Relevant bibliographies by topics / Angian Pectoris

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Angian Pectoris'

Author: Grafiati
Published: 4 June 2021
Last updated: 12 February 2022

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Journal articles on the topic "Angian Pectoris"

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Abduljabbar, Ahmed Abduljalal, and Parween Abdulsamad Ismail. "Investigation of Malondialdehyde (MDA), Homocysteine (Hcy) and C- reactive protein (CRP) in sera of patients with Angina Pectoris." Al-Mustansiriyah Journal of Science 30, no. 1 (August 15, 2019): 68. http://dx.doi.org/10.23851/mjs.v30i1.463.

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Homocysteine (Hcy) has been considered as an independent risk factor for coronary artery disease (CAD Oxidative stress and free radicals are known to have important roles in the development ofAngina Pectoris. Oxidative stress is present in cardiovascular diseases (CVDs), and hyperhomocysteinemia, an independent risk factor for these diseases, may play a role in inducing production of oxygen free radicals. The aim of this study was to determine a possible relationship between blood serum Hcy levels and lipid peroxidation in patients suffering from Angina Pectoris (AP).To evaluate the possible role of homocysteine (Hcy) in inducing oxidative stress in Angina Pectoris(AP), plasma homocysteine( Hcy), plasma malondialdehyde (MDA) and C reactive protein (CRP) were measured in 60 unstable Angina Pectoris patients, we tested 30 healthy volunteers. Hcy was measured by an enzymatic colorimetric method and MDA, an index of lipid peroxidation, by spectrophotometer. Serum Hcy levels were significantly higher in angina pectories (AP) patients than the controls (23.2±8.0 vs 10.76 ± 2.55 micromol/L; P
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Chaloupka, Václav. "Angina, its diagnosis and treatment." Cor et Vasa 49, no. 9 (September 1, 2007): 334–40. http://dx.doi.org/10.33678/cor.2007.116.

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Rajanit, Sojitra, Mukesh Dungrani, Paras Virani, and Hasumati Raj. "A review on Nifedipine co-administered with Metoprolol succinate for the treatment of hypertension." International Journal of Advances in Scientific Research 1, no. 3 (April 30, 2015): 129. http://dx.doi.org/10.7439/ijasr.v1i3.1795.

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Hypertension and Angina pectoris area major public health problem in the developed Countries recently. Hypertension and Angina Pectoris are frequently treated with antihypertensive drugs like calcium-channel blockers, angiotensin-converting enzyme inhibitors, and angiotensin II (AT1) receptor blockers, and statins. Nifedipine is calcium-channel blockers and widely used in treatment of Angina pectoris condition. Metoprolol Succinate is Beta-adrenoreceptor blocker and widely used in treatment of hypertension condition. Combination of Nifedipine and Metoprolol Succinate is used in the treatment of cardiovascular diseases like hypertension and Angina Pectoris. So this combination therapy gives antihypertensive and Angina Pectoris effects in the treatment of cardiac diseases.
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Seyidov, V. G., A. Ya Fissun, V. V. Yevsyukov, I. V. Lyubchuk, S. Ye Bobyrev, and E. V. Arutyunov. "Long-term coronary shunting results for 5 years of observation. Factors influencing on angina pectoris recurrence after coronary shunting." Bulletin of Siberian Medicine 5, no. 3 (September 30, 2006): 105–11. http://dx.doi.org/10.20538/1682-0363-2006-3-105-111.

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Aimed at o comparing coronary shunting long-term results with drug treatment of angina pectoris as well as estimating influence of dyslipidemia, diabetes mellitus, hypoglycemic therapy nature, arterial hypertension and increased systemic inflammation on angina pectoris recurrence after the surgery, we examined 793 patients 5 years after coronary shunting and 81 patients who were treated by standard methods. Five years after the surgery, we noted decreased number of patients without angina pectoris symptoms and increased number of patients with angina pectoris. Increased levels of cholesterol, low density lipoproteins, α-lipoproteids, C-reactive proteins and diabetes mellitus of the 2-nd type II—III stage contribute to angina pectoris recurrence rate after the surgery. Operated on patients revealed angina pectoris recurrences, myocardial infarction frequency, repeated hospitalizations and annual mortality more seldom compared with patients who underwent drug treatment.
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YASUE, HIROFUMI. "Angina pectoris." Nihon Naika Gakkai Zasshi 86, no. 2 (1997): 189–90. http://dx.doi.org/10.2169/naika.86.189.

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O'Rourke, M. F. "ANGINA PECTORIS." Australian and New Zealand Journal of Medicine 15, no. 4 (August 1985): 409. http://dx.doi.org/10.1111/j.1445-5994.1985.tb02760.x.

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NEWTON, JANICE L. "Angina pectoris." Nursing 28, no. 8 (August 1998): 58–60. http://dx.doi.org/10.1097/00152193-199808000-00025.

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McGoon, Michael D. "Angina Pectoris." Mayo Clinic Proceedings 61, no. 1 (January 1986): 83. http://dx.doi.org/10.1016/s0025-6196(12)61416-5.

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Bittner, Vera. "Angina Pectoris." Circulation 117, no. 12 (March 25, 2008): 1505–7. http://dx.doi.org/10.1161/circulationaha.108.764217.

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Seim, Sigurd. "Angina Pectoris." Acta Medica Scandinavica 166, no. 4 (April 24, 2009): 255–67. http://dx.doi.org/10.1111/j.0954-6820.1960.tb17377.x.

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Dissertations / Theses on the topic "Angian Pectoris"

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Kent, Marjorie Anne. "Coping with angina pectoris following diagnosis." Thesis, University of British Columbia, 1985. http://hdl.handle.net/2429/24415.

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The purpose of this study was to determine how angina pectoris patients experience and cope with their chronic illness following diagnosis. Qualitative methodology, using phenomenological theory, was used to guide the study. The study was conducted with a convenience sample of six male angina patients, ranging in age from 42 to 72 years, with recency of diagnosis from two to thirty-four months. None of these subjects were known to have had a myocardial infarction (MI). Using a semi-structured interview guide with open-ended questions, the investigator interviewed each subject at home, on two occasions. Data coding and analysis were approached using the constant comparative method developed by Glaser and Strauss (1967). The findings of the study revealed angina patients progressing through four phases of coping experience as they adjusted to their illness and its concomitant need for risk modification. These phases were identified as: (1) Initial Coping Response; (2) Managing Specific Adjustment Areas; (3) Secondary Coping Response; and (4) Awareness of Resulting Changes. The findings supported the use of a chronic illness framework adapted from Strauss et al. (1984) for identifying some of the adjustment areas encountered and coping strategies utilized by the angina patients investigated. Nurses and other health professionals are angina patients investigated. Nurses and other health professionals are in a critical position to assist angina patients in coping with their illness. The phases of coping experience identified in this study may serve as a useful guide to help these health professionals assess the nature of problems and concerns related to coping with angina so that better adjustment in daily life will ensue.
Applied Science, Faculty of
Nursing, School of
Graduate
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Würbel, Helga. "Lokaler Kälteexpositionstest der Nagelfalzkapillaren bei Patienten mit vasospastischer Angina pectoris (Prinzmetal) und bei Patienten mit stabiler Angina pectoris /." [S.l : s.n.], 1988. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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Jerrewing, Magdalena Sohlin Madeleine. "Patienters kunskap och upplevelser av angina pectoris." Thesis, Mittuniversitetet, Institutionen för hälsovetenskap, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:miun:diva-13461.

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Forslund, Lennart. "Prognostic implications of exercise induced and ambulatory ischemia in patients with stable angina pectoris /." Stockholm, 1999. http://diss.kib.ki.se/1999/91-628-3894-6/.

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Borge, Fredrik, and Lena Winberg. "Patienters erfarenheter av att leva med angina pectoris." Thesis, Högskolan i Halmstad, Akademin för hälsa och välfärd, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:hh:diva-28211.

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Ett stort antal personer insjuknar varje år i kranskärlssjukdom, vilket inkluderar angina pectoris. Sjuksköterskan behöver ha goda kunskaper om hur patienter med angina pectoris erfar olika symptom relaterat till sjukdomsbilden, samt hur patienter anterar och lever med sin sjukdom. Detta för att kunna ge bästa stöd och arbeta på ett personcentrerat sätt. Syftet var att beskriva patienters erfarenheter av att leva med angina pectoris. Metoden som användes var litteraturstudie vilken grundades på tolv vetenskapliga artiklar vilka analyserades och sammanställdes. Resultatet visade att patienter med angina pectoris har olika erfarenheter av sjukdomen. Tre teman framkom i resultatet; Patienters erfarenheter av smärta och obehag, Patienters erfarenheter av stress och oro samt Patienters erfarenheter av stöd. Då befintlig forskning mer generellt inriktas på patienter med kranskärlssjukdomar, behövs mer specifik forskning där situationen för patienter med angina pectoris belyses.
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Welsh, Colin John Percy. "Oestrogen replacement in postmenopausal women with angina pectoris." Thesis, University of Glasgow, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.421120.

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Chen, Fei. "Studies on vascular remodeling in acute coronary artery disease /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-467-8/.

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Rück, Andreas. "Myocardial gene therapy and gene expression in angina pectoris /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-648-4/.

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Silva, Silvia Sidnéia da. ""Angina pectoris instável: perfil de clientes de uma instituição privada"." Universidade de São Paulo, 2003. http://www.teses.usp.br/teses/disponiveis/22/22132/tde-27082004-093303/.

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Trata-se de estudo descritivo com abordagem de análise quantitativa que tem por objetivo identificar o perfil de clientes portadores de angina pectoris instável atendidos numa instituição privada, de um município do interior paulista. O referencial teórico utilizado foi o modelo de “Campo de Saúde” de Lalonde que analisa elementos como a biologia humana, meio ambiente, estilo de vida e organização dos serviços de saúde diante da ocorrência de patologias. A amostra constituiu-se de 58 clientes, com idade entre 34 e 88 anos, de ambos os sexos, a maioria aposentados, que reinternaram na Unidade Coronariana da referida instituição, no período de 01 de setembro de 2002 a 31 de março de 2003. A coleta de dados se deu no domicílio dos clientes, através de entrevista semi-estruturada; aplicada pela pesquisadora. Quanto à biologia humana 32,7% clientes apresentaram obesidade; os pais destes tiveram como causa mortis o infarto agudo do miocárdio e angina pectoris com índices de 24,1% e 20,7%, respectivamente; os antecedentes familiares mais freqüentes quanto às patologias foram a ocorrência de infarto agudo do miocárdio em 22,4% dos pais e hipertensão arterial sistêmica em 41,3% dos parentes próximos; 84,4% clientes possuíam hipertensão arterial sistêmica; 46,5% eram hipercolesterolêmicos; 27,5% tiveram doença vascular periférica e 17,2% apresentaram quadro de acidente vascular cerebral; valores de PAS≥140 mmHg e PAD≥90 mmHg, identificados em 71,1% e 55,7% dos clientes, respectivamente; além de valores de glicemia de jejum >110 mg/dl apresentados por 34,5% da amostra. Com relação ao meio ambiente, 55,1% dos clientes possuíam 1º grau incompleto e 8,6% eram analfabetos; 82,7% dos clientes eram casados, 65,5% não exerciam atividade remunerada e tinham renda familiar entre 03 e 06 salários mínimos. No que concerne ao estilo de vida, 100% dos clientes relacionaram a doença com fatores de risco como a hipertensão arterial sistêmica, o estresse, história familiar, dieta inadequada, tabagismo e falta de atividade física; 24,1% referiram o consumo de bebida alcoólica; 55,2% eram ex-fumantes; 37,9% dos clientes realizavam atividade física; 48,3% referiram fatos ocorridos antes da dor anginosa; 55,2% dos clientes relataram alterações de sono; inatividade sexual em 43,1% dos clientes sendo que 88,0% das clientes já estavam na menopausa e apenas 13,6% faziam terapia de reposição hormonal. No tocante ao atendimento de saúde, todos os clientes eram conveniados mas apenas 13,8% dos clientes utilizavam o serviço de medicina preventiva disponibilizado pelo convênio para prevenir doenças; o serviço de saúde pública é referência para a amostra no tocante à aquisição de medicamentos. Os achados confirmaram a interferência dos elementos referenciados pelo modelo de “Campo de Saúde” na ocorrência das patologias e identificaram a necessidade de trabalhar a mudança no estilo de vida dos clientes, através da prevenção dos fatores de risco para as doenças cardiovasculares e promoção da saúde, em geral.
Descriptive study which was carried out in order to identify customer’s profiles with unstable pectoris angina attended in a Sao Paulo’s state’s town. The theoretical model used was the “Lalonde’s Health Field Model”, which analyses elements like human biology, environment, lifestyle, and health services’ organization ahead diseases occurrences. The sample was composed by 58 customers, aged between 34 and 88 year old, masculine and feminine, most of them retired, which were readmitted in a the mentioned Coronary Unity’s institution. The data were collected at the customer’s residences and a semi- structured interview was used. Concerning about human biology, 32.7% male customers presented obesity; the parents died under acute infarct and pectoris angina and the percentual was 24,1% and 20,7%, respectively. The most common relatives’ diseases were related to the parent’s acute infarct (22,4%) and systemic arterial hypertension for the near relatives; 84.4% of the costumers suffered by systemic arterial hypertension; 46.5% had high cholesterol; 27.5% had peripheral vascular diseases and 17.2% demonstrated cerebral vascular accidents. Values like PAS≥140mmHg and PAD≥90 mmHg, identified in 71,1% and 55,7% among the customers, respectively; beyond glycemia’s values over 110mg/d, presented by 34,4% among the costumers. According to the environment, 55,1% customers didn’t study high school and 8,6% were illiterate; 82,7% were married and among them 65.5% didn’t have remunerate activity and their familiar remuneration was between 03 and 06 Brazil’s minimum salary. Related to the life style, 100% customers related the disease with risk factors’diseases as systemic arterial hypertension, stress, family history, inadequate diet, smoking and physical activity’s lack; 24,1% referred alcoholics drinks consume; 55,2% were ex-smokers; 37,9% practiced physical activity; 55.2% related sleeping problems; 43,1% related sexual inactivity and among them 88,0% female costumers already had menopause and only 13.6% were doing hormonal replacement therapy. About health services, all the costumers were had health policies but only 13.8% customers had used preventive medicine. The public health service is a reference in medicines’ acquisition. The data confirm the mentioned health field model elements in the happening of diseases and identify the need of working on changes in the costumers’ life style, through risk factors prevention for the cardiovascular diseases and health promotion.
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Mohammad, Razhan, and Peyam Eizuldeen. "Radiologiska undersökningsmetoder vid diagnostisering av Stabil Angina Pectoris : En litteraturstudie." Thesis, Örebro universitet, Institutionen för hälsovetenskaper, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:oru:diva-50532.

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Books on the topic "Angian Pectoris"

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Purcell, Julia Ann. Angina pectoris. Atlanta, Ga: Pritchett & Hull, 1985.

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Ardissino, Diego, Lionel H. Opie, and Stefano Savonitto, eds. Drug Evaluation in Angina Pectoris. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2628-5.

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Kaski, Juan Carlos. Essentials in Stable Angina Pectoris. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-41180-4.

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Shapiro, Leonard M. A colour atlas of angina pectoris. London: Wolfe Medical, 1987.

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Jackson, Graham. Angina. 2nd ed. London: Dunitz, 1995.

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Jackson, Graham. Angina. London: Dunitz, 1991.

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M, Blatt Charles, ed. Angina pectoris: Management strategies and guide to interventions. 2nd ed. Caddo, Okla: Professional Communications, Inc., 1997.

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Avanzas, Pablo, and Juan Carlos Kaski, eds. Pharmacological Treatment of Chronic Stable Angina Pectoris. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-17332-0.

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Cleophas, Ton J. M. Beta-Blockers in Hypertension and Angina Pectoris. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-011-0129-5.

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Living with angina. Oakland, CA: New Harbinger Publications, 1996.

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Book chapters on the topic "Angian Pectoris"

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Zdrenghea, Dumitru, and Dana Pop. "Angina Pectoris." In Cardiac Rehabilitation Manual, 121–35. London: Springer London, 2010. http://dx.doi.org/10.1007/978-1-84882-794-3_5.

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Liu, Zhanwen. "Angina Pectoris." In Essentials of Chinese Medicine, 55–65. London: Springer London, 2009. http://dx.doi.org/10.1007/978-1-84882-596-3_6.

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Buser, P., S. Osswald, M. Pfisterer, H. R. Zerkowski, W. Brett, and H. H. Osterhues. "Angina pectoris." In Kardiologie und Kardiochirurgie, 5–8. Heidelberg: Steinkopff, 2003. http://dx.doi.org/10.1007/978-3-642-57371-2_1.

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Raehl, Cynthia L., and Paul E. Nolan. "Angina Pectoris." In Applied Therapeutics, 213–36. London: Palgrave Macmillan UK, 1992. http://dx.doi.org/10.1007/978-1-349-13175-4_11.

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Scharnagl, Hubert, Winfried März, Markus Böhm, Thomas A. Luger, Federico Fracassi, Alessia Diana, Thomas Frieling, et al. "Angina Pectoris." In Encyclopedia of Molecular Mechanisms of Disease, 90–91. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_108.

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Zdrenghea, Dumitru, and Dana Pop. "Angina Pectoris." In Cardiac Rehabilitation Manual, 137–51. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-47738-1_5.

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Abrams, David B., J. Rick Turner, Linda C. Baumann, Alyssa Karel, Susan E. Collins, Katie Witkiewitz, Terry Fulmer, et al. "Angina Pectoris." In Encyclopedia of Behavioral Medicine, 94–95. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_1244.

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Lorimer, A. Ross, and W. Stewart Hillis. "Angina Pectoris." In Treatment in Clinical Medicine, 29–52. London: Springer London, 1985. http://dx.doi.org/10.1007/978-1-4471-3120-5_3.

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Ye, Siqin. "Angina Pectoris." In Encyclopedia of Behavioral Medicine, 113. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39903-0_1244.

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Buuren, M. J. Zaagman-van, and G. P. M. Geerlings-Bakker. "Angina pectoris." In Verpleegkundig Vademecum, 30–34. Houten: Bohn Stafleu van Loghum, 2008. http://dx.doi.org/10.1007/978-90-313-7326-0_6.

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Conference papers on the topic "Angian Pectoris"

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Falk, E. A. "UNSTABLE ANGINA PECTORIS: PATHOLOGIC ASPECTS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643711.

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Unstable angina pectoris represents a common and important manifestation of acute ischemic heart disease encompassing the broad spectrum of clinical syndromes between stable effort angina and acute myocardial infarction. This group of patientsisfar from uniform concerning underlying pathogenetic mechanisms and prognosis, but generally the risk of infarction or deathis increased during the unstable period. Most patients are presenting with new or worsening effort angina or angina at rest,and especially patients with rest anginaassociated with transient ECG changes seem to constitute a high risk subgroup. Transient reductions in coronary blood flow,rather than increases in myocardial oxygen demand, seem to play the major role in rest angina, indicating an underlying 'dynamic' coronary stenosis.Furthermore, unstable angina seems to beagood clinicalmarker for actively progressing coronary-artery disease.Pathologically, a rapidly evolving coronary-artery lesion represented by a disrupted atherosclerotic plaque with variable degree of plaque hemorrhage and luminalthrombosis usually is present in patientscoming to autopsy after a period of rest angina. The thrombus at the rupture site may be mural and limited (just sealing therupture) or occlusive depending on the degree of preexisting atherosclerotic stenosis. An occlusive thrombus is seldom seen over ruptured plaques causing less tha15% stenosis (histologic area stenosis), but is found with increasing frequency when stenosis severety increases beyond 15%.Most occlusive thrombi have a layered structure with thrombus material of differing age indicating an episodic growth by repeated mural deposits. Aggregated platelets usually can be identified in the mostrecent part of the thrombus, while older parts are more homogeneous due to fibrin infiltration/stabilization. Additionally,microemboli and microinfarcts are frequently found in the myocardium downstream tocoronary thrombi. So, the period of unstable angina preceding a fatal heart attackseems to be characterized by an ongoing thrombotic process in a major coronary artery where recurrent mural thrombus formation alternates with intermittent thrombus fragmentation and peripheral embolization. Such a dynamic thrombosis (with or without a concomitant focal vasospastic phenomenon) at the site of an unstable (ruptured) atherosclerotic lesion obviously may lead to the other clearly thrombus-related acute ischemic events: myocardial infarction or sudden death.Clinical studies using coronary angiography and coronary angioscopy during the acute phase of unstable angina have revealed a high frequency of ulcerated (unstable) atherothrombotic lesion in arteries responsible for the acute ischemia. Furthermore, episodic platelet activation (usually associated with chest pain) has recently been demonstrated in patients with unstable angina.The mechanism underlying pain/ischemia(predominantly spasm?) and the rapid plaque progression (plaque hemorr.hage/luminal thrombosis?) during unstable angina maydiffer. Accordingly, therapy directed against a possible spasm (nitrates, calcium antagonists) usually relieves pain effectively without having any documented effect on infarction/survival, while antithr-ombotic therapy (aspirin, heparin) clearlyimproves the prognosis without apparent antianginal effect. Therefore, with the objective not only of relieving pain but also of improving the prognosis, more attention should be paid to the potentially fatal thrombotic process that apparently isgoing on in a major coronary artery of many patients with unstable angina.
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Gulba, D., and P. Lichtlen. "FOUR CASES WITH STABILIZATION OF UNSTABLE ANGINA PECTORIS BY THROMBOLYTIC THERAPY." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643007.

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Acute myocardial infarctions in the vast majority of cases are caused by coronary artery thrombosis at the site of complicated arteriosclerotic plaques. By several trials evidence has been given, that myocard can be preserved, when thrombolytic therapy is started within a short period after thrombotic coronary occlusion. Recently, angioscopic evidence has been given, that the unstable angina pectoris syndrome frequently is assciated with coronary artery thrombosis, too. Thus, thrombolytic therapy should be of comparable benefit for patients suffering from unstable angina pectoris syndrome. Up to now, we have treated four patients suffering from unstable angina pectoris syndrome (two with documented spontaneous reversible ST-segment elevations, two with newly complained recurrent nocturnal episodes of severe angina) with thrombolytic therapy (Pat. 1: 1.5 Mio IE Streptokinase; Pat. 2: 100 mg rt-PA; Pat. 3: 150 mg rt-PA; Pat. 4: 60 mg scu-PA plus 200 000 IE UK). After thrombolytic therapy, all four patients were free of symptoms for at least 60 h. Pat. 3 had recurrance of chest pain with spontaneous reversible ST-segment elevations on the third day after therapy. Pat. 1, 2, and 4 were without clinical symptoms until angiography and secondary intervention (angioplasty (PTCA) /bypass operation (CABG)). Cardiac catheterization was performed within one week after thrombolytic therapy. In all four patients, ischemia related coronary artery was patent at angiography. We conclude, that in unstable angina pectoris syndromes with newly developed nocturnal symptoms and/or spontaneous reversible ST-segment elevations in the ECG can be stabilized by thrombolytic therapy. After thrombolysis, however, recurrance of chest pain may be soon, and PTCA or CABG should be performed as soon as possible.
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Koringer, C., R. Jäger, K. Huber, and K. lechner. "LEVELS OF PLASMINOGEN ACTIVATOR INHIBITOR IN PATIENTS WITH ANGINA PECTORIS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644453.

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Several groups have shown that fibrinolytic capacity is impaired in survivors of myocardial infarction, due to increased levels of the fast-acting plasminogen activator inhibitor (PAI). In order to study the behaviour of PAI in patients with coronary heart disease, 180 patients with angina pectoris were investigated. They were 148 males and 32 females, ages ranging from 29 to 70 years (52.8 ± 8.2, mean ± S.D.). A sex- and age- matched normal population served as a control (n=105, age-range 30 to 69 years, 52.4 ± 7.9). PAI was determined by a functional titration assay, and its activity expressed as arbitrary units (AU). PAI levels were significantly (p <0.005) higher in patients with angina (24.3 ± 10.3 AU/ml, range 10.1 to 112.0 AU/ml) than in normals (20.4 ± 4.6 AU/ml, range 10.5 to 31.6 AU/ml). PAI levels were unrelated to sex or age, in both the patient and the control groups. As expected, plasma triglyceride levels were correlated to PAI in patients (r=0.19, p<0.01) and in normals (r=0.20, p<0.05). Patients with a history of previous myocardial infarction (n=114) had similar PAI levels as patients without infarction (24.2 ± 11.1 AU/ml as compared to 24.4 ± 9.6 AU/ml). It is concluded that PAI levels are elevated in patients with coronary heart disease, whether myocardial infarction has taken place or not.
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Terres, W., C. Hamm, W. Kupper, and W. Bleifeld. "PLATELET AGGREGABLLITY AND METABOLISM IN PATIENTS WITH UNSTABLE ANGINA PECTORIS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643777.

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Several platelet products indicating platelet activation have been detected in blood and urine of patients (PTS) with angina pectoris (AP) at rest. Platelet activation mainly depends on local changes in the morphology or biochemical behaviour of the vessels. Whether platelet hyperaggregability is of additional importance in the pathogenesis of unstable AP is up to now unclear. In a prospective trial we therefore evaluated 32 patients (PTS) with coronary heart disease, 16 with AP at rest during the last 8 hours before blood collection and 16 age and sex matched controls with stable exertional AP. Platelet aggregation was measured upon stimulation with ADP (0.5, 1 and 10 μmol/l) and collagen (1and 5μg/ml), and c-AMP was determined in platelet rich plasma before, and, as an estimate of platelet adenylate cyclase activity, after stimulation of this enzyme with PGE 1 (10 μmol/l for 30 s). For all concentrations of both ADP and collagen no significant differences in the rates and extents of aggregation could be found between the groups. Correspondingly, the mean (±. 2 SEM) concentrations of c-AMP were similar, basally (4.1 ±.1.4 pmol/ml for PTS withunstable AP and 5.3 t 1.3 pmol/ml for PTS with stable AP)and after stimulation of platelet adenylate cyclase with PGE 1 (14.8 ± 4.1 vs. 17.2 ± 2.8 pmol/ml).Conclusion: No generalized platelet hyperaggregability could be detected in our PTS with unstable AP when compared to controls with stable exertional AP.
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Ha, Sung Ho, Zhen Yu Zhang, and Eun Kyoung Kwon. "A Hybrid Method to Predict Angina Pectoris through Mining Emergency Data." In 2010 International Conference on Information Science and Applications. IEEE, 2010. http://dx.doi.org/10.1109/icisa.2010.5480410.

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Barbieri, Enrico, Gianni Destro, Massimo Oliva, and Piero Zardini. "Acetylcholine test in patients with angina pectoris and normal coronary angiography." In Europto Biomedical Optics '93, edited by Kazuhiko Atsumi, Cornelius Borst, Frank W. Cross, Herbert J. Geschwind, Dieter Jocham, Jan Kvasnicka, Hans H. Scherer, Mario A. Trelles, and Eberhard Unsoeld. SPIE, 1994. http://dx.doi.org/10.1117/12.169117.

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Agustika, Dyah Kurniawati, Juli Astono, Sumarna, and Agus Purwanto. "Classification Between Normal Heartbeat and Angina Pectoris in Phonocardiograph Using Neural Network." In International Conference on Educational Research and Innovation (ICERI 2019). Paris, France: Atlantis Press, 2020. http://dx.doi.org/10.2991/assehr.k.200204.032.

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Beliaeva, V. A. "Daily dynamics of the frequency of emergency calls to patients with angina pectoris." In Научный диалог: Вопросы медицины. ЦНК МОАН, 2018. http://dx.doi.org/10.18411/spc-15-12-2018-02.

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Zhao, Zhilei, and Ruiyu Li. "Effects of Tongluo Huoxue Recipe Combined with Nicorandil on Cardiac Function of Stable Angina Pectoris." In 2018 9th International Conference on Information Technology in Medicine and Education (ITME). IEEE, 2018. http://dx.doi.org/10.1109/itme.2018.00177.

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Yang, Tao, Rong-Rong Jiang, Cheng-Yu Wu, and Kan-Kan She. "Combination Law of the Disease Locations and Features of Angina Pectoris due to Coronary Heart Disease." In 2015 International Conference on Medicine and Biopharmaceutical. WORLD SCIENTIFIC, 2016. http://dx.doi.org/10.1142/9789814719810_0072.

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Reports on the topic "Angian Pectoris"

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Xue, Yixuan, Qiqi Yang, Xiaolei Zhang, Yanji Zhang, and Wei Huang. Acupuncture and related therapies for stable angina pectoris: a protocol for network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2020. http://dx.doi.org/10.37766/inplasy2020.11.0035.

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Liu, Wei, Xingxing Li, Zongjing Fan, Jie Cui, and Yang Wu. The efficacy of Xuefu Zhuyu decoction combined with trimetazidine on unstable angina pectoris: A meta-analysis of randomized clinical trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2021. http://dx.doi.org/10.37766/inplasy2021.3.0073.

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Fan, Maoxia, Ying Tian, and Dong Guo. Efficacy and safety of Xinkeshu in the treatment of angina pectoris of coronary heart disease: A systematic review and meta-analysis protocol. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2021. http://dx.doi.org/10.37766/inplasy2021.9.0026.

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Huang, Langlang, Ri Xu, Xin Huang, Yusa Wang, Jianan Wang, Yanwei Liu, and Zhongyong Liu. Traditional Chinese Medicine Injection for Promoting Blood Circulation and Removing Blood Stasis in Treating Angina Pectoris of Coronary Heart Disease: A protocol for systematic review and network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2021. http://dx.doi.org/10.37766/inplasy2021.3.0103.

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