Relevant bibliographies by topics / Angiotensin-1 converting enzyme

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Angiotensin-1 converting enzyme'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Angiotensin-1 converting enzyme"

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Shaha, Kunal Bikram, Ashok Adhikari, Jung Rae Cho, Bimal Pandey, Yubaraj Sharma, and Sajjad Safi. "Angiotensin-Converting Enzyme Inhibitor / Angiotensin II Receptor Blocker in COVID-19: a Double-edged Sword or a Myth." Nepalese Medical Journal 3, no. 1 (2020): 309–12. http://dx.doi.org/10.3126/nmj.v3i1.28726.

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Angiotensin-converting enzyme-2 receptor has been unearthed as a prime site of entry of Severe Acute Respiratory Syndrome Coronavirus 2 owing to its strong affinity towards spike protein of Severe Acute Respiratory Syndrome Coronavirus 2, resulting in down-regulation of Angiotensin-converting enzyme -2 receptors and hyperstimulation of Angiotensin-converting enzyme-1 pathway. This proposed theory has led to the birth of a new controversy regarding the use of Angiotensin-converting enzyme inhibitors/angiotensin receptor blockers in Coronavirus disease 2019 patients. A theory is against the use
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Ferrario, Carlos M. "Angiotensin-Converting Enzyme 2 and Angiotensin-(1-7)." Hypertension 47, no. 3 (2006): 515–21. http://dx.doi.org/10.1161/01.hyp.0000196268.08909.fb.

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Chadwick, I. G., M. Kraskiewicz, W. W. Yeo, K. S. Higgins, P. R. Jackson, and L. E. Ramsay. "No Relation between Angiotensin-Converting Enzyme Gene Polymorphism and Dermal Responses to Bradykinin in Healthy Subjects." Clinical Science 91, no. 5 (1996): 617–20. http://dx.doi.org/10.1042/cs0910617.

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1. The dermal wheal response to bradykinin is increased by drugs which inhibit angiotensin-converting enzyme, and thus provides a measure of angiotensin-converting enzyme activity at the tissue level. An insertion/deletion polymorphism of the angiotensin-converting enzyme gene predicts serum angiotensin-converting enzyme activity, but its relation to angiotensin-converting enzyme activity in tissue is unclear. 2. The relations between angiotensin-converting enzyme genotype and wheal responses to intradermal bradykinin were studied in 105 healthy subjects: 30 of genotype DD, 51 of genotype ID a
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Ferrario, Carlos M., and Jasmina Varagic. "The ANG-(1–7)/ACE2/mas axis in the regulation of nephron function." American Journal of Physiology-Renal Physiology 298, no. 6 (2010): F1297—F1305. http://dx.doi.org/10.1152/ajprenal.00110.2010.

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The study of experimental hypertension and the development of drugs with selective inhibitory effects on the enzymes and receptors constituting the components of the circulating and tissue renin-angiotensin systems have led to newer concepts of how this system participates in both physiology and pathology. Over the last decade, a renewed emphasis on understanding the role of angiotensin-(1–7) and angiotensin-converting enzyme 2 in the regulation of blood pressure and renal function has shed new light on the complexity of the mechanisms by which these components of the renin angiotensin system
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Li, Ruyin, Tongtong Tang, Feifei Ma, et al. "Identification of Three Novel Angiotensin-I-Converting Enzyme Inhibitory Peptides from Cassia Obtusifolia Seeds and Evaluation of their Inhibition Mechanisms." Current Topics in Nutraceutical Research 22, no. 1 (2023): 108–15. http://dx.doi.org/10.37290/ctnr2641-452x.22:108-115.

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Angiotensin-I-converting enzyme inhibitory peptides were isolated from Cassia obtusifolia seeds by alcalase hydrolysis. Using ultrafiltration, the peptides were divided into four fractions (<1, 1–3, 3–5, >5 kDa). The fraction below 1 kDa exhibited the appropriate ACE inhibition (IC50 = 65.88 μg/mL), and was further purified by gel filtration chromatography, which displayed better angiotensin-I-converting enzyme inhibitory activity (IC50 = 53.67 μg/mL). The amino acid sequences of three novel angiotensin-I-converting enzyme inhibitory peptides were identified by liquid chromatography with
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Stevens, B. R., A. Fernandez, and C. del Rio Martinez. "Angiotensin converting enzyme in brush-border membranes of avian small intestine." Journal of Experimental Biology 135, no. 1 (1988): 1–8. http://dx.doi.org/10.1242/jeb.135.1.1.

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Angiotensin converting enzyme activity was identified in brush-border membranes purified from the small intestinal epithelium of the common grackle, Quiscalus quiscula. Angiotensin converting enzyme was enriched 20-fold in the membrane preparation, compared with intestinal epithelial cell scrapes, and was coenriched with the brush-border markers, alkaline phosphatase and aminopeptidase N. The kinetics of hydrolysis of N-[3-(2-furyl)acryloyl]-L-phenylalanylglycylglycine (FAPGG) gave a Vmax of 907 +/− 41 units g-1 and a Km of 55 +/− 6 mumol l-1. The avian intestinal angiotensin converting enzyme
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Chappell, Mark C., Nancy T. Pirro, Angela Sykes, and Carlos M. Ferrario. "Metabolism of Angiotensin-(1–7) by Angiotensin-Converting Enzyme." Hypertension 31, no. 1 (1998): 362–67. http://dx.doi.org/10.1161/01.hyp.31.1.362.

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Praddaude, Françoise, Tuan Tran-Van, Jeannine Marchetti, Jean-Pierre Girolami, and Jean-Louis Ader. "Effects of Chronic Administration of an Angiotensin-Converting Enzyme Inhibitor on Angiotensin-Converting Enzyme Activity in the Pars Recta of Rabbits." Clinical Science 79, no. 1 (1990): 29–35. http://dx.doi.org/10.1042/cs0790029.

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1. To determine whether chronic angiotensin-converting enzyme inhibition induces a decrease in proximal tubular angiotensin-converting enzyme activity, urine and blood samples were collected in conscious New Zealand rabbits before and after 16 days administration in drinking water of low doses of captopril (2.6 ± 0.6 mg 24 h−1 kg−1), high doses of captopril (7.6 ± 0.9 mg 24 h−1 kg−1) or no captopril (controls). The kidneys were then removed and angiotensin-converting enzyme activity was determined in isolated pars recta of microdissected nephrons as pmol of tritiated hippurylglycylglycine subs
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Moniwa, Norihito, Jasmina Varagic, Stephen W. Simington, et al. "Primacy of angiotensin converting enzyme in angiotensin-(1–12) metabolism." American Journal of Physiology-Heart and Circulatory Physiology 305, no. 5 (2013): H644—H650. http://dx.doi.org/10.1152/ajpheart.00210.2013.

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Angiotensin-(1–12) [ANG-(1–12)], a new member of the renin-angiotensin system, is recognized as a renin independent precursor for ANG II. However, the processing of ANG-(1–12) in the circulation in vivo is not fully established. We examined the effect of angiotensin converting enzyme (ACE) and chymase inhibition on angiotensin peptides formation during an intravenous infusion of ANG-(1–12) in normotensive Wistar-Kyoto rats (WKY) and spontaneously hypertensive rats (SHR). WKY and SHR were assigned to a short ANG-(1–12) infusion lasting 5, 15, 30, or 60 min ( n = 4–10 each group). In another exp
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Butler, R., A. D. Morris, and A. D. Struthers. "Angiotensin-Converting Enzyme Gene Polymorphism and Cardiovascular Disease." Clinical Science 93, no. 5 (1997): 391–400. http://dx.doi.org/10.1042/cs0930391.

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1. The crucial role played by the renin-angiotensin—aldosterone system in the cardiovascular system and the immense therapeutic potential of angiotensin-converting enzyme inhibitors and, more recently, angiotensin II receptor blocking agents, in both heart failure and post-myocardial infarction is becoming increasingly evident. Polymorphisms within the genes controlling this enzyme system are candidates for the elucidation of the pathogenesis of cardiovascular disease and this link is both intriguing and provocative. Recently, an association between a polymorphism of the angiotensin-converting
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Dissertations / Theses on the topic "Angiotensin-1 converting enzyme"

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Hocharoen, Lalintip. "Catalytic Metallopeptide Promoted Inactivation of Enzyme Targets Related to Disease: Angiotensin Converting Enzyme-1 and SortaseA." The Ohio State University, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=osu1354634371.

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Serfözö, Peter Daniel [Verfasser]. "Angiotensin-Converting Enzyme 2- and Prolyl Carboxypeptidase-Independent Conversion of Angiotensin II to Angiotensin-(1-7) in Circulation and Peripheral Tissues / Peter Daniel Serfözö." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2020. http://d-nb.info/1218076844/34.

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Dean, Stephanie A. "Regulation of angiotensin converting enzyme and angiotensin II type 1 receptor by 17beta-estradiol in female rats: Implications following experimental myocardial infarction." Thesis, University of Ottawa (Canada), 2005. http://hdl.handle.net/10393/26883.

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The present studies tested the hypothesis that 17beta-estradiol (E2) downregulates ACE and AT1R in several tissues important to cardiovascular regulation, including the brain and heart, and that this downregulation attenuates the progression of LV dysfunction following myocardial infarction (MI). In Experiment 1, female Wistar rats were randomized into one of four groups: (1) sham-ovariectomized (OVX); (2) OVX+vehicle (veh); (3) OVX+E2 replacement at physiological levels and (4) OVX+high E2. Five weeks following OVX, ACE and ATIR were increased 15-90% in the heart, several cardiovascular nucle
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Chisi, John Eugenes. "The regulatory role of AcSDKP and angiotensin 1-converting enzyme (ACE) inhibitors on haematopoietic stem and progenitor cell proliferation." Thesis, University of St Andrews, 1999. http://hdl.handle.net/10023/14971.

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Negative regulatory factors inhibit the proliferation of haematopoietic stem cells thus protecting them from differentiation pressures. One of the negative regulators of stem cell proliferation is the tetrapeptide Acetyl-Seryl-Aspartyl-Lysyl-Proline (AcSDKP). This peptide is endogenously produced in vivo and long term bone marrow cultures and is degraded by angiotensin 1-converting enzyme (ACE) both in vivo and in vitro. The aim of these investigations was to study the role of ACE on haematopoietic stem and progenitor cell proliferation. Since the N-domain ACE active has been implicated in AcS
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Wang, Yu. "THE ROLE OF THE ACE2/ANG-(1-7)/MASR AXIS IN THE DEVELOPMENT OF OBESITY-HYPERTENSION IN MALE AND FEMALE MICE." UKnowledge, 2016. http://uknowledge.uky.edu/pharmacol_etds/13.

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Obesity is strongly associated with hypertension and cardiovascular diseases. An activated renin-angiotensin system (RAS) has long been suggested as a critical contributor to elevated blood pressure with obesity. Angiotensin II (AngII), the main effector of an activated RAS, can be catabolized by angiotensin-converting enzyme 2 (ACE2) to form angiotensin-(1-7) (Ang-(1-7)), which, acting through the mas receptor (MasR), has been shown to oppose the effects of an activated RAS. Therefore, further understanding of the mechanisms of this counter-regulatory arm, called the ACE2/Ang-(1-7)/MasR axis,
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Raji, Ismaila. "Effect of Tulbaghia violacea on the blood pressure and heart rate in male spontaneously hypertensive wistar rats." Thesis, University of the Western Cape, 2011. http://etd.uwc.ac.za/index.php?module=etd&action=viewtitle&id=gen8Srv25Nme4_5212_1367481132.

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<p>Tulbaghia violacea Harv. (Alliaceae) is a small bulbous herb which belongs to the family, Alliaceae, most commonly associated with onions and garlic. In South Africa (SA), this&nbsp<br>herb has been traditionally used in the treatment of various ailments, including fever, colds, asthma, paralysis, hypertension (HTN) and stomach problems. The aim of this study&nbsp<br>was to evaluate the effect of methanol leaf extracts (MLE) of T. violacea on the blood pressure (BP) and heart rate (HR) in anaesthetized male spontaneously hypertensive rats<br>&nbsp<br>and to find out the mechanism(s) by whic
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Glover, Zoe. "Acer, a homologue of the human angiotensin-1 converting enzyme, modulates the response of sleep, glycogen storage, lifespan, fecundity and stress resistance to diet in Drosophila melanogaster." Thesis, Lancaster University, 2017. http://eprints.lancs.ac.uk/86909/.

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Human angiotensin-1–converting-enzyme (ACE) plays a primary role in the regulation of blood pressure and electrolytes as part of the Renin-Angiotensin System (RAS) in humans. Renin cleaves angiotensinogen to angiotensin I and ACE regulates the vasoconstriction of blood vessels by converting angiotensin-1 to angiotensin-2 (a potent vasoconstrictor) and also by breaking down bradykinin (a potent vasodilator). Renin is regulated by a feedback loop mechanism and is inhibited by higher concentrations of angiotensin-2. A lack of or inhibition of ACE can lead to a reduction in blood pressure (BP) and
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Cobas, Roberta Arnoldi. "Polimorfismo I/D do gene da enzima conversora de angiotensina e C242T do gene do componente p22phox da NADPH oxidase em pacientes com diabetes tipo 1." Universidade do Estado do Rio de Janeiro, 2009. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=1694.

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O sistema renina-angiotensina e o estresse oxidativo têm participação importante na fisiopatologia das complicações crônicas do diabetes. No presente estudo, foram avaliados 103 pacientes com diabetes tipo 1 (DM1) com idade de 28,810,6 anos e duração de doença de 13,18,5 anos e 158 controles não diabéticos quanto à presença dos polimorfismos I/D da ECA e C242T do p22phox, componente essencial para a ativação da NADPH oxidase. Esta análise foi realizada por reação de polimerase em cadeia para ambos os polimorfismos, seguida de restrição enzimática para avaliação do polimorfismo C242T p22phox.
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Al, Bacha Jeanne D'Arc. "Nouvelles fonctions de l'enzyme de conversion de l'angiotensine et du récepteur de la (pro)rénine en pathologies cardiovasculaires et neurologiques." Thesis, Paris 6, 2015. http://www.theses.fr/2015PA066415.

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De nouveaux composants du système rénine angiotensine (SRA) continuent d'être découverts et leurs fonctions étudiées. Cette thèse présente de nouvelles relations entre enzyme de conversion de l’angiotensine (ECA), thrombose et maladies cardiovasculaires (MCV), d’une part, et d’autre part, un rôle pour le récepteur de la (pro)rénine dans le développement du système nerveux central (SNC). Tout d’abord, nous avons étudié l'association de la double mutation de l’ECA et l’inhibiteur de l’activateur du plasminogène-1 (PAI-1) sur l’incidence d’évènements cardiovasculaires chez les patients Libanais h
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Wiklund, Per-Gunnar. "Genetic aspects of stroke : association and linkage studies in a northern Swedish population." Doctoral thesis, Umeå : Public Health and Clinical Medicine, 2005. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-668.

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Books on the topic "Angiotensin-1 converting enzyme"

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Dilsizian, Vasken, Ines Valenta, and Thomas H. Schindler. Myocardial Viability Assessment. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199392094.003.0021.

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Heart failure may be a consequence of ischemic or non-ischemic cardiomyopathy. Etiologies for LV systolic dysfunction in ischemic cardiomyopathy include; 1) transmural scar, 2) nontransmural scar, 3) repetitive myocardial stunning, 4) hibernating myocardium, and 5) remodeled myocardium. The LV remodeling process, which is activated by the renin-angiotensin system (RAS), stimulates toxic catecholamine actions and matrix metalloproteinases, resulting in maladaptive cellular and molecular alterations5, with a final pathway to interstitial fibrosis. These responses to LV dysfunction and interstiti
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Dhaun, Neeraj, and David J. Webb. Endothelins and their antagonists in chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0114_update_001.

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The endothelins (ETs) are a family of related peptides of which ET-1 is the most powerful endogenous vasoconstrictor and the predominant isoform in the cardiovascular and renal systems. The ET system has been widely implicated in both cardiovascular disease and chronic kidney disease (CKD). ET-1 contributes to the pathogenesis and maintenance of hypertension and arterial stiffness, as well endothelial dysfunction and atherosclerosis. By reversal of these effects, ET antagonists, particularly those that block ETA receptors, may reduce cardiovascular risk. In CKD patients, antagonism of the ET s
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(Editor), J. Rosenthal, and K. O. Stumpe (Editor), eds. Angiotensin-Converting-Enzym-Hemmer Bei Hypertonie, Teil 1 - Angiotensin Converting Inhibitors in Treatment of Hypertension: Schwerpunkt Auf CI (Angiotensin-Converting-Enzym-Hemmer ... Bei Hypertonie, Teil 1 -). Not Avail, 1993.

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Lynch, Bernadette, and Aine Burns. The patient with scleroderma. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0165.

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Scleroderma is tightness, thickening, and non-pitting induration of skin. Two forms of the skin disease are described. Limited cutaneous systemic sclerosis (lcSSc) which occurs distal to the wrists (or ankles) and/or over the face and neck, often associated with longstanding Raynaud’s phenomenon, and diffuse cutaneous systemic sclerosis (dcSSc) where truncal as well as acral skin involvement occurs as well as tendon friction rubs. In this latter condition the onset of the skin changes occurs within 1 year of onset of Raynaud’s phenomenon; however, the skin involvement may precede onset of vasc
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Lai, Kar Neng, and Sydney C. W. Tang. Immunoglobulin A nephropathy. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0068_update_001.

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Immunoglobulin A nephropathy is characteristically slowly evolving, and studies from autopsies and kidney donors show that deposition of immunoglobulin A is quite common and not necessarily associated with overt disease. However, series of biopsy-diagnosed patients that extend to 20 or 30 years report rates of end-stage renal failure of up to 40–50%. A very approximate overall rate of end-stage renal disease of 1% per year has been suggested. Proteinuria, glomerular filtration rate (GFR), and possibly some features on renal biopsies enable risk stratification, but all patients need long-term m
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Ferro, Charles J., and Khai Ping Ng. Recommendations for management of high renal risk chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0099.

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Poorer renal function is associated with increasing morbidity and mortality. In the wider population this is mainly as a consequence of cardiovascular disease. Renal patients are more likely to progress to end-stage renal disease, but also have high cardiovascular risk. Aiming to reduce both progression of renal impairment and cardiovascular disease are not contradictory. Focusing on the management of high-risk patients with proteinuria and reduced glomerular filtration rates, it is recommended that blood pressure should be kept below 140/90, or 130/80 if proteinuria is &gt; 1 g/24 h (protein:
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Gnudi, Luigi, Giorgio Gentile, and Piero Ruggenenti. The patient with diabetes mellitus. Edited by Giuseppe Remuzzi. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0149_update_001.

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About one third of patients with type 1 diabetes develop diabetic nephropathy long-term (usually not before at least 10 years of diabetes), though this proportion is falling as standards of care have risen. Nephropathy is strongly associated with other microvascular complications of diabetes, so that some degree of retinopathy is to be expected, and evidence of neuropathy is common. Patients with type 2 diabetes are equally susceptible, but this is an older group in which vascular disease and other pathologies are also more likely. The rise in type 2 diabetes accounts for diabetes being the mo
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Kriesel, Kristin. Tubuläre Atrophie unter chronischer Blockade des Angiotensin-Converting-Enzyms bei Ratten mit 1-Nieren, 1-Clip-Goldblatt-Hypertonie. 1993.

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Book chapters on the topic "Angiotensin-1 converting enzyme"

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Daliri, Eric Banan-Mwine, and Byong H. Lee. "Angiotensin 1-Converting Enzyme Inhibitory Postbiotics from Fermented Soybean." In Methods and Protocols in Food Science. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3421-9_36.

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Trask, Aaron J., Jasmina Varagic, Sarfaraz Ahmad, and Carlos M. Ferrario. "Angiotensin-(1-7), Angiotensin-Converting Enzyme 2, and New Components of the Renin Angiotensin System." In Renin Angiotensin System and Cardiovascular Disease. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60761-186-8_10.

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Ferrario, Carlos M., David B. Averill, K. Bridget Brosnihan, et al. "Regulation of Cardiovascular Control Mechanisms by Angiotensin-(1–7) and Angiotensin-Converting Enzyme 2." In Hypertension and Hormone Mechanisms. Humana Press, 2007. http://dx.doi.org/10.1007/978-1-59259-987-5_3.

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Ferrario, Carlos M., Jewell A. Jessup, and Jasmina Varagic. "Newer Insights into the Biochemical Physiology of the Renin–Angiotensin System: Role of Angiotensin-(1-7), Angiotensin Converting Enzyme 2, and Angiotensin-(1-12)." In The Local Cardiac Renin-Angiotensin Aldosterone System. Springer US, 2009. http://dx.doi.org/10.1007/978-1-4419-0528-4_2.

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Benter, Ibrahim F., Mariam H. M. Yousif, Jasbir S. Juggi, and Saghir Akhtar. "The Angiotensin-Converting Enzyme 2/Angiotensin-(1-7)/Mas Receptor Axis: A Potential Target for Treating Diabetic Cardiovascular Disease." In Diabetic Cardiomyopathy. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-9317-4_22.

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Celik, Gulperi. "Diabetic Nephropathy and Current Approach to Therapy." In Current Perspective on Diabetes Mellitus in Clinical Sciences. Nobel Tip Kitabevleri, 2023. http://dx.doi.org/10.69860/nobel.9786053359111.13.

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Diabetic nephropathy is a common and serious complication of diabetes mellitus (DM), characterized by progressive kidney damage that can lead to end-stage renal disease (ESRD). It primarily affects individuals with both type 1 and type 2 diabetes, and its pathogenesis involves complex interplay of metabolic, hemodynamic, and inflammatory factors. Key mechanisms include hyperglycemia-induced oxidative stress, activation of the renin-angiotensin-aldosterone system (RAAS), and inflammatory pathways. Current therapeutic approaches aim to delay or prevent the progression of diabetic nephropathy. Ti
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Akchunov, A. A., Z. Golubenko, and N. Sosnina. "Isolation and Characterization of Biological Properties of Inhibitors Angiotensin-1-Converting Enzyme from the Spider Venom Latrodectus Tredecimguttatus." In Recent Progress on Kinins. Birkhäuser Basel, 1992. http://dx.doi.org/10.1007/978-3-0348-7321-5_59.

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Özkaya, Esen, and Kurtuluş Didem Yazganoğlu. "Angiotensin-Converting Enzyme Inhibitors." In Adverse Cutaneous Drug Reactions to Cardiovascular Drugs. Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-6536-1_2.

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Oparil, Suzanne, Qingcheng Meng, Shuang-dan Sun, Yiu-Fai Chen, and Louis J. Dell’Italia. "Tissue Angiotensin Converting Enzyme." In Vascular Endothelium. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-0355-8_15.

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Mountokalakis, Theodore D. "Angiotensin Converting Enzyme Inhibitors." In Contemporary Concepts in Cardiology. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-5007-5_12.

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Conference papers on the topic "Angiotensin-1 converting enzyme"

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Westwood, Brian M., Hossam A. Shaltout, and Mark C. Chappell. "Modeling of Angiotensin Peptide Metabolism in Renal Proximal Tubules." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-190990.

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The recent discovery of angiotensin converting enzyme 2 (ACE2) as a functional peptidase within the renin-angiotensin system (RAS) has added a new layer of complexity to the enzymatic cascade of this hormonal system. ACE2 is highly expressed in the proximal tubules of the kidney, an important tissue site involved in blood pressure regulation. Therefore, we derived a model for the processing of Ang I which is the immediate precursor to the biologically active peptides Ang II and Ang-(1-7) based on metabolism data in isolated proximal tubules of the sheep kidney (1). Given the individual experim
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Furlam, Tiago de Oliveira, Ewelin Wasner Machado da Silva, Isadora Gonçalves Roque, et al. "Renin-Angiotensin System 24 hours after mild traumatic brain injury: a case- control study." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.651.

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Background: The Renin-Angiotensin System (RAS) has been associated with several neuropathologies, including traumatic brain injury (TBI). Objectives: Assess the relationship between RAS and mild TBI within 24 hours after trauma. Design and setting: A case-control study developed by the Federal University of Minas Gerais and conducted at the Hospital João XXIII, Belo Horizonte, Minas Gerais, Brazil. Methods: Sociodemographic data and blood samples were collected from 52 individuals, of whom 28 suffered mild TBI in the 24 hours prior to collection and 24 healthy individuals made up the control g
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Bruyakin, S. D., and D. A. Makarevich. "MODELING OF POTENTIAL PROTEIN S1 SARS-COV-2 LIGANDS." In SAKHAROV READINGS 2021: ENVIRONMENTAL PROBLEMS OF THE XXI CENTURY. International Sakharov Environmental Institute of Belarusian State University, 2021. http://dx.doi.org/10.46646/sakh-2021-2-27-30.

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The S1 protein of SARS-CoV-2 (hereinafter referred to as the S1 protein) is probably the main factor in the pathogenesis of COVID-19. In our opinion, the elimination or decrease in the concentration of this protein will reduce the inflammatory process and, accordingly, damage to organs and tissues by the activated immune system. An analysis of the complexes of the Angiotensin-converting enzyme 2 (ACE2) and the S1 protein (ACE2-S1) will determine the oligopeptides that are ligands for binding the S1 protein, the timely removal of which from the blood of patients with COVID-19 will prevent the d
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Gause, S., M. L. Ribeiro Neto, and D. A. Culver. "Assessing the Clinical Utility of Serum Angiotensin-Converting Enzyme, Soluble IL-2 Receptor, and 1-25 Vitamin D in the Diagnosis of Extra-Pulmonary Sarcoidosis." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4309.

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Krentz, Andrew J. "Cardio-Renal-Metabolic connection – Do we have possibilities for solving this problem?" In 7th International Congress of Cardionephrology KARNEF 2025. Punta Niš, 2025. https://doi.org/10.46793/karnef25.190k.

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Within the continuum of cardiometabolic diseases, a cardiovascular-kidney-metabolic (CKM) syndrome has been conceptualised. The CKM syndrome is a systemic disorder characterised by pathophysiological interactions between metabolic risk factors, chronic kidney disease (CKD), and the cardiovascular system. CKM syndrome is characterised by multiorgan dysfunction and confers a high risk of adverse cardiovascular outcomes. Relevant haemodynamic and neurohormonal pathologies of the include the metabolic syndrome (central adiposity, insulin resistance, hypertension, hyperglycaemia, atherogenic dyslip
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Jacobs, M., L. Lahousse, H. P. Van Eeckhoutte, et al. "Effect of ACE1 (Angiotensin Converting Enzyme 1) Polymorphism Rs1799752 on Protein Levels of ACE2, the SARS-CoV-2 (Severe Acute Respiratory Syndrome Coronavirus 2) Entry Receptor, in Alveolar Lung Epithelium." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a1272.

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Baudin, B., L. Drouet, J. L. Carrier, M. Bérard, and Q. Y. Wu. "DISTRIBUTION OF ENDOTHELIAL MARKERS ALONG THE VASCULAR TREE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643357.

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Few specific markers of the endothelial cells are available. Von Willebrand factor has been recognized as the most specific but we have shown that its intracellular content and extracellular release varies largely along the vascular tree of the pig. Synthesis is maximal in capillaries and in pulmonary artery endothelial cells. Synthesis is almost nil in the aorta. Intermediary values are encountered in the inferior vena cava. We studied the distribution of angiotensin converting enzyme (ACE) in endothelial cells along the vascular tree, as synthesis, storage, membrane expression and release of
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Shokina, V. A., S. A. Doronin, and A. V. Kudryavtsev. "DEVELOPMENT OF NEUTRALIZING RECOMBINANT NANOBODIES FOR TREATMENT AND PREVENTION OF COVID-19." In X Международная конференция молодых ученых: биоинформатиков, биотехнологов, биофизиков, вирусологов и молекулярных биологов — 2023. Novosibirsk State University, 2023. http://dx.doi.org/10.25205/978-5-4437-1526-1-396.

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The rapid variability of coronavirus forces the use of alternative strategies to protect humans from COVID-19. Therefore, we have developed a nanobody that blocks the binding of RBD domain of the S-protein of SARS-CoV-2 virus to the angiotensin-converting enzyme type 2 (ACE2).
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Saldeen, K., R. Moalli, and T. Saldeen. "EFFECT OF A FIBRIN-DERIVED PEPTIDE ON PULMONARY ANGIOTENSIN CONVERTING ENZYME (ACE) ACTIVITY AND ON PRESSURE RESPONSES TO BRADYKININ (BK)." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644331.

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Decreased ACE activity is a common finding in patients with adult respiratory distress syndrome (ARDS) and in animal models of lung injury. The nature of this effect is unknown. Fibrin, also a common finding in lung injury, is degraded to small peptides by proteolytic enzymes. Peptide 6A, corresponding to amino acid residues 43-47 of the BB-chain of fibrin (cgen), is produced by plasmin degradation of fibrin and has been shown to inhibit ACE in vitro. The purpose of the present investigation was to study whether peptide 6A inhibits pulmonary ACE in vivo and, if so, determine its effect of hemo
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Reports on the topic "Angiotensin-1 converting enzyme"

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Wu, Xiaoqi, Jisen Zhao, Maoxia Fan, and Dongo Guo. Quality of Evidence Supporting the Effects of Xinmailong injection in Heart Failure: An Overview of Systematic Reviews and Meta-Analyses. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.10.0023.

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Review question / Objective: 2.1.1 type of research SRs/MAs of RCT (randomized controlled trial) of Xinmailong injection for the treatment of heart failure. 2.1.2 Subject investigated All included patients met internationally recognized diagnostic criteria for heart failure.There are no limitations on age, gender, ethnicity, time of onset, source of cases and language of publication. 2.1.3 Type of Intervention The control group was treated with conventional basic Western medicine recommended by the guidelines related to heart failure[1, 11], including antiplatelet drugs, anticoagulants, vasodi
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กาญจนทัต, อภิชาติ. โปรตีนไฮโดรไลเสตจากเมล็ดผลไม้ไทยเพื่อการบำบัดโรค : รายงานวิจัยฉบับสมบูรณ์ (ปีที่ 1). จุฬาลงกรณ์มหาวิทยาลัย, 2014. https://doi.org/10.58837/chula.res.2014.89.

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Blood pressure regulation is partially dependent on the renin-angiotensin system; renin acts on angiotensinogen to release angiotensin-I, which is further converted into the angiotensin II by the angiotensin I-converting enzyme (ACE). ACE plays a key physiological role in the regulation of blood pressure by virtue of two different reactions that it catalyzes: conversion of the inactive angiotensin I to the powerful vasoconstrictor angiotensin II, and inactivation of the vasodilator bradykinin. Crude extract and ammonium sulphate cut protein extracts, and their pepsin-pancreatin hydrolysates, f
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