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Books on the topic 'Aneurysmata'

Author: Grafiati

Published: 4 June 2021

Last updated: 31 January 2023

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1

Transfemoral endovascular aneurysm management. Universiteit Utrecht], 1996.

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2

Arterial aneurysms: A historical review. [publisher not identified], 2004.

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3

1960-, Forsting M., and Cognard C, eds. Intracranial vascular malformations and aneurysms: From diagnostic work-up to endovascular therapy. Springer, 2004.

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4

Langmoen, Iver A., Tryggve Lundar, Rune Aaslid, and Hans-J. Reulen, eds. Neurosurgical Management of Aneurysmal Subarachnoid Haemorrhage. Springer Vienna, 1999. http://dx.doi.org/10.1007/978-3-7091-6377-1.

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5

Bonser, Robert S., Domenico Pagano, Axel Haverich, and Jorge Mascaro, eds. Controversies in Aortic Dissection and Aneurysmal Disease. Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-5622-2.

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6

Surgical techniques for saccular and giant intracranial aneurysms. Williams & Wilkins, 1990.

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7

Savage, Edward Bruce. Hydrocortisone induces aortic rupture in inbred blotchy mice: Implications for abdominal aortic aneurysmal disease in humans. s.n.], 1985.

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8

Aneurysmal Subarachnoid Hemorrhage. W.B. Saunders Company, 2010.

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9

Sartor, K., and Michael Forsting. Intracranial Vascular Malformations and Aneurysms: From Diagnostic Work-Up to Endovascular Therapy. Springer London, Limited, 2006.

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10

Cognard, C., M. Knaut, Isabel Wanke, A. Dörfler, and Michael Forsting. Intracranial Vascular Malformations and Aneurysms: From Diagnostic Work-Up to Endovascular Therapy. Springer London, Limited, 2008.

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11

Management Of Aneurysmal Subarachnoid Hemorrhage. E.M.I.S. MEDICAL PUBLISHING, 1993.

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12

Langmoen, I. A. Neurosurgical Management of Aneurysmal Subarachnoid Haemorrhage. Langmoen I a, 2012.

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13

Reulen, Hans J., I. A. Langmoen, Tryggve Lundar, and Rune Aaslid. Neurosurgical Management of Aneurysmal Subarachnoid Haemorrhage. Springer, 2012.

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14

Haverich, Axel, Robert S. Bonser, Jorge Mascaro, and Domenico Pagano. Controversies in Aortic Dissection and Aneurysmal Disease. Springer, 2016.

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15

Haverich, Axel, Robert S. Bonser, Jorge Mascaro, and Domenico Pagano. Controversies in Aortic Dissection and Aneurysmal Disease. Springer, 2014.

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16

Haverich, Axel, Robert S. Bonser, and Domenico Pagano. Controversies in Aortic Dissection and Aneurysmal Disease. Springer, 2014.

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17

Savvina, Irina, ed. Aneurysmal Subarachnoid Hemorrhage: From Diagnosis to Treatment. Nova Science Publishers, 2022. http://dx.doi.org/10.52305/dngf4073.

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18

Savvina, Irina Alexandrovna. Aneurysmal Subarachnoid Hemorrhage: From Diagnosis to Treatment. Nova Science Publishers, Incorporated, 2022.

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19

Savvina, Irina Alexandrovna. Aneurysmal Subarachnoid Hemorrhage: From Diagnosis to Treatment. Nova Science Publishers, Incorporated, 2022.

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20

Aneurysmal Disease of the Thoracic and Abdominal Aorta. InTech, 2011.

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21

Bush, Ruth, ed. Aneurysmal Disease of the Thoracic and Abdominal Aorta. InTech, 2011. http://dx.doi.org/10.5772/1038.

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22

Lo, Benjamin W. Y., Aurora W. M. Lo, Adrian Upton, Benedict Beng-Teck Taw, and Hidetoshi Kasuya. Pathophysiology of Brain-Body Interactions in Aneurysmal Subarachnoid Hemorrhage. Nova Science Publishers, Incorporated, 2017.

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23

(Editor), I. A. Langmoen, Tryggve Lundar (Editor), Rune Aaslid (Editor), and Hans-J. Reulen (Editor), eds. Neurosurgical Management of Aneurysmal Subarachnoid Haemorrhage (Acta Neurochirurgica Supplementum). Springer, 1999.

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24

Waschke, Jacqueline. Beule Im Kopf: Ein Stück Vom Leben Mit Aneurysma. Independently Published, 2018.

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25

Hasan, David. The Natural History of Cerebral Aneurysms. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0109.

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Subarachnoid hemorrhage (SAH) secondary to rupture of cerebral aneurysms represents a relatively small fraction of strokes (5%) but morbidity and mortality associated with aneurysm rupture remain very high despite advances in the treatment of aneurysmal SAH. Cerebral vasospasm (CV) is the leading cause of delayed morbidity and mortality following aneurysmal subarachnoid hemorrhage, as well as delayed neurological dysfunction 1 to 2 weeks after rupture. Endothelial dysfunction is one of the primary contributing factors to CV following aneurysmal SAH, and this is associated with alterations in i

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26

Perkins, Jeremy. Peripheral arterial disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0104.

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Peripheral arterial disease is defined as an alteration to the blood supply to a limb, caused by an occlusion or stenosis in the arteries supplying that limb. The acuteness of the arterial compromise, and its severity and extent, will determine the symptoms experienced by the patient. Aneurysmal disease is defined as a localized dilatation of an artery and is most commonly seen in the infrarenal abdominal aorta. An infrarenal abdominal aorta is defined as being aneurysmal if its maximum anteroposterior diameter is 3 cm or greater.

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27

Mcgrath, Anthony, Kesavan Sri-Ram, Eric Yeung, et al. Benign bone tumours. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.002005.

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♦ Unicameral bone cyst♦ Aneurysmal bone cyst♦ Non-ossifying fibroma/fibrous cortical defect♦ Bone island♦ Fibrous dysplasia♦ Osteochondroma♦ Enchondroma♦ Langerhans cell histiocytosis♦ Chondroblastoma♦ Giant cell tumour♦ Osteoblastoma/osteoid osteoma♦ 12.Periosteal chondroma.

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28

Chong, Ji Y., and Michael P. Lerario. Worst Headache of Her Life. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190495541.003.0035.

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Aneurysmal subarachnoid hemorrhage typically causes sudden severe headache. Diagnosis is made by CT scan in most cases, but lumbar puncture may be needed if CT findings are normal and suspicion is high. Rapid evaluation and treatment are important because of the high morbidity and mortality associated with rebleeding and vasospasm.

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29

Chong, Ji Y., and Michael P. Lerario. Thunderclap Headache. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190495541.003.0024.

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Reversible cerebral vasoconstriction syndrome can cause thunderclap headache, subarachnoid hemorrhage, and stroke. The clinical presentation can be similar to aneurysmal subarachnoid hemorrhage and therefore requires rapid angiography. Angiographic findings of vasculopathy and resolution of clinical symptoms and imaging abnormalities support this diagnosis. This syndrome is most often incited following pregnancy or the ingestion of a vasoactive medication.

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30

Fukuda, Hitoshi, Benjamin W. Y. Lo, Yusuke Nishimura, Yee-Yin Wan, and Aurora W. M. Lo. Brain-Body Interactions: Contemporary Outcome Prediction in Aneurysmal Subarachnoid Hemorrhage Using Bayesian Neural Networks and Fuzzy Logic. Nova Science Publishers, Incorporated, 2014.

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31

Pia, H. W., C. Langmaid, and J. Zierski. Cerebral Aneurysms: Advances in Diagnosis and Therapy. Springer London, Limited, 2012.

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32

Eichstädt, Hermann. Quantitative Myokardszintigraphie Bei Koronaroperationen: Darstellung Eines Methodischen Quantifizierungsversuchs in der Perioperativen Diagnostik der Koronaren Herzerkrankung Bei Aneurysma- und Bypasspatienten. Springer London, Limited, 2013.

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33

Perrott, Jerrold L., and Steven C. Reynolds. Neuroprotective agents in critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0048.

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The prevention and reduction of secondary injury following primary CNS insult is an important goal in critically-ill patients. Numerous pharmacological therapies have been studied as potential neuroprotective agents with few translating from research to clinical benefit. These are nimodipine and statins in aneurysmal subarachnoid haemorrhage and phenytoin in traumatic brain injury. Additionally, in traumatic brain injury, clinical studies have identified that corticosteroids and albumin colloid resuscitation are associated with increased risk of mortality, and as such should be avoided. Future

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34

Spittell, Peter C. Vascular Diseases. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0728.

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Peripheral vascular diseases, such as aneurysmal disease, intermittent claudication, acute arterial occlusion, and thromboangiitis, are prevalent in current medical practice. Vasospastic disorders, another class of vascular disease, are characterized by episodic color changes of the skin resulting from intermittent spasm of the small arteries and arterioles of the skin and digits. Vasospastic disorders are important because they frequently are a clue to another underlying disorder, such as arterial occlusive disease, connective tissue disorders, neurologic disorders, or endocrine disease. Char

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35

Kahn, S. Lowell. Reverse Deployment of the Gore Excluder Contralateral Iliac Limbs for Aortoiliac Interventions. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0009.

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Aortoiliac occlusive disease and aneurysmal disease are common pathologies encountered by the interventionalist. There are a multitude of commercially available bifurcated grafts for use in aortoiliac disease, but these devices are costly and require at least a 14 Fr femoral access for deployment. This chapter describes a simple and safe method for reversing deployment of the Excluder contralateral limb. This has great utility not only for aortoiliac interventions but also for central venous stenoses/occlusions. Reversal of the limb allows a proximal diameter of 12–27 mm with a fixed distal di

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36

Wijdicks, Eelco F. M., and Sarah L. Clark. Vasopressors and Inotropes. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190684747.003.0012.

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Vasopressors and inotropes are used in the neurosciences intensive care unit to treat hypotension and to augment blood pressure. Hypotension can be attributed to abnormal cardiac output, abnormal intravascular volume or abnormal systemic vascular resistance. Vasopressors are needed to manage hemodynamic augmentation in patients with severe cerebral vasospasm and aneurysmal subarachnoid hemorrhage, in patients with critical carotid or basilar artery stenosis producing marginal blood flow, or when patients are maintained in drug-induced comas. The main incentive is to maintain adequate perfusion

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37

Kahn, S. Lowell. Use of Two Bifurcated Stent Grafts for Creation of an Aorto-Uni-Iliac Endograft. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0010.

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Abdominal aortic aneurysms are a common pathology encountered by the interventionalist. Most endovascular repairs are performed with conventional bifurcated devices. However, there are situations in which the use of an aorto-uni-iliac (AUI) device is required because the use of a bifurcated graft is not feasible. Standard indications for use of an AUI include a narrow aortic segment precluding delivery and adequate expansion of a bifurcated graft, unilateral iliac occlusion, tortuosity, severe stenosis, and the presence of iliac aneurysmal disease. Occasionally, an AUI may be used for aortoili

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38

Neurosurgery of Complex Tumors and Vascular Lesions. Churchill Livingstone, 1997.

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39

Dewhurst, Alexander Timothy, and Brigitta Brandner. Intensive care management after vascular surgery. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0370.

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Vascular patients require admission to an intensive care unit at a number of stages during their hospital stay. They often have multiple co-morbidities and are at risk of major complications. Their management strategy requires a multidisciplinary approach with locally agreed pathways taking national frameworks into account. Vascular emergencies require immediate resuscitation and transfer to a tertiary cardiovascular centre. Vascular disease occurs throughout the arterial vascular tree, affecting both large and small vessels. The major cause is atherosclerosis. The management of vascular condi

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40

Michel, Jean-Baptiste. Biology of vascular wall dilation and rupture. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0016.

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Arterial pathologies, important causes of death and morbidity in humans, are closely related to modifications in the circulatory system during evolution. With increasing intraluminal pressure and arterial bifurcation density, the arterial wall becomes the target of interactions with blood components and outward convection of plasma solutes and particles, including plasma zymogens and leukocyte proteases. Abdominal aortic aneurysms of atherothrombotic origin are characterized by the presence of an intraluminal thrombus (ILT), a major source of proteases, including plasmin, MMP-9, and elastase.

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