Relevant bibliographies by topics / Anisocoria

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Anisocoria'

Author: Grafiati
Published: 4 June 2021
Last updated: 20 February 2023

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Journal articles on the topic "Anisocoria"

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Bakbak, Berker, and Şansal Gedik. "Anisocoria." Turkish Journal of Ophthalmology 42, no. 1 (December 1, 2012): 68–72. http://dx.doi.org/10.4274/tjo.42.s14.

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Williams, David. "Anisocoria." Companion Animal 12, no. 7 (September 2007): 79–82. http://dx.doi.org/10.1111/j.2044-3862.2007.tb00206.x.

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Falardeau, Julie. "Anisocoria." International Ophthalmology Clinics 59, no. 3 (2019): 125–39. http://dx.doi.org/10.1097/iio.0000000000000276.

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Kawasaki, Aki. "ANISOCORIA." CONTINUUM: Lifelong Learning in Neurology 15 (August 2009): 218–35. http://dx.doi.org/10.1212/01.con.0000348841.03232.bb.

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Sitzman, B. Todd, David L. Bogdonoff, Thomas P. Bleck, Burkhard F. Spiekermann, and Cherylee W. J. Chang. "Postoperative Anisocoria." Anesthesia & Analgesia 83, no. 3 (September 1996): 633–35. http://dx.doi.org/10.1097/00000539-199609000-00035.

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Sitzman, B. Todd, David L. Bogdonoff, Thomas P. Bleck, Burkhard F. Spiekermann, and Cherylee W. J. Chang. "Postoperative Anisocoria." Anesthesia & Analgesia 83, no. 3 (September 1996): 633–35. http://dx.doi.org/10.1213/00000539-199609000-00035.

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González Martín-Moro, J., B. Pilo de la Fuente, A. Clement Corral, Y. Fernández de Miguel, F. Gómez Sanz, and R. Cobo Soriano. "Everyday anisocoria: Anisocoria epidemiology in a secondary care setting." Archivos de la Sociedad Española de Oftalmología (English Edition) 87, no. 7 (July 2012): 206–15. http://dx.doi.org/10.1016/j.oftale.2012.04.008.

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Riddick, Frank A. "Cruise Ship Anisocoria." Annals of Internal Medicine 117, no. 1 (July 1, 1992): 95. http://dx.doi.org/10.7326/0003-4819-117-1-95_1.

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Lust, Karin. "Nebulizer-Induced Anisocoria." Annals of Internal Medicine 128, no. 4 (February 15, 1998): 327. http://dx.doi.org/10.7326/0003-4819-128-4-199802150-00041.

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Iosson, Neil. "Nebulizer-Associated Anisocoria." New England Journal of Medicine 354, no. 9 (March 2, 2006): e8. http://dx.doi.org/10.1056/nejmicm050851.

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Dissertations / Theses on the topic "Anisocoria"

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Brown, Daniel Allan. "Automated Pupillometry Following Sport-Related Concussion in National Level Rugby League Athletes." Thesis, Griffith University, 2018. http://hdl.handle.net/10072/380064.

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Background Sport-related concussion accounts for 9% of all sporting injuries. Within elite rugby league athletes, 17% will suffer a concussion in a three-year period. Although there is increasing knowledge of incidence and risk of SRC in rugby league, gaps regarding the optimal diagnostic and return to play protocols remain. The National Rugby League requires teams to follow a strict concussion protocol, the Head Injury Assessment, which incorporates the Sport Concussion Assessment Tool (3rd ed). The effectiveness of the Sport Concussion Assessment Tool 3rd Edition protocol, along with existing cognitive assessment tools to diagnose and predict a return to play have been disputed, which has highlighted the need for objective biomarkers. The pupil light reflex is an autonomic nervous system function that occurs when there is a light stimulus upon the pupil. Emerging evidence has been suggested that pupil light reflex may be useful as a potential objective physiological biomarker for neuroanatomical pathway disruption. The aim of the present study was to determine whether there was a change in the pupil light reflex and anisocoria (pupil asymmetry) following a sport-related concussion in national level rugby league athletes. Aims and Objectives The study aimed to determine whether a change in the PLR and anisocoria is detectable following an acute sport-related concussion in national level rugby league athletes. To achieve this, comparisons of the PLR, anisocoria, variability and time-frame variations between concussed and non-concussed athletes were undertaken. Methods Fifty-five male volunteer athletes (age: 23 ± 4.5 years) were recruited from a local National Rugby League club. Eight pupil light reflex parameters were measured via a handheld monocular pupillometer during the rugby league pre-season. During the 2017 National Rugby League season, the pupil light reflex of nine athletes without a sport-related concussion diagnosis was monitored. Sixteen athletes were diagnosed with sport-related concussion and the pupil light reflex was recorded over a ten-day time frame, and broken down to three periods, 0 – 3 days, 4 – 6 days and 7 – 10 days, to measure recovery. Absolute change scores between the pupil light reflex parameter values were calculated for each athlete. Independent t-tests and Mann-Whitney U tests were used to test the study objectives. Results The neurological pupil index was significantly lower in the sport-related concussion group compared to the no-sport-related concussion (p = 0.0002). There was no statistical difference in the additional seven pupil light reflex parameters between the two groups. Given the variability within sport-related concussion and no-sport-related concussion athletes, absolute change scores were calculated to determine difference between groups. The absolute change score for resting pupil diameter (p = 0.001) and minimum pupil diameter (p < 0.0001) were statistically larger for athletes in the sport-related concussion group compared to no-sport-related concussion. No significant differences were found for the remaining six PLR parameters between groups. Throughout the acute phase of sport-related concussion, resting pupil diameter and minimum pupil diameter were found to be statistically different at 0 – 3 days, 4 – 6 days and 7 – 10 days following a sport-related concussion (p < 0.05). Maximum constriction velocity was statistically different at 0 – 3 days (p = 0.04) following a sport-related concussion, however no difference was observed at the remaining time frames (p > 0.05). Conclusion The pupil light reflex has previously been observed to have a statistical difference between control groups and mild traumatic brain injury groups. The present study found scientifically significant changes in the pupil light reflex following acute sport-related concussion in national level rugby league athletes compared to non-concussed athletes. The neurological pupil index for athletes with sport-related concussion were found to be statistically smaller than non-concussed athletes. Similar to previous research investigating mild traumatic brain injuries, the resting and minimum pupil diameter were found to be significantly different between a sport-related concussion group and non-concussion group. Results of the present study suggests the pupil light reflex may provide an objective physiological biomarker for diagnosis and recovery monitoring of an acute sport-related concussion in national level rugby league athletes. The change in the pupil light reflex observed in the present study suggest the potential application within the diagnosis and recovery monitoring of an acute sport-related concussion in national level rugby league athletes.
Thesis (Masters)
Master of Medical Research (MMedRes)
School of Medical Science
Griffith Health
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Haus, Sebastian. "Anisokorie im Schockraum – Prognosefaktoren für das klinische Outcome." Doctoral thesis, 2017. http://hdl.handle.net/11858/00-1735-0000-0023-3E04-7.

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Books on the topic "Anisocoria"

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Misulis, Karl E., and E. Lee Murray. Approach to Visual Deficits. Edited by Karl E. Misulis and E. Lee Murray. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190259419.003.0006.

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The most common visual complaints seen by the hospital neurologist are visual loss, diplopia, papilledema, anisocoria, and ptosis. The differential diagnosis of these and other ocular presentations is discussed.
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Book chapters on the topic "Anisocoria"

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D’Souza, Neil M., Sumayya J. Almarzouqi, Michael L. Morgan, and Andrew G. Lee. "Drugs; Anisocoria." In Encyclopedia of Ophthalmology, 1–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-35951-4_1183-1.

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Falco, Jeff, Sumayya J. Almarzouqi, and Andrew G. Lee. "Physiologic Anisocoria." In Encyclopedia of Ophthalmology, 1–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-35951-4_1297-1.

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DeBacker, Danielle L., Andrew R. Davis, Sumayya J. Almarzouqi, and Andrew G. Lee. "Pharmacologic Anisocoria." In Encyclopedia of Ophthalmology, 1–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-35951-4_1301-1.

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D’Souza, Neil M., Sumayya J. Almarzouqi, Michael L. Morgan, and Andrew G. Lee. "Drugs: Anisocoria." In Encyclopedia of Ophthalmology, 661–62. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-540-69000-9_1183.

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Falco, Jeff, Sumayya J. Almarzouqi, and Andrew G. Lee. "Physiologic Anisocoria." In Encyclopedia of Ophthalmology, 1378–79. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-540-69000-9_1297.

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DeBacker, Danielle L., Andrew R. Davis, Sumayya J. Almarzouqi, and Andrew G. Lee. "Pharmacologic Anisocoria." In Encyclopedia of Ophthalmology, 1364–65. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-540-69000-9_1301.

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Rahman, Effie Z., Angelina Espino Barros Palau, and Andrew G. Lee. "Anisocoria: Big Pupil." In Encyclopedia of Ophthalmology, 1–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-35951-4_526-1.

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Rahman, Effie Z., Angelina Espino Barros Palau, and Andrew G. Lee. "Anisocoria: Big Pupil." In Encyclopedia of Ophthalmology, 123–26. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-540-69000-9_526.

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Morgan, Michael L., Sumayya J. Almarzouqi, and Andrew G. Lee. "Anisocoria of the Small Pupil." In Encyclopedia of Ophthalmology, 1–4. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-35951-4_1159-1.

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Morgan, Michael L., Sumayya J. Almarzouqi, and Andrew G. Lee. "Anisocoria of the Small Pupil." In Encyclopedia of Ophthalmology, 120–23. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-540-69000-9_1159.

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Conference papers on the topic "Anisocoria"

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Alves, Pedro Vinicius Brito, Coralia Gabrielle Vieira Silveira, Jorge Fernando de Miranda Pereira, Isabela Fonseca Risso, Paulo Eduardo Lahoz Fernandez, Victoria Veiga Ribeiro Gonçalves, Paula Baleeiro Rodrigues Silva, et al. "Horner`s Syndrome after internal jugular vein catheterization: a case report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.322.

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Context: Central venous catheterization of the internal jugular vein is a common procedure that can be complicated with Horner`s Syndrome, caused by a direct lesion of cervical sympathetic pathways, pneumothorax compression, or carotid dissection. This entity should be considered when assessing new anisocoria in intensive care scenarios. Case Report: We report the case of a 64-year-old woman, who presented anisocoria during an intensive care unit hospitalization. She had been admitted with severe COVID-19 and need for mechanical ventilation. Her anisocoria was more evident in the dark, with right miosis, ipsilateral semi-ptosis, and preserved photoreaction reflexes. Before the anisocoria, she had a venous catheter inserted in her right jugular vein. Further evaluation showed a right pneumothorax, which was promptly drained after the mispuncture. The cervical arterial angiotomography showed no signs of carotid dissection. Conclusion: New anisocoria in critical patients is usually associated with impairment of the parasympathetic tonus, either by the use of topic or inhalatory anticholinergic drugs or cerebral herniation syndrome. However, in these situations, the anisocoria is more appreciable in light, with disruption of photoreaction in the greater pupil and ipsilateral ptosis. Therefore, we believe our patient developed an iatrogenic Horner`s Syndrome, secondary to a catheterization mispuncture, leading to a direct lesion of sympathetic pathways and their compression by the pneumothorax. Beyond parasympathetic pharmacologic blockade or cerebral herniation, Horner`s Syndrome constitutes a valuable differential diagnosis when evaluating patients with new anisocoria in the ICU.
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Miyahira, Clara Kimie, Beatriz Medeiros Correa, Raphael Palomo Barreira, Thomas Zurga Markus Torres, Natália Figueiredo Miranda, Natasha Soares Cutolo, Thiago da Cruz Marques, Vanessa Moraes Rossette, and Eduardo de Almeida Guimarães Nogueira. "Artery of Percheron territory infarct in a young patient with thrombophilia." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.026.

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Context: Stroke, when affecting territories dependent on posterior circulation, affects the thalamus and mesencephalic structures, regions irrigated by the paramedian arteries and perforating arteries of the posterior communicating artery and posterior cerebral artery. However, there is an anatomical variant of the paramedian arteries, the Percheron artery, when occluded causes bilateral thalamic infarction. Case report: a 53-year-old woman who presented blurred vision, holocranial headache, hypertensive peak, decreased level of consciousness, anisocoria, conjugate eye deviation. Tomography was normal and after 48 hours there was periventricular hypodensity of white matter, corona radiate and bilateral thalamic. Cerebral angiography confirmed moderate stenosis in the cavernous segments of the internal carotid arteries. In the investigation, a heterozygous mutation for factor V of Leiden was found. The patient evolved well, after blood pressure control and secondary prophylaxis. Clinical picture brings the classic triad of Percheron Artery infarction: decreased level of consciousness, vertical changes in the look and cognitive changes in memory and learning. Other symptoms could be found hypersomnia, oculomotor paralysis and ataxia. The diagnosis is clinical and radiological by visualizing bilateral thalamic involvement. Predisposing factors should be investigated, such as the Leiden factor V mutation, which increases thromboembolic events. Conclusion: It is important to research risk factors and execute an early diagnosis for acute intervention and prophylaxis of stroke.
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Darella, Amanda Xavier, Stefany Elias, and Gladys Lentz Martins. "Aortic dissection and stroke associated with COVID-19: case report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.242.

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Case Report A 58-year-old man, previously healthy, presented with headache, myalgia, retrosternal chest pain associated with paresthesia and paresis in upper limbs and lower limbs of acute onset. He denies fever, cough, dyspnea, or contact with patients with COVID-19. There was a difference in blood pressure and pulse in the upper limbs. No particularities in the neurological examination were noted. The electrocardiogram and enzymes of myocardial necrosis did not show abnormalities. PCR for SARS-CoV-2 was positive. Chest computed angiotomography showed acute type IA aortic dissection. The patient underwent cardiac surgery, without complications. After 48 hours of the procedure, the patient progresses with a lower level of consciousness, left hemiplegia, and anisocoria. The brain computed tomography showed extensive ischemic injury in the right middle cerebral artery territory. The patient underwent decompression craniectomy. The patient was discharged from the hospital, maintaining hemiplegia on the left side of the body. Discussion SARS-CoV-2 is a RNA virus responsible for the current COVID-191 pandemic. Moderate to severe forms of the disease may present with acute respiratory distress syndrome (ARDS), myocarditis, and thrombotic events such as pulmonary venous thromboembolism and ischemic stroke2. There are few reports in the literature about acute aortic dissection in patients with COVID-193,4. Acute aortic dissection is characterized by rupture of the intimal layer of the vessel with exposure of the middle layer and cystic necrosis and formation of a false lumen5. The mechanisms associated with thromboembolic phenomena in SARSCoV-2 infection remain poorly elucidated in the literature. This case report highlights a patient with severe complications of COVID-19, with the viral trigger being a possible contributor to the condition of acute aortic dissection and stroke.
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