Relevant bibliographies by topics / Animals ascarriers of disease

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Animals ascarriers of disease'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Animals ascarriers of disease"

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Keymer, I. F. "Disease in wild animals." Veterinary Record 161, no. 7 (August 18, 2007): 244. http://dx.doi.org/10.1136/vr.161.7.244-b.

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Liu, Si-kwang. "Metabolic Disease in Animals." Seminars in Musculoskeletal Radiology 06, no. 4 (2002): 341–46. http://dx.doi.org/10.1055/s-2002-36733.

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Shmakova, Anna A., and Lyubov V. Androsova. "Modeling Alzheimer’s disease in animals." Psychiatry 77 (2018): 97–108. http://dx.doi.org/10.30629/2618-6667-2018-77-97-108.

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Müller, M., and G. Brem. "Disease resistance in farm animals." Experientia 47, no. 9 (September 1991): 923–34. http://dx.doi.org/10.1007/bf01929883.

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Darke, P. G. G. "Myocardial disease in small animals." British Veterinary Journal 141, no. 4 (July 1985): 342–48. http://dx.doi.org/10.1016/0007-1935(85)90082-x.

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Forrester, Donald J. "Essentials of Disease in Wild Animals." Journal of Wildlife Diseases 43, no. 1 (January 2007): 145–49. http://dx.doi.org/10.7589/0090-3558-43.1.145.

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Andrew, Stacy E. "Corneal fungal disease in small animals." Clinical Techniques in Small Animal Practice 18, no. 3 (August 2003): 186–92. http://dx.doi.org/10.1016/s1096-2867(03)90015-6.

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Hardy, Anne, Dr. "Animals, Disease, and Man: Making Connections." Perspectives in Biology and Medicine 46, no. 2 (2003): 200–215. http://dx.doi.org/10.1353/pbm.2003.0021.

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Hannigan, Chloe. "Diagnosing blood disease in domestic animals." Veterinary Record 170, no. 16 (April 20, 2012): 418.1–418. http://dx.doi.org/10.1136/vr.e2815.

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Bader, Michael, Holger Bohnemeier, Frank S. Zollmann, Ora E. Lockley-Jones, and Detlev Ganten. "Transgenic Animals in Cardiovascular Disease Research." Experimental Physiology 85, no. 6 (November 2000): 713–31. http://dx.doi.org/10.1111/j.1469-445x.2000.02096.x.

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Dissertations / Theses on the topic "Animals ascarriers of disease"

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Nsubuga, Rebecca Namugabwe. "Statistical inference for infectious disease data of animals." Thesis, University of Edinburgh, 2004. http://hdl.handle.net/1842/30591.

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This thesis primarily explores parameter methods as applied to data generated from an experimental infection with foot and mouth disease (FMD) virus in sheep. Data were generated from two experiments involving four groups of sheep, housed under restricted mixing, where sheep in the initial group were inoculated with type O FMD virus. The aim of the analysis is to investigate the presence of any trend in the infection rate with increased generation. The infection process of FMD virus in sheep can be modelled using chain binomial models and generalized linear models. However, application of these methods requires that the epidemic chain of infection pathways be known. The set of true pathways is an unobservable quantity and, in general, infectious disease data will be incomplete because the infection process is only partially observed. One proposed strategy is subjectively to assign an epidemic chain to the data and to analyse it on this basis. This approach is evaluated. An alternative to modelling the FMD infection process for individual sheep is to consider the transmission among groups of sheep, thus avoiding the need to make inference about individual infection pathways. Martingale methods and maximum likelihood estimation methods are used to estimate the typical infection rate β applying to groups of sheen where the aim is to investigate whether the infection rate changes across groups. The expected total infection exposure for each group is estimated. This entails knowledge of the time of infection, the latent period and the infectious period for each infected sheep. Parameters for the latent period and infectious period distributions are estimated from the data. A joint distribution of time to infection and latent period is formulated from which expected values for time to infection and the latent period for each infected sheep are estimated.
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Millar, Douglas Spencer. "Mycobacterium paratuberculosis, mycobacteria and chronic enteritis in humans and animals." Thesis, St George's, University of London, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.308932.

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Jewell, Chris. "Real-time inference and risk-prediction for notifable disease of animals." Thesis, Lancaster University, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.536005.

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Hingley, P. J. "Problems in modelling responses of animals to foot and mouth disease vaccine." Thesis, University of Reading, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.356751.

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Persson, Anna, and Jessica Löfgren. "Reactions of persons with Alzheimer’s disease on the presence of animals in nursing." Thesis, Kristianstad University College, Department of Health Sciences, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:hkr:diva-3547.

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The results showed that in the presence of animals both verbal and non verbal communication, problem behaviours and different physical parameters were improved for persons with Alzheimer’s disease. The authors think that animals can provide for some needs which easily remain unmet in the daily life; e.g. to care for and take care of another living being. Possibilities are seen for an introduction of animals into nursing as they can have a positive influence on persons with Alzheimer’s disease. The number of persons with Alzheimer’s disease may increase in the future. Today, there is no known medical cure for the disease. The environment has proved to be of importance in their care. Animals can contribute to a good environment. The purpose was to describe reactions of persons with Alzheimer’s disease in the presence of animals in nursing. A general literature study was made from ten articles found at different databases on the web. Additions were made through manual searching in the articles´ list of references. The results of the articles were processed through a division into codes, subcategories and main categories.

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Williams, Alison Clare. "Construction of infectious disease resistant animals by manipulation of the acute phase response." Thesis, University of Leicester, 2000. http://hdl.handle.net/2381/30333.

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Crockford, Melanie. "Partial characterisation of pilchard herpesvirus and the associated disease in pilchards." Crockford, Melanie (2007) Partial characterisation of pilchard herpesvirus and the associated disease in pilchards. PhD thesis, Murdoch University, 2007. http://researchrepository.murdoch.edu.au/445/.

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In 1995 and again in 1998, millions of pilchards (Sardinops sagax neopilchardus) were found dead or dying off the coast of Australia and also in New Zealand. The epizootics moved progressively, at a rapid speed against the prevailing currents. A previously unrecognised herpesvirus, Pilchard herpesvirus (PHV), was identified as the causative agent. Until recently, rapid and sensitive methods to detect PHV were not available and based on a previously identified and conserved 373 bp region of the genome, polymerase chain reaction (PCR), in situ hybridisation and real-time PCR methods were developed for the specific detection of PHV in formaldehyde-fixed and frozen tissues of pilchards. Real-time PCR was shown to have greater sensitivity than a conventional PCR and in situ hybridisation for the detection of PHV infection. The PCR assay and sequence analysis of the amplification products was used to compare the 373 bp region of the genome from strains obtained during the 1995 and 1998 epidemics. Significant differences between the strains were not detected.Additional sequence data was obtained adjacent to the 373 bp of known PHV sequence, which did not match any sequence in any of the genetic databases, and this will be invaluable for further study of the pilchard herpesvirus and the development of improved detection methods. The molecular-based methods of virus detection developed were applied to a re- examination of virus in paraffin-embedded tissues taken from fish during an attempt to transmit the virus to wild caught pilchards in 1999. The results obtained confirmed previously equivocal results that transmission of PHV to wild caught pilchards was achieved, although this experiment failed to demonstrate thattransmission of the virus resulted in severe lesions typical of those seen in the epizootics. Using formaldehyde-fixed samples from fish collected during the 1998 PHV epizootic, virus was detected in fish collected 4 days prior to the occurrence of the epizootic even though the fish then appeared clinically normal, during the epizootic, and 8 days after mortalities had ceased. An investigation of wild pilchards collected from 4 Australian pilchard sub-populations using real-time PCR demonstrated that PHV was present in the gills of 13.75% of 800 fish sampled, indicating that the virus is now endemic in the Australian pilchard population. Variation in the prevalence of PHV infection in the 4 subpopulations was detected, higher in western and southern populations than in populations from the east coast. The endemic nature of PHV infection in the pilchard population explains why there have been no further epizootics with mass mortalities since 1998.
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Knight-Jones, Theo. "Field evaluation of foot-and-mouth disease vaccination in Turkey." Thesis, Royal Veterinary College (University of London), 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618321.

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Loh, Richmond Cern-Wan. "The pathology of devil facial tumour disease in Tasmanian devils (Sarcophilus harrisii) /." Access via Murdoch University Digital Theses Project, 2006. http://wwwlib.murdoch.edu.au/adt/browse/view/adt-MU20061019.131524.

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Arab, Fahad Ahmad H. "Qualitative and quantitative studies on infection of E. Hungarensis (Levine & Ivens, 1965) in the wood mouse Apodemus Sylvaticus and labaratory mouse Mus musculus." Thesis, University of Reading, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.332042.

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Books on the topic "Animals ascarriers of disease"

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Wobeser, Gary A. Disease in Wild Animals. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-48978-8.

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Ware, Wendy A., John D. Bonagura, and Brian A. Scansen. Cardiovascular Disease in Companion Animals. 2nd ed. Boca Raton: CRC Press, 2021. http://dx.doi.org/10.1201/9780429186639.

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Kruiningen, H. J. Van. Daniels' health & disease management of animals. 2nd ed. Storrs, CT: University of Connecticut, 1999.

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Kruiningen, H. J. Van. Daniels' health & disease management of animals. 2nd ed. Storrs, CT: University of Connecticut, 1999.

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Gram, W. Dunbar, Rowan J. Milner, and Remo Lobetti, eds. Chronic Disease Management for Small Animals. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781119201076.

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Wobeser, Gary A. Essentials of disease in wild animals. Ames, Iowa: Blackwell Pub., 2006.

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Van Der Zijpp, A. J., and W. Sybesma. Improving Genetic Disease Resistance in Farm Animals. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-1057-7.

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Bishop, S. C., R. F. E. Axford, F. W. Nicholas, and J. B. Owen, eds. Breeding for disease resistance in farm animals. Wallingford: CABI, 2010. http://dx.doi.org/10.1079/9781845935559.0000.

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Investigation and management of disease in wild animals. New York: Plenum Press, 1994.

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Swanson, Janice C. Toxoplasmosis: A protozoan disease of animals and man. Beltsville, Md: National Agricultural Library, 1990.

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Book chapters on the topic "Animals ascarriers of disease"

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Lassnig, Caroline, and Mathias Müller. "Disease-Resistant Transgenic Animals disease-resistant transgenic animals." In Encyclopedia of Sustainability Science and Technology, 2963–76. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0851-3_10.

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Lassnig, Caroline, and Mathias Müller. "Disease-Resistant Transgenic Animals disease-resistant transgenic animals." In Sustainable Food Production, 747–60. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-5797-8_10.

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Fresneda, Karina C., and Francisco R. Carvallo Chaigneau. "Tyzzer's Disease." In Clostridial Diseases of Animals, 281–91. Hoboken, NJ: John Wiley & Sons, Inc, 2016. http://dx.doi.org/10.1002/9781118728291.ch24.

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Ware, Wendy A., John D. Bonagura, and Brian A. Scansen. "Heartworm Disease." In Cardiovascular Disease in Companion Animals, 841–76. 2nd ed. Boca Raton: CRC Press, 2021. http://dx.doi.org/10.1201/9780429186639-45.

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Scansen, Brian A. "Thromboembolic Disease." In Cardiovascular Disease in Companion Animals, 745–74. 2nd ed. Boca Raton: CRC Press, 2021. http://dx.doi.org/10.1201/9780429186639-41.

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Lassnig, Caroline, and Mathias Müller. "Disease-Resistant Transgenic Animals." In Encyclopedia of Sustainability Science and Technology, 1–17. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4939-2493-6_10-3.

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Houston, Doreen M., Andrew Moore, Denise A. Elliott, and Vincent C. Biourge. "Stone Disease in Animals." In Urinary Tract Stone Disease, 131–50. London: Springer London, 2010. http://dx.doi.org/10.1007/978-1-84800-362-0_10.

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Sundberg, J. P. "Papillomavirus Infections in Animals." In Papillomaviruses and Human Disease, 40–103. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71097-1_3.

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Robertson, Lucy J., Camilla Björkman, Charlotte Axén, and Ronald Fayer. "Cryptosporidiosis in Farmed Animals." In Cryptosporidium: parasite and disease, 149–235. Vienna: Springer Vienna, 2013. http://dx.doi.org/10.1007/978-3-7091-1562-6_4.

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Vaden, Shelly L., and Gregory F. Grauer. "Glomerular Disease." In Nephrology and Urology of Small Animals, 538–46. West Sussex, UK: John Wiley & Sons, Ltd., 2014. http://dx.doi.org/10.1002/9781118785546.ch53.

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Conference papers on the topic "Animals ascarriers of disease"

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Fougere, B. "Medicinal Plants for Chronic Disease in Small Animals." In GA 2017 – Book of Abstracts. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1608014.

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Knazovicky, D., M. Freire, B. Case, D. Gearing, and B. Lascelles. "Pilot Evaluation of The Effect of Anti-nerve Growth Factor Antibody on Sensory System Function in Dogs with Degenerative Joint Disease-Associated Pain." In Pain in Animals Workshop 2017: Abstracts. Schattauer GmbH, 2018. http://dx.doi.org/10.1055/s-0038-1660881.

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Davies, R., HS Jin, K. Sime, JO Williams, CR Thompson, GW Jones, K. Allen-Redpath, et al. "THU0025 Il-6 trans-signaling causes accelerated atherosclerosis in disease prone animals." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.2871.

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Tanyildizl, Erkan, and Gokce Yildirim. "Performance Comparison of Classification Algorithms for The Diagnosis of Mastitis Disease in Dairy Animals." In 2019 7th International Symposium on Digital Forensics and Security (ISDFS). IEEE, 2019. http://dx.doi.org/10.1109/isdfs.2019.8757469.

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Stemeier, K., J. Mertin, J. Pill, and F. Hartig. "EFFECTS OF THROMBOXANE RECEPTOR BLOCKER BM 13.505 ON THE DEVELOPMENT OF PROTEINURIA IN AUTOIMMUNE NZB/W MICE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643757.

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Female F1 hybrid of New Zealand black and white mice(NZB/W) spontaneouslydevelop an autoimmune disease characterize by afatal immune complex glomerulonephritis.Theyare considered to be a relevant model of human systemic lupus erythematosus. We observeda doubling of the concentration of TXB2 in urine at the same time when onset of proteinuria was noticed. This suggests that TXA2 synthetized by mesangial and epithelial cells of the glomeruli as well as by some inflammatory cells and platelets might be an important mediator in the pathogenesis of thi auto immune-mediated glomerular disease. Weused BM 13.505 as an long-acting TX receptor blocker for evaluating the importance of TXA2 on the development of proteinuria and compared its effects with that of the immunsuppressive agent cyclophosphamide.NZB/W mice were distributed to vehicle-treated (V-)group 20 mg/kg BM 13.505 (BM-) group and 20 mg/kgcyclophosphamide (C-) group( = 13 -14).Daily dosing by gavage was startedat the age of12 weeks. Every fourth week theurinary concentrations of proteins were measured by th biuret method and TXB2by a RIA. An increasein TXB2 was seen in the V- and BM-group, while in the C-group TXB2 was lowered. At 36 weeks of age 8of 14 animals of the V-group were proteinuria positive (>100 mg/100 ml). The study was finished at 44 weeks because more than 2/3 of the animals of the V-group haddeveloped a proteinuria. Previously four animals died and in most of other the disease was faradvanced. In the BM-group no animal had diedor showed signs of illness. However seven ofthe animals had slightly elevated protein concentrations in urine and two moderate elevated values. In the C-group no proteinuria was detected. Histological examinations of thekidneys showed a correlation in individualanimals of the V-group between the duration and extent of proteinuria and changes in the morphology of the glomeruli. In the BM-treated animals slight to moderate protein deposits were detectable, while in cyclophosphamide-treated animals glomeruli were of normal structure. This study presents someevidence that TXA2 may be an important mediator in the pathogenesis of this immune-mediated renal disease. Manifestation of this disease is delayed by the administration of thespecific TX receptor blocker BM 13.505.
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Yazawa, Toru, Yukio Shimoda, Satoru Shimizu, and Tomoo Katsuyama. "Neurodynamical Control of the Heart of Freely Moving Animals Including Humans." In ASME 2012 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/imece2012-85872.

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Two kinds of nerves, acceleratory and inhibitory cardio-regulator nerves, innervate the heart. They are known to discharge concurrently to maintain an equilibrium state of the body. The nerves are also known to change their frequency of discharge in a reflexive manner to meet the demand from the periphery; such as augmentation of oxygen supply or vice versa. Consequently, the heart exhibits dynamic change in its pumping rate and force of contraction. If the control system fails, the heart exhibits unhealthy state. However, assessment of healthy/unhealthy status is uneasy because we are not able to monitor the nerve activities by non-invasive methods. Therefore, we challenged to detect state of the heart without nerve-recordings. We used the detrended fluctuation analysis (DFA) applying to heartbeat interval time series because DFA has been believed that it can quantify the state of heart. We performed DFA on the EKGs (electrocardiograms) from various living organisms including humans. The objective of this research was to determine whether the analytical technology, DFA, could function as a useful method for the evaluation of the subject’s quality of cardiovascular-related illness and transition to and from a normal healthy state. We found that DFA could describe brain-heart interaction quantitatively: the scaling exponents of (1) healthy, (2) sick-type (such as stressful or arrhythmic states), and (3) unpredictable-death type (such as ischemic heart disease) were corresponded to individuals who exhibited, (1) nearly one, (2) less than one, and (3) greater than one, respectively. We conclude that scaling exponents could determine whether the subjects are under sick or healthy conditions on the basis of cardiac physiology.
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Erzhen, Zen, Lu Yung-Cai, Wang Jain, Shi Fang, Lia Xiaoqing, Zhou Yulin, Jia Xudong, and Gou Zhaozheng. "EXPERIMENTAL STUDIES ON THE MECHANISM OF CHINESE MEDICINAL RHAPONTICUM UNIFLORUM DC IN PREVENTING CORONARY HEART DISEASE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643029.

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The mechanism of Rhaponticum Uniflorum DC in preventing coronary heart disease was studied in vivo and in vitro. TBA fluorescent method was used to determine lipid peroxides (Lpo) and double analysis method was used to determine glutathione peroxidase (GSH-Px) activity and fluorescent polarization of DPH probed membrane fluidity of smooth muscle cell (SMC).Rabbits were fed with high fat diet for 120 days. At the end of experiment, all the animals acquired hyperlipidemia and developed atheroma lesions in aorta and/or coronary. It was found that hyperlipidemia caused a rising of Lpo in blood (from 2.6±0.56 in control up to 8.48±3.28 nmol/ml) and in arterial wall (from 6.75±0.59 in control up to 31.94±4.20 nmol/g protein) and a decreasing of GSH-Px activity in arterial wall (from 0.210±0.095 down to 0.056±0.026 EU/g protein); concomitantly, an increase in microviscosity of arterial SMC membrane (from 1.93±0.04 in control up to 3.49±0.92 poise) which reflects a decrease in fluidity of SMC membrane. Lpo level was higher in plaque area (113.70±46.14 nmol/g protein) than in non-plaque area (58.32±12.69 nmol/g protein). GSH-Px activity level was lower in plaque area (0.0052±0.0014 EU/g protein) than in non-plaque area (0.015+0.0014 EU/g protein). Microviscosity of SMC membrane was higher in plaque area (2.92±0.35 poise) than in non-plaque area (2.26±0.24 poise, p<0.02). By comparison, the rabbits received Rhaponticum Uniflorum DC and VE showed much lowering of Lpo level in arterial wall (down to 10.74±1.61 and 9.93±1.17 nmol/g protein) and decreasing of microviscosity (down to 2.05+0.45 and 2.08+0.50 poise) that is increasing of membrane fluidity of arterial SMC membrane, but GSH-Px activity in arterial wall was keeping at lower level (0.036±0.027 and 0.051±0.027 EU/g protein). The atheroma lesions develped in these two group animals were less severe and fewer in number.
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Booth, R. F. G., J. F. Martin, S. Moncada, and A. C. Honey. "IS ATHEROSCLEROSIS A DISEASE OF THE OUTSIDE OF ARTERIES?" In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643083.

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The early stages of atherosclerosis are characterised by: (i) Intimal infiltration by macrophages, (ii) Intimal smooth muscle cell (SMC) proliferation and (iii) Accumulation of cholesterol within the vessel wall. Many studies have suggested that these changes occur subsequent to endothelial cell (EC) damage and platelet deposition. We now describe a new animal model with characteristics of early human atherosclerosis but without observable EC damage or platelet deposition. 12 rabbits were separated into 2 groups; each was fed normal laboratory chow and one group was supplemented with lg/day cholesterol. At day 0, under anaesthesia, a silastic collar (internal volume 0.3ml) was placed around the left carotid artery. The collar was filled with whole blood and sealed without causing Constriction of the vessel. The right carotid arteries were sham operated. The vessels were replaced and the animals allowed to recover. After 14 days both carotids were perfuse-fixed in situ. The left carotid arteries were separated into:(A)A mid-region from within the collar,(B)A region proximal to the collar and (C)A distal region. These regions were subdivided for histology and scanning E.M. Medial/Intimal ratios of transverse sections of each region were analysed by computerised image analysis and shown below.Histology demonstrated an intense proliferation within 14 days which resembled early atherosclerosis. There was intimal monocyte infiltration and SMC proliferation and loss of SMC from the medial layer. Scanning E.M. revealed no observable EC damage or platelet adhesion. The lesion was highly focal and generally restricted to the collar region. Dietary cholesterol did not enhance proliferation but did cause foam cell formation. Sham operated vessels showed no significant atherosclerotic changes. Thus, early atherosclerotic lesions may be the result of damage to the outer layers of blood vessels.
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Henak, Corinne R., and Jeffrey A. Weiss. "Regional Hyperelastic Properties of Human Hip Cartilage." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80470.

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Cartilage material properties are necessary to make accurate predictions of cartilage stress during joint loading and to understand the effects of aging and disease on cartilage mechanics. Human hip cartilage material properties for loading situations that are relevant to activities of daily living are unavailable. Investigators have needed to use material properties from the joints of other animals as a substitute (e.g., [1]).
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Abdrakhmanov, S. K., E. E. Mukhanbetkaliyev, A. A. Sultanov, and S. B. Tyulegenov. "EVALUATION OF POST-VACCINAL IMMUNITY AGAINST FMD BROKEN DOWN BY AGE AND GENDER GROUPS IN THE REPUBLIC OF KAZAKHSTAN." In STATE AND DEVELOPMENT PROSPECTS OF AGRIBUSINESS. DSTU-PRINT, 2020. http://dx.doi.org/10.23947/interagro.2020.1.23-25.

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The article provides an analysis of monitoring of post-vaccination immunity against foot and mouth disease, stratified by age and gender groups. Evaluation of induced immunity broken down by age and gender groups is the main element of post-vaccination monitoring, which allows not only to assess the quality of vaccination, but also to determine the timing for achieving the necessary immunity among vaccinated animals.
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Reports on the topic "Animals ascarriers of disease"

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Vantassel, Stephen M., and Mark A. Klng. Wildlife Carcass Disposal. U.S. Department of Agriculture, Animal and Plant Health Inspection Service, July 2018. http://dx.doi.org/10.32747/2018.7207733.ws.

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Abstract:
Many wildlife management situations require the disposal of animal carcasses. These can include the lethal removal of wildlife to resolve damage or conflicts, as well as clean-up after mortalities caused by vehicle collisions, disease, oil spills or other natural disasters. Carcasses must be disposed of properly to protect public sensitivities, the environment, and public health. Improper disposal of carcasses can result in public outrage, site contamination, injury to animals and people, and the attraction of other animals that may lead to wildlife damage issues. Concern over ground water contamination and disease transmission from improper carcass disposal has resulted in increased regulation. Successful carcass disposal programs are cost-effective, environmentally sound, and protective of public health. In addition, disposal practices must demonstrate sensitivity to public perception while adhering to state and local guidelines. This publication discusses the range of options available for the responsible disposal of animal carcasses.
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Mengak, Michael T. Wildlife Translocation. U.S. Department of Agriculture, Animal and Plant Health Inspection Service, July 2018. http://dx.doi.org/10.32747/2018.7210105.ws.

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Abstract:
Many people enjoy wildlife. Nationwide, Americans spend over $144 billion annually on fishing, hunting, and wildlife-watching activities. However, wildlife is not always welcome in or near homes, buildings, or other property and can cause significant damage or health and safety issues. Many people who experience a wildlife conflict prefer to resolve the issue without harming the offending animal. Of the many options available (i.e., habitat modification, exclusion, repellents) for addressing nuisance wildlife problems, translocation—capturing and moving—of the offending animal is often perceived to be effective. However, trapping and translocating wild animals is rarely legal nor is it considered a viable solution by wildlife professionals for resolving most nuisance wildlife problems. Reasons to avoid translocating nuisance wildlife include legal restrictions, disease concerns, liability issues associated with injuries or damage caused by a translocated animal, stress to the animal, homing behavior, and risk of death to the animal. Translocation is appropriate in some situations such as re-establishing endangered species, enhancing genetic diversity, and stocking species in formerly occupied habitats. The main focus of this publication, however, is to address nuisance wildlife issues that may be commonly encountered by homeowners and nuisance wildlife control professionals.
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