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Relevant bibliographies by topics / Angiogenesis, Autophagy, ROP

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Journal articles
Dissertations / Theses

Academic literature on the topic 'Angiogenesis, Autophagy, ROP'

Author: Grafiati

Published: 14 March 2023

Last updated: 31 July 2025

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Journal articles on the topic "Angiogenesis, Autophagy, ROP"

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Maugeri, N., G. A. Ramirez, A. Monno, et al. "POS0152 PLATELET EXTRACELLULAR VESICLES MODULATE NEUTROPHIL STATE IN SYSTEMIC SCLEROSIS VIA HMGB1." Annals of the Rheumatic Diseases 82, Suppl 1 (2023): 297. http://dx.doi.org/10.1136/annrheumdis-2023-eular.3082.

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BackgroundPlatelet extracellular vesicles (Plt-EVs) expressing the damage-associated molecular pattern prototypic signal, high mobility group box 1 (HMGB1), accumulate in the blood of patients with systemic sclerosis (SSc) and per se promote autoimmunity, fibrosis and vascular inflammation [1, 2]. A poorly understood defect in critical players in sprouting angiogenesis, angiopoietins and the angiopoietin receptor, TIE2 is also an hallmark of SSc. Normal neutrophils respond to angiopoietin 1 and other stimuli by undergoing activation and upregulating the Tie2 expression [3].ObjectivesWe reasone

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Baral, April J., Gabriel C. Nwokolo, Sarah Mcgowan, Steven D. Shnyder, and Robert A. Falconer. "Abstract 5586: In vitro investigation of the proliferative and metastatic role of CD13 in cancer." Cancer Research 85, no. 8_Supplement_1 (2025): 5586. https://doi.org/10.1158/1538-7445.am2025-5586.

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CD13 (Aminopeptidase N,APN) is a glycosylated ectopeptidase with diverse physiological functions including proliferation, differentiation, migration and angiogenesis1. CD13 is a marker for neoangiogenesis that is detected in tumor neovasculature, but not in the normal vasculature, contributing with tumor growth, invasion, and metastasis. In vitro studies have revealed association between CD13 various cancer types including human glioblastoma2 and prostate3 cancers. Thus, CD13 is an appealing target for the delivery of drugs in CD13-expressing tumors which can reduce tumor growth and progressio

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Liu, Xianyang, Qian Zhou, Jiayu Meng, et al. "Autophagy‐mediated activation of the AIM2 inflammasome enhances M1 polarization of microglia and exacerbates retinal neovascularization." MedComm 5, no. 8 (2024). http://dx.doi.org/10.1002/mco2.668.

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AbstractRetinopathy of prematurity (ROP) is a retinal neovascularization (RNV) disease that is characterized by abnormal blood vessel development in the retina. Importantly, the etiology of ROP remains understudied. We re‐analyzed previously published single‐cell data and discovered a strong correlation between microglia and RNV diseases, particularly ROP. Subsequently, we found that reactive oxygen species reduced autophagy‐dependent protein degradation of absent in melanoma 2 (AIM2) in hypoxic BV2 cells, leading to increased AIM2 protein accumulation. Furthermore, we engineered AIM2 knockout

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Ash, Dipankar, Sudhahar Varadarajan, Seock-Won Youn, et al. "Abstract 13610: Endothelial Cu Transporter Atp7a Promotes Vegfr2 Signaling and Post-ischemic Neovascularization via Regulating Autophagy." Circulation 142, Suppl_3 (2020). http://dx.doi.org/10.1161/circ.142.suppl_3.13610.

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Background: VEGFR2 (KDR/Flk1) signaling in endothelial cells (ECs) plays a central role in angiogenesis. Copper (Cu) is essential micronutrient and has been implicated in angiogenesis. The P-type ATPase transporter ATP7A is key regulator of Cu homeostasis but its role in VEGFR2 signaling in ECs and post-ischemic neovascularization is entirely unknown. Results: Here we show that ATP7A expression was dramatically increased in the angiogenic ECs in mice hindlimb ischemia model. EC-specific ATP7A deficient mice or Cu transporter-dysfunctional ATP7A mut mice showed significant decrease in blood flo

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Ash, Dipankar, Varadarajan Sudhahar, Seock-Won Youn, et al. "The P-type ATPase transporter ATP7A promotes angiogenesis by limiting autophagic degradation of VEGFR2." Nature Communications 12, no. 1 (2021). http://dx.doi.org/10.1038/s41467-021-23408-1.

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AbstractVEGFR2 (KDR/Flk1) signaling in endothelial cells (ECs) plays a central role in angiogenesis. The P-type ATPase transporter ATP7A regulates copper homeostasis, and its role in VEGFR2 signaling and angiogenesis is entirely unknown. Here, we describe the unexpected crosstalk between the Copper transporter ATP7A, autophagy, and VEGFR2 degradation. The functional significance of this Copper transporter was demonstrated by the finding that inducible EC-specific ATP7A deficient mice or ATP7A-dysfunctional ATP7Amut mice showed impaired post-ischemic neovascularization. In ECs, loss of ATP7A in

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Li, Baolong, Zhengtai Chen, Xiaobin Luo, et al. "Butylphthalide Inhibits Autophagy and Promotes Multiterritory Perforator Flap Survival." Frontiers in Pharmacology 11 (January 29, 2021). http://dx.doi.org/10.3389/fphar.2020.612932.

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Multiterritory perforator flap is an important plastic surgery technique, yet its efficacy can be limited by partial necrosis at the choke Ⅱ zone. Butylphthalide (NBP) has been used for many diseases but has not been studied in the multiterritory perforator flap. With the effect of NBP, we observed increasing in capillary density, inhibition of autophagy and oxidative stress, and a reduction in apoptosis of cells, all consistent with increased flap survival. However, the protective effect of NBP on multiterritory perforator flap was lost following administration of the autophagy agonist rapamy

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Li, Zihao, Qing Wei, Yijun Li, et al. "Dexmedetomidine regulates the anti-oxidation and autophagy of adipose-derived stromal cells under H2O2-induced oxidative stress through Nrf2/p62 pathway and improves the retention rate of autologous fat transplantation." Frontiers in Pharmacology 15 (November 25, 2024). http://dx.doi.org/10.3389/fphar.2024.1453938.

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To investigate the protective mechanism of dexmedetomidine (DEX) on adipose-derived stromal cells (ADSCs) under oxidative stress model and its promotion effect on the retention rate of adipose granule transplantation by in vitro and in vivo experiments. The experiment was divided into control group, model group (ADSCs + H2O2+normal serum), DEX group (ADSCs + H202+DEX drug-containing serum), autophagy agonist group (ADSCs + H2O2+rapamycin (RAP)+normal serum), RAP + DEX group (ADSCs + H2O2+normal serum), RAP + DEX drug-containing serum), autophagy inhibitor group (ADSCs + H2O2+chloroquine (CQ)+n

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Wang, Yan-Xia, Hong-Yu Zheng, Kun Zhou, et al. "Multifaceted Nature of HuR in Atherosclerosis Development." Current Medicinal Chemistry 31 (January 15, 2024). http://dx.doi.org/10.2174/0109298673279032231214110313.

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Abstract: HuR (Human antigen R) is an RNA binding protein (RBP) that specifically binds to certain RNA sequences, influencing post-transcriptional regulation. HuR is primarily involved in tumor regulation, as well as cell growth, proliferation, inflammation, and angiogenesis. HuR is implicated in endothelial activation, smooth muscle proliferation, inflammatory response, macrophage apoptosis, lipid regulation, and autophagy, playing a crucial regulatory role in atherosclerosis. Accumulating evidence suggests that HuR has dual roles in AS. On the one hand, HuR expedites the development of AS by

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Xue, Lianguo, Tao Jia, Yuanxin Zhu, Lidong Zhao, and Jianping Mao. "Down-regulation of circ_0058058 suppresses proliferation, angiogenesis and metastasis in multiple myeloma through miR-338-3p/ATG14 pathway." Journal of Orthopaedic Surgery and Research 16, no. 1 (2021). http://dx.doi.org/10.1186/s13018-021-02867-8.

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Abstract Background Multiple myeloma (MM) is one of the most frequently diagnosed hematological malignancy. Dysregulation of circular RNAs (circRNAs) has important impacts on MM process. Herein, this work aimed to investigate the role and mechanism of circ_0058058 in MM progression. Methods Levels of genes and proteins were detected by real-time reverse transcription PCR (RT-qPCR) and Western blot. CCK-8 assay, colony formation assay, EdU assay, flow cytometry, tube formation assay, transwell assay and Western blot were utilized to detect the proliferation, apoptosis, angiogenesis and metastas

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Dolomatov, S.I., та W. Zukow. "Эпигенетика почек = Kidneys epigenetics". 7 липня 2019. https://doi.org/10.5281/zenodo.3270754.

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<strong>Dolomatov S.I., Zukow W. </strong><strong>Эпигенетика почек</strong><strong> = Kidney</strong><strong>s</strong><strong> epigenetics</strong><strong>. </strong><strong>RSW. Radom,</strong><strong> 144 </strong><strong>p. ISBN </strong><strong>9780359774524</strong><strong>.</strong><strong> DOI </strong><strong>http://dx.doi.org/10.5281/zenodo.3270699</strong><strong> PBN Poland </strong><strong>https://pbn.nauka.gov.pl/sedno-webapp/works/917606</strong>           <strong>Radomska Szkoła Wyższa w Radomiu, Radom, Poland</strong>         &nbsp

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Dissertations / Theses on the topic "Angiogenesis, Autophagy, ROP"

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Pesce, Noemi Anna. "MOLECULAR MECHANISMS OF ANGIOGENESIS-RELATED OCULAR DISEASES IN PRECLINICAL MODELS." Doctoral thesis, Università di Siena, 2021. http://hdl.handle.net/11365/1148688.

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Neovascularization in the eye contributes to visual loss in several ocular diseases, including proliferative diabetic retinopathy (PDR), neovascular glaucoma (NVG) and retinopathy of prematurity (ROP). In these diseases, the neovascular mechanisms originate from the retina, but some advanced stages of PDR or nG can lead to the development of rubeosis iridis (RI), a clinical manifestation characterized by iris angiogenesis. Vascular endothelial growth factors (VEGFs) and their receptor (VEGFRs) are the key promoters of angiogenesis, playing a crucial role in both physiological and pathological

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