bleise, Rene Viso, Esteban Scrivano, and Pedro Lylyk. "Paraophthalmic Internal Carotid Artery Aneurysm: Spontaneous Subarachnoid Hemorrhage Caused by the Rupture of a Paraophthalmic Aneurysm, Treated with Coils and Complicated by Severe Vasospasm, Treated with Pharmaceutical Vessel Dilatation and Proximal Balloon Angioplasty; Diffuse Distal Vasospasm Treated with the NeuroFlo Device with Good Clinical Outcome." In The Aneurysm Casebook. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-70267-4_119-1.

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Weir, Bryce. "Epidemiology and Associated Disease States." In Subarachnoid Hemorrhage: Causes And Cures. Oxford University PressNew York, NY, 1998. http://dx.doi.org/10.1093/oso/9780195128758.003.0003.

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Abstract An aneurysm is a dilatation on a blood vessel,1 usually an artery. Many different types of aneurysms affect the nervous system (Table 3-1). The most common type is a saccular or ovoid protrusion attached by a small neck to the artery of origin, historically called a “berry.” Ruptured aneurysms account for most cases of subarachnoid hemorrhage (SAH), and the incidence of aneurysmal SAH is directly related to age. Aneurysmal rupture is extremely uncommon in infancy, becoming steadily less so during aging. For this reason the term “congenital” applied to aneurysms is a misnomer.
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Lee, Christine U., and James F. Glockner. "Case 17.27." In Mayo Clinic Body MRI Case Review, edited by Christine U. Lee and James F. Glockner. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199915705.003.0445.

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60-year-old man with hypertension resistant to medical therapy MIP images from noncontrast 3D SSFP renal MRA (Figure 17.27.1) demonstrate poor visualization of the renal arteries, with low contrast and low SNR. VR images from CE 3D MRA (Figure 17.27.2) reveal 2 left renal arteries and aneurysmal dilatation of the distal right main renal artery....
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Li, Yanliang, Monique Munro, Gerardo Ledesma-Gil, and William F. Mieler. "Takayasu arteritis: Bilateral progressive loss of vision with aneurysmal dilatation." In Clinical Cases in Medical Retina. Elsevier, 2025. http://dx.doi.org/10.1016/b978-0-12-822720-6.00057-4.

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Conference papers on the topic "Aneurysmal dilatation"

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Guzmán, Amador M., Nelson O. Moraga, and Cristina H. Amon. "Pulsatile Non-Newtonian Flow in a Double Aneurysm." In ASME 1997 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1997. http://dx.doi.org/10.1115/imece1997-0241.

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Abstract An aneurysm is a permanent balloon-like, blood-filled dilatation on an arterial vessel. In an abdominal aortic aneurysm (AAA), the growth and eventual rupture of aneurysmal lesions depends on the interaction between the aneurysm wall and the blood motion along it. Rupture of an aneurysm occurs when the shear stresses exceed the strength of the wall [1]. When an AAA ruptures, 50 percent of patients die before reaching the operating room and 54 percent of the remaining will die within 30 days [2]. Therefore, AAA represents a great danger to the patient [3]. The objective of this study i
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Ferruzzi, J., M. S. Enevoldsen, and J. D. Humphrey. "On the Mechanical Behavior of Healthy and Aneurysmal Abdominal Aorta." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53852.

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Abdominal aortic aneurysm (AAA) is a pathological condition of the infrarenal aorta characterized by a local dilatation of the arterial wall. The main histopathologic features of an AAA are smooth muscle cell death and loss of elastin. The biomechanical behavior of AAAs has been widely studied to determine the rupture potential according to the principles of material failure. However, most prior approaches are limited by the use of data from uniaxial tensile testing and by the assumption of material isotropy, leading to inaccurate characterization of the 3D multiaxial mechanical response of th
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Fazel Bakhsheshi, Mohammad, Florian Vixege, and Dana Grecov. "Reduction of the Aortic Aneurysm Sac Pressure Using a Stent With Venturi Structure: A Numerical Study." In 2018 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/dmd2018-6919.

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Aortic aneurysm is a common disorder which is due to weakening of the aortic wall [1]. Aneurysm rupture is a potentially life threatening complication [2]. The stent graft implantation is one of the potential alternatives for treating patients at high risk for an open surgical procedure. The short-term outcome for stent implantation is promising; however, as with any medical procedure, this has potential limitations such as side branch occlusion, device malfunctions, dilatation at the proximal portion and the so called ‘endoleak’. An endoleak is the persistent blood flow into and within the an
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Ene, Florentina, Carine Gachon, Patrick Delassus, and Liam Morris. "Investigating the Effect of Intraluminal Thrombus in Abdominal Aortic Aneurysm by Computational and Experimental Methods." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206636.

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Abdominal aortic aneurysm (AAA) represents an abnormal dilatation and weakening of the abdominal aorta with high risk of rupture. Most aneurysms of the infrarenal aorta possess an asymmetrical fusiform morphology.
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Zhao, Xuefeng, Madhavan L. Raghavan, and Jia Lu. "Identifying the Distribution of Heterogeneous Anisotropic Elastic Properties in Cerebral Aneurysms." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206659.

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Cerebral aneurysms are focal dilatations of the intracranial arterial wall, whose rupture risk is likely related to pressure induced wall stress. Fundamental to stress and strain prediction in aneurysms is the constitutive behavior of wall tissue. However, delineating the constitutive equation of aneurismal tissue, in particular, experimental determination of the material parameters, presents some significant challenges due to the nonlinear, anisotropic and heterogeneous nature of the aneurysmal tissue.
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Trachet, Bram, Marjolijn Renard, Joris Bols, Steven Staelens, Bart Loeys, and Patrick Segers. "Hemodynamics in Ascending and Abdominal Aorta Aneurysm Formation in the ApoE−/− Angiotensin II Mouse Model." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80243.

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Aortic aneurysm is a pathological dilatation of the aorta that can be life-threatening when it ruptures. Aneurysms occur throughout the entire aorta but there is a predisposition for the ascending and the abdominal aorta, an observation that cannot be fully explained by the current knowledge of the disease pathophysiology. ApoE −/− mice infused with angiotensin II have recently been reported to develop not only abdominal [1], but also ascending aortic aneurysms [2]. These animals thus provide the perfect model to compare aneurysm progression in both aortic locations and to investigate whether
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Kroon, Martin, and Gerhard Holzapfel. "A Theoretical Model for Saccular Cerebral Aneurysm Growth: Deformation and Stress Analysis." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176857.

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Aneurysms are abnormal dilatations of arteries, and these lesions are found almost exclusively in humans. Saccular cerebral aneurysms occur most frequently in the Circle of Willis, which is a circuit of arteries supplying the brain with blood. Aneurysms of this kind appear in a few percent of the human population in the Western world. Only a few percent of these lesions do actually rupture, but once rupture occurs the consequences are severe, often with death as outcome. Once a cerebral aneurysm is detected, clinicians need to decide whether operation is required or not. These decisions are ma
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Ma, Jiayao, Zhong You, and James Byrne. "A Novel Flow Diverter for Direct Treatment of Cerebral Aneurysms." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14013.

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Cerebral aneurysm (CA) is a localised dilatation in the wall of the brain vasculature which can cause it to swell out like a balloon. If not treated, it will continue to grow and eventually tear or rupture, resulting in severe disability from stroke and, in around 25% of cases, death [1]. CAs affect up to 5% of the adult population with 1% of detected aneurysms rupture every year [2].
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9

Kwon, Chi-Ho, Ki-Won Lee, and Young-Ho Kim. "Fluid-Structure Interactions Abdominal Aortic Aneurysm Models Under the Pulsatile Flow Condition." In ASME 2000 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2000. http://dx.doi.org/10.1115/imece2000-2542.

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Abstract Fluid-structure interaction studies were performed on various abdominal aortic aneurysm (AAA) models under the pulsatile flow condition. Eight aneurysm models were made with four different dilatation sizes and two different wall thickness. Stresses and deformations of the aneurysm wall were significantly affected by the dilatation size as well as the wall thickness. The change in wall thickness increased with the more dilated aneurysm. In spite of considerable radial deformations, axial deformations of the aneurysm wall were dominant. The present study showed the strong possibility to
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10

Trachet, Bram, Daniel Devos, Julie De Backer, Anne De Paepe, Bart L. Loeys, and Patrick Segers. "Patient-Specific Modelling of Aortic Arch Wall Shear Stress Patterns in Patients With Marfan Syndrome." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206340.

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Marfan syndrome (MFS) is a genetic connective tissue disorder with a high prevalence of aortic aneurysm formation (a pathological dilatation of the aorta), typically at the aortic root. The disorder is caused by mutations in the gene encoding fibrillin-1 [1]. Recently, it has been shown in mouse models that selected manifestations of MFS, such as aortic aneurysm formation, can be explained by excessive signaling by the transforming growth factor–beta (TGF-beta) family of cytokines [2]. Although the footprint of the disease is clearly genetic, there is still a role for (computational) biomechan
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