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Journal articles on the topic 'Angina-pectoris'

Author: Grafiati

Published: 4 June 2021

Last updated: 31 July 2024

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1

MALIK, ISHTIAQ AHMED, KHALID MAHMOOD, and ALI NAWAZ KHAN. "ANGINA PECTORIS." Professional Medical Journal 16, no. 02 (June 10, 2009): 202–8. http://dx.doi.org/10.29309/tpmj/2009.16.02.2902.

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Objective: To assess the clinical efficacy, cost effectiveness and side effect profile of trimetazidine in the managementof stable angina pectoris. Patients a n d M e t h o d s : An open label, uncontrolled study was conducted in 200 patients with stable angina inarmed forces institute of cardiology, Rawalpindi Pakistan. Patients were treated for 4weeks with modified release trimetazidine tablet (35mg)twice daily in addition to their conventional therapy. R e s u l t s : As compared to base line trimetazidine significantly reduced the number ofanginal episodes per week from 10 to 3 (p<0.005), improved exercise duration time on standard exercise tolerance test (ETT) (410 vs. 370sec; p<0.01), time to onset of typical angina (380 vs, 290sec; p<0.05), time to 1mm or more ST segment depression (340 vs. 290 sec;p<0.01)).There was no drop out of patients due to side effects or non compliance. C o n c l u s i o n : These results indicate that trimetazidineis effective and well tolerated when used in combination with existing antianginal therapy in patients with angina pectoris

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2

YASUE, HIROFUMI. "Angina pectoris." Nihon Naika Gakkai Zasshi 86, no. 2 (1997): 189–90. http://dx.doi.org/10.2169/naika.86.189.

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3

Goldschlager, Nora. "Angina pectoris." Postgraduate Medicine 80, no. 6 (November 1986): 147. http://dx.doi.org/10.1080/00325481.1986.11699596.

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4

Bittner, Vera. "Angina Pectoris." Circulation 117, no. 12 (March 25, 2008): 1505–7. http://dx.doi.org/10.1161/circulationaha.108.764217.

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5

O'Rourke, M. F. "ANGINA PECTORIS." Australian and New Zealand Journal of Medicine 15, no. 4 (August 1985): 409. http://dx.doi.org/10.1111/j.1445-5994.1985.tb02760.x.

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6

McGoon, Michael D. "Angina Pectoris." Mayo Clinic Proceedings 61, no. 1 (January 1986): 83. http://dx.doi.org/10.1016/s0025-6196(12)61416-5.

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7

Seim, Sigurd. "Angina Pectoris." Acta Medica Scandinavica 166, no. 4 (April 24, 2009): 255–67. http://dx.doi.org/10.1111/j.0954-6820.1960.tb17377.x.

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8

NEWTON, JANICE L. "Angina pectoris." Nursing 28, no. 8 (August 1998): 58–60. http://dx.doi.org/10.1097/00152193-199808000-00025.

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9

Aldana B., Jairo, and Alberto Suárez N. "Angina pectoris." Universitas Médica 53, no. 4 (September 9, 2012): 431–42. http://dx.doi.org/10.11144/javeriana.umed53-4.anpe.

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Se cumplen 200 años de la descripción de la angina de pecho, publicada en el New England Journal of Medicine. En este articulo se ha traducido el artículo original “Remarks on Angina Pectoris”, publicado en 1812, por el dr. John Warren.

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10

. "Angina pectoris." Huisarts en Wetenschap 47, no. 4 (April 2004): 776. http://dx.doi.org/10.1007/bf03083974.

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11

Rajanit, Sojitra, Mukesh Dungrani, Paras Virani, and Hasumati Raj. "A review on Nifedipine co-administered with Metoprolol succinate for the treatment of hypertension." International Journal of Advances in Scientific Research 1, no. 3 (April 30, 2015): 129. http://dx.doi.org/10.7439/ijasr.v1i3.1795.

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Hypertension and Angina pectoris area major public health problem in the developed Countries recently. Hypertension and Angina Pectoris are frequently treated with antihypertensive drugs like calcium-channel blockers, angiotensin-converting enzyme inhibitors, and angiotensin II (AT1) receptor blockers, and statins. Nifedipine is calcium-channel blockers and widely used in treatment of Angina pectoris condition. Metoprolol Succinate is Beta-adrenoreceptor blocker and widely used in treatment of hypertension condition. Combination of Nifedipine and Metoprolol Succinate is used in the treatment of cardiovascular diseases like hypertension and Angina Pectoris. So this combination therapy gives antihypertensive and Angina Pectoris effects in the treatment of cardiac diseases.

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12

BORTOLOTTI, M. "Angina pectoris and oesophageal angina." Gut 45, no. 4 (October 1, 1999): 630. http://dx.doi.org/10.1136/gut.45.4.630.

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13

Seyidov, V. G., A. Ya Fissun, V. V. Yevsyukov, I. V. Lyubchuk, S. Ye Bobyrev, and E. V. Arutyunov. "Long-term coronary shunting results for 5 years of observation. Factors influencing on angina pectoris recurrence after coronary shunting." Bulletin of Siberian Medicine 5, no. 3 (September 30, 2006): 105–11. http://dx.doi.org/10.20538/1682-0363-2006-3-105-111.

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Aimed at o comparing coronary shunting long-term results with drug treatment of angina pectoris as well as estimating influence of dyslipidemia, diabetes mellitus, hypoglycemic therapy nature, arterial hypertension and increased systemic inflammation on angina pectoris recurrence after the surgery, we examined 793 patients 5 years after coronary shunting and 81 patients who were treated by standard methods. Five years after the surgery, we noted decreased number of patients without angina pectoris symptoms and increased number of patients with angina pectoris. Increased levels of cholesterol, low density lipoproteins, α-lipoproteids, C-reactive proteins and diabetes mellitus of the 2-nd type II—III stage contribute to angina pectoris recurrence rate after the surgery. Operated on patients revealed angina pectoris recurrences, myocardial infarction frequency, repeated hospitalizations and annual mortality more seldom compared with patients who underwent drug treatment.

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14

Iablonska, Victoriia, Leonid Kholopov, Olena Khyzhnyak, and Viktoriia Batashova-Halinska. "The effectiveness of cytoprotection in the treatment of stable angina in patients with arterial hypertension and hyperuricemia, taking into account the peculiarities of the course of coronary artery disease in wartime." ScienceRise: Medical Science, no. 6 (51) (November 30, 2022): 4–7. http://dx.doi.org/10.15587/2519-4798.2022.268523.

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The aim of the study is to establish the effectiveness of treatment of severe angina pectoris in patients with hyperuricemia, taking into consideration the peculiarities of the course of coronary artery disease (CAD) in wartime, using ranolazine – a selective inhibitor of the late sodium flow in combined pharmacotherapy. Materials and methods. We studied the anti-anginal effect of ranolazine in 14 patients with CAD, stable angina pectoris III-IV functional class (FC), hyperuricemia and arterial hypertension (AH) during 6 months of the 2022 year. The effectiveness of the study drug on the clinical course of angina pectoris was assessed by questionnaire and clinical examination after three months of treatment. The results. At the end of the second week of ranolazine use, angina attacks at rest, which were registered before the start of the observation, stopped in all patients with angina pectoris III FC and 50 % with angina pectoris IV FC. In 78.6 %, the number of angina attacks and the use of nitrates decreased by more than 2 times; 21.4 % no longer had angina attacks. At the end of the first month, anginal attacks were not observed in all patients with angina pectoris III FC and 50 % with angina pectoris FC IV. In 2 patients with angina pectoris IV FC (50 %), anginal attacks continued to be registered during physical exertion and emotional stress, but no more than once a week. At the same time, there were no angina attacks at rest. The same results were obtained during the survey of patients at the end of three months of observation. Conclusions. Ranolazine is an effective component of anti-anginal therapy, significantly affecting the patient's quality of life. Therefore, we can recommend ranolazine for patients with hyperuricemia in angina attacks that persist with insufficient effectiveness of zasic therapy with first-line drugs, especially during military conflicts

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15

Mostafa A. Abdulrahman. "The relationship between hypertension and angina pectoris; physiological & Biochemical study of patients Samarra city." Tikrit Journal of Pure Science 20, no. 1 (February 8, 2023): 1–5. http://dx.doi.org/10.25130/tjps.v20i1.1127.

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This study was conducted for the period from 09/2013 to 02/2014 at Samarra General Hospital labs with patients admitted in the corridors of recovery for the patients with angina pectoris; ratio to the disease of hypertension is the pressure measurement during a hospital, (50-65) age nonsmoker. Reveiew as results showed high significant in hypertension for people in hypertension , angina pectoris compared with the control group in,as well as high significant in lipid profile (cholesterol, triglycerides). the high increase in angina pectoris group compared hypertension with group compared with control group at the level( P <0.05 ). As for the lipoproteins, a decrease significant in the concentration of HDL-C was the largest decrease in angina pectoris when compared with hypertension group and the control group. at the same time high LDL-C concentration in a angina pectoris when compared with the control group, as to the biochemical variety results for increase significant in the concentration of blood sugar in the two study groups .show results high in angina pectoris when compared with hypertension group and the control group ,uric acid show results high in angina pectoris when compared with hypertension group and the control group.

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16

Mandsorwale, Deepti, and Bindu Sharma. "Experimental and validation of significance and accuracy of oxidized low-density lipoproteins and myeloperoxidase in the screening of cardio-vascular disease." ScienceRise: Medical Science, no. 2(47) (March 31, 2022): 33–38. http://dx.doi.org/10.15587/2519-4798.2022.254042.

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The aim. To access the superiority of myeloperoxidase & oxidized low-density lipoproteins over each other acts as a better predictive marker gaining information regarding the severity of cardiovascular disease. Materials and methods. 215 subjects are taken into consideration of which 54 are healthy controls, 52 are from stable angina pectoris, 53 are taken from unstable angina pectoris and 56 subjects are from acute myocardial infarction. Lipid profile parameters, oxidative stress markers, plasma myeloperoxidase and plasma oxidized low density lipoproteins were estimated by kit methods, thiobarbituric acid reactive substances method, and colorimetric assay, sandwich and competitive enzyme linked immunosorbent assay techniques, respectively. Results were present as mean ± SD, p-values <0.05 as significant, and Student’s unpaired “t” test. Comparative analysis by box and whiskers plot to check skewness and deviations within the values. Data analysis was performed by software package SPSS version 17.0. Results. The oxidized low density lipoproteins levels found significantly elevated in all three cases subgroup contrary to insignificant levels of myeloperoxidase in stable angina pectoris compared to control. Box and whisker plot of myeloperoxidase levels showed no skewness in stable angina pectoris (non-significant), whereas unstable angina pectoris and acute myocardial infarction showed right skewness (highly significant), whereas plots of oxidized low-density lipoproteins show extensive interquartile range in the stable angina pectoris subgroup, suggesting scattered deviation in the mean values compared to unstable angina pectoris and acute myocardial infarction subgroup. Conclusions. The study concluded that significantly elevated level of oxidized low-density lipoproteins in stable angina pectoris, unstable angina pectoris, and acute myocardial infarction subgroups with a scattered deviation of oxidized low density lipoproteins levels in the stable angina pectoris subgroup reflects its low prognostic reliability compared to plasma myeloperoxidase with marginal deviation and in insignificant elevation in stable angina pectoris. Thus, plasma myeloperoxidase and oxidized low density lipoproteins levels serve as independent predictors of cardiovascular disease, but plasma myeloperoxidase levels predict an increased risk over oxidized low density lipoproteins for subsequent cardiovascular events in stable and unstable angina and extend the prognostic information gained from traditional biochemical markers

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17

Henderson, Robert, and Adam Timmis. "Stable angina pectoris." Srce i krvni sudovi 31, no. 4 (2012): 38–47. http://dx.doi.org/10.5937/siks1201038h.

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18

Yeghiazarians, Yerem, Joel B. Braunstein, Arman Askari, and Peter H. Stone. "Unstable Angina Pectoris." New England Journal of Medicine 342, no. 2 (January 13, 2000): 101–14. http://dx.doi.org/10.1056/nejm200001133420207.

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19

Tijssen, J. G. P., M. L. Simoons, P. J. De Feijter, P. G. Hugenholtz, and J. Lubsen. "Unstable angina pectoris." European Heart Journal 8, suppl H (October 2, 1987): 3–15. http://dx.doi.org/10.1093/eurheartj/8.suppl_h.3.

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20

Bankwala, Zehyani, and Lyle J. Swenson. "Unstable angina pectoris." Postgraduate Medicine 98, no. 6 (December 1995): 155–65. http://dx.doi.org/10.1080/00325481.1995.11946092.

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21

YEGHIAZARIANS, YEREM, JOEL B. BRAUNSTEIN, ARMAN ASKARI, and PETER H. STONE. "Unstable Angina Pectoris." Survey of Anesthesiology 44, no. 6 (December 2000): 327–28. http://dx.doi.org/10.1097/00132586-200012000-00007.

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22

Pfisterer, M. E., and Ch Kaiser. "Instabile Angina pectoris." Hämostaseologie 20, no. 01 (2000): 53–58. http://dx.doi.org/10.1055/s-0037-1619469.

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ZusammenfassungIn den letzten Jahren wurden bei der instabilen Angina pectoris sowohl im Bereich des pathophysiologischen Verständnisses, der Risikostratifizierung als auch der Therapie große Fortschritte unternommen. Nebst besserer klinischer Risikostratifizierung stehen uns heute ebenfalls biochemische Marker wie z.B. das Troponin zur initialen Risikoabschätzung zur Verfügung. Zusätzlich zur Standardtherapie mit Azetylsalizylsäure und der intravenösen Vollheparinisierung verfügen wir über neuere Medikamente wie die niedermolekularen Heparine sowie die GPIIb/IIIa-Rezeptorantagonisten, welche in größeren Studien mit oder ohne koronare Intervention ihre Wirksamkeit bewiesen haben, vorerst jedoch vor allem den Hochrisikopatienten vorbehalten sein sollten. Die interventionelle Therapie wurde insbesondere durch die Entwicklung von koronaren Stents verbessert, die Wahl des richtigen Zeitpunkts der Intervention bleibt jedoch kontrovers.

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23

Kim, Michael C., Annapoorna Kini, and Samin K. Sharma. "Refractory angina pectoris." Journal of the American College of Cardiology 39, no. 6 (March 2002): 923–34. http://dx.doi.org/10.1016/s0735-1097(02)01716-3.

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24

Becker, Richard C., and Joseph S. Alpert. "Variant Angina Pectoris." Chest 92, no. 6 (December 1987): 963–65. http://dx.doi.org/10.1378/chest.92.6.963b.

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25

Strauer, Bodo. "Stabile Angina pectoris." Der Klinikarzt 37, no. 10 (October 2008): 457. http://dx.doi.org/10.1055/s-0028-1103380.

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26

. "angina pectorisAngina pectoris." Medisch-Farmaceutische Mededelingen 38, no. 4 (April 2000): 86. http://dx.doi.org/10.1007/bf03057527.

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27

. "Instabiele angina pectoris." Medisch-Farmaceutische Mededelingen 39, no. 1 (January 2001): 20. http://dx.doi.org/10.1007/bf03057666.

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28

RIDDERVOLD, FRIDTJOV, OTTO A. SMISETH, KOLBJØRN FORFANG, and TOR FRØYSAKER. "Unstable Angina Pectoris." Acta Medica Scandinavica 224, no. 1 (April 24, 2009): 19–23. http://dx.doi.org/10.1111/j.0954-6820.1988.tb16733.x.

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29

Maseri, Attilio, Sergio Chierchia, and Juan Carlos Kaski. "Mixed angina pectoris." American Journal of Cardiology 56, no. 9 (September 1985): E30—E33. http://dx.doi.org/10.1016/0002-9149(85)91173-7.

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30

Garratt, Kirk N. "Stable angina pectoris." Current Treatment Options in Cardiovascular Medicine 2, no. 2 (April 2000): 161–72. http://dx.doi.org/10.1007/s11936-000-0009-y.

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31

Dörr, Rolf. "Stabile Angina pectoris." Herz Kardiovaskuläre Erkrankungen 31, no. 9 (December 2006): 827–35. http://dx.doi.org/10.1007/s00059-006-2938-z.

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32

Wallace, W. A., J. F. Richeson, and P. N. Yu. "Unstable angina pectoris." Clinical Cardiology 13, no. 10 (October 1990): 679–86. http://dx.doi.org/10.1002/clc.4960131002.

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33

Chaloupka, Václav. "Angina, its diagnosis and treatment." Cor et Vasa 49, no. 9 (September 1, 2007): 334–40. http://dx.doi.org/10.33678/cor.2007.116.

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34

Mathenge, Njambi, Wenjun Fan, Nathan D. Wong, Calvin Hirsch, Chris (Joseph) Delaney, Ezra A. Amsterdam, Bruce Koch, Rico Calara, and Julius M. Gardin. "Pre-diabetes, diabetes and predictors of incident angina among older women and men in the Cardiovascular Health Study." Diabetes and Vascular Disease Research 17, no. 1 (November 28, 2019): 147916411988847. http://dx.doi.org/10.1177/1479164119888476.

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Diabetes mellitus and angina pectoris are important conditions in older persons. The utility of pre-diabetes mellitus, diabetes mellitus and other risk factors as predictors of incident angina pectoris among older adults has not been characterized. We examined incident angina pectoris rates by sex and diabetes mellitus status in 4511 adults aged ⩾65 years without coronary heart disease at baseline from the Cardiovascular Health Study. Cox regression examined predictors of incident angina pectoris, including pre-diabetes mellitus or diabetes mellitus adjusted for sociodemographic characteristics and other risk factors, over 12.2 ± 6.9 years of follow-up. Overall, 39.1% of participants had pre-diabetes mellitus, 14.0% had diabetes mellitus and 532 (11.8%) had incident angina pectoris. Incident angina pectoris rates per 1000 person-years in those with neither condition, pre-diabetes mellitus, and diabetes mellitus were 7.9, 9.0 and 12.3 in women and 10.3, 11.2 and 14.5 in men, respectively. Pre-diabetes mellitus and diabetes mellitus were not independently associated with incident AP; however, key predictors of AP were male sex, low-density lipoprotein-cholesterol, triglycerides, systolic blood pressure, antihypertensive medication and difficulty performing at least one instrumental activity of daily living (all p < 0.05 to p < 0.01). In our cohort of older adult participants, while the incidence of AP is greater in those with diabetes mellitus, neither diabetes mellitus nor pre-diabetes mellitus independently predicted incident angina pectoris.

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35

S., V. "To the pathogenesis and treatment of true angina pectoris. Palya (Wien. Klin. Woch., 1926, No. 22)." Kazan medical journal 22, no. 7 (September 4, 2021): 865. http://dx.doi.org/10.17816/kazmj79534.

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36

Maya Arfania, Adinda Khoirun Nissa, Khoirul Haniatin, and Novita Andriyani. "Effectiveness of Nitrate and Beta-Blocker Drugs in Patients with Angina Pectoris: A Systematic Literature Review." Eureka Herba Indonesia 4, no. 3 (June 16, 2023): 280–84. http://dx.doi.org/10.37275/ehi.v4i3.83.

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Angina pectoris is the most common clinical manifestation of myocardial ischemia and often occurs when the heart needs more blood. The main goals of treatment in patients with unstable angina are relief of symptoms, slowing disease progression, and reducing future events, especially myocardial infarction, and death. This study aimed to explore the literature regarding the effectiveness of nitrate and beta-blocker drugs in angina pectoris patients. The literature search process was carried out on various databases (PubMed, Web of Sciences, EMBASE, Cochrane Libraries, and Google Scholar) regarding risk factors and clinical overview of acute kidney injury. The search was performed using the terms: (1) "nitric oxide" OR "beta-blockers" OR "morbidity" OR "mortality" AND (2) "angina pectoris". The literature is limited to clinical studies and published in English. Sublingual nitroglycerin has become the mainstay of treatment for angina pectoris. This drug can be used to relieve acute angina or as a prophylaxis, namely before activities that can trigger angina. Beta-blockers are a type of drug that can be used to relieve angina symptoms and prevent ischemic events through the mechanism of reducing myocardial oxygen demand, reducing heart rate, and myocardial contractility. These drugs work by competitively inhibiting the action of circulating catecholamines at cell membrane beta-adrenergic receptors. In conclusion, nitrate and beta-blocker drugs are the main choices in relieving angina pectoris symptoms.

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37

Tiwari, Ishwar, Raphael M. Herr, Adrian Loerbroks, and Shelby S. Yamamoto. "Household Air Pollution and Angina Pectoris in Low- and Middle-Income Countries: Cross-Sectional Evidence from the World Health Survey 2002–2003." International Journal of Environmental Research and Public Health 17, no. 16 (August 11, 2020): 5802. http://dx.doi.org/10.3390/ijerph17165802.

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The evidence regarding the effects of household air pollution on angina pectoris is limited in low-and middle-income countries (LMICs). We sought to examine the association between household air pollution and angina pectoris across several countries. We analyzed data of individuals from 46 selected countries participating in the cross-sectional World Health Survey (WHS) 2002–2003. Pooled and stratified (sex, continent) logistic regression with sampling weights was used to estimate adjusted odds ratios (ORs) and 95% confidence intervals (CIs) to quantify associations between the use of different household fuels with angina pectoris. In the pooled sample, we observed lower odds of angina pectoris with electricity use (OR: 0.68, 95% CI: 0.56–0.83) compared to those households reporting the use of gas as a household fuel. Increased odds of angina pectoris were observed with the use of agriculture/dung/shrub/other (OR: 1.65, 95% CI: 1.30–2.09), mixed (solid and non-solid fuels) (OR: 1.31, 95% CI: 1.09–1.56), and mixed solid fuel use (OR: 1.59, 95% CI: 1.12–2.25). Higher odds of angina pectoris were observed mainly with solid fuel use. The results highlight the importance of addressing these issues, especially in regions with a high proportion of solid fuel users and increasing levels of cardiovascular disease.

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38

Shang, Qinghua, Hao Xu, Zhaolan Liu, Keji Chen, and Jianping Liu. "OralPanax notoginsengPreparation for Coronary Heart Disease: A Systematic Review of Randomized Controlled Trials." Evidence-Based Complementary and Alternative Medicine 2013 (2013): 1–12. http://dx.doi.org/10.1155/2013/940125.

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This systematic review aims to evaluate current evidence for the benefit and side effect of oralPanax notoginsengpreparation for coronary heart disease (CHD). We included 17 randomized clinical trials (17 papers and 1747 participants). Comparing with no intervention on the basis of conventional therapy, oralPanax notoginsengdid not show significant effect on reducing cardiovascular events, but it could alleviate angina pectoris (including improving the symptoms of angina pectoris [RR 1.20; 95% CI 1.12 to 1.28; 7 trials,n=791], improving electrocardiogram [RR 1.35; 95% CI 1.19 to 1.53; 8 trials,n=727], decreasing the recurrence of angina pectoris [RR 0.38; 95% CI 0.16 to 0.94; 1 trials,n=60], duration of angina pectoris [RR −1.88; 95% CI −2.08 to −1.69; 2 trials,n=292], and dosage of nitroglycerin [MD −1.13; 95% CI −1.70 to −0.56; 2 trials,n=212]); oralPanax notoginsenghad no significant difference compared with isosorbide dinitrate on immediate effect for angina pectoris [RR 0.96; 95% CI 0.81 to 1.15; 1 trial,n=80]. In conclusion, oralPanax notoginsengpreparation could relieve angina pectoris related symptoms. However, the small sample size and potential bias of most trials influence the convincingness of this conclusion. More rigorous trials with high quality are needed to give high level of evidence, especially for the potential benefit of cardiovascular events.

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39

Laederach-Hofmann, Kurt, Clemens Turniger, Lutz Mussgay, and Ralph Jürgensen. "Sensorische und affektive Komponenten im Gebrauch von Schmerzwörtern bei Patienten mit Angina Pectoris und koronarer Herzkrankheit oder Syndrom-X." Zeitschrift für Klinische Psychologie und Psychotherapie 30, no. 3 (July 2001): 182–88. http://dx.doi.org/10.1026/0084-5345.30.3.182.

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Zusammenfassung. Ziel: Untersuchung von Patienten mit Syndrom-X im Vergleich zu solchen mit koronarstenotisch bedingter Angina pectoris im Hinblick auf Unterschiede in verbal-inhaltlichen Aspekten der Schmerzbeschreibung. Methode: Einundsechzig Patienten mit Angina pectoris wurden untersucht, 32 davon mit Syndrom-X (Angina pectoris, abnorme Ergometrie und normale Koronarangiographie) und 29 mit koronarer Herzkrankheit (Angina Pectoris, abnorme Ergometrie, angiographisch dokumentierte koronare 1-3-Gefässerkrankung, kein vorgängiger Myokardinfarkt). Neben einem klinisch semi-strukturierten Interview werden dafür die Hamburger Schmerz Adjektiv Liste, die Schmerzempfindungsskala und die Berner Version des McGill Pain Questionnaire verwendet. Ergebnisse: Patienten mit Syndrom-X weisen bei sensorischen Adjektiven in der Hamburger Schmerz Adjektiv Liste und in der Schmerzempfindungsskala signifikant geringere Werte auf als solche mit koronare Herzkrankheit. Für die affektiven Adjektive gibt es lediglich signifikante Unterschiede in der Hamburger Schmerz Adjektiv Liste zwischen beiden Patientengruppen. Im McGill Pain Questionnaire erwiesen sich die Unterschiede zwischen den Gruppen sowie zwischen den Fragebögen als nicht signifikant. Schlußfolgerungen: Die Unterschiede in den Fragebogendaten erlauben zwar keine diagnostische Trennung der beiden Gruppen, zeigen jedoch eine höhere affektive Schmerzbewertung bei Angina pectoris mit koronarer Herzkrankheit.

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40

Kassim, Wissam jabbar. "Assessment of physical Domain for patients with angina pectoris." Kufa Journal for Nursing Sciences 2, no. 3 (December 25, 2012): 111–23. http://dx.doi.org/10.36321/kjns.vi20123.2538.

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Objective: To assess the physical domain for patients with angina pectoris. Methodology: A descriptive study was carried out, in March 15th 2012 to June 15th 2012, to assess physical domain for patients with angina pectoris. A purposive sample of (50) patients with angina pectoris was selected out of four coronary care unites hospital in Baghdad in coronary care units. A questionnaire was adapted for purpose of the study, which comprised of four major parts in which there were (24) items. The data were analyzed by through the use of descriptive statistical approach data analysis (frequency, percentage, mean of scores) and inferential statistical data analysis approach (chi-square). Results: The findings of the study indicated that there was relationship between sex and physical domains and their age. In conclusion, the results indicated that individuals with angina pectoris were old age group and the majorities were males. Recommendation: The study recommended that an education program should be designed to increase individuals' information toward daily physical activity (physical domains) and disease prevention and to reduce the risk of angina pectoris.

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Yanto, Suyanto Yanto. "PENGGUNAAN TEKNIK GUIDED IMAGERY DALAM MENURUNKAN NYERI DADA PASIEN ANGINA PECTORIS." Jurnal Keperawatan dan Kesehatan 12, no. 1 (April 30, 2021): 7–12. http://dx.doi.org/10.54630/jk2.v12i1.142.

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ABSTRACT The main problem in Angina pectoris is transient chest pain or a feeling of pressure that occurs when the heart muscle is deprived of oxygen. Non-pharmacological therapy is a decision to help pharmacological therapy in reducing the scale of pain quickly, one of which is the Guided Imagery technique. This study aims to determine the effect of Guided imagery techniques on reducing pain in patients with Angina pectoris in the ICVCU Room of the Regional General Hospital Dr. Moewardi, Surakarta in 2019. This type of research is experimental with pre-experimental design with one-group type pre-test design. The instrument of data collection in this study was the Standard Operational Procedure Guided Imagery and observation sheet which included the name, age, sex and scale of pain before and after Guided imagery techniques. The subjects in this study were Angina pectoris patients according to inclusion and exclusion criteria. The analysis used was the Wilcoxon test. The results of data analysis on 40 respondents who were given Guided imagery relaxation showed a decrease in pain scale as evidenced by a significance value of 0,000, where the value of p <0.05 so that H0 was rejected. There is an effect of the Guided imagery technique on reducing patient pain with Angina pectoris as evidenced by changes in pain scale. Keywords: Angina pectoris, Guided imagery, Pain. Abstrak Permasalahan utama pada Angina pectoris adalah nyeri dada sementara atau suatu perasaan tertekan yang terjadi jika otot jantung mengalami kekurangan oksigen. Terapi non farmakologi merupakan suatu keputusan untuk membantu terapi farmakologi dalam menurunkan skala nyeri secara cepat salah satunya dengan teknik Guided imagery. Penelitian ini bertujuan untuk mengetahui pengaruh teknik Guided imagery terhadap penurunan nyeri pasien dengan Angina pectoris di Ruang ICVCU RSUD Dr. Moewardi, Surakarta tahun 2019. Jenis penelitian ini adalah eksperimental dengan desain pra eksperimen dengan jenis one-grup pra post-test design. Instrumen pengumpulan data pada penelitian ini adalah Standar Operational Procedure Guided imagery dan lembar observasi yang meliputi nama, umur, jenis kelamin dan skala nyeri sebelum serta setelah dilakukan teknik Guided imagery. Subyek pada penelitian ini adalah pasien Angina pectoris sesuai kriteria inklusi dan ekslusi. Analisis yang digunakan adalah uji wilcoxon. Hasil analisa data pada 40 responden yang diberikan relaksasi Guided imagery menunjukkan adanya penurunan skala nyeri dibuktikan dengan nilai significancy 0,000, dimana nilai p < 0,05 sehingga H0 ditolak. Ada pengaruh teknik Guided imagery terhadap penurunan nyeri pasien dengan Angina pectoris dibuktikan dengan perubahan skala nyeri. Kata Kunci : Angina pectoris, Guided imagery, Nyeri

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Tobing, Erna R., Jusak Nugraha, and Muhammad Amminuddin. "DIAGNOSTIC CONCORDANCE BETWEEN NEXT-GENERATION AND HIGH SENSITIVE TROPONIN-I IN ANGINA PECTORIS PATIENTS." INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY 24, no. 1 (March 29, 2018): 64. http://dx.doi.org/10.24293/ijcpml.v24i1.1158.

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Angina pectoris merupakan gejala klinis Sindrom Koroner Akut (SKA) yang mengarah pada penyakit jantung koroner. Sindromkoroner akut terdiri dari Unstable Angina dan Infark Miokard Akut (IMA). Kadar Troponin I (TnI) dapat mendukung penegakkandiagnosis IMA di pasien angina pectoris. Beberapa metode pemeriksaan TnI semakin berkembang diantaranya TnI high sensitive (TnI hs)dan TnI next-generation (TnI ng). Tujuan penelitian ini adalah menganalisis kesesuaian diagnostik antara kadar TnI ng yang diperiksamenggunakan metode Fluorescent Enzyme Transfer Latex (FETL) [Alere Triage MeterPro®] dan TnI hs dengan metode ChemiluminescentImmunoassay (CLEIA) [Mitsubishi PathFast®] di pasien angina pectoris. Penelitian dilaksanakan di RSUD Dr.Soetomo Surabaya masawaktu Maret-Juli 2016 dengan rancangan penelitian potong lintang. Sebanyak 82 subjek penelitian dengan gejala angina pectorisdiperiksakan kadar Troponin-I menggunakan kedua metode. Subjek penelitian sebanyak 44% didiagnosis SKA, dan 56% non SKA. Nilaikesesuaian koefisien kappa antara TnI ng dan TnI hs di pasien angina pectoris adalah 0,738 (p<0,01). Kepekaan dan kekhasan TnI ngterhadap TnI hs untuk diagnosis IMA dengan cut off 0,02 ng/mL adalah 94% dan 78%. Analisis kenasaban antara kadar TnI ng danTnI hs dengan koefisien kenasaban Spearman rho (ρ) adalah 0,826 (p<0,01). Terdapat kesesuaian diagnostik antara TnI ng dan TnI hsdi pasien angina pectoris. Kedua metode pemeriksaan TnI dapat digunakan untuk membantu menegakkan diagnosis di pasien anginapectoris. Penelitian lebih lanjut diperlukan untuk mengetahui nilai prognosis TnI.

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Jiang, Li, Zhaoxia Deng, Hongcai Zhang, Yuan Li, Tingting Wang, and Wen Xie. "Acupoint for angina pectoris." Medicine 100, no. 3 (January 22, 2021): e24080. http://dx.doi.org/10.1097/md.0000000000024080.

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&NA;. "Chronic stable angina pectoris." Inpharma Weekly &NA;, no. 1193 (June 1999): 4. http://dx.doi.org/10.2165/00128413-199911930-00006.

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Kaplinsky, Elieser. "Management of Angina Pectoris." Drugs 43, Supplement 1 (1992): 9–14. http://dx.doi.org/10.2165/00003495-199200431-00004.

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Brest, Albert N. "Management of Angina Pectoris." Primary Care: Clinics in Office Practice 12, no. 1 (March 1985): 91–100. http://dx.doi.org/10.1016/s0095-4543(21)01242-2.

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GERHOLD, WALTER M. "Surgery for Angina Pectoris." Annals of Internal Medicine 103, no. 3 (September 1, 1985): 477. http://dx.doi.org/10.7326/0003-4819-103-3-477_2.

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Dalal, J. N., and A. C. Jain. "Chronic stable angina pectoris." Postgraduate Medicine 91, no. 4 (March 1992): 165–77. http://dx.doi.org/10.1080/00325481.1992.11701251.

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Eff, Jack, John Godfrey, Ronald Garutti, and Paolo Capone. "Celiprolol in Angina Pectoris." Journal of Cardiovascular Pharmacology 8, Supplement 4 (1986): S132—S134. http://dx.doi.org/10.1097/00005344-198608004-00030.

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Schweizer, Walter. "ISOPROPYLHYDRAZIDES IN ANGINA PECTORIS." Annals of the New York Academy of Sciences 80, no. 3 (December 15, 2006): 1016–19. http://dx.doi.org/10.1111/j.1749-6632.1959.tb49276.x.

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