Log in

Relevant bibliographies by topics / Angiotensins. Hormones / Books

To see the other types of publications on this topic, follow the link: Angiotensins. Hormones.

Books on the topic 'Angiotensins. Hormones'

Author: Grafiati

Published: 4 June 2021

Last updated: 6 February 2022

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 16 books for your research on the topic 'Angiotensins. Hormones.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse books on a wide variety of disciplines and organise your bibliography correctly.

1

International Symposium on Cellular and Molecular Biology of the Adrenal Cortex (5th 1992 Avignon, France). Cellular and molelcular biology of the adrenal cortex: Proceedings of the 5th International Symposium on Cellular and Molecular Biology of the Adrenal Cortex held in Avignon (France) August 27-29, 1992. Paris, France: INSERM, 1992.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

2

Kidney Hormones: Erythropoietin (Kidney Hormones). Academic Press, 1997.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

3

Fisher, J. W. Kidney Hormones. Academic Press, 1986.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

4

(Editor), J. M. Saez, and et al (Editor), eds. Cellular and Molecular Biology of the Adrenal Cortex (Colloques Inserm). John Libbey & Co Ltd, 1992.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

5

Cellular and Molecular Biology of the Adrenal Cortex (Colloques INSERM). J. Libbey Eurotext, 1996.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

6

K, Raizada Mohan, Phillips M. Ian, Sumners Colin, and International Symposium on the Cellular and Molecular Aspects of Angiotensin Receptors (1st : 1994 : Gainsville, Fla.), eds. Recent advances in cellular and molecular aspects of angiotensin receptors. New York: Plenum Press, 1996.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

7

R, Ruffolo Robert, ed. Angiotensin II receptors. Boca Raton: CRC Press, 1994.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

8

Ghazi, Nooshin. Variations in AII release from the rat brain during the estrous cycle. 1994.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

9

Grove, Kevin Lyle. Angiotensin II receptors in the hypothalamus of the adult and developing female rat brain. 1994.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

10

Vitamin Biosynthesis (Vitamins and Hormones, Volume 60) (Vitamins and Hormones). Academic Press, 2000.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

11

Sardinia, Michael Francis. AT₄ receptor binding characteristics: A study of the structural requirements of the angiotensin IV peptide requisite for high affinity, specific ligand-receptor interaction. 1994.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

12

Butler, Gary, and Jeremy Kirk. Adrenal gland disorders. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199232222.003.0068.

Full text

Abstract:

Physiology 236Adrenal insufficiency 238Adrenal steroid excess 246Further reading 247The adrenal cortex, which produces steroid hormones, is under the control of both the hypothalamo–pituitary–adrenal (HPA) endocrine axis, which regulates cortisol secretion, and the renin–angiotensin system, which regulates aldosterone secretion (Figs 8.1 and ...

APA, Harvard, Vancouver, ISO, and other styles

13

Endlich, Karlhans, and Rodger Loutzenhiser. Regulation of vasomotor tone in the afferent and efferent arterioles. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0208.

Full text

Abstract:

Pre-glomerular vessels are regulated by membrane potential alterations affecting the activity of L-type voltage-activated Ca2+ channels; whereas voltage-independent mechanisms regulate the efferent arteriole, notably influenced by angiotensin II and therefore by angiotensin converting enzyme inhibitors, and by non-steroidal anti-inflammatory drugs.These properties underlie the physiologic control of glomerular capillary pressure, for example, by prostaglandin E2, during conditions of reduced renal perfusion and the stabilization of glomerular capillary pressure when the kidney is exposed to pressure fluctuations. A wealth of hormones affects the tone of renal vessels. The chapter focuses on basic regulatory and signalling mechanisms, emphasizing the unique aspects of the renal vasculature and the underlying features that facilitate the independent regulation of pre-glomerular and post-glomerular tone.

APA, Harvard, Vancouver, ISO, and other styles

14

Robberecht, W. Control of Growth Hormone and Prolactin Release by Angiotensin Peptides in Reaggregate Cell Cultures of the Rat Pituitary. Leuven University Press, 1990.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

15

Rahimi, Kazem. Chronic heart failure. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0092.

Full text

Abstract:

The European Society of Cardiology defines heart failure as a clinical syndrome in which patients have the following features: symptoms typical of heart failure (breathlessness, fatigue, ankle swelling); signs typical of heart failure (tachycardia, tachypnoea, pulmonary crackles, pleural effusion, raised jugular venous pressure, peripheral oedema, hepatomegaly); and objective evidence of a structural or functional abnormality of the heart at rest (cardiomegaly, third heat sound, cardiac murmurs, abnormality on the echocardiogram, raised natriuretic peptide concentration). Heart failure results in activation of the sympathetic nervous system and the renin–aldosterone–angiotensin system, and release of a number of hormones such as natriuretic peptides, and cytokines, including tumour necrosis factor amongst others. While neurohormone activation is initially compensatory and helps in the short term to maintain circulatory needs, ultimately it has detrimental effects on the myocardium and compromises its function further. These mechanisms are therefore therapeutic targets to improve symptoms and lessen the risk of death.

APA, Harvard, Vancouver, ISO, and other styles

16

Arroyo, Vicente, Mónica Guevara, and Javier Fernández. Renal failure in cirrhosis. Edited by Norbert Lameire. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0247.

Full text

Abstract:

A major event in liver cirrhosis is the development of a progressive deterioration of circulatory function due to splanchnic arterial vasodilation and impairment in cardiac function. This feature determines a homeostatic activation of the renin–angiotensin–aldosterone system, sympathetic nervous system, and antidiuretic hormone. The splanchnic microcirculation is resistant to the vasoconstrictor effect of these systems. Therefore, the homeostasis of arterial pressure in cirrhosis occurs in the extrasplanchnic, mainly renal circulation. The activation of these systems produces renal fluid retention, which accumulates as ascites, and water retention and dilutional hyponatraemia. In the latest phase of cirrhosis, when circulatory dysfunction is severe, renal vasoconstriction is intense and patients develop type 2 hepatorenal syndrome (HRS) and refractory ascites.Type 1 HRS is an acute and rapidly progressive renal failure that occurs in the setting of a precipitating event, commonly an infection. Patients with type 1 HRS also present with rapid deterioration of liver function (encephalopathy, jaundice) and relative adrenal insufficiency. The mechanism of this multiorgan failure is an acute deterioration in circulatory function due to both an accentuation of arterial vasodilation and of cardiac dysfunction.There is no specific test for the diagnosis of HRS. The most accepted diagnostic criteria are those proposed by the International Ascites Club which are based on the exclusion of other types of renal failure. The course of renal failure following treatment of the precipitating event of HRS is another important diagnostic feature.The treatment of choice of tense ascites in cirrhosis is paracentesis associated with intravenous albumin infusion. Moderate sodium restriction and diuretics (spironolactone alone or associated with furosemide) are subsequently given to prevent re-accumulation of ascites. Diuretics are the treatment of choice in patients with moderate ascites. Patients with type 2 HRS and refractory ascites (not responding to diuretics) could be treated by frequent paracentesis or by the insertion of a transjugular intrahepatic portosystemic shunt (TIPS).Terlipressin plus albumin is the treatment of choice in type 1 HRS

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!